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Libros sobre el tema "Clostridium botulinum"

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Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 28 de julio de 2024

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1

Smith, Louis DS. Botulism: The organism, its toxins, the disease. 2a ed. Springfield, Ill., USA: Thomas, 1988.

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2

Hauschild, Andreas H. W., 1929- y Dodds Karen L. 1957-, eds. Clostridium botulinum: Ecology and control in foods. New York: M. Dekker, 1993.

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3

National Institutes of Health (U.S.). Office of Medical Applications of Research, ed. Consensus statement. Bethesda, MD: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, Office of Medical Applications of Research, 1993.

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4

O, Sang-sŏk. Chŏ sansŏng milbong pʻojang sikpʻum ŭi kijun, kyugyŏk kukchehwa saŏp =: Establishment of global standards for low acid canned foods in hermetically sealed container. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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5

Rasetti-Escargueil, Christine y Susanne Surman-Lee. Clostridium botulinum: A spore forming organism and a challenge to food safety. New York: Nova Science Publishers, 2012.

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6

Szczawiński, Jacek. Wpływ peklowania, pasteryzacji i napromieniowania mięsa na wytwarzanie toksyny przez Clostridium botulinum. Warszawa: Wydawn. SGGW-AR, 1987.

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7

Sharkey, Freddie. Toxin gene expression in clostridium botulinum type E under different growth conditions. 2S.l: The author], 2002.

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8

Sharkey, Freddie. Toxin gene expression in clostridium botulinum type E under different growth conditions. [S.l: The author], 2002.

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9

Sharkey, Freddie. Toxin gene expression in clostridium botulinum type E under different growth conditions. 2S.l: The author], 2002.

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10

Loch, Petra Vera. Kulturell-biochemische und molekularbiologische Untersuchungen sowie Pathogenitätsprüfung von Clostridium botulinum-Stämmen: Ein Beitragzur Taxonomie. Hannover: [s.n.], 2000.

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11

Post, Robert C. Scheme and critical variables for a limited study on the effects of vacuum packaging and irradiation on the outgrowth and toxin production of Clostridium botulinum in pork loins. [Washington, D.C.?]: U.S. Dept. of Agriculture, Food Safety and Inspection Service, 1986.

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12

Yusof, Farida Z. The distribution of toxin genes among isolates of Clostridium botulinum Group II from different environments. [S.l: The Author], 2000.

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13

Daly, Dyan Jennifer. A study of the distribution of toxigenic and non-toxigenic group II non-proteolytic clostridium botulinum strains. [S. l: The Author], 1996.

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14

Klarmann, Dieter. Untersuchungen zum Vorkommen von Clostridium botulinum in Kotproben von Rind and Schwein sowie in Rohmaterialien und Tierkörpermehlen verschiedener Tierkörperbeseitigungsanstalten. Hannover: [s.n.], 1987.

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15

Hauschild. Clostridium Botulinum. Taylor & Francis Group, 2019.

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16

Clostridium botulinum. [Washington, D.C.]: United States Department of Agriculture, Food Safety and Inspection Service, 2010.

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17

Robert, Graham. Clostridium Botulinum Type C. Creative Media Partners, LLC, 2022.

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18

Clostridium botulinum: Ecologyand controlin foods. New York: Marcel Dekker, 1993.

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19

Smith, Louis De Spain. Botulism: The Organism, Its Toxins, the Disease (American Lecture Series in Clinical Microbiology). Charles C. Thomas, Publisher Ltd, 1988.

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20

Hauschild. Clostridium Botulinum (Food Science and Technology). CRC, 1992.

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21

Clostridium Botulinum: A Practical Approach to the Organism and Its Control in Foods. Wolters Kluwer Legal & Regulatory U.S., 1998.

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22

Fernández, Emiliano José Quinto. Clostridium botulinum: Una aproximación práctica al microorganismo y su control en los alimentos. Editorial Acribia, S.A., 2005.

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23

Hauschild. Clostridium Botulinum: Ecology and Control in Foods. Taylor & Francis Group, 2018.

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24

Hauschild. Clostridium Botulinum: Ecology and Control in Foods. Taylor & Francis Group, 2018.

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25

Hauschild. Clostridium Botulinum: Ecology and Control in Foods. Taylor & Francis Group, 2018.

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26

Hauschild. Clostridium Botulinum: Ecology and Control in Foods. Taylor & Francis Group, 2018.

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27

Bell, Chris y Alec Kyriakides. Clostridium Botulinum: A Practical Approach to the Organism and Its Control in Foods. Blackwell Publishing Limited, 2000.

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28

Reuner, Karl Heinrich. Wirkungsmechanismus von Clostridium-botulinum-C2-Toxin in intakten Zellen. 1989.

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29

Fritz, Michaela Sylvia. Struktur-Funktions-Untersuchungen des binären Clostridium botulinum C2-Toxins. 2004.

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30

Katirji, Bashar. Case 24. Editado por Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0028.

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Botulism is an extremely rare neuromuscular disorder, caused by botulinum toxin which is produced by the anaerobic bacteria Clostridium botulinum. It has several forms: classic foodborne, infantile, wound, intestinal, and iatrogenic forms. The presentation is often acute and severe but may be occasionally subacute and moderate. The diagnosis may be difficult and requires a high index of suspicion. This case presents an adult with classic foodborne botulism and highlights the clinical and electrodiagnostic findings that distinguish this disorder from other neuromuscular junction disorders including myasthenia gravis and Lambert-Eaton myasthenic syndrome. Specifically, the findings on repetitive nerve stimulation are discussed and distinguished from the results seen in other neuromuscular junction disorders.
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31

McGrath, Susan. The development of an RNA assay for Clostridium botulinum toxin gene expression. S. l.[The Author], 1999.

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32

Foster, Keith A., Peter Hambleton y Clifford C. Shone. Treatments from Toxins: The Therapeutic Potential of Clostridial Neurotoxins. Taylor & Francis Group, 2010.

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33

(Editor), Keith Alan Foster, Peter Hambleton (Editor) y Clifford C. Shone (Editor), eds. Treatments from Toxins: The Therapeutic Potential of Clostridial Neurotoxins. CRC, 2006.

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34

Lipman, Jeffrey y Robert J. Boots. Diagnosis, assessment, and management of tetanus, rabies, and botulism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0245.

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Tetanus, rabies and botulism are all infections characterized by the production of a neurotoxin, and generally do not give rise to a systemic inflammatory response. Typically tetanus result from the infection of wounds by the ubiquitious soil-borne bacteria Clostridium tetanii, botulism is most commonly due to toxin produced in food contaminated with Clostridium botulinum. Rabies usually results from an animal bite infected with the rabies virus of the Lyssavirus group. Neurological involvement by all three infections is characterized by paralysis and autonomic instability with tetanus also being associated with muscular rigidity. Importantly, the autonomic dysfunction of tetanus can be severe and may necessitate prolonged treatment in an intensive care unit (ICU). Active immunization can prevent or minimize the symptoms of tetanus and rabies, while passive immunization may slow symptom progression in botulism. Intensive care support is often required to manage respiratory failure and autonomic dysfunction. Rabies is typically fatal in the absence of prior immunization.
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35

Bartone, John C. y Abbe Research Division. Bioterrorism and Biology of Botulism -Clostridium Botulinum: Index of New Information and Guide-Book for Consumers, Reference and Research. Abbe Pub Assn of Washington Dc, 2002.

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36

Bartone, John C. y Abbe Research Division. Bioterrorism and Biology of Botulism Clostridium Botulinum: Index of New Information and Guide-Book for Consumers, Reference and Research. Abbe Pub Assn of Washington Dc, 2001.

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37

Stafstrom, Carl E. Disorders Caused by Botulinum Toxin and Tetanus Toxin. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0156.

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Anaerobic organisms of the genus Clostridia (C) can cause significant human disease. Exotoxins secreted by C botulinum and C tetani cause botulism and tetanus, respectively (summarized in Table 156.1). Botulinum neurotoxin causes neuromuscular blockade by interfering with vesicular acetylcholine release, leading to cholinergic blockade at the neuromuscular junctions of skeletal muscle, and consequently, symmetric flaccid paralysis. Tetanus toxin prevents release of inhibitory neurotransmitters at central synapses, leading to overactivity of motor neurons and muscle rigidity and spasms. This chapter reviews clinical features of botulism and tetanus and discusses their pathophysiological basis.
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38

Montecucco, Cesare. Clostridial Neurotoxins. Springer, 2013.

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39

Montecucco, Cesare. Clostridial Neurotoxins: The Molecular Pathogenesis of Tetanus and Botulism. Springer London, Limited, 2013.

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40

Montecucco, C., ed. Clostridial Neurotoxins: The Molecular Pathogenesis of Tetanus and Botulism. SPRINGER-VERLAG, 1995.

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