Academic literature on the topic 'Vitamin D'
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Journal articles on the topic "Vitamin D"
Köse, Nurgül. "Blood vitamin D levels in patients with fibromyalgia and the effectiveness of vitamin D treatment." Dicle Medical Journal/Dicle Tıp Dergisi 40, no. 4 (January 12, 2013): 585–88. http://dx.doi.org/10.5798/diclemedj.0921.2013.04.0337.
Full textJ. Mustafa, Awat, and Aso F. Salih. "VITAMIN D INTOXICATION IN INFANTS WITH VITAMIN D SUPPLEMENTS." Journal of Sulaimani Medical College 10, no. 1 (March 21, 2020): 67–71. http://dx.doi.org/10.17656/jsmc.10241.
Full textLucock, Mark, Patrice Jones, Charlotte Martin, Emma Beckett, Zoe Yates, John Furst, and Martin Veysey. "Vitamin D." Journal of Evidence-Based Complementary & Alternative Medicine 20, no. 4 (April 15, 2015): 310–22. http://dx.doi.org/10.1177/2156587215580491.
Full textPusparini, Pusparini. "DEFISIENSI VITAMIN D TERHADAP PENYAKIT (Vitamin D Deficiency and Diseases)." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 21, no. 1 (April 15, 2018): 90. http://dx.doi.org/10.24293/ijcpml.v21i1.1265.
Full textGašparini, Dora, and Marija Kaštelan. "Vitamin D i koža." Medicina Fluminensis 57, no. 4 (December 1, 2021): 356–64. http://dx.doi.org/10.21860/medflum2021_264890.
Full textKroll, Dorothy. "Update on Vitamins: Vitamin D." Alternative and Complementary Therapies 1, no. 3 (April 1995): 172–76. http://dx.doi.org/10.1089/act.1995.1.172.
Full textBİLGİLİ, Ali, and Onur ALAÇIK. "Levels of Vitamin D in Some Vitamin D Veterinary Preparations Kept in Different Storage Conditions." ISPEC Journal of Agricultural Sciences 6, no. 2 (June 15, 2022): 195–201. http://dx.doi.org/10.46291/ispecjasvol6iss2id287.
Full textNguyen, Timothy. "Vitamin D and Vitamin D Analogs." Journal for Nurse Practitioners 12, no. 3 (March 2016): 208–9. http://dx.doi.org/10.1016/j.nurpra.2015.12.020.
Full textS.A. Mursalova, S.S. Safarova, Kh.M. Mirzayeva, S.N. Valiyeva, T.F. Jafarova, and A.N. Khudiyeva. "Qadın reproduktiv sağlamlığı və vitamin D." Actual Questions of Modern Gynecology and Perinatology 10, no. 2 (July 19, 2023): 27–32. http://dx.doi.org/10.28942/mgpam.v10i2.109.
Full textKoprivica, Marko, and Svetlana Kašiković-Lečić. "Causes, consequences, and treatment of vitamin D deficiency in humans." Medicinski casopis 57, no. 3 (2023): 119–24. http://dx.doi.org/10.5937/mckg57-43860.
Full textDissertations / Theses on the topic "Vitamin D"
Sultan, Arwa. "Vitamin D and Depression in Women of Reproductive Age: Exploring Women’s Use of Vitamin D and Vitamin Supplements." Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/24787.
Full textMolin, Arnaud. "Etude des causes génétiques de dérégulation du métabolisme de la vitamine D." Thesis, Normandie, 2019. http://www.theses.fr/2019NORMC405/document.
Full textThe vitamin D (D3 or cholecalciferol from animal kingdom and D2 or ergosterol from plan kingdom) is a pleiotropic hormone who has numerous biological effects including the regulation of calcium and phosphate metabolism. In humans, this compound is synthetized in skin in an inactive form. Thus, we call vitamin D metabolism the biological process which leads to the production of active metabolites (by enzymes 25- and 1α-hydroxylases encoded by CYP2R1 and CYP27B1 genes) and its degradation by vitamin D 24-hydroxylase (gene CYP24A1). The expression of 1α- and 24-hydroxylases is tightly and inversely regulated to maintain calcium and phosphate homeostasis, thanks to several feedback loops including 1,25-dihydroxyvitamin D and its receptor VDR, serum calcium and parathormone, serum phosphate and FGF23. Vitamin D deficiency and vitamin D activation deficiency are associated with rickets, while vitamin D excess are associated with hypercalcemia-hypercalciuria due to vitamin D intoxication (overdose) or hypersensitivity to vitamin D (activation excess or degradation deficiency).Our aim is to identify genetic causes of vitamin D metabolism deregulation and to specify pathophysiologic mechanisms describing phenotype. Thus, we jointly used the tools of genetics (next-generation and Sanger sequencing) and biochemistry (vitamin D metabolites assay) in a cohort of human patients ascertained thanks to the national center for rare diseases of calcium and phosphate metabolism.This work allowed us to specify the role of two genes in diseases of vitamin D metabolism, CYP2R1 and CYP24A1, showing loss of function mutations in patients with rickets and low 25-hydroxyvitamin D and hypersensitivity to vitamin D, respectively. Our study brought new phenotypic elements in these affections. In our cohort of patients, the identification of mutations leading to phosphate deregulation (in SLC34A1 and SLC34A3) highlights the putative role of regulators of vitamin D 1α- and 24-hydroxylases activities in pathophysiology.No significant variation have been identified in the proximal promoting regions of CYP27B1 and CYP24A1. We could not go further considering the lack of knowledge in regulating regions and factors in humans. Identifying distal regulators will allow to study their implication in rare diseases of vitamin D metabolism
Lowe, Lorraine Claire. "Vitamin D and the vitamin D receptor in breast cancer." Thesis, St George's, University of London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428038.
Full textFrota, Karine de Holanda. "Ponto de corte para adequação da concentração sérica de 25 hidroxivitamina D em adultos e idosos: estudo de base populacional - ISA-Capital." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/6/6138/tde-05092012-102812/.
Full textIntroduction - The serum concentration of vitamin D may vary in individuals of different age groups and geographic regions and may be influenced by sun exposure, season and by BMI and parathyroid hormone (PTH). The classification widely used as a cut-off for appropriate vitamin D status refers serum 25 (OH) D above 30 ng/mL. However, this classification may be inappropriate for the Brazilian population, due to the particular food and the climate of our population. Objective - To determine the mean serum concentrations of 25(OH)D and PTH and correlate them with BMI, sunlight exposure and season and to identify the cutoff values of 25 (OH) D associated with elevation in PTH. Methods For this dissertation, one original article were developed. Original article describe cross-sectional study performed with subjects from the ISA Capital, multicenter population-based. We investigated 589 individuals were of both sexes, age groups: 20-59 (adults) and 60 (elderly). Blood samples for laboratory measurements of 25(OH)D and PTH were collected. Individuals, who agreed to participate in blood collection, also answered a questionnaire on sunlight exposure. Statistical analysis included ROC curve, Student t test, correlation tests, ANOVA. The calculations were performed by the software SPSS version 17.0. and p 0.05 was considered significant. Results - In the original article, the mean age of participants was 54.83 (19.21) years, 61.3 per cent female and 38.7 per cent were male. The mean serum 25 (OH) D was 50.02 (22.69) ng/mL, between the groups was 47.48 (23.03) (adults) and 52.68 (22.06) ng/ mL (elderly) and significant difference between them (p = 0.005). A seasonal variation in serum 25 (OH) D was observed and positive correlation between 25(OH)D and BMI (r = 0.114, p = 0.006). The new cutoff value 55.8 ng / mL, determined by ROC curve analysis found 67.6 per cent of subjects insufficient 25 (OH) D and between groups 72.1 per cent (adults) and 62.8 per cent (elderly). Conclusion - The results demonstrate the presence of seasonal variation in serum 25 (OH) D in the municipality of Sao Paulo. The cutoff point proposed for our population showed a high prevalence of insufficient vitamin D. Therefore, public policy is needed to prevent vitamin D insufficiency in order to beneficial effects on health and quality of life in this population.
Ellfolk, Maria. "Regulation of Vitamin D 25-hydroxylases : Effects of Vitamin D Metabolites and Pharmaceutical Compounds on the Bioactivation of Vitamin D." Doctoral thesis, Uppsala universitet, Avdelningen för farmaceutisk biokemi, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9412.
Full textKe, Liang. "Hypertension and Vitamin D." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/14220.
Full textClark, W. Andrew. "Falls and Vitamin D." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/2526.
Full textClark, W. Andrew. "Falls and Vitamin D." Digital Commons @ East Tennessee State University, 2013. https://dc.etsu.edu/etsu-works/2528.
Full textMartori, Muntsant Clara. "Vitamin D and canine leishmaniasis." Doctoral thesis, Universitat Autònoma de Barcelona, 2021. http://hdl.handle.net/10803/673957.
Full textLas leishmaniosis son un grupo de enfermedades causadas por protozoos del género Leishmania que se transmiten por vectores. La leishmaniosi visceral (LV) humana puede ser mortal si no se trata, resultando en 26 000-65 000 muertes por año. Los cánidos son el principal reservorio y huéspedes de Leishmania infantum, el agente causante de la LV zoonótica en la cuenca mediterránea. Se desconoce el mecanismo que regula el resultado final de la infección, pero se sabe que el sistema inmunitario juega un papel clave en el control de la enfermedad. Varios estudios han demostrado que la vitamina D tiene un rol importante en la respuesta inmune, activando el sistema inmunitario innato y modulando la respuesta adaptativa. Además, se ha descrito la relación entre la deficiencia de vitamina D y el riesgo de sufrir algunas enfermedades. El objetivo de la tesis fue estudiar si la vitamina D tiene una contribución relevante en la leishmaniosis canina (LCan). Para ello se determinó la concentración de vitamina D en muestras de suero de una población de perros sanos y enfermos de leishmaniosis residentes en una zona altamente endémica y se estudió la relación de ésta con parámetros parasitológicos e inmunológicos. Los perros enfermos mostraron niveles de vitamina D significativamente más bajos que los no infectados y que los infectados asintomáticos. Además, la deficiencia de vitamina D se correlacionó con parámetros relacionados con la progresión de la enfermedad. También investigamos si las variaciones genéticas en el locus del gen del receptor de la vitamina D se asocia con la progresión de LCan, pero las frecuencias alélicas de los polimorfismos (SNPs) encontrados no resultaron ser estadísticamente diferentes entre grupos. Posteriormente se analizó la concentración de vitamina D en muestras de suero tomadas en diferentes periodos del año en una cohorte de perros sanos. Los resultados mostraron que no hay una variación estacional de los niveles de vitamina D en perros. También se analizó retrospectivamente la concentración de vitamina D en perros con leishmaniosis clínica y perros no infectados al inicio del estudio, cuando todos los animales eran negativos a Leishmania, y un año después. Mientras que los perros sanos no mostraron cambios en los niveles de vitamina D durante el estudio, los que desarrollaron leishmaniosis mostraron una reducción significativa al final del estudio. Por lo tanto, la concentración de vitamina D no es un factor de riesgo para desarrollar LCan, sino que disminuye con el curso de la enfermedad. Un modelo in vitro demostró que añadir vitamina D activa (1,25(OH)2D3) conlleva una reducción significativa de la carga de L. infantum en macrófagos caninos. Analizando la expresión de genes relacionados con la vía de la vitamina D en monocitos caninos primarios demostramos que la expresión de la β-defensina CBD103 aumenta significativamente con la adición de 1,25(OH)2D3. Los resultados corroboraron que la vitamina D juega un papel en el control del parásito. Por últimos, se estudió la viabilidad de la vitamina D como adyuvante para potenciar el efecto de una vacuna frente la leishmaniosis. Se administró vitamina D junto a una vacuna de ADN encapsulada en liposomas a ratones BALB/c. Dos semanas después de la vacunación los animales se infectaron con L. infantum. Se determinó la carga parasitaria en órganos diana y se evaluó la respuesta inmune antes de la infección y seis semanas después. La vacuna no redujo significativamente la carga parasitaria, pero con la coadministración de vitamina D se apreció una tendencia a reducirla. El estudio de la respuesta inmunológica sugirió que el aumento de linfocitos T CD4+ y CD8+ podrían haber contribuido a la protección parcial conseguida cuando se administró vitamina D como potenciador junto a la vacuna.
Leishmaniasis are a group of neglected vector-borne diseases caused by obligate intracellular protozoan parasites of the genus Leishmania. Human visceral leishmaniasis (VL) can be fatal if left untreated, resulting in 26 000-65 000 deaths per year. Canids are the main reservoir and hosts of L. infantum, the causative agent of zoonotic VL in the Mediterranean Basin. The mechanisms that regulate the outcome of the infection are undisclosed, although it is well known that immune system plays a key role in leishmaniasis disease control. Several studies have shown that vitamin D plays an important immunomodulatory role by activating innate immune system and modulating the adaptive immune response. Furthermore, the relationship between vitamin D deficiency and the risk of suffering from a plethora of health disorders has been described. The aim of the thesis was to study if vitamin D have a relevant contribution in canine leishmaniasis (CanL). Because of that, we measured vitamin D concentration in serum samples from a cohort of healthy and ill dogs from a highly endemic area and we have also studied the relationship of vitamin D concentration with parasitological and immunological parameters. The sick dogs presented significantly lower vitamin D levels than their non-infected and the asymptomatic counterparts. In addition, vitamin D deficiency correlated with several parameters linked to leishmaniasis progression. We also aimed to investigate whether genetic variation within the vitamin D receptor gene locus is associated with the progression of CanL, but the allelic frequencies of the four single nucleotide polymorphisms (SNPs) found were not statistically different between groups. Afterwards, we analysed retrospectively vitamin D concentration in serum samples from a cohort of healthy dogs collected in different periods of the year. The results showed that there is not a seasonal variation of vitamin D concentration in dogs. We also analysed retrospectively vitamin D concentration in serum samples from dogs with clinical leishmaniasis and non-infected healthy dogs, in which we measured vitamin D levels at the beginning of the study, when all dogs were negative for Leishmania, and 1 year later. Whereas non-infected dogs showed no changes in vitamin D levels along the study, those developing clinical leishmaniasis showed a significant vitamin D reduction at the end of the study. Therefore, vitamin D concentration is not a risk factor for developing canine leishmaniasis, but it diminishes with the onset of clinical disease. An in vitro model using a canine macrophage cell line proved that adding active vitamin D (1,25(OH)2D3) leads to a significant reduction in L. infantum load. Analyzing expression of genes related to vitamin D pathway on primary canine monocytes, we showed that defensin CBD103 expression was significantly enhanced after active vitamin D addition. The in vitro results corroborated that vitamin D plays a role in parasitic control. Finally, we studied the suitability of vitamin D as an adjuvant to enhance the effect of a DNA vaccine against VL. BALB/c mice were treated with vitamin D concomitantly with a DNA vaccine encapsulated in liposomes. Two weeks after vaccination, the animals were infected with L. infantum parasites. Parasite load was measured in target tissues and immune response was evaluated before challenge and six weeks post-infection. Our DNA vaccine did not significantly reduce parasite load in liver nor spleen, but vitamin D coadministration showed a tendency to diminish parasite load in target organs. The study of cell response in splenocytes suggested that higher levels of CD4+ and CD8+ T cells may be responsible for the partial protection mediated by the DNA vaccine with vitamin D as enhancer.
Universitat Autònoma de Barcelona. Programa de Doctorat en Farmacologia
Ball, Lindsay Clare. "Cystic fibrosis and vitamin D supplementation." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2010. https://www.mhsl.uab.edu/dt/2010m/ball.pdf.
Full textBooks on the topic "Vitamin D"
Wesley, Pike J., S. Adams John, and ScienceDirect (Online service), eds. Vitamin D. 3rd ed. San Diego: Academic Press [Imprint], 2011.
Find full textRona, Zoltan P. Vitamin D: The sunshine vitamin. Summertown, Tenn: Books Alive, 2009.
Find full text1939-, Feldman David, Glorieux Francis H, and Pike J. Wesley, eds. Vitamin D. 2nd ed. Amsterdam: Elsevier Academic Press, 2005.
Find full textTangpricha, Vin, ed. Vitamin D. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26176-8.
Full textHolick, Michael F., ed. Vitamin D. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9.
Full textHolick, Michael F., ed. Vitamin D. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-4757-2861-3.
Full text1939-, Feldman David, Glorieux Francis H, and Pike J. Wesley, eds. Vitamin D. San Diego: Academic Press, 1997.
Find full textMilne, G. W. A., and M. Delander, eds. Vitamin D Handbook. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2007. http://dx.doi.org/10.1002/9780470238165.
Full textMalone, Stephanie R. Vitamin D: Nutrition, side effects, and supplements. Hauppauge, N.Y: Nova Science, 2010.
Find full textDowd, James. The vitamin D cure. Hoboken, N.J: John Wiley & Sons, 2012.
Find full textBook chapters on the topic "Vitamin D"
Holick, Michael F. "Vitamin D and Health: Evolution, Biologic Functions, and Recommended Dietary Intakes for Vitamin D." In Vitamin D, 3–33. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_1.
Full textSilver, Justin, and Tally Naveh-Many. "Vitamin D and the Parathyroids." In Vitamin D, 235–54. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_10.
Full textCarlberg, Carsten. "Diversity of Vitamin D Target Genes." In Vitamin D, 255–74. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_11.
Full textBikle, Daniel D. "Extrarenal Synthesis of 1,25-Dihydroxyvitamin D and Its Health Implications." In Vitamin D, 277–95. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_12.
Full textLiu, Philip T., Martin Hewison, and John S. Adams. "Vitamin D and the Innate Immunity." In Vitamin D, 297–310. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_13.
Full textCross, Heide S., and Meinrad Peterlik. "Vitamin D and Colon Cancer." In Vitamin D, 311–23. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_14.
Full textLarriba, María Jesús, and Alberto Muñoz. "Mechanisms of Resistance to Vitamin D Action in Human Cancer Cells." In Vitamin D, 325–34. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_15.
Full textHarvey, Louise, Thomas Burne, Xiaoying Cui, Alan Mackay-Sim, Darryl Eyles, and John McGrath. "Vitamin D and the Brain: A Neuropsychiatric Perspective." In Vitamin D, 335–44. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_16.
Full textZemel, Michael B., and Xiaocun Sun. "Vitamin D Modulation of Adipocyte Function." In Vitamin D, 345–58. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_17.
Full textCalvo, Mona S., and Susan J. Whiting. "Determinants of Vitamin D Intake." In Vitamin D, 361–82. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_18.
Full textConference papers on the topic "Vitamin D"
Berg, Isaac, Corrine K. Hanson, Harlan R. Sayles, Debra Romberger, Amy J. Nelson, Jane L. Meza, Bruce Miller, Lisa D. Edwards, and Stephen I. Rennard. "Vitamin D, Vitamin D Binding Protein, And Airflow In COPD." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3749.
Full textBellocchia, Michela, Monica Boita, Filippo Patrucco, Cinzia Ferrero, Giulia Verri, Daniela Libertucci, Francesco Coni, Giuseppe Tabbia, Alessio Mattei, and Caterina Bucca. "Vitamin D deficiency and COPD exacerbations: Effect of vitamin D supplementation." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa3961.
Full textHsia, Connie C. W., Madhuri Poduri, Elaine Isaminger, and Khashayar Sakhaee. "Inverse Relationship Between Vitamin D Metabolites During Vitamin D Repletion In Sarcoidosis." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3004.
Full textKocak, A., D. Harmancı, M. Birlik, and G. Guner. "AB0183 Vitamin d and vitamin d receptor in patients with scleroderma subtypes." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2158.
Full textLehouck, An, Els Wauters, Chantal Mathieu, Claudia Carremans, Marc Decramer, Diether Lambrechts, and W. Janssens. "Vitamin D Binding Protein Phenotypes Have An Impact On Vitamin D Substitution In COPD." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2979.
Full textS. ABDULREDHA, Wafa, Amel S. Abdulredha, Haider O. Shuhaib, Afrah THIAB, and Amall Hussein ANATHEIL. "VITAMIN D AND PHYSICAL ACTIVITY: REVIEW." In VI.International Scientific Congress of Pure,Applied and Technological Sciences. Rimar Academy, 2022. http://dx.doi.org/10.47832/minarcongress6-34.
Full textAksan, A., J. Stötzel, L. Tessmer, O. Schröder, and J. Stein. "Effect of vitamin D supplementation on vitamin D, its metabolites and vitamin D binding protein in inflammatory bowel disease and their relation to inflammation." In Viszeralmedizin 2023 77. Jahrestagung der DGVS mit Sektion Endoskopie Herbsttagung der Deutschen Gesellschaft für Allgemein- und Viszeralchirurgie mit den Arbeitsgemeinschaften der DGAV und Jahrestagung der CACP. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1771765.
Full textBatai, Ken, Ebony Shah, Maria Ruden, Jennifer Newsome, Adam Murphy, and Rick A. Kittles. "Abstract 4481: Vitamin D pathway gene variants associated with vitamin D deficiency in African Americans." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4481.
Full textHewison, Martin. "Vitamin D, Immunity and covid-19." In Virtual 2021 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2021. http://dx.doi.org/10.21748/am21.128.
Full textMazur, Anna, Katarzyna Koziorowska, Klaudia Dynarowicz, and David Aebisher. "Vitamin D Supplementation and Photodynamic Therapy." In IECN 2022. Basel Switzerland: MDPI, 2022. http://dx.doi.org/10.3390/iecn2022-12382.
Full textReports on the topic "Vitamin D"
Janowsky, Esther. Vitamin D and Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, April 1997. http://dx.doi.org/10.21236/ada327042.
Full textJanowsky, Esther. Vitamin D and Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, July 1997. http://dx.doi.org/10.21236/ada334101.
Full textJanowsky, Esther. Vitamin D and Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, July 1995. http://dx.doi.org/10.21236/ada298702.
Full textHansen, Natasha. Vitamin D Deficiency in Athletes. Ames (Iowa): Iowa State University, January 2019. http://dx.doi.org/10.31274/cc-20240624-1591.
Full textLiu, Tang, Zhimin Yang, Zhihong Li, and Xiaolei Ren. Differential Effects of Vitamin D And Active Vitamin D Supplements for Preventing Falls: A Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2020. http://dx.doi.org/10.37766/inplasy2020.9.0018.
Full textBalint, Eva. Vitamin D, Breast Cancer and Bone Health. Fort Belvoir, VA: Defense Technical Information Center, October 2008. http://dx.doi.org/10.21236/ada499634.
Full textFeldman, David. Interactions between Vitamin D and Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2008. http://dx.doi.org/10.21236/ada502825.
Full textSchlichting, Christine L., and David J. Styer. Vitamin D Status of Submariners during Patrol. Fort Belvoir, VA: Defense Technical Information Center, January 1989. http://dx.doi.org/10.21236/ada206776.
Full textFeldman, David. Interactions Between Vitamin D and Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, July 2010. http://dx.doi.org/10.21236/ada537364.
Full textFeldman, David, and Robert Carlson. Interactions Between Vitamin D and Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2009. http://dx.doi.org/10.21236/ada518246.
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