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Libros sobre el tema "Generic peptide drug"

Autor: Grafiati
Publicado: 10 de marzo de 2023

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1

NATO Advanced Research Workshop on Advanced Drug Delivery Systems for Peptides and Proteins (1986 Copenhagen, Denmark). Delivery systems for peptide drugs. New York: Plenum Press, 1986.

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2

Proteins, enzymes, genes: The interplay of chemistry and biology. New Haven, CT: Yale University Press, 1999.

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3

Jun-Lin, Guan, ed. Signaling through cell adhesion molecules. Boca Raton, Fla: CRC Press, 1999.

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4

Davis, S. S. Delivery Systems for Peptide Drugs. Springer, 2013.

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5

Tomlinson, E., Lisbeth Illum y S. S. Davis. Delivery Systems for Peptide Drugs. Springer London, Limited, 2013.

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6

Delivery Systems for Peptide Drugs (Nato Asi Series a, Life Sciences, Vol 125) (Nato Science Series: A:). Springer, 1987.

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7

Dhagat, Swasti, Jujjvarapu Satya Eswari y Manisha Yadav. Computer-Aided Design of Antimicrobial Lipopeptides As Prospective Drug Candidates. Taylor & Francis Group, 2019.

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8

Dhagat, Swasti, Jujjvarapu Satya Eswari y Manisha Yadav. Computer-Aided Design of Antimicrobial Lipopeptides As Prospective Drug Candidates. Taylor & Francis Group, 2019.

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9

Dhagat, Swasti, Jujjvarapu Satya Eswari y Manisha Yadav. Computer-Aided Design of Antimicrobial Lipopeptides As Prospective Drug Candidates. Taylor & Francis Group, 2019.

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10

Jujjavarapu, Satya Eswari, Swasti Dhagat y Manisha Yadav. Computer-Aided Design of Antimicrobial Lipopeptides As Prospective Drug Candidates. Taylor & Francis Group, 2021.

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11

Dhagat, Swasti, Jujjvarapu Satya Eswari y Manisha Yadav. Computer-Aided Design of Antimicrobial Lipopeptides As Prospective Drug Candidates. Taylor & Francis Group, 2019.

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12

Soloviev, Mikhail, Chris Shaw y Per Andrén. Peptidomics: Methods and Applications. Wiley-Interscience, 2007.

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13

The LDN book: How a little-known generic drug, low dose naltrexone, could revolutionize treatment for autoimmune diseases, cancer, autism, depression, and more. 2016.

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14

Wójcik-Gładysz, Anna. Ghrelin – hormone with many faces. Central regulation and therapy. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_awg_2020.

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Discovered in 1999, ghrelin, is one of the peptides co-creating the hypothalamicgastrointestinal axis, otherwise known as the brain-gut axis. Ghrelin participates in many physiological processes and spectrum of its activity is still being discovered. This 28 amino acid peptide ‒ a product of the ghrl gene, was found in all vertebrates and is synthesized and secreted mainly from enteroendocrine X/A cells located in the gastric mucosa of the stomach. Expression of the ghrelin receptor has been found in many nuclei of the hypothalamus involved in appetite regulation. Therefore it’s presumed that ghrelin is one of the crucial hormones deciphering the energy status required for the maintenance of organism homeostasis. Ghrelin acts as a signal of starvation or energy insufficiency and its level in plasma is reduced after the meal. Neuropeptide Y (NPY) and agouti-related peptide (AgRP; NPY/AgRP) neurons located in the arcuate nucleus (ARC) area are the main target of ghrelin in the hypothalamus. This subpopulation of neurons is indispensable for inducing orexigenic action of ghrelin. Moreover ghrelin acting as a neurohormone, mainly in the hypothalamus area, plays an important role in the regulation of growth and reproduction processes. Indeed, ghrelin action on reproductive processes has been observed in the systemic effects exerted at both hypothalamus-pituitary and gonadal levels. Similarly the GH-releasing ghrelin action was observed both on the hypothalamus level and directly on the somatotrophic cells in the pituitary and this dose-related GH releasing activity was found in in vitro as well as in in vivo experiments. In recent years, numerous studies revealed that ghrelin potentially takes part in the treatment of diseases associated with serious disturbances in the organism energy balance and/or functioning of the gastrointestinal tract. It was underlined that ghrelin may be a hormone with a broad spectrum of therapeutic effect on obesity and anorexia nervosa, as well as may also have protective effect on neurodegenerative diseases, inflammatory disorders or functional changes in the body caused by cancers. In overall, ghrelin treatment has been tested in over 100 preclinical studies with healthy volunteers as well as patients with various types of cancer, eating disorders such as anorexia nervosa and bulimia nervosa. It was observed that ghrelin has an excellent clinical safety profile and emerging side effects occurred only in 3–10% of patients and did not constitute a sufficient premise to discontinue the therapy. In general, it can be concluded that ghrelin may be sufficiently used as a prescription drug.
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15

Peptidomics: Methods and applications. Hoboken, N.J: John Wiley & Sons, 2007.

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16

(Editor), A. M. Egorov y Gennady Zaikov (Editor), eds. New Research on Biotechnology And Medicine. Nova Biomedical Books, 2006.

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17

Ryutaro, Utsumi, ed. Bacterial signal transduction: Networks and drug targets. New York: Springer Science+Business Media, 2008.

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18

Utsumi, Ryutaro. Bacterial Signal Transduction: Networks and Drug Targets. Springer London, Limited, 2008.

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19

Schulkin, Jay. The CRF Signal. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780198793694.001.0001.

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This book discusses just how diverse a peptide corticotrophin-releasing factor (CRF) is, as demonstrated by its presence in various tissues in the body, including the skin, the placenta, and various regions of the brain. As Dobzhansky (1962) noted, in light of Darwin (1874), and beyond, CRF must be placed in the larger world of regulatory biology. Evolutionary trends do not proceed in a continuous one-dimensional direction; there are starts, turns, and abrupt ends. The study of CRF is mostly about diverse functions in physiological and behavioral regulation of the internal milieu and adapting to an ecological and or social context. The book begins with a depiction of the evolutionary origins of CRF in living things, dating back hundreds of millions of years. The book pushes the conception of CRF beyond the HPA axis and common knowledge. We study the role of CRF in metamorphosis and parturition. Further, CRF is a contributor to fear and anxiety, and the book explains how excessive fear is tied to anxiety disorders and vulnerability to the breakdown of mental and physical health. Also discussed is CRF in approach/avoidance behaviors across pre- and postnatal events. CRF is intimately involved in organ development, but it is also linked to devolution of function and conditions of danger. Cravings, addictions, and how CRF is tied both to the ingestion of diverse drugs and to withdrawal are explored. CRF is considered as an epistemic object, addressing what constitutes an information molecule, in general, and CRF, in particular.
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20

Septic Shock: Methods and Protocols (Methods in Molecular Medicine). Humana Press, 2000.

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21

Guan, Jun-Lin. Signaling Through Cell Adhesion Molecules. Taylor & Francis Group, 2019.

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22

Guan, Jun-Lin. Signaling Through Cell Adhesion Molecules. Taylor & Francis Group, 2019.

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23

Guan, Jun-Lin. Signaling Through Cell Adhesion Molecules. Taylor & Francis Group, 2019.

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