Dissertations / Theses on the topic 'Vitamin E – Physiological effect'
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Howell, Anne C. "Effects of antioxidant vitamin treatment on UV-irradiated cells." Virtual Press, 1995. http://liblink.bsu.edu/uhtbin/catkey/941360.
Full textDepartment of Biology
Bryant, Rebecca Jane. "Effects of vitamins E and C on exercise-induced lipid peroxidation." Virtual Press, 1996. http://liblink.bsu.edu/uhtbin/catkey/1020147.
Full textDepartment of Family and Consumer Sciences
Winmill, Catherine Anne 1955. "EFFICACY OF CALCIUM AND VITAMIN D SUPPLEMENTATION IN REDUCING DIASTOLIC BLOOD PRESSURE IN A MILD HYPERTENSIVE MALE POPULATION." Thesis, The University of Arizona, 1987. http://hdl.handle.net/10150/276386.
Full textAbel, Stefan. "The physiological effect of vitamin B12 deficiency in human blood." Thesis, Stellenbosch : Stellenbosch University, 1990. http://hdl.handle.net/10019.1/69031.
Full textENGLISH ABSTRACT: The main aim of this workpiece was to establish the physiological parameters against which a vitamin Bu deficiency could be measured. A comparison between the hematological values of healthy patients and those suffering from pernicious anemia due to vitamin Bu deficiency was done. A specific case of pernicious anemia was used in the comparison of abnormal values to the values of normal healthy patients. The comparison consisted of blood analyses with the help of specified instruments, photomicrographs of bone marrow and blood smears and statistical data. A Coulter Counter Model ZF was used for the hematological analyses of blood, a radio-isotope assay for serum vitamin B u was done and photomicrographs were taken with a NIKON Microflex camera with photomicrographic attachments. The importance of vitamin Bu has been shown in this workpiece. With the use of techniques and certain instruments, the effects of a shortage of vitamin Bu has been shown. Analyses of the blood from normal ,healthy patients was compared to that of patients suffering from pernicious anemia. It was demonstrated that pernicious anemia is characterized by a low erythrocyte count, hematocrit (Het), hemoglobin (Hb) and vitamin Bu levels together with a higher mean corpuscular hemoglobin (MCH) and mean corpuscular volume (MCV). In severe cases of pernicious anemia these levels are extremely high or low as the case may be. Together with these values, the investigation of pernicious anemic blood and bone marrow smears revealed abnormally large erythrocyte precursors and fewer leucocytes than normal. Abnormally shaped cells, called macrocytes, were seen which was due to the disruption in deoxyribonucleic acid (DNA) synthesis caused by the vitamin Bu deficiency. This study produced a set of hematological reference values. The comparative study between healthy and pernicious anemic patients demonstrated a significant drop in serum vitamin B12 values during pernicious anemia. The hematological effect was illustrated by the Coulter Counter blood analysis results and the microscopic examination revealed the presence of megaloblastic erythrocytes, oval erythrocytes, pear-shaped poikilocytes and polymorphonuclear neutropbils with hypersegmented nuclei in blood smears I during vitamin B12 deficiency. This dianoses can be supported by the presence of megaloblasts and metamyelocytes in pernicious anemic bone marrow.
AFRIKAANSE OPSOMMING: Die hoof doel van hierdie werkstuk was om fisiologiese grense te bepaal waarteen 'n vitamien B12 tekort gemeet kan word. 'n Vergelyking tussen die hematologiese waardes van gesonde persone en die van pasiente met pernisieuse anemie wat ontstaan het as gevolg van 'n vitamien B12 tekort was uitgevoer. Die waardes verkry vanaf 'n spesifieke geval van pernisieuse anemie. was vergelyk met waardes vanaf normale gesonde persone. Hierdie vergelyking het bestaan uit bloed analises, fotomikrograwe van bloed en beenmurg smere en statistiese data. Die hematologiese bloed analises was met behulp van 'n Coulter Teller model ZF uitgevoer. 'n Radio-isotoop bepaling vir serum vitamien B12 was gedoen en fotomikrograwe was met 'n NIKON Microflex kamera geneem. Die belang van 'n vitamien B12 tekort was in hierdie werkstuk gedemonstreer. Die effek van hierdie tekort is deur die gebruik van sekere instrumente en tegnieke aangedui en die resultate hiervan is vergelyk tussen gesonde persone en pasiente met 'n vitamien B12 tekort. Hierdie studie het bevestig dat pernisieuse anemie gekenmerk word deur verlaagde eritrosiet, hematokrit (Het), hemoglobien (Hb) en vitamien B12 vlakke tesame met verhoogde gemene korpuskulere hemoglobien (GKH) en gemene korpuskulere volume (GKV) vlakke. Gedurende ernstige gevalle van pernisieuse anemie kan hierdie waardes uitermatig hoog of laag wees. Benewens hierdie waardes het die ondersoek van bloed en beenmurg gedurende vitamien B12 tekort, abnormale groot eritrosiet voorgangers en 'n verminderde hoeveelheid leukosiete getoon. Abnormale sel vorms was ook sigbaar a.g.v. die onderbreking in DNA sintese wat deur 'n vitamien B12 tekort veroorsaak word. Pernisieuse anemie word verkry wanneer daar 'n vitamien B12 en/of folaat tekort in die dieet is of wanneer hierdie vitamiene nie geabsorbeer kan word nie. Die teenwoordigheid van makrosiete, ovaal eritrosiete, peervormige poikilosiete en polimorfonuklere neutrofiele met hipergesegmenteerde keme in bloedsmere dui op 'n megaloblastiese anemie. Hierdie diagnose kan ondersteun word deur die aanwesigheid van megaloblaste en reuse metamielosiete in die beenmurg. Die bepaling van vitamien B12 en folaat vlakke in die bloed kan as addisionele bewysstukke vir 'n volledige diagnose dien. Gedurende hierdie studie is daar 'n stel hematologiese verwysingswaardes vasgestel. Die vergelykende studie tussen gesonde persone en pasiente met pernisieuse anemie het getoon dat daar 'n beduidende verlaging in serum vitamien B12 waardes gedurende pernisieuse anemie is. Die hematologiese effek was ook duidelik waameembaar in die Coulter teller se bloed analiese en mikroskopiese ondersoeke het die · teenwoordigheid van makrosiete, ovaal eritrosiete, peervormige poikilosiete en polimorfenuklere neutrofiele met hipersegmenteerde keme in bloedsmere aangedui. Hierdie diagnose kan ondersteun word deur die aanwesigheid van megaloblaste en reuse metamielosiete in die beenmurg.
This study was financially aided by a bursary from the CSIR.
Farmer, Bertrand. "The status of beta carotene and vitamin A in Quebec dairy herds and their effects on reproductive performance /." Thesis, McGill University, 1985. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=63167.
Full textMorton, Lincoln William. "The role of dietary phenolic compounds in the detoxification of reactive nitrogen species." University of Western Australia. Dept. of Medicine, 2003. http://theses.library.uwa.edu.au/adt-WU2003.0026.
Full textAvery, Neva G. "The effects of vitamin E supplementation on the recovery from repeated bouts of resistance exercise." Virtual Press, 2002. http://liblink.bsu.edu/uhtbin/catkey/1231405.
Full textSchool of Physical Education
Poirier, Johanne 1959. "The effects of selenium and vitamin E intake on diet-induced oxidative stress and hyperlipidemia /." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31526.
Full textClarke, Michael William. "Vitamin E metabolism in humans." University of Western Australia. School of Medicine and Pharmacology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0191.
Full textEiselstein, Emily M. "The effects of vitamin E supplementation and/or resistance exercise on insulin responsiveness in the elderly." Virtual Press, 2002. http://liblink.bsu.edu/uhtbin/catkey/1239215.
Full textSchool of Physical Education
Wanamaker, Scot E. "The effects of vitamin E supplementation and resistance training on muscle function in elderly subjects." Virtual Press, 2002. http://liblink.bsu.edu/uhtbin/catkey/1231404.
Full textSchool of Physical Education
Austin, Nicole. "Vitamin D, neuromuscular control and falling episodes in Australian postmenopausal women." University of Western Australia. School of Medicine and Pharmacology, 2009. http://theses.library.uwa.edu.au/adt-WU2010.0009.
Full textMcMahon, Jennifer A., and n/a. "The effect of homocysteine lowering vitamins on cognitive performance in older people : a randomised controlled trial." University of Otago. Department of Human Nutrition, 2006. http://adt.otago.ac.nz./public/adt-NZDU20070426.154857.
Full textMurphy, Stephanie A. "Effects of selenium and vitamin B-6 on growth of chemically- induced transplanted tumors in BALB/c inbred mice." Thesis, Virginia Tech, 1989. http://hdl.handle.net/10919/43906.
Full textMaster of Science
Mason, Shelley S. "Exploring Tissue Engineering: Vitamin D3 Influences on the Proliferation and Differentiation of an Engineered Osteoblast Precursor Cell Line During Early Bone Tissue Development." PDXScholar, 2013. https://pdxscholar.library.pdx.edu/open_access_etds/1000.
Full textCobb, Jennifer L. "Validation of a Sun-Exposure Questionnaire for Adolescent Girls." Fogler Library, University of Maine, 2001. http://www.library.umaine.edu/theses/pdf/CobbJL2001.pdf.
Full textLiu, Chia-chi. "Oxidation of ascorbate by protein radicals in simple systems and in cells." Phd thesis, Australia : Macquarie University, 2007. http://hdl.handle.net/1959.14/16746.
Full textBibliography: leaves 295-322.
Generation of peroxide groups in proteins exposed to a wide variety of reactive oxygen species (ROS) requires an initial formation of protein carbon-centred or peroxyl free radicals, which can be reduced to hydroperoxides. Both protein radicals and protein hydroperoxides are capable of oxidizing important biomolecules and thus initiate biological damage. In this study, we investigated the inhibition of protein hydroperoxide formation by ascorbate and GSH in gamma-irradiated HL-60 cells.--We used HL-60 cells as a model for general protection of living organisms by ascorbate (Asc) and glutathione (GSH) from the deleterious effects of protein hydroperoxides generated by radicals produced by gamma radiation. Measurement by HPLC indicated that incubation of HL-60 cells with Asc in the presence of ascorbate oxidase resulted in the accumulation of intracellular Asc. The intracellular Asc levels were lowered by irradiation, demonstrating intracellular consumption of Asc by the radiation-generated radicals. Exposure of HL-60 cells to increasing gamma irradiation doses resulted in increasing accumulation of protein peroxides in the cells. This was measured by the FOX assay. A significant decrease in intracellular protein hydroperoxides was noted when the cells were treated with ascorbic acid before irradiation. A dose-dependent protective effect of Asc was observed. Asc loading also provided strong protection from radiation-generated protein hydroperoxides independently of the composition of the external medium, showing that only the radicals formed within the cells were effective in oxidizing the cell proteins. Similarly, protein peroxidation was inhibited in cells with enhanced levels of GSH and increased when the intracellular GSH concentration was reduced. These findings indicate that ascorbate and GSH are important antioxidants in protecting cells from oxidative stress associated with the generation of protein hydroperoxide.
Mode of access: World Wide Web.
xxix, 322 leaves ill
Johnson, Lyn. "Regulation of hepatic catabolism in the physiological economy of vitamin D." Thesis, University of Cambridge, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254250.
Full textUdagawa, Miho. "Studies on Distribution and Physiological Function of Vitamin K in Fish." Kyoto University, 2001. http://hdl.handle.net/2433/150325.
Full textZeferino, Cynthia Pieri [UNESP]. "Resposta fisiológica, qualidade da carne e expressão gênica no músculo esquelético de frangos de corte sob estresse por calor que receberam antioxidantes na dieta." Universidade Estadual Paulista (UNESP), 2012. http://hdl.handle.net/11449/104104.
Full textCoordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)
Universidade Estadual Paulista (UNESP)
A temperatura ambiente elevada representa o principal fator limitante do desenvolvimento da produção avícola em regiões de clima quente. O objetivo do trabalho foi verificar se a dieta suplementada com vitaminas C e E, seria capaz de neutralizar, ou reduzir, os efeitos do estresse por calor, aplicado dos 28 aos 42 dias de idade, sobre a resposta fisiológica, o desempenho, rendimento de carcaça e qualidade da carne de frangos. Foram utilizados 384 frangos de corte machos distribuídos num delineamento inteiramente casualizado, com arranjo fatorial 2 x 3 (com e sem suplementação da dieta com vitaminas e temperaturas ambientais associadas ao pair-feeding) e 16 repetições. As aves foram mantidas em termoneutralidade até os 28 dias. A partir desta idade, foram alojadas em grupos de quatro por gaiola, em três salas climatizadas: duas termoneutras (22,6 e 22,5ºC) e uma de estresse por calor (31,7ºC). Metade das aves recebeu dieta suplementada com vitaminas C (536 mg/kg) e E (127 mg/kg). Na sala de estresse por calor as aves tiveram livre acesso à ração; nas salas termoneutras metade das aves recebeu ração à vontade e a outra metade recebeu quantidade limitada, no sistema pair-feeding. Foram avaliados a temperatura retal e da superfície da pele e características de desempenho, rendimento de carcaça e qualidade instrumental da carne. As análises de qualidade de carne foram realizadas no músculo pectoralis major (peito) 24 horas após o abate. A suplementação da dieta com vitaminas C e E não foi capaz de neutralizar, nem de reduzir, os efeitos negativos do estresse por calor sobre as características de desempenho, rendimento de carcaça e qualidade de carne das aves. A semelhança nos resultados de desempenho entre aves em estresse por calor e em pair-feeding sugere que a queda no desempenho sob estresse por calor deveu-se, principalmente, à redução no consumo de alimentos
Elevated ambient temperature represents the main limiting factor for the development of chicken production in hot climate regions. The objective of this study was to evaluate if the diet supplemented with vitamins C and E would be able to reduce, or neutralize the negative effects of heat stress applied between 28 and 42 days of age, on the physiological response, performance, slaughter yield and meat quality of chickens. A total of 384 male broiler chickens were assigned to a completely randomized design, with a 2 x 3 factorial arrangement (diet with or without vitamins supplementation and ambient temperatures associated with pair-feeding) and 16 replications. The chickens were kept in thermoneutral conditions up to 28 days of age. From this age and on, were housed in groups of four per cage, in three environmentally controlled chambers: two were thermoneutral (22,6 e 22,5ºC) and one for heat stress (31,7ºC). Half the chickens were offered a diet supplemented with vitamins C (536 mg/kg) and E (127 mg/kg). In the heat stress chamber, the chickens had free access to the feed; in the thermoneutral chambers half the chickens had free access to the feed and the other half received a limited amount, in a pair-feeding system. Rectal and body surface temperatures and performance, slaughter yield and meat quality traits were evaluated. Meat quality analyses were performed in the pectoralis major muscle 24 hours after slaughter. Diet supplementation with vitamins C and E were not able to neutralize, neither to reduce, the negative effects of heat stress on the performance, slaughter yield and meat quality traits. The similarity of performance results between the chickens submitted to heat stress and pair-feeding, suggested that the reduction in performance under heat stress were due mainly to the drop in feed consumption
Zeferino, Cynthia Pieri 1981. "Resposta fisiológica, qualidade da carne e expressão gênica no músculo esquelético de frangos de corte sob estresse por calor que receberam antioxidantes na dieta /." Botucatu : [s.n.], 2012. http://hdl.handle.net/11449/104104.
Full textBanca: José Roberto Sartori
Banca: Dirlei Antonio Berto
Banca: Luiz Lehmann Coutinho
Banca: José F. M. Mendes
Resumo: A temperatura ambiente elevada representa o principal fator limitante do desenvolvimento da produção avícola em regiões de clima quente. O objetivo do trabalho foi verificar se a dieta suplementada com vitaminas C e E, seria capaz de neutralizar, ou reduzir, os efeitos do estresse por calor, aplicado dos 28 aos 42 dias de idade, sobre a resposta fisiológica, o desempenho, rendimento de carcaça e qualidade da carne de frangos. Foram utilizados 384 frangos de corte machos distribuídos num delineamento inteiramente casualizado, com arranjo fatorial 2 x 3 (com e sem suplementação da dieta com vitaminas e temperaturas ambientais associadas ao pair-feeding) e 16 repetições. As aves foram mantidas em termoneutralidade até os 28 dias. A partir desta idade, foram alojadas em grupos de quatro por gaiola, em três salas climatizadas: duas termoneutras (22,6 e 22,5ºC) e uma de estresse por calor (31,7ºC). Metade das aves recebeu dieta suplementada com vitaminas C (536 mg/kg) e E (127 mg/kg). Na sala de estresse por calor as aves tiveram livre acesso à ração; nas salas termoneutras metade das aves recebeu ração à vontade e a outra metade recebeu quantidade limitada, no sistema pair-feeding. Foram avaliados a temperatura retal e da superfície da pele e características de desempenho, rendimento de carcaça e qualidade instrumental da carne. As análises de qualidade de carne foram realizadas no músculo pectoralis major (peito) 24 horas após o abate. A suplementação da dieta com vitaminas C e E não foi capaz de neutralizar, nem de reduzir, os efeitos negativos do estresse por calor sobre as características de desempenho, rendimento de carcaça e qualidade de carne das aves. A semelhança nos resultados de desempenho entre aves em estresse por calor e em pair-feeding sugere que a queda no desempenho sob estresse por calor deveu-se, principalmente, à redução no consumo de alimentos
Abstract : Elevated ambient temperature represents the main limiting factor for the development of chicken production in hot climate regions. The objective of this study was to evaluate if the diet supplemented with vitamins C and E would be able to reduce, or neutralize the negative effects of heat stress applied between 28 and 42 days of age, on the physiological response, performance, slaughter yield and meat quality of chickens. A total of 384 male broiler chickens were assigned to a completely randomized design, with a 2 x 3 factorial arrangement (diet with or without vitamins supplementation and ambient temperatures associated with pair-feeding) and 16 replications. The chickens were kept in thermoneutral conditions up to 28 days of age. From this age and on, were housed in groups of four per cage, in three environmentally controlled chambers: two were thermoneutral (22,6 e 22,5ºC) and one for heat stress (31,7ºC). Half the chickens were offered a diet supplemented with vitamins C (536 mg/kg) and E (127 mg/kg). In the heat stress chamber, the chickens had free access to the feed; in the thermoneutral chambers half the chickens had free access to the feed and the other half received a limited amount, in a pair-feeding system. Rectal and body surface temperatures and performance, slaughter yield and meat quality traits were evaluated. Meat quality analyses were performed in the pectoralis major muscle 24 hours after slaughter. Diet supplementation with vitamins C and E were not able to neutralize, neither to reduce, the negative effects of heat stress on the performance, slaughter yield and meat quality traits. The similarity of performance results between the chickens submitted to heat stress and pair-feeding, suggested that the reduction in performance under heat stress were due mainly to the drop in feed consumption
Doutor
Haaften, Rachel I. M. van. "Effect of vitamin E on glutathione-dependent enzymes." [Maastricht] : Maastricht : UMP, Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 2003. http://arno.unimaas.nl/show.cgi?fid=6339.
Full textHogan, Kyla B. "The hemostatic responses to exercise in hot and cold temperatures." Virtual Press, 2008. http://liblink.bsu.edu/uhtbin/catkey/1398717.
Full textSchool of Physical Education, Sport, and Exercise Science
Xu, Fan 1960. "Effect of prolonged exercise on running economy." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=68149.
Full textAlbenali, Lujain. "The effect of vitamin D supplementation on atopic dermatitis." Thesis, University of Sheffield, 2013. http://etheses.whiterose.ac.uk/4510/.
Full textPietrzak, Ewa M. "Vitamin E as an index of tissue peroxidation: The effect of vitamin C deficiency and ischemia/reperfusion." Thesis, University of Ottawa (Canada), 1993. http://hdl.handle.net/10393/6464.
Full textGallo, Sina. "An examination of parental compliance with vitamin D recommendations and the physiological responses to vitamin D isoform and dosage in breastfed infants from greater Montréal." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=116878.
Full textLa vitamine D est importante pour maintenir l'homéostasie du calcium et pour promouvoir le développement des os. Les nouveau-nés allaités ont un risque accru en carence de vitamine D car le lait maternel est insuffisant en vitamine D. Santé Canada recommande que tous les nourrissons allaités reçoivent 400 UI/jour de vitamine D dès la naissance et jusqu'à ce qu'ils puissent obtenir cette quantité par l'alimentation. La production alimentaire et endogène de la vitamine D est généralement mesurée en utilisant le métabolite 25-hydroxyvitamine D (25(OH)D) car il a une demi-vie plus longue et est moins contrôlé par les hormones que la forme active, soit la 1,25-dihydroxyvitamine D. L'Institute of Médicine (IOM) définit la quantité nécessaire pour répondre aux besoins de 97,5% d'une population avec l'apport nutritionnel recommandé (ANR), mais en raison de l'insuffisance des données scientifiques pour le groupe d'âge 0 à 12 mois, un apport suffisant de 400 UI/jour a été créé à la place. Cette quantité est jugée suffisante pour maintenir les concentrations plasmatique de 25(OH)D dans la gamme de 40-50 nmol/L, suffisante pour la santé des os. D'autres soutiennent que des concentrations >75 nmol/L sont nécessaires et peuvent nécessiter 800 UI/ jour de vitamine D pour certains nourrissons et enfants. Malgré ces recommandations, certains nourrissons au Canada ont des concentrations faibles de 25(OH)D. Il est difficile de savoir si ces faibles concentrations sont dues à un non suivi de la posologie ou parce que les recommandations sont insuffisant pout atteindre des niveau sanguins de 25(OH)D désirée. À ce jour, les résultats quant a savoir si les apports en vitamine D de >400 UI/jour pendant l'enfance ont des conséquences bénéfiques sur l'accumulation du contenu minéral osseux n'ont pas étés explorés scientifiquement. Il existe deux isoformes de la vitamine D qui sont disponibles sous forme de suppléments au Canada, soit cholécalciférol (vitamine D3) et ergocalciférol (vitamine D2). Il y a une controverse quant à l'efficacité biologique de ces deux formes ayant pour effet d'augmenter les niveaux sanguins de 25(OH)D. Santé Canada à stipulé dans sa recommandation de 2004, que la vitamine D2 est moins efficace que la D3 hors, aucune étude n'a comparé l'efficacité de ces isoformes directement chez les nourrissons. L'objectif global de cette thèse est de: (1) décrire les pratiques de suppléments de la vitamine D par la mère du nouveau-né, dans un grand centre urbain au Canada, (2) de comparer la réponse biologique à un supplément de vitamine D2 vs. D3 chez les nourrissons allaités de 1 à 4 mois de la vie, et (3) définir le dosage quotidien de la vitamine D3 en fonction de la quantité nécessaire pour (a) élever les niveaux plasmatique de 25(OH)D ≥75 nmol /L dans 97,5% des nourrissons (telle qu'était utilisée dans la détermination de l'ANR) et (b) utilisant des mesures de l'homéostasie minérale, de la croissance et du contenu de la masse osseuse. L'étude 1 a consisté d'une enquête téléphonique auprès de 342 mères ayant accouchées à l'hôpital Royal Victoria (Montréal, Québec), représentant environ 10% de toutes les naissances annuelles à Royal Victoria. Les mères ont étés contactées entre 6 à 12 mois post-partum, et questionnées sur les pratiques quant aux suppléments en vitamine D, en particulier sur la quantité et la fréquence des doses ainsi que les raisons du non-respect. L'étude 2 était un essai clinique aléatoire (ECR) de 52 nourrissons allaités d'un mois d'âge. Ces nourrissons ont reçu 400 UI/jour de vitamine D2 ou D3 pour une durée de 3 mois. Le changement dans les niveau plasmatique de 25(OH)D ainsi que la proportion de nourrissons qui ont atteint ≥50 et ≥75 nmol/L de 25(OH)D ont été comparés entre les deux groupes. L'étude 3 était un ECR dont 132 bébés allaités d'un mois d'âge, en bonne santé, ont étés assignés au hasard à recevoir l'une des 4 doses de vitamine D3 (400, 800, 1200 ou 1600 UI/jour).
Anderson, Dawn E. "Effects of caffeine on the metabolic and catecholamine responses to exercise in 5 and 28p0sC environments." Virtual Press, 1992. http://liblink.bsu.edu/uhtbin/catkey/833465.
Full textHuman Performance Laboratory
Martin, David E. "The effect of heat stress on excess post exercise oxygen consumption." Virtual Press, 1992. http://liblink.bsu.edu/uhtbin/catkey/834623.
Full textSchool of Physical Education
Shi, Xiaocai. "Effect of sodium and water intake on plasma aldosterone during prolonged exercise in warm environment." Virtual Press, 1990. http://liblink.bsu.edu/uhtbin/catkey/722232.
Full textSchool of Physical Education
Majewicz, Jonathan. "The effect of vitamin E and vitamin C on the differential gene expression in monocytes and endothelial cells." Thesis, University of Reading, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434331.
Full textHeyns, Gerhardus Johannes. "Influence of macro- versus microcooling on the physiological and psychological performance of the human operator." Thesis, Rhodes University, 1995. http://hdl.handle.net/10962/d1016247.
Full textLoewen, Anisa Joy. "Optimizing the loading of vitamin A and vitamin D into re-assembled casein micelles and investigating the effect of micellar complexation on vitamin D stability." Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/50865.
Full textLand and Food Systems, Faculty of
Graduate
Morgan, Michael J. "Opioid-dopamine interactions in analgesia in the formalin test." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74326.
Full textPoirier, Patrick. "Effect of chronic stress on prefrontal cortical function." Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=86861.
Full textFirst, contrary to previous evidences, chronic stress induces working memory performance alterations differentially in two populations of rats. One group displayed a decrease of performance only at 30 second delay, while the other had a decrease and increase at 0 and 30 seconds respectively.
Then, the effect of chronic stress on synaptic plasticity induction in the hippocampus-PFC network was investigated. High-frequency tetanic stimulation (HFS) of the dorsal hippocampus that induced long-term potentiation (LTP) in the prelimbic and infralimbic cortex in normal conditions was unable to induce LTP after chronic stress in the infralimbic cortex, whereas long-term depression (LTD) instead of LTP was induced in the prelimbic cortex.
Given that synaptic plasticity has been shown to depend on NMDA receptors in the PFC, NMDA subunit expressions before and after chronic stress was examined. There was a decrease of NR1 subunits expression in the prelimbic, but not infralimbic cortex. In contrast, the NR2A/NR2B ratio was increased in the infralimbic, but not prelimbic cortex. These results suggest that chronic stress disrupts PFC functions through dynamic modulation of distinct neural networks within the PFC.
Le cortex préfrontal (PFC) est une région du cerveau qui contrôle les fonctions cognitives comme la mémoire de travail. Dans cette thèse, l'effet du stress chronique sur des fonctions du PFC a été analysé chez des rats adultes.
Premièrement, les performances de la mémoire de travail ont été mesurées avant et après exposition au stress chronique. Nous avons constaté que le stress chronique induit des changements de performances de la mémoire de travail différemment selon deux populations de rats. Une des populations a démontré une diminution de performance seulement à 30 secondes de délai. Au contraire, l'autre a démontré une diminution de performance à 0 seconde et une amélioration de performance à 30 secondes.
En plus, nous avons évalué l'effet du stress chronique sur l'induction de la plasticité synaptique dans le réseau reliant l'hippocampe au PFC. Dans les conditions initiales, une stimulation tétanique à haute fréquence (HFS) dans l'hippocampe dorsal provoquait une potentialisation à long terme (LTP) dans le cortex prélimbique et infralimbique Or après exposition au stress chronique, une stimulation tétanique à haute fréquence n'a pas entraîné de potentialisation à long terme dans le cortex infralimbique. De plus, une exposition au stress chronique a provoqué l'apparition dans le cortex prélimbique d'une dépression à long terme (LTD) plutôt qu'une potentialisation à long terme.
Étant donné que la plasticité synaptique dépend des récepteurs de NMDA dans le PFC, nous avons examiné l'expression de sous-unité de NMDA avant et après exposition au stress chronique. En accord avec les changements synaptiques distincts de plasticité entre le cortex prélimbique et infralimbique après exposition au stress chronique, nous avons observé que l'expression de la sous-unité NR1 a diminué dans le prélimbique, mais non dans l'infralimbique. En revanche, le ratio de NR2A/NR2B a augmenté dans le cortex infralimbique, mais non dans le prélimbique. Ces résultats suggèrent que le stress chronique perturbe les fonctions du PFC par la modulation dynamique des réseaux distincts neurologiques dans le PFC.
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