Bibliografías temáticas / Enzyme activation / Libros

Libros sobre el tema "Enzyme activation"

Siga este enlace para ver otros tipos de publicaciones sobre el tema: Enzyme activation.
Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 5 de marzo de 2023

Crea una cita precisa en los estilos APA, MLA, Chicago, Harvard y otros

Elija tipo de fuente:

Consulte los 50 mejores mejores libros para su investigación sobre el tema "Enzyme activation".

Junto a cada fuente en la lista de referencias hay un botón "Agregar a la bibliografía". Pulsa este botón, y generaremos automáticamente la referencia bibliográfica para la obra elegida en el estilo de cita que necesites: APA, MLA, Harvard, Vancouver, Chicago, etc.

También puede descargar el texto completo de la publicación académica en formato pdf y leer en línea su resumen siempre que esté disponible en los metadatos.

Explore libros sobre una amplia variedad de disciplinas y organice su bibliografía correctamente.

1

Ottaway, J. H. Regulation of enzyme activity. Oxford: IRL Press, 1988.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
2

H, Stenmark, ed. Phosphoinositides in subcellular targeting and enzyme activation. Berlin: Springer, 2004.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
3

Stein, Ross L. Kinetics of enzyme action: Essential principles for drug hunters. Hoboken, N.J: John Wiley, 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
4

Stenmark, Harald, ed. Phosphoinositides in Subcellular Targeting and Enzyme Activation. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18805-3.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
5

Krupi͡anko, V. I. Vektornyĭ metod predstavlenii͡a fermentativnykh reakt͡siĭ. Moskva: "Nauka", 1990.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
6

Alan, Wiseman, ed. Enzyme induction, mutagen activation, and carcinogen testing in yeast. Chichester, West Sussex, England: E. Horwood, 1987.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
7

H, Schmid-Schönbein, Wurzinger L. J, Zimmermann R. E y Commission of the European Communities. Committee on Medical and Public Health Research., eds. Enzyme activation in blood-perfused artificial organs: Proceedings of an interdisciplinary meeting. Boston: Nijhoff, 1985.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
8

Mordechai, Liscovitch, ed. Signal-activated phospholipases. Austin: R.G. Landes Co., 1994.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
9

Akira, Arimura, Saʻīd Sāmī y International Symposium on VIP, PACAP, and Related Peptides (2nd : 1995 : New Orleans, La.), eds. VIP, PACAP, and related peptides: Second international symposium. New York: New York Academy of Sciences, 1996.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
10

Light, P. E. An investigation into factors controlling activation of the enzyme protein Kinase C in the regulation of neurotransmitter release at the frog neuromuscular junction. Birmingham: University of Birmingham, 1990.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
11

Chen, Chang-Hwei. Activation and Detoxification Enzymes. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1049-2.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
12

1923-, Lorand Laszlo y Mann Kenneth G, eds. Proteolytic enzymes in coagulation, fibrinolysis, and complement activation. San Diego: Academic Press, 1993.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
13

Rossomando, Edward F. HPLC in enzymatic analysis. 2a ed. New York: Wiley, 1998.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
14

M, Cunningham Fiona, ed. Lipid mediators. London: Academic Press, 1994.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
15

C, Murphy Robert y Fitzpatrick Frank A, eds. Arachidonate related lipid mediators. San Diego: Academic Press, 1990.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
16

L, Schramm Vern y Purich Daniel L, eds. Enzyme kinetics and mechanism. 1999.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
17

Enzyme kinetics and mechanism. San Diego: Academic Press, 2002.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
18

Stein, Ross L. Kinetics of Enzyme Action. Wiley & Sons, Incorporated, John, 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
19

Stenmark, Harald. Phosphoinositides in Subcellular Targeting and Enzyme Activation. Springer London, Limited, 2012.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
20

Stein, Ross L. Kinetics of Enzyme Action: Essential Principles for Drug Hunters. Wiley & Sons, Incorporated, John, 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
21

Stein, Ross L. Kinetics of Enzyme Action: Essential Principles for Drug Hunters. Wiley & Sons, Incorporated, John, 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
22

Stein, Ross L. Kinetics of Enzyme Action: Essential Principles for Drug Hunters. Wiley & Sons, Incorporated, John, 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
23

Stenmark, Harald. Phosphoinositides in Subcellular Targeting and Enzyme Activation. Springer, 2012.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
24

Stenmark, Harald. Phosphoinositides in Subcellular Targeting and Enzyme Activation. Springer, 2003.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
25

Engel, P. C. Enzymology LabFax (Labfax Series). Academic Press, 1996.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
26

Baici, Antonio. Kinetics of Enzyme-Modifier Interactions: Selected Topics in the Theory and Diagnosis of Inhibition and Activation Mechanisms. Baici Antonio, 2015.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
27

Baici, Antonio. Kinetics of Enzyme-Modifier Interactions: Selected Topics in the Theory and Diagnosis of Inhibition and Activation Mechanisms. Springer, 2016.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
28

Baici, Antonio. Kinetics of Enzyme-Modifier Interactions: Selected Topics in the Theory and Diagnosis of Inhibition and Activation Mechanisms. Springer London, Limited, 2015.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
29

Activation And Detoxification Enzymes Functions And Implications. Springer, 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
30

Cooper, A. The Enzyme Catalysis Process:Energetics, Mechanism and Dynamics. Springer, 1989.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
31

Cascorbi, Ingolf. Polymorphic cytochrome P450 2D6 as the responsible enzyme of activation. Editado por Paul Farquhar-Smith, Pierre Beaulieu y Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0079.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
Resumen
The landmark paper discussed in this chapter is ‘Bioactivation of the narcotic drug codeine in human liver is mediated by the polymorphic monooxygenase catalyzing debrisoquine 4-hydroxylation (cytochrome P-450 dbl/bufI)’, published by Dayer et al. in 1988. Codeine is an old but frequently prescribed drug used for the treatment of mild-to-moderate pain. However, its use is nowadays restricted after observations of partly fatal respiratory repression in children. Codeine itself exhibits no analgesic effect, but is partly activated by O-demethylation to morphine by cytochrome P450 2D6 (CYP2D6). The identification of the polymorphic CYP2D6 as the enzyme responsible for activation was achieved by Dayer et al. in 1998 and was an important milestone contributing to the widely observed inter-individual differences of drug action and side effects of codeine. Translating the pharmacogenetics of codeine into clinical practice is currently underway in clinical trials, to identify ineffective analgesics and, in particular, avoid severe adverse events.
32

Soda, Kenji y Toshio Fukui. Molecular Aspects of Enzyme Catalysis. Wiley & Sons, Incorporated, John, 2008.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
33

Molecular aspects of enzyme catalysis. Tokyo: Kodansha, 1994.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
34

Fukui, Tukui y Kenji Soda. Molecular Aspects of Enzyme Catalysis. Vch Pub, 1994.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
35

Soda, Kenji y Toshio Fukui. Molecular Aspects of Enzyme Catalysis. Wiley & Sons, Limited, John, 2007.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
36

Lyle, Stephen. The sulfate-activation pathway: A bifunctional enzyme which utilizes intermediate channeling. 1993.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
37

Clarke, Andrew. Temperature and reaction rate. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199551668.003.0007.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
Resumen
All other things being equal, physiological reaction rate increases roughly exponentially with temperature. Organisms that have adapted over evolutionary time to live at different temperatures can have enzyme variants that exhibit similar kinetics at the temperatures to which they have adapted to operate. Within species whose distribution covers a range of temperatures, there may be differential expression of enzyme variants with different kinetics across the distribution. Enzymes adapted to different optimum temperatures differ in their amino acid sequence and thermal stability. The Gibbs energy of activation tends to be slightly lower in enzyme variants adapted to lower temperatures, but the big change is a decrease in the enthalpy of activation, with a corresponding change in the entropy of activation, both associated with a more open, flexible structure. Despite evolutionary adjustments to individual enzymes involved in intermediary metabolism (ATP regeneration), many whole-organism processes operate faster in tropical ectotherms compared with temperate or polar ectotherms. Examples include locomotion (muscle power output), ATP regeneration (mitochondrial function), nervous conduction and growth.
38

Brown, Stephanie Leanne. The biosynthesis of poly (ribitol-phosphate) wall teichoic acids and the roles of individual tailoring modifications. 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
39

Smerage, Jeffrey. Activated Transcription of the Glycolytic Enzyme Genes of Saccharomyces Cerevisiae: The Chromatin Structures of TP11 and Mechanisms of RAP1P Mediated Activation. Dissertation Discovery Company, 2018.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
40

Smerage, Jeffrey. Activated Transcription of the Glycolytic Enzyme Genes of Saccharomyces Cerevisiae: The Chromatin Structures of TP11 and Mechanisms of RAP1P Mediated Activation. Creative Media Partners, LLC, 2018.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
41

Chen, Chang-Hwei. Activation and Detoxification Enzymes: Functions and Implications. Springer, 2014.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
42

Chen, Chang-Hwei. Activation and Detoxification Enzymes: Functions and Implications. Springer, 2011.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
43

Deegan, Patrick. Porphyria. Editado por Patrick Davey y David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0179.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
Resumen
This chapter discusses six diseases caused by inborn errors of metabolism affecting the biosynthesis of haem. Haem is a tetracyclic metal-binding compound involved in oxygen transport (in haemoglobin and myoglobin) and redox reactions (e.g. in the cytochrome P450 system). Each of these conditions is caused by a single gene defect in one of the enzymes involved in the biosynthesis of haem. Inheritance is usually autosomal dominant with incomplete penetrance. The enzyme defect results in disease, not as a result of deficiency of the reaction product, but as a result of accumulation of precursors. Early, soluble precursors, 5-aminolaevulinic acid, and porphobilinogen (not porphyrins as such) are neurotoxic and, when present in great excess, as occurs when flux through the haem synthetic pathway is increased in response to particular medications or hormones, lead to acute neurovisceral crises. Later cyclical precursors (porphyrins) in the pathway are also water soluble and excreted in urine, but are susceptible to activation by electromagnetic radiation in the visible spectrum and are converted to free-radical metabolites that cause pain, inflammation, and tissue damage in the skin. The final haem precursors (also porphyrins) are hydrophobic and excreted in the bile and faeces and are also activated by light to toxic metabolites.
44

O’Neal, M. Angela. Pain in the Back. Editado por Angela O’Neal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190609917.003.0006.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
Resumen
This case explores how bone health can be affected by antiepileptic drugs (AEDs), and why this is an important issue in women with epilepsy (WWE). AEDs affect bone health through activation of the cytochrome P450 system in the liver, leading to increased metabolism of vitamin D. The enzyme-inducing AEDs include phenytoin, carbamazepine, phenobarbital, and primidone. Risk factors for osteoporosis include female gender, low body mass, inadequate vitamin D intake, and smoking. The specific AEDs used and the length of treatment confer additional risks. Furthermore, in WWE, risks are magnified related to falls, either from seizures or related to medication toxicity. Screening with bone density and measures to promote bone health are extremely important in these at-risk women.
45

Hester, Charles K. Mitogen-Activated Protein Kinases: Activation, Functions and Regulation. Nova Science Publishers, Incorporated, 2019.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
46

Rossomando, Edward F. HPLC in Enzymatic Analysis. Wiley & Sons, Incorporated, John, 2009.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
47

Frenkel, Joost y Hans R. Waterham. Mevalonate Kinase Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0039.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
Resumen
Mevalonate kinase deficiency (MKD) is an autosomal recessive inborn error of isoprenoid biosynthesis, a pathway yielding sterols and nonsterol isoprenoids.In patients, the enzyme activity of mevalonate kinase is severely reduced due to mutations in the encoding gene, MVK. The substrate, mevalonate, accumulates and is elevated in blood and urine. Shortage of certain downstream products of the pathway, nonsterol isoprenoids, leads to dysregulation of the innate immune system, activation of inflammasomes, and interleukin (IL)-1 mediated inflammation.Symptoms start in early childhood with recurrent attacks of fever, vomiting, diarrhea, headache, sore throat, abdominal pain, arthralgias, painful lymphadenopathy, hepatosplenomegaly, skin rash, and mucosal ulcers. Severely affected patients have additional symptoms, such as intellectual impairment, progressive cerebellar ataxia, and tapetoretinal degeneration. Complications include intestinal obstruction, AA-amyloidosis, hemophagocytosis, and severe infection.Management of MKD is directed at controlling inflammation.
48

(Editor), John N. Abelson, Melvin I. Simon (Editor), Laszlo Lorand (Editor) y Kenneth G. Mann (Editor), eds. Proteolytic Enzymes in Coagulation, Fibrinolysis, and Complement Activation, Part B, Volume 223: Volume 223: Proteolytic Enzymes in Coagulation, Fibrinolysis ... Activation Part B (Methods in Enzymology). Academic Press, 1993.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
49

Rider, Lisa G. y Frederick W. Miller. Outcome assessment in the idiopathic inflammatory myopathies. Editado por Hector Chinoy y Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0016.

Texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
Resumen
Due to their rarity, heterogeneity, and multispecialty nature, the myositis syndromes have limited data-driven consensus on appropriate outcome measures. Recently, two international, multispecialty consortia developed new tools and consensus on core set measures of myositis disease activity and damage, as well as response criteria that are now recommended for use as clinical trial endpoints but will also be useful in clinical practice. Magnetic resonance imaging, muscle ultrasound, selected laboratory tests, and immunological biomarkers—including cytokines, chemokines, lymphocyte flow cytometry, and endothelial activation markers—can all be helpful adjuncts to serum muscle enzyme levels in assessing disease activity and damage, but these have not yet been fully validated. Definitions of clinically inactive disease, complete clinical response, and remission have also been proposed but require further validation. These advances should enhance the development of therapies by standardizing our ability to demonstrate their efficacy in treating the idiopathic inflammatory myopathies.
50

Abelson, John N., Laszlo Lorand, Melvin I. Simon y Kenneth G. Mann. Proteolytic Enzymes in Coagulation, Fibrinolysis, and Complement Activation, Part B. Elsevier Science & Technology Books, 1993.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
También puede estar interesado en las bibliografías sobre el tema "Enzyme activation" para otros tipos de fuentes:
Artículos de revistas Tesis

Pasar a la bibliografía