Literatura académica sobre el tema "Champ cardiaque"
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Artículos de revistas sobre el tema "Champ cardiaque"
Villemin, C., F. Abel, G. Dispersyn, M. Cotty-Eslous y S. Marchand. "La réalité virtuelle comme antidouleur : une revue systématique de la littérature". Douleur et Analgésie 34, n.º 3 (septiembre de 2021): 163–70. http://dx.doi.org/10.3166/dea-2021-0171.
Texto completoRoy, Jean-Yves. "Médecine : crise et défi". Articles 16, n.º 1 (12 de abril de 2005): 43–67. http://dx.doi.org/10.7202/055674ar.
Texto completoGercek, Cihan, Patrice Roth, Djilali Kourtiche, Pierre Schmitt, Isabelle Magne, Martine Souques, Mustapha Nadi y Jacques Lambrozo. "Test d’implants cardiaques pour une exposition professionnelle à un champ électrique 50 Hz". Archives des Maladies Professionnelles et de l'Environnement 77, n.º 3 (junio de 2016): 492. http://dx.doi.org/10.1016/j.admp.2016.03.316.
Texto completoBell, Dawn M., Thomas E. Johns y Larry M. Lopez. "Endothelial Dysfunction: Implications for Therapy of Cardiovascular Diseases". Annals of Pharmacotherapy 32, n.º 4 (abril de 1998): 459–70. http://dx.doi.org/10.1345/aph.17084.
Texto completoCharniot, J. C. "But : évaluation des interférences des champs magnétiques sur les stimulateurs cardiaques". Archives des Maladies du Coeur et des Vaisseaux - Pratique 2005, n.º 138 (abril de 2005): 11–12. http://dx.doi.org/10.1016/s1261-694x(05)88067-8.
Texto completoSouques, M., I. Magne, A. Trigano, R. Frank, M. Héro, P. Schmitt, M. Nadi, F. Audran y J. Lambrozo. "Implants cardiaques et exposition aux champs électromagnétiques 50Hz en environnement professionnel". Archives des Maladies Professionnelles et de l'Environnement 69, n.º 4 (septiembre de 2008): 547–52. http://dx.doi.org/10.1016/j.admp.2008.02.019.
Texto completoSouques, M., J. A. Trigano, R. Frank, I. Magne, O. Blandeau, J. P. Gernez y J. Lambrozo. "Effets d’un champ magnétique de 50 Hz 100μT sur le fonctionnement des stimulateurs cardiaques implantés". Archives des Maladies Professionnelles et de l'Environnement 67, n.º 2 (mayo de 2006): 426–27. http://dx.doi.org/10.1016/s1775-8785(06)78279-0.
Texto completoSouques, Martine. "Influence des champs électromagnétiques non ionisants sur les dispositifs cardiaques médicaux implantables". La Presse Médicale 33, n.º 22 (diciembre de 2004): 1611–15. http://dx.doi.org/10.1016/s0755-4982(04)99003-3.
Texto completoMagne, I., M. Souques y M. Héro. "Quelle conduite tenir après l’implantation d’un défibrillateur cardiaque chez un travailleur exposé aux champs magnétiques ?" Radioprotection 42, n.º 2 (abril de 2007): 211–18. http://dx.doi.org/10.1051/radiopro:2007001.
Texto completoSouques, M., I. Magne, M. Hero y J. Lambrozo. "Quelle conduite tenir après l’implantation d’un défibrillateur cardiaque chez un travailleur exposé aux champs magnétiques ?" Archives des Maladies Professionnelles et de l'Environnement 67, n.º 2 (mayo de 2006): 427. http://dx.doi.org/10.1016/s1775-8785(06)78280-7.
Texto completoTesis sobre el tema "Champ cardiaque"
Bajolle, Fanny. "Second champ cardiaque et morphogenèse de la voie efférente : de la souris à l'homme". Paris 5, 2006. http://www.theses.fr/2006PA05P634.
Texto completoKnowledge about the morphogenesis of the heart is crucial to understanding congenital heart disease. The discovery of the second heart field and its contribution to the outflow tract has changed our view of how the heart forms. The expression profiles of two transgenic mouse lines expressed in subdomains of the second heart field, together with lineage analysis of cardiomyocytes, suggest that the myocardium at the base of the great arteries is prefigured in subdomains of the second heart field. Outflow tract myocardium rotates prior to the positioning of the great arteries and this rotation is disturbed in mutants with outflow tract defects. In Fgf10 mutant embryos, the outflow tract is normal when FgfR2IIIb mutant embryos have malformations of the outflow tract. Analysis of Nkx2. 5 mutant embryos has shown that second heart field markers fail to be repressed in differentiated myocardial cells. In conclusion, data from animal models give new insights into congenital heart disease
Oumellal, Hammou. "Modelisation du champ electrique cardiaque : mise au point de la methodologie sur un modele a 2d". Caen, 1988. http://www.theses.fr/1988CAEN2015.
Texto completoOumellal, Hammou. "Modélisation du champ électrique cardiaque mise au point de la méthodologie sur un modèle à 2D /". Grenoble 2 : ANRT, 1988. http://catalogue.bnf.fr/ark:/12148/cb37617238h.
Texto completoRoux, Marine. "Etude du rôle des gènes HOX dans le développement du cœur chez la souris". Thesis, Aix-Marseille, 2013. http://www.theses.fr/2013AIXM5086.
Texto completoHox genes are known to be involved in the establishment of cell position and identity along the anterior-posterior axis in embryos and could act as key downstream effectors of retinoic acid during heart development. In situ hybridization experiments show that Hoxb1, Hoxa1 and Hoxa3 define sub-domains within the second heart field (SHF). Our genetic lineage analysis reveals the contribution of Hoxb1+ cardiac progenitors to the atria and to the inferior wall of the outflow tract (OFT), which then gives rise to the myocardium at the base of the pulmonary trunk. Interestingly, Hoxa1+ progenitors contribute to the distal part of the OFT suggesting that these anterior Hox genes could play a role in its proximo-distal patterning. No cardiac anomalies had been reported so far in Hoxb1 mutant mice. However, our detailed study shows that mutant fetuses exhibit OFT misalignment and ventricular septal defects associated or not with ventricular wall and epicardium anomalies. Using a marker of the sub-pulmonary myocardium, we observe an abnormal contribution of SHF cells in Hoxb1-/- hearts. This defect is the consequence of the dysregulation of the signaling pathways controlling SHF regulation. Accordingly, those embryos exhibit a shorter OFT. The study of Hoxa1 mutant embryos reveals pharyngeal arch arteries patterning defects causing anomalies of the aortic arch and right subclavian artery at fetal stages. Using compound mutants, we show an increase in the penetrance and severity of these defects, suggesting a synergistic interaction between Hoxa1 and Hoxb1 during aortic arch patterning. Together, these data support a crucial role for anterior Hox genes in cardiac development
Phua, Chee Teck. "Développement d'une nouvelle méthode de mesure du rythme cardiaque et du débit sanguin fondée sur les perturbations localisées d'un champ magnétique". Phd thesis, Université Paris-Est, 2012. http://tel.archives-ouvertes.fr/tel-00794495.
Texto completoPhua, Chee Teck. "Développement d’une nouvelle méthode de mesure du rythme cardiaque et du débit sanguin fondée sur les perturbations localisées d’un champ magnétique". Thesis, Paris Est, 2012. http://www.theses.fr/2012PEST1099/document.
Texto completoContinuous pulse rate, blood pressure and blood flow monitoring are important for the assessment of physiological vital signs as these are able to provide continuous feedback on the health condition of an individual. Existing commercial, medical and research methods to continuously acquire such these physiological vital signs require good electrical or optical contact. During this research, a magnetic based sensing method, at room temperature, for blood pulse, flow and pressure is developed to achieve data acquisition through fabric, environmental contaminants and body-fluids. This method is named Modulated Magnetic Signature of Blood (MMSB) and physical measurements were conducted on multiple subjects, mathematically modelled and simulated in a multi-physics environment with verification through use of measurement data. Measurement results, using MMSB, for blood pressure and blood flow were compared, and found to be well correlated, with lifestyle device and medical research instruments respectively. In addition, two devices are developed, and are in the midst of commercialization, to support lifestyle applications
Mony, Marie-Cécile. "Étude des cartographies chimiques élémentaires et des concentrations relatives des cations du tissu cardiaque en microscopie ionique analytique : aspects physiologiques et pharmacologiques". Paris 11, 1989. http://www.theses.fr/1989PA112313.
Texto completoGercek, Cihan. "Immunité des implants cardiaques actifs aux champs électriques de 50/60 Hz". Thesis, Université de Lorraine, 2016. http://www.theses.fr/2016LORR0226/document.
Texto completoThe European Directive 2013/ 35 / EU specify minimum requirements for the protection of workers exposed to electromagnetic fields and define with implants as “workers at particular risk”. Regarding the implantable cardioverter defibrillator wearers (ICD) or pacemaker (PM), exposure to electric or magnetic field of extremely low frequency creates inductions inside the human body that generate interference voltage which may cause the dysfunction of the implant. This thesis investigates the electromagnetic compatibility of cardiac implants subjected to an electric field low frequency (50/60 Hz). Computational simulations are effectuated in order to design an experimental bench for the exposure of a phantom including pacemakers or implantable defibrillators. A provocative study is established to define the electric field thresholds for preventing any malfunction of the implant. In numerical simulations; a virtual human model (digital phantom containing a cardiac implant) was placed in an upright position in a vertical exposure to an electric field. The finite element method was used to define the inductions in the cardiac implant level with a resolution of 2 mm (CST® software). In the experimental part, a test bench designed to allow generating an electric field up to 100 kV / m at frequencies 50-60 Hz was constructed, optimized and employed to investigate the behavior of cardiac implants.Several configurations were studied. 54 active cardiac implants (43 pacemakers and 11 defibrillators) are submitted to very high electric field of 50-60 Hz (up to 100 kV / m) inside the experimental bench. No failure was observed for public exposure levels for most configurations (+ 99%) except for six pacemakers in the case of a configuration clinically almost inexistent: unipolar mode with maximum sensitivity and atrial sensing.The implants configured with a nominal sensitivity in bipolar mode are resistant to electric fields exceeding the low action levels (ALs), even for the most high ALs, as defined by 2013 / 35 / EU
Rammah, Mayyasa. "Characterization of cardiopharyngeal progenitor cells and transcriptional regionalisation in the cardiac outflow tract". Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM4061.
Texto completoThe vertebrate heart develops from the heart tube and the contribution of mesodermal progenitors termed second heart field (SHF). Perturbation in SHF addition leads to congenital heart defects (CHD). The outflow tract (OFT) myocardium is entirely derived from the SHF. Distinct regions of the embryonic OFT have been shown to give rise to subaortic and subpulmonary myocardium of the heart. The work described here focuses on SHF progenitor subpopulations in mouse giving rise to distinct OFT domains and characterizes the regional transcriptional identity and regulation of future subaortic and subpulmonary myocardium. We identified Notch-dependent subaortic myocardial SHF progenitors in anterior pharyngeal mesoderm. We demonstrated that Notch/Hes1 and Tbx1/Pparg cross regulatory cascades are important to establish functionally important OFT regional domains. Explant and embryo culture experiments revealed that Pparg is required for both the deployment of SHF cells and transcriptional regulation of the future subpulmonary myocardial domain. We also found that Dlk1, a negative regulator of Pparg, is expressed in the complementary subaortic domain upstream of Notch receptor activation and potentially participates in the establishment of OFT regional identity. We also report an overlapping transcriptional profile between future subaortic myocardium and subpopulation of epicardial cells at fetal stages. Finally, we provide evidence for the existence of conserved bipotential myogenic progenitors in cardiopharyngeal mesoderm coexpressing Nkx2-5 and Tbx1. Overall this work identifies novel pathways and genes in cardiopharyngeal mesoderm that may contribute to clinically relevant CHD
Chauvin, Michel. "Interactions entre champs electriques et magnetiques et stimulation cardiaque permanente. Risques induits par l'instrumentation biomedicale". Université Louis Pasteur (Strasbourg) (1971-2008), 1991. http://www.theses.fr/1991STR13044.
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