Tesis sobre el tema "ERM PROTEINS"
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Hebert, Alan. "Functional Relationship between Merlin and the ERM Proteins". Thesis, Harvard University, 2012. http://dissertations.umi.com/gsas.harvard:10567.
Texto completoMachicoane, Michaël. "The role of ERM proteins in cell division". Paris 6, 2013. http://www.theses.fr/2013PA066285.
Texto completoThe control of cell division axis is crucial for embryogenesis, cell differentiation and adult tissue homeostasis, and relies on the cortical localization of protein complexes able to pull on the mitotic apparatus. One of these force generators is the evolutionary conserved Gαi / LGN / NuMA module, which recruits Dynein motors at the cortex. On top of this, intrinsic and extrinsic cues can then modulate the activity and localization of this complex. Particularly, the F-actin cortex has recently been involved in the dialogue between astral microtubules and force generators. Identifying the proteins involved in F-actin organization at the cortex is thus a major challenge. During my thesis work, I focused my attention on the proteins of the ERM (Ezrin-Radixin-Moesin) family. I investigated the role of these membrane-actin linkers in the orientation of the mitotic spindle during oriented cell division. Our work demonstrated that ERM are strongly and directly activated by the SLK kinase at mitotic entry in mammalian cells. Using micro-fabricated adhesive substrates to control the axis of cell division, we found that the activation of ERM plays a key role in guiding the orientation of the mitotic spindle. Accordingly, impairing ERM activation in apical progenitors of the mouse embryonic neocortex severely disturbed spindle orientation in vivo. At the molecular level, ERM activation promotes the polarized association at the mitotic cortex of NuMA. We propose that F-actin and activated ERM at the mitotic cortex are critical for the correct localization of force generator complexes and hence for proper spindle orientation
Matsui, Takeshi. "Activation of ERM (ezrin/radixin/moesin) proteins by the small GTP binding protein Rho". Kyoto University, 2000. http://hdl.handle.net/2433/180838.
Texto completoBissen, Philippe [Verfasser] y Volker [Akademischer Betreuer] Gerke. "ERM proteins - regulators of mitosis? / Philippe Bissen ; Betreuer: Volker Gerke". Münster : Universitäts- und Landesbibliothek Münster, 2019. http://d-nb.info/1181188873/34.
Texto completoZarrouk, Marouan. "The role of ezrin/radixin/moesin(ERM) proteins in L-selectin-dependent signalling". Thesis, Imperial College London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.510756.
Texto completoOlsson, Per-Anders. "MIR, a novel ERM-like protein in the nervous system". Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5061-X/.
Texto completoLegg, James William. "Regulation of the hyaluronan receptor CD44 by the ezrin/radixin/moesin (ERM) family of proteins". Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392688.
Texto completoHayashi, Ken. "Immunofluorescence detection of ezrin/radixin/moesin (ERM) proteins with their carboxyl-terminal threonine phosphorylated in cultured cells and tissues : application of a novel fixation protocol using trichloroacetic acid (TCA) as a fixative". Kyoto University, 1999. http://hdl.handle.net/2433/181750.
Texto completoBRUNO, MARINA. "ROLE OF BIOACTIVE SPHINGOLIPIDS IN INNER EAR AND SKELETAL MUSCLE BIOLOGY". Doctoral thesis, Università di Siena, 2019. http://hdl.handle.net/11365/1072579.
Texto completoTan, Yu Pei. "The development of Lactococcus lactis as an antimicrobial agent". Thesis, Queensland University of Technology, 2010. https://eprints.qut.edu.au/39143/1/Yu_Pei_Tan_Thesis.pdf.
Texto completoGründel, Anne. "Funktion glykolytischer Enzyme von Mycoplasma pneumoniae in der Wirt-Erreger-Interaktion". Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-213500.
Texto completoLo, Kin Ho. "Activation of signal transducer and activator of transcription 3 (STAT3) by G[alpha]16 and G[alpha]14 via a c-Src/JAK-and ERK-dependent mechanism /". View abstract or full-text, 2004. http://library.ust.hk/cgi/db/thesis.pl?BICH%202004%20LO.
Texto completoIncludes bibliographical references (leaves 92-111). Also available in electronic version. Access restricted to campus users.
Owen, Jo. "Structural and functional studies of fibulin-1 EGF domains". Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270656.
Texto completoPanaganti, Shilpa. "Parallel SVM with Application to Protein Structure Prediction". Digital Archive @ GSU, 2004. http://digitalarchive.gsu.edu/cs_theses/3.
Texto completoEl, Sabeh Rana. "Pleiotropism of MyD88, as Determined by its Multiple Protein-Protein Interactions". Thesis, Lyon 1, 2014. http://www.theses.fr/2014LYO10168.
Texto completoMyD88 is a protein that is at the interface between inflammation and cancer. It is the key adaptor protein used by TLRs/IL-1R to mediate their downstream signaling, resulting in NF-κB and MAPK activation, and inflammatory cytokine production. MyD88 also plays a role in tumorigenesis via two mechanisms, an inflammatory one dependent on its function in TLRs/IL- 1R signaling, and an intrinsic, cell-autonomous mechanism mediated by its interaction with the kinase Erk. Based on the different roles played by MyD88, this thesis work consisted in studying how MyD88 protein-protein interactions can regulate its function. We show that MyD88 interacts with Ubc9, resulting in its sumoylation and subsequent negative regulation of MyD88- mediated inflammation. We also demonstrate that MyD88 interacts with the nuclear receptor ER-α, an interaction necessary for the inflammatory response. Finally, we have studied the importance of the MyD88/Erk interaction in the maintenance of the transformed phenotype of Ras-dependent tumors. These findings could eventually be exploited to target MyD88 and its interactions in the treatment of inflammatory disorders and cancer
Pust, Sascha. "Einfluss der ERM-Proteine auf die Protrusion-Ausbildung und Zell-Zell-Ausbreitung von Listeria monocytogenes". [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=967112273.
Texto completoDoarn, Michael C. "Altered differentiation in acute myeloid leukemias; role of ERG and FUS-ERG fusion protein". Connect to this title online, 2005. http://hdl.handle.net/1811/373.
Texto completoTitle from first page of PDF file. Document formattted into pages: contains, 45 p.; also includes graphics. Includes bibliographical references (p. 42-45). Available online via Ohio State University's Knowledge Bank.
Samuels, Ivy S. "The roles of ERK₁ and ERK₂ MAP kinase in neural development and disease". Cleveland, Ohio : Case Western Reserve University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=case1214495630.
Texto completoGandhi, Neha Sureshchandra. "Molecular modelling of the interactions of complex carbohydrates with proteins". Thesis, Curtin University, 2011. http://hdl.handle.net/20.500.11937/2027.
Texto completoLubart, Quentin. "Les protéines ERM , Interactions entre la membrane cellulaire et le cytosquelette : une approche biomimétique". Thesis, Université Grenoble Alpes (ComUE), 2016. http://www.theses.fr/2016GREAI108/document.
Texto completoERM (ezrin, radixin, moesin) proteins play a central role in cellulo in a large number of physiological and pathological processes, including cell infection, migration and cell division. Among the ERMs, moesin is particularly involved in the formation of the immunological synapse, viral and bacterial infection, and cancer metastasis. From a structural point of view, ERMs can be in inactive (closed) conformation or active (open), which enable them to interact on one side with the cytoskeleton (actin and tubulin) via their C-terminal domain and on the other side with the plasma membrane via their FERM domain. Binding to the plasma membrane is mediated via a specific lipid of the phosphoinositide family, the phosphatidylinositol(4,5)bisphosphate (PIP2). In addition, ERM can be phosphorylated, which contribute to their structural opening. To date, the role of the phosphorylation in ERM/membrane and ERM/cytoskeleton interactions, although widely studied in cellulo, remains poorly understood at the molecular level.The aim of this PhD thesis is precisely to study, at the molecular level and using biomimetic systems, interactions between recombinant proteins and biomimetic membranes containing PIP2. To this end, we have engineered lipid membranes in the form of large and small unilamellar vesicles and supported lipid bilayers. These biomimetic membranes are used to characterize interactions between proteins and membranes by complementary biophysical techniques, notably quantitative cosedimentation, fluorescence microscopy and spectroscopy, and quartz crystal microbalance with dissipation monitoring. In a first part, we studied the role of double phosphorylation on moesin, achieved via a site-specific mutation on threonine residues, on moesin/biomimetic membrane interactions, in comparison to the wild type protein. The recombinant proteins and mutants were produced in our laboratory.Our results show that there is a specific and cooperative interaction for the double phosphomimetic mutant while interactions is 1:1 in the case of the wild type protein. In a second part, we used supported lipid bilayers containing PIP2 to study the molecular adsorption mechanism of the viral protein Gag and of its mutants. The methodologies that were developed in this work open perspectives in molecular biophysics since they are easily adaptable to other proteins on model lipid membranes containing phosphoinositidesKeywords: Ezrin-Radixin-Moesin, phosphoinositides, PIP2, protein/lipid interactions, biomimetic lipid membrane, Gag viral protein, cytoskeleton
McCosker, Helen Clare. "Prognostic significance of IGF and ECM induced signalling proteins in breast cancer patients". Thesis, Queensland University of Technology, 2012. https://eprints.qut.edu.au/53580/1/Helen_McCosker_Thesis.pdf.
Texto completoRobinson, J. D. "G protein-coupled receptor kinase 2 is a Rho-dependent scaffold protein for the ERK MAPK cascade". Thesis, University College London (University of London), 2011. http://discovery.ucl.ac.uk/1336072/.
Texto completoPaunola, Eija. "Protein folding before and efter translocation into the yeast endoplasmic reticulum". Helsinki : University of Helsinki, 2000. http://ethesis.helsinki.fi/julkaisut/eri/biote/vk/paunola/.
Texto completoDvořák, Pavel. "Biomedicínské aplikace polykaprolaktonových nanovlákenných membrán". Master's thesis, Vysoké učení technické v Brně. Fakulta chemická, 2021. http://www.nusl.cz/ntk/nusl-444549.
Texto completoMazzetti, Scott A. "Akt and ERK activation in human skeletal muscle : dose-dependency of responses to increasing muscle contractions". Virtual Press, 2003. http://liblink.bsu.edu/uhtbin/catkey/1259313.
Texto completoHuman Performance Laboratory
Van, Kanegan Michael J. "Regulation of nerve growth factor signaling by protein phosphatase 2A". Diss., University of Iowa, 2008. https://ir.uiowa.edu/etd/279.
Texto completoLam, King-yin Andy. "Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling". Click to view the E-thesis via HKUTO, 2010. http://sunzi.lib.hku.hk/hkuto/record/B44251014.
Texto completoLam, King-yin Andy y 林敬賢. "Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44251014.
Texto completoSUWASTIKA, I. NENGAH. "Plant Specific Subcellular Targeting mechanism: studies on SYP 7s, Qc-SNARE Family Proteins, and on Plant Obg-Era Homologue of small GTPase Proteins". 京都大学 (Kyoto University), 2009. http://hdl.handle.net/2433/123813.
Texto completoGiollo, Manuel. "Computational Approaches to Address the Next-Generation Sequencing Era". Doctoral thesis, Università degli studi di Padova, 2015. http://hdl.handle.net/11577/3424280.
Texto completoIn questa tesi, vengono proposti nuovi algoritmi e modelli per affrontare problemi biologici. L'informatica svolge un ruolo chiave nella proteomica e nella ricerca genetica dovuto alla gestione delle grandi moli di dati biologici. Nel contesto dello studio di proteine, ho sviluppato nuovi metodi per la predizione delle loro funzioni basati su principi di reperimento dell'informazione. Utilizzando fonti eterogenee di conoscenza, come la ricerca su grafi e la similarità di sequenze, ho progettato uno strumento chiamato INGA che può essere utilizzato per annotare interi genomi. Questo è stato valutato imparzialmente dal Critical Assessment of Function Annotation, e ha dimostrato di essere uno degli approcci più efficaci per l'inferenza di funzione. Per meglio caratterizzare le proteine dal punto di vista strutturale, ho proposto una strategia di rilevamento delle conformazioni delle proteine basata su rete di interazione di residui (RIN). Le reti RIN sono state quindi estese per gestire le fluttuazioni temporali delle coordinate atomiche. Tali grafi sono stati infine generati automaticamente da algoritmi di clustering. Un'implementazione chiamata RING MD ha evidenziato efficacemente i principali amminoacidi noti per essere funzionalmente rilevanti nell'Ubiquitina. Questi aminoacidi sono infatti molto importanti per spiegare la dinamica strutturale della proteina. Con la stessa logica, sono stati usati i grafi RIN anche per prevedere l'impatto delle mutazioni all'interno di una struttura proteica. Combinando informazioni sul nodo mutante in una rete e le sue caratteristiche, una rete neurale artificiale è stata addestrata per stimare la variazione di energia libera di Gibbs all'interno di una proteina. Cambiamenti estremi nell'energia interna potrebbe portare all'unfolding della proteina, ed eventualmente ad una malattia. D'altro canto, anche la riduzione della flessibilità proteica può ostacolare la sua funzione. Ad esempio, le fluttuazioni estreme osservate nelle proteine intrinsecamente disordinate (IDP) sono fondamentali per le loro attività. Per studiare le IDP, ho contribuito alla raccolta del più grandi dataset di regioni disordinate mai esistito. Nella seguente analisi è stato dimostrato quali sono le funzioni tipiche di queste sequenze e i processi biologici in cui sono coinvolte. Data l'importanza della loro identificazione, una valutazione globale di predittori del disordine è stata eseguita per mostrare quali sono i metodi più efficaci e le loro limitazioni. Nel contesto della genetica, mi sono concentrato sulla previsione di fenotipi. Durante il Critical Assessment of Genome Interpretation (CAGI), ho proposto nuovi approcci per l'analisi dei dati dell'esoma progettati per valutare il rischio di morbo di Crohn e di ipercolesterolemia. Queste sono spesso definite come malattie complesse, dal momento che il meccanismo alla base della loro insorgenza è ancora sconosciuto. Nel mio studio, i campioni umani con un arricchimento di mutazioni in geni critici sono stati predetti come soggetti a rischio genetico elevato. Oltre ai geni associati alla malattia, le reti di interazione proteiche sono state considerate per valutare l'accumulo di varianti in pathway biologici. Tale strategia ha dimostrato di essere tra le migliori secondo gli organizzatori del CAGI. Nel caso più semplice dei tratti mendeliani, con BOOGIE ho progettato un metodo per la predizione dei gruppi sanguigni umani basata su dati di esoma. Esso utilizza una versione specializzata dell'algoritmo nearest neighbour al fine di far corrispondere le varianti genetiche in un esoma non annotato con quelle disponibili in una base di conoscenza di riferimento. L'esempio più simile è usato per trasferire il gruppo sanguigno. Con una precisione superiore al 90%, BOOGIE è un prototipo che mostra le potenziali applicazioni della predizione genetica, e può essere facilmente esteso a qualsiasi tratto mendeliano. Riassumendo, questa tesi è una risposta parziale alla crescita esponenziale di sequenze disponibili che necessitano ulteriori esperimenti. Integrando informazioni eterogenee e la progettazione di nuovi modelli predittivi basati su apprendimento automatico, ho sviluppato nuovi strumenti per l'analisi di dati biologici e per la loro classificazione. Tutte le implementazioni sono liberamente disponibili per la comunità e potrebbero essere utili durante indagini future come in studi di malattie e nella progettazione di farmaci.
López, Ceballos Pablo. "Elucidating how protein turnover in cell-ECM adhesion stabilizes tissue structure during development". Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/57622.
Texto completoMedicine, Faculty of
Graduate
Leugers, Chad Jeremy. "Modulation of growth factor-induced ERK signaling by the microtubule associated protein tau". Diss., University of Iowa, 2010. https://ir.uiowa.edu/etd/541.
Texto completoOsta, Muhammad Samir Ahmed. "Characterisation of ECM protein processing mechanisms underlying simple peritoneal sclerosis and encapsulating peritoneal sclerosis". Thesis, University of Sheffield, 2015. http://etheses.whiterose.ac.uk/11406/.
Texto completoParisis, Nikolaos. "Identification of PAR-2-regulated ERK substrates and (Beta)-arrestin-interacting proteins in invasive breast cancer cells". Thesis, University of Essex, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520107.
Texto completoRomán, Pérez Erick. "The role of ERa, ERß and phytoestrogens from soy in P53-mediated response to DNA damage in mammary epithelium". Amherst, Mass. : University of Massachusetts Amherst, 2009. http://scholarworks.umass.edu/dissertations/AAI3372274/.
Texto completoOn title page, the 'a' in ERa is symbolized by the Greek symbol for alpha. Includes bibliographical references (p. 108-124). Print copy also available.
Conley, Travis B. "The influence of training status on ERK and AKT phosphorylation in human skeletal muscle". Virtual Press, 2005. http://liblink.bsu.edu/uhtbin/catkey/1319219.
Texto completoSchool of Physical Education, Sport, and Exercise Science
Lundberg, Ida. "Fibroblasts and ECM in colorectal cancer : Analysis of subgroup specific protein expression and matrix arrangement". Thesis, Umeå universitet, Patologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-85606.
Texto completoLee, Vivian Wing Yan. "Regulation of extracellular signal-regulated protein kinases (ERK) 1 and 2 in synaptic nerve terminals". Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1446422/.
Texto completoKitajima, Sakihito. "Molecular analysis of gene expression of tobacco osmotin-like protein and its transcription factor ERF". Kyoto University, 1999. http://hdl.handle.net/2433/181925.
Texto completoDurrett, Timothy P. "Understanding Arabidopsis ion homeostasis in the post-genomic era assigning function to two proteins involved in iron metabolism /". Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4437.
Texto completoThe entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on April 27, 2009) Vita. Includes bibliographical references.
Shim, Hoon. "THE ROLE OF R7 REGULATORS OF G PROTEIN SIGNALING IN THE RETINA". VCU Scholars Compass, 2012. http://scholarscompass.vcu.edu/etd/580.
Texto completoAzzam, Diana Galil. "Extracellular signal regulated kinase/mitogen activated protein kinase (ERK/MAPK) regulation of the androgen receptor in breast cancer cells". University of Western Australia. School of Pathology and Laboratory Medicine, 2008. http://theses.library.uwa.edu.au/adt-WU2009.0024.
Texto completoHodge, Jacob G. "Regulation of the MEK/ERK signaling cascade by ADAM12 in triple-negative breast cancer cells". Thesis, Kansas State University, 2016. http://hdl.handle.net/2097/35228.
Texto completoBiochemistry and Molecular Biophysics Interdepartmental Program
Anna Zolkiewska
Mitogen-activated protein kinase (MAPK) signaling plays an important role in the proliferation, survival, and therapy resistance of breast cancer cells. Two important protein kinases involved in the MAPK pathway are MEK and ERK. The MEK/ERK signaling cascade can be stimulated by activation of the epidermal growth factor receptor (EGFR) upon binding of EGF-like ligands, which are released from cells by ADAM proteases. EGFR is frequently overexpressed in triple-negative breast cancer (TNBC), a particularly aggressive form of breast cancer. Our analysis of clinical data revealed that high expression of ADAM12, but not other ADAMs, in TNBC is associated with poor patient survival. Thus, we hypothesized that ADAM12 plays a critical role in the progression of TNBC, possibly by stimulating MEK/ERK activity in an EGFR-dependent manner. To test this hypothesis, ADAM12 was knocked-down (KD) in SUM159PT TNBC cells, which express high levels of the endogenous ADAM12 protein. An antibody array assay indicated a significant decrease in the activation of the MAPK pathway in SUM159PT cells after ADAM12 KD. The decrease in MAPK activity was further confirmed by Western blotting using phospho-MEK and phospho-ERK specific antibodies. Additionally, conditioned media from ADAM12-deficient SUM159PT cells failed to support the survival of MCF10A cells, suggesting that ADAM12 KD reduced the release of pro-survival growth factors from SUM159PT cells. Based upon this data, we propose that ADAM12 is a novel regulator of the MAPK pathway and a potential therapeutic target in breast cancer.
Kisielnicka, Edyta. "SCF-mediated degradation of the two translational regulators, CPB-3 and GLD-1, during oogenesis in C. elegans". Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2018. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-234186.
Texto completoLeandro, Luciana de Oliveira. "Controle de qualidade em imuno-histoquimica: o modelo de deteccao da oncoproteina C-erB-2". Sao Paulo : [s.n.], 2004. http://10.188.1.43/lildbi/docsonline/5/2/125-Tese%5FCIP%5FLeandro,%5FLuciana%5FO%5F2004.pdf.
Texto completoSalery, Marine. "Rôle de la protéine Arc (Activity-regulated cytoskeleton-associated protein) dans les adaptations moléculaires et comportementales induites par la cocaïne". Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066400/document.
Texto completoMolecular and cellular adaptations induced by drugs of abuse in the reward system play a key role in long-term behavioral alterations encountered in addiction. This work falls within an approach of understanding the cellular processes rapidly engaged by cocaine that could underlie the persistent alteration of neuronal physiology and behaviors. Arc protein is a major player in neuronal plasticity. Arc is induced in many behavioral paradigms and is essential for long-term synaptic plasticity and memory consolidation. The aim of this study was to characterize the profile and modality of Arc induction within the mouse striatum in response to cocaine administration. Our study shows that Arc expression is rapidly and transiently increased in the striatum after acute cocaine in an ERK-dependent fashion. This work revealed that cocaine-induced Arc protein rapidly and transiently accumulates in the nucleus of striatal neurons. In the nucleus, Arc is preferentially expressed in active transcription regions and localizes at the vicinity of phosphorylated histones H3. In vitro Arc overexpression decreased glutamate-induced Histones H3 phosphorylation showing that Arc interferes with activity-dependent chromatin remodeling. In vivo genetic invalidation of Arc expression in a transgenic mouse model was associated with a decreased chromatin compaction and increased RNA Polymerase II activity suggesting a repressive role of Arc on transcriptional mechanisms. Total Arc loss of expression leads to increased sensitivity to cocaine and promotes long-term behavioral alterations induced by low doses of cocaine
Stevens, Payton D. "THE FUNCTION OF ERBIN, A SCAFFOLD PROTEIN, AS A TUMOR SUPPRESSOR IN COLON CANCER". UKnowledge, 2018. https://uknowledge.uky.edu/biochem_etds/36.
Texto completoLok, Tsz-mei y 駱芷薇. "Aberrant activation of ERK/FOXM1 signaling axis promotes cell migration/invasion in ovarian cancer". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45208104.
Texto completoChen, Xi. "The role of PI3K and ERK/MAPK signal transduction cascades in long-term memory formation /". Thesis, Connect to this title online; UW restricted, 2004. http://hdl.handle.net/1773/6248.
Texto completoNovotny, Marian. "Applications of Structural Bioinformatics for the Structural Genomics Era". Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis Acta Universitatis Upsaliensis, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7593.
Texto completo