Relevant bibliographies by topics / Vestibular apparatus

Academic literature on the topic 'Vestibular apparatus'

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Published: 4 June 2021
Last updated: 1 August 2024

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Journal articles on the topic "Vestibular apparatus"

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Naryshkin, A. G., I. V. Galanin, A. L. Gorelik, R. Yu Seliverstov, and T. A. Skoromets. "Conceptual Aspects of Vestibular Neuromodulation." Физиология человека 49, no. 4 (July 1, 2023): 115–23. http://dx.doi.org/10.31857/s0131164623700297.

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The review highlights the development of the vestibular system in phylo- and ontogenesis, also its influence on the forming and mature brain. Based on recent studies, neuronal networks formed under the influence of the vestibular apparatus (VA) have been described. The basic function of the VA is gravitational sensitivity, which is detected by the otolithic apparatus of the vestibule. Because of this peculiarity of the vestibular apparatus, according to the authors, the main property of the vestibular apparatus is its dominant participation in multimodal synthetic processes. Different methods of vestibular neuromodulation (VNM) and its possibilities in the treatment of various brain diseases are considered. The authors believe that the “point of application” of VNM is its effect on the macular vestibular apparatus, which explains its effectiveness in various diseases of the brain.
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Efimova, V. L. "THE ROLE OF VESTIBULAR HEARING IN SPEECH PERCEPTION (REVIEW OF FOREIGN RESEARCH)." Educational Psychology in Polycultural Space 66, no. 2 (2024): 25–34. http://dx.doi.org/10.24888/2073-8439-2024-66-2-25-34.

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The article presents a review of foreign studies on the participation of the vestibular system in speech perception. Although for a long time it was believed that the vestibular system is involved only in the management of balance and motor skills, there is increasing evidence that it is involved in cognitive processes such as memory, attention, and speech. There is no doubt that auditory perception is necessary for speech perception. The peripheral parts of the vestibular and auditory analyzers are anatomically closely related. But there is not enough research on how vestibular organs affect auditory perception. Evolutionarily, the vestibular apparatus appeared in animals much earlier than the peripheral hearing organs. The human vestibular apparatus consists of five paired sections: three semicircular canals and two otolith organs – the utriculus and sacculus. In the process of evolution, the semicircular canals of humans and other mammals underwent maximum changes. The ability to register auditory information was preserved only in one of the parts of the vestibular apparatus – sacculus. Vestibular (saccular) hearing allows us to register low-frequency sounds in the range from 100 to 1000 Hz. This helps speech perception, as this frequency range is associated with the perception of intonation and other prosodic components of the utterance. Saccular hearing also helps speech perception in noise. Data on the role of the vestibular apparatus in speech perception are useful for all speech and language specialists. The ability of the sacculus to respond to sound is used for instrumental diagnostics of the vestibular system – cervical vestibular myogenic evoked potentials (cVEMP).
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Trinus, K. F., and K. F. Claussen. "International clinical protocol of vestibular disorders (dizziness)." East European Journal of Neurology, no. 4(4) (September 20, 2015): 4–47. http://dx.doi.org/10.33444/2411-5797.2015.4(4).4-47.

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Vertigo - a common symptom, which traditionally presents the results of vestibular dysfunction and non vestibular dysfunction. Vertigo refers to violations of orientation in space. The main symptoms, which produce the patients in the survey: dizziness, subjective vertigo, dizziness objective, pseudo vertigo, imbalance, kinetosis. In many cases, dizziness is functional, rather than an organic nature. Diagnosis of the causes of vertigo arises from the concept of the structure of the vestibular apparatus, the main idea of which is the formation of vertigo in the vestibular system. Morpho-physiological, vestibular system consists of four main projections: vestibular-cortical (sensory), vestibular-motor, vestibular-autonomic and vestibular-limbic. One of the main methods of research of a condition the vestibular apparatus and projections are vestibular evoked potentials (VEP). Owing to this method, have been established objective limits to the sensitivity of the movement. Research of vestibular-spinal responses are based on Romberg test, Unterberger-Fukuda test and Uemura test. Evaluation and treatment of patients with dizziness will differ significantly once the category of dizziness has been determined
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Efimova, Victoria L., and Irina P. Volkova. "Vestibular system and human cognitive functions." Pediatrician (St. Petersburg) 14, no. 6 (May 7, 2024): 71–78. http://dx.doi.org/10.17816/ped626401.

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The article is a review of scientific research on the influence of the vestibular system on human cognitive functions. The vestibular apparatus is well studied. Research in recent decades using functional tomography techniques has shown that it has extensive connections with the subcortical and cortical structures of the brain that provide cognitive activity. Hypotheses are put forward that the conduction and processing of bioelectric impulses by the brain, which are recorded by the vestibular apparatus, creates the necessary background for the course of all cognitive processes. The vestibular apparatus has connections with the limbic system, hippocampus, striatum and neocortex. Therefore, vestibular dysfunctions can reduce the ability to learn, cause impaired attention, memory, executive functions, cause disorientation, and affect stress levels. An urgent area of research is the study of the influence of vestibular sensory reactivity on children’s learning ability. This influence has long been underestimated, since it was generally assumed that motor and cognitive development occur independently of each other. The mechanisms linking vestibular dysfunction with cognitive impairment have not yet been sufficiently studied. Further studies are needed to assess the possible impact of vestibular dysfunctions on attention, memory, and speech. Such studies are already underway. Their results are most relevant for patients with neurodegenerative disorders and for children with special needs.
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NAKAI, YOSHIAKI. "Structure and pathology of vestibular apparatus." Equilibrium Research 46, no. 2 (1987): 111–19. http://dx.doi.org/10.3757/jser.46.111.

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Virre, E. "Virtual reality and the vestibular apparatus." IEEE Engineering in Medicine and Biology Magazine 15, no. 2 (1996): 41–43. http://dx.doi.org/10.1109/51.486717.

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Pereira, Emanuel, Bruno Ferreira, Ana Margarida Amorim, and Paulo Menezes. "Immersive Technologies for Vestibular Rehabilitation." International Journal of Creative Interfaces and Computer Graphics 13, no. 1 (January 1, 2022): 1–20. http://dx.doi.org/10.4018/ijcicg.311835.

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Vestibular vertigo accounts for about a quarter of dizziness complaints. Loss of vestibular function is a debilitating condition that causes nausea, spontaneous nystagmus, or disequilibrium, which is known to highly influence day-to-day tasks. Therefore, recovery should start as soon as possible, targeting the affected side of the vestibular apparatus, by performing a set of prescribed exercises. Aside from being a long and tiresome process, patients must perform these exercises, while trying to stimulate optokinetic, angular vestibulo-ocular, and vestibulospinal reflexes. The article presents the development of virtual reality serious games that can be played at home or in a rehabilitation clinic. The main objective is to increase patients' motivation, specifically during telerehabilitation which is essential for a faster recovery process. A preliminary evaluation was carried out to compare the users' experiences using a smartphone-based headset and a standalone commercial head-mounted display, the Oculus Quest.
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Efimova, Viktoriya Leonidovna, Elena Ivanovna Nikolaeva, Leonid Gennad'evich Buinov, Evgenii Gennad'evich Vergunov, Natal'ya Olegovna Nikolaeva, Antonina Leonidovna Khasnutdinova, and Irina Sergeevna Mazurova. "The influence of vestibular training on dynamic visual acuity in primary school students with learning difficulties." Психология и Психотехника, no. 3 (March 2023): 1–13. http://dx.doi.org/10.7256/2454-0722.2023.3.40581.

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The relevance of the study is determined by the increasing number of younger schoolchildren with difficulties in mastering reading and writing. In recent decades, the hypothesis has been confirmed that one of the causes of difficulties in mastering reading and writing in elementary school may be reduced sensory reactivity of the vestibular system. The article presents the results of an experimental study of dynamic visual acuity in children with learning difficulties. Dynamic visual acuity reflects the quality of interaction between the visual and vestibular systems. Its violations can complicate the development of reading and writing. The study involved 35 primary school students with learning difficulties. The study was conducted on the basis of a pediatric neurological clinic, instrumental studies and vestibular training were conducted as prescribed by a neurologist. At the first stage, functional diagnostics of vestibular function was carried out. The functions of the otolith part of the vestibular apparatus were evaluated by the method of cervical vestibular evoked potentials. The functions of the horizontal semicircular channels of the vestibular apparatus were evaluated by measuring the duration of post-rotational nystagmus. The experimental group included children whose learning difficulties were combined with sensory hyperactivity of the vestibular system or asymmetry of sensory reactivity of the vestibular system. Then the children underwent vestibular training on a riding simulator with visual biofeedback. The duration of the training is 14 days. The assessment of dynamic visual acuity was carried out by the standard method in sitting and standing positions before and after the training. Dynamic visual acuity depends on the quality of the vestibulocular reflex. It is shown that vestibular training on a riding simulator with visual biofeedback significantly improves dynamic visual acuity in a group of children with hyperactivity, symptoms of astheno-neurotic syndrome, asymmetry of sensory reactivity of the vestibular apparatus. Further research is needed to assess the impact of the training results on the success of children in learning.
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Černý, Rudolf, Ondřej Čakrt, and Jaroslav Jeřábek. "Laboratory methods for investigating the vestibular apparatus." Neurologie pro praxi 18, no. 3 (July 1, 2017): 163–69. http://dx.doi.org/10.36290/neu.2017.080.

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Bellé, Marcieli, Sílvia do Amaral Sartori, and Angela Garcia Rossi. "Alcoholism: effects on the cochleo-vestibular apparatus." Brazilian Journal of Otorhinolaryngology 73, no. 1 (January 2007): 110–16. http://dx.doi.org/10.1016/s1808-8694(15)31132-0.

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Dissertations / Theses on the topic "Vestibular apparatus"

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Li, Chuan. "Spatial coding of gravitational input to the vestibuloolivary pathway and its refinement in development." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31539609.

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Lee, Son Gregory Martin. "Vestibular connectivity to soleus motor units during quiet stance." Thesis, University of British Columbia, 2007. http://www.oregonpdf.org.

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Manczak, Tiago. "Estimulador galvânico vestibular para fMRI." Universidade Tecnológica Federal do Paraná, 2012. http://repositorio.utfpr.edu.br/jspui/handle/1/406.

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Este trabalho apresenta o desenvolvimento de um estimulador galvânico vestibular para ser usado em experimentos de imageamento por ressonância magnética funcional (fMRI). Em experiências de fMRI é necessário a produção de estímulos somatossensoriais no paciente. Os estímulos devem ser sincronizados com a sequência de pulsos da fMRI. O estimulador foi dividido em circuitos analógicos (colocados dentro da sala do magneto) e circuitos digitais (sala de comando do sistema de MRI). A comunicação entre os circuitos é feita através de fibra óptica. Experimentos de fMRI realizados com voluntários demonstraram que o estimulador proposto é capaz de manter a sincronização com sistema de fMRI e pode ser usado para localizar as áreas do cérebro que são ativados pelo sistema vestibular.
This work presents the development of a galvanic vestibular stimulator to be used in functional magnetic resonance imaging experiments (fMRI). In fMRI experiments it is required the production of somatosensory stimuli in the patient must be sincronized with the fMRI pulse sequence. The stimulator circuits were divided in analog circuits (placed within the magnet room) and digital circuits (placed in the MRI command room). The communication between the circuits is made through optical fiber. fMRI experiments performed with volunteers demonstrated that the proposed stimulator is able to keep the sincronization with the MRI system and can be used to locate the brain areas that are activated by the vestibular system.
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Li, Chuan, and 李川. "Spatial coding of gravitational input to the vestibuloolivary pathway and its refinement in development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31539609.

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Sansom, Andrew J., and n/a. "The role of calcium-dependent pathways in vestibular compensation." University of Otago. Department of Pharmacology & Toxicology, 2005. http://adt.otago.ac.nz./public/adt-NZDU20070418.145158.

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Damage to one vestibular apparatus (unilateral vestibular deafferentation, UVD) results in severe postural and ocular motor disturbances (such as spontaneous nystagmus, SN) that recover over time in a process known as vestibular compensation. However, the underlying neurochemical mechanisms of vestibular compensation are poorly understood. While UVD affects many areas in the CNS, attention has focused upon the partially deafferented second order neurons in the vestibular nuclei complex (VNC). Several converging lines of evidence suggest that Ca�⁺-permeable ion channels (N-methyl-D-aspartate receptors and L-type voltage-gated Ca�⁺-channels) and intracellular Ca�⁺-dependent protein kinases play an important role in vestibular compensation. However, the nature of this involvement and the locus of these changes are unknown. The aim of this thesis was to investigate the role of Ca�⁺ signalling pathways in the VNC during vestibular compensation in guinea pig. These issues were investigated in three separate experiments that utilised two methodological approaches: i) in vitro assays were used to determine the nature and extent of protein phosphorylation within the VNC at various stages of compensation; and ii) ion channel blockers or cell-permeable kinase inhibitors were injected directly into the VNC immediately before UVD to determine whether or not these systems were causally involved in compensation. The results of experiment 1 (Chapter 5) showed that a bolus intra-VNC injection of an uncompetitive NMDA receptor antagonist, but not an L-type voltage-gated Ca�⁺ channel antagonist, temporarily reduced SN frequency at the earliest measurement time (6 hours post-UVD). These results suggested that the initial expression of SN required, in part, the activation of NMDA receptors in the VNC on the side of the UVD, and by inference, Ca�⁺ entry through the ion channel. The results of experiment 2 (Chapter 6) revealed that the medial VNC contains abundant Ca�⁺/calmodulin-dependent and Ca�⁺/phospholipid-dependent protein kinase activities. The same VNC tissue removed from animals at various times after UVD, showed that vestibular compensation is accompanied by specific changes in the phosphorylation of several major protein kinase C substrates. These included an unidentified 46-kDa band, and a 75-kDa band with similar characteristics to the myristoylated alanine-rich C kinase substrate (MARCKS). These results suggest that protein kinase C signalling pathways may be involved in vestibular compensation. The results of experiment 3 (Chapter 7) are consistent with these results showing that intra-VNC infusion of a protein kinase C inhibitor, but not a Ca�⁺/calmodulin-dependent protein kinase II inhibitor, significantly increased SN at the earliest measurement times (6 and 8 hours), but had no effect upon the time taken to achieve compensation or on postural compensation. These results suggest that the induction of SN compensation involves protein kinase C activity in the VNC. Taken together, these findings suggest that the mechanisms underlying the expression of SN (e.g., Ca�⁺ influx via NMDA receptors) are possibly distinct from those that initiate its compensation (e.g., PKC activation). The downstream effects of raised intracellular Ca�⁺ may involve protein kinase C-dependent phosphorylation of key intracellular proteins that initiate long-lasting changes in cellular function within the VNC.
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Goddard, Matthew John, and n/a. "Cognitive and emotional effects of vestibular damage in rats and their medial temporal lobe substrates." University of Otago. Department of Pharmacology & Toxicology, 2008. http://adt.otago.ac.nz./public/adt-NZDU20080923.091605.

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Psychiatric disorders and cognitive impairment are increasingly being described in patients with vestibular pathology. Yet frameworks that describe the link between emotion, memory and the vestibular system have yet to reach maturity, partly because studies have not yet provided detailed accounts of behavioral changes in experimental animals, or in man. One of the goals of this thesis was to use experimental psychology to define changes in memory and emotional behaviour in rats given bilateral vestibular deafferentation (BVD, n=18) or sham surgery (Sham, n=17). In an elevated-plus maze task, BVD rats made up to 166% greater open arm entries and spent up to 42% more time in the open arms compared to Sham rats. In an elevated-T maze task, BVD rats failed to develop a normal learned inhibition response to open space. In an open field maze BVD rats consistently showed 50-60% greater movement velocity, spent on average 35% more time in the inner most aversive part of the arena, and failed to show the normal boundary-seeking behaviour (thigmotaxis) typical of untreated or Sham rats. In a social interaction test BVD rats spent up to 34% less time engaged in social contact compared to Sham rats. In a hyponeophagia test, BVD rats� latency to eat was 70% greater than Sham rats at 3-weeks post-op., however this difference disappeared at 3- and 5-months. These findings suggest that BVD treatment may in some cases disrupt normal behavioral inhibition. Memory performance was also affected. In a T-maze task BVD rats achieved 40-60% correct arm entries, compared to 90-100% for Sham controls. In a foraging task carried out in darkness, BVD rats� initial homing angle was random, homing paths were ~70% longer, and reference memory errors were ~56% greater compared to Sham rats. To elucidate possible neurochemical substrates for these behavioral changes, western blot assays on monoamine proteins were carried out on tissue from a naïve set of rats (BVD n=6; Sham n=6). In BVD rats, serotonin transporter protein expression was 39% lower in CA1 hippocampus and 27% lower in the forebrain region, despite forebrain tryptophan hydroxylase expression being 34% upregulated. Tyrosine hydroxylase expression in the forebrain region was 27% lower in BVD rats. Proteins related to synaptogenesis were also investigated. In the dentate gyrus SNAP-25 was 37% upregulated in BVD rats, while in area CA2/3 of the hippocampus neurofilament-L was 13% upregulated. Forebrain and entorhinal cortex drebrin expression was 28% and 38% downregulated in BVD rats. Neurofilament-L was also 31% downregulated in the forebrain region of BVD rats. To test whether any of these behavioral or biochemical changes may have been attributable to chronic physiological stress, a corticosterone assay was carried out at the conclusion of behavioral testing; however, the no significant between treatment differences were found. In conclusion, vestibular information appears to be needed for the acquisition of spatial and reference memory as well as the normal expression of emotional behaviour. The neurochemical changes described herein point toward possible substrates for these behaviors, however their full significance has yet to be determined.
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Lau, Yau-pok. "Postnatal development of thalamic neurons in response to vertical movement /." View the Table of Contents & Abstract, 2007. http://sunzi.lib.hku.hk/hkuto/record/B3834810X.

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劉友璞 and Yau-pok Lau. "Postnatal development of thalamic neurons in response to vertical movement." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B45011369.

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Knox, Craig A. "A model for morphological change in the hominid vestibular system in association with the rise of bipedalism." Virtual Press, 2007. http://liblink.bsu.edu/uhtbin/catkey/1371468.

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This study re-examines the morphological data and conclusions of Spoor, Wood, and Zonneveld concerning the morphology of the vestibular apparatus in relation to locomotor behavior in hominids (1994). The pedal and labyrinthine morphology of early hominid taxa are functionally analyzed for classification as either obligate bipeds or habitual bipeds with primarily arboreal locomotion. The bony labyrinth is investigated since the anatomy of the semicircular canals of the vestibular auditory system can be determined in fossil crania through computed tomographical analysis. It is thought that a relationship exists between semicircular canal size and locomotor behavior. Functionally modern pedal morphology precedes modern vestibular morphology in the fossil record. Complete modern pedal morphology, however, appears concurrently with modern vestibular morphology first at Homo erectus. A comparison of the genes involved in the development of both pedal and labyrinthine morphology was undertaken. It was found that only fibroblast growth factor 8 (FgfS) and sonic hedgehog (Shh) are shared between these systems in the determination of positional information. It is found that the function of Fgf8 in otic induction and in limb bud formation is very different. It is also found that the function of Shh in vestibular and pedal morphogenesis is different. Therefore, it is unlikely for alteration in the function or in the expression of either gene to result in the observed differences in pedal and vestibular morphology between early hominid taxa: Australopithecus afarensis, Australopithecus africanus, Homo habilis; and Homo erectus. My examination of the data on the timing of changes in pedal morphology rejects Spoor, Wood, and Zonneveld's conclusion. Moreover I find no gene mutation which could account for simultaneous change in the shape of the semicircular canals and the proportions of the metatarsals and pedal phalanges. Instead, it is postulated that the change to modern vestibular morphology at Homo erectus is in response to a concurrent enlargement in cranial capacity. It is also postulated that persistence of panid vestibular morphology in the semicircular canals of hominid taxa: Australopithecus afarensis, Australopithecus africanus, and Homo habilis is a functionally neutral trait in regard to bipedal locomotor capability.
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Moravec, William J. "Tuning in vestibular hair cells of a turtle Trachemys scripta /." Ohio : Ohio University, 2006. http://www.ohiolink.edu/etd/view.cgi?ohiou1149291414.

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Books on the topic "Vestibular apparatus"

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M, Highstein Stephen, Fay Richard R, and Popper Arthur N, eds. The vestibular system. New York: Springer, 2004.

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Japan) International Symposium on Vestibular Disorders (1994 Hiroshima-shi. International Symposium on Vestibular Disorders, Hiroshima, January, 1994. Edited by Harada Yasuo 1931-. Stockholm, Sweden: Scandinavian University Press, 1995.

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Ettore, Pirodda, and Pompeiano O, eds. Neurophysiology of the vestibular system: Selected papers of the Bárány Society Meeting, Bologna, June 1-4, 1987. Basel: Karger, 1988.

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Ettore, Pirodda, ed. Clinical testing of the vestibular system: Selected papers of the Bárány Society Meeting, Bologna, June 1-4, 1987. Basel: Karger, 1988.

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1934-, Honrubia Vicente, ed. Clinical neurophysiology of the vestibular system. 2nd ed. Philadelphia: F.A. Davis Co., 1990.

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Clendaniel, Richard A. Vestibular rehabilitation. 4th ed. Philadelphia: F.A. Davis, 2014.

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Susan, Herdman, ed. Vestibular rehabilitation. 2nd ed. Philadelphia: Davis, 2000.

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J, Yates Bill, and Miller Alan D, eds. Vestibular autonomic regulation. Boca Raton: CRC Press, 1996.

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H, Anderson John, and Beitz Alvin J, eds. Neurochemistry of the vestibular system. Boca Raton, FL: CRC Press, 2000.

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1932-, Nomura Yasuya, Schuknecht Harold F. 1917-, and International Symposium on Basic and Clinical Otology (1983 : Tokyo, Japan), eds. Hearing loss and dizziness. Tokyo: Igaku-Shoin, 1985.

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Book chapters on the topic "Vestibular apparatus"

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Dohlman, G. F. "Excretion and Absorption of Endolymph in the Vestibular Apparatus." In Novartis Foundation Symposia, 138–47. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470719565.ch9.

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Sembulingam, K., and Prema Sembulingam. "Vestibular Apparatus." In Essentials of Medical Physiology, 825. Jaypee Brothers Medical Publishers (P) Ltd., 2006. http://dx.doi.org/10.5005/jp/books/10283_158.

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Sembulingam, K., and Prema Sembulingam. "Vestibular Apparatus." In Essentials of Medical Physiology, 880. Jaypee Brothers Medical Publishers (P) Ltd., 2010. http://dx.doi.org/10.5005/jp/books/11093_158.

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Sembulingam, K., and Prema Sembulingam. "Vestibular Apparatus." In Essentials of Physiology for Dental Students, 659. Jaypee Brothers Medical Publishers (P) Ltd., 2016. http://dx.doi.org/10.5005/jp/books/12902_105.

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Pal, Gopal, Pravati Pal, and Nivedita Nanda. "Vestibular Apparatus." In Comprehensive Textbook of Medical Physiology (Volume 2), 1097. Jaypee Brothers Medical Publishers (P) Ltd., 2017. http://dx.doi.org/10.5005/jp/books/12961_51.

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Sembulingam, K., and Prema Sembulingam. "Vestibular Apparatus." In Essentials of Medical Physiology, 919. Jaypee Brothers Medical Publishers (P) Ltd., 2012. http://dx.doi.org/10.5005/jp/books/11696_74.

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Sembulingam, K., and Prema Sembulingam. "Vestibular Apparatus." In Essentials of Physiology for Dental Students, 599. Jaypee Brothers Medical Publishers (P) Ltd., 2011. http://dx.doi.org/10.5005/jp/books/11397_112.

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Bijlani, RL. "Chapter-13.11 The Vestibular Apparatus." In Understanding Medical Physiology, 657–63. Jaypee Brothers Medical Publishers (P) Ltd, 2011. http://dx.doi.org/10.5005/jp/books/11448_28.

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R.L., Bijlani. "Chapter 13.11 The Vestibular Apparatus." In Understanding Medical Physiology A Textbook for Medical Students (3rd Edition), 769–77. Jaypee Brothers Medical Publishers (P) Ltd., 2004. http://dx.doi.org/10.5005/jp/books/10999_109.

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"The Vestibular Apparatus and Balance System." In Space and Life, 69–81. CRC Press, 2004. http://dx.doi.org/10.1201/9780203602102.ch8.

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Conference papers on the topic "Vestibular apparatus"

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Gastaldi, L., S. Pastorelli, and M. Sorli. "Vestibular apparatus: dynamic model of the semicircular canals." In BIOMED 2009. Southampton, UK: WIT Press, 2009. http://dx.doi.org/10.2495/bio090211.

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Mach, Ondrej, Jan Boril, Josef Repka, Ladislav Gogh, Boris Oniscenko, Vladimir Socha, Lenka Hanakova, and Erik Blasch. "Vestibular Apparatus Training in Czech Air Force Analysis and Opportunities." In 2023 IEEE/AIAA 42nd Digital Avionics Systems Conference (DASC). IEEE, 2023. http://dx.doi.org/10.1109/dasc58513.2023.10311300.

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Reports on the topic "Vestibular apparatus"

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Training Simulator for Correct Walking Formation and Vestibular Apparatus Training. Nicholas M. Belokrylov, Ludmila V. Sharova, Alexsey V. Shepalov, Svetlana V. Annenkova, December 2016. http://dx.doi.org/10.14526/01_1111_168.

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Students’ vestibular apparatus development using physical exercises from combat sports. Vadim Yu. Ziambetov, March 2019. http://dx.doi.org/10.14526/2070-4798-2019-14-1-191-197.

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