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Artículos de revistas sobre el tema "AIDS (Disease)"

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Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 10 de marzo de 2023

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1

Thomsen, Hauke, Xinjun Li, Kristina Sundquist, Jan Sundquist, Asta Försti y Kari Hemminki. "Familial associations for Addison’s disease and between Addison’s disease and other autoimmune diseases". Endocrine Connections 9, n.º 11 (noviembre de 2020): 1114–20. http://dx.doi.org/10.1530/ec-20-0328.

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Design Addison’s disease (AD) is a rare autoimmune disease (AID) of the adrenal cortex, present as an isolated AD or part of autoimmune polyendocrine syndromes (APSs) 1 and 2. Although AD patients present with a number of AID co-morbidities, population-based family studies are scarce, and we aimed to carry out an unbiased study on AD and related AIDs. Methods We collected data on patients diagnosed with AIDs in Swedish hospitals and calculated standardized incidence ratios (SIRs) in families for concordant AD and for other AIDs, the latter as discordant relative risks. Results The number of AD patients was 2852, which accounted for 0.4% of all hospitalized AIDs. A total of 62 persons (3.6%) were diagnosed with familial AD. The SIR for siblings was remarkably high, reaching 909 for singleton siblings diagnosed before age 10 years. It was 32 in those diagnosed past age 29 years and the risk for twins was 323. SIR was 9.44 for offspring of affected parents. AD was associated with 11 other AIDs, including thyroid AIDs and type 1 diabetes and some rarer AIDs such as Guillain–Barre syndrome, myasthenia gravis, polymyalgia rheumatica and Sjögren’s syndrome. Conclusions The familial risk for AD was very high implicating genetic etiology, which for juvenile siblings may be ascribed to APS-1. The adult part of sibling risk was probably contributed by recessive polygenic inheritance. AD was associated with many common AIDs; some of these were known co-morbidities in AD patients while some other appeared to more specific for a familial setting.

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2

Kamradt, T., D. Niese y F. Vogel. "SLIM DISEASE (AIDS)". Lancet 326, n.º 8469-8470 (diciembre de 1985): 1425. http://dx.doi.org/10.1016/s0140-6736(85)92588-7.

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3

Marquart, K. H., H. A. G. Müller, J. Sailer y R. Moser. "SLIM DISEASE (AIDS)". Lancet 326, n.º 8465 (noviembre de 1985): 1186–87. http://dx.doi.org/10.1016/s0140-6736(85)92707-2.

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4

Chen, Yahong, Jinjin Yuan, Xianlin Han, Xiaolong Liu, Xiao Han y Hanhui Ye. "Coexpression Analysis of Transcriptome on AIDS and Other Human Disease Pathways by Canonical Correlation Analysis". International Journal of Genomics 2017 (2017): 1–10. http://dx.doi.org/10.1155/2017/9163719.

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Acquired immune deficiency syndrome is a severe disease in humans caused by human immunodeficiency virus. Several human genes were characterized as host genetic factors that impact the processes of AIDS disease. Recent studies on AIDS patients revealed a series disease is complicating with AIDS. To resolve gene interaction between AIDS and complicating diseases, a canonical correlation analysis was used to identify the global correlation between AIDS and other disease pathway genes expression. The results showed that HLA-B, HLA-A, MH9, ZNED1, IRF1, TLR8, TSG101, NCOR2, and GML are the key AIDS-restricted genes highly correlated with other disease pathway genes. Furthermore, pathway genes in several diseases such as asthma, autoimmune thyroid disease, and malaria were globally correlated with ARGs. It suggests that these diseases are a high risk in AIDS patients as complicating diseases.

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5

Simon, Douglas y Lawrence J. Brandt. "Biliary tract disease in aids: Aids vs. nonaids". Hepatology 12, n.º 3 (septiembre de 1990): 618–19. http://dx.doi.org/10.1002/hep.1840120330.

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6

McGuinness, G., J. F. Gruden, M. Bhalla, T. J. Harkin, J. S. Jagirdar y D. P. Naidich. "AIDS-related airway disease." American Journal of Roentgenology 168, n.º 1 (enero de 1997): 67–77. http://dx.doi.org/10.2214/ajr.168.1.8976923.

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7

Golbus, Joseph. "Rheumatic disease and AIDS". Postgraduate Medicine 92, n.º 4 (15 de septiembre de 1992): 99–110. http://dx.doi.org/10.1080/00325481.1992.11701468.

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8

McGowan, Ian y Peter Anton. "AIDS and intestinal disease". Current Opinion in Gastroenterology 13, n.º 1 (enero de 1997): 18–23. http://dx.doi.org/10.1097/00001574-199701000-00004.

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9

Metze, K. y J. A. Maciel. "AIDS and Chagas' disease". Neurology 43, n.º 2 (1 de febrero de 1993): 447. http://dx.doi.org/10.1212/wnl.43.2.447-a.

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10

Rieve, Julia A. "AIDS Disease Management Programs". Case Manager 11, n.º 5 (septiembre de 2000): 38–39. http://dx.doi.org/10.1067/mcm.2000.110320.

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11

Goodman, Philip C. "Mycobacterial disease in AIDS". Journal of Thoracic Imaging 6, n.º 4 (septiembre de 1991): 22–27. http://dx.doi.org/10.1097/00005382-199109000-00007.

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12

Navarro, Willis H. y Lawrence D. Kaplan. "AIDS-related lymphoproliferative disease". Blood 107, n.º 1 (1 de enero de 2006): 13–20. http://dx.doi.org/10.1182/blood-2004-11-4278.

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Abstract Not long after the recognition of HIV as the causative agent of AIDS, it was evident that individuals infected with HIV developed lymphoma at a greater rate than the population at large. Approximately two thirds of AIDS-related lymphoma (ARL) cases are categorized as diffuse large B-cell type, with Burkitt lymphomas comprising 25% and other histologies a much smaller proportion. Typically, these individuals have presented with advanced extranodal disease and CD4+ lymphocyte counts of less than 200/mm3. Recent clinical trials have demonstrated a better outcome with chemotherapy for ARL since the introduction of combination antiretroviral treatment, termed highly active antiretroviral therapy (HAART). For patients with relapses, solid evidence points to the safety and utility of hematopoietic-cell transplantation as a salvage modality. Coinfection with other viruses such as Epstein-Barr virus and Kaposi sarcoma-associated herpesvirus have led to the genesis of previously rare or unrecognized lymphoma subtypes such as plasmablastic and primary effusion lymphomas. The immunosuppressive impact of treatment for patients with ARL receiving chemotherapy with HAART appears transient and opportunistic infections have become less problematic than prior to HAART. Significant progress has been made in the understanding and management of ARL but outcomes still remain inferior compared to those achieved in HIV- individuals.

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13

Pinto, Amélia Nogueira. "AIDS and Cerebrovascular Disease". Stroke 27, n.º 3 (marzo de 1996): 538–43. http://dx.doi.org/10.1161/01.str.27.3.538.

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14

Williams, Jennet y Mark Bower. "AIDS-related malignant disease". Medicine 41, n.º 8 (agosto de 2013): 430–34. http://dx.doi.org/10.1016/j.mpmed.2013.05.013.

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15

Dalla Pria, Alessia y Mark Bower. "AIDS-related malignant disease". Medicine 46, n.º 6 (junio de 2018): 365–69. http://dx.doi.org/10.1016/j.mpmed.2018.03.002.

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16

Mönkemüller, Klaus E. y Martín Olmos. "Gastric disease in AIDS". Techniques in Gastrointestinal Endoscopy 4, n.º 2 (abril de 2002): 66–70. http://dx.doi.org/10.1053/tgie.2002.33013.

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17

Ames, Elizabeth D., Michael S. Conjalka, Arthur F. Goldberg, Richard Hirschman, Sushil Jain, Ariel Distenfeld y Craig E. Metroka. "Hodgkin's Disease and AIDS". Hematology/Oncology Clinics of North America 5, n.º 2 (abril de 1991): 343–56. http://dx.doi.org/10.1016/s0889-8588(18)30446-5.

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18

Lopez-Herce Cid, JoseAndres, Jesus Lopez-Herce Cid, EduardoFlores Sañudo, Javier Menarguez y JaimeCosin Ochaita. "AIDS AND HODGKIN'S DISEASE". Lancet 328, n.º 8515 (noviembre de 1986): 1104–5. http://dx.doi.org/10.1016/s0140-6736(86)90510-6.

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19

Temple, J. J. y W. Abe Andes. "AIDS AND HODGKIN'S DISEASE". Lancet 328, n.º 8504 (agosto de 1986): 454–55. http://dx.doi.org/10.1016/s0140-6736(86)92155-0.

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20

Kuo, Irene y Narsing A. Rao. "Ocular disease in AIDS". Springer Seminars in Immunopathology 21, n.º 2 (junio de 1999): 161–77. http://dx.doi.org/10.1007/bf00810248.

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21

Kuo, Irene y Narsing A. Rao. "Ocular disease in AIDS". Springer Seminars in Immunopathology 21, n.º 2 (26 de julio de 1999): 161–77. http://dx.doi.org/10.1007/s002810050060.

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22

Peppercorn, Mark A. "Anorectal disease in AIDS". Gastroenterology 104, n.º 6 (junio de 1993): 1883. http://dx.doi.org/10.1016/0016-5085(93)90684-5.

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23

Collins, Frank M. "AIDS-related mycobacterial disease". Springer Seminars in Immunopathology 10, n.º 4 (diciembre de 1988): 375–91. http://dx.doi.org/10.1007/bf02053847.

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24

Corti, Marcelo. "AIDS and Chagas' Disease". AIDS Patient Care and STDs 14, n.º 11 (noviembre de 2000): 581–88. http://dx.doi.org/10.1089/10872910050193752.

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25

Ives, David V. "Cytomegalovirus disease in AIDS". AIDS 11, n.º 15 (diciembre de 1997): 1791–97. http://dx.doi.org/10.1097/00002030-199715000-00002.

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26

Berger, Joseph R., Jonathan O. Harris, Jocelyn Gregoriost y Michael Norenbergt. "Cerebrovascular disease in AIDS". AIDS 4, n.º 3 (marzo de 1990): 239–44. http://dx.doi.org/10.1097/00002030-199003000-00010.

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27

Nyamathi, Adeline. "AIDS-related heart disease". Journal of Cardiovascular Nursing 3, n.º 4 (agosto de 1989): 65–76. http://dx.doi.org/10.1097/00005082-198908000-00009.

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28

Brettman, Lee R. "Liver disease in aids". Hepatology 7, n.º 2 (marzo de 1987): 403–5. http://dx.doi.org/10.1002/hep.1840070238.

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29

Wilcox, C. Mel y Klaus E. Mönkemüller. "Hepatobiliary Diseases in Patients with AIDS: Focus on AIDS Cholangiopathy and Gallbladder Disease". Digestive Diseases 16, n.º 4 (1998): 205–13. http://dx.doi.org/10.1159/000016868.

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30

Valenti, William M. "Update on AIDS". Infection Control 6, n.º 2 (febrero de 1985): 85–86. http://dx.doi.org/10.1017/s0195941700062664.

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Recent advances in our knowledge of the Acquired Immune Deficiency Syndrome (AIDS) could have a major impact on practitioners in both infection control and employee health. The discovery that a retrovirus, the human T-cell leukemia virus (HTLV-III) is the cause of AIDS could be used to help health care workers better understand this disease. Those of us who are trying to provide our employees with up-to-date information on AIDS should use this new information to help employees understand the nature of the disease, its transmissibility and non-transmissibility. Now that the disease can be linked to a specific agent, employees may find AIDS to be much less mysterious. The retrovirus link is a major step forward in understanding this disease and may eventually lead to more effective treatment and possibly a vaccine. Our knowledge continues to evolve in much the same way as our understanding of Legionnaires' Disease and Toxic Shock Syndrome.The Acquired Immune Deficiency Syndrome (AIDS) was discussed extensively at recent infectious diseases meetings in Washington, D.C. The results of original research conducted by investigators at the National Institutes of Health (NIH), Centers for Disease Control (CDC), Pasteur Institute in Paris, and medical centers throughout the world were presented at the meetings. A number of issues have been clarified and the pieces of the puzzle are starting to come together.

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31

Samuels, Hadas, Malki Malov, Trishna Saha Detroja, Karin Ben Zaken, Naamah Bloch, Meital Gal-Tanamy, Orly Avni, Baruh Polis y Abraham O. Samson. "Autoimmune Disease Classification Based on PubMed Text Mining". Journal of Clinical Medicine 11, n.º 15 (26 de julio de 2022): 4345. http://dx.doi.org/10.3390/jcm11154345.

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Autoimmune diseases (AIDs) are often co-associated, and about 25% of patients with one AID tend to develop other comorbid AIDs. Here, we employ the power of datamining to predict the comorbidity of AIDs based on their normalized co-citation in PubMed. First, we validate our technique in a test dataset using earlier-reported comorbidities of seven knowns AIDs. Notably, the prediction correlates well with comorbidity (R = 0.91) and validates our methodology. Then, we predict the association of 100 AIDs and classify them using principal component analysis. Our results are helpful in classifying AIDs into one of the following systems: (1) gastrointestinal, (2) neuronal, (3) eye, (4) cutaneous, (5) musculoskeletal, (6) kidneys and lungs, (7) cardiovascular, (8) hematopoietic, (9) endocrine, and (10) multiple. Our classification agrees with experimentally based taxonomy and ranks AID according to affected systems and gender. Some AIDs are unclassified and do not associate well with other AIDs. Interestingly, Alzheimer’s disease correlates well with other AIDs such as multiple sclerosis. Finally, our results generate a network classification of autoimmune diseases based on PubMed text mining and help map this medical universe. Our results are expected to assist healthcare workers in diagnosing comorbidity in patients with an autoimmune disease, and to help researchers in identifying common genetic, environmental, and autoimmune mechanisms.

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32

Janaki, CR, GS Selvi, B. Parveen, N. Gomathy y Anupama Roshan. "AIDS defining disease: Disseminated cryptococcosis". Indian Journal of Dermatology 51, n.º 3 (2006): 202. http://dx.doi.org/10.4103/0019-5154.27988.

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33

Northup, George W. "AIDS: Disease of the 80s". Journal of the American Osteopathic Association 85, n.º 11 (1 de noviembre de 1985): 46–47. http://dx.doi.org/10.1515/jom-1985-851110.

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34

Silberner, J. "AIDS: Disease, Research Efforts Advance". Science News 127, n.º 17 (27 de abril de 1985): 260. http://dx.doi.org/10.2307/3969504.

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35

Ryder, Mark I., Wipawee Nittayananta, Maeve Coogan, Deborah Greenspan y John S. Greenspan. "Periodontal disease in HIV/AIDS". Periodontology 2000 60, n.º 1 (22 de agosto de 2012): 78–97. http://dx.doi.org/10.1111/j.1600-0757.2012.00445.x.

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36

Lewis, Laurie. "AIDS as a Chronic Disease". AIDS Patient Care 3, n.º 3 (junio de 1989): 28–30. http://dx.doi.org/10.1089/apc.1989.3.28.

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37

Faria, PR, PA Vargas, PHN Saldiva, GM Böhm, T. Mauad y OP Almeida. "Tongue disease in advanced AIDS". Oral Diseases 11, n.º 2 (marzo de 2005): 72–80. http://dx.doi.org/10.1111/j.1601-0825.2004.01070.x.

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38

Cello, John P. "AIDS-Related Biliary Tract Disease". Gastrointestinal Endoscopy Clinics of North America 8, n.º 4 (octubre de 1998): 963–73. http://dx.doi.org/10.1016/s1052-5157(18)30242-3.

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39

Yu, Qianli, Douglas F. Larson y Ronald R. Watson. "Heart disease, methamphetamine and AIDS". Life Sciences 73, n.º 2 (mayo de 2003): 129–40. http://dx.doi.org/10.1016/s0024-3205(03)00260-1.

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40

Yeager, Barbara. "AIDS not an infectious disease?" Geriatric Nursing 12, n.º 1 (enero de 1991): 4. http://dx.doi.org/10.1016/s0197-4572(06)80299-5.

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41

Freeman, William R. "Retinal disease associated with AIDS*". Australian and New Zealand Journal of Ophthalmology 21, n.º 2 (mayo de 1993): 71–78. http://dx.doi.org/10.1111/j.1442-9071.1993.tb00757.x.

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42

Kohan, Darius, Paul E. Hammerschlag y Roy A. Holliday. "Otologic Disease in AIDS Patients". Laryngoscope 100, n.º 12 (diciembre de 1990): 1326???1330. http://dx.doi.org/10.1288/00005537-199012000-00016.

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43

Enns, R. "AIDS cholangiopathy: “an endangered disease”". American Journal of Gastroenterology 98, n.º 10 (octubre de 2003): 2111–12. http://dx.doi.org/10.1016/s0002-9270(03)00757-3.

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44

Kelly, R. D. "AIDS: Therapeutics in HIV Disease". Biochemical Education 16, n.º 4 (octubre de 1988): 238. http://dx.doi.org/10.1016/0307-4412(88)90146-x.

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45

Pratt, Robert J. "AIDS—Therapeutics in HIV disease". International Journal of Nursing Studies 26, n.º 1 (enero de 1989): 84. http://dx.doi.org/10.1016/0020-7489(89)90052-7.

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46

Weissler, Jonathan C. y Ann R. Mootz. "Pulmonary Disease in AIDS Patients". American Journal of the Medical Sciences 300, n.º 5 (noviembre de 1990): 330–43. http://dx.doi.org/10.1097/00000441-199011000-00010.

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47

Lowe, G. D. O. "AIDS—therapeutics of HIV disease". Fibrinolysis 2, n.º 4 (octubre de 1988): 259. http://dx.doi.org/10.1016/0268-9499(88)90022-7.

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48

Enns, Robert. "AIDS cholangiopathy: "an endangered disease"". American Journal of Gastroenterology 98, n.º 10 (octubre de 2003): 2111–12. http://dx.doi.org/10.1111/j.1572-0241.2003.07724.x.

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49

Brettle, R. P. "AIDS therapeutics in HIV disease". Sexually Transmitted Infections 64, n.º 6 (1 de diciembre de 1988): 398. http://dx.doi.org/10.1136/sti.64.6.398-b.

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50

Coleman, D. G., E. J. Beck, B. S. Peters, J. R. Harris y A. J. Pinching. "Changing disease patterns in AIDS." BMJ 304, n.º 6830 (28 de marzo de 1992): 839. http://dx.doi.org/10.1136/bmj.304.6830.839-a.

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