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Libros sobre el tema "Cerebral arteriovenous malformations"

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Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 23 de febrero de 2023

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1

Samson, Duke S., H. Hunt Batjer y Philip E. Stieg. Intracranial arteriovenous malformations. New York: Informa Healthcare, 2007.

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2

Shokei, Yamada, ed. Arteriovenous malformations in functional areas of the brain. Armonk, NY: Futura Pub. Co., 1999.

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3

Yaşargil, Mahmut Gazi. AVM of the brain. Stuttgart: Thieme, 1987.

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4

J, Teddy P., Valavanis A y Duvernoy Henri M, eds. AVM of the brain, histology, embryology, pathological considerations, hemodynamics, diagnostic studies, microsurgical anatomy. Stuttgart: Georg Thieme, 1987.

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5

1953-, Steiger Hans-Jakob, ed. Neurosurgery of arteriovenous malformations and fistulas: A multimodal approach. Wien: Springer, 2002.

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6

Cavernous malformations of the nervous system. Houndmills, Basingstoke, Hampshire: Cambridge University Press, 2011.

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7

Kathie's miracle. [Place of publication not identified]: Xlibris, 2010.

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8

Maciunas, Robert J. Endovascular neurological intervention. Park Ridge, Ill: American Association of Neurological Surgeons, 1995.

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9

Awad, Issam. Dural Arteriovenous Malformations. Not Avail, 1993.

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10

Batjer, H. Hunt, Philip E. Stieg y Duke Samson. Intracranial Arteriovenous Malformations. Taylor & Francis Group, 2006.

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11

(Editor), Philip E. Stieg, H. Hunt Batjer (Editor) y Duke Samson (Editor), eds. Intracranial Arteriovenous Malformations. Informa Healthcare, 2006.

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12

Yamada, Shokei. Arteriovenous Malformations in Functional Areas of the Brain. Wiley & Sons, Incorporated, John, 2008.

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13

Cavernous Malformations. American Association of Neurological Surgeons, 1992.

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14

Schmid-Elsaesser, Robert, Alexander Muacevic, Hartmut Brückmann y Berndt Wowra. Neurosurgery of Arteriovenous Malformations and Fistulas: A Multimodal Approach. Springer London, Limited, 2012.

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15

J, Jafar Jafar, Awad Issam A y Rosenwasser Robert H, eds. Vascular malformations of the central nervous system. Philadelphia: Lippincott Williams & Wilkins, 1999.

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16

(Editor), Jafar J. Jafar, Issam A. Awad (Editor) y Robert H. Rosenwasser (Editor), eds. Vascular Malformations of the Central Nervous System. Lippincott Williams & Wilkins, 1999.

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17

Pasqualin, A. y R. Da Pian. New Trends in Management of Cerebro-Vascular Malformations: Proceedings of the International Conference, Verona, Italy, June 8-12, 1992. Springer-Verlag Telos, 1994.

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18

1928-, Pasqualin A., Da Pian R. 1951-, Scienza R y International Conference "New Trends in Management of Cerebro-vascular Malformations" (1992 : Verona, Italy), eds. New trends in management of cerebro-vascular malformations: Proceedings of the international conference, Verona, Italy, June 8-12, 1992. Wien: Springer-Verlag, 1994.

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19

Spinelli, Allison y Liang Huang. Arteriovenous Malformation. Editado por David E. Traul y Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0006.

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Arteriovenous malformation (AVM) is an abnormal connection between arteries and veins, bypassing the capillary system, which may occur in the brain or the spinal cord. AVMs are challenging to diagnose, and the symptoms may be subtle or dramatic. The location and extent of the lesion will determine its potential for morbidity as these lesions also affect a young population. Most importantly, the technology and evolution of treatment for AVMs has changed dramatically over the past two decades and now incorporates interventional neuroradiology and stereotactic radiation. Management options include conservative management, surgical resection, endovascular embolization, and stereotactic surgery. This chapter’s discussion will cover surgical and interventional modes of treatment of cerebral AVMs.
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20

Renfrow, Jaclyn J., Aqib H. Zehri, Kyle M. Fargen, Jasmeet Singh, John A. Wilson y Stacey Q. Wolfe. Management of Intracranial Vascular Lesions During Pregnancy. Editado por Emma Ciafaloni, Cheryl Bushnell y Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0016.

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Management of cerebral vascular lesions in pregnancy requires special consideration to an altered natural history in the pregnant patient, such as a higher rupture rate of arteriovenous malformations. Additionally, treatment challenges exist including radiation exposure, medication selection, optimal treatment timing, and modalities. If identified prior to a pregnancy most vascular lesions warrant a definitive treatment discussion to circumvent the risks associated with an intracranial hemorrhage during pregnancy. The treatment team consists of a multidisciplinary approach involving neurosurgeons, anesthesiologists, neurointensivists, and obstetricians.
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21

Prout, Jeremy, Tanya Jones y Daniel Martin. Neuroanaesthesia and neurocritical care. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199609956.003.0022.

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This chapter describes the general conduct of anaesthesia for neurosurgery with particular reference to techniques for reducing intracranial pressure, safe positioning, and recognition and management of air embolus. Management for specific common procedures such as shunt surgery, haematomas, traumatic brain injury and pituitary surgery is described. Neurosurgical conditions such as cerebral aneurysms and arteriovenous malformations may be managed in neuroradiology and the special considerations for the provision of anaesthesia for these cases are detailed. The principles of management of traumatic brain injury in critical care which aim to reduce secondary brain injury are explained.
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22

Harley, Kim y Sue Jones. Neurological and spinal surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642663.003.0023.

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Neurological assessment is performed on admission for surgery, as a routine part of medical examination. This is done to diagnose whether symptoms of illness in the patient are due to neurological conditions and, if so, where in the nervous system the pathological lesions are located. Hydrocephalus is either an acute or chronic condition whereby the cerebrospinal fluid pressure rises, causing symptoms of raised intracranial pressure. Patients at risk of raised intracranial pressure should be nursed by staff trained and experienced in neurological assessment using the Glasgow coma scale. This chapter looks at neurological assessment, raised intracranial pressure, head injuries, and brain and spinal tumours. This chapter also discusses the management of subarachnoid haemorrhage, cerebral aneurysm, arteriovenous malformations, and epilepsy. Finally, the chapter provides an overview of degenerative diseases of the spine and peripheral nerve injury.
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23

Maciunas, Robert J. Endovascular Neurological Intervention. American Association of Neurological Surgeons, 1995.

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24

Kevin Luk, K. H. y Deepak Sharma. Subarachnoid Hemorrhage. Editado por David E. Traul y Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0024.

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Subarachnoid hemorrhage (SAH) is commonly caused by rupture of an intracranial aneurysm, arteriovenous malformation, or due to trauma. Prompt diagnosis and intervention are required to control intracranial pressure, maintain cerebral perfusion, and prevent rebleeding. Clinical grading of the bleed predicts morbidity and mortality, whereas imaging grading predicts risk of cerebral vasospasm. Hydrocephalus can occur as a result of SAH, which requires treatment with an external ventricular drain. Endovascular and open microsurgical procedures are available for securing the vascular abnormalities. Patients are typically monitored in a neurocritical care unit for up to 21 days post-bleed to monitor for the development of cerebral vasospasm/delayed cerebral ischemia (DCI). Mainstay of treatment for DCI includes induced hypertension, balloon angioplasty, and intraarterial vasodilator therapy. In addition, patient may experience significant derangement in their cardiac, pulmonary, and endocrine systems, requiring inotropic support, mechanical ventilation, or insulin infusion therapy.
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25

Laureno, Robert. Asymptomatic Disease. Editado por Robert Laureno. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190607166.003.0008.

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This chapter on “Asymptomatic Disease” examines the discovery and management of asymptomatic neurologic diseases. Considered are carotid stenosis, berry aneurysm, arteriovenous malformation, and pseudotumor cerebri. Asymptomatic disease can be discovered by routine physical examination or by diagnostic tests that have been ordered for some unrelated reason. Discovery of asymptomatic disease is not always accidental; practitioners sometimes search for asymptomatic brain disease. Such testing can be prompted by the family history, the patient’s personal history, or the clinical situation. After an asymptomatic condition has been found, the practitioner must decide how to use the information. Judgment, in such cases, can be difficult.
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26

Sharma, Deepak y Julia Metzner. Nontraumatic Intracranial Hemorrhage. Editado por Matthew D. McEvoy y Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0062.

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Nontraumatic subarachnoid, intraventricular, or intraparenchymal hemorrhage can be caused by either rupture of an aneurysm or arteriovenous malformation or by coagulopathy, hypertension, or vasculitis. Pituitary apoplexy results from spontaneous hemorrhage or infarction into a pituitary tumor. Additionally, anesthesiologists must be prepared to manage intraoperative bleeding during craniotomies. Successful management of nontraumatic intracranial hemorrhage requires (1) careful preoperative evaluation and preparation considering extracranial manifestations of intracranial bleeding; (2) administration of balanced anesthesia to facilitate surgical exposure and neurophysiological monitoring; (3) maintenance of cerebral perfusion by preserving circulating volume, judicious use of blood product transfusion and vasopressors, and avoidance of excessive hyperventilation; and, when possible, (4) providing timely emergence from anesthesia to allow neurological assessment. Close communication between the surgical and anesthesia teams is critical for optimizing the potential for good patient outcomes.
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