Literatura académica sobre el tema "BRAF isoforms"
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Artículos de revistas sobre el tema "BRAF isoforms"
Marranci, Andrea, Andrea Tuccoli, Marianna Vitiello, Elisa Mercoledi, Samanta Sarti, Simone Lubrano, Monica Evangelista et al. "Identification of BRAF 3′UTR Isoforms in Melanoma". Journal of Investigative Dermatology 135, n.º 6 (junio de 2015): 1694–97. http://dx.doi.org/10.1038/jid.2015.47.
Texto completoBayer, Abraham L., Jodie Pietruska, Jaymes Farrell, Siobhan McRee, Pilar Alcaide y Philip W. Hinds. "AKT1 Is Required for a Complete Palbociclib-Induced Senescence Phenotype in BRAF-V600E-Driven Human Melanoma". Cancers 14, n.º 3 (23 de enero de 2022): 572. http://dx.doi.org/10.3390/cancers14030572.
Texto completoTadijan, Ana, Francesca Precazzini, Nikolina Hanžić, Martina Radić, Nicolò Gavioli, Ignacija Vlašić, Petar Ozretić et al. "Altered Expression of Shorter p53 Family Isoforms Can Impact Melanoma Aggressiveness". Cancers 13, n.º 20 (18 de octubre de 2021): 5231. http://dx.doi.org/10.3390/cancers13205231.
Texto completoVlašić, Ignacija, Anđela Horvat, Ana Tadijan y Neda Slade. "p53 Family in Resistance to Targeted Therapy of Melanoma". International Journal of Molecular Sciences 24, n.º 1 (21 de diciembre de 2022): 65. http://dx.doi.org/10.3390/ijms24010065.
Texto completoDillon, Martha, Antonio Lopez, Edward Lin, Dominic Sales, Ron Perets y Pooja Jain. "Progress on Ras/MAPK Signaling Research and Targeting in Blood and Solid Cancers". Cancers 13, n.º 20 (10 de octubre de 2021): 5059. http://dx.doi.org/10.3390/cancers13205059.
Texto completoSorokin, Alex, Lea Bitner, Ji Wu, David Menter, Scott Kopetz y Van Karlyle Morris. "Antitumor activity of panRAF inhibition in BRAF V600E metastatic colorectal cancer." Journal of Clinical Oncology 35, n.º 4_suppl (1 de febrero de 2017): 616. http://dx.doi.org/10.1200/jco.2017.35.4_suppl.616.
Texto completoBroseghini, Elisabetta, Emi Dika, Eric Londin y Manuela Ferracin. "MicroRNA Isoforms Contribution to Melanoma Pathogenesis". Non-Coding RNA 7, n.º 4 (27 de septiembre de 2021): 63. http://dx.doi.org/10.3390/ncrna7040063.
Texto completoNgeow, Kao Chin, Hans J. Friedrichsen, Linxin Li, Zhiqiang Zeng, Sarah Andrews, Laurent Volpon, Hannah Brunsdon et al. "BRAF/MAPK and GSK3 signaling converges to control MITF nuclear export". Proceedings of the National Academy of Sciences 115, n.º 37 (27 de agosto de 2018): E8668—E8677. http://dx.doi.org/10.1073/pnas.1810498115.
Texto completoFarrell, Jaymes, Jodie Pietruska, Siobhan McRee, Philip Tsichlis y Philip Hinds. "Abstract PR14: Defining isoform-specific roles for AKTs in BRAFV600E-driven melanoma". Cancer Research 80, n.º 19_Supplement (1 de octubre de 2020): PR14. http://dx.doi.org/10.1158/1538-7445.mel2019-pr14.
Texto completoJournal, Baghdad Science. "Detection of RAF fusion transcripts in FFPE samples of Medullablastoma and Ependymom in Iraqi children with RT-RQPCR assays". Baghdad Science Journal 11, n.º 3 (7 de septiembre de 2014): 1411–19. http://dx.doi.org/10.21123/bsj.11.3.1411-1419.
Texto completoTesis sobre el tema "BRAF isoforms"
Duggan, Megan C. "The Role of Novel NRAS Isoforms in Melanoma Disease Progression and BRAF Inhibitor Resistance". The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1491229758308229.
Texto completoLUBRANO, SIMONE. "Yeast S. cerevisiae as a tool to study BRAFV600E kinase isoforms". Doctoral thesis, Università di Siena, 2018. http://hdl.handle.net/11365/1040150.
Texto completoAlatawi, Abdullah Salem S. "Alternative Splicing of MDM4 in Human Melanomas". Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1598021739996938.
Texto completoValluet, Agathe. "Utilisation de modèles murins pour l'étude du rôle physiologique des isoformes de BRaf et du rôle des protéines Raf dans le lignage mélanocytaire". Paris 7, 2010. http://www.theses.fr/2010PA077167.
Texto completoOur team works on the ERK pathway and we mainly focus on the Raf proteins family. We have demonstrated that the BRAF gene encoded several isoforms resulting from alternative splicing of exons 8b and 9b. The presence of these sequences modulates the biochemical and oncogenic properties of the protein. The aim of my project was to analyse the phenotype of knockout mice for each £/to/alternatively spliced exons. Constitutive ablation revealed no obvious defects during embryogenesis and adulthood. However, behavioural analyses revealed a specific role for exon 9b-containing BRaf isoforms in certain types of hippocampal-dependent learning and memory. BRaf and CRaf protein kinases have recently emerged as critical players in cutaneous melanoma but little is known about their putative role in the melanocyte lineage in vivo. The aim of my second project was to analyse the phenotype of mice deleted for both BRaf and CRaf in this lineage. Surprisingly, the double knockouts displayed normal pigmentation at birth and did not show signifîcant defect in melanoblasts. However, fbllowing the first hair | molting, the double knockout animals unveiled a progressive hair graying phenotype resulting from depletion of j melanocyte stem cells (MSC). In vitro cultures of melanocytic cells derived from knockout animals could not sustain growth in the presence of TPA but proliferated in the presence of the Kit ligand, SCF. Taken together, our results show that Raf signalling is required for proper MSC maintenance, but dispensable for early melanocyte lineage development. Our observations reveal an unexpected uncoupling between Kit and Raf signalling in the melanocyte lineage
Actas de conferencias sobre el tema "BRAF isoforms"
McRee, Siobhan K., Jodie R. Pietruska, Elisabeth Dignan y Philip W. Hinds. "Abstract B07: Differential roles for AKT isoforms in BRAF mutant melanoma". En Abstracts: AACR Precision Medicine Series: Targeting the Vulnerabilities of Cancer; May 16-19, 2016; Miami, FL. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1557-3265.pmccavuln16-b07.
Texto completoPoulikakos, Poulikos I. "Abstract PR-1: Acquired resistance to RAF inhibitors is mediated by splicing isoforms of BRAF(V600E) that dimerize in a RAS-independent manner." En Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 12-16, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1535-7163.targ-11-pr-1.
Texto completoPeng, Sheng-Bin, James Henry, Michael Kaufman, Wei-Ping Lu, Bryan D. Smith, Subha Vogeti, Scott Wise et al. "Abstract DDT02-02: Identification of LY3009120 as a pan inhibitor of Raf isoforms and dimers with minimal paradoxical activation and activities against BRaf or Ras mutant tumor cells". En Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-ddt02-02.
Texto completoDuggan, Megan, Andrew Stiff, Gonzalo Olaverria Salavaggione, Maryam Bainazar, Nicholas Latchana, Joseph Markowitz, Albert de la Chapelle, Ann-Kathrin Eisfeld y William Carson. "Abstract 301: Identification of NRAS isoform 2 overexpression as a novel mechanism facilitating BRAF inhibitor resistance in malignant melanoma (MM)". En Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-301.
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