Bibliografías temáticas / AHAS/ALS inhibitor resistance

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Literatura académica sobre el tema "AHAS/ALS inhibitor resistance"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 18 de febrero de 2022

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Artículos de revistas sobre el tema "AHAS/ALS inhibitor resistance"

1

Gerwick, B. Clifford, Linda C. Mireles y Robert J. Eilers. "Rapid Diagnosis of Als/Ahas-Resistant Weeds". Weed Technology 7, n.º 2 (junio de 1993): 519–24. http://dx.doi.org/10.1017/s0890037x00027986.

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A method to rapidly identify acetolactate synthase/acetohydroxyacid synthase (ALS/AHAS)-resistant weeds is described based upon the differential accumulation of acetoin in the presence and absence of an ALS/AHAS inhibitor herbicide. Acetoin accumulation is induced by inhibition of ketol-acid reductoisomerase (KARI), the enzyme immediately following ALS/AHAS in the biosynthesis of branched-chain amino acids. Inhibition of ALS/AHAS prevents the build up of acetoin and forms the basis for distinguishing between sensitive and resistant biotypes. A new inhibitor of KARI, 1,1-cyclopropanedicarboxylic acid (CPCA), is described and was found to cause acetoin accumulation in velvetleaf leaf disks over the concentration range of 2 to 100 000 μM. In the presence of CPCA, a number of species important to monitor for ALS/AHAS resistance were found to accumulate acetoin at rates sufficient for resistance diagnosis in 2 to 8 h. In velvetleaf, the youngest apical leaf was found to be the most active in acetoin accumulation. The resistance diagnosis method was validated by clearly distinguishing between imazaquin-sensitive and imazaquin-resistant cocklebur biotypes.
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Lebaron, Homer M. "Weed Science in the 1990s: Will It be Forward or in Reverse?" Weed Technology 4, n.º 3 (septiembre de 1990): 671–89. http://dx.doi.org/10.1017/s0890037x00026208.

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Dr. Homer M. LeBaron is Senior Research Fellow in the New Technology and Basic Research Department of CIBA-GEIGY Corporation, where He has the responsibility for coordinating and directing outside basic research on all of CIBA-GEIGY agricultural products. He has been employed in various R&D positions with Geigy and CIBA-GEIGY for 27 years. From 1960 to 1964, Dr. LeBaron was employed as a plant physiologist at the Virginia Tech Experiment Station in Norfolk, Virginia, mainly researching weed problems in vegetables and fruit crops.LeBaron was born May 13, 1926 in Southern Alberta, Canada, the third in a family of 10 children, and grew up on a diversified irrigation farm. He obtained his B.S. and M.S. degrees from Utah State University in 1955 and 1957. He received his Ph.D. degree from Cornell in 1960.LeBaron, in addition to WSSA, is a member of the American Society of Agronomy, American Chemical Society, Entomological Society of America, American Phytopathological Society, CAST, Sigma Xi, Aquatic Plant Management Society, European Weed Research Society, and all of the regional weed science societies.He has held numerous positions in several of these and other scientific societies. Homer served as president of NEWSS in 1969–70 and as president of the SWSS in 1986–87. He served on several WSSA committees, including the Executive Board of Directors. In 1978, Dr. LeBaron was elected a Fellow in the WSSA, and received the Distinguished Service Award in the SWSS in 1984.He is author of over 70 scientific publications, and has been editor and author of five books on herbicide and pesticide resistance and biotechnology. He is currently serving on the parent Herbicide Resistance Action Committee (HRAC) under GIFAP, as well as on the ALS/AHAS Inhibitors Resistance Working Group and as Chairman of the Triazine Resistance Working Group. He is on the Planning Committee and Co-Chair of the Weed Resistance Management Working Group within the International Organization for Pest Resistance Management (IOPRM). In addition to his busy professional schedule, Dr. LeBaron has always been involved in church and community affairs. He has 7 children and 20 grandchildren.
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Yu, Qin y Stephen B. Powles. "Resistance to AHAS inhibitor herbicides: current understanding". Pest Management Science 70, n.º 9 (20 de enero de 2014): 1340–50. http://dx.doi.org/10.1002/ps.3710.

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Norazua, Zakaria, Ahmad-Hamdani Muhammad Saiful y Juraimi Abdul Shukor. "Patterns of resistance to AHAS inhibitors in Limnocharis flava from Malaysia". Plant Protection Science 54, No. 1 (24 de noviembre de 2017): 48–59. http://dx.doi.org/10.17221/131/2016-pps.

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Limnocharis flava (L.) Buchenau is among the most problematic rice weeds in Malaysia and is also reported to have developed multiple resistance to AHAS inhibitor bensulfuron-methyl and synthetic auxin 2,4-D. In this study, resistance across different AHAS inhibitors was characterised in a L. flava population infesting rice fields in Pulau Pinang, Malaysia. Dose-response experiments were conducted to determine the level of resistance to sulfonylureas, imidazolinone, triazolopyrimidine, and pyrimidinyl-thiobenzoate. Cross-resistance across different AHAS inhibitors was observed in the resistant L. flava population, exhibiting a high level of resistance to bensulfuron-methyl, while exhibiting a moderate level of resistance to metsulfuron-methyl and a low level of resistance to pyrazosulfuron-ethyl and pyribenzoxim. However, all resistant L. flava individuals were still sensitive to imazethapyr, penoxsulam, and bispyribac-sodium. Based on the results, it is likely that resistance to AHAS inhibitors in L. flava is conferred by target-site resistance mechanisms.
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Rodriguez, Jeanette, Amber Hauvermale, Arron Carter, Rachel Zuger y Ian C. Burke. "An ALA 122 THR substitution in the AHAS / ALS gene confers imazamox‐resistance in Aegilops cylindrica". Pest Management Science 77, n.º 10 (25 de junio de 2021): 4583–92. http://dx.doi.org/10.1002/ps.6498.

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Eberlein, Charlotte V., Mary J. Guttieri, Philip H. Berger, John K. Fellman, Carol A. Mallory-Smith, Donn C. Thill, Roger J. Baerg y William R. Belknap. "Physiological consequences of mutation for ALS-inhibitor resistance". Weed Science 47, n.º 4 (agosto de 1999): 383–92. http://dx.doi.org/10.1017/s0043174500091967.

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Biochemical and physiological effects of target site resistance to herbicides inhibiting acetolactate synthase (ALS) were evaluated using sulfonylurea-resistant (R) and -susceptible (S) near isonuclearLactuca sativa‘Bibb’ lines derived by backcrossing the resistance allele fromLactuca serriolaL. intoL. sativa.Sequence data suggest that resistance inL. sativais conferred by a single-point mutation that encodes a proline197to histidine substitution in Domain A of the ALS protein; this is the same substitution observed in RL. serriola. Kmapp(pyruvate) values for ALS isolated from R and SL. sativawere 7.3 and 11.1 mM, respectively, suggesting that the resistance allele did not alter the pyruvate binding domain on the ALS enzyme. Both R and S ALS had greater affinity for 2-oxobutyrate than for pyruvate at the second substrate site. Ratios of acetohydroxybutyrate: acetolactate produced by R ALS across a range of 2-oxobutyrate concentrations were similar to acetohydroxybutyrate: acetolactate ratios produced by S ALS. Specific activity of ALS from RL. sativawas 46% of the specific activity from SL. sativa, suggesting that the resistance allele has detrimental effects on enzyme function, expression, or stability. ALS activity from R plants was less sensitive to feedback inhibition by valine, leucine, and isoleucine than ALS from S plants. Valine, leucine, and isoleucine concentrations were about 1.5 times higher in R seed than in S seed on a per gram of seed basis, and concentrations of valine and leucine were 1.3 and 1.6 times higher, respectively, in R leaves than in S leaves. Findings suggest that the mutation for resistance results in altered regulation of branched-chain amino acid synthesis.
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Beckie, Hugh J. y Xavier Reboud. "Selecting for Weed Resistance: Herbicide Rotation and Mixture". Weed Technology 23, n.º 3 (septiembre de 2009): 363–70. http://dx.doi.org/10.1614/wt-09-008.1.

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Herbicide rotations and mixtures are widely recommended to manage herbicide resistance. However, little research has quantified how these practices actually affect the selection of herbicide resistance in weeds. A 4-yr experiment was conducted in western Canada from 2004 to 2007 to examine the impact of herbicide rotation and mixture in selecting for acetolactate synthase (ALS) inhibitor resistance in the annual broadleaf weed, field pennycress, co-occurring in wheat. Treatments consisted of the ALS-inhibitor herbicide, ethametsulfuron, applied in a mixture with bromoxynil/MCPA formulated herbicide (photosystem-II inhibitor/synthetic auxin), or in rotation with the non-ALS inhibitor at an ALS-inhibitor application frequency of 0, 25, 50, 75, and 100% (i.e., zero to four applications, respectively) over the 4-yr period. The field pennycress seed bank at the start of the experiment contained 5% ethametsulfuron-resistant seed. Although weed control was only marginally reduced, resistance frequency of progeny of survivors increased markedly after one ALS-inhibitor application. At the end of the experiment, the level of resistance in the seed bank was buffered by susceptible seed, increasing from 29% of recruited seedlings after one application to 85% after four applications of the ALS inhibitor. The level of resistance in the seed bank for the mixture treatment after 4 yr remained similar to that of the nontreated (weedy) control or 0% ALS-inhibitor rotation frequency treatment. The results of this study demonstrate how rapidly ALS-inhibitor resistance can evolve as a consequence of repeated application of herbicides with this site of action, and supports epidemiological information from farmer questionnaire surveys and modeling simulations that mixtures are more effective than rotations in mitigating resistance evolution through herbicide selection.
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Beckie, H. J., L. M. Hall, F. J. Tardif y G. Séguin-Swartz. "Acetolactate synthase inhibitor-resistant stinkweed (Thlaspi arvense L.) in Alberta". Canadian Journal of Plant Science 87, n.º 4 (1 de octubre de 2007): 965–72. http://dx.doi.org/10.4141/cjps06019.

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Two stinkweed populations from southern and central Alberta were not controlled by acetolactate synthase (ALS)-inhibiting herbicides in 2000. This study reports on their cross-resistance to ALS-inhibiting herbicides, molecular basis of resistance, and inheritance of resistance. Both putative herbicide-resistant biotypes responded similarly to increasing doses of the herbicides. The biotypes were highly resistant to ethametsulfuron and exhibited a low level of resistance to metsulfuron and imazethapyr. However, both biotypes were not resistant to florasulam, a triazolopyrimidine ALS inhibitor, or sulfometuron, a non-selective sulfonylurea ALS inhibitor. The cross-resistance pattern was consistent with the confirmed target-site mutation. Sequence analysis of the ALS gene detected a Pro197Leu mutation in both biotypes. Similar to many other ALS inhibitor-resistant weed biotypes, resistance was conferred by a single dominant gene. This study confirms the first global occurrence of herbicide resistance in this species. Key words: ALS-inhibitor resistance, ALS sequence, herbicide resistance, target-site mutation
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Shergill, Lovreet S., Jenna Malone, Peter Boutsalis, Christopher Preston y Gurjeet Gill. "Basis of ACCase and ALS inhibitor resistance inHordeum glaucumSteud". Pest Management Science 73, n.º 8 (15 de febrero de 2017): 1638–47. http://dx.doi.org/10.1002/ps.4501.

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Osuna, M. D. y R. De Prado. "Conyza albida : a new biotype with ALS inhibitor resistance". Weed Research 43, n.º 3 (9 de mayo de 2003): 221–26. http://dx.doi.org/10.1046/j.1365-3180.2003.00337.x.

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Tesis sobre el tema "AHAS/ALS inhibitor resistance"

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Friesen, Lincoln Jacob Shane. "Identification of the mechanisms of wild radish herbicide resistance to PSII inhibitors, auxinics, and AHAS inhibitors". University of Western Australia. School of Plant Biology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0106.

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The objective of this Ph.D. research was to identify new and novel mechanisms of wild radish (Raphanus raphanistrum L.) resistance to photosystem II (PSII) inhibitors, auxinics, and acetohydroxyacid synthase (AHAS) inhibitors. PSIIinhibitor resistance was demonstrated to be target-site based, and conferred by a Ser264 to Gly substitution of the D1 protein. Auxinic resistance was associated with reduced herbicide translocation to the meristematic regions of resistant wild radish plants. Two new resistance mutations of wild radish AHAS were discovered, including one encoding the globally rare Asp376 to Glu substitution, and another encoding an Ala122 to Tyr substitution, which has never been identified or assessed for resistance in plants previously. Characterization of the frequency and distribution of AHAS resistance mutations in wild radish from the WA wheatbelt revealed that Glu376 was widespread, and that some mutations of AHAS are more common than others. Computer simulation was used to examine the molecular basis of resistance-endowing AHAS target-site mutations. Furthermore, through the computer-aided analysis, residues were identified with the potential to confer resistance upon substitution, but which have not previously been assessed for this possibility. Results from this Ph.D. research demonstrate that diverse, unrelated mechanisms of resistance to PSII inhibitors, auxinics, and AHAS inhibitors have evolved in wild radish of the WA wheatbelt, and that these mechanisms have accumulated in some populations.
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2

Ferguson, Gabrielle Mary. "ALS-inhibitor resistance in populations of Amaranthus powellii S. Wats. and Amaranthus retroflexus L". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0023/MQ51062.pdf.

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Iwakami, Satoshi. "Molecular mechanism of resistance in a multiple-herbicide resistant Echinochloa phyllopogon". Kyoto University, 2013. http://hdl.handle.net/2433/180368.

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Kyoto University (京都大学)
0048
新制・課程博士
博士(農学)
甲第17830号
農博第2015号
新制||農||1016(附属図書館)
学位論文||H25||N4787(農学部図書室)
30645
京都大学大学院農学研究科農学専攻
(主査)教授 稲村 達也, 教授 冨永 達, 教授 奥本 裕
学位規則第4条第1項該当
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4

Weerasooriya, Dilooshi Kumari. "Genetic analysis of interveinal chlorosis and reduced seedling vigor as related to agronomic performance in sorghum resistant to ALS inhibitor herbicides". Diss., Kansas State University, 2016. http://hdl.handle.net/2097/32896.

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Doctor of Philosophy
Department of Agronomy
Tesfaye T. Tesso
The lack of effective post-emergence weed control options is often highlighted as one of the major factors behind dwindling acreage under sorghum (Sorghum bicolor (L.) Moench) in the United States. The discovery of herbicide resistance sources in wild sorghum population and subsequent efforts to incorporate them into cultivated sorghum was received with much optimism to change weed management practices in sorghum. As the development of the technology advances, especially of the Acetolactate synthase (ALS) resistance, concerns over the temporary interveinal chlorosis and reduced seedling vigor in some of the resistant families became heightened. This thesis research is designed to shed light on the genetic basis of seedling chlorosis and assess its impacts on yield potential. The study has three parts; the first part is focused on identifying the genetic causes and plant mechanisms associated with the chlorotic phenotype. ALS herbicide resistant sister-lines expressing normal and chlorotic phenotypes were analyzed via RNA sequencing at four time points during seedling growth. The study identified several variants of genes coding chloroplast precursors and those that cause epigenetic modifications. Once confirmed, genetic markers can be developed to track these gene variants in the breeding population and eliminate segregates genetically prone to chlorosis/yellowing. The second part of the study focuses on assessing the effect of ALS resistance associated chlorosis on agronomic and nutritional parameters of sorghum inbred lines. A set of ALS resistant lines expressing different levels of the chlorotic phenotype were evaluated in replicated field trials and laboratory methods. Results showed that interveinal chlorosis delays flowering but does not have negative effect on yield and nutritional parameters with and without herbicide treatment. The last part addresses whether there is any yield drag that may be associated with herbicide resistance traits and foliar interveinal chlorosis. For this, we synthesized a large set (182) of hybrids from ALS resistant, ACCase resistant and regular (susceptible) seed and pollinator parents. The hybrids were then evaluated in three sets at multiple locations during the 2014 and 2015 crop seasons along with commercial checks. The results revealed that resistance to both herbicides do not cause any drag to grain yield. The traits also do not have any negative impact on grain and nutritional quality of resistant hybrids.
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Kershner, Kellan Scott. "Herbicide resistance in grain sorghum". Diss., Kansas State University, 2010. http://hdl.handle.net/2097/13069.

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Doctor of Philosophy
Department of Agronomy
Kassim Al-Khatib
Mitchell R. Tuinstra
Sorghum acreage is declining throughout the United States because management options and yield have not maintained pace with maize improvements. The most extreme difference has been the absence of herbicide technology development for sorghum over the past twenty years. The objectives of this study were to evaluate the level of resistance, type of inheritance, and causal mutation of wild sorghums that are resistant to either acetyl-coenzyme A carboxylase (ACCase)-inhibiting herbicides or acetohydroxyacid synthase (AHAS)-inhibiting herbicides. ACCase-inhibiting herbicides used in this study were aryloxyphenoxypropionate (APP) family members fluazifop-P and quizalofop-P along with cyclohexanedione (CHD) family members clethodim and sethoxydim. The level of resistance was very high for APP herbicides but low to nonexistent to CHD herbicides. With genetic resistance to APP herbicides, the resistance factors, the ratio of resistance to susceptible, were greater than 54 to 64 for homozygous individuals and greater than 9 to 20 for heterozygous individuals. Resistance to CHD herbicides was very low with resistance factors ranging from one to about five. Genetic segregation studies indicate a single gene is the cause of resistance to APP herbicides. Sequencing identified a single mutation that results in cysteine replacing tryptophan (Trp-2027-Cys). Trp-2027-Cys has previously been reported to provide resistance to APP but not CHD herbicides. The other wild sorghum evaluated in this study was resistant to AHAS-inhibiting herbicides including imidazolinone (IM) family member, imazapyr, and sulfonylurea (SU) family member, nicosulfuron. Resistance factors in this genotype were very high, greater than 770 for the IM herbicide and greater than 500 for the SU herbicide, for both herbicide chemical families. Genetic segregation studies demonstrate that resistance was controlled by one major locus and two modifier loci. DNA sequencing of the AHAS gene identified two mutations, Val-560-Ile and Trp-574-Leu. Val-560-Ile is of unknown importance, but valine and isoleucine are similar and residue 560 is not conserved. Trp-574 is a conserved residue and Leu-574 is a known mutation that provides strong cross resistance to IM and SU herbicides. The results of these studies suggest that these sources of APP, SU, and IM resistance may provide useful herbicide resistance traits for use in sorghum.
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6

Park, Kee-Woong. "ALS-inhibitor resistant downy brome (Bromus tectorum L.) biotypes in Oregon : mechanism of resistance, fitness, and competition". Thesis, 2003. http://hdl.handle.net/1957/30435.

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Capítulos de libros sobre el tema "AHAS/ALS inhibitor resistance"

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Eberlein, C. V., M. J. Guttieri, C. A. Mallory-Smith y D. C. Thill. "Effects of Mutation for ALS-Inhibitor Resistance on ALS Activity in Resistant and Susceptible Near-Isonuclear Lactuca Lines". En Weed and Crop Resistance to Herbicides, 191–97. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5538-0_21.

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Actas de conferencias sobre el tema "AHAS/ALS inhibitor resistance"

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Ouyang, Xiaoming, Ashley Barling, Aletha Lesch, Jeffrey Tyner, Sophia Jeng, Christina Zheng, Sara A. Courtneidge, Shannon McWeeney y Molly Kulesz-Martin. "Abstract 78: Inhibiting anaplastic lymphoma kinase (ALK) overcomes epidermal growth factor receptor (EGFR) inhibitor resistance in head and neck squamous cell carcinoma (HNSCC) patient-derived models". En Abstracts: AACR-AHNS Head and Neck Cancer Conference: Optimizing Survival and Quality of Life through Basic, Clinical, and Translational Research; April 23-25, 2017; San Diego, CA. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1557-3265.aacrahns17-78.

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