Bibliografías temáticas / Cl11

Literatura académica sobre el tema "Cl11"

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Publicado: 25 de julio de 2024
Última modificación: 26 de julio de 2024

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Artículos de revistas sobre el tema "Cl11"

1

Sonnenberg, Karsten, Patrick Pröhm, Nico Schwarze, Carsten Müller, Helmut Beckers y Sebastian Riedel. "Untersuchungen chlorreicher Polychloride: [Cl11 ]− , [Cl12 ]2− und [Cl13 ]−". Angewandte Chemie 130, n.º 29 (21 de junio de 2018): 9274–78. http://dx.doi.org/10.1002/ange.201803486.

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Pathak, Parul, Monika Singh, Ananya Naskar, Sandeep Kumar Singh, Nikunj Bhardwaj y Ajay Kumar. "Isolation and Phenotypic Microarray Profiling of Different Pseudomonas Strains Isolated from the Rhizosphere of Curcuma longa L." Stresses 3, n.º 4 (13 de noviembre de 2023): 749–61. http://dx.doi.org/10.3390/stresses3040051.

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In the present study, different Pseudomonas strains were isolated from the rhizospheric soil of Curcuma longa (turmeric) and further identified and characterized based on morphological, biochemical, and molecular characteristics through the 16S rRNA gene sequencing analysis. The identified bacterial strains belong to the Pseudomonas genus viz. Pseudomonas sp. CL10, Pseudomonas sp. CL11, and P. fluorescence CLI4. However, the isolated strains tested positive for IAA production, siderophore production, and the solubilization of tricalcium phosphate during plant growth promoting traits analysis. Further phenotype microArray (PM) technology was used to evaluate the antibiotic and chemical sensitivity of the isolated bacterial strains. The antibiotics phleomycin, oxacillin, vancomycin, novobiocin, spiramycin, and rifampicin, as well as chemicals like, 5-7 dichloro-8-hydroxy quanaldine, 5-7 dichloro-8-hydroxyquinoline, domophenbrobide, and 3-5 dimethoxy benzyl alcohol, showed resistance in all the rhizobacterial strains. However, upon further detailed study, Pseudomonas sp. CL10 exhibited resistance to thirteen antibiotics, CL11 to fourteen, and CL14 showed resistance against seventeen antibiotics and chemical classes. The results of the study indicate that some of these strains can be used as bioinoculum to enhance the plant growth and health.
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3

Sonnenberg, Karsten, Patrick Pröhm, Nico Schwarze, Carsten Müller, Helmut Beckers y Sebastian Riedel. "Investigation of Large Polychloride Anions: [Cl11 ]− , [Cl12 ]2− , and [Cl13 ]−". Angewandte Chemie International Edition 57, n.º 29 (21 de junio de 2018): 9136–40. http://dx.doi.org/10.1002/anie.201803486.

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4

Rios, Patrícia R. P., Elineide B. Silveira, Luíza S. S. Martins, Edson B. Silva Neto y Andréa M. A. Gomes. "Caracterização de isolados de Colletotrichum lagenarium de pepino com base em marcadores isoenzimáticos". Horticultura Brasileira 22, n.º 4 (diciembre de 2004): 729–33. http://dx.doi.org/10.1590/s0102-05362004000400012.

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O conhecimento da variabilidade genética de isolados de Colletotrichum lagenarium é de grande importância para o sucesso de programas de melhoramento genético visando resistência à antracnose do pepino. Dezenove isolados de C. lagenarium oriundos de plantios comerciais de pepino da Zona da Mata e do Agreste de Pernambuco foram comparados por análise eletroforética de isoenzimas em gel de poliacrilamida a 7%, utilizando os sistemas esterase, fosfatase ácida, fosfatase alcalina e malato desidrogenase. A avaliação foi realizada pelo número e pela posição das bandas reveladas nos géis, calculando-se a mobilidade relativa. As distâncias genéticas e similaridades foram sumarizadas em análises de agrupamento, utilizando-se o software NTSYS. A análise mostrou variação no número e posição das bandas no gel, dentro de cada sistema estudado, revelando diferenças fenotípicas dentro da população. As enzimas malato desidrogenase e fosfatase alcalina mostraram menor polimorfismo, enquanto, a esterase e fosfatase ácida apresentaram-se mais polimórficas. A análise de agrupamento permitiu separar os genótipos em cinco grupos distintos: 1 (CL1, CL3 e CL22), 2 (CL14), 3 (CL4, CL38, CL15, CLl7, CL5, CL8, e CL10), 4 (CL28) e 5 (CL23, CL20, CL34, CL36, CL35, CL37, e CL16). O menor índice de similaridade genética (27,3%) foi observado entre o isolado CL1 e os isolados CL20, CL35, CL37 e CL16. O maior índice de similaridade (100%) foi observado entre os isolados CL5 e CL8; CL5 e CL10; CL8 e CL10; CL35 e CL37; CL35 e CL16; e entre CL37 e CL16.
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5

Zhuang, Hong-Mei, Xiao Hu, Shu-Rong Hong, Ping-Ping Zhou, Yu Zhang y Ling Li. "18α-Glycyrrhetinic acid inhibits the viability of HR5-CL11 cervical carcinoma cells through induction of apoptosis and DNA damage". Bangladesh Journal of Pharmacology 11, n.º 3 (26 de agosto de 2016): 750. http://dx.doi.org/10.3329/bjp.v11i3.26592.

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<p class="Abstract">The present study was aimed to investigate the effect of 18α-glycyrrhetinic acid on induction of apoptosis and DNA-damage in HR5-CL11 cervical carcinoma cells. The results revealed 73.6% reduction in HR5-CL11 cell viability on treatment with 5 µM concentration of 18α-glycyrrhetinic acid for 48 hours. The DNA of 18α-glycyrrhetinic acid-treated cells showed a ladder-like pattern. Fragmentation of DNA in the 18α-glycyrrhetinic acid-treated cells was markedly higher compared to the control cells. Examination of the DNA damage in HR5-CL11 cells after treatment with 18α-glycyrrhetinic acid showed breakage in DNA strands and formation of comet-like structures. The frequency of comet formation in 18α-glycyrrhetinic acid treated cells was found to be 7.8 after 48 hours. The population of cells with more than four γH2AX foci was increased to 38.6% on treatment with 5 µM concentration of 18α-glycyrrhetinic acid. Thus, 18α-glycyrrhetinic acid inhibits the viability of HR5-CL11 cervical cancer cells through induction of apoptosis by DNA damage and can be used for the treatment of cervical cancer.</p><p class="Abstract"><strong>Video Clip:</strong></p><p class="Abstract"><a href="https://www.youtube.com/v/XZQHm07d2sk">MTT assay</a>: 1 min 13 sec</p><p> </p>
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6

WANG, CHUNYANG, JINQUAN WANG, JOSHUA GONG, HAI YU, JENNIFER C. PACAN, ZHONGXIANG NIU, WEIDUO SI y PARVIZ M. SABOUR. "Use of Caenorhabditis elegans for Preselecting Lactobacillus Isolates To Control Salmonella Typhimurium". Journal of Food Protection 74, n.º 1 (1 de enero de 2011): 86–93. http://dx.doi.org/10.4315/0362-028x.jfp-10-155.

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Host-specific probiotics have been used to control enteric pathogens, including foodborne pathogens, in food animal production. However, evaluation of the efficacy of these probiotics requires costly in vivo assays in the target animal. The nematode Caenorhabditis elegans has been used for prescreening of antimicrobial agents and for studies of host-pathogen interactions. In the present study, 17 Lactobacillus isolates from chicken and pig intestines were tested with C. elegans, and the ability of these isolates to prevent death from Salmonella infection was variable. Two Lactobacillus isolates (S64, which gave full protection, and CL11, which gave no protection) were further studied. Both isolates exhibited a similar colonization profile in the C. elegans intestine. Although different culture fractions of CL11 were not protective, both live and heat-killed S64 cells provided full or partial protection of C. elegans from death caused by Salmonella infection. In contrast, different culture fractions from both isolates had similar effects on the colonization of the nematode intestine by Salmonella Typhimurium DT104. Our preliminary results from a pig performance trial revealed a correlation between the degree of protection in the C. elegans survival assay and the performance of 35-day-old weaned piglets that were treated with the same Lactobacillus isolates, suggesting that C. elegans can be used as a laboratory animal model for preselecting probiotics for control of Salmonella infections.
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7

Kroupin, P. Yu, A. I. Yurkina, A. A. Kocheshkova, D. S. Ulyanov, G. I. Karlov y M. G. Divashuk. "Comparative assessment of the copy number of satellite repeats in the genome of Triticeae species". Vavilov Journal of Genetics and Breeding 27, n.º 8 (28 de diciembre de 2023): 947–57. http://dx.doi.org/10.18699/vjgb-23-109.

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Satellite repeats are a significant component of the genome of Triticeae and play a crucial role in the speciation. They are a valuable tool for studying these processes. Pseudoroegneria species play a special role among grasses, as they are considered putative donors of the St-genome in many polyploid species. The aim of this study was to compare the copy number of satellite repeats in the genomes of Triticeae species. Quantitative real-time PCR was applied to determine the copy numbers of 22 newly discovered satellite repeats revealed in the whole-genome sequences of Pseudoroegneria species and one additional repeat previously identified in the genome of Aegilops crassa. The study focused on seven species of Pseudoroegneria, three species of Thinopyrum, Elymus pendulinus, Ae. tauschii, Secale cereale, and Triticum aestivum. Based on the copy number level and coefficients of variation, we identified three groups of repeats: those with low variability between species (medium-copy CL82), those with medium variability (low- and medium-copy CL67, CL3, CL185, CL119, CL192, CL89, CL115, CL95, CL168), and those with high coefficients of variation (CL190, CL184, CL300, CL128, CL207, CL69, CL220, CL101, CL262, CL186, CL134, CL251, CL244). CL69 exhibited a specific high copy number in all Pseudoroegneria species, while CL101 was found in both Pseudoroegneria and Th. junceum, CL244 in Th. bessarabicum, CL184 in P. cognata and S. cereale. CL95, CL128, CL168, CL186, CL207, and CL300 exhibited higher copy numbers in P. cognata compared to other species; CL3, CL95, CL115, CL119, CL190, CL220, CL207, and CL300 in P. kosaninii; CL89 in P. libanotica; CL134 in P. geniculata. Our assessment of the copy number of new satellite repeats in the St-genome and the analysis of their amplification specificity between species can contribute to the molecular-genetic and chromosome markers used for evolutionary, phylogenetic, and population studies of Triticeae species.
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8

Jenne, Carsten y Bianca Wegener. "Silver Salts of the Weakly Coordinating Anion [Me3 NB12 Cl11 ]-". Zeitschrift für anorganische und allgemeine Chemie 644, n.º 18 (2 de octubre de 2018): 1123–32. http://dx.doi.org/10.1002/zaac.201800358.

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9

Ghorai, Bithin y Santiram Chatterjee. "Effect of Near-Surface Crustal Layers on Undrained Vertical Penetration Response of Subsea Pipelines". International Journal of Offshore and Polar Engineering 28, n.º 2 (1 de junio de 2018): 218–24. http://dx.doi.org/10.17736/ijope.2018.cl11.

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10

Rohdenburg, Markus, Martin Mayer, Max Grellmann, Carsten Jenne, Tobias Borrmann, Florian Kleemiss, Vladimir A. Azov, Knut R. Asmis, Simon Grabowsky y Jonas Warneke. "Superelektrophiles Verhalten eines Anions demonstriert durch spontane Bindung von Edelgasen an [B12 Cl11 ]−". Angewandte Chemie 129, n.º 27 (31 de mayo de 2017): 8090–96. http://dx.doi.org/10.1002/ange.201702237.

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Tesis sobre el tema "Cl11"

1

Duncan, Peter I. "Characterisation of the murine CLK1 dual-specificity kinase". Thesis, University of Ottawa (Canada), 1997. http://hdl.handle.net/10393/10402.

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Murine Clk1 (also known as Sty) was identified as a dual-specificity kinase capable of phosphorylating serine, threonine and tyrosine residues when expressed in bacteria. Expression of Clk1 is deveolpmentally regulated at the level of RNA expression. Embryonic cells express two mRNAs of similar size, whereas differentiated cells express two additional transcripts of larger size. Little is known about the bochemical and biological activity of Clk1 in mammalian cells. We demonstrate that the two embryonically expressed mRNAs are derived through alternative splicing of a unique exon (exon EB) within the Clk1 precursor mRNA. These mRNAs encode full-length catalytically active Clk1 and a truncated inactive polypeptide, Clk1$\rm\sp{T}.$ Larger incompletely spliced Clk1 transcripts accumulate in differentiated cells. When expressed in mammalian cells Clk1 possesses dual-specificity kinase activity and is capable of forming complexes with other molecules of Clk1 and Clk1$\rm\sp{T}.$ The regions involved in binding map to the amino-terminal non-catalytic domain of Clk1. Phosphorylation sites map to the amino acids encoded by the alternatively splice exon EB. Clk1$\rm\sp{T}$ and catalytic mutants of Clk1 co-localise with splicing factors in intranuclear speckles, whereas catalytically active Clk1 causes a redistribution of these factors within the nucleus. This activity requires the presence of amino acids encoded by exon EB. These results suggest a role for Clk1 and Clk1$\rm\sp{T}$ in the regulation of RNA splicing. Splicing of a Clk1 mini-gene, encompassing exon EB, in vivo is regulated by Clk1 and Clk1$\rm\sp{T}.$ Catalytically active Clk1 stimulates exclusion of EB leading to the production of Clk1$\rm\sp{T}$ mRNA. In contrast, Clk1$\rm\sp{T}$ promotes EB inclusion leading to production of Clk1 mRNA. Two Clk1-related human kinases, hClk2 and hClk3, also exhibit dual-specificity kinase activity and cause the redistribution of nuclear splicing factors. Similar to Clk1$\rm\sp{T},$ the hClk truncated isoforms, hClk2$\rm\sp{T}$ and hClk3, co-localise with splicing factors in nuclear speckles. hClk2 and hClk3 are able to influence the splicing pattern of a murine Clk1 mini-gene in vivo, indicating that they can also regulate precursor mRNA splicing. Taken together these results imply a role for the Clk family of kinases in the regulation of gene expression at the level of RNA processing.
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2

Tibbles, Katherine L. "Regulation of Clb1 during meiosis in Saccharomyces cerevisiae". Thesis, University of Warwick, 2013. http://wrap.warwick.ac.uk/60444/.

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Meiosis is a specialised form of cell division in which diploid cells divide to form four non-identical spores containing half the genetic complement of the parent. During this cell division program, much of the usual machinery regulating cell division is put to alternate use to allow the cells to undergo an extra round of division without an intervening phase of DNA synthesis. In particular, the end of the first division, meiosis I, must be regulated differently than the end of the mitotic division. We used the model organism Saccharomyces cerevisiae to determine some of these differences in regulation. The cell division program is driven by the sequential association of cyclins with the CDK (cyclin dependent kinase), leading to waves of kinase activity. Exit from mitosis requires the downregulation of CDK activity, and is coordinated by two signalling networks, the FEAR (Cdc14 Early Anaphase Release) network and the MEN (Mitotic Exit Network). Both networks initiate the release of the phosphatase Cdc14 from its inhibitor, Net1, to counter CDK activity. Exit from meiosis I similarly relies on Cdc14 activity, but is driven only by the FEAR network. Experimental results showed that the phosphorylation state and subcellular localisation of the meiotic cyclin, C1b1, are altered in meiosis I. We investigated this relationship and aimed to determine the kinase responsible. We used modelling techniques to explore several rationales for the specific regulation of C1b1. We examined the functional significance of C1b1 localisation, using localisation mutants, and made an investigation into Cdc14 release in meiosis I.
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3

Lange, Jenny. "Neuron-glia interactions in infantile neuronal ceroid lipofuscinosis (CLN1 disease)". Thesis, King's College London (University of London), 2016. https://kclpure.kcl.ac.uk/portal/en/theses/neuronglia-interactions-in-infantile-neuronal-ceroid-lipofuscinosis-cln1-disease(ceed9c8f-f40d-4796-be7b-e799fe88b431).html.

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The neuronal ceroid lipofuscinoses (NCLs) are the most common cause of childhood dementia and are invariably fatal. Early, localised glial activation occurs in all forms of NCL and has been shown to be an accurate predictor of areas in which neuronal loss is most pronounced. Indeed, in a tissue culture model of juvenile NCL, glial cells have been shown to actively contribute to neuronal dysfunction and death. So far the role of glial cells has not been established in other forms of NCL and in order to assess glial function in infantile NCL (INCL, CLN1 disease), a series of different tissue cultures were generated from Ppt1 deficient mice (Ppt1-/- ). These studies revealed that both Ppt1-/-astrocytes and microglia exhibit a more activated phenotype under basal conditions, as well as alterations to their protein expression profile following pharmacological stimulation. Ppt1-/- astrocytes displayed moderately reduced glutamate uptake, as well as changes in lactate release. Perhaps as a consequence of these changes, Ppt1- /-astrocyte survival was severely impaired. In addition the morphological phenotypes of Ppt1-/-neurons were explored, revealing decreased neurite outgrowth, complexity and a reduction in cell body size. Ppt1-/-neuronal cultures contained significantly fewer inhibitory neurons and displayed decreased cell survival after prolonged time in culture. Most importantly, using a co-culture system, the presence of Ppt1-/- glial cells appeared to increase cell death in both WT and Ppt1-/- cultures. Notably, Ppt1-/- microglia appeared to trigger increased Ppt1-/- neuronal death, whereas Ppt1-/- astrocytes also exhibited increased cell death. Ppt1-/- glial cells also affected both wild type and Ppt1-/- neuronal morphology, including further reduced neurite outgrowth. In contrast, wild type glial cells ameliorated some of the morphological defects observed in Ppt1-/- neurons, and this was most apparent when wild type astrocytes where grown in co-cultures with Ppt1-/- neurons. Taken together, these finding present novel evidence for compromised glial function in Cln1 disease, demonstrating that both Ppt1-/- microglia and astrocytes may potentially contribute to the neurodegeneration observed in CLN1 disease. These data highlight the importance of targeting glial cells in the development of therapeutic interventions for CLN1 disease. Furthermore, although sharing some similarities Ppt1-/-glial phenotypes were broadly different from those observed in the tissue culture model of juvenile NCL, suggesting that the role of glia may differ between forms of NCL.
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4

Nelvagal, Hemanth Ramesh. "Spinal cord pathology in CLN1 disease : a novel therapeutic target". Thesis, King's College London (University of London), 2017. https://kclpure.kcl.ac.uk/portal/en/theses/spinal-cord-pathology-in-cln1-disease(8b1dc3ed-dfd9-442d-a427-43ded0d82a6a).html.

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The neuronal ceroid lipofuscinoses (NCLs) are a group of up to 14 inherited progressive neurodegenerative lysosomal storage disorders affecting children and young adults. Together, they are the most common pediatric neurodegenerative storage disorders. Symptoms include loss of vision, epileptic seizures and the loss of cognitive and motor function. A lack of any effective therapies means that all forms are fatal. CLN1 disease or Infantile NCL is one of the most rapidly progressing forms of the disease and is caused by a deficiency of the lysosomal enzyme palmitoyl protein thioesterase – 1 (PPT1). Ppt1 deficient (Ppt1-/-) mice recapitulate features of the human disease and have proved to be a useful tool in characterizing disease progression and pathology in the brain. However, these pathological changes fail to fully explain the sensorimotor deficits seen in this mouse model as well as in human CLN1 disease. Along with the limited success of various brain directed therapies, this led us to analyze the spinal cord. Our analysis revealed unexpectedly profound and rapidly progressing disease pathology in the spinal cords of these mice, which occurs earlier than similar events in the brain. This included regional atrophy, neuroinflammation, and significant neuron loss at all levels of the cord as well as novel phenotypes indicating a postnatal developmental delay and significant white matter pathology. Automated gait analysis also showed novel early phenotypes in these mice including an increased dependence on the forelimbs for locomotion. Similar spinal cord pathology was also demonstrated in human INCL autopsy samples as well as in mouse models of the other major forms of NCL. Targeting the spinal cords of Ppt1-/-mice with enzyme replacement therapy (ERT) and gene therapy significantly improved disease pathology, motor function and lifespan in these mice, demonstrating the clinical significance of spinal cord pathology in these mice. Furthermore, combining intracranial and intrathecal gene therapy showed a synergistic effect, showing the greatest improvements for any CLN1 disease therapy to date. Taken together, these findings highlight the spinal cord as not only being significantly affected in CLN1 disease, but also as a novel and effective therapeutic target.
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5

GARSUAULT, SOPHIE. "Degradation et fonction de cln1 identification de sequences en cis responsables de la degradation et de la fonction de cln1, une cycline g1 de saccharomyces cerevisiae". Paris 6, 1998. http://www.theses.fr/1998PA066496.

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Ces travaux ont consiste a identifier des signaux en cis necessaires a la degradation et a la fonction de cln1, une cycline g1 de s. Cerevisiae. Les cyclines g1 sont au coeur de la regulation du cycle cellulaire eucaryote, controlant la transition entre proliferation et differenciation. Chez la levure saccharomyces cerevisiae, cln1 et cln2 s'accumulent dans la cellule en phase g1, declenchent l'engagement irreversible dans un nouveau cycle en activant la kinase cdc28, puis disparaissent brutalement en raison de leur haute instabilite. L'association a cdc28 se fait par l'intermediaire d'une region tres conservee chez toutes les cyclines, la cyclin-box, situee dans la region n-terminale, et correspondant au premier domaine de repliement defini par cristallographie (cyclin-fold). Nous montrons que la derniere helice du second cyclin-fold joue un role determinant dans la fonction de cln1. Peu d'exemples de sequences situees hors de la cyclin-box et contribuant a la fonctionnalite de la cycline ont ete rapportes jusqu'a maintenant. Des deletions carboxy-terminales progressives ont permis de montrer que les determinants de la degradation sont repartis dans la region c-terminale de cln1, et sont similaires a ceux identifies chez cln2 et cln3. Les sites de phosphorylation semblent etre les determinants majeurs, mais la region c-terminale contient d'autres signaux non identifies, necessaires pour une degradation rapide. Les determinants de proteolyse contenus dans la region carboxy-terminale de cln1 ne fonctionnent pas comme des signaux autonomes de degradation, contrairement a ce qui a ete observe chez cln2 et cln3. La mutation ponctuelle dans la cyclin-box d'un residu hautement conserve, l'arginine 72, est suffisante pour abolir la fixation a cdc28, et a stabiliser significativement la cycline. Ainsi, la fixation de la cycline a la kinase est un pre-requis a sa degradation rapide. Nous montrons par ailleurs que les cyclines libres - non fixees a cdc28 - sont reconnues par une voie de degradation operant plus lentement, ne reconnaissant probablement pas les memes signaux que la voie de degradation des cyclines associees a cdc28.
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6

Zou, Meilin. "Haplotypes of cleistogamous flowering gene cly1 and association with temperature stress". Thesis, Zou, Meilin (2017) Haplotypes of cleistogamous flowering gene cly1 and association with temperature stress. Honours thesis, Murdoch University, 2017. https://researchrepository.murdoch.edu.au/id/eprint/37942/.

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Cleistogamy refers to a type of sexual breeding system, a floret type with closed flowers. Cleistogamous flower sheds its pollen before flower opening, so this behavior leads to a great mass of autogamy. Furthermore, the cleistogamy has been found and presented widely in the angiosperm. In recent years, the cleistogamy has been regarded as an important reproductive system in various plant taxa and already attracted widespread attention in the world. However, the understanding of the molecular mechanism of the cleistogamous trait was very limited. It is valuable to explore the cleistogamy gene cly1 in barley. Cly1 has been cloned, and two SNPs within the open reading frame region were identified to be associated with floral types. New markers were developed to genotype 672 barley accessions. Among them, the floral types of 436 lines were investigated. A total of 45 novel lines were identified as the genotype of the two markers could not explain their phenotypes. Five novel lines which showed non-cleistogamous types in the field but had cleistogamous genotype were sequenced. Thirteen SNPs were detected among the gene region. But no SNPs were associated with non-cleistogamy. Promoter region, methylation, and miR172 gene sequence need to be investigated in the future. The purposes of this article are to investigate the occurrence mechanism of cleistogamy, particularly on genetic aspects in barley. Temperature stress including frost stress and heat stress is one of the biggest obstacles to crop production. So the impacts of cleistogamy against temperature stress were discussed in the present study. Grain fertility rate was investigated in the four field trials. Katanning experienced frost stress in 2016 with the lowest temperature of -3.4°C. Grain fertility percentage was as low as 85% in Katanning, while in other three trials, the grain fertility percentage ranged from 92% to 96%. The correlation coefficient of grain fertility between cleistogamous type and non-cleistogamous type was analyzed. No significant difference was detected in the four trials between the two floral types.
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7

Aigner, Christian [Verfasser] y Franz [Akademischer Betreuer] Bracher. "Entwicklung neuer CLK1-Inhibitoren mit antiviraler Aktivität / Christian Aigner ; Betreuer: Franz Bracher". München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2017. http://d-nb.info/1138566160/34.

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Durkan, Anne Maria. "The expression of CX₃CL1 (fractalkine) in renal tubular epithelial cells and the regulation of CX₃CL1 by stimulation of the thromboxane prostanoid receptor". Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/24546.

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Most renal diseases have a common end inflammatory pathway, which is associated with a leukocytic infiltrate. Chemokines are small proteins that are responsible for the chemoattraction of leukocytes into areas of injury or insult. CX3CL1, also known as fractalkine, exists as a transmembrane protein as well as a soluble protein. It acts as a cell adhesion molecule in addition to its chemoattractant properties. This thesis firstly examines the distribution of CX3CL1 in renal tubular epithelial cells (RTEC) and in the second part of the thesis the regulation of CX3CL1 by stimulation of the thromboxane prostanoid (TP) receptor is examined. The localisation of CX3CL1 was initially demonstrated primarily on the apical surface of tubular epithelial cells in human renal biopsy specimens with histological diagnoses of acute tubular necrosis and acute allograft rejection. A cell model was then developed in MDCK cells to examine the distribution more closely. There are a limited number of mechanisms potentially responsible for the trafficking of CX3CL1 to the apical membrane and it was established that N-glycosylation of CX3CL1 is required for its presence on the apical membrane of RTEC. The mobility of CX3CL1 within the cell membrane was next assessed and it was shown to be relatively immobile. We hypothesized that this would promote cell adhesion and indeed further experiments confirmed that CX3CL1 in RTEC does promote adhesion of cells bearing the cognate receptor. Given that CX3CL1 and thromboxane A2 are both found in similar inflammatory conditions, are both present early in the inflammatory process and that stimulation of the TP receptor has been shown to regulate other chemokines, we next evaluated the effect of stimulation of TP on CX3CL1. We found that both total and surface cellular levels of CX3CL1 were reduced following stimulation of TP. A maximal nadir was present after 30-60 minutes and the levels returned to baseline by 4 hours. The mechanism for the loss of CX3CL1 was then assessed. CX3CL1 is known to recycle between the cell surface and an internal compartment. No effect of TP stimulation was seen on the endocytosis or exocytosis of CX3CL1. Stimulation of TP was however, shown to stimulate tumour necrosis factor-a converting enzyme (TACE) via ERK phosphorylation. TACE inducibly cleaves CX3CLI, releasing the soluble chemokine. TACE siRNA was used to knock down TACE gene expression and this prevented the loss of cellular CX3CL1, confirming that TP stimulation induces TACE cleavage of CX3CL1. T he results of further experiments are discussed in the discussion chapter.
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9

Hedden, J. J. "The role of Clp1 and Pcf11 in transcription and pre-mRNA 3’-end processing". Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1369193/.

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Eukaryotic transcripts require a number of complex cotranscriptional modifications and processing events before translation to protein. Clp1 and Pcf11 are subunits of cleavage factor IA (CFIA), an essential component of the Saccharomyces cerevisiae pre-mRNA 3’-end processing machinery. The crystal structure of a Clp1-Pcf11 complex was determined previously and revealed the binding of ATP to a highly-conserved P-loop motif and a tight Pcf11-Clp1 interaction facilitated by a number of highly-conserved Pcf11 residues. Nonetheless, the biological function of both Clp1-ATP binding and the Pcf11-Clp1 interaction was not well understood. The work in this thesis combines an in vitro and in vivo investigation of the Clp-ATP and Clp-Pcf11 interactions in an effort to understand the function of these factors in transcription and pre-mRNA 3’-end processing. It is demonstrated that the interaction of ATP and Pcf11 with Clp1 are linked events: Loss of Clp1-ATP binding results in the abrogation of the Pcf11-Clp1 interaction and leads to Clp1 instability in vitro, and similarly, mutations that directly uncouple the Pcf11-Clp1 interaction also disrupt Clp1-ATP binding and cause Clp1 instability in vitro. An in vivo mutational analysis in S. cerevisiae revealed that both Clp1-ATP binding and the Pcf11-Clp1 interaction are essential for yeast survival. Further cell and immunoprecipitation studies demonstrated that one essential function of Clp1 is as a chaperone of Pcf11, and RT-qPCR analysis of mRNA from a sample set of yeast genes points to a role for these proteins in transcription and transcription termination rather than in poly(A) site selection.
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Isosomppi, Juha. "Molecular and cell biology of infantile (CLN1) and varaint late infantile (CLN5) neuronal ceroid lipofuscinoses". Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/laa/haart/vk/isosomppi/.

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Libros sobre el tema "Cl11"

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Technik, SIAT Architektur +., ed. CL21: Die CargoLifter-Werft = The Cargolifter Shipyard. München: SIAT Architektur + Technik, 2001.

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Companion to Greek Architecture. Wiley & Sons, Limited, John, 2020.

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Sadler. Cps:(Butler)Computer Literacy Cl101. Irwin, 1994.

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Peterson's. Peterson's Prac Test:PSAT #101-Expl/CL10. Peterson's, 2006.

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Peterson's. Peterson's Practice Test: PSAT #101-CL10. Peterson's, 2006.

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Davis, F. A. UB, Classics, CL151: University of Buffalo. Davis Company, F. A., 2021.

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Caillaud, Catherine y Frédéric Sedel. Neuronal Ceroid Lipofuscinoses. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0059.

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Neuronal ceroid lipofuscinoses (NCLs) are inherited neurodegenerative disorders beginning mainly in childhood, rarely in adults. They are characterized by the accumulation of autofluorescent lipopigments in brain, especially in neurons. Their clinical heterogeneity is now explained by the huge number of genes (from CLN1 to CLN14) involved in their pathogenesis. Their diagnosis is possible using enzymatic tests and/or direct sequencing of the corresponding genes. Different therapeutic approaches are in development for these diseases such as enzyme replacement therapy or gene transfer.
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Elmenzo, Marlene. CL1 90V 20 MA Simple Temperature-Compensated Constant-Current LED Driver IC Data Sheet. Microchip Technology Incorporated, 2018.

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Capítulos de libros sobre el tema "Cl11"

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Dempsey, Brian R., Anne C. Rintala-Dempsey, Gary S. Shaw, Yuan Xiao Zhu, A. Keith Stewart, Jaime O. Claudio, Constance E. Runyan et al. "STY (CLK1)". En Encyclopedia of Signaling Molecules, 1810. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101299.

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Villars, P., K. Cenzual, J. Daams, R. Gladyshevskii, O. Shcherban, V. Dubenskyy, V. Kuprysyuk y I. Savysyuk. "Cs4[Sc6C]Cl13". En Structure Types. Part 9: Space Groups (148) R-3 - (141) I41/amd, 818. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-02702-4_591.

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Perkins, Michael R. "Clinical pragmatics". En Handbook of Pragmatics, 139–63. Amsterdam: John Benjamins Publishing Company, 2022. http://dx.doi.org/10.1075/hop.m2.cli1.

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Perkins, Michael R. "Clinical pragmatics". En Handbook of Pragmatics, 1–31. Amsterdam: John Benjamins Publishing Company, 2011. http://dx.doi.org/10.1075/hop.15.cli1.

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Perkins, Michael R. "Clinical pragmatics". En Handbook of Pragmatics, 1–29. Amsterdam: John Benjamins Publishing Company, 2003. http://dx.doi.org/10.1075/hop.7.cli1.

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Villars, P., K. Cenzual, J. Daams, R. Gladyshevskii, O. Shcherban, V. Dubenskyy, V. Kuprysyuk, I. Savysyuk y R. Zaremba. "(NH4)6[{Ta5(NH)4}Cl17]". En Structure Types. Part 10: Space Groups (140) I4/mcm – (136) P42/mnm, 630. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19662-1_536.

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Froehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, Jan Rémi, Soheyl Noachtar, Werner J. Heppt, Annette Cryer et al. "Neuronal Ceroid Lipofuscinosis (CLN1-10), Autosomal Recessive". En Encyclopedia of Molecular Mechanisms of Disease, 1459–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1277.

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Hönerlage, B. "Cu Cl1–x Br x : phonon wavenumbers". En New Data and Updates for I-VII, III-V, III-VI and IV-VI Compounds, 101–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-48529-2_19.

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Hönerlage, B. "Cu Cl1–x Br x : electron mobility, drift velocity". En New Data and Updates for I-VII, III-V, III-VI and IV-VI Compounds, 103. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-48529-2_20.

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Dawra, Dishu, Mayank Dimri, A. K. Singh, Rakesh Kumar Pandey, Alok K. S. Jha y Man Mohan. "Influence of Dense Plasma Environment on the He-α and He-β Transitions of Cl15+ Ion". En Springer Proceedings in Physics, 85–104. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-7691-8_8.

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Actas de conferencias sobre el tema "Cl11"

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Borodin, N. I., A. G. Okhrimchuk y A. V. Shestakov. "Polarizing Spectroscopy of Y3Al5O12, SrAl2O4, CaAl2O4 Crystals Containing Cr4+". En Advanced Solid State Lasers. Washington, D.C.: OSA, 1992. http://dx.doi.org/10.1364/assl.1992.cl11.

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Kass, Daniel, Guoying Yu, Rehan A. Kahloon, Erich Scheller, Luai Huleihel, Giuseppe Deiuliis, John Tedrow et al. "CLF1 Gene Expression Is Increased In Lung Injury And Development". En American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5546.

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Wu, Feng Chang, Yu-Fen Lin, Liang-Chuan Lai, Eric Y. Chuang y Mong-Hsun Tsai. "Abstract 2914: To study DNA repair function of two closely related human lung adenocarcinoma cell lines, CL1-0 and CL1-5." En Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2914.

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Marchand, Pascal. "Dibenzofuran derivatives inspired from cercosporamide as dual inhibitors of Pim and CLK1 kinases". En 7th International Electronic Conference on Medicinal Chemistry. Basel, Switzerland: MDPI, 2021. http://dx.doi.org/10.3390/ecmc2021-11607.

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Meilman, M. L. y M. G. Livshitz. "Origin of Lasing in Forsterite: Additional Data and Analysis". En Advanced Solid State Lasers. Washington, D.C.: OSA, 1992. http://dx.doi.org/10.1364/assl.1992.cl10.

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Deka, C., M. Bass, B. H. T. Chai y X. X. Zhang. "Spectroscopic Studies of Cr4+ in Different Hosts". En Advanced Solid State Lasers. Washington, D.C.: OSA, 1992. http://dx.doi.org/10.1364/assl.1992.cl12.

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Cerqua-Richardson, Kathleen, B. Peng y T. Izumitani. "Spectroscopic Investigation of Cr+4-Doped Glasses". En Advanced Solid State Lasers. Washington, D.C.: OSA, 1992. http://dx.doi.org/10.1364/assl.1992.cl13.

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Payne, S. A., W. F. Krupke, L. K. Smith, W. L. Kway y L. D. DeLoach. "Wing-Pumping of the Cr:LiSAF Laser Material". En Advanced Solid State Lasers. Washington, D.C.: OSA, 1992. http://dx.doi.org/10.1364/assl.1992.cl14.

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Cooper, Rebecca E., Jessica Finck, Rene Hoover, Clara S. Chan y Kirsten Küsel. "Genomic Insights into the Metabolic Flexibility of the Acid-Tolerant Fe(II)-oxidizer Sideroxydans sp. CL21". En Goldschmidt2020. Geochemical Society, 2020. http://dx.doi.org/10.46427/gold2020.476.

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Polyushkevich, L. O., M. S. Gancheva, I. E. Doduyev y L. A. Lutova. "The role of the CLV1, TDR genes in the development of potato tubers (Solanum tuberosum L.)". En IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-359.

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Informes sobre el tema "Cl11"

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Morris, Victor y Herb A. Winston. Laser Ceilometer CL51 Demonstration Field Campaign Report. Office of Scientific and Technical Information (OSTI), mayo de 2016. http://dx.doi.org/10.2172/1254298.

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CALS TEST NETWORK WRIGHT-PATTERSON AFB OH. TMSS Parsing Test MIL-M-9977J. Appendix I. NATO Stage B Cross-Servicing Checklist (CL1) Document Type Definition. Fort Belvoir, VA: Defense Technical Information Center, junio de 1994. http://dx.doi.org/10.21236/ada306310.

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