Relevant bibliographies by topics / Wag/Rij

Academic literature on the topic 'Wag/Rij'

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Published: 11 March 2023

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Journal articles on the topic "Wag/Rij"

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Габова, А. В., К. Ю. Саркисова, Е. А. Федосова, А. Б. Шацкова, and А. А. Морозов. "ВОЗРАСТНЫЕ ИЗМЕНЕНИЯ ПИК-ВОЛНОВЫХ РАЗРЯДОВ У КРЫС ЛИНИИ WAG/Rij С ГЕНЕТИЧЕСКОЙ АБСАНСНОЙ ЭПИЛЕПСИЕЙ, "Российский физиологический журнал им. И.М. Сеченова"." Российский физиологический журнал им. И. М. Сеченова, no. 10 (2018): 1176–89. http://dx.doi.org/10.7868/s0869813918100052.

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Крысы линии WAG/Rij являются моделью генетической абсансной эпилепсии, характеризующейся наличием пик-волновых разрядов (ПВР) на ЭЭГ. Известно, что первые ПВР у крыс линии WAG/Rij возникают в возрасте 2-3 месяцев, а в дальнейшем их число и длительность увеличиваются. Однако эволюция ПВР в процессе прогрессивного развития абсансной эпилепсии у крыс линии WAG/Rij остается неисследованной. Цель настоящей работы - выяснить возрастные изменения частотно-временной динамики, частотного спектра и морфологических характеристик ПВР у крыс линии WAG/Rij. Для достижения этой цели у одних и тех же крыс в возрасте от 2 до 12 месяцев исследовали эволюцию ПВР. Установлено, что ПВР формируются в возрасте 2-4 месяцев, в дальнейшем наблюдают морфологические изменения ПВР. Показано, что в процессе прогрессивного развития абсансной эпилепсии ПВР проходят 3 стадии «созревания». Предполагается, что связанная с возрастом эволюция ПВР у крыс линии WAG/Rij является отражением прогрессивных электрофизиологических изменений в соматосенсорной коре - области мозга, с которой связывают генерацию и генерализацию ПВР.
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Zhuravlev, Maxim, Anastasiya Runnova, Kirill Smirnov, and Evgenia Sitnikova. "Spike-Wave Seizures, NREM Sleep and Micro-Arousals in WAG/Rij Rats with Genetic Predisposition to Absence Epilepsy: Developmental Aspects." Life 12, no. 4 (April 12, 2022): 576. http://dx.doi.org/10.3390/life12040576.

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The current study was done in Wistar Albino Glaxo Rijswijk (WAG/Rij) rats, which are genetically prone to develop spontaneous spike-wave discharges (SWDs) and are widely used as a genetic model of absence epilepsy. Here, we examined functional links between sleep and spike-wave epilepsy in aging WAG/Rij rats using advanced techniques of EEG analysis. SWDs, periods of NREM sleep and micro-arousals were automatically detected in three-channel epidural EEG recorded in freely moving WAG/Rij rats consequently at the age 5, 7 and 9 months. We characterized the developmental profile of spike-wave epilepsy in drug-naïve WAG/Rij rats and defined three epi-phenotypes—severe, mild and minor epilepsy. Age-related changes of SWDs were associated with changes in NREM sleep. Several signs of NREM sleep fragmentation were defined in epileptic WAG/Rij rats. It seems that spike-wave epilepsy per se promotes micro-arousals during NREM sleep. However, subjects with a higher number of micro-arousals (and NREM sleep episodes) at the age of 5 months were characterized by a reduction of SWDs between 5 and 7 months of age.
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Ilbay, Gul, Aymen Balıkcı, Sibel Köktürk, Melda Yardımoglu Yılmaz, Nurbay Ates, Canan Baydemır, and Sibel Balcı. "Neonatal Tactile Stimulation Downregulates Dendritic Spines in Layer V Pyramidal Neurons of the WAG/Rij Rat Somatosensory Cortex." Neural Plasticity 2022 (April 12, 2022): 1–9. http://dx.doi.org/10.1155/2022/7251460.

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Objective. The aim of our study is to examine the effects of neonatal tactile stimulations on the brain structures that previously defined as the focus of epilepsy in the Wistar-Albino-Glaxo from Rijswijk (WAG/Rij) rat brain with genetic absence epilepsy. Methods. In the present research, morphology and density of dendritic spines were analyzed in layer V pyramidal neurons of the somatosensory cortex (SoCx) of WAG/Rij rats (nonstimulated control, tactile-stimulated, and maternal separated rats) and healthy Wistar (nonepileptic) rats. To achieve this, a Golgi-Cox method was used. Results. Dendritic spine number in layer V of the SoCx has been detected significantly higher in adult WAG/Rij rats at postnatal day 150 in comparison to nonepileptic adult control Wistar rats ( p < 0.001 ). Moreover, quantitative analyses of dendrite structure in adult WAG/Rij rats showed a decrease in dendrite spine density of pyramidal neurons of SoCx which occurred in early neonatal exposure to maternal separation (MS) and tactile stimulation (TS) ( p < 0.001 ). Conclusions. Our findings provide the first evidence that tactile stimulations during the early postnatal period have a long-term impact on dendrite structure in WAG/Rij rat’s brain and demonstrate that neonatal tactile stimulation can regulate dendritic spines in layer V in pyramidal neurons of SoCx in epileptic brains.
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Musina, L. A., S. S. Baigil'din, and Z. R. Khismatullina. "Morphological study of the retina of WAG/Rij rats with pigmentary degeneration in postnatal ontogenesis." Journal of Anatomy and Histopathology 9, no. 3 (October 10, 2020): 42–48. http://dx.doi.org/10.18499/2225-7357-2020-9-3-42-48.

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The aim of the study was to detect morphofunctional features of the retina of WAG/Rij rats during postnatal development from the 1st to the 360th day after birth.Material and methods. The study included retina of the inbred WAG/Rij rats (60 eyeballs from 30 rats totally) from the 1st to the 360th day of life. Standard histological studies were performed on paraffin sections stained with hematoxylin and eosin. Immunohistochemical method was used to determine the expression of acidic glial fibrillar protein GFAP in the rat retina. Mouse monoclonal antibodies (Santa Cruz Biotechnology) and a universal secondary detection system (NovocastraTM) were used for imaging. The degree of protein expression in the retina of WAG/Rij rats was compared in different age groups.Results. It was found that after birth, the retina of rat models of the WAG/Rij line is formed in the same way as the retina of rats of other strains and acquires a definitive structure only by the end of the second week (correlates with the opening of the eyes). On the 20th day, the first signs of dystrophic and destructive processes appear in the retina of WAG / Rij rats progressing as they grow older and leading to retinal gliosis. The increase in the expression of acidic glial fibrillar protein GFAP begins from the 30th day and increases with age as destructive processes in the retina increase. Conclusion. The early postnatal development of the retina of the WAG/Rij rats, which correlates in character with the postnatal retina development of rats of other strains, is interrupted by the launch of destructive processes in the retina soon after its complete differentiation. The further intensifying cascade of degeneration over time leads to the death of retinal neurons and their replacement by glial cells.
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Celikyurt, Ipek Komsuoglu, Guner Ulak, Oguz Mutlu, Furuzan Yildiz Akar, Faruk Erden, and Sezer Sener Komsuoglu. "Lamotrigine effects sensorimotor gating in WAG/Rij rats." Journal of Neurosciences in Rural Practice 03, no. 02 (May 2012): 126–30. http://dx.doi.org/10.4103/0976-3147.98207.

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ABSTRACTIntroduction: Prepulse inhibition (PPI) is a measurable form of sensorimotor gating. Disruption of PPI reflects the impairment in the neural filtering process of mental functions that are related to the transformation of an external stimuli to a response. Impairment of PPI is reported in neuropsychiatric illnesses such as schizophrenia, Huntington’s disease, Parkinson’s diseases, Tourette syndrome, obsessive compulsive disorder, and temporal lobe epilepsy with psychosis. Absence epilepsy is the most common type of primary generalized epilepsy. Lamotrigine is an antiepileptic drug that is preferred in absence epilepsy and acts by stabilizing the voltage-gated sodium channels. Aim: In this study, we have compared WAG-Rij rats (genetically absence epileptic rats) with Wistar rats, in order to clarify if there is a deficient sensorimotor gating in absence epilepsy, and have examined the effects of lamotrigine (15, 30 mg/kg, i.p.) on this phenomenon. Materials and Methods: Depletion in PPI percent value is accepted as a disruption in sensory-motor filtration function. The difference between the Wistar and WAG/Rij rats has been evaluated with the student t test and the effects of lamotrigine on the PPI percent have been evaluated by the analysis of variance (ANOVA) post-hoc Dunnett’s test. Results: The PPI percent was low in the WAG/Rij rats compared to the controls (P<0.0001, t:9,612). Although the PPI percent value of the control rats was not influenced by lamotrigine, the PPI percent value of the WAG/Rij rats was raised by lamotrigine treatment (P<0.0001, F:861,24). Conclusions: As a result of our study, PPI was disrupted in the WAG/Rij rats and this disruption could be reversed by an antiepileptic lamotrigine.
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Ilbay, Gul, Zeynep Ikbal Dogan, Aymen Balıkcı, Seyda Erdogan, and Akfer Karaoglan Kahilogulları. "Effects of Postnatal Caffeine Exposure on Absence Epilepsy and Comorbid Depression: Results of a Study in WAG/Rij Rats." Brain Sciences 12, no. 3 (March 8, 2022): 361. http://dx.doi.org/10.3390/brainsci12030361.

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The present study aims to investigate effect of early caffeine exposure on epileptogenesis and occurrence of absence seizures and comorbid depression in adulthood. For this purpose, Wistar Albino Glaxo from Rijswijk (WAG/Rij) rats were enrolled in a control and two experimental groups on the 7th day after the delivery. The rats in experimental groups received either 10 or 20 mg/kg caffeine subcutaneously while animals in control group had subcutaneous injections of 0.9% saline. The injections started at postnatal day 7 (PND7) and were continued each day for 5 days. At 6–7 months of age, electroencephalogram (EEG) recordings and behavioral recordings in the forced swimming test, sucrose consumption/preference test and locomotor activity test were carried out. At 6 months of age, 10 mg/kg and 20 mg/kg caffeine-treated WAG/Rij rats showed increased immobility latency and active swimming duration in forced swimming test when compared with the untreated controls. In addition, 20 mg/kg caffeine treatment decreased immobility time. In sucrose preference/consumption tests, WAG/Rij rats in 10 mg/kg caffeine group demonstrated higher sucrose consumption and preference compared to untreated controls. The rats treated with 20 mg/kg caffeine showed higher sucrose preference compared to control rats. The exploratory activity of rats in the 10 mg/kg caffeine-treated group was found to be higher than in the 20 mg/kg caffeine-treated and control groups in the locomotor activity test. At 7 months of age, caffeine-treated animals showed a decreased spike-wave discharge (SWD) number compared to the control animals. These results indicate that postnatal caffeine treatment may decrease the number of seizure and depression-like behaviors in WAG/Rij rats in later life. Caffeine blockade of adenosine receptors during the early developmental period may have beneficial effects in reducing seizure frequency and depression-like behaviors in WAG/Rij rat model.
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De Miguel, M., J. M. Fernández-Santos, I. Trigo-Sánchez, I. Matera, I. Ceccherini, I. Martín, G. Romeo, and H. Galera-Davidson. "The Ret proto-oncogene in the WAG/Rij rat strain: an animal model for inherited C-cell carcinoma?" Laboratory Animals 37, no. 3 (July 1, 2003): 215–21. http://dx.doi.org/10.1258/002367703766453065.

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WAG/Rij rat strain has been suggested as an animal model for the study of inherited human medullary thyroid carcinoma (MTC), due to its high incidence of spontaneous C-cell thyroid tumours. Although the role of the Ret proto-oncogene mutations, as responsible for human MTC, is well established, nothing has been published concerning this putative animal model. Based upon the previously reported rat Ret sequence, exons 10, 11, 13, 14, 15, and 16, known to carry activating mutations in humans, have been analysed in the WAG/Rij rat by PCR, single strand conformational polymorphism (SSCP) and direct sequencing. Neither the germline nor MTC samples showed any Ret sequence difference in the exons when analysed in comparison to a non-MTC-susceptible rat strain. Our results indicate that Ret exons relevant in humans are not involved in WAG/Rij rat MTC, as expected, and this questions the validity of this strain as a model for the human disease, and suggests there must be additional mechanisms for the genesis and progression of rat MTC.
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Poolos, Nicholas P. "H-Channel Dysfunction in Generalized Epilepsy: It Takes Two." Epilepsy Currents 6, no. 3 (May 2006): 88–90. http://dx.doi.org/10.1111/j.1535-7511.2006.00107.x.

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Impaired Regulation of Thalamic Pacemaker Channels Through an Imbalance of Subunit Expression in Absence Epilepsy Budde T, Caputi L, Kanyshkova T, Staak R, Abrahamczik C, Munsch T, Pape HC J Neurosci 2005;25(43):9871–9882 The role of hyperpolarization-activated, cyclic nucleotide-modulated (HCN) channel isoforms and hyperpolarization-activated cation current ( Ih) for seizure-related burst firing in thalamocortical (TC) neurons was investigated in a rat genetic model of absence epilepsy [Wistar Albino Glaxo rats, bred in Rijswijk (WAG/Rij)]. Burst discharges in TC neurons locked to seizure activity in vivo were prolonged during blockade of Ih by Cs+ and ZD7288 (4-ethylphenylamino-1,2-dimethyl-6-methylaminopyrimidinium chloride). In vitro analyses revealed a hyperpolarizing shift of half-maximal Ih activation ( Vh) in WAG/Rij ( Vh = −93.2 mV) compared with nonepileptic controls [August × Copenhagen-Irish (ACI) ( Vh = −88.0 mV)]. This effect is explained by a shift of the responsiveness of Ih to cAMP toward higher concentrations in TC neurons from WAG/Rij, as revealed by application of 8-bromo-cAMP and the phosphodiesterase inhibitor IBMX. During blockade of adenylyl cyclase activity, Ih activation was similar in the two strains, whereas the difference in cAMP responsiveness persisted, thereby voting against different ambient cAMP levels between strains. Increasing the intracellular cAMP level and shifting Ih activation led to a change from burst to tonic firing mode in WAG/Rij but not in ACI rats. Furthermore, HCN1 expression was significantly increased on mRNA and protein levels, with no changes in HCN2–4 expression. In conclusion, there is an increase in HCN1 expression in the epileptic thalamus, associated with a decrease in cAMP responsiveness of Ih in TC neurons and resulting impairment to control the shift from burst to tonic firing, which, in turn, will prolong burst activity after recruitment of Ih during absence seizures.
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Sitnikova, Evgenia, Alexander E. Hramov, Vadim Grubov, and Alexey A. Koronovsky. "Age-Dependent Increase of Absence Seizures and Intrinsic Frequency Dynamics of Sleep Spindles in Rats." Neuroscience Journal 2014 (June 23, 2014): 1–6. http://dx.doi.org/10.1155/2014/370764.

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The risk of neurological diseases increases with age. In WAG/Rij rat model of absence epilepsy, the incidence of epileptic spike-wave discharges is known to be elevated with age. Considering close relationship between epileptic spike-wave discharges and physiologic sleep spindles, it was assumed that age-dependent increase of epileptic activity may affect time-frequency characteristics of sleep spindles. In order to examine this hypothesis, electroencephalograms (EEG) were recorded in WAG/Rij rats successively at the ages 5, 7, and 9 months. Spike-wave discharges and sleep spindles were detected in frontal EEG channel. Sleep spindles were identified automatically using wavelet-based algorithm. Instantaneous (localized in time) frequency of sleep spindles was determined using continuous wavelet transform of EEG signal, and intraspindle frequency dynamics were further examined. It was found that in 5-months-old rats epileptic activity has not fully developed (preclinical stage) and sleep spindles demonstrated an increase of instantaneous frequency from beginning to the end. At the age of 7 and 9 months, when animals developed matured and longer epileptic discharges (symptomatic stage), their sleep spindles did not display changes of intrinsic frequency. The present data suggest that age-dependent increase of epileptic activity in WAG/Rij rats affects intrinsic dynamics of sleep spindle frequency.
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ÇELİK, Zülfinaz Betül, Emre Soner TİRYAKİ, Elif TÜRKDÖNMEZ, M. Nusret ÇİÇEKLİ, Ahmet ALTUN, and Caner GÜNAYDIN. "Parallel changes in the promoter methylation of voltage-gated T-type calcium channel alpha 1 subunit G and histone deacetylase activity in the WAG/Rij model of absence epilepsy." Journal of Health Sciences and Medicine 6, no. 1 (January 12, 2023): 92–98. http://dx.doi.org/10.32322/jhsm.1207399.

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Objective: In the last two decades, research on epigenetic mechanisms has expanded dramatically. Recent studies demonstrated that epigenetic mechanisms regulate epilepsy and epileptogenic pathologies. In this study, we aimed to investigate changes in the promoter methylation status of the voltage-gated T-type calcium channel alpha 1 subunit G (CACNA1G) gene and total histone deacetylase activity in Wistar Albino Glaxo/Rijswijk (WAG/Rij) rats which is one of the commonly used genetic absence rat models of epilepsy in the three different age groups (3, 6, and 9 months old) on both sexes. Material and Method: Evaluation of changes in the spike-wave discharges (SWDs) was performed with electrocorticography (ECoG). The promoter methylation status of the CACNA1G gene was determined by methylation-specific PCR (MSP), and histone deacetylase (HDAC) activity was determined spectrophotometrically. Results: Our results demonstrated that the number of SWDs increased time-dependent in WAG/Rij. Additionally, it was observed that CACNA1G promoter methylation decreased, and total HDAC activity increased with age in both sexes. Conclusion: Our results provide further support for epigenetic regulation in the absence epilepsy phenotype and suggest that the underlying mechanism behind the increase in the number of SWDs with age in the WAG/Rij animals might be regulated by CACNA1G promoter methylation or HDAC activity.
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Dissertations / Theses on the topic "Wag/Rij"

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Levin, April Robyn. "Early Seizure Blockade: Preventing Long-Term Epileptic Activity in Wag/Rij Rats." Yale University, 2008. http://ymtdl.med.yale.edu/theses/available/etd-08152007-130206/.

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The purpose of this study was to determine how early seizure blockade with ethosuximide (ESX) would influence ion channel expression and long-term spike-wave discharge (SWD) activity in epileptic WAG/Rij rats. The goal was to elucidate the question Do seizures beget seizures? in a genetically prone model and if so, to attempt to interrupt this cycle by early intervention. In our first experiment, we used immunocytochemistry to determine the effect of ESX on cortical expression of ion channels in treated and untreated WAG/Rij rats and age-matched Wistar controls. This experiment revealed that treatment with ESX blocked the upregulation of Nav1.1 and Nav1.6 as well as the downregulation of HCN1 that is associated with epileptic activity in rats (p < .05). In a second experiment, WAG/Rij rats were divided into 3 groups: untreated (H2O), temporary early treatment (ESX 4 month), and continuous early treatment (ESX continuous), and SWD activity was measured by electroencephalogram (EEG) at various timepoints. This second experiment revealed that animals in the ESX 4 month group spent less percent time in SWD (0.242 ± .068 SEM) than animals in the H2O group (0.769 ± .060 SEM, p < .001), although they spent slightly more percent time in SWD than animals in the ESX continuous group (0.020 ± .065 SEM, p = .003). This effect was predominantly due to seizure number, and average seizure duration did not vary among the three groups. Additionally, power spectrum analysis revealed a significant correlation when the difference between power spectra for H2O and ESX 4 month rats was compared to the power spectrum of a seizure (Pearson correlation equals 0.955, 2-tailed significance < .000000001), suggesting quantitatively that seizures were reduced by temporary early treatment. This suggests that early prevention of SWD may reduce the burden of seizures later in life, and that possibilities for prevention of genetic absence epilepsy should be further investigated.
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Lucas, Susanne. "Anreicherungsverhalten von Kontrastmittel und Zytostatika enthaltenden Liposomen in der Leber von WAG-Rij-Ratten." [S.l.] : [s.n.], 2000. http://www.diss.fu-berlin.de/2001/190/index.html.

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Regenstein, Jens. "Therapiemonitoring im Langzeitversuch am CC-531-Adenokarzinom in der Leber von WAG-RIJ-Ratten mit Hilfe der Magnetresonanztomographie." [S.l.] : [s.n.], 2000. http://www.diss.fu-berlin.de/2001/191/index.html.

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OUSAID-OUAZZANI, LATIFA. "Etude de l'expression de la somatostatine dans les cellules normales et tumorales d'un modele murin du cancer medullaire humain (rat wag/rij). Actions de la somatostatine sur la secretion et la proliferation des cellules c tumorales in vivo et in vitro." Paris 11, 1993. http://www.theses.fr/1993PA112403.

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Le cancer medullaire de la thyroide (cmt) provient de la tumorisation des cellules c. Elles secretent principalement la calcitonine (ct). La presence de somatostatine (ss) a ete signalee chez diverses especes aussi bien dans la thyroide normale que dans certains cas de cmt. Compte tenu des effets antisecretoire et antiproliferatif de ce peptide, nous avons examine d'eventuelles correlations entre sa secretion locale et l'evolution de la tumeur, dans un modele murin du cmt humain, le rat wag/rij. Nous avons d'abord localise, in vivo, la biosynthese de ss par les cellules c des tumeurs spontanees lors des etapes successives du developpement de la maladie. La ss a ete detectee dans quelques cellules c de l'hyperplasie diffuse. Dans la plupart des nodules et des cmt, les cellules ss+ sont regroupees en plages d'importance variable, suggerant une origine clonale de ces cellules. Les cellules ss+ sont une sous-population des cellules ct+ et la co-localisation est constante entre les immunoreactivites ss et ct. L'activite mitotique des cellules ss+ est significativement plus faible que celle des cellules depourvues de ss immunoreactive. Les cellules ct+/ss+ representent la sous-population tumorale la plus differenciee et la moins agressive. Par ailleurs, le traitement in vitro, des cellules tumorales par un analogue synthetique de la ss, la sandostatine, augmente le nombre de cellules immunoreactives a la ct et augmente leur contenu hormonal. D'autre part, la sandostatine inhibe la proliferation de ces cellules. Ces effets antisecretoire et antiproliferatif sont dose-dependants et reversibles
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Lucas, Susanne [Verfasser]. "Anreicherungsverhalten von Kontrastmittel und Zytostatika enthaltenden Liposomen in der Leber von WAG-Rij-Ratten / vorgelegt von Susanne Lucas." 2000. http://d-nb.info/963665669/34.

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Regenstein, Jens [Verfasser]. "Therapiemonitoring im Langzeitversuch am CC-531-Adenokarzinom in der Leber von WAG-RIJ-Ratten mit Hilfe der Magnetresonanztomographie / vorgelegt von Jens Regenstein." 2000. http://d-nb.info/963779656/34.

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Karakizlis, Hristos [Verfasser]. "Effekt der Tiefenhirnstimulation des Nucleus subthalamicus auf die Anfallslänge und -frequenz im WAG-Rij- und im Kindling-Modell der Ratte / vorgelegt von Hristos Karakizlis." 2007. http://d-nb.info/985327847/34.

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Book chapters on the topic "Wag/Rij"

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Sitnikova, Evgenia, Kirill Smirnov, and Vladimir V. Raevsky. "Sensory Inflow from Whiskers Modulates Development of Absence Epilepsy in WAG/Rij Rats." In Advances in Cognitive Research, Artificial Intelligence and Neuroinformatics, 510–19. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71637-0_59.

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S., A. "Two Types of Epilepsy Models and Processes of Cognition: Pentylenetetrazole Kindling and Absence Epilepsy of WAG/Rij Rats Strain." In Underlying Mechanisms of Epilepsy. InTech, 2011. http://dx.doi.org/10.5772/21579.

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"Summary of Potential Models for Aging and Carcinogenesis Research Using Bn/Bi, W Ag/Rij, and (Wag × Bn)F1 Rats." In Pathology of Aging Rats, edited by Joe D. Burek, 203–4. CRC Press, 2018. http://dx.doi.org/10.1201/9781351075435-13.

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Conference papers on the topic "Wag/Rij"

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Kenul, Abbasova, Darya Sydorenko, and Sergey Titov. "PTZ-KINDLING OF WAG/RIJ RATS." In XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2022.sudak.ns2021-17/44.

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Grubov, Vadim V., Evgenia Y. Sitnikova, Vladimir O. Nedaivozov, and Alexey A. Koronovskii. "Proepileptic patterns in EEG of WAG/Rij rats." In Saratov Fall Meeting 2017: Fifth International Symposium on Optics and Biophotonics: Laser Physics and Photonics XIX; Computational Biophysics and Analysis of Biomedical Data IV, edited by Vladimir L. Derbov and Dmitry E. Postnov. SPIE, 2018. http://dx.doi.org/10.1117/12.2315128.

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Smirnov, Kirill, Daria Tsvetaeva, and Evgenia Sitnikova. "NEONATAL WHISKER TRIMMING IN WAG/RIJ PUPS ENCOURAGES MATERNAL CARE." In XIV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2018. http://dx.doi.org/10.29003/m197.sudak.ns2018-14/426-427.

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De, Amiyangshu, Amit Konar, Amalesh Samanta, Souvik Biswas, and Piyali Basak. "Seizure prediction using low frequency EEG wavesfrom WAG/Rij rats." In 2017 2nd International Conference for Convergence in Technology (I2CT). IEEE, 2017. http://dx.doi.org/10.1109/i2ct.2017.8226129.

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Grubov, Vadim V., and Evgenia Sitnikova. "Detection of proepileptic activity patterns in EEG of WAG/Rij rats." In Saratov Fall Meeting 2018: Computations and Data Analysis: from Nanoscale Tools to Brain Functions, edited by Dmitry E. Postnov. SPIE, 2019. http://dx.doi.org/10.1117/12.2527707.

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Sayli, Omer. "Detection of spike-wave discharges in the EEG signals of WAG/Rij rats." In 2015 23th Signal Processing and Communications Applications Conference (SIU). IEEE, 2015. http://dx.doi.org/10.1109/siu.2015.7130161.

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Shishelova, Anna. "CRITICAL PERIODS IN A “MOTHER-INFANTS” SYSTEM IN WISTAR И WAG/RIJ RATS." In XIV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2018. http://dx.doi.org/10.29003/m203.sudak.ns2018-14/541-542.

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8

Grubov, Vadim V., Evgenia Y. Sitnikova, Alexey N. Pavlov, Marina V. Khramova, Alexey A. Koronovskii, and Alexander E. Hramov. "Age-dependent seizures of absence epilepsy and sleep spindles dynamics in WAG/Rij rats." In SPIE BiOS, edited by Valery V. Tuchin, Kirill V. Larin, Martin J. Leahy, and Ruikang K. Wang. SPIE, 2015. http://dx.doi.org/10.1117/12.2076734.

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Makarov, Vladimir V., Vladimir A. Maximenko, Gilles van Luijtelaar, Annika Lüttjohann, and Alexander E. Hramov. "Control of epileptic seizures in WAG/Rij rats by means of brain-computer interface." In Dynamics and Fluctuations in Biomedical Photonics XV, edited by Valery V. Tuchin, Kirill V. Larin, Martin J. Leahy, and Ruikang K. Wang. SPIE, 2018. http://dx.doi.org/10.1117/12.2291696.

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Gabova, Alexandra, Karine Sarkisova, Ekaterina Fedosova, Alla Shatskova, and Alexei Morozov. "AGE-RELATED CHANGES OF SPIKE-WAVE DISCHARGES IN WAG/RIJ RATS WITH GENETIC ABSANCE EPILEPSY." In XIV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2018. http://dx.doi.org/10.29003/m181.sudak.ns2018-14/147.

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You might also be interested in the extended bibliographies on the topic 'Wag/Rij' for particular source types:
Journal articles
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