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Libros sobre el tema "Endocrine and paracrine regulations"

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Publicado: 7 de diciembre de 2024

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1

Reproductive Endocrinology Study Section Workshop on Autocrine and Paracrine Mechanisms in Reproductive Endocrinology (1988 Shrewsbury, Mass.). Autocrine and paracrine mechanisms in reproductive endocrinology. New York: Plenum Press, 1989.

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2

Nikula, Hannu. Endocrine and paracrine modulation of gonadotropin action in the rat testis. Turku: Turun Yliopisto, 1990.

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3

Walter, Helen Jane. The spatio-temporal modulation of the insulin-like growth factor[s] axis in the lesioned central nervous system: Implications for endocrine, paracrine and autocrine activities. Birmingham: University of Birmingham, 1996.

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4

F, Piva y International Symposium on "Cell to Cell Communication in Endocrinology" (1987 : Florence, Italy), eds. Cell to cell communication in endocrinology. New York: Raven Press, 1988.

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5

Ezrin, Calvin. The endocrine control diet: How to beat the metabolic trap and lose weight permanently. New York: Harper & Row, 1990.

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6

1927-, Assenmacher Ivan, Boissin Jean y Centre d'études biologiques des animaux sauvages (France), eds. Endocrine regulations as adaptive mechanisms to the environment =: Régulations endocriniennes et adaptations à l'environnement : Colloque international, Centre d'études biologiques des animaux sauvages, Forêt de Chizé, 1er-5 juillet 1985. Paris: Editions du Centre national de la recherche scientifique, 1986.

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7

Guraya, Sardul S. Comparative Cellular and Molecular Biology of Testis in Vertebrates: Trends in Endocrine, Paracrine and Autocrine Regulation of Structure and Functions. Science Publishers, 2001.

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8

Goldberg, Erwin, Barry Zirkin, Vassilios Papadopoulos y Polina V. Lishko, eds. Endocrine and Paracrine Regulation of Spermatogenesis - A Collection of Up to Date Research Contributions on Testis Formation and Function. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-859-2.

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9

Krey, L. Autocrine and Paracrine Mechanisms in Reproductive Endocrinology. Springer London, Limited, 2012.

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10

Krey, L. Autocrine and Paracrine Mechanisms in Reproductive Endocrinology. Springer London, Limited, 2013.

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11

Dussaule, Jean-Claude, Martin Flamant y Christos Chatziantoniou. Function of the normal glomerulus. Editado por Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0044_update_001.

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Glomerular filtration, the first step leading to the formation of primitive urine, is a passive phenomenon. The composition of this primitive urine is the consequence of the ultrafiltration of plasma depending on renal blood flow, on hydrostatic pressure of glomerular capillary, and on glomerular coefficient of ultrafiltration. Glomerular filtration rate (GFR) can be precisely measured by the calculation of the clearance of freely filtrated exogenous substances that are neither metabolized nor reabsorbed nor secreted by tubules: its mean value is 125 mL/min/1.73 m² in men and 110 mL/min/1.73 m² in women, which represents 20% of renal blood flow. In clinical practice, estimates of GFR are obtained by the measurement of creatininaemia followed by the application of various equations (MDRD or CKD-EPI) and more recently by the measurement of plasmatic C-cystatin. Under physiological conditions, GFR is a stable parameter that is regulated by the intrinsic vascular and tubular autoregulation, by the balance between paracrine and endocrine agents acting as vasoconstrictors and vasodilators, and by the effects of renal sympathetic nerves. The mechanisms controlling GFR regulation are complex. This is due to the variety of vasoactive agents and their targets, and multiple interactions between them. Nevertheless, the relative stability of GFR during important variations of systemic haemodynamics and volaemia is due to three major operating mechanisms: autoregulation of the afferent arteriolar resistance, local synthesis and action of angiotensin II, and the sensitivity of renal resistance vessels to respond to NO release.
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12

Erben, Reinhold G. y L. Darryl Quarles, eds. Endocrine and Paracrine Role of FGF23 and Klotho in Health and Disease. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88945-805-9.

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13

Dacke, Christopher, C. Dacke y I. Caple. Comparative Endocrinology of Calcium Regulations (Endocrine Updates). Society for Endocrinology, 1996.

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14

Mclver, Bryan, Peter J. Tebben y Pankaj Shah. Endocrinology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0200.

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Numerous chemical messages control various functions at the levels of cells, organs, and organ systems. Such messages may be autocrine (the chemical message directly affects the cell producing it), paracrine (the message has local effects), or endocrine (the message has distant sites of action). Typically, endocrine effects are caused by hormones that are produced by specialized organs, although several important endocrine functions are performed by nonglandular tissues, most prominently the liver and the kidney. Disorders of the hypothalamus, ovaries, testes, and pituitary, thyroid, and adrenal glands are reviewed.
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15

Roberge, Sylvie. Endrocrine profiles in cystic ovarian diseased dairy cows and postpartum beef cows. 1991.

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16

Yang, Zhihong y Xiu-Fen Ming. Adventitia and perivascular adipose tissue—the integral unit in vascular disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0020.

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Obesity and obesity-associated metabolic disorders are highly associated with cardiovascular disease. Abnormal ectopic deposition and accumulation of adipose tissue in organs, including perivascular space (perivascular adipose tissue, PVAT) in obesity are emerging to contribute to vascular disease development through pathological paracrine and/or endocrine secretion of cytokines, namely adipokines, which are vasoactive factors including vascular relaxing and contracting factors, smooth muscle growth promoting and inhibiting factors, and pro- and anti-inflammatory factors. In obesity, production of these factors from PVAT is altered and in imbalance which favours vascular contraction, pathological remodelling, and inflammation. In cross-talk with the endothelium, the functional changes of adventitia and PVAT are detrimental and importantly contribute to the acceleration of vascular atherosclerosis and complications associated with obesity and metabolic disorders
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17

Ezrin, Calvin y Robert E. Kowalski. The Endocrine Control Diet: How to Beat the Metabolic Trap and Lose Weight Permanently. HarperCollins, 1989.

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18

Ezrin, Calvin y Robert E. Kowalski. The Endocrine Control Diet: How to Beat the Metabolic Trap and Lose Weight Permanently. HarperCollins, 1989.

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19

Turner, Helen E., Richard Eastell y Ashley Grossman, eds. Endocrinology (Oxford Desk Reference). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199672837.001.0001.

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Oxford Desk Reference: Endocrinology provides an overview of the principles of endocrinology, a detailed pathophysiology of disorders of the endocrine system, and practical advice on the clinical presentation of the spectrum of endocrine disease. Written by over 100 international experts, it discusses the diagnosis, management, and relevant genetic and immunological aspects of endocrine disorders. Whilst discussion of common endocrine conditions is comprehensive, it also includes rare syndromes with useful guidance on screening and follow-up. Providing clinical advice to endocrinologists, general physicians, and specialist nurses, it also includes background to biochemical, immunological, genetic, and epidemiological aspects of endocrinology. It is extensively cross-referenced, with suggestions for further reading, includes links to recent international guidelines, and is illustrated throughout with diagrams, tables, and radiological images. There is a quick reference section which covers algorithms for investigation and management of commonly encountered clinical scenarios for use in the clinic. There is an outpatient resource for explanation of endocrine conditions to patients using diagrams of common conditions while in the clinic, in addition to patient support groups and advice, and discussion on legal aspects of medicine and driving regulations. This should be a very useful resource for all involved in the assessment and management of any patient with any form of a possible endocrine disorder.
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20

Weil, Andrew. Integrative Environmental Medicine. Editado por Aly Cohen y Frederick S. vom Saal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190490911.001.0001.

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Integrative Environmental Medicine looks at the history and changing landscape of environmental issues in the United States, including water supply, air quality, extensive plastic pollution, harmful chemicals in cleaning and personal care products, radiofrequency radiation, food additives, pesticides, and medications. The unique properties of compounds such as endocrine-disrupting chemicals are explored along with their ability to disturb the body’s normal signaling pathways, genetic profile, and gut microbiome. Resulting molecular derangements promote thyroid and other autoimmune diseases, diabetes, cardiovascular disease, cancer, and influence developmental problems in children. Analysis of current research identifies ways to reduce exposures and health risks, improve regulations and appropriate testing for chemicals, remediate environmental pollution, and design healthier products for the future. Best practices are considered for clinicians to ascertain exposure history, test for toxins, and teach patients how to avoid harmful exposures. Patients will be prepared and empowered with information about healthier food choices and cooking practices, appropriate supplement use, water filtration, cleaning and personal care product selection, improved sleep, stress reduction, sauna, fasting, chelation, safe cell phone use, and other means of reducing harmful environmental exposures. Solutions at every level require interdisciplinary collaboration to advance assessment, design, stewardship, and regulation of chemicals to promote environmental and human health.
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