Bibliografías temáticas / Blood proteins Physiology

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Literatura académica sobre el tema "Blood proteins Physiology"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 29 de enero de 2023

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Artículos de revistas sobre el tema "Blood proteins Physiology"

1

Davies, Peter L., Choy L. Hew, and Garth L. Fletcher. "Fish antifreeze proteins: physiology and evolutionary biology." Canadian Journal of Zoology 66, no. 12 (December 1, 1988): 2611–17. http://dx.doi.org/10.1139/z88-385.

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Many marine teleosts have adapted to ice-laden seawater by evolving antifreeze proteins and glycoproteins. These proteins are synthesized in the liver for export to the blood where they circulate at levels of up to 20 mg/mL. There are at least four distinct antifreeze protein classes differing in carbohydrate content, amino acid composition, protein sequence, and secondary structure. In addition to antifreeze structural diversity, fish species differ considerably with respect to mechanisms controlling seasonal regulation of plasma antifreeze concentrations. Some species synthesize antifreeze p
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2

Jia, Ruizhe, Jingyun Li, Can Rui, Hui Ji, Hongjuan Ding, Yuanqing Lu, Wei De, and Lizhou Sun. "Comparative Proteomic Profile of the Human Umbilical Cord Blood Exosomes between Normal and Preeclampsia Pregnancies with High-Resolution Mass Spectrometry." Cellular Physiology and Biochemistry 36, no. 6 (2015): 2299–306. http://dx.doi.org/10.1159/000430193.

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Background/Aims: Exosomes are extracellular vesicles that are involved in several biological processes. The roles of proteins from human umbilical cord blood exosomes in the pathogenesis of preeclampsia remains poorly understood. Methods: In this study, we used high-resolution LC-MS/MS technologies to construct a comparative proteomic profiling of human umbilical cord blood exosomes between normal and preeclamptic pregnancies. Results: A total of 221 proteins were detected in human umbilical cord blood exosomes, with 14 upregulated and 15 downregulated proteins were definitively identified bet
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3

Croxatto, HR. "How Many Peptidic Hormones Can Derive From Blood Plasma Proteins?" Physiology 5, no. 5 (October 1, 1990): 201–4. http://dx.doi.org/10.1152/physiologyonline.1990.5.5.201.

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Peptides having structures identical or related to some already known hormones have been obtained using pepsin as a hydrolytic agent acting at acid pH upon plasma substrates. The data suggest the existence of systems similar to renin-angiotensin for the generation of active peptides other than angiotensin or kinins.
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4

Ohno, H., K. Yamashita, R. Doi, K. Yamamura, T. Kondo, and N. Taniguchi. "Exercise-induced changes in blood zinc and related proteins in humans." Journal of Applied Physiology 58, no. 5 (May 1, 1985): 1453–58. http://dx.doi.org/10.1152/jappl.1985.58.5.1453.

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Effects of cycle ergometer exercise (approximately 75% maximum ventilatory O2 consumption for 30 min) on the concentrations of zinc and related proteins in erythrocytes and/or plasma were studied on 11 sedentary male students. Lower concentrations of total zinc and of zinc derived from carbonic anhydrase I type (CA-I) in erythrocytes were observed immediately after exercise, but they disappeared after 30 min of rest. The change in total zinc concentration in erythrocytes correlated well with that in CA-I concentration immediately after exercise, as well as after rest. The concentration of carb
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5

Teahan, Carmel G., Hugh A. McKenzie, and Mervyn Griffiths. "Some monotreme milk “whey” and blood proteins." Comparative Biochemistry and Physiology Part B: Comparative Biochemistry 99, no. 1 (January 1991): 99–118. http://dx.doi.org/10.1016/0305-0491(91)90014-5.

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Richardson, Samantha J., Julie A. Monk, Caroline A. Shepherdley, Lars O. E. Ebbesson, Frank Sin, Deborah M. Power, Peter B. Frappell, Josef Köhrle, and Marilyn B. Renfree. "Developmentally regulated thyroid hormone distributor proteins in marsupials, a reptile, and fish." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 288, no. 5 (May 2005): R1264—R1272. http://dx.doi.org/10.1152/ajpregu.00793.2004.

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Thyroid hormones are essential for vertebrate development. There is a characteristic rise in thyroid hormone levels in blood during critical periods of thyroid hormone-regulated development. Thyroid hormones are lipophilic compounds, which readily partition from an aqueous environment into a lipid environment. Thyroid hormone distributor proteins are required to ensure adequate distribution of thyroid hormones, throughout the aqueous environment of the blood, and to counteract the avid partitioning of thyroid hormones into the lipid environment of cell membranes. In human blood, these proteins
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7

Synelnyk, T. B., O. O. Kravchenko, O. S. Kostiuk, O. M. Savchuk, S. A. Sukhodolia, and L. I. Ostapchenko. "DISTRIBUTION OF SERINE PROTEASES IN BLOOD PLASMA AND PANCREAS IN CHRONIC PANCREATITIS AND ONCOPATHOLOGY." Fiziolohichnyĭ zhurnal 68, no. 6 (December 8, 2022): 31–43. http://dx.doi.org/10.15407/fz68.06.031.

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The aim of our study was to evaluate the trypsin-like serine proteases (TLPs) distribution between systemic circulation and pancreatic tissue and to investigate the peculiarities of their involvement in the extracellular matrix components degradation in patients with pancreatic pathologies with electrophoretic analysis methods using. Тhe Khmelnitsky Regional Clinical Hospital patients aged 28-89 were selected for this study: 20 people with chronic pancreatitis (group CP); 20 people with pancreatic cancer (group PC); 20 conditionally healthy persons (control). Blood plasma samples and pancreati
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8

Lampe, L., K. Wienhold, G. Meyer, F. Baisch, H. Maass, W. Hollmann, and R. Rost. "Effects of simulated microgravity (HDT) on blood fluidity." Journal of Applied Physiology 73, no. 4 (October 1, 1992): 1366–69. http://dx.doi.org/10.1152/jappl.1992.73.4.1366.

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Exposures to microgravity and head-down tilt (HDT) produce similar changes in body fluid. This causes an increase in hematocrit that significantly affects hemorheological values. Lack of physical stimulation under bed rest conditions and the relative immobility of the crew during spaceflight also affects the blood fluidity. A group of six healthy male subjects participated as volunteers, and blood samples were collected 10 days before, on day 2 and day 9, and 2 days after the HDT phase. Blood rheology was quantified by plasma viscometry, red cell aggregability, and red cell deformability. A re
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9

Martino, Tami A., Nazneen Tata, Georg A. Bjarnason, Marty Straume, and Michael J. Sole. "Diurnal protein expression in blood revealed by high throughput mass spectrometry proteomics and implications for translational medicine and body time of day." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 293, no. 3 (September 2007): R1430—R1437. http://dx.doi.org/10.1152/ajpregu.00183.2007.

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Molecular gene cycling is useful for determining body time of day (BTOD) with important applications in personalized medicine, including cardiovascular disease and cancer, our leading causes of death. However, it impractically requires repetitive invasive tissue sampling that is obviously not applicable for humans. Here we characterize diurnal protein cycling in blood using high-throughput proteomics; blood proteins are easily accessible, minimally invasive, and can importantly serve as surrogates for what is happening elsewhere in the body in health and disease. As proof of the concept, we us
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van de Graaf, Stan F. J., Joost G. J. Hoenderop, and René J. M. Bindels. "Regulation of TRPV5 and TRPV6 by associated proteins." American Journal of Physiology-Renal Physiology 290, no. 6 (June 2006): F1295—F1302. http://dx.doi.org/10.1152/ajprenal.00443.2005.

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The epithelial Ca2+ channels TRPV5 and TRPV6 are the most Ca2+-selective members of the TRP channel superfamily. These channels are the prime target for hormonal control of the active Ca2+ flux from the urine space or intestinal lumen to the blood compartment. Insight into their regulation is, therefore, pivotal in our understanding of the (patho)physiology of Ca2+ homeostasis. The recent elucidation of TRPV5/6-associated proteins has provided new insight into the molecular mechanisms underlying the regulation of these channels. In this review, we describe the various means of TRPV5/6 regulati
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Tesis sobre el tema "Blood proteins Physiology"

1

Su, Linlin, and 苏琳琳. "Drug transporters and blood-testis barrier dynamics." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47752816.

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Bexis, Sotiria. "The relationship between vascular structure, contractile proteins, vascular reactivity and blood pressure in animal models of hypertension /." Title page, contents and abstract only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09phb572.pdf.

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Mwaikambo, Bupe Rose. "Emerging roles for the CD36 scavenger receptor in neovascular ocular disease." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=115899.

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Ocular neovascularization (NV) associated with corneal NV, ischemic retinopathies and age-related macular degeneration is a leading cause of severe vision loss. While numerous contributing factors have been identified, the potential role of the CD36 scavenger receptor has been largely overlooked notwithstanding its crucial involvement in normal retinal function. Accordingly, the central aim of this work was to elucidate the contribution and regulation of CD36 during ocular NV using the cornea as a model.<br>Initial work investigating the role of CD36 10 maintaining corneal avascularity, an imp
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4

Jiang, Liying. "Exposure of endothelial cells to shear stress stimulates protein tryosine phosphorylation." Thesis, Georgia Institute of Technology, 1996. http://hdl.handle.net/1853/25421.

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Liang, Yan, and 梁艳. "Endothelial LKB1/AMPK signaling pathway in regulating energy and vascular homeostasis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193460.

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Liver kinase B1 (LKB1), a serine/threonine kinase, is responsible for the activation of AMP-activated protein kinase (AMPK), the master regulator of energy metabolism. LKB1/AMPK signaling pathway possesses a wide range of biological functions in regulating cell cycle progression, cell polarity, senescence and inflammation. In cultured endothelial cells, the pro-senescence function of LKB1/AMPK signaling pathway has been observed. However, the mechanisms by which LKB1 is regulated in endothelial cells remain largely uncharacterized. Furthermore, little is known about the effects of activated en
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6

Waghulde, Harshal B. "Mapping and CRISPR/Cas9 Gene Editing for Identifying Novel Genomic Factors Influencing Blood Pressure." University of Toledo Health Science Campus / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=mco1470402637.

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Henninger, Nils. "Inhibiting Axon Degeneration in a Mouse Model of Acute Brain Injury Through Deletion of Sarm1." eScholarship@UMMS, 2017. http://escholarship.umassmed.edu/gsbs_diss/900.

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Traumatic brain injury (TBI) is a leading cause of disability worldwide. Annually, 150 to 200/1,000,000 people become disabled as a result of brain trauma. Axonal degeneration is a critical, early event following TBI of all severities but whether axon degeneration is a driver of TBI remains unclear. Molecular pathways underlying the pathology of TBI have not been defined and there is no efficacious treatment for TBI. Despite this significant societal impact, surprisingly little is known about the molecular mechanisms that actively drive axon degeneration in any context and particularly followi
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8

Chakaroun, Rima. "Effects of weight loss and exercise on chemerin serum concentrations and adipose tissue expression in human obesity." Doctoral thesis, Universitätsbibliothek Leipzig, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-158639.

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Chemerin is a chemoattractant adipokine that regulates adipogenesis and may induce insulin resistance. Chemerin serum concentrations are elevated in obese, insulin-resistant, and inflammatory states in vivo. Here we investigate the role of omental (OM) and subcutaneous (SC) adipose tissue chemerin and CMKLR1 messenger RNA (mRNA) expression in human obesity. In addition, we test the hypothesis that changes in chemerin serum concentrations are primarily associated with reduced body fat mass in the context of 3 weight loss intervention studies. Chemerin serum concentration was measured in 740 ind
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9

Julien, Mathéau A. "Mechanical Strain-Mediated Syndecan Regulation and Its Effects on Adhesion of Vascular Smooth Muscle Cells." Diss., Georgia Institute of Technology, 2005. http://hdl.handle.net/1853/7007.

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An injured vascular system has a substantial impact on an individuals overall health, and an understanding of the mechanisms that underlie blood vessel pathophysiology is required for the development of rational and effective treatment strategies. The phenotypic modulation of smooth muscle cells (SMC) during vascular injury, characterized by altered adhesion, migration and synthetic behavior, plays an important role in the eventual outcome. Specifically, the ability of SMCs to adhere to and remodel their extracellular environment via regulation of the syndecan class of cell adhesion molecule
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10

Babin, Patrick. "Lipoproteines et apolipoproteines plasmatiques chez les poissons teleosteens." Paris 6, 1987. http://www.theses.fr/1987PA066032.

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Caracterisation des lipoproteines plasmatiques et de leurs apolipoproteines chez salmo gairdneri. Determination de leur masse moleculaire et de leur densite. L'etude au long du cycle annuel de reproduction a permis de demontrer la presence de proteines vitellines ovulaires dans le plasma. De plus, le role des lipoproteines, plasmatiques dans la steroidogenese ovarienne a ete etudie
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Libros sobre el tema "Blood proteins Physiology"

1

Andreeva, Alla Michailovna. Structural and functional organization of fish blood proteins. Hauppauge, N.Y: Nova Science, 2011.

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2

Hepatic plasma proteins: Mechanisms of function and regulation. San Diego: Academic Press, 1993.

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3

Job, Harenberg, and Heidelberger Akademie der Wissenschaften, eds. New trends in haemostasis: Coagulation proteins, endothelium, and tissue factors. Berlin: Springer-Verlag, 1990.

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4

International, Meeting on Anion Transport Protein of the Red Blood Cell Membrane as well as Kidney and Diverse Cells (1989 Fukuoka-shi Japan). Anion transport protein of the red blood cell membrane: Proceedings of the International Meeting on Anion Transport Protein of the Red Blood Cell Membrane as well as Kidney and Diverse Cells, Fukuoka, 1-3 May 1989. Amsterdam: Elsevier, 1989.

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1949-, Agre Peter, and Cartron Jean Pierre, eds. Protein blood group antigens of the human red cell: Structure, function, and clinical significance. Baltimore: Johns Hopkins University Press, 1992.

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S, Pagano Irwin, and Strait Nathan B, eds. HDL and LDL cholesterol physiology and clinical significance. Hauppauge, NY: Nova Science Publishers, 2009.

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R, Harris James, ed. Erythroid cells. New York: Plenum Press, 1990.

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Edwin J. Cohn and the development of protein chemistry: With a detalied account of his work on the fractionation of blood during and after World War II. [Boston, Mass.]: Published by Center for Blood Research, Inc., 2002.

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A, Schreiber, ed. Primate phylogeny from a human perspective: A study based on the immunological technique of comparative determinant analysis (CDA). Stuttgart: G. Fischer, 1996.

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service), ScienceDirect (Online, ed. The HDL handbook: Biological functions and clinical implications. London: Academic, 2010.

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Capítulos de libros sobre el tema "Blood proteins Physiology"

1

Bauer, C., and W. Wuillemin. "Blood, Plasma Proteins, Coagulation, Fibrinolysis, and Thrombocyte Function." In Comprehensive Human Physiology, 1651–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60946-6_84.

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Ogawa, Kishiko, and Elvira Fehrenbach. "Exercise Intensity and Duration Affect Blood-Soluble HSP72." In Heat Shock Proteins and Whole Body Physiology, 253–65. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-3381-9_15.

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Nag, Sukriti, Dan Kilty, and Shruti Dev. "Extracellular Matrix Proteins in Cerebral Vessels in Chronic Hypertension." In Biology and Physiology of the Blood-Brain Barrier, 327–31. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_53.

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Stanimirovic, Danica B., Rita Ball, Josée Wong, and Jon P. Durkin. "The Role of Protein Kinase C and Marcks Protein Phosphorylation in Rat Cerebromicrovascular Endothelial Cell Proliferation Induced by Astrocyte-Derived Factors." In Biology and Physiology of the Blood-Brain Barrier, 213–20. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_36.

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Calvo, Charles-Félix, and Michel Mallat. "Expression of Macrophage Chemotactic Protein-1 in Rat Glial Cells." In Biology and Physiology of the Blood-Brain Barrier, 271–77. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_44.

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Mannucci, P. M., S. Viganò, S. Antoncecchi, and N. Ciavarella. "Protein C — An Inhibitor of Blood Coagulation: Biochemistry, Physiology, Clinical Aspects." In Advances in Hemostasis and Thrombosis, 149–57. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4615-9424-6_15.

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Stanimirovic, Danica B., Paul Morley, Edith Hamel, Rita Ball, Geoff Mealing, and Jon P. Durkin. "Calcium and Protein Kinase C Signaling in Response to Vasoactive Peptides in Human Cerebromicrovascular Endothelial Cells." In Biology and Physiology of the Blood-Brain Barrier, 221–27. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_37.

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Passow, Hermann. "Molecular aspects of band 3 protein-mediated anion transport across the red blood cell membrane." In Reviews of Physiology, Biochemistry and Pharmacology, Volume 103, 61–203. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/3540153330_2.

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Sembulingam, K., and Prema Sembulingam. "Blood and Plasma Proteins." In Essentials of Physiology for Dental Students, 38. Jaypee Brothers Medical Publishers (P) Ltd., 2011. http://dx.doi.org/10.5005/jp/books/11397_6.

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Pramanik, Debasis. "Composition of blood and plasma proteins." In Principles of Physiology, 87. Jaypee Brothers Medical Publishers (P) Ltd., 2015. http://dx.doi.org/10.5005/jp/books/12674_12.

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Actas de conferencias sobre el tema "Blood proteins Physiology"

1

Comp, P. C., and C. T. Esmon. "Defects in the protein C pathway." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643715.

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Activated protein C functions as an anticoagulant by enzymatically degrading factors Va and Villa in the clotting cascade. Protein C may be converted to its enzymatically active form bythrombin. The rate at which thrombin cleavage of the zymogen occurs is greatly enhanced when thrombin is bound to an endothelial cell receptor protein, thrombomodulin. Activated proteinC has a relatively long half-life in vivo and the formation of activated protein C in response to low level thrombin infusion suggests that the protein C system may provide a feedback mechanism to limit blood clotting. Clinical su
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2

Wachtfogel, Yanina T., Yizhar Floman, Meir Liebergall, Robert W. Colman, and Amiram Eldor. "PLATELET ALPHA2-ADRENERGIC RECEPTOR ABNORMALITIES IN PATIENTS WITH IDIOPATHK: SCOLIOSIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644567.

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Idiopathic scoliosis is a genetic multisystem disease involving skeletal, biochemical, central nervous svstem, muscle and blood platelet abnormalities. Platelets of patients with idiopathic scoliosis have been shown to have decreased adenosine diphosphate and epinephrine-induced aggregation. Similarities between the contractile protein system of platelets and muscle have made the platelet a popular model for certain aspects of muscle physiology. This study confirmed that 64% of the patient platelets tested exhibited a significantly decreased sensitivity to aggregation bv epinephrine. In seven
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Tran, Phat L., Jessica R. Gamboa, Katherine E. McCracken, Jeong-Yeol Yoon, and Marvin J. Slepian. "Interaction With Nanoscale Topography: The Use of Nanowell-Trapped Charged Ligand-Bearing Nanoparticle Surfaces To Modulate Physiological Focal Adhesions in Endothelial Cells." In ASME 2013 2nd Global Congress on NanoEngineering for Medicine and Biology. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/nemb2013-93345.

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Achieving cell adhesion, growth and homeostasis on an underlying biomaterial surface may be a desirable feature in implant device design and tissue engineering. Insight has been gained from numerous cell patterning strategies where spatial cues and physical constraints have been shown to regulate the structure and function of cells. Despite significant advances in modifying substrates for cellular attachment, migration and proliferation, the achievement of confluent and aligned growth of functional endothelial cells on cardiovascular blood-contacting implants under physiologically significant
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