Relevant bibliographies by topics / Viral

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Viral'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 August 2024

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Journal articles on the topic "Viral"

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US, A. Dürdal. "Viral Superantigens." Mikrobiyoloji Bulteni 50, no. 3 (July 29, 2016): 491–504. http://dx.doi.org/10.5578/mb.24250.

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Garcia-Iglesias, Jaime, Maurice Nagington, and Peter Aggleton. "Viral times, viral memories, viral questions." Culture, Health & Sexuality 23, no. 11 (October 19, 2021): 1465–69. http://dx.doi.org/10.1080/13691058.2021.1976564.

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Kaplún, Gabriel. "Viral y vital. Ciudadanía, educación y comunicación." Question/Cuestión 1, junio (June 6, 2020): e349. http://dx.doi.org/10.24215/16696581e349.

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Deiman, Birgit A. L. M., and Cornelis W. A. Pleij. "Pseudoknots: A Vital Feature in Viral RNA." Seminars in Virology 8, no. 3 (1997): 166–75. http://dx.doi.org/10.1006/smvy.1997.0119.

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Kara, İsmail Hamdi. "Acute viral hepatitis." Turkiye Aile Hekimligi Dergisi 12, no. 1 (2008): 39–43. http://dx.doi.org/10.2399/tahd.08.039.

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Rainetová, Petra. "Viral intestinal infections - viral gastroenteritides." Pediatrie pro praxi 18, no. 1 (April 4, 2017): 44–49. http://dx.doi.org/10.36290/ped.2017.009.

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McGill, Paul E. "Viral infections: α-viral arthropathy." Baillière's Clinical Rheumatology 9, no. 1 (February 1995): 145–50. http://dx.doi.org/10.1016/s0950-3579(05)80151-7.

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Fujinami, R., and T. Weber. "Viral neuroimmunology/Viral neuropathogenesis I." Journal of Neurovirology 8, no. 3 (January 2002): 16–17. http://dx.doi.org/10.1080/13550280290049840.

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Bauckhage, Christian, Fabian Hadiji, and Kristian Kersting. "How Viral Are Viral Videos?" Proceedings of the International AAAI Conference on Web and Social Media 9, no. 1 (August 3, 2021): 22–30. http://dx.doi.org/10.1609/icwsm.v9i1.14598.

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Within only a few years after the launch of video sharing platforms, viral videos have become a pervasive Internet phenomenon. Yet, notwithstanding growing scholarly interest, the suitability of the viral metaphor seems not to have been studied so far. In this paper, we therefore investigate the attention dynamics of viral videos from the point of view of mathematical epidemiology. We introduce a novel probabilistic model of the progression of infective diseases and use it to analyze time series of YouTube view counts and Google searches. Our results on a data set of almost 800 videos show that their attention dynamics are indeed well accounted for by our epidemic model. In particular, we find that the vast majority of videos considered in this study show very high infection rates.
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Kara, İsmail Hamdi. "Acute viral hepatitis C." Turkiye Aile Hekimligi Dergisi 12, no. 2 (2008): 89–94. http://dx.doi.org/10.2399/tahd.08.089.

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Dissertations / Theses on the topic "Viral"

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Ekberg, Johan, and Fredrik Isaksson. "Viral Marknadsföring." Thesis, Linköping University, Department of Management and Economics, 2000. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-705.

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Bakgrund: I massmedia har det på senare tid börjat uppmärksammas ett nytt fenomen inom internetbaserad marknadsföring som kallas för viral marknadsföring. Det har dock inte gjorts några studier om vad begreppet egentligen innebär.

Syfte: Att ge en innebörd åt begreppet viral marknadsföring och att undersöka de faktorer som kan påverka möjligheten att kunna utnyttja viral marknadsföring.

Avgränsningar: I denna studie har vi avgränsat oss genom att säga att teorierna kring viral marknadsföring endast går att applicera på Internet. I studien behandlas endast konsumentrelaterade produkter. Genomförande: Undersökningen har genomförts via studier av artiklar och litteratur och med en empirisk del som består av enkätundersökning, intervju samt data från ett fallföretag.

Resultat: Viral marknadsföring handlar om att låta utomstående personer marknadsföra det egna företaget via Internet. Spridning av viral marknadsföring kan ske aktivt eller passivt. I uppsatsen identifieras olika typer av viral marknadsföring beroende på vilken typ av produkt eller tjänst som marknadsförs. Avslutningsvis redovisas en modell som beskriver de faktorer som bör beaktas vid utformandet av en viral marknadsföringsstrategi, detta med avseende på produkten, kunden och marknaden.

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Azevedo, Lúcia Alexandra Fernandes. "Bronquiolite viral aguda." Master's thesis, Universidade da Beira Interior, 2009. http://hdl.handle.net/10400.6/889.

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Contextualização: A bronquiolite é a infecção das vias respiratórias inferiores mais comum em crianças com idade inferior a dois anos, sendo uma importante causa de internamento nos meses de Inverno. Resulta da infecção e inflamação da mucosa respiratória das vias aéreas distais por uma grande variedade de vírus sazonais, sendo o Vírus Sincicial Respiratório o agente etiológico mais frequentemente implicado. O diagnóstico é baseado na história clínica típica e no exame físico. O tratamento é essencialmente de suporte, contudo persiste o recurso a terapêuticas cuja prática não é suficientemente sustentada por evidência científica. Apesar de ser uma doença frequente existe pouco consenso acerca da melhor abordagem diagnóstica e terapêutica. Objectivos: O objectivo principal deste estudo foi analisar a orientação diagnóstica e terapêutica das crianças com bronquiolite. Como objectivo secundário foi determinada a frequência dos diversos agentes etiológicos virais isolados e analisada a gravidade da doença em função destes. Metodologia: Foi conduzido um estudo prospectivo descritivo em crianças com bronquiolite, com idade inferior a 24 meses, que recorreram ao Serviço de Urgência Pediátrica do Centro Hospitalar da Cova da Beira, entre 1 de Novembro de 2008 e 31 de Março de 2009. Procedeu-se ao registo de informações relativas aos dados demográficos, manifestações clínicas, exames complementares de diagnóstico solicitados e intervenções terapêuticas efectuadas. Resultados: Foram incluídas no estudo 78 crianças, 60% eram do sexo masculino e a média de idade foi de 8,5 meses. Cinquenta e três por cento das crianças necessitaram de internamento tendo sido a duração média deste de 7 dias. A prova terapêutica com salbutamol foi realizada a 59% das crianças, tendo sido continuado em 32,4% das tratadas no domicílio e em 56,1% das internadas. A antibioterapia sistémica foi prescrita em 19,2% das crianças. Durante o internamento 95% das crianças necessitaram de oxigénio suplementar, a aspiração de secreções foi realizada em 56%, e em 61% houve necessidade de hidratação endovenosa. A cinesioterapia respiratória foi realizada em 22% das crianças internadas. A radiografia do tórax foi realizada em 38,5%, o hemograma e PCR em 28,2% e a hemocultura em 17,9% das crianças. A pesquisa de vírus respiratórios foi positiva em 78,7%, tendo sido detectada infecção concomitante com dois vírus em 23% das crianças. O VSR foi identificado em 69,3% e o BoVh em 22,7%. Em 88% das crianças com amostras positivas para o BoVh foi detectada simultaneamente infecção com o VSR. As crianças com infecção concomitante com VSR e BoVh necessitaram mais frequentemente de internamento comparativamente às crianças com infecção simples por VSR (80% vs 60%). Conclusões: Tendo em consideração a evidência actual em relação à abordagem diagnóstica e terapêutica da bronquiolite detectaram-se dois aspectos passíveis de optimização que são a redução da administração de salbutamol e do recurso a exames complementares de diagnóstico. Os resultados do estudo confirmam o VSR como o principal agente etiológico da bronquiolite, e destacam o BoVh como um vírus também frequentemente associado a esta doença, demonstrando ainda uma elevada taxa co-detecção deste com o VSR. Neste estudo não foi possível associar a co-infecção com VRS e BoVh a uma maior gravidade da doença, comparativamente à infecção simples por VSR.
Backgroud: The bronchiolitis is the most common lower tract respiratory infection in children under two years and is a major cause of hospitalization during the winter months. It´s the result of infection and inflammation of the distal airways respiratory mucosa the by a variety of seasonal virus, and Respiratory Syncytial Virus is the etiologic agent most frequently implicated. The diagnosis is based on typical clinical history and physical examination. Treatment is essentially supportive however, therapeutics not sufficiently supported by scientific evidence remain being used. Despite being a common disease there is little consensus about the best diagnostic and therapeutic approach. Objectives: The main objective of this study was to analyze therapeutic and diagnostic approaches of children with bronchiolitis. The secondary objective was to determine the frequency of various viral pathogens isolated and analyze the severity of the disease according to these. Methodology: We conducted a prospective descriptive study in children with bronchiolitis, with the age below 24 months, which have been taken at Pediatric Emergency Service of Hospital of Cova da Beira, between 1 November 2008 and 31 March 2009. It has been registrated information related to demographics, clinical manifestations, additional diagnostic tests and therapeutic interventions. Results: We included 78 children in the study, 60% were male and mean age was 8.5 months. Fifty-three percent of children required hospitalization and the average duration of that was 7 days. Proof therapy with salbutamol was performed at 59% of children, and was continued in 32.4% of those treated at home and in 56.1% of those hospitalized. Systemic antibiotics were prescribed in 19.2% of children. Along hospitalization 95% of children required supplemental oxygen. Nasopharyngeal suction was performed in 56%, and 61% of the children needed intravenous hydration. Chest physiotherapy was performed in 22% of hospitalized children. Chest X-ray was performed in 38.5%, blood count and PCR in 28.2% and blood culture in 17.9% of children. In viral diagnostic testing at least one virus was detected in 78.7% and concomitant infection with two viruses was detected in 23% of children. The RSV was identified in 69.3% and BoVh in 22.7% of children. In 88% of children with positive samples for BoVh it has been detected simultaneously RSV infection. Children with concomitant infection with RSV and BoVh required more often hospitalization compared with children infected with RSV alone (80% vs 60%). Conclusions: Given the current evidence regarding the diagnostic approach and treatment of bronchiolitis there were found two issues that are likely to be optimized: the reduction of salbutamol prescription and the use of complementary tests of diagnosis. These results confirm RSV as the primary etiologic agent of bronchiolitis, and highlight the BoVh as a virus often associated with this disease, also showing a high rate of co-detection with RSV. Given the limitations of the study it wasn’t possible to associate the co-infection with RSV and BoVh with a greater severity of illness, compared to infection by RSV alone.
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Kymalainen, Hanna. "Development of viral & non-viral episomal vectors for gene therapy applications." Thesis, University of London, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589000.

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Gene therapy consists of methods which attempt to repair or replace defective genes responsible for disease, or to add genes to a therapeutic effect. To achieve this, two episomally maintained recombinant viral vectors have shown promising results: integration-deficient lentiviral vectors (IDLVs), and adeno-associated virus (AA V) vectors. The non-integrating nature of these vectors improves their safety profile but also limits transgene retention as nuclear episomes generally get lost during cell division. In the present study, the establishment of stable replicating episomes via transduction with AA V and IDL V gene therapy vectors was examined in CHO cells. Different DNA elements and cell culture conditions were evaluated, and in particular the effects of (i) DNA elements called S/MARs (scaffold/matrix attachment regions) which are involved in chromatin organisation, transcription and replication, and (ii) induction of transient cell cycle arrest in transfected and transduced cell populations. In the case of both AA V and IDL V vectors, the incorporation of S/MAR elements into vector transcription units had only marginal effects on the establishment of stable transgene- positive cell populations, either with or without induction oftransient cell cycle arrest. However, a striking general result was observed in cell populations transduced with IDL Vs and subjected to a transient cell cycle arrest soon after transduction. Under these conditions, following release from cell cycle arrest and in the absence of any selection pressure, substantial populations (10-25%) of proliferating and stably transduced cells emerged and were maintained over at least 100 population doublings. This establishment of stable transduction was seen only with IDLVs, was crucially . dependent on the induction of a period of transient cell cycle arrest, occurred independently of the presence of S/MAR elements, and resulted in transgene-positive cell populations which could be isolated and propagated as stable clonal cell lines. In these polyclonal and clonal IDL V -transduced cell lines, the existence of non-integrated vector genomes in the form of multi-copy nuclear episomes was confirmed by evidence from linear amplification -mediated PCR, deep sequencing, Southern blotting and FISH (fluorescent in situ hybridisation). 2 The cumulative evidence suggests that transduction of eHO cells with IDL Vs followed by a short period of induced cell cycle arrest leads to the establishment of stable IDL V- based nuclear episomes which are transcriptionally active and undergo replication and segregation during cell division without the need for antibiotic-based or other positive selection pressure. Preliminary investigations were also done to test the capacity of combined IDL V transduction and transient cell cycle arrest to establish stable episome Hel.a cells and murine haematopoietic stem cells. However, further experiments are required either to optimise the protocol in these cells or to find other clinically relevant cell types in which the protocol can be implemented. The transfer of this technology to a variety of clinically relevant human stem or progenitor cell populations could improve the safety profile of a range of gene therapy strategies currently under investigation. 3
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Paton, David James. "Bovine viral diarrhoea virus : studies of viral epitopes and of porcine infections." Thesis, University of Surrey, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317374.

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Ruoss, Sven. "Erfolgsfaktoren des Viral Marketing." St. Gallen, 2008. http://www.biblio.unisg.ch/org/biblio/edoc.nsf/wwwDisplayIdentifier/02600278002/$FILE/02600278002.pdf.

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Stirnemann, Sandra. "Verhaltenswirksamkeit des Viral Marketing." St. Gallen, 2008. http://www.biblio.unisg.ch/org/biblio/edoc.nsf/wwwDisplayIdentifier/02601128002/$FILE/02601128002.pdf.

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Gangadharan, Bevin. "Proteomics in viral disease." Thesis, University of Oxford, 2006. http://ora.ox.ac.uk/objects/uuid:c66c53ed-a824-4f99-8f2b-d2bc65a984c7.

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The separation, identification, and characterisation of the proteins present in a tissue or biological sample is called ‘proteomics’. This technique can be used for example to identify biomarkers and investigate signalling pathways. Increasingly, proteomics is being applied to the analysis of virus related samples; here two such examples are described. Presently there is no reliable non-invasive way of assessing liver fibrosis. Here a novel 2D-PAGE based proteomics study was used to identify potential fibrosis biomarkers. Serum from patients with varying degrees of hepatic scarring induced by infection with the hepatitis C virus (HCV) was analysed. Several proteins associated with liver scarring and/or viral infection were identified. The most prominent changes were observed when comparing serum samples from cirrhotic patients with healthy controls: Expression of inter-α-trypsin inhibitor heavy chain H4 fragments, α1 antichymotrypsin, apolipoprotein L1 (Apo L1), prealbumin and albumin was decreased in cirrhotic serum, whereas CD5 antigen like protein (CD5L) and β2 glycoprotein I (β2GPI) increased. In general, α2 macroglobulin (a2M) and immunoglobulin components increased with hepatic fibrosis whereas haptoglobin and complement components (C3, C4 and factor H-related protein 1) decreased. Novel proteins associated with HCV-induced fibrosis include the inter-alpha-trypsin inhibitor heavy chain H4 fragments, complement factor H-related protein 1, CD5L, Apo L1, β2GPI and the increase in thiolester cleaved products of a2M. The relationship between these changes is discussed. One of the accessory genes of the HIV viral genome encodes for the Nef protein. Nef is present in lipid rafts and increases viral replication within infected host cells by binding to a guanine nucleotide exchange factor, Vav. This leads to activation of a GTPase, Cdc42, however, the signalling pathway is poorly understood. 2D-PAGE based proteomics was used to identify differentially expressed raft-associated proteins by comparing T cells in the presence and absence of Nef. A ubiquitin conjugating enzyme UbcH7, which acts in conjugation with c-Cbl, was absent from the rafts of Nef-transfected cells. Vav ubiquitination was also absent from these rafts. In collaboration with Dr. Alison Simmons and Prof. Andrew McMichael the absence of UbcH7 in rafts was found to be caused by β-Pix forming a ternary complex with c-Cbl and activated Cdc42. Vav ubiquitination was restored and viral replication was diminished when β-Pix was knocked down providing a new candidate target for inhibiting HIV replication. This thesis demonstrates the use of proteomics in providing novel information for virus related samples. This influential technology benefits in both biomarker discovery to aid clinicians with early diagnosis of diseased individuals and in the elucidation of novel signalling pathways in infected cells to provide new candidate targets.
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Phillips, Angela Marie Ph D. Massachusetts Institute of Technology. "Chaperoning viral protein evolution." Thesis, Massachusetts Institute of Technology, 2018. http://hdl.handle.net/1721.1/118276.

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Thesis: Ph. D. in Biological Chemistry, Massachusetts Institute of Technology, Department of Chemistry, 2018.
Cataloged from PDF version of thesis. Vita.
Includes bibliographical references.
Preventing viral pandemics and developing effective antiviral therapeutics demands understanding the molecular mechanisms that both potentiate and constrain viral evolution. The rapid evolution of viruses is mediated in part by their high mutation rates, enabling resistance to antiviral drugs, seasonal vaccines, and innate and adaptive immune responses. Fortunately for us, the same mutations responsible for resistance are often biophysically deleterious to viral proteins. Thus, viral evolution is inherently constrained by the proper folding of viral proteins into functional, stable conformations. In cells, protein folding and homeostasis are assisted by complex networks of chaperones and quality control machinery. Though the evolutionary implications of most chaperones and quality control factors remain unexplored, the HSP90 chaperone can buffer and potentiate the phenotypic effects of mutations in endogenous client proteins in bacteria, fungi, plants, and other eukaryotic organisms. Viruses acquire mutations at a rate several orders of magnitude above that of the aforementioned organisms, yet they do not encode any machinery to assist destabilized protein variants to their folded, functional conformations. However, viral proteins are known to interact with host chaperones and quality control machinery. My graduate work has focused on determining whether and how host proteostasis machinery modulates viral protein evolution. First, I employed a serial passaging approach to evolve influenza in host cells with remodeled proteostasis capacities, revealing that cytosolic host proteostasis capacity is indeed a critical determinant of influenza evolutionary trajectories. This work motivated systematic quantification of influenza protein mutational tolerance upon perturbation of host proteostasis, for which I applied deep mutational scanning to comprehensively profile the mutational tolerance of influenza nucleoprotein and hemagglutinin in modulated cytosolic and endoplasmic reticulum (ER) folding environments, respectively. The nucleoprotein work provides the first experimental evidence that host chaperones can enhance the accessibility of biophysically deleterious, adaptive viral protein variants. The hemagglutinin work establishes evolutionary implications for the ER proteostasis machinery, and demonstrates that ER proteostasis mechanisms enhance mutational tolerance across the entire HA protein. Overall, it is clear that host chaperones and quality control machinery crucially impact viral protein fitness, and likely also impact the fitness of endogenous variants.
by Angela Marie Phillips.
Ph. D. in Biological Chemistry
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Larsson, Tom. "Kan jag bli viral?" Thesis, Malmö högskola, Fakulteten för teknik och samhälle (TS), 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-20056.

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Integrationen av sociala medier i dagens samhälle har bidragit till framväxten av en mersammankopplad och global värld. En värld som erbjuder fler tekniska möjligheter än tidigare, men som även bidrar med nya utmaningar. En av dessa utmaningar är hur man ska agera för att bli positivt utmärkt på den alltmer konkurrensutsatta arbetsmarknaden. Denna studie syftar till att angripa denna utmaning genom att undersöka hur den sociala applikationen Instagram kan användas som en kanal för personlig marknadsföring. Studiens slutsatser, vilka är baserade på en kombination av resultat från litteraturstudier samt kvalitativt inriktade intervjuer och innehållsanalyser, tyder på att Instagram kan användas som ett personligt marknadsföringsverktyg. Resultatet av studien indikerar att man bör börja med att klargöra sina unika och attraktiva egenskaper, mål och färdigheter för att skapa sig ett personligt varumärke.Detta varumärke kan sedan kommuniceras på Instagram genom skapandet och applicerande av en intresseveckande markandsföringsstrategi. För att höja chanserna att strategin ska lyckas bör den vara autentisk, tydlig, konsekvent och tilltalande för potentiella arbetsgivare. Resultaten tyder även på att det är viktigt att t.ex. använda hashtags och kommentarer för att skapa positiva interaktioner och uppmärksamhet kring ens varumärke/Instagramkonto. Slutligen bör man även utvärdera resultatet av ens ansträngningar för att se om ens mål blivit uppfyllda.
The integration of social media in today's society has largely contributed to the development of a more accessible and global world. Even if this evolution raises countless possibilities it also creates challenges, one of those is how to become positively distinguished in the increasingly competitive employment market. This study addresses this challenge by investigating how the new and popular social application Instagram can be used as a tool for personal branding. The findings, which are based on a combination of literature reviews, qualitatively oriented interviews and content analysis, concludes that Instagram can be used a personal branding tool. The recommended way to do this is by first clarify one's unique brandable attributes, values and goals by constructing a personal brand. This brand can then be communicated through Instagram by the use of a captivating strategy, which are evaluated and reviewed after some time. This in order to gradually develop the value offered so that the brand remains contemporary and competitive. To increase the chances of succeeding with the strategy, it should be authentic, clear, consistent and appealing for prospective employers. Findings also show that it is important to use e.g. hashtags and comments to create positive interactions and buzz around one's brand/Instagram account.
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Nishikawa, Hiroki. "Development of Novel Anti-viral Agents against Class I Viral Membrane Fusion Process." 京都大学 (Kyoto University), 2011. http://hdl.handle.net/2433/142504.

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Books on the topic "Viral"

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Pon̲n̲uccāmi, Mēlāṇmai Ce. Viral-- viral-- viral--: Cir̲ukatait tokuppu. Nākappaṭṭin̲am: Kumarip Patippakam, 1995.

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Lemon, Stanley M., Arie Jeremy Zuckerman, and H. C. Thomas. Viral hepatitis. 3rd ed. Malden, Mass: Blackwell Pub., 2005.

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Reichs, Kathy. Viral. Paris: Pocket, 2011.

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Avshalom, Mizrahi, ed. Viral vaccines. New York: Wiley-Liss, 1990.

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Samuels, Robert. Viral Rhetoric. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73895-2.

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Langner, Sascha. Viral Marketing. Wiesbaden: Gabler, 2009. http://dx.doi.org/10.1007/978-3-8349-8258-2.

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Kurstak, Edouard. Viral Hepatitis. Vienna: Springer Vienna, 1993. http://dx.doi.org/10.1007/978-3-7091-4437-4.

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Thomas, Howard, Stanley Lemon, and Arie Zuckerman, eds. Viral Hepatitis. Oxford, UK: Blackwell Publishing Ltd, 2005. http://dx.doi.org/10.1002/9780470987131.

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Callea, Francesco, Mario Zorzi, and Valeer J. Desmet, eds. Viral Hepatitis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71350-7.

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Langner, Sascha. Viral Marketing. Wiesbaden: Gabler Verlag, 2005. http://dx.doi.org/10.1007/978-3-8349-9078-5.

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Book chapters on the topic "Viral"

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Said, Adnan, and Aiman Ghufran. "Viral Hepatitis: Other Viral Hepatides." In Liver Disorders, 165–72. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-30103-7_12.

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Damrhung, Pornrat. "From Viral Hit to Vital Troupe." In Dance On!, 125–36. London: Routledge, 2023. http://dx.doi.org/10.4324/9781003307624-21.

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Samuels, Robert. "Viral Culture." In Viral Rhetoric, 43–59. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73895-2_4.

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García-Iglesias, Jaime, Maurice Nagington, and Peter Aggleton. "Viral times." In Viral Times, 1–9. London: Routledge, 2024. http://dx.doi.org/10.4324/9781003322788-1.

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Gibbs, Craig S. "Viral response to therapy: viral dynamics." In Hepatitis Prevention and Treatment, 157–73. Basel: Birkhäuser Basel, 2004. http://dx.doi.org/10.1007/978-3-0348-7903-3_9.

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Al-Shaalan, Mohammad. "Viral Exanthem." In Textbook of Clinical Pediatrics, 881–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_72.

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Weese, J. Scott, and Martha B. Fulford. "Viral Diseases." In Companion Animal Zoonoses, 241–74. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9780470958957.ch3.

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Davenport, Andrew, Todd W. Costantini, Raul Coimbra, Marc M. Sedwitz, A. Brent Eastman, David V. Feliciano, David V. Feliciano, et al. "Viral Meningitis." In Encyclopedia of Intensive Care Medicine, 2458. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_2422.

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Kopera, Daisy. "Viral Infections." In Laser and IPL Technology in Dermatology and Aesthetic Medicine, 285–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-03438-1_20.

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Nomura, Yasuya, Yasuya Nomura, and Yasuya Nomura. "Viral Diseases." In Morphological Aspects of Inner Ear Disease, 117–46. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54204-9_6.

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Conference papers on the topic "Viral"

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Wendell, Patrick, and Michael J. Freedman. "Going viral." In the 2011 ACM SIGCOMM conference. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/2068816.2068867.

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Bessi, Alessandro, Fabio Petroni, Michela Del Vicario, Fabiana Zollo, Aris Anagnostopoulos, Antonio Scala, Guido Caldarelli, and Walter Quattrociocchi. "Viral Misinformation." In WWW '15: 24th International World Wide Web Conference. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2740908.2745939.

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Hemsley, Jeff, and Sikana Tanupabrungsun. "Viral Design." In SMSociety '18: International Conference on Social Media and Society. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3217804.3217901.

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Berrigan, Caitlin. "Viral confections." In SIGGRAPH07: Special Interest Group on Computer Graphics and Interactive Techniques Conference. New York, NY, USA: ACM, 2007. http://dx.doi.org/10.1145/1280120.1280150.

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Hansen, Derek L., and Christianne Johnson. "Veiled viral marketing." In the 2nd ACM SIGHIT symposium. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2110363.2110393.

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Lakshmanan, Laks V. S. "Viral marketing 2.0." In SIGMOD/PODS'16: International Conference on Management of Data. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/2980523.2980526.

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Jiang, Lu, Yajie Miao, Yi Yang, Zhenzhong Lan, and Alexander G. Hauptmann. "Viral Video Style." In ICMR '14: International Conference on Multimedia Retrieval. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2578726.2578754.

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Sadeh, Gil, Lior Wolf, Tal Hassner, Nachum Dershowitz, and Daniel Stokl Ben-Ezra. "Viral transcript alignment." In 2015 13th International Conference on Document Analysis and Recognition (ICDAR). IEEE, 2015. http://dx.doi.org/10.1109/icdar.2015.7333854.

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Sulic, Susana. "Cyvers viral city." In SIGGRAPH07: Special Interest Group on Computer Graphics and Interactive Techniques Conference. New York, NY, USA: ACM, 2007. http://dx.doi.org/10.1145/1280120.1280250.

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Hoang, Tuan-Anh, and Ee-Peng Lim. "Retweeting: An Act of Viral Users, Susceptible Users, or Viral Topics?" In Proceedings of the 2013 SIAM International Conference on Data Mining. Philadelphia, PA: Society for Industrial and Applied Mathematics, 2013. http://dx.doi.org/10.1137/1.9781611972832.63.

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Reports on the topic "Viral"

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Edmundson, Scott, Michael Huesemann, Sherry Cady, Li-Jung Kuo, Brady Anderson, and Daman Reynolds. VITAL- Viral InhibiTors from ALgae: Generating Extracts for Antiviral Activity Assays. Office of Scientific and Technical Information (OSTI), October 2020. http://dx.doi.org/10.2172/1776864.

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Zhou, C., and J. Smith. Viral Metagenomics: MetaView Software. Office of Scientific and Technical Information (OSTI), October 2007. http://dx.doi.org/10.2172/923102.

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Tignor, Gregory H. Drug Development against Viral Diseases. Fort Belvoir, VA: Defense Technical Information Center, February 1987. http://dx.doi.org/10.21236/ada201949.

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Danny Colombara, Danny Colombara. Viral Causes of Lung Cancer. Experiment, December 2012. http://dx.doi.org/10.18258/0065.

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Papisov, Mikhail. Viral Oncolytic Therapeutics for Neoplastic Meningitis. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada609948.

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Walter Moss, Walter Moss. Can viral molecular structures cause cancer? Experiment, January 2014. http://dx.doi.org/10.18258/1929.

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Kuruppu, Kumudu D. Viral Oncolytic Therapeutics for Neoplastic Meningitis. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada566647.

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Spears, William M., Lora Billings, and Ira B. Schwartz. Modeling Viral Epidemiology in Connected Networks. Fort Belvoir, VA: Defense Technical Information Center, March 2001. http://dx.doi.org/10.21236/ada389278.

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Kuruppu, Kumudu D. Viral Oncolytic Therapeutics for Neoplastic Meningitis. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada592240.

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Papisov, Mikhail, and Betty Diamond. Viral Oncolytic Therapeutics for Neoplastic Meningitis. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada600901.

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