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Libros sobre el tema "Endocryne tumour"

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Publicado: 11 de marzo de 2023

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1

Donal, O'Shea y Bloom Stephen Robert, eds. Gastrointestinal endocrine tumours. London: Baillière Tindall, 1996.

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2

H, Clark Orlo y American Cancer Society, eds. Endocrine tumors. Hamilton, Ont: BC Decker, 2003.

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3

1936-, Mazzaferri Ernest L. y Samaan Naguib A. 1925-, eds. Endocrine tumors. Boston: Blackwell Scientific Publications, 1993.

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4

Chapman, Catherine Eluned. Radioimmunodetection of endocrine tumours. Birmingham: University of Birmingham, 1987.

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5

Socinski, Mark A. Endocrine tumours and malignancies. London: Mosby-Wolfe, 1996.

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6

Andrew, Arnold, ed. Endocrine neoplasms. Boston: Kluwer Academic Publishers, 1997.

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7

M, Polak Julia y Bloom Stephen Robert, eds. Endocrine tumours: The pathobiology ofregulatory peptide-producing tumours. Edinburgh: Churchill Livingstone, 1985.

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8

Solcia, Enrico, Günter Klöppel y Leslie H. Sobin. Histological Typing of Endocrine Tumours. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-59655-1.

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9

M, Polak Julia y Bloom Stephen Robert, eds. Endocrine tumours: The pathobiology of regulatory peptide-producing tumours. Edinburgh: Churchill Livingstone, 1985.

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10

Surgical pathology of endocrine and neuroendocrine tumors. New York, NY: Humana Press, 2009.

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11

Endocrine tumor syndromes and their genetics. Basel: Karger, 2013.

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12

Gosney, John R. Pulmonary endocrine pathology: Endocrine cells and endocrine tumors of the lung. Oxford: Butterworth-Heinemann, 1992.

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13

L, Barkan Ariel, ed. Medical therapy of endocrine tumors. Philadelphia: Saunders, 1989.

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14

Lutwin, Becker, ed. Hormone-related malignant tumors. Berlin: Springer-Verlag, 1990.

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15

M, Polak Julia, ed. Diagnostic histopathology of neuroendocrine tumours. Edinburgh: Churchill Livingstone, 1993.

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16

Baudin, Éric y Michel Ducreux. Tumeurs endocrines thoraciques et digestives. Paris: Springer Paris, 2008. http://dx.doi.org/10.1007/978-2-287-35574-5.

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17

Beger, Hans G., Akimasa Nakao, John P. Neoptolemos, Shu You Peng y Michael G. Sarr, eds. Pancreatic Cancer, Cystic Neoplasms and Endocrine Tumors. Oxford, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118307816.

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18

Khan, Ashraf, ed. Surgical Pathology of Endocrine and Neuroendocrine Tumors. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-396-1.

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19

R, Pasqualini Jorge y Katzenellenbogen Benita S. 1945-, eds. Hormone-dependent cancer. New York: M. Dekker, 1996.

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20

Richard, Sheaves, Jenkins Paul J. MRCP y Wass J. A. H, eds. Clinical endocrine oncology. Osney Mead, Oxford: Blackwell Science, 1997.

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21

Sturgeon, Cord. Endocrine Neoplasia. Boston, MA: Springer Science+Business Media, LLC, 2010.

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22

D, Hay Ian y Wass J. A. H, eds. Clinical endocrine oncology. 2a ed. Malden, Mass: Blackwell Pub., 2008.

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23

D, Hay Ian y Wass J. A. H, eds. Clinical endocrine oncology. 2a ed. Malden, Mass: Blackwell, 2008.

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24

National Institutes of Health (U.S.). Clinical Center, ed. Understanding multiple endocrine neoplasia type 1. [Bethesda, Md.?]: Clinical Center, National Institutes of Health, 1988.

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25

Schock, Roxanne. Understanding multiple endocrine neoplasia type 1 (MEN 1). [Bethesda, Md.?]: Clinical Center Communications, National Institutes of Health, 1988.

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26

O’Dorisio, T. M., ed. Sandostatin® in the Treatment of Gastroenteropancreatic Endocrine Tumors. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-61328-9.

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27

P, Hollander Vincent, ed. Hormonally responsive tumors. Orlando: Academic Press, 1985.

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28

Jonathan, Waxman, ed. Molecular endocrinology of cancer. Cambridge: Cambridge University Press, 1996.

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29

Jolly, Elaine, Andrew Fry y Afzal Chaudhry, eds. Endocrine. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0007.

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Chapter 7 covers the basic science and clinical topics relating to the endocrine system which trainees are required to learn as part of their basic training and demonstrate in the MRCP. It covers endocrine physiology, acid-base balance, thyrotoxicosis, hypothyroidism, goitre and thyroid nodule, Cushing syndrome, acromegaly, hyperprolactinaemia, hypopituitarism, diabetes insipidus, adrenal incidentaloma, primary hyperaldosteronism, adrenal insufficiency, phaeochromocytoma and paraganglioma , male hypogonadism and Gynaecomastia, menstrual disorders and anovulation, hirsutism and the polycystic ovarian syndrome, multiple endocrine neoplasia and other genetic endocrine tumour syndromes, neuroendocrine tumours, acid-base disorders, sodium disorders, potassium disorders, hypocalcaemia, hypercalcaemia, hyperparathyroidism, osteoporosis, osteomalacia, Paget disease, dyslipidaemia, porphyria, adult inborn errors of metabolism, and obesity.
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30

Bower, Mark, Louise Robinson y Sarah Cox. Endocrine and metabolic complications of advanced cancer. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0142.

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Cancer produces endocrine and metabolic complications in two ways. Firstly, the primary tumour or its metastases may interfere with the function of endocrine glands, kidneys, or liver by invasion or obstruction. Secondly, tumours may give rise to remote effects without local spread and these actions are termed paraneoplastic manifestations of malignancy. Generally, these paraneoplastic syndromes arise from secretion by tumours of hormones, cytokines, and growth factors, but also occur when normal cells secrete products in response to the presence of tumour. This chapter reviews the pathogenesis, epidemiology, and management of the commonest paraneoplastic endocrinopathies including hypercalcaemia, Cushing’s syndrome, the syndrome of inappropriate antidiuresis, non-islet cell tumour hypoglycaemia, enteropancreatic hormone syndromes, Carcinoid syndrome, phaeochromocytoma, gonadotrophin secretion syndromes, prolactin and oxytocin secretion, and paraneoplastic pyrexia. The chapter concludes with a brief discussion of the management of metabolic disease in the context of advanced malignancy including hyperglycaemia, thyroid dysfunction, metabolic bone disease, renal failure, liver failure, and lactic acidosis.
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31

Ajithkumar, Thankamma, Ann Barrett, Helen Hatcher y Natalie Cook. Endocrine tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235636.003.0014.

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Malignancies of the thyroid gland are the commonest endocrine malignancy but comprise <1% of cancer incidence overall (Coleman et al. 1999). The incidence is increasing slowly. The highest incidence is seen in North and Central America, and Australasia, with the lowest incidence in Africa.
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32

Polak, Julia M. Endocrine Tumours: The Pathobiology of Regulatory Peptide-Producing Tumours (Current Problems in Tumour Pathology). Churchill Livingstone, 1985.

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33

Raheja, Amol y William T. Couldwell. Endocrine Active Pituitary Tumor. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0015.

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This chapter presents an illustrative case demonstrating the principles of diagnosis and management in endocrine active pituitary tumor. The index case involves a 30-year-old male patient who presented with phenotypic markers of acromegaly. On radiological and endocrinological evaluation, growth hormone–secreting pituitary macroadenoma was identified. The philosophy of decision-making and management paradigm is discussed to demonstrate the pros and cons of medical, radiation, and surgical options. Technical nuances of the surgical procedure and its complication management are stressed. A brief review of literature is included to elaborate on the current evidence, including outcomes for the various management options for such tumors, with special emphasis on multimodality management.
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34

Arnold, Andrew. Endocrine Neoplasms. Springer, 2012.

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35

Arnold, Andrew. Endocrine Neoplasms. Springer, 2012.

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36

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne y Gareth Morris-Stiff. Endocrine cancers. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199689842.003.0018.

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This chapter examines the aetiology, diagnosis, and management of malignant tumours of the oesophagus, stomach, and small intestine as well as those of the liver, pancreas, gallbladder and the biliary tree. It highlights the multidisciplinary approach to these tumours and illustrates the improved results now being seen through this approach.
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37

Altieri, Barbara, Antongiulio Faggiano y Enzo Lalli, eds. Rare Endocrine Tumors. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-623-9.

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38

Endocrine dependent tumors. New York: Raven Press, 1991.

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39

Staff, CIBA Foundation Symposium. Hormone Production in Endocrine Tumours. Wiley & Sons, Limited, John, 2008.

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40

DeLellis, R. A., G. Klöppel, E. Solcia, C. Capella y L. H. Sobin. Histological Typing of Endocrine Tumours. Springer London, Limited, 2012.

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41

Gosney, John R. Pulmonary Endocrine Pathology: Endocrine Cells and Endocrine Tumours of the Lung. Elsevier Science & Technology Books, 2013.

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42

Epidemiology of Endocrine Tumors. Elsevier, 2020. http://dx.doi.org/10.1016/c2019-0-04370-1.

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43

Singh, Harminder, Smeer Salam y Theodore H. Schwartz. Endocrine Silent Pituitary Tumors. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0016.

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Pituitary adenomas are the most common intracranial neoplasms in adults, with a prevalence of 7% to 17%. Clinically, they can be divided into 2 categories based on whether they secrete pituitary hormones: functional (secretory) and nonfunctional (nonsecretory or endocrine silent) adenomas. The biologic latency of nonfunctional (endocrine silent) adenomas makes them usually diagnosed at the stage of macro (>1 cm) and giant (>4 cm) adenomas. Because these tumors are nonfunctioning, their primary symptoms are due to mass effect, particularly on the optic chiasm and normal pituitary gland and stalk superiorly, and the cavernous sinus laterally. Visual field disturbance is the most common presenting complaint, followed by pituitary dysfunction and headaches. Surgical outcomes, therefore, are aimed at determining visual outcome in addition to rates of gross total resection, recurrence, and postoperative pituitary dysfunction. Several recent case series have documented the increased success of the endonasal endoscopic transsphenoidal approach for resecting nonfunctioning pituitary adenomas, particularly in relation to the classic open cranial and microsurgical transsphenoidal techniques.
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44

Moini, Jahangir, Raheleh Ahangari y Craig Badolato. Epidemiology of Endocrine Tumors. Elsevier, 2021.

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45

Khan, Ashraf. Surgical Pathology of Endocrine and Neuroendocrine Tumors. Humana Press, 2016.

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46

Stratakis, C. A., ed. Endocrine Tumor Syndromes and Their Genetics. S. Karger AG, 2013. http://dx.doi.org/10.1159/isbn.978-3-318-02331-2.

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47

WHO Classification of Tumours of Endocrine Organs. World Health Organization, 2017.

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48

Stassi, Giorgio, Simone Di Franco, Michaela Luconi y Natalia Simona Pellegata, eds. Cancer Stem Cells in Endocrine Tumors. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-256-4.

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49

Bronstein, Marcello D. Pituitary Tumors in Pregnancy (Endocrine Updates). Springer, 2001.

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50

Hessman, Ola. Genetic Studies of Endocrine Abdominal Tumors. Uppsala Universitet, 2001.

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