Academic literature on the topic 'Vector-borne diseases'

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Journal articles on the topic "Vector-borne diseases"

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GUBLER, D. J. "Vector-borne diseases." Revue Scientifique et Technique de l'OIE 28, no. 2 (August 1, 2009): 583–88. http://dx.doi.org/10.20506/rst.28.2.1904.

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Kidd, Linda. "Vector-Borne Diseases." Veterinary Clinics of North America: Small Animal Practice 52, no. 6 (November 2022): i. http://dx.doi.org/10.1016/s0195-5616(22)00122-x.

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Choi, Young Hwa. "Vector-borne infectious diseases." Journal of the Korean Medical Association 60, no. 6 (2017): 449. http://dx.doi.org/10.5124/jkma.2017.60.6.449.

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Wang, Penghua, Fengwei Bai, Gong Cheng, Jianfeng Dai, and Michael J. Conway. "Vector-Borne Viral Diseases." BioMed Research International 2015 (2015): 1. http://dx.doi.org/10.1155/2015/582045.

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Rajagopalan, P. K. "Aspects of Vector Borne Disease Control." Journal of Communicable Diseases 50, no. 01 (March 29, 2018): 28–31. http://dx.doi.org/10.24321/0019.5138.201806.

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Dhopte, Pragati, and Irrusappan Hari. "VECTOR-BORNE DISEASES IN INDIA." International Journal of Advanced Research 8, no. 10 (October 31, 2020): 1055–67. http://dx.doi.org/10.21474/ijar01/11933.

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Vectors are transmitted diseases from person to person that diseases are known as vactor borne diseases. There are mainly six vector borne diseases present in India, tropical and subtropical rigion also. As per current medical importance, geographic distribution, epidemiology and potential spreading of vector borne diseases, Malaria total cases were 29340 and deaths 2 and Japanese encephalitis total cases were 111. Chikungunya and Kala azar total cases were 700 and no deaths were found in 2020 respectively. 87.25% of MDA were supplied to total population and the dengue cases were 136422 and deaths 132 were observed in 2019. The vector borne diseases in India are reviewed in this article.
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Copping, Leonard G. "Vector-Borne Diseases in Europe." Outlooks on Pest Management 20, no. 4 (August 1, 2009): 174–75. http://dx.doi.org/10.1564/20aug08.

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Zorrilla-Vaca, A. "Bedbugs and Vector-Borne Diseases." Clinical Infectious Diseases 59, no. 9 (July 16, 2014): 1351–52. http://dx.doi.org/10.1093/cid/ciu575.

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O’Kelly, Brendan, and John S. Lambert. "Vector-borne diseases in pregnancy." Therapeutic Advances in Infectious Disease 7 (January 2020): 204993612094172. http://dx.doi.org/10.1177/2049936120941725.

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Vector-borne infections cause a significant proportion of world-wide morbidity and mortality and many are increasing in incidence. This is due to a combination of factors, primarily environmental change, encroachment of human habitats from urban to peri-urban areas and rural to previously uninhabited areas, persistence of poverty, malnutrition and resource limitation in geographical areas where these diseases are endemic. Pregnant women represent the single largest ‘at risk’ group, due to immune-modulation and a unique physiological state. Many of these diseases have not benefitted from the same level of drug development as other infectious and medical domains, a factor attributing to the ‘neglected tropical disease’ title many vector-borne diseases hold. Pregnancy compounds this issue as data for safety and efficacy for many drugs is practically non-existent, precluding exposure in pregnancy to many first-line therapeutic agents for ‘fear of the unknown’ or overstated adverse pregnancy-foetal outcomes. In this review, major vector-borne diseases, their impact on pregnancy outcomes, current treatment, vaccination and short-comings of current medical practice for pregnant women will be discussed.
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Senior, Kathryn. "Vector-borne diseases threaten Europe." Lancet Infectious Diseases 8, no. 9 (September 2008): 531–32. http://dx.doi.org/10.1016/s1473-3099(08)70192-0.

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Dissertations / Theses on the topic "Vector-borne diseases"

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El, Moustaid Fadoua. "Modeling Temperature Effects on Vector-Borne Disease Dynamics." Diss., Virginia Tech, 2019. http://hdl.handle.net/10919/102579.

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Vector-borne diseases (VBDs) cause significant harm to humans, plants, and animals worldwide. For instance, VBDs are very difficult to manage, as they are governed by complex interactions. VBD transmission depends on the pathogen itself, vector-host movement, and environmental conditions. Mosquito-borne diseases are a perfect example of how all these factors contribute to changes in VBD dynamics. Although vectors are highly sensitive to climate, modeling studies tend to ignore climate effects. Here, I am interested in the arthropod small vectors that are sensitive to climate factors such as temperature, precipitation, and drought. In particular, I am looking at the effect of temperature on vector traits for two VBDs, namely, dengue, caused by a virus that infects humans and bluetongue disease, caused by a virus that infects ruminants. First, I collect data on mosquito traits' response to temperature changes, this includes adult traits as well as juvenile traits. Next, I use these traits to model mosquito density, and then I incorporate the density into our mathematical models to investigate the effect it has on the basic reproductive ratio R0, a measure of how contagious the disease is. I use R0 to determine disease risk. For dengue, my results show that using mosquito life stage traits response to temperature improves our vector density approximation and disease risk estimates. For bluetongue, I use midge traits response to temperature to show that the suitable temperature for bluetongue risk is between 21.5 �C and 30.7 �C. These results can inform future control and prevention strategies.
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Xue, Ling. "Modeling and analysis of vector-borne diseases on complex networks." Diss., Kansas State University, 2013. http://hdl.handle.net/2097/16788.

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Doctor of Philosophy
Department of Electrical and Computer Engineering
Caterina Scoglio
Vector-borne diseases not only cause devastating economic losses, they also significantly impact human health in terms of morbidity and mortality. From an economical and humane point of view, mitigation and control of vector-borne diseases are essential. Studying dynamics of vector-borne disease transmission is a challenging task because vector-borne diseases show complex dynamics impacted by a wide range of ecological factors. Understanding these factors is important for the development of mitigation and control strategies. Mathematical models have been commonly used to translate assumptions concerning biological (medical, demographical, behavioral, immunological) aspects into mathematics, linking biological processes of transmission and dynamics of infection at population level. Mathematical analysis translates results back into biology. Classical deterministic epidemic models do not consider spatial variation, assuming space is homogeneous. Spatial spread of vector-borne diseases observed many times highlights the necessity of incorporating spatial dynamics into mathematical models. Heterogeneous demography, geography, and ecology in various regions may result in different epidemiological characteristics. Network approach is commonly used to study spatial evolution of communicable diseases transmitted among connected populations. In this dissertation, the spread of vector-borne diseases in time and space, is studied to understand factors that contribute to disease evolution. Network-based models have been developed to capture different features of disease transmission in various environments. Network nodes represent geographical locations, and the weights represent the level of contact between regional pairings. Two competent vector populations, Aedes mosquitoes and Culex mosquitoes, and two host populations, cattle and humans were considered. The deterministic model was applied to the 2010 Rift Valley fever outbreak in three provinces of South Africa. Trends and timing of the outbreak in animals and humans were reproduced. The deterministic model with stochastic parameters was applied to hypothetical Rift Valley fever outbreak on a large network in Texas, the United States. The role of starting location and size of initial infection in Rift Valley fever virus spread were studied under various scenarios on a large-scale network. The reproduction number, defined as the number of secondary infections produced by one infected individual in a completely susceptible population, is typically considered an epidemic threshold of determining whether a disease can persist in a population. Extinction thresholds for corresponding Continuous-time Markov chain model is used to predict whether a disease can perish in a stochastic setting. The network level reproduction number for diseases vertically and horizontally transmitted among multiple species on heterogeneous networks was derived to predict whether a disease can invade the whole system in a deterministic setting. The complexity of computing the reproduction number is reduced because the expression of the reproduction number is the spectral radius of a matrix whose size is smaller than the original next generation matrix. The expression of the reproduction number may have a wide range of applications to many vector-borne diseases. Reproduction numbers can vary from below one to above one or from above one to below one by changing movement rates in different scenarios. The observations provide guidelines on executing movement bans in case of an epidemic. To compute the extinction threshold, corresponding Markov chain process is approximated near disease free equilibrium. The extinction threshold for Continuous-time Markov chain model was analytically connected to the reproduction number under some assumptions. Numerical simulation results agree with analytical results without assumptions, proposing a mathematical problem of proving the existence of the relationships in general. The distance of the extinction threshold were shown to be closer to one than the reproduction number. Consistent trends of probability of extinction varying with disease parameters observed through numerical simulations provide novel insights into disease mitigation, control, and elimination.
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McOdimba, Francis Awuor. "Epidemiology of vector-borne diseases in cattle from SE Uganda." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/30498.

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Institutions involved in vector-borne diseases research, epidemiological studies as well as vaccine development require reliable and sensitive assays to support the development of vaccine products and new drugs for treatment. These diagnostic assays also aid in identifying disease control target populations, and to monitor infection during trials for assessing the efficacy of preventive or curative drug. Molecular techniques such as the polymerase chain reaction (PCR) amplification have been used in detecting parasites of several species, sub-species and types and are favoured over microscopic examination of blood or the immunological methods because of their superior sensitivity and higher throughput. Two of the most commonly used diagnostic methods, microscopy and molecular techniques for pathogen detection and species characterization, were evaluated for their sensitivity and specificity and subsequently used in screening cattle for parasites in the blood of cattle kept under traditional mixed farming management system. Molecular methods revealed higher VBD prevalence in the cattle from the villages of Tororo and Busia districts of SE Uganda. The prevalence of trypanosome species pathogenic to livestock was found to be higher than previously documented in this area. Based on the data obtained by PCR amplification the effect of prophylactic drug intervention against trypanosomiasis was assessed over a period of six months. While isometamidium chloride treatment of cattle appeared to control trypanosomiasis in areas with low prevalence, the drug had no effect in controlling the disease in high prevalence areas. It would therefore be necessary to combine the use of drug intervention with other methods such as vector control, to reduce the prevalence, in order to realize effective control of trypanosomiasis.
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Shearer, Freya. "Improving geospatial models of risk for vector-borne, zoonotic diseases." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:cfe8ffa9-453b-4e10-9009-e387a39db6de.

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Public health surveillance data are often incomplete, particularly where resources are lacking, but geospatial models can help to fill the gaps by providing estimates where data are sparse. By combining information on locations where diseases have been recorded with geographic data on environmental and socioeconomic covariates known to affect disease transmission using machine-learning models (such as boosted regression trees), niche modelling can generate fine-resolution, evidence-based risk maps for a variety of diseases of public health importance. This thesis investigates the geographical distribution of two vector-borne, zoonotic diseases of public health importance: Plasmodium knowlesi malaria and yellow fever (YF). A number of new methodological approaches to niche modelling are developed for: mapping diseases whose distributions are impacted by multiple host and vector species, ameliorating spatial bias in disease reporting rates, and accounting for human vaccination coverage. Chapter 2 investigates spatial variation in risk of human P. knowlesi infection across Southeast Asia. The infection risk model for P. knowlesi malaria is based on improvements to a standard niche modelling approach, and incorporates a novel joint distribution model to leverage data from a number of host species. Chapter 3 estimates YF vaccination coverage through time across all age cohorts in every district/municipality of countries at risk of YF, globally. These estimates are used to estimate the additional vaccination coverage needed to prevent further YF outbreaks, and they provide information needed to account for population immunity when estimating YF infection risk. Chapter 4 describes the development of a novel Poisson point process niche model, which is then used to predict YF infection risk in humans and demonstrates how vaccination coverage can be efficiently accounted for in disease niche models. The disease risk maps of P. knowlesi malaria and YF produced through this thesis will act as resources to improve the targeting, implementation and evaluation of disease prevention, surveillance and control strategies. Methods developed to account for vaccination coverage, reporting rate biases, and complex transmission systems will be applicable to risk mapping for a range of vector-borne, zoonotic diseases of public health importance.
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PEPA, A. DELLA. "VECTOR-BORNE DISEASES IN COLONY STRAY CATS OF MILAN CITY." Doctoral thesis, Università degli Studi di Milano, 2013. http://hdl.handle.net/2434/219128.

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LEISHMANIA INFANTUM INFECTION IN STRAY CATS IN A NON-ENDEMIC AREA IN NORTHERN ITALY E. Spada, DVM, PhD, Researcher 1, A. Della Pepa, DVM 1, A. Migliazzo, DVM, PhD 2, G. Bagnagatti De Giorgi, DVM 1, R. Perego, DVM, PhD 1, D. Proverbio, DVM, PhD, Professor 1 1Dipartimento di Scienze Veterinarie per la Salute, la Produzione Animale e la Sicurezza Alimentare, Universita degli Studi di Milano, Milan, Italy 2Centro di Referenza Nazionale per le Leishmaniosi,Istituto Zooprofilattico Sperimentale della Sicilia, Palermo, Italy Tipologia: Ricerca Originale Area di interesse: Medicina interna Purpose of the work. To assess the prevalence of leishmaniosis in a large representative sample of stray cats from this non-endemic area, and to analyze the results according to clinical, laboratory and infectious data 2. MOLECULAR STUDY ON VECTOR-BORNE INFECTIONS IN URBAN STRAY COLONY CAT IN NORTHENRN ITALY Eva Spada§, DVM, PhD, Researcher Daniela Proverbio§, DVM, PhD, Professor Alessandra Della Pepa§, DVM Paola Galluzzo*, Biologist Roberta Perego§, DVM, PhD Giada Bagnagatti De Giorgi§, DVM Abstract Feline vector-borne diseases are caused by a wide range of pathogens, which are transmitted by arthropods. Many of these infections have zoonotic implications and feral cats may potentially act as sentinels of human and pet health. The present study investigated the prevalence of vector-borne infections in feral colony cats in the city of Milan in northern Italy. Blood samples from 260 feral cats were evaluated, with conventional PCR, for the presence of DNA associated with hemoplasmas (Mycoplasma haemofelis and Mycoplasma haemominutum), Rickettsia spp., Anaplasma phagocytophilum, Ehrlichia spp. and Babesia microti. Odd ratios (OR) were calculated to identify risk factors for infection with vector-borne pathogens. Positive PCR was found in 156 out of 260 subjects (60%), with a prevalence of 33.1% for hemoplasmas, 31.9% for Rickettsia spp., 17.7% for A. phagocytophilum , 6.7% for Ehrlichia spp. (out of 30 samples), and 1.2% for B. microti spp (out of 168 samples). Statistical analysis revealed a correlation between infections with Rickettsia spp. and hemoplasmas (OR=1.95, P=0.02). Additionally, Rickettsia spp. infection was associated with ocular infection (OR=2.21, P=0.02). We conclude that vector-borne infections, including zoonotic diseases, are present in feral cats of Milan. Thus, domestic cats exposed to the outdoors should be routinely monitored and treated for ectoparasites to minimize disease onset and potential transmission of zoonotic agents to humans. Moreover, as these vector-borne infections are transmitted through blood, feline blood donors from this area should be screened by PCR.
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Twiddy, Sally Susanna. "The molecular epidemiology and evolution of dengue virus." Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269490.

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Shukullari, Enstela [Verfasser]. "Parasites and Vector-borne Diseases in Client-owned Dogs in Albania / Enstela Shukullari." München : Verlag Dr. Hut, 2016. http://d-nb.info/1135988994/34.

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Morin, Cory William. "Climate and Environmental Influences on the Ecology of Vectors and Vector-borne Diseases." Diss., The University of Arizona, 2012. http://hdl.handle.net/10150/241951.

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Recently researchers have recognized the potential effects of climate variability and climate change on infectious disease ecology. Mosquito-borne diseases are of considerable concern due to their reliance on temperature to regulate vector reproduction, survival, and vector and agent development. Precipitation is also influential because it helps maintain habitat for immature mosquitoes. The interactions between climate, vector, and agent are complex, however, and thus assessing the overall impact of climate on disease occurrence is difficult. Discerning the influence of climate on mosquito-borne diseases requires an interdisciplinary synthesis of knowledge about the relationships between components of the disease system and analysis techniques that account for the individual and interacting roles that each element contributes to the ecology of the disease. In this dissertation, climate and climate change influences on dengue fever and West Nile virus are identified through process based modeling to simulate changes in vector and viral transmission dynamics. Analysis of the literature pertaining to climate influences on dengue virus ecology reveals that climate variables often interact interdependently to influence dengue virus transmission. Statistical techniques correlating or modeling climate-dengue relationships are often inconsistent and location specific. Process based modeling has been employed to better simulate the intricacies and non-linear dynamics involved, but most models focus only on vector populations. Therefore, models should incorporate viral development and transmission components to better simulate dengue virus ecology. A model of West Nile virus vector dynamics across the southern United States reveals that impacts from climate change are very location and context-specific. While temperatures generally increase the season length of vector activity, changes in precipitation and evapotranspiration dynamics often lead to lower summer mosquito populations and limited population development in water-stressed areas. A simulation of dengue fever cases in San Juan County, Puerto Rico with a coupled vector-epidemiological model showed strong agreement when compared with reported case data (Willmott's d = 0.90 and r2 = 0.71). The model indicates that certain climate variables became disease limiting during specific times of the year. Temperature limits virus transmission during the winter by slowing viral development while lower precipitation limits spring transmission by suppressing vector populations.
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Federico, Stefano. "Towards innovative tools against vector-borne diseases: focusing on Plasmodium and Leishmania spp." Doctoral thesis, Università di Siena, 2022. http://hdl.handle.net/11365/1194525.

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Up to date, the World Health Organization (WHO) recognizes twenty conditions belonging to neglected tropical diseases (NTDs) caused by parasites, viruses, bacteria, and snake envenoming that affect some of the World’s poorest areas, predominantly in Africa, Asia, and the Americas. NTDs, that affect more than a billion people worldwide, are referred to as “neglected” as they receive inadequate attention, e.g., in terms of research funding, when compared to other diseases. Of the twenty NTDs recognized by the WHO, twelve are caused by parasites. Based on data provided by the 2019 Global Burden of Disease Study (GBD), over 20 million disability adjusted life years (DALYs) are caused by NTDs and approximately 750,000 people died because of NTDs and malaria. Taken together, these data lead malaria and NTDs to be the 15th leading cause of death worldwide. Regarding malaria, based on our previous study on bridged bicyclic 2,3-dioxabicyclo[3,3,1]dioxanes as antimalarial agents, in this work we aimed at improving the potency and the pharmacokinetic profiles of the latter by developing two new classes of bridged bicyclic endoperoxides. The introduction of protonable chains at R1 led to a marked increase in potency with respect to previous derivatives; additionally, the introduction of until-now unexplored triazine-based R1 substituents paved the way for the rational design of novel optimized antimalarial agents. Both classes of endoperoxides showed good inhibitory potency toward P. falciparum, and these results were also rationalized by in silico analysis of the interaction between the peroxide bridge and Fe(II)-heme. Furthermore, taking inspiration from the anticancer properties of ART-derived dimers, three new sets of endoperoxide-based dimers were designed and synthesized. The study design aimed at unveiling the main feature required for the explication of the antitumor activity. Preliminary biological investigation performed in human leukemia HL-60 cell line highlighted compounds 66d and 66g as the most promising derivatives of the series. In conclusion, 24 new chemical entities were synthesized and subjected to biological investigation. As per NTDs, we have identified 25 new chemical entities active against Leishmania (and possibly other trypanosomatids) trypanothione reductase, derived from the hit compound 138a. The potent and selective TR inhibitor 138a, identified by screening of GSK LeishBox, acts by selectively bind the TS2 binding pocket of TR (with respect to hGR). Further structural information were obtained by crystallography studies, which led to the resolution of the co-crystal structure of 138a in complex with TbTR, thus confirming the mechanism of inhibition. The intensive SAR analysis led to the identification of the most important features of the parent compound. The most promising derivatives, in terms of IC50 values against LiTR, were also evaluated in phenotypic assays against axenic amastigote and microphage-infecting promastigote life cycle stages of L. infantum. Moreover, the toxicity profile for some of the best compound was assessed in 3T3 and HepG2 cell lines to get preliminary information about the selectivity of the latter versus human hosts. Further biological studies are ongoing to validate the therapeutic potential of this new class of TR inhibitors in an in vivo murine model of Leishmania infection.
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Alonso, Wladimir Jimenez. "Vector host choice and the environmental context of mosquito-borne virus transmission." Thesis, University of Oxford, 2003. http://ora.ox.ac.uk/objects/uuid:bc3632b8-321a-4751-8797-80b40098ec27.

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The present thesis explored ethological and geographical approaches for the investigation of vector-borne parasites. In the first part, the role of associative learning on vector preferences for hosts was investigated through a comprehensive series of behavioural experiments using the vector of dengue and yellow fever diseases, the mosquito Aedes aegypti. To this end, the possibility that the mosquitoes were able to associate unconditional stimuli with particular odours and visual patterns to which they were responsive was explored, but no evidence supporting the hypothesis that associative learning abilities are present in adults of this species was found. A critical review of the literature on learning in mosquitoes conducted afterward allowed the reinterpretation of findings in the field, narrowing the scope of evidence suggesting the existence of these cognitive abilities in some species. In the second part of the thesis, the distribution and evolution of mosquito-borne viruses was investigated with the use of geo-coded environmental data and spatial statistics. Initially, the eco-climates associated with the distribution of Japanese encephalitis virus were described and modelled, allowing the production of a worldwide predictive map defining the probability of each region to develop this disease in the future. Predominating amongst those areas shown to be under high risk were the equatorial regions of South America and Africa. The methodology used to infer such patterns – non-linear discriminant analysis – was subsequently explored with a number of simulations. Overall, differences in the choice of parameters required for the analysis were shown to lead to differences in the final outputs produced, basically in those cases where the environmental range for which predictions are generated is not rigorously limited. Finally, eco-climate surrogates for the evolution of the Japanese encephalitis serocomplex were investigated, but the current environmental distances between the viruses did not seem to be associated with the events leading to their speciation.
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Books on the topic "Vector-borne diseases"

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Society for General Microbiology. Symposium. Microbe-vector interactions in vector-borne diseases. Cambridge [Eng.]: Cambridge University Press, 2004.

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W, Service M., ed. Demography and vector-borne diseases. Boca Raton, Fla: CRC Press, 1989.

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International Symposium of Vectors & Vector-borne Diseases (8th 2006 Madurai, India). Vector-borne diseases: Epidemiology and control. Edited by Tyagi B. K and Indian Council of Medical Research. Centre for Research in Medical Entomology. Jodhpur: Scientific Publishers, India on behalf of Centre for Research in Medical Entomology, ICMR, 2008.

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Mack, Alison, ed. Global Health Impacts of Vector-Borne Diseases. Washington, D.C.: National Academies Press, 2016. http://dx.doi.org/10.17226/21792.

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Takken, Willem, and Bart G. J. Knols, eds. Emerging pests and vector-borne diseases in Europe. The Netherlands: Wageningen Academic Publishers, 2007. http://dx.doi.org/10.3920/978-90-8686-626-7.

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W, Takken, and Knols B. G. J, eds. Emerging pests and vector-borne diseases in Europe. Wageningen: Wageningen Academic Publishers, 2007.

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Serap, Aksoy, ed. Transgenesis and the management of vector-borne disease. New York: Springer Science+Business Media, 2008.

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Garros, Claire, Jérémy Bouyer, Willem Takken, and Renate C. Smallegange, eds. Pests and vector-borne diseases in the livestock industry. The Netherlands: Wageningen Academic Publishers, 2018. http://dx.doi.org/10.3920/978-90-8686-863-6.

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Emmanuel, Camus, House James A, Uilenberg G, and Vector-Borne Pathogens: Challenges for the 21st Century and International Trade and Animal Diseases (1996 : San José, Costa Rica), eds. Vector-borne pathogens: International trade and tropical animal diseases. New York, N.Y: New York Academy of Sciences, 1996.

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K, Panigrahi Srikanta, and Anand Mona, eds. Vector borne diseases in India: Environmental health & policy perspectives. New Delhi: Manak Publications, 2007.

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Book chapters on the topic "Vector-borne diseases"

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Müller, Ruth, Friederike Reuss, Vladimir Kendrovski, and Doreen Montag. "Vector-Borne Diseases." In Biodiversity and Health in the Face of Climate Change, 67–90. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-02318-8_4.

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Seifert, Horst S. H. "Vector-borne Diseases." In Tropical Animal Health, 149–270. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0147-6_5.

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Wilder-Smith, Annelies. "Vector-borne diseases." In Essential Travel Medicine, 65–73. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118597361.ch7.

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Sharma, Satyavan. "Vector-borne diseases." In Progress in Drug Research / Fortschritte der Arzneimittelforschung / Progrès des recherches pharmaceutiques, 365–485. Basel: Birkhäuser Basel, 1990. http://dx.doi.org/10.1007/978-3-0348-7133-4_8.

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Martcheva, Maia. "Vector-Borne Diseases." In Texts in Applied Mathematics, 67–89. Boston, MA: Springer US, 2015. http://dx.doi.org/10.1007/978-1-4899-7612-3_4.

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Gupta, Pankaj. "Vector-Borne Diseases." In Environmental Health and Occupational Safety, 210–26. Boca Raton: CRC Press, 2024. http://dx.doi.org/10.1201/9781003464785-9.

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Goddard, Jerome. "Miscellaneous Vector-Borne Diseases." In Infectious Diseases and Arthropods, 153–76. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-400-5_7.

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Balakrishnan, Indran, and Stephen H. Gillespie. "Vector-Borne Parasitic Diseases." In Principles and Practice of Travel Medicine, 91–124. Chichester, UK: John Wiley & Sons, Ltd, 2002. http://dx.doi.org/10.1002/0470842512.ch8.

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Goddard, Jerome. "Miscellaneous Vector-Borne Diseases." In Infectious Diseases and Arthropods, 173–95. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-75874-9_7.

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Goddard, Jerome. "Miscellaneous Vector-Borne Diseases." In Infectious Diseases and Arthropods, 151–73. Totowa, NJ: Humana Press, 2000. http://dx.doi.org/10.1007/978-1-59259-721-5_7.

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Conference papers on the topic "Vector-borne diseases"

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Anguelov, Roumen, Jean Lubuma, and Yves Dumont. "Mathematical analysis of vector-borne diseases on plants." In 2012 IEEE 4th International Symposium on Plant Growth Modeling, Simulation, Visualization and Applications (PMA). IEEE, 2012. http://dx.doi.org/10.1109/pma.2012.6524808.

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ROGERS, DAVID. "NEW APPROACHES FOR STUDYING VECTORS AND VECTOR-BORNE DISEASES." In International Seminar on Nuclear War and Planetary Emergencies 40th Session. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789814289139_0033.

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NETESOV, SERGEY. "VECTOR-BORNE DISEASES IN THE ASIAN PART OF RUSSIA." In International Seminar on Nuclear War and Planetary Emergencies 40th Session. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789814289139_0037.

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Kitron, Uriel D. "Local modulation of climate change impact on vector-borne diseases." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94728.

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McAllister, Janet. "Entomology at CDC: Protecting the public from vector-borne diseases." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.103181.

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S, Katyayani Akella N., and Mandaar B. Pande. "Utilising Data From Social Media In Modelling Vector-Borne Diseases." In 2021 IEEE 2nd International Conference on Technology, Engineering, Management for Societal impact using Marketing, Entrepreneurship and Talent (TEMSMET). IEEE, 2021. http://dx.doi.org/10.1109/temsmet53515.2021.9768688.

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Panda, G. K., Indrani Kumari Sahu, and Susant Kumar Dash. "A Rough Set Approach for Knowledge Acquisition on Vector-Borne Diseases." In 2019 Second International Conference on Advanced Computational and Communication Paradigms (ICACCP). IEEE, 2019. http://dx.doi.org/10.1109/icaccp.2019.8882970.

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Pandya, Darshanaben Dipakkumar, Shailesh Kantilal Patel, Abdul Hamid Qureshi, Akashgiri Jashvantgiri Goswami, Sheshang Degadwala, and Dhairya Vyas. "Multi-Class Classification of Vector Borne Diseases using Convolution Neural Network." In 2023 2nd International Conference on Applied Artificial Intelligence and Computing (ICAAIC). IEEE, 2023. http://dx.doi.org/10.1109/icaaic56838.2023.10140654.

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McVey, D. Scott. "Predictive biology and control strategies for vector-borne diseases of livestock." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.107331.

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Wood, Byron, John Vesecky, Jim Lawless, Louisa Beck, and Joan Salute. "Medsat: A satellite system for surveillance of tropical vector-borne diseases." In The earth and space science information system (ESSIS). AIP, 1993. http://dx.doi.org/10.1063/1.44501.

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Reports on the topic "Vector-borne diseases"

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Ross, S. G., M. C. Thomson, and T. Pultz. RADARSAT-1 for Monitoring Vector-borne Diseases in Tropical Environments: A Review. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2001. http://dx.doi.org/10.4095/219826.

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Estrugo, Andrés, and Rodrigo García Ayala. Assessing the Effects of Climate and Socioeconomic Factors on Vulnerability to Vector-Borne Diseases in Latin America. Inter-American Development Bank, July 2014. http://dx.doi.org/10.18235/0011647.

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Climate change is imposing a large burden on the most vulnerable populations, particularly in the developing world. Establishing consistent causal relationships, however, is difficult because a multiplicity of climatic, economic and sociodemographic elements are combined to create the conditions for an outbreak of vector-borne disease. Based on a two-step procedure, this paper presents and tests an approach to estimating the effects of epidemic outbreaks on health vulnerability. The model proposed is empirically tested for five countries in Latin America where dengue is a national health priority. Using data from national censuses, satellite climate information and data from a newly developed disease outbreak surveillance online platform, the paper finds that climate has nonnegligible effects on health vulnerability. The evidence found and the vulnerability index constructed can be used to analyze the main determinants of vulnerability in order to address policy concerns.
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Ibáñez, Ana María, Sandra Rozo, and Maria J. Urbina. Forced Migration and the Spread of Infectious Diseases. Inter-American Development Bank, November 2020. http://dx.doi.org/10.18235/0002894.

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We examine the role of Venezuelan forced migration on the propagation of 15 infectious dis-eases in Colombia. For this purpose, we use rich municipal-monthly panel data. We exploit the fact that municipalities closer to the main migration entry points have a disproportionate ex-posure to infected migrants when the cumulative migration flows increase. We find that higher refugee inflows are associated with increments in the incidence of vaccine-preventable dis-eases, such as chickenpox and tuberculosis, as well as sexually transmitted diseases, including AIDS and syphilis. However, we find no significant effects of migration on the propagation of vector-borne diseases. Contact with infected migrants upon arrival seems to be the main driving mechanism.
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Valencia-Amaya, Mauricio G., and Dolores de la Mata. The Health Impacts of Severe Climate Shocks in Colombia. Inter-American Development Bank, July 2014. http://dx.doi.org/10.18235/0011649.

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This paper studies the link between severe weather shocks in Colombia and municipality-level incidence of dengue and malaria. The unexpectedly high variability of the 2010 rainfalls relative to previous periods and their regional heterogeneity are exploited as an identification strategy. A differences-indifferences DD) strategy is thereby implemented where the period 2007-2009 is defined as the pre-treatment period and 2010-2011 as the post-treatment period. The treatment group is all municipalities that experienced higher intra-year rain variability in 2010 than in 2007-2009. The results from the different specifications confirm that the relationship between climate events and vector-borne diseases is intricate. The 2010 weather shocks are associated with not only an increase in the number of dengue cases, in the case of high variability (but not extreme) yearly rain, but also a decrease in its incidence, in particular in the presence of extreme rain events. Floods seem to have decreased the number of dengue cases.
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Gottlieb, Yuval, Bradley Mullens, and Richard Stouthamer. investigation of the role of bacterial symbionts in regulating the biology and vector competence of Culicoides vectors of animal viruses. United States Department of Agriculture, June 2015. http://dx.doi.org/10.32747/2015.7699865.bard.

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Symbiotic bacteria have been shown to influence host reproduction and defense against biotic and abiotic stressors, and this relates to possible development of a symbiont-based control strategy. This project was based on the hypothesis that symbionts have a significant impact on Culicoides fitness and vector competence for animal viruses. The original objectives in our proposal were: 1. Molecular identification and localization of the newly-discovered symbiotic bacteria within C. imicola and C. schultzei in Israel and C. sonorensis in California. 2. Determination of the prevalence of symbiotic bacteria within different vector Culicoides populations. 3. Documentation of specific symbiont effects on vector reproduction and defense: 3a) test for cytoplasmic incompatibility in Cardinium-infected species; 3b) experimentally evaluate the role of the symbiont on infection or parasitism by key Culicoides natural enemies (iridescent virus and mermithid nematode). 4. Testing the role(s) of the symbionts in possible protection against infection of vector Culicoides by BTV. According to preliminary findings and difficulties in performing experimental procedures performed in other insect symbiosis systems where insect host cultures are easily maintained, we modified the last two objectives as follows: Obj. 3, we tested how symbionts affected general fitness of Israeli Culicoides species, and thoroughly described and evaluated the correlation between American Culicoides and their bacterial communities in the field. We also tried alternative methods to test symbiont-Culicoides interactions and launched studies to characterize low-temperature stress tolerances of the main US vector, which may be related to symbionts. Obj. 4, we tested the correlation between EHDV (instead of BTV) aquisition and Cardinium infection. Culicoides-bornearboviral diseases are emerging or re-emerging worldwide, causing direct and indirect economic losses as well as reduction in animal welfare. One novel strategy to reduce insects’ vectorial capacity is by manipulating specific symbionts to affect vector fitness or performance of the disease agent within. Little was known on the bacterial tenants occupying various Culicoides species, and thus, this project was initiated with the above aims. During this project, we were able to describe the symbiont Cardinium and whole bacterial communities in Israeli and American Culicoides species respectively. We showed that Cardinium infection prevalence is determined by land surface temperature, and this may be important to the larval stage. We also showed no patent significant effect of Cardinium on adult fitness parameters. We showed that the bacterial community in C. sonorensis varies significantly with the host’s developmental stage, but it varies little across multiple wastewater pond environments. This may indicate some specific biological interactions and allowed us to describe a “core microbiome” for C. sonorensis. The final set of analyses that include habitat sample is currently done, in order to separate the more intimately-associated bacteria from those inhabiting the gut contents or cuticle surface (which also could be important). We were also able to carefully study other biological aspects of Culicoides and were able to discriminate two species in C. schultzei group in Israel, and to investigate low temperature tolerances of C. sonorensis that may be related to symbionts. Scientific implications include the establishment of bacterial identification and interactions in Culicoides (our work is cited in other bacteria-Culicoides studies), the development molecular identification of C. schultzei group, and the detailed description of the microbiome of the immature and matched adult stages of C. sonorensis. Agricultural implications include understanding of intrinsic factors that govern Culicoides biology and population regulation, which may be relevant for vector control or reduction in pathogen transmission. Being able to precisely identify Culicoides species is central to understanding Culicoides borne disease epidemiology.
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Klement, Eyal, Elizabeth Howerth, William C. Wilson, David Stallknecht, Danny Mead, Hagai Yadin, Itamar Lensky, and Nadav Galon. Exploration of the Epidemiology of a Newly Emerging Cattle-Epizootic Hemorrhagic Disease Virus in Israel. United States Department of Agriculture, January 2012. http://dx.doi.org/10.32747/2012.7697118.bard.

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In September 2006 an outbreak of 'Bluetongue like' disease struck the cattle herds in Israel. Over 100 dairy and beef cattle herds were affected. Epizootic hemorrhagic disease virus (EHDV) (an Orbivirusclosely related to bluetongue virus (BTV)), was isolated from samples collected from several herds during the outbreaks. Following are the aims of the study and summary of the results: which up until now were published in 6 articles in peer-reviewed journals. Three more articles are still under preparation: 1. To identify the origin of the virus: The virus identified was fully sequenced and compared with the sequences available in the GenBank. It appeared that while gene segment L2 was clustered with EHDV-7 isolated in Australia, most of the other segments were clustered with EHDV-6 isolates from South-Africa and Bahrain. This may suggest that the strain which affected Israel on 2006 may have been related to similar outbreaks which occurred in north-Africa at the same year and could also be a result of reassortment with an Australian strain (Wilson et al. article in preparation). Analysis of the serological results from Israel demonstrated that cows and calves were similarly positive as opposed to BTV for which seropositivity in cows was significantly higher than in calves. This finding also supports the hypothesis that the 2006 EHD outbreak in Israel was an incursive event and the virus was not present in Israel before this outbreak (Kedmi et al. Veterinary Journal, 2011) 2. To identify the vectors of this virus: In the US, Culicoides sonorensis was found as an efficient vector of EHDV as the virus was transmitted by midges fed on infected white tailed deer (WTD; Odocoileusvirginianus) to susceptible WTD (Ruder et al. Parasites and Vectors, 2012). We also examined the effect of temperature on replication of EHDV-7 in C. sonorensis and demonstrated that the time to detection of potentially competent midges decreased with increasing temperature (Ruder et al. in preparation). Although multiple attempts were made, we failed to evaluate wild-caught Culicoidesinsignisas a potential vector for EHDV-7; however, our finding that C. sonorensis is a competent vector is far more significant because this species is widespread in the U.S. As for Israeli Culicoides spp. the main species caught near farms affected during the outbreaks were C. imicolaand C. oxystoma. The vector competence studies performed in Israel were in a smaller scale than in the US due to lack of a laboratory colony of these species and due to lack of facilities to infect animals with vector borne diseases. However, we found both species to be susceptible for infection by EHDV. For C. oxystoma, 1/3 of the Culicoidesinfected were positive 11 days post feeding. 3. To identify the host and environmental factors influencing the level of exposure to EHDV, its spread and its associated morbidity: Analysis of the cattle morbidity in Israel showed that the disease resulted in an average loss of over 200 kg milk per cow in herds affected during September 2006 and 1.42% excess mortality in heavily infected herds (Kedmi et al. Journal of Dairy Science, 2010). Outbreak investigation showed that winds played a significant role in virus spread during the 2006 outbreak (Kedmi et al. Preventive Veterinary Medicine, 2010). Further studies showed that both sheep (Kedmi et al. Veterinary Microbiology, 2011) and wild ruminants did not play a significant role in virus spread in Israel (Kedmi et al. article in preparation). Clinical studies in WTD showed that this species is highly susceptibile to EHDV-7 infection and disease (Ruder et al. Journal of Wildlife Diseases, 2012). Experimental infection of Holstein cattle (cows and calves) yielded subclinical viremia (Ruder et al. in preparation). The findings of this study, which resulted in 6 articles, published in peer reviewed journals and 4 more articles which are in preparation, contributed to the dairy industry in Israel by defining the main factors associated with disease spread and assessment of disease impact. In the US, we demonstrated that sufficient conditions exist for potential virus establishment if EHDV-7 were introduced. The significant knowledge gained through this study will enable better decision making regarding prevention and control measures for EHDV and similar viruses, such as BTV.
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VanderNoot, Victoria A., Deanna Joy Curtis, Chung-Yan Koh, Benjamin H. Brodsky, and Todd Lane. Enhanced vector borne disease surveillance of California Culex mosquito populations reveals spatial and species-specific barriers of infection. Office of Scientific and Technical Information (OSTI), August 2014. http://dx.doi.org/10.2172/1154713.

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Delivering Climate Agenda for LAC: IDB Group Actions to 2020. Inter-American Development Bank, December 2017. http://dx.doi.org/10.18235/0006053.

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Climate change presents challenges to ensuring global sustainable and inclusive development. The United Nations Framework Convention on Climate Change (UNFCCC) Paris Agreement and the new Sustainable Development Goals (SDGs) reflect an unprecedented level of commitment to deliver sustainable development to meet these challenges. The transformation of the global economy needs to start immediately if we are to meet the Paris objective for keeping global temperature rise well below 2°C by the end of the century, which most estimate will mean achieving net zero greenhouse gas (GHG) emissions by 2050. The Agreement recognizes the need to achieve zero net emissions, calls for the alignment of all financial flows to a pathway for low-carbon and climate-resilient development, and urges industrial countries to jointly increase climate finance to US$100 billion per year by 2020. Latin America and the Caribbean (LAC) is particularly vulnerable to the effects of climate change such as sea level rise, natural disasters, water and vector-borne diseases, and changing precipitation and temperature patterns that jeopardize health, productivity, and food-producing crops. These impacts threaten poverty, inequality reduction and sustainable growth in the region. In the context of the Paris Agreement, 25 of the Inter-American Development Bank Group’s (IDBG) 26 borrowing member countries have submitted Intended Nationally Determined Contributions or Nationally Determined Contributions (referred to as NDCs throughout this document), that include emissions reduction commitments and climate resilience plans (as of September 2017). Progress toward implementation of pledges will determine the rate at which the world will be able to act to tackle climate change.
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