Books: 'Viral antigen'

1

1929-, Laver William Graeme, Air Gillian, and Cold Spring Harbor Laboratory, eds. Immune recognition of protein antigens. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1985.

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2

Pollwein, Peter. Spezifische Bindungsstellen von SV40 T-Antigen im zellulären Mausgenom. Konstanz: Hartung-Gorre, 1987.

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3

1941-, Thomas D. Brian, ed. Viruses and the cellular immune response. New York: Marcel Dekker, 1993.

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4

Kyuhei, Tomonari, ed. Viral superantigens. Boca Raton: CRC Press, 1997.

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5

Kang, Chʻang-yul. Pairŏsŭ pektʻŏ ro hyŏngjil toiptoen hangwŏn chesi sepʻo ŭi myŏnyŏk chʻiryoje yuhyosŏng pʻyŏngka mit sihŏmpŏp yŏnʼgu =: Development and estimation of immunotherapeutic cell-based vaccine approaches using antigen presenting cells transduced with viral vector. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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6

R, Burton Dennis, ed. Antibodies in viral infection. Berlin: Springer, 2001.

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7

International Symposium on the Immunobiology of Proteins and Peptides (3rd 1984 Tahoe City, Calif.). Immunobiology of proteins and peptides III: Viral and bacterial antigens. New York: Plenum Press, 1985.

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8

V, Quinnan Gerald, ed. Vaccinia viruses as vectors for vaccine antigens: Proceedings of the Workshop on Vaccinia Viruses as Vectors for Vaccine Antigens, held November 13-14, 1984, in Chevy Chase, Maryland, U.S.A. New York: Elsevier, 1985.

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9

1937-, Cruse Julius M., and Lewis R. E. 1947-, eds. Antigenic variation: Molecular and genetic mechanisms of relapsing disease. Basel: Karger, 1987.

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10

T, Huber Brigitte, and Palmer Ed 1952-, eds. Superantigens: A pathogen's view of the immune system. Plainview, N.Y: Cold Spring Harbor Laboratory Press, 1993.

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11

Van Regenmortel, M. H. V. and Neurath A. Robert, eds. Immunochemistry of viruses II: The basis for serodiagnosis and vaccines. Amsterdam: Elsevier, 1990.

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12

G, Craig Alister, and Scherf Artur, eds. Antigenic variation. London: Academic, 2003.

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13

Edouard, Kurstak, ed. New vaccines and chemotherapy. New York: Plenum Medical Book Co., 1988.

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14

Gruss, Claudia. Bindung des T-Antigens aus einer SV40 transformierten Zellinie an virale und zelluläre DNA-Sequenzen. Konstanz: Hartung-Gorre, 1988.

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15

G, Giraldo, Campania (Italy), and Istituto dei tumori di Napoli., eds. From oncogenes to tumor antigens: Proceedings of the First International Symposium on Recent Advances in Tumor Immunology. Amsterdam: Elsevier Science Publishers, 1985.

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16

Grant, McFadden, ed. Viroceptors, virokines and related immune modulators encoded by DNA viruses. Austin: R.G. Landes, 1995.

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17

Workshop on Mechanisms and Specificity of HIV Entry into Host Cells (1989 San Francisco, Calif.). Mechanisms and specificity of HIV entry into host cells. New York: Plenum Press, 1991.

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18

Rud, Debrah Shurman. Indentification of FAM111A as a novel simian virus 40 large T antigen binding protein that affects viral host range. 2009.

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19

Greenlee, John E. Cerebrospinal Fluid. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0145.

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Evaluation of cerebrospinal fluid is of essential importance in the diagnosis of central nervous system infections, with a major role in the diagnosis of meningitis and encephalitis. In bacterial and viral meningitis, CSF evaluation is usually straightforward, and well-defined serological and molecular techniques are available for CSF examination in many of the more common viral encephalitides. This is in contrast to CSF evaluation in cases of tuberculous and chronic meningitis, in which organisms may be difficult to detect by culture or polymerase chain methods or antigen detection. This chapter first discusses the anatomy and physiology of CSF production, reviews concepts of CSF analysis, and then reviews CSF changes in the major categories of central nervous system infections.

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20

Alexander, D. J., N. Phin, and M. Zuckerman. Influenza. Edited by I. H. Brown. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0037.

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Influenza is a highly infectious, acute illness which has affected humans and animals since ancient times. Influenza viruses form the Orthomyxoviridae family and are grouped into types A, B, and C on the basis of the antigenic nature of the internal nucleocapsid or the matrix protein. Infl uenza A viruses infect a large variety of animal species, including humans, pigs, horses, sea mammals, and birds, occasionally producing devastating pandemics in humans, such as in 1918 when it has been estimated that between 50–100 million deaths occurred worldwide.There are two important viral surface glycoproteins, the haemagglutinin (HA) and neuraminidase (NA). The HA binds to sialic acid receptors on the membrane of host cells and is the primary antigen against which a host’s antibody response is targeted. The NA cleaves the sialic acid bond attaching new viral particles to the cell membrane of host cells allowing their release. The NA is also the target of the neuraminidase inhibitor class of antiviral agents that include oseltamivir and zanamivir and newer agents such as peramivir. Both these glycoproteins are important antigens for inducing protective immunity in the host and therefore show the greatest variation.Influenza A viruses are classified into 16 antigenically distinct HA (H1–16) and 9 NA subtypes (N1–9). Although viruses of relatively few subtype combinations have been isolated from mammalian species, all subtypes, in most combinations, have been isolated from birds. Each virus possesses one HA and one NA subtype.Last century, the sudden emergence of antigenically different strains in humans, termed antigenic shift, occurred on three occasions, 1918 (H1N1), 1957 (H2N2) and 1968 (H3N2), resulting in pandemics. The frequent epidemics that occur between the pandemics are as a result of gradual antigenic change in the prevalent virus, termed antigenic drift. Epidemics throughout the world occur in the human population due to infection with influenza A viruses, such as H1N1 and H3N2 subtypes, or with influenza B virus. Phylogenetic studies have led to the suggestion that aquatic birds that show no signs of disease could be the source of many influenza A viruses in other species. The 1918 H1N1 pandemic strain is thought to have arisen as a result of spontaneous mutations within an avian H1N1 virus. However, most pandemic strains, such as the 1957 H2N2, 1968 H3N2 and 2009 pandemic H1N1, are considered to have emerged by genetic re-assortment of the segmented RNA genome of the virus, with the avian and human influenza A viruses infecting the same host.Influenza viruses do not pass readily between humans and birds but transmission between humans and other animals has been demonstrated. This has led to the suggestion that the proposed reassortment of human and avian influenza viruses takes place in an intermediate animal with subsequent infection of the human population. Pigs have been considered the leading contender for the role of intermediary because they may serve as hosts for productive infections of both avian and human viruses, and there is good evidence that they have been involved in interspecies transmission of influenza viruses; particularly the spread of H1N1 viruses to humans. Apart from public health measures related to the rapid identification of cases and isolation. The main control measures for influenza virus infections in human populations involves immunization and antiviral prophylaxis or treatment.

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21

Immunobiology Prot & Pept. Plenum Press, 1985.

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22

Smith, Rebecca. Smallpox. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0063.

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Symptoms of the smallpox virus include fever and a progressive papular rash that becomes vesicular and then pustular. A systemic inflammatory response syndrome (SIRS) leads to septic shock and death in 30% of cases. The definitive diagnosis can be confirmed via blood samples, lesion contents, or scrapings from crusts analyzed using electron microscopy, viral antigen immunohistochemistry, or polymerase chain reaction. The suspicion of a single smallpox case should lead to immediate notification of local public health authorities and the hospital epidemiologist. Because the disease does not exist in nature, smallpox should be considered the result of a bioterrorist attack until proven otherwise. An epidemiologic investigation is essential for determining the perimeter of the initial release so that tracking and quarantine of those exposed can be completed. Patients are extremely contagious and must be placed on contact, droplet, and airborne precautions in a negative pressure room.

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23

Burton, Dennis R. Antibodies in Viral Infection. Springer Berlin / Heidelberg, 2010.

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24

(Editor), Madeleine W. Cunningham, and Robert S. Fujinami (Editor), eds. Molecular Mimicry, Microbes, and Autoimmunity. ASM Press, 2000.

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25

Atassi, M. Z. Immunobiology of Proteins and Peptides-III: Viral and Bacterial Antigens. Springer London, Limited, 2012.

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26

Atassi, M. Z. Immunobiology of Proteins and Peptides-III: Viral and Bacterial Antigens. Springer, 2012.

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27

Biological Significance of Superantigens (Chemical Immunology). S. Karger AG (Switzerland), 1992.

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28

Vaccines '85: Molecular and Chemical Basis of Resistance to Parasitic, Bacterial, and Viral Diseases (Vaccines). Cold Spring Harbor Laboratory Pr, 1985.

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29

The Europa world year book. 3rd ed. London: Europa, 1998.

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30

The Europa World Year Book 1994 (The Europa World Year Book). 3rd ed. Europa Pubns, 1994.

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31

Lai, Kar Neng, and Sydney C. W. Tang. Immunoglobulin A nephropathy. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0069_update_001.

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A working hypothesis is that patients with immunoglobulin A (IgA) nephropathy have inherited defects in B cells producing galactose-deficient polymeric IgA1. Additional cofactors are required to form immune complexes and their deposition in glomeruli. Molecular characterization of IgG autoantibodies that recognize abnormally underglycosylated IgA1 reveals a specific amino acid substitution in the variable region of the IgG1 heavy chain. This substitution greatly enhances IgG1 binding to the galactose-deficient IgA1. The triggering antigens may include viral or bacterial antigens, or possibly by ingested food epitopes. Antiglycan IgG1 antibodies are one of the additional risk factors, or a second/multiple hit, which predisposes to disease development.

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32

Kurstak, Edouard, R. G. Marusyk, and F. A. Murphy. New Vaccines and Chemotherapy. Springer, 2013.

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33

Kurstak, Edouard, M. H. V. Van Regenmortel, R. G. Marusyk, and F. A. Murphy. New Vaccines and Chemotherapy. Springer, 2013.

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34

Kurstak, Edouard, M. H. V. Van Regenmortel, R. G. Marusyk, and F. A. Murphy. New Vaccines and Chemotherapy. Springer London, Limited, 2013.

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35

(Editor), Edouard Kurstak, R. G. Marusyk (Editor), F. A. Murphy (Editor), and M.H.V. Van Regenmortel (Editor), eds. Applied Virology Research: New Vaccines and Chemotherapy: Volume 1 (Annals of Theoretical Psychology). Springer, 1988.

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36

Mesquita, Emersom C., and Fernando A. Bozza. Diagnosis and management of viral haemorrhagic fevers in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0293.

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In a globalized scenario where widespread international travel allows viral agents to migrate from endemic to non-endemic areas, health care providers and critical care specialists must be able to readily recognize a suspected case of viral haemorrhagic fever (VHF). Early suspicion is pivotal for improving patient outcome and to ensure that appropriate biosafety measures be applied. VHFs are acute febrile illnesses marked by coagulation disorders and organ specific syndromes. VHFs represent a great medical challenge because diseases are associated with a high mortality rate and many VHFs have the potential for person-to-person transmission (Filoviruses, Arenavioruses, and Bunyaviroses). Dengue is the most frequent haemorrhagic viral disease and re-emergent infection in the world and, due to its public health relevance, severe dengue will receive special attention in this chapter. The diagnosis of VHFs is made by detecting specific antibodies, viral antigens (ELISA) and viral nucleic acid (RT-PCR) on blood samples. Supportive care is the cornerstone in the treatment of VHFs. Ribavirin should be started as soon as a case of VHF is suspected and discontinued if a diagnosis of Filovirus or Flavivirus infection is established. Adjunctive antimicrobial therapy is usually implemented to treat co-existing or secondary infections. Antimalarial treatment should also be initiated if a malaria test (thick blood films) is not quickly available and/or reliable and patients travel history is compatible. It is always recommended to apply appropriate biosafety measures and notify local infection control unit and state and national authorities.

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37

Petersen, Ruby. Antigenic cross-reactivity between caprine arthritis-encephalitis, visna and progressive pneumonia viruses involves all virion-associated proteins and glycoproteins. 1985.

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38

Nucleic Acid Sensors and Antiviral Immunity. Taylor & Francis Group, 2012.

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39

Virally infected cells. New York: Plenum, 1989.

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40

Virally Infected Cells (Subcellular Biochemistry). Springer, 1989.

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41

(Editor), G. De-The, W. Henle (Editor), and F. Rapp (Editor), eds. Oncogenesis and Herpesviruses III: Part I DNA of herpesviruses, viral antigens, cell-virus interaction (International Agency for Research on Cancer). IARC Scientific Publications, 1986.

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42

Düzgünes, Nejat. Mechanisms and Specificity of HIV Entry into Host Cells. Springer London, Limited, 2012.

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43

Mechanisms and Specificity of HIV Entry into Host Cells. Springer, 1991.

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44

Düzgünes, Nejat. Mechanisms and Specificity of HIV Entry into Host Cells. Springer, 2012.

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45

Sole, Richard, and Santiago F. Elena. Viruses as Complex Adaptive Systems. Princeton University Press, 2018. http://dx.doi.org/10.23943/princeton/9780691158846.001.0001.

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Viruses are everywhere, infecting all sorts of living organisms, from the tiniest bacteria to the largest mammals. Many are harmful parasites, but viruses also play a major role as drivers of our evolution as a species and are essential regulators of the composition and complexity of ecosystems on a global scale. This book draws on complex systems theory to provide a fresh look at viral origins, populations, and evolution, and the coevolutionary dynamics of viruses and their hosts. New viruses continue to emerge that threaten people, crops, and farm animals. Viruses constantly evade our immune systems, and antiviral therapies and vaccination campaigns can be powerless against them. These unique characteristics of virus biology are a consequence of their tremendous evolutionary potential, which enables viruses to quickly adapt to any environmental challenge. This book presents a unified framework for understanding viruses as complex adaptive systems. It shows how the application of complex systems theory to viral dynamics has provided new insights into the development of AIDS in patients infected with HIV-1, the emergence of new antigenic variants of the influenza A virus, and other cutting-edge advances. The book also extends the analogy of viruses to the evolution of other replicators such as computer viruses, cancer, and languages.

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46

Alexander, Kevin. Myocarditis and Pericarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0019.

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Infectious myocarditis is a primary, inflammatory cardiomyopathy that can lead to cardiomyocyte toxicity via direct myocyte invasion, toxin production, and/or stimulation of a chronic inflammatory response through antigenic mimicry. Its incidence is difficult to determine due to significant disease heterogeneity and the lack of a noninvasive gold standard for diagnosis. Often, the causative pathogen is not identified; in cases where it is, appropriate anti-infective agents may be used. Treatment is primarily supportive. Acute infectious pericarditis involves inflammation of the parietal and visceral layers of the pericardial sac that surround the heart. Because infectious pericarditis usually has a viral etiology, antibiotics are only started if blood or pericardial effusion cultures demonstrate a bacterial or fungal cause. Purulent pericarditis and cardiac tamponade should be treated with drainage via either pericardiocentesis or a pericardiotomy. Pericardial resection is the only treatment for constrictive pericarditis.

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47

Muche, Marion, and Seema Baid-Agrawal. Hepatitis B. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0185_update_001.

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Hepatitis B virus (HBV) has been causally linked to a variety of renal diseases, the most common being glomerular diseases and systemic autoimmune disease. Membranous nephropathy (MN) is the commonest HBV-associated glomerulonephritis (HBV-GN), followed by membranoproliferative glomerulonephritis (MPGN), mesangial proliferative glomerulonephritis, immunoglobulin (Ig)-A nephropathy, and focal segmental glomerulosclerosis (FSGS). Polyarteritis nodosa is a rare manifestation. The incidence of HBV-associated renal diseases seems to be decreasing with the introduction of vaccination programmes.HBV-MN is the most frequent cause of nephrotic syndrome in children in countries with high endemicity of HBV infection. The clinical course is usually benign in children with high rates of spontaneous remission rates and low risk of progression to renal failure. The prognosis is worse in adults. Of the systemic autoimmune disorders associated with HBV infection that involve the kidneys, the strongest link has been found with polyarteritis nodosa (PAN), a lesion that causes arteritis of medium-sized renal vessels. HBV-associated PAN (HBV-PAN) usually manifests in the first year after infection, and is clinically indistinguishable from classic PAN.Diagnosis of HBV-GN or -PAN is based on the clinical picture, histological findings, evidence of viral replication in serum and/or liver and detection of HBV antigens or DNA in the tissue. Besides deposition of immune complexes, other mechanisms such as virus-induced cytopathic damage have been proposed to explain the pathogenesis.HBV-GN and HBV-PAN appear to respond to antiviral treatment. Both show remission after HBeAg seroconversion. The available studies predominantly employed first-generation agents like interferon alpha and lamivudine, which showed suppression of viral replication and clinical remission of HBV-associated renal disease. Immunosuppressive therapy appears to be inevitable for the control of severe HBV-PAN and could be helpful in addition to antiviral treatment for cases of HBV-GN not responding clinically to antiviral treatment.

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48

Götz, Christopher M., and Kitty F. Emery, eds. La Arqueologia de los Animales de Mesoamerica. Lockwood Press, 2014. http://dx.doi.org/10.5913/2014123.

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El reconocimiento del papel de los animales en las antiguas dietas, en las economias, politicas y los rituales, es vital para poder entender a las culturas del pasado en su totalidad. Por el otro lado, seguir las claves que se obtienen de restos de animales preteritos puede aproximarnos a entender la antigua relacion que existia entre los humanos y el mundo que les rodeaba. En respuesta a un creciente interes en el campo de la zooarqueologia, este libro presenta investigaciones que representan a las multiples culturas y regiones de Mesoamerica, tratando especificamente los aspectos mas recurrentes en la literatura zooarqueologica. Desde el punto de vista geografico, los ensayos reunidos aqui informan acerca del uso de animals por parte de los pueblos indigenas de toda el area mesoamericana, ubicada entre los confines nortenos de Mexico y la frontera sur, en Centroamerica. Esto incluye culturas tan diversas como los olmecas, mayas, mixtecos, zapotecos e indigenas de Centroamerica. El marco temporal del libro se extiende desde el Preclasico y Clasico, sobre el Posclasico, los tiempos coloniales e historicos, hasta la epoca actual. Los capitulos del libro, escritos por expertos en la materia de la zooarqueologia mesoamericana, proporcionan un fondo de conocimiento general e importante acerca del uso domestico y ritual durante los tiempos tempranos y clasicos de Mesoamerica y Centroamerica, pero abarcan tambien aspectos especificos de la relacion entre humanos y animales, tales como la domesticacion temprana y el simbolismo de animales, asi como otros puntos aun pobremente entendidos, relacionados a la tafonomia y a la metodologia zooarqueologica.

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49

Sepúlveda, Jovanny. Derecho, filosofía y sociedad. CUA - Medellin, 2020. http://dx.doi.org/10.52441/der202002.

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Cuando recordamos la definición aristotélica del hombre, que lo señala como zoon politikón, nos viene a la mente la concepción del hombre como un ser que es capaz de estar con otros en medio de una sociedad diversa (polis) con derechos y deberes. Tanto para Platón como para el Estagirita, las relaciones de los hombres en sociedad deben estar regidas por principios para que el bien común se preserve y la vida de todos sea llevadera, además de considerar que se requiere de un gobierno que los administre. Platón consideraba que la mejor forma de gobierno sería la proveniente del gobernante sabio y el Estagirita pensaba que solo los mejores hombres deberían ser quienes gobernasen. Que Platón propusiera el gobierno del sabio era una clara evidencia de la sospecha de vicios que surgirían en otro sistema del que seríamos herederos: la democracia. Ahora bien, no se trata aquí de la concepción de sabiduría depositada en la seguridad de un acervo de información adquirido por erudición y estudio académico. En Aristóteles, heredado de su maestro Platón, el sentido de la política estará vinculado a la idea de la virtud. Y si entendemos la virtud como la tendencia a obrar el bien (en tanto que disposición voluntaria adquirida para lograr el punto medio entre dos extremos viciosos), descubrimos una relación inextricable entre el gobierno de la polis y el actuar con los demás. Es bien interesante que proponga que es el hombre virtuoso el que gobierne y ejerza el derecho porque, en definitiva, la imparcialidad, la justicia y la verdad preceden al hombre que obra con medida y prudencia (phrónesis). Conviene recordar que el Estagirita clasifica las virtudes en dos grupos: las dianoéticas (que perfeccionan el intelecto: la sabiduría y la prudencia) y las éticas1 (que perfeccionan la voluntad). No debemos dejar de lado, después de anunciar esta estrecha relación entre ética y política presente en el pensamiento griego antiguo, que para Aristóteles el fin del hombre es la felicidad. Suena raro, en un ambiente laboral, económico, social y político como en el que vivimos hoy que se nos proponga que el único fin valedero de nuestras vidas sea lograr la felicidad. Aunque la traducción no es del todo justa, una ética de la eudaimonía implica superar los esquemas que nos proponen que se vive para triunfar y adquirir fama, para enriquecernos o experimentar todos los placeres posibles… Si hay algo de lo que nos damos cuenta constantemente, sobre todo desde la experiencia de limitación y de precariedad del hombre, es que nos gastamos una buena parte de nuestras vidas en cosas que no nos llevan a vivir bien. Tenemos y hacemos muchas cosas innecesarias, cosas que tornan cosas a los que deberían ser más importantes y vitales en nuestra experiencia vital (vida política y vida ética cotidiana). Pero fue la Modernidad, de la mano del pensamiento de Emmanuel Kant −en gran medida−, la que prefiguró el divorcio de esta estrecha relación. Así, el deber por el deber hizo surgir una escisión entre la ética y la política, lo que ha significado la aparición de interpretaciones acomodadas de asuntos tan importantes como la ley, los principios, los derechos, por solo mencionar algunos. Los efectos de una hermenéutica acomodada e interesada han provocado vicios en la ley que son delatados por los juristas y filósofos del derecho que reconocen la necesidad de develar la mentira que se oculta tras verdades a medias. Urge hoy una reflexión de este carácter, que nos recuerde el derecho y que actualice su vigencia para una sociedad que requiere justicia y verdad.

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50

Martinho, Neudson Johnson, Carolina Carbonell Demori, and João Vitor Andrade. Ciências da saúde: aprendizados, ensino e pesquisa no cenário contemporâneo. Editora Amplla, 2021. http://dx.doi.org/10.51859/amplla.csa511.1121-0.

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Novos desafios surgem a cada dia na área de saúde, os quais vem demonstrando que somente a partir da confluência de saberes e fazeres das diversas áreas do conhecimento os sistemas de saúde estarão fortalecidos para o enfrentamento deles, tendo em vista a complexidade das ações que se fazem necessárias em todos os níveis de atenção: primário, secundário e terciário. As rápidas mudanças epidemiológicas e demográficas, concomitante ao surgimento de pandemias e recrudescimento de doenças transmissíveis e não transmissíveis no mundo, evocam novos olhares e fazeres que requerem transformações desde a formação dos profissionais, produção de conhecimentos com vistas à processos de trabalhos colaborativos nos serviços de saúde, nos quais ocorram uma compreensão quanto a complementariedade dos diversos saberes e fazeres que compõem a equipe de saúde, desconstruindo a cultura das ações sobrepostas ou, a falácia que algumas profissões são mais importantes e portanto, hegemônicas frente as outras. O mundo é complexo e dinâmico, logo, os seres humanos que nele existem também o são em suas dimensões existenciais considerando que estão imbricados no mundo e, por esta característica fenomenológica, o saber / fazer de uma única profissão jamais será capaz de compreender e atender as complexas necessidades factuais destes seres em todo o seu ciclo vital, tendo em vista a imprevisibilidades e incertezas oriundas delas. Com base nas afirmações supracitadas e partindo do princípio de que o trabalho em saúde não tem um produto concreto final, sendo seu resultado consequência das relações profissionais e interpessoais que subjazem suas ações de cuidado e não apenas resultante do saber/fazer de um único profissional, é importante enfatizar que toda a centralidade do processo de trabalho em saúde deve ser o usuário e suas complexas necessidades, as quais só poderão ser realmente atendidas com um cuidado interprofissional / colaborativo. Portanto, mudanças no modus operandi e modus faciendi na formação dos profissionais de saúde e no processo de trabalho suscitam o desenvolvimento de novas competências e habilidades que devem permear a arte de ensinar, pesquisar e extensionar, reverberando no verdadeiro trabalho em equipe nos serviços de saúde, algo somente alcançável através da implementação de uma educação interprofissional nas universidades e cursos técnicos, assim como, a criação de políticas indutoras para o trabalho colaborativo, no qual os diversos saberes e fazeres profissionais sejam valorizados e reconhecidos quanto sua importante complementariedade, visando decisões terapêuticas compartilhadas no atendimento às necessidades de saúde no contexto mundial vigente frente a pandemia de COVID-19 e outras demandas biopsicossociais de cuidado humano. Nesta perspectiva, pensar em ciências da saúde é refletir sobre novos aprendizados, novas metodologias de ensino e novas formas de pesquisar em um cenário contemporâneo que desvela um mix de processos antigos que recrudescem e novos que surgem, apontando a urgência de novas tomadas de decisões, as quais sejam rápidas e eficazes em responder às complexas necessidades dos usuários do sistema de saúde, contribuindo para o resgate e manutenção da qualidade de vida. Desejamos a todos uma profícua leitura desta obra, a qual pela diversidade de temáticas abordadas, permitirá ao leitor não somente uma leitura do mundo sob o prisma dos autores ao socializarem os resultados alcançados em seus estudos, mas, sobretudo, poderá instigá-lo de certa forma à “reescrevê-lo”, no sentido de transformálo a partir da apreensão e ressignificação de conhecimentos contidos nas diferentes pesquisas que corporificam este e-book, permitindo um movimento de cruzamento e interdependência entre variadas competências.

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