Relevant bibliographies by topics / Virology

Academic literature on the topic 'Virology'

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Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "Virology"

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White, David. "Virology." Medical Journal of Australia 145, no. 11-12 (December 1986): 646. http://dx.doi.org/10.5694/j.1326-5377.1986.tb139533.x.

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White, David. "Virology." Medical Journal of Australia 150, no. 9 (May 1989): 539. http://dx.doi.org/10.5694/j.1326-5377.1989.tb136671.x.

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Goudsmit, Jaap. "Virology." Current Opinion in Infectious Diseases 8, no. 1 (February 1995): 54–58. http://dx.doi.org/10.1097/00001432-199502000-00012.

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Schiller, John. "Virology." Papillomavirus Report 14, no. 2 (March 2003): 85–87. http://dx.doi.org/10.1179/095741903235001920.

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Tyring, Stephen K. "Virology." Dermatologic Clinics 20, no. 2 (April 2002): xi. http://dx.doi.org/10.1016/s0733-8635(02)00003-7.

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Flewett, T. H. "Virology." Postgraduate Medical Journal 66, no. 771 (January 1, 1990): 73. http://dx.doi.org/10.1136/pgmj.66.771.73-a.

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Ratner, Lee, and Eva Maria Fenyö. "Virology." AIDS 10, Supplement (January 1996): S1–2. http://dx.doi.org/10.1097/00002030-199601001-00001.

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Weiss, Robin A., and Jay A. Levy. "Virology." AIDS 2 (1988): S1–2. http://dx.doi.org/10.1097/00002030-198800001-00001.

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Kurth, Reinhard, and Flossie Wong-Staal. "Virology." AIDS 7 (January 1992): S1—S2. http://dx.doi.org/10.1097/00002030-199201001-00001.

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Kurth, Reinhard, and Flossie Wong-Staal. "Virology." AIDS 7 (January 1993): S1—S2. http://dx.doi.org/10.1097/00002030-199301001-00001.

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Dissertations / Theses on the topic "Virology"

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Johansson, Susanne. "Genomic Organization and Capsid Architecture of Ljungan Virus : a Novel Member of the Picornaviridae." Doctoral thesis, Högskolan i Kalmar, Naturvetenskapliga institutionen, 2002. http://urn.kb.se/resolve?urn=urn:nbn:se:hik:diva-46.

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Ljungan virus är ett virus som isolerades i Sverige i mitten på nittiotalet. Under perioden 1989-1992 avled flera svenska elitorienterare plötsligt i hjärtmuskelinflammation. Man misstänkte att orienterarna kunde ha utsatts för en vektorburen infektion eftersom de exponeras för djur som finns i skog och mark under träning och tävling. Det är sedan tidigare känt att sorkar är den naturliga reservoaren för ett annat virus (Puumala virus) som kan orsaka njurskada hos människor. Sorkantalet i vissa delar av Sverige varierar kraftigt från år till år i ett cykliskt förlopp. Man fann ett samband mellan antalet sorkar och förekomsten av hjärtmuskelinflammation, typ 1 diabetes och Guillain-Barre's syndrom vilket ledde till att ett tidigare okänt virus, Ljungan virus, kunde isoleras från sorkar. Detta virus är ett litet RNA-virus som tillhör familjen Picornavirus. Till denna familj hör också flera kända virus såsom många av våra vanligt förekommande förkylningsvirus, men också virus som kan orsaka svåra sjukdomar, till exemel poliovirus och mul- och klövsjukevirus. Ljungan virus är ett nyupptäckt virus och därför är kunskapen om viruset begränsad. För att öka vår förståelse om viruset så har arvsmassan för tre svenska isolat (87-012, 174F och 145SL) av Ljungan virus kartlagts (artikel IV). Denna studie visade att Ljungan virusets arvmassa har flera unika egenskaper. Släktskapstudier visade att Ljungan virus är endast avlägset släkt med redan kända picornavirus och viruset bör därför utgöra en egen undergrupp i familjen (artikel III). Med kunskap om Ljungan virusets arvsmassa så var det möjligt att visa att Ljungan virus förekommer även på andra ställen än i Sverige (artikel VI). I mitten på 60-talet isolerades ett virus, M1146, från sork som fångats i Oregon, USA. Baserat på egenskaper hos proteinhöljet (kapsiden) så antog man då att M1146 var ett picornavirus. Studier av arvsmassan för detta virus visade att M1146 är närmast besläktat med de svenska Ljungan virus isolaten och har samma unika egenskaper i sin arvsmassa (artikel VI). Dessa studier har varit möjliga eftersom vi tidigare har utvecklat en metod för att producera stora mängder av hela arvsmassan (artikel I och II). Slutligen har det proteinhölje som innesluter och skyddar Ljungan virusets arvsmassa studerats (artikel V). Dessa studier visade att kapsiden är uppbyggd av tre proteiner och inte fyra som hos de flesta picornavirus. Dessa studier underlättades av att en enkel och effektiv metod för att odla Ljungan virus i provröret har utvecklats (artikel V). Sammantaget så har dessa studier försett oss med nya kunskaper om Ljungan virus som möjliggör fortsatta studier av dess biologi och eventuella förmåga att orsaka sjukdom hos människor och djur.
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Kindberg, Elin. "Host genetic risk factors to viral diseases - a double-edged sword : Studies of norovirus and tick-borne encephalitis virus." Doctoral thesis, Linköpings universitet, Molekylär virologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-54923.

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It is today well known that the outcome of a certain infection depends on factors of both the host and the pathogen. Studies of host genetic susceptibility to infectious diseases aim to increase the understanding of why some individuals are more susceptible than others, to a certain infection. Knowledge of genetic susceptibility to a viral disease may be used in development of new therapeutic means, and also to recognize individuals who are at increased risk of severe symptoms if infected with a pathogen. It seems however that a risk factor for one disease may play a protective role in another situation; like a double-edged sword. In this thesis I have studied genetic factors affecting susceptibility to norovirus (NoV) and factors affecting the risk of developing tick-borne encephalitis (TBE) after infection with TBE virus (TBEV). NoV is the cause of the “winter vomiting disease”, affecting millions of people every year, and causing up to 200,000 fatalities among children in developing countries, each year. It is today recognized that the secretor status of an individual, i.e. the ability to express ABO blood groups and related antigens, in secretions and on mucosa, affect the risk of being infected by NoV. By studying authentic NoV outbreaks in Denmark, Spain and Sweden and by comparing the secretor status of affected and unaffected individuals we were able to confirm that secretor status have indeed great impact on susceptibility to some NoV strains, but also that there are strains circulating, which infect individuals regardless of secretor status. TBEV is endemic in many parts of Europe and Asia but studies have shown that 70-95% of all infections are asymptomatic or sub-clinical. Some individuals do however develop TBE, a severe disease including meningitis or encephalitis with or without myelitis. Also, many patients suffer from long-time sequelae and TBEV infections may in worst case be fatal. The reason for difference in disease outcome is not known and we have chosen to study if genetic factors affecting the immune response may play a role in disease outcome. To do this we used a prospectively collected Lithuanian material with samples from patients with TBE, AME (aseptic meningoencephalitis) and matched healthy controls. So far we have found that a deletion in chemokine receptor 5 (CCR5), a gene encoding a receptor involved in cell migration, is a risk factor for developing disease. We have also data showing that toll-like receptor 3 (TLR3), a receptor recognizing double stranded RNA (dsRNA), which is a product of TBEV replication, may instead of being protective increase the risk of TBE.
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Backström, Ellenor. "Regulation of Adenoviral Gene Expression by the L4-33K and L4-22K Proteins." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-101324.

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The splicing pattern during an adenovirus infection is shifted at the late phase towards using weaker splice sites, splicing out larger introns. Splicing of weak 3´ splice sites usually requires recognition of the 3´AG dinucleotide before the first catalytic step of splicing. Such splicing events are said to be AG-dependent and requires an interaction of both subunits of the cellular splicing factor U2AF with the 3´ splice site. We show that splicing of transcripts that are AG-dependent in uninfected nuclear extracts (NE) becomes AG-independent in nuclear extracts prepared form adenovirus late-infected HeLa cells (Ad-NE). Further we demonstrate that the first step in splicing of a model transcript, IgM, becomes completely U2AF-independent in Ad-NE. This finding supports our working model that 3´ splice site recognition in Ad-NE is altered, and in fact might be U2AF-independent. We further show that the adenovirus late protein L4-33K acts as a virus encoded alternative splicing factor. L4-33K activates splicing of both cellular and viral transcripts containing weak 3´ splice sites. This supports the hypothesis that adenovirus alter splicing during the infection to favour usage of weak, suboptimal 3´ splice sites. However, we were unable to find an alternative U2AF-related factor that could stimulate L4-33K splicing enhancer activity. Furthermore, we demonstrate that the serine residues in the C-terminal part of L4-33K are important for the splicing enhancer activity but also for its nuclear localisation. The adenovirus major late promoter is highly activated after the onset of viral genome replication. Protein complexes binding to downstream elements of the promoter are required for full enhancement of this promoter. We show that an L4-33K-related protein, L4-22K, stimulates transcription from the major late promoter. This stimulation is mainly via the downstream elements and does not require the viral IVa2 protein, which is a transcription factor of the major late promoter.
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Vildevall, Malin. "The Norovirus Puzzle : Characterization of human and bovine norovirus susceptibility patterns." Doctoral thesis, Linköpings universitet, Molekylär virologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-68386.

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Winter vomiting disease is caused by norovirus (NoV) and affects millions of people every year resulting in 200.000 deaths among children in developing countries. It was observed early that not all individuals exposed to the norovirus became ill. The reason for this is now recognized to be dependent upon the secretor status of an individual. The secretor status determines the ability of an individual to express histo-blood group antigens (HBGA) on mucosa and in saliva. A non-secretor is unable to express HBGAs due to a mutation in a gene called FUT2. In this thesis, I have investigated the antibody prevalence and titer in humans in Sweden and Nicaragua to the most common GII NoV and the correlation to secretor status, Lewis status and ABO. I found that secretors had significantly higher antibody prevalence and titer to GII NoV than non-secretors suggesting that non-secretors are less prone to be infected by the GII NoV. In Nicaragua, I also found several different NoV strains circulating at the same time. The NoVs have been circulating and evolving in the human population for some time and the same individuals seems to be infected over and over again with the same virus. This suggests that there is no long-term immunity present but possibly short-term immunity, which would make it very difficult to produce a vaccine against NoV. However, recent studies have shown the possibility of using virus like particles as a vaccine candidate and have demonstrated long-term immunity. The bovine NoV (boNoV) cause gastroenteritis in cattle and are closely related to the human NoV. The possibility of zoonotic transfer to humans is currently being investigated. I found that 26% of Swedish blood donors have antibodies to the boNoV suggesting that they have been exposed to the virus. The human NoV has been observed to be able to infect and cause disease in cattle, could the boNoV do the same in humans? To date, no boNoV strain has been found in humans. The proposed receptor structure for boNoV is the αGal epitope, which is present in many mammals like cow, pig, horse, sheep and rabbit but not in humans. This indicates that humans are not at risk for boNoV infection because we lack the proper receptor structure. However, recombinations between different NoV strains have been demonstrated and the possibility of more than one receptor being present has been suggested. I found that aa position 365-379 on the boNoV capsid seems to be important for binding to erythrocytes. In this thesis, I hope to add some new pieces to the Norovirus Puzzle.
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Somberg, Monika. "Cellular and Viral Factors that Control Human Papillomavirus Type 16 Late Gene Expression." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-150706.

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Human papillomavirus type 16 (HPV-16) is the major cause of cervical cancer. We speculate that inhibition of HPV-16 late gene expression is a prerequisite for establishment of persistence and progression to cervical cancer. This is based on the findings that the late proteins are found only in the nuclei of terminally differentiated epithelium, and are never detected in human papillomavirus infected cervical cancer cells. It is therefore of great importance to understand how HPV-16 controls the onset of the immunogenic proteins L1 and L2 in an infected cancer cell. HPV-16 late gene expression is tightly regulated by differentiation-dependent transcription as well as by post-transcriptional mechanisms. The long-term goal of these studies was to understand how HPV late gene expression is regulated. The specific aim of this thesis was to identify cellular and viral factors that force the virus to switch on the late genes, and to determine the mechanism of action of these factors. This will help us to understand under which circumstances HPV establish persistent infections that could progress to cancer. We found three cellular factors; PTB, ASF/SF2 and SRp30c, and one viral factor; AdE4orf4, that in four distinctive ways were involved in the regulation of HPV-16 late gene expression. Interestingly, over-expression of PTB, AdE4orf4 or SRp30c produced different types of spliced late mRNAs. PTB induced the unspliced L2/L1 mRNA, while AdE4orf4 and SRp30c induced the spliced L1 and L1i mRNA, respectively. The three proteins had different mechanisms of action and different target sites within the HPV-16 genome, which revealed the many and complex pathways in HPV-16 gene regulation. These findings have contributed to a broader understanding of how the expression of HPV-16 late genes is controlled.
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Polstra, Abeltje Mette. "Human herpesvirus 8: virology and disease." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2004. http://dare.uva.nl/document/74171.

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Jensen, Stephanie Meryl, and Stephanie Meryl Jensen. "A Bioorthogonal Approach to Chemical Virology." Diss., The University of Arizona, 2016. http://hdl.handle.net/10150/621769.

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Dengue virus (DENV) is a mosquito-transmitted flavivirus that threatens approximately half of the world's population. In this dissertation, the use of bioorthogonal chemistry as a tool for researching emerging viral diseases, including DENV is explored. To this end, a bioorthogonally-modified amino acid was successfully installed within the proteome of DENV, which was used for the pull down of a known virus-protein interaction. This technology is intended to be broadly used for the determination of any virus-host interaction, through the installment of a non-perturbing modification that 1) does not hinder viral infectivity and 2) can be selectively discriminated by any complimentary probe. En route to using this technology, a new viral purification strategy was developed for DENV that reduces the overall purification time by 10 hours, and improves retention of virion infectivity. This method and a survey of other viral purification methods used with DENV is contained herein. Furthermore, a chemical scaffold that was repurposed for exploration of protein-protein crosslinking, namely for release of a reactive chemical warhead under acidic conditions, was used for the surface modification of DENV. This triazabutadiene probe was found to be activated by light. In this dissertation is reported the first time aryl diazonium ions for protein crosslinking have been generated on a protein or viral surface through UV-irradiation. The advantages and limitations of this chemistry are presented herein.
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Watanabe, Aripuanã Sakurada Aranha [UNESP]. "Pesquisa do vírus influenza HRSV e hMPV em uma população de idosos da cidade de Botucatu - São Paulo, Brasil." Universidade Estadual Paulista (UNESP), 2006. http://hdl.handle.net/11449/89984.

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Made available in DSpace on 2014-06-11T19:24:16Z (GMT). No. of bitstreams: 0 Previous issue date: 2006Bitstream added on 2014-06-13T20:51:50Z : No. of bitstreams: 1 watanabe_asa_me_botfm.pdf: 435949 bytes, checksum: e2d8389a510d8ed3643dbe728b2b723a (MD5)
From about 200 virus that cause respiratory infections, only 8 are responsible for severe illness in children, immunodeficient adults and elderly, inc1uding Adenovirus, Influenza A and B, Parainfluenza 1, 2 and 3, Human RespÍratory Syncytial Vírus (HRSV) and Human Metapneumovirus (hMPV). Three of these virus are responsible for a significant morbidity in elderly: Influenza, HRSV and hMPV. The nosocomial infection caused by these vÍruses can be fatal in hospitalized children and patients with other pathologies. With the advance of the of molecular biology techniques, the diagnosis and the characterization of these vírus became more effective. Beside vírus detection by PCR, isolation of vírus in specific cellular cultures to increase the amount of pathogens have been used in severallaboratories. Studies on prevalence of respÍratory vírus in elderly are rare. The objective of this research was to evaluate the occurrence of Influenza, HRSV and of the hMPV and the risk factors involved in the diseases caused by these vÍruses. Nasopharyngeal swabs were collected and used for inoculation in cells culture and dÍrect analysis by RT-PCR. The results from RT-PCR of Influenza Vírus and RSV were negative. We also tested the samples with GeneScan, that is a technique based on fluorescent primers specific to the studied vírus. Results for Flu virus and HRSV were the same ofRT-PCR, but 1 MPV sample was positive. 55,32% ofthe 47 studied elderly were vaccinated against Influenza; 14,9% had tabagism habits and 70,2% were women. These risk factors might had influenced in the absence of positive samples for the Influenza vírus andHRSV.
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Groussaud, Damien. "Modulation de la machinerie de O-GlcNACylation par l’oncoprotéineTax du virus HTLV-1 et implication dans la transactivation du promoteur viral." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB252/document.

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Le virus HTLV-1 est le seul rétrovirus humain oncogène découvert à ce jour à l’origine d’une lymphoprolifération maligne à cellules T, la leucémie aiguë de l’adulte (ATL). L’oncoprotéine Tax du virus HTLV-1 joue un rôle majeur dans l’établissement de la leucémie mais aussi dans la réplication du virus. Tax régule la transcription du promoteur viral situé au sein du LTR 5’ du virus, gouvernant ainsi sa propre production. Pour ce faire, Tax recrute des dimères du facteur de transcription cellulaire CREB, sous forme phosphorylée, au niveau d’éléments de réponse à l’AMP cyclique (vCRE) situés dans la partie U3 du LTR. La phosphorylation de CREB est un processus crucial pour la transactivation du promoteur viral. CREB est aussi ciblé et régulé par OGlcNAcylation, modification post traductionnelle impliquée dans de nombreuses pathologies comme le cancer, dont la régulation dépend de deux enzymes, l’OGA et l’OGT, formant le complexe OGlcNAzyme. Du fait que Tax dérégule de nombreuses machineries de modifications post traductionnelles et que la OGlcNAcylation régule avec la phosphorylation la stabilité et l‘activité de facteurs de transcriptions, nous avons évalué pour la première fois le statut de la OGlcNAcylation de CREB dans le cadre de l’infection par HTLV-1 et son implication sur la régulation du promoteur viral. Nous avons pu montrer que dans le cadre de l’infection par HTLV-1, la protéine Tax dérégulait la machinerie de OGlcNAcylation en inhibant l’activité de l’OGA. Pour ce faire, Tax interagit avec le complexe OGlcNAzyme entrainant une augmentation de la OGlcNAcylation de CREB favorisant ainsi la transcription du LTR 5’. Ainsi nous avons pu établir pour la première fois une relation entre Tax et la OGlcNAcylation permettant de proposer un nouveau modèle de régulation du LTR viral
HTLV-1 virus is the only oncogenic human retrovirus discovered to date. It is responsible for a T cell malignant lymphoproliferation named Adult T cell Leukemia. HTLV-1 Tax oncoprotein plays a major role in the development of the leukemia and also in the viral replication. Tax regulates the transcription from the viral promoter located in the virus 5’LTR, favoring its own transcription. In order to do this, Tax recrutes dimers of the phosphorylated cellular transcription factor CREB to viral cyclic AMP response elements (vCRE) situated in the U3 part of the LTR. CREB phosphorylation is a crucial process to allow the transactivation of the viral promoter. CREB is also targeted and regulated by OGlcNAcylation which is a post-translational modification implicated in many pathologies such as cancer. Its regulation depends on two enzymes, OGA and OGT forming the OGlcNAzyme complex. As Tax deregulates many post-translational modification machineries and as OGlcNAcylation regulates the stability and activity of transcriptional factors together with phosphorylation, we have evaluated for the first time the OGlcNAcylation status of CREB in the context of HTLV-1 infection and its implication in the regulation of the viral promoter. We have shown that in the context of HTLV-1 infection, Tax protein was deregulating the OGlcNAcylation machinery leading to an increase in CREB OGlcNAcylation which favors the transcription from the 5’LTR. Thus we have established for the first time a relationship between Tax and OGlcNAcylation allowing us to propose a new model of regulation of the viral LTR
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Sanfilippo, Luiz Francisco. "Epidemiologia e caracterização molecular do vírus da Influenza em quatro espécies de pinguins na Região Antártica." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/42/42132/tde-11082011-105843/.

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O Vírus da influenza, apesar de todas as epidemias e pandemias referirem-se a infecções em seres humanos, não está restrita a espécie humana e é capaz de causar debilidade ou mortalidade em várias outras espécies, incluindo cavalos, suínos, mamíferos marinhos e aves, entre outros. Estudos ecológicos das viroses de influenza conduziram a hipótese que todas as que acometem mamíferos derivam de reservatórios destes vírus em aves. Mesmo com programas de monitoramento contínuo de aves silvestres em alguns países do mundo que possuem casos originados pelos vírus aviário H5N1, pouco foi feito na Antártica e por isso, o presente trabalho foi realizado nas estações de verão antártico de 2006, 2007 e 2008 em duas localidades no território Antártico, a Península Keller, localizada na Ilha Rei George e na ilha Elefante 61°08S, 55°07W, a primeira onde está situada a Estação Antártica Comandante Ferraz-EACF e a segunda onde está localizada uma base de apoio a estudos avançados. Para este estudo foi realizada a coleta de 283 amostras de quatro diferentes espécies de pinguins: Pygoscelis adeliae; P. papua; P. antarctica; Aptenodytes patagonicus. Para o diagnóstico das amostras colhidas, foi aplicada a detecção direta dos produtos amplificados pelo método de RT-PCR em gel de agarose confirmados pelo método de Real-Time PCR (Applied Biosystems) e pelo RT-PCR-GeneScan no laboratório de Virologia Clínica e Molecular, do Departamento de Microbiologia, da Universidade de São Paulo. Os resultados obtidos em nosso estudo foram 8 amostras positivas em pinguins para o vírus Influenza A. As amostras positivas por RT-PCR foram encaminhadas para o laboratório de Influenza do Department of Infectious Diseases, St. Jude Children\'s Research Hospital, Memphis, TN, USA, para isolamento em ovos embrionados, não havendo crescimento de vírus da influenza A. Quatro destas amostras positivas puderam ser sequenciadas e comparadas com sequências de Influenza A depositadas no Genbank apresentando uma identidade de 96,8 % a 100 % entre elas e o controle tendo este último uma identidade de 100% com as do banco de dados, confirmando a presença do vírus nestas aves.
Epidemics and pandemics of influenza usually refer to infections in human beings. The influenza virus is not, however, restricted to humans and can cause infirmity and death in other species including horses, swine, marine mammals, birds, and others. Ecological studies of viral infections have led to the hypothesis that the influenza viruses that attack mammals have their origin in the accumulation of these viruses in birds (avian flu). In some countries with influenza cases caused by the avian H5N1 virus, there was monitoring of wild birds but little had been done in Antarctica. The present work was therefore carried out during the Antarctic summer seasons of 2006, 2007, and 2008 in two Antarctic locations: The Commander Ferraz Antarctic Station, on the Keller Peninsula of King George Island, and at the Base of Advanced Studies located on Elephant Island (61°08S, 55°07W). Two hundred eighty-three (283) samples from four different penguin species Pygoscelis adeliae, Pygoscelis papua, Pygoscelis antarctica; and Aptenodytes patagonicus were collected for this study. Diagnoses of the samples were performed not only by application of direct detection and amplification according to the RT-PCR method in agar-gel, but also by Real-Time PCR (Applied Biosystems), and by RT-PCR gene scan at the Laboratory of Clinical and Molecular Virology of the Department of Microbiology of the University of Sao Paulo. Eight of the penguin samples tested positive for the Influenza-A virus. The positive samples, as determined by RT-PCR, were sent to the Influenza Laboratory of the Department of Infectious Diseases of the St. Jude Research Hospital in Memphis, Tennessee, USA, to be isolated in egg embryos where no further growth of the Influenza-A virus took place. Four of these positive samples could be sequenced and compared with those of Influenza-A on deposit at the Gene Bank and ranged from 96.85 to 100% when compared with the control samples (100% positive), thus confirming the presence of the virus in the tested birds.
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Books on the topic "Virology"

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Hull, Roger, Fred Brown, and Chris Payne. Virology. London: Palgrave Macmillan UK, 1989. http://dx.doi.org/10.1007/978-1-349-07945-2.

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K, Joklik Wolfgang, ed. Virology. 3rd ed. Norwalk, Conn: Appleton & Lange, 1988.

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Stringfellow, Dale A., 1944- ed. and Upjohn Company, eds. Virology. Kalamazoo, Mich: Upjohn, 1986.

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Fraenkel-Conrat, Heinz. Virology. 2nd ed. Englewood Cliffs, N.J: Prentice-Hall International, 1988.

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Dulbecco, Renato. Virology. 2nd ed. Philadelphia: Lippincott, 1988.

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K, Joklik Wolfgang, ed. Virology. 2nd ed. Norwalk, Conn: Appleton-Century-Crofts, 1985.

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1946-, Kimball Paul C., and Levy Jay A, eds. Virology. 2nd ed. Englewood Cliffs, N.J: Prentice-Hall, 1988.

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N, Fields Bernard, ed. Virology. New York: Raven Press, 1985.

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1910-, Fraenkel-Conrat Heinz, and Owens Robert A. 1947-, eds. Virology. 3rd ed. Englewood Cliffs, N.J: Prentice Hall, 1994.

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Wang, Aiming, and Yi Li, eds. Plant Virology. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1835-6.

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Book chapters on the topic "Virology"

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Georgiev, Vassil St. "Virology." In National Institute of Allergy and Infectious Diseases, NIH, 41–42. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-297-1_7.

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Briones, Carlos. "Virology." In Encyclopedia of Astrobiology, 1745. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-11274-4_1659.

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Murray, Paul G. "Virology." In Encyclopedia of Cancer, 1–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_6195-2.

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Briones, Carlos. "Virology." In Encyclopedia of Astrobiology, 2605. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-44185-5_1659.

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Jeffries, Don. "Virology." In The Management of AIDS Patients, 53–63. London: Macmillan Education UK, 1986. http://dx.doi.org/10.1007/978-1-349-18079-0_3.

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Kotyk, Arnošt. "Virology." In Quantities, Symbols, Units, and Abbreviations in the Life Sciences, 77–81. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-206-7_12.

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Jeffries, D. J. "Virology." In AIDS and Obstetrics and Gynaecology, 3–13. London: Springer London, 1988. http://dx.doi.org/10.1007/978-1-4471-3150-2_1.

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Boland, G. J. "Virology." In Molecular Diagnostics, 33–50. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-4511-0_3.

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Murray, Paul G. "Virology." In Encyclopedia of Cancer, 4815–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_6195.

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Murray, Paul G. "Virology." In Encyclopedia of Cancer, 3915–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_6195.

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Conference papers on the topic "Virology"

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Savenkova, D. A., D. A. Alhireenko, S. A. Bodnev, E. Yu Prudnikova, A. V. Zaikovskaya, Yu A. Golovko, I. R. Imatdinov, A. B. Ryzhikov, O. V. Pyankov, and D. V. Yudkin. "TRANSGENIC CELL LINES IN VIROLOGY." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-119.

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Cell cultures are used as classic models for virology. Development of transgenesis and genome editing methods leads to a new possibility in creation of transgenic cell lines that can serve as more suitable models for virological research. This work describes the obtaining transgenic cell lines and the study of their susceptibility to viral infections.
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de Pablo, Pedro. "Physical Virology with Atomic Force Microscopy." In Microscience Microscopy Congress 2021 incorporating EMAG 2021. Royal Microscopical Society, 2021. http://dx.doi.org/10.22443/rms.mmc2021.265.

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Moubarak, Joanna, Eric Filiol, and Maroun Chamoun. "The Blockchain Potential in Computer Virology." In 4th International Workshop on FORmal methods for Security Engineering. SCITEPRESS - Science and Technology Publications, 2020. http://dx.doi.org/10.5220/0009371607960803.

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Zaitsev, Boris. "Atomic Force Microscopy as a Tool for Applied Virology and Microbiology." In SCANNING TUNNELING MICROSCOPY/SPECTROSCOPY AND RELATED TECHNIQUES: 12th International Conference STM'03. AIP, 2003. http://dx.doi.org/10.1063/1.1639726.

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Lee, Junghoon, Peter C. Doerschuk, and John E. Johnson. "Simultaneous 3-D Image Reconstruction and Classication with Applications to Structural Virology*." In Signal Recovery and Synthesis. Washington, D.C.: OSA, 2007. http://dx.doi.org/10.1364/srs.2007.ptua1.

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Ahmed, MI, S. Kader, S. Bandi, and J. Tang. "G371(P) Correlating virology and severity of bronchiolitis: use of bronchiolitis severity index." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 24–26 May 2017, ICC, Birmingham. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313087.364.

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Ibrahim, Mohamed. "AdaPool: Multi-Armed Bandits for Adaptive Virology Screening on Cyber-Physical Digital-Microfluidic Biochips." In MLCAD '20: 2020 ACM/IEEE Workshop on Machine Learning for CAD. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3380446.3430626.

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Held, C., J. Wenzel, R. Webel, M. Marschall, R. Lang, R. Palmisano, and T. Wittenberg. "Using multimodal information for the segmentation of fluorescent micrographs with application to Virology and microbiology." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6091601.

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Ezegbe, Henrietta, and Malcolm Steinberg. "P400 Quality improvement initiatives to strengthen viral suppression among ALHIVs in institute of human virology sites in abuja." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.492.

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Chinarro Vadillo, David, Francisco Javier Diaz Perez, Adib Guardiola Mouhaffel, and Maria Rosa Pino Otín. "HOW TO INTRODUCE IN A VIROLOGY CLASS THE COVID-19'S EPIDEMIC RESEARCH EXPERIENCES THROUGH CHALLENGE-BASED METHODOLOGIES." In 13th annual International Conference of Education, Research and Innovation. IATED, 2020. http://dx.doi.org/10.21125/iceri.2020.1045.

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Reports on the topic "Virology"

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van der Poel, W. H. M. One health, one virology. Wageningen: Wageningen University & Research, 2019. http://dx.doi.org/10.18174/478362.

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Sergeev, Alexander, Nikolay Litusov, Ekaterina Voroshilina, Alexey Kozlov, Yulia Grigoryeva, Danila Zornikov, Vasily Petrov, et al. Electronic educational resource Microbiology, virology and immunology. SIB-Expertise, January 2024. http://dx.doi.org/10.12731/er0769.29012024.

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"The electronic educational resource has been prepared as information support for the independent work of students, residents and graduate students mastering the main educational programs of higher professional education of the enlarged group of specialties Healthcare, developed on the basis of the Federal State Educational Standards of Higher Education and Professional Standards and providing for the formation of knowledge in microbiology and virology and immunology. The electronic educational resource provides information on the morphology, physiology, genetics, ecology of microorganisms, the basics of infectology and the epidemiology of infectious diseases. Each section is accompanied by control questions and practice tests. The manual contains extensive illustrative material that contributes to the assimilation of the issues under study. The electronic educational resource will help in the work of students, residents and graduate students both directly in practical classes and in preparation for classes, tests and exams."
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Sullivan, Matthew. Environmental Virology Workshop Summary, Tucson, Arizona, Jan 7-12, 2013. Office of Scientific and Technical Information (OSTI), February 2015. http://dx.doi.org/10.2172/1170243.

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Irit Davidson, Irit. The Knowledge that Human Tumor Virology can Gain from Studies on Avian Tumor Viruses. Libertas Academica, December 2009. http://dx.doi.org/10.4137/atv_1784.

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Wasi, Chantapong. Virus Diseases: The Global Challenge to Health for All. Asia-Pacific Congress of Medical Virology (2nd) Held in Bangkok, Thailand on November 17-22, 1991. Abstracts. Fort Belvoir, VA: Defense Technical Information Center, July 1992. http://dx.doi.org/10.21236/ada258158.

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Mawassi, Munir, and Valerian Dolja. Role of RNA Silencing Suppression in the Pathogenicity and Host Specificity of the Grapevine Virus A. United States Department of Agriculture, January 2010. http://dx.doi.org/10.32747/2010.7592114.bard.

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RNA silencing is a defense mechanism that functions against virus infection and involves sequence-specific degradation of viral RNA. Diverse RNA and DNA viruses of plants encode RNA silencing suppressors (RSSs), which, in addition to their role in viral counterdefense, were implicated in the efficient accumulation of viral RNAs, virus transport, pathogenesis, and determination of the virus host range. Despite rapidly growing understanding of the mechanisms of RNA silencing suppression, systematic analysis of the roles played by diverse RSSs in virus biology and pathology is yet to be completed. Our research was aimed at conducting such analysis for two grapevine viruses, Grapevine virus A (GVA) and Grapevine leafroll-associated virus-2 (GLRaV- 2). Our major achievements on the previous cycle of BARD funding are as follows. 1. GVA and GLRaV-2 were engineered into efficient gene expression and silencing vectors for grapevine. The efficient techniques for grapevine infection resulting in systemic expression or silencing of the recombinant genes were developed. Therefore, GVA and GLRaV-2 were rendered into powerful tools of grapevine virology and functional genomics. 2. The GVA and GLRaV-2 RSSs, p10 and p24, respectively, were identified, and their roles in viral pathogenesis were determined. In particular, we found that p10 functions in suppression and pathogenesis are genetically separable. 3. We revealed that p10 is a self-interactive protein that is targeted to the nucleus. In contrast, p24 mechanism involves binding small interfering RNAs in the cytoplasm. We have also demonstrated that p10 is relatively weak, whereas p24 is extremely strong enhancer of the viral agroinfection. 4. We found that, in addition to the dedicated RSSs, GVA and GLRaV-2 counterdefenses involve ORF1 product and leader proteases, respectively. 5. We have teamed up with Dr. Koonin and Dr. Falnes groups to study the evolution and function of the AlkB domain presents in GVA and many other plant viruses. It was demonstrated that viral AlkBs are RNA-specific demethylases thus providing critical support for the biological relevance of the novel process of AlkB-mediated RNA repair.
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Comunicación de las Ciencias, Centro. Fake news: Grupo de virología UA responde. Universidad Autónoma de Chile, March 2020. http://dx.doi.org/10.32457/2050012728/9604202038.

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Desde que la epidemia del coronavirus empezó a expandirse por el mundo, han sido múltiples las noticias de cómo frenar la pandemia, qué hacer para no contagiarse, cómo actuar en definitiva ante el Covid-19. Pero igual que ha habido muchas recomendaciones y la mayoría de gran valor y ayuda para todos, también han circulado muchos fake news que no han hecho más que confundir a la población. El grupo de virología de la Universidad Autónoma de Chile repasa algunas de estas falsas noticias para frenar su difusión y entregar información que permita tomar decisiones con evidencia científica.
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Usme Ciro, José Aldemar, María Cristina Navas Navas, Katherine Laiton Donato, and Daniel Bernardo Ramírez Osorio. X Simposio Colombiano y VI Congreso Latinoamericano de Virología. Ediciones Universidad Cooperativa de Colombia, November 2023. http://dx.doi.org/10.16925/eccr.08.

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El X Simposio Colombiano y VI Congreso Latinoamericano de Virología es el evento insignia de la Asociación Colombiana de Virología (ACV). En esta edición, el evento es liderado por la Universidad Cooperativa de Colombia, en asocio con el Instituto Nacional de Salud, el Centro Internacional de Agricultura Tropical (CIAT) y la ACV. Importantes empresas del sector se han vinculado como patrocinadores y diversas entidades públicas y privadas a nivel nacional e internacional también han brindado su apoyo para garantizar el máximo nivel de la agenda científica. El presente compendio incluye los resúmenes de 17 conferencias magistrales y centrales y 139 resúmenes de trabajos libres presentados en modalidades oral y cartel. Los trabajos libres han sido organizados por áreas temáticas (la mayoría transversales), para facilitar la integración entre investigadores de diferentes disciplinas y modelos de estudio. Los resúmenes compilados reflejan el alto nivel de la investigación en virología en Colombia y en Latinoamérica. La investigación en este campo del conocimiento continúa creciendo y su importancia en la toma de decisiones trascendentales ha sido ratificada durante los recientes retos en la salud pública global, donde el conocimiento básico aplicado a la integración de desarrollos tecnológicos con nuevas estrategias 0de análisis se convierte en una herramienta sin precedentes para el seguimiento, en tiempo real, de las infecciones virales y la implementación de estrategias efectivas para su control.
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Comunicación de las Ciencias, Centro. Pandemia de COVID-19: Lecciones desde España. Universidad Autónoma de Chile, March 2020. http://dx.doi.org/10.32457/2050012728/9607202041.

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Tanto Chile como España están enfrentando junto con la Pandemia de COVID-19, una “infodemia”, término utilizado por la Organización Mundial de la Salud (OMS) para referirse a la sobreabundancia de información falsa y su rápida propagación. En medios españoles el neurovirólogo José Antonio López Guerrero (JAL), divulgador y Profesor Titular del Departamento de Biología Molecular de la Universidad Autónoma de Madrid (UAM), es un invitado frecuente para aclarar las dudas de la ciudadanía y combatir las “Fakes News”, porque además de investigar antivirales contra coronavirus en su laboratorio, es uno de los responsables de comunicación de la Sociedad Española de Virología. En entrevista con el Centro de Comunicación de las Ciencias, el científico español nos aclaró algunas dudas respecto al nuevo coronavirus o como él le llama “el pequeño jinete del apocalipsis”.
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Microbial Evolution: This report is based on a colloquium convened by the American Academy of Microbiology on August 28-30, 2009, in San Cristobal, Ecuador. American Society for Microbiology, August 2011. http://dx.doi.org/10.1128/aamcol.28aug.2009.

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The year 2009 marked both the 200th anniversary of Darwin's birth and the 150th anniversary of the publication of his landmark book, On the Origin of Species. In August 2009, to celebrate these milestones, the American Academy of Microbiology convened a colloquium in the Galapagos Islands, where Darwin made some of his most crucial observations, to consider a new question: what would Darwin have made of the microbial world? The ability to sail to remote sites like the Galapagos, and access to specimens collected by himself and other avid naturalists, gave Darwin the information he needed to develop a conceptual framework for understanding life's visible diversity. Today, new discoveries and technical capabilities in microbiology are providing information that for the first time makes it possible to develop a conceptual framework for deepening our understanding of the diversity of the microbial world. Darwin focused his attention on visible life forms, which actually make up only a small fraction of the living world—the invisible world of microorganisms was as yet largely unexplored in his time. Yet Darwin's theory has proven remarkably robust; despite some fundamental differences between microorganisms and the rest of the living world, the two lynchpins of Darwin's theory—descent with modification and natural selection—have proven as powerful in explaining microbial evolution as they have in explaining macrobial evolution. Since Darwin, the advent of Mendelian Genetics and the Modern Synthesis have provided a wealth of new tools to evolutionists; these tools are also of fundamental importance in the modern study of microbiology. The scientists gathered at the colloquium considered two fundamental questions: ▪ Is the balance of evolutionary mechanisms, for example natural selection or drift, or individual and group selection, consistent among microbes and similar between microbes and macrobes? ▪ How are the mode and tempo of microbial evolution influenced by Earth's diversity of environments, and the changing global environment, and how are microbes themselves driving these changes? The colloquium provided an opportunity for individuals with expertise in evolutionary biology, genetic engineering, mycology, virology, microbial ecology, and other fields to discuss these issues and review the areas in which research is needed to fill gaps in our understanding.
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