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Libros sobre el tema "Cellular Migration"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 16 de noviembre de 2024

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1

Nguyen, Laurent y Simon Hippenmeyer, eds. Cellular and Molecular Control of Neuronal Migration. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-007-7687-6.

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2

1949-, Husband Alan J., ed. Migration and homing of lymphoid cells. Boca Raton, Fla: CRC Press, 1988.

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3

Laboratory, Cold Spring Harbor, ed. The Cell surface. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1992.

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4

Laboratory, Cold Spring Harbor, ed. The cell surface. Plainview, N.Y: Cold Spring Harbor Laboratory Press, 1992.

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5

Cellular Migration and Formation of Neuronal Connections. Elsevier, 2013. http://dx.doi.org/10.1016/c2011-0-07731-1.

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6

Nguyen, Laurent y Simon Hippenmeyer. Cellular and Molecular Control of Neuronal Migration. Springer, 2016.

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7

Nguyen, Laurent y Simon Hippenmeyer. Cellular and Molecular Control of Neuronal Migration. Springer London, Limited, 2013.

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8

Cellular And Molecular Control Of Neuronal Migration. Springer, 2013.

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9

Nguyen, Laurent y Simon Hippenmeyer. Cellular and Molecular Control of Neuronal Migration. Springer, 2013.

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10

Cellular Migration and Formation of Axons and Dendrites. Elsevier, 2020. http://dx.doi.org/10.1016/c2017-0-00859-5.

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11

Olszewski, Waldemar. In Vivo Migration of Immune Cells. Taylor & Francis Group, 2021.

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12

In vivo migration of immune cells. Boca Raton, Fla: CRC Press, 1987.

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13

Rakic, Pasko y John Rubenstein. Cellular Migration and Formation of Neuronal Connections: Comprehensive Developmental Neuroscience. Elsevier Science & Technology Books, 2013.

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14

Rakic, Pasko, Bin Chen, John Rubenstein y Kenneth Y. Kwan. Cellular Migration and Formation of Neuronal Connections: Comprehensive Developmental Neuroscience. Elsevier Science & Technology Books, 2020.

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15

Rakic, Pasko, Bin Chen, John Rubenstein y Kenneth Y. Kwan. Cellular Migration and Formation of Neuronal Connections: Comprehensive Developmental Neuroscience. Elsevier Science & Technology, 2020.

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16

Rakic, Pasko y John Rubenstein. Cellular Migration and Formation of Neuronal Connections: Comprehensive Developmental Neuroscience. Elsevier Science & Technology Books, 2013.

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17

Symposia on Quantitative Biology Vol. LVII, 1992. Cold Spring Harbor Laboratory Press, 1993.

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18

Oncogenes. 2a ed. Boca Raton, Fla: CRC Press, 1989.

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19

Oncogenes. Boca Raton, Fla: CRC Press, 1986.

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20

Pimentel, Enrique. Oncogenes. Taylor & Francis Group, 2020.

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21

Pimentel, Enrique. Oncogenes. Taylor & Francis Group, 2020.

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22

Pimentel, Enrique. Oncogenes. Taylor & Francis Group, 2020.

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23

Pimentel, Enrique. Oncogenes. Taylor & Francis Group, 2019.

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24

Pimentel, Enrique. Oncogenes. Taylor & Francis Group, 2020.

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25

Dyer, Laura A. y Margaret L. Kirby. The role of the neural crest in cardiac development. Editado por José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso y Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0019.

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Resumen
The cardiac neural crest (CNC) plays pivotal roles in numerous steps of cardiac development. Every aspect of the CNC cell’s lifespan is highly orchestrated, from its induction in the dorsal neural tube to its migration to its differentiation at its final destination. During migration, CNC cells are affected by their environment and simultaneously modulate the extra-cellular milieu through which they migrate. In the pharyngeal arches, CNC cells repattern the originally symmetrical arch arteries, producing the great arteries. Because the cardiac neural crest is essential for many aspects of heart development, it is unsurprising that human CNC-related syndromes have severe phenotypes. This chapter describes how CNC cells are formed and contribute to their final destinations. Essential signalling pathways are presented in the context of CNC development, and CNC-related syndromes are included to highlight this population’s broad importance during development.
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26

Lopez-Arvizu, Carmen, Carmel Bogle y Harolyn M. E. Belcher. Neurobiology of Fetal Alcohol Spectrum Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0179.

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Prenatal exposure to ethanol can result in a wide range of clinical presentations that are grouped under the term “Fetal Alcohol Spectrum Disorders” (FASD). The direct cellular teratogenic effects of ethanol on fetal neurodevelopment include damage to cell survival, proliferation, and migration mechanisms. Dysregulation of neurotransmission and alteration of genetic transcription have also been implicated in the neurotoxic effects of prenatal ethanol exposure. These deleterious events lead to brain volume reduction, corpus callosum dysgenesis, cerebellar, and other neuroanatomical anomalies that have been observed in individuals with FASD. Beyond direct ethanol-induced insults, the impact that ethanol has on maternal nutrition, metabolism, hormonal regulation, and placental physiology also adversely effects fetal development. The complex interactions between numerous neurobiological and psychosocial mechanisms that hinder optimal fetal neurodevelopment are reflected by the heterogeneous clinical presentation of FASD, including impaired growth, dysmorphic facial features, and cognitive and behavioral disorders.
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27

Luckner, Martin. Secondary Metabolism and Cell Differentiation. Springer, 2011.

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28

Stickel, Eberhard y Martin Luckner. Secondary Metabolism and Cell Differentiation. Springer, 2011.

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