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Books on the topic 'Virus induced diseases'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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1

Sandberg, Kristian. Characterization of the interferon-alpha/beta producing leukocytes and their responses to viral inducers. Uppsala: Sveriges Lantbruksuniversitet, 1991.

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2

Steven, Specter, Bendinelli Mauro, and Friedman Herman 1931-, eds. Virus-induced immunosuppression. New York: Plenum Press, 1989.

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3

M, Goldman J., Epstein M. A, and Leukaemia Research Fund, eds. Vaccine intervention against virus-induced tumours. Basingstoke, Hampshire: Macmillan, 1986.

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4

Vaccine Intervention Against Virus-induced Tumors (Leukaemia & Lymphoma Research). Mcgraw-Hill (Tx), 1986.

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5

Baker, Carlye Ann. Production and characterization of polyclonal and monoclonal antibodies to three virus-induced proteins of papaya ringspot virus type W. 1989.

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6

Jackson, Michael Keven. Nucleotide sequence of S reading frame and viral-induced trans-activation of caprine arthritis-encephalitis virus. 1990.

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7

Knowles, Donald P. Studies of the pathogenesis of viral-induced arthritis and the transcriptional organization of caprine arthritis-encephalitis virus. 1988.

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8

Norbert, Gilmore, Wainberg Mark A, Medical Research Council (Canada), Canada. National Advisory Committee on AIDS., and Workshop on Viral Mechanisms of Immunosuppression (1984 : Montréal, Quebec), eds. Viral mechanisms of immunosuppression: Proceedings of a workshop. New York: Liss, 1985.

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9

AIDS: Virus- or Drug Induced? Springer, 2011.

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10

Peter, Duesberg, ed. AIDS: Virus or drug induced? Dordrecht: Kluwer Academic Publishers, 1996.

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11

(Editor), R. D. Hunt, ed. Nonhuman Primates I (Monographs on Pathology of Laboratory Animals). Intl Life Sciences Inst, 1993.

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12

Duesberg, Peter. AIDS: Virus or Drug Induced? (Contemporary Issues in Genetics and Evolution). 9th ed. Springer, 1996.

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13

Duesberg, Peter. AIDS: Virus or Drug Induced? (Contemporary Issues in Genetics and Evolution). 9th ed. Springer, 1996.

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14

Bale, James F. Congenital and Perinatal Viral Infections. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0160.

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Despite remarkable advancements in the treatment and prevention of infectious diseases, congenital (also known as intrauterine) and perinatal (also known as neonatal) infections remain major causes of permanent neurodevelopmental disabilities worldwide. Fortunately, relatively few viral pathogens can infect the developing fetus or the newborn postnatally and induce neurological disease. These pathogens include cytomegalovirus, rubella virus, herpes simplex virus types 1 and 2, varicella zoster virus, lymphocytic choriomeningitis virus, the nonpolio enteroviruses, parechovirus, and human immunodeficiency virus. This chapter describes the clinical manifestations, diagnosis, treatment, and outcome of these congenital and perinatal viral infections.

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15

Muche, Marion, and Seema Baid-Agrawal. Hepatitis B. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0185_update_001.

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Hepatitis B virus (HBV) has been causally linked to a variety of renal diseases, the most common being glomerular diseases and systemic autoimmune disease. Membranous nephropathy (MN) is the commonest HBV-associated glomerulonephritis (HBV-GN), followed by membranoproliferative glomerulonephritis (MPGN), mesangial proliferative glomerulonephritis, immunoglobulin (Ig)-A nephropathy, and focal segmental glomerulosclerosis (FSGS). Polyarteritis nodosa is a rare manifestation. The incidence of HBV-associated renal diseases seems to be decreasing with the introduction of vaccination programmes.HBV-MN is the most frequent cause of nephrotic syndrome in children in countries with high endemicity of HBV infection. The clinical course is usually benign in children with high rates of spontaneous remission rates and low risk of progression to renal failure. The prognosis is worse in adults. Of the systemic autoimmune disorders associated with HBV infection that involve the kidneys, the strongest link has been found with polyarteritis nodosa (PAN), a lesion that causes arteritis of medium-sized renal vessels. HBV-associated PAN (HBV-PAN) usually manifests in the first year after infection, and is clinically indistinguishable from classic PAN.Diagnosis of HBV-GN or -PAN is based on the clinical picture, histological findings, evidence of viral replication in serum and/or liver and detection of HBV antigens or DNA in the tissue. Besides deposition of immune complexes, other mechanisms such as virus-induced cytopathic damage have been proposed to explain the pathogenesis.HBV-GN and HBV-PAN appear to respond to antiviral treatment. Both show remission after HBeAg seroconversion. The available studies predominantly employed first-generation agents like interferon alpha and lamivudine, which showed suppression of viral replication and clinical remission of HBV-associated renal disease. Immunosuppressive therapy appears to be inevitable for the control of severe HBV-PAN and could be helpful in addition to antiviral treatment for cases of HBV-GN not responding clinically to antiviral treatment.

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16

Jean-Marie, Andrieu, Bach Jean-François, and Even P, eds. Autoimmune aspects of HIV infection. London: Royal Society of Medicine Services, 1988.

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17

(Editor), P. Even, ed. Autoimmune Aspects of HIV Infection (International Congress and Symposium Series No 141). Royal Society of Medicine, 1988.

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18

Казачинская, Е. И. ВИРУС ДЕНГЕ. Академическое изд-во «Гео», 2021. http://dx.doi.org/10.21782/b978-5-6043022-6-2.

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The review is devoted to the analysis of literature data on the history research of dengue fever, the discovery of the etiological infectious agent of this disease-dengue virus and its serotypes. A taxonomic overview of the }lavivirus family, genome organization, structure and function of viral proteins, mosquito species-viral vectors and virus transmission cycles, theories of its origin are presented. As well as the evolution, characteristics and epidemiology of viral serotypes, cellular receptors for dengue virus penetration, pathogenicity for human and factors for the development of severe disease, induced immunity, applied methods and markers for diagnosis, principles of disease treatment and drug development (more information about monoclonal antibodies-potential therapeutic drugs), vaccine options and their effectiveness are considered. The book is intended for students, graduate students, employees of research institutions and universities, as well as doctors involved in the study of }laviviruses and the problem of differential diagnosis of flavivirus infections.

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19

Transformation and regeneration of groundnut, and utilization of viral genes to induce resistance to virus diseases: Summary and recommendations of a meeting, 24-27 Apr 1992. Patancheru: International Crops Research Institute for the Semi-Arid Tropics, 1992.

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20

Reddy, D. V. Transformation and Regeneration of Groundnut, and Utilization of Viral Genes to Induce Resistance to Virus Diseases Summary and Recommendations of a Meeting 24-27 Apr. 1992. International Crops Research Institute for the Semi-Arid Tropics (ICRISAT), 1992.

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21

International Crops Research Institute for the Semi-arid Tropics., ed. Transformation and regeneration of groundnut, and utilization of viral genes to induce resistance to virus diseases: Summary and recommendations of a meeting, 24-27 Apr 1992, Virology Department, Wageningen Agricultural University, The Netherlands. Patancheru, Andhra Pradesh, India: ICRISAT, 1992.

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22

International Crops Research Institute for the Semi-arid Tropics. and Wageningen Universiteit. Virology Dept., eds. Transformation and regeneration of groundnut, and utilization of viral genes to induce resistance to virus diseases: Summary and recommendations of a meeting, 24-27, Apr 1992, Virology Department, Wageningen Agricultural University, the Netherlands. Patancheru: International Crops Research Institute for the Semi-Arid Tropics, 1992.

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23

International Crops Research Institute for the Semi-arid Tropics. and Wageningen Universiteit. Virology Dept., eds. Transformation and regeneration of groundnut, and utilization of viral genes to induce resistance to virus diseases: Summary and recommendations of a meeting, 24-27, Apr 1992, Virology Department, Wageningen Agricultural University, the Netherlands. Patancheru: International Crops Research Institute for the Semi-Arid Tropics, 1992.

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