Books on the topic 'Verapamil'

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1

Bender, F., and K. Greeff, eds. Kombinationstherapie der Herzrhytmusstörungen mit Chinidin und Verapamil. Heidelberg: Steinkopff, 1985. http://dx.doi.org/10.1007/978-3-642-85346-3.

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2

Weinstock, Barry S. Influence of verapamil on total and regional intravascular volume in the dog. [New Haven: s.n.], 1987.

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3

International Symposium, Calcium Antagonists in Cardiovascular Care (1991 : Basle, Switzerland), ed. Verapamil-- a cardioprotective strategy: Highlights from a satellite symposium to the International Symposium, Calcium Antagonists in Cardiovascular Care, Basle, Switzerland, Feb. 13, 1991. New York, NY: Raven Health Care Communications, 1991.

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4

Parker, Philip M., and James N. Parker. Verapamil: A medical dictionary, bibliography, and annoted research guide to Internet references. San Diego, CA: ICON Health Publications, 2004.

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5

Niels, Gadsbøll, Hansen Jørgen Fischer, Jespersen Christian M, and Sigurd Bjarn, eds. Danish verapamil infarction trials: Festschrift in honour of Jorgen Fischer Hansen's 60th birthday. [Copenhagen: Dept. of Cardiovascular Medicine, University of Copenhagen, Bispebjerg Hospital, 1997.

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6

Dubois, Natalie C. Inhibition of small cell lung cancer cell line (H-69) with the calcium channel blocker, Verapamil. Sudbury, Ont: Laurentian University, 1995.

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7

Albrecht, Fleckenstein, and Laragh John H. 1924-, eds. Hypertension--the next decade: Verapamil in focus : proceedings of an international symposium, Berlin ICC, 10-11 October 1985. Edinburgh: Churchill Livingstone, 1987.

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8

Muredda, Mario. The effects of calcium channel antagonist verapamil on the growth and energy metabolism of human MCF7 breast cancer cells. Sudbury, Ont: Laurentian University, 1998.

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9

J, Norris R., Wale Laurence, and Revlon Health Care Group, eds. Sustained release verapamil workshop. London: Medical News Tribune Group, 1985.

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10

Kombinationstherapie Der Herzrhytmusst Rungen Mit Chinidin Und Verapamil. Steinkopff-Verlag Darmstadt, 2012.

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11

Maria, Hacks, and Rohwedder Dirk, eds. Verapamil: A drug on the threshold of the next decade. Hamburg: Wellingsbüttel Scientific Pub. Co., 1992.

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12

Effects of [CA²+] and verapamil on muscle injury immediately after exercise. 1991.

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13

Effects of [CA²⁺] and verapamil on muscle injury immediately after exercise. 1991.

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14

Ali, Isam El-Din Mohamed. Comparative effects of Loperamide, Verapamil and Nifedipine on contractions of guinea-pigileum. Bradford, 1988.

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15

Effects of [CAp2+s] and verapamil on muscle injury immediately after exercise. 1990.

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16

Publications, ICON Health. Verapamil - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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17

(Editor), F. Bender, and K. Greeff (Editor), eds. Kombinationstherapie der Herzrhythmusstörungen mit Chinidin und Verapamil: Berichte des ersten Cordichin-Symposiums. Steinkopff-Verlag Darmstadt, 1985.

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18

Greeff, K., and F. Bender. Kombinationstherapie der Herzrhytmusstörungen Mit Chinidin und Verapamil: Berichte des Ersten Cordichin-Symposiums. Steinkopff, Dietrich, 2013.

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19

Fleckenstein, A. A. Hypertension the Next Decade: Verapamil in Focus : Proceedings of an International Symposium, Berlin. Churchill Livingstone, 1987.

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20

Hill, Susan Sarah. Comparative effects of verapamil, bepridil, and loperamide on agonist-evoked contractions of guinea-pig ileum. Bradford, 1986.

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21

Fahey, Jeanne Marie. The hematological effects of age, exercise and the calcium antagonists nimodipine and verapamil on sodium nitrite-induced methemoglobinemia. 1987.

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22

Albrecht, Fleckenstein, Laragh John H. 1924-, and Knoll AG (Ludwigshafen am Rhein, Germany), eds. Hypertension--the next decade: Verapamil in focus : proceedings of an international symposium, Berlin ICC, 10-11 October 1985. Edinburgh: Churchill Livingstone, 1987.

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23

Isbister, Geoffrey, and Colin Page. Management of β‎-blocker and calcium channel blocker poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0325.

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β‎-blocker and calcium channel-blockers can cause life-threatening toxicity due to cardiogenic shock. Both β‎-blockers and calcium channel-blockers are heterogenous groups of drugs and particular drugs, such as propranolol, diltiazem, and verapamil are far more toxic than the others in their class. The most important investigations in β‎-blocker and calcium channel-blocker overdose are an electrocardiogram, blood glucose measurement, and electrolytes. Like most overdoses, supportive treatment is the most important, with emphasis on the primary pathophysiology. Early decontamination should be considered based on the severity of the poisoning. Treatment of β‎-blockers and calcium channel-blockers poisoning, using absolute blood pressure as an endpoint can be misleading and measuring cardiac output can be more informative in gauging response to treatment. There are no specific antidotes, although β‎-agonists may be effective in β‎-blocker overdose and calcium has been shown to be effective in calcium channel-blocker overdose. The choice of inotropes and/or vasopressors will differ for β‎-blockers and calcium channel-blockers. These include isoprenaline, high dose insulin euglycaemia, phosphodiesterase inhibitors, and other catecholaminergic inotropes for β‎-blocker poisoning and adrenaline, high dose insulin euglycaemia and vasopressors for calcium channel-blocker poisoning.
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