Relevant bibliographies by topics / Vasomotor reactivity

Academic literature on the topic 'Vasomotor reactivity'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Vasomotor reactivity"

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Friedman, E. H. "Vasomotor reactivity." Neurology 45, no. 11 (November 1, 1995): 2115. http://dx.doi.org/10.1212/wnl.45.11.2115.

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Brothers, R. Matthew, Chansol Hurr, Kiyoung Kim, Joshua F. Lee, and Rong Zhang. "Cerebral Vasomotor Reactivity." Medicine & Science in Sports & Exercise 46 (May 2014): 13. http://dx.doi.org/10.1249/01.mss.0000493201.13477.46.

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Rogers, Robert L., John S. Meyer, Karl F. Mortel, Roderick K. Mahurin, and John Thornby. "Age-Related Reductions in Cerebral Vasomotor Reactivity and the Law of Initial Value: A 4-Year Prospective Longitudinal Study." Journal of Cerebral Blood Flow & Metabolism 5, no. 1 (March 1985): 79–85. http://dx.doi.org/10.1038/jcbfm.1985.11.

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A group of 51 neurologically normal, middle-aged and elderly volunteers (aged 35–86 years; mean age 63.24 years) with and without risk factors for stroke were given annual tests of cerebral vasomotor reactivity to assess any changes in the cerebral vascular capacitance associated with advancing age that might alter cerebral vasomotor reactivity. Cerebral vasomotor reactivity was estimated as the difference in bihemisphere gray matter CBF measured by the 133Xe inhalation method in the steady state breathing room air, followed by a second measurement during inhalation of 100% oxygen. There were significant and progressive reductions in cerebral vasomotor reactivity during the 4-year longitudinal study. Positive linear correlations were apparent between initial steady-state mean bihemisphere gray matter CBF levels and degrees of vasomotor reactivity, suggesting that the Law of Initial Value plays an important role. This should be borne in mind when analyzing scores of cerebral vasomotor reactivity. In the present communication, analysis of covariance was used to correct for influences of initial CBF levels on vasomotor responses tested while breathing pure oxygen.
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DENOVELLIS, V. "Aging and vasomotor reactivity." Pharmacological Research 26 (September 1992): 14. http://dx.doi.org/10.1016/1043-6618(92)90739-x.

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Ribigan, A. C., F. A. Antochi, E. O. Terecoasa, M. Popa, O. Rusu, C. Coclitu, A. Ciobotaru, C. Tiu, and O. A. Bajenaru. "CEREBRAL VASOMOTOR REACTIVITY IN PATIENTS WITH ARTERIAL HYPERTENSION AND COGNITIVE IMPAIRMENT." Romanian Journal of Neurology 15, no. 4 (December 31, 2016): 168–73. http://dx.doi.org/10.37897/rjn.2016.4.4.

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Objectives. The aim of our study is to determine whether cerebral vasomotor reactivity is impaired in patients with arterial hypertension and cognitive impairment and how this hemodynamic parameter is associated with different functions of cognition. Materials and methods. We included 87 patients with arterial hypertension divided into two groups, one with neurocognitive impairment ranging from mild to severe aged between 47 and 90 years (70.2 ± 11.4) and the second group without cognitive impairment aged between 41 and 86 years (60.1 ± 11.4). We excluded patients with significant hemodynamic cervico-cerebral arterial stenoses, arrhythmias and other diseases that may impair cerebral vasomotor reactivity. All the patients underwent assessment of vasomotor reactivity and neurocognitive functions. Results. BHI values were significantly lower in the first group of patients compared to the second one. The percent of patients with impaired cerebral vasomotor reactivity was significantly higher in the group of patients with cognitive impairment, as compared to the other group (54.35% vs 29.27%, p=0.01). There was a significant statistical difference between the MMSE, MOCA and clock test scores among patients with and without impaired vasomotor reactivity. This difference was also maintained for visuospatial/executive, naming and language domains of the MOCA test. Conclusions. Impaired cerebral vasomotor reactivity is more frequent in patients with arterial hypertension and cognitive impairment. Patients with arterial hypertension and impaired vasomotor reactivity have poorer cognitive performance, cognitive functions most affected in patients with impaired vasomotor reactivity being language, visuospatial and executive ones.
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Lucic-Prokin, Aleksandra, Petar Slankamenac, and Pavle Kovacevic. "Transcranial doppler methods in the assessment of cerebral vasomotor reactivity." Medical review 73, no. 1-2 (2020): 21–28. http://dx.doi.org/10.2298/mpns2002021l.

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Introduction. Transcranial Doppler is the only non-invasive neuroimaging modality in the diagnosis and monitoring of various neurovascular diseases. Apart from assessing cerebral hemodynamics of blood flow in the basal brain arteries, transcranial Doppler provides physiological data and anatomical images. Quantification analysis of vasomotor reactivity. Various transcranial Doppler methods evaluate cerebral vasomotor reactivity, providing important information on the properties of arterioles under induced hemodynamic conditions. Exogenous and endogenous vasoactive stimuli of different potency (apnea, acetazolamide, carbon dioxide, L-arginine) are most commonly used, making transcranial Doppler a prognostic indicator of future ischemic events. This article reviews principles of various transcranial Doppler methods in the evaluation of vasomotor reactivity, emphasizing their advantages and disadvantages. Transcranial Doppler in the field of reduced vasomotor reactivity. Evaluation of vasomotor reactivity has a role in the prediction of future ischemic events, evaluation of revascularization effect after carotid endarterectomy, but also in the increasingly significant choice of the right time to perform it. In recent years, transcranial Doppler methods have found application in other areas of dysfunctional cerebral hemodynamics: dementia, hypertension, migraines, and sepsis. Conclusion. Due to an excellent temporal resolution, non-invasive approach, good cost-benefit ratio, bedside monitoring, relative simplicity in terms of interpretation and performance, and portability, transcranial Doppler in vasomotor reactivity may be the ideal tool in the evaluation of cerebral hemodynamics, arterial perfusion integrity and collateral capacity.
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Carod-Artal, Francisco Javier. "Statins and Cerebral Vasomotor Reactivity." Stroke 37, no. 10 (October 2006): 2446–48. http://dx.doi.org/10.1161/01.str.0000239656.59618.d4.

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Kaşıkç, Mehmet Tayfun, and Güray Koç. "Vasomotor reactivity in the ophthalmic artery." Gulhane Medical Journal 62, no. 1 (March 13, 2020): 33–37. http://dx.doi.org/10.4274/gulhane.galenos.2019.784.

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Giannopoulos, S., B. Boden-Albala, J. H. Choi, E. Carrera, M. Doyle, T. Perez, and R. S. Marshall. "Metabolic syndrome and cerebral vasomotor reactivity." European Journal of Neurology 17, no. 12 (November 18, 2010): 1457–62. http://dx.doi.org/10.1111/j.1468-1331.2010.03087.x.

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Ameriso, S. F., J. G. Mohler, M. Suarez, and M. Fisher. "Morning reduction of cerebral vasomotor reactivity." Neurology 44, no. 10 (October 1, 1994): 1907. http://dx.doi.org/10.1212/wnl.44.10.1907.

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Dissertations / Theses on the topic "Vasomotor reactivity"

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Beltrame, John Francis. "Vasomotor reactivity studies of small and large coronary arteries /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phb453.pdf.

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Ohtani, Ryo. "Cerebral vasomotor reactivity to postural change is impaired in patients with cerebrovascular white matter lesions." Kyoto University, 2004. http://hdl.handle.net/2433/147505.

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Beltrame, John Francis. "Vasomotor reactivity studies of small and large coronary arteries / John Francis Beltrame." Thesis, 1998. http://hdl.handle.net/2440/19544.

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Errata and corrigenda inserted at end of thesis.
Bibliography: leaves 290-337.
xxii, 337 leaves : ill. ; 30 cm.
The objective of this thesis is to examine vasomotor reactivity in both large and small arterial vessels utilising both basic and clinical models. In 4 sections, the thesis: 1. summarises fundamental morphological and physiological principles of the coronary circulation, methods of assessing coronary vasomotor reactivity and characteristics of clinical disorders with vasomotor dysfunction ; 2. investigates regional heterogeneity of vasomotor reactivity in sheep epicardial coronary arteries ; 3. involves studies of the coronary flow phenomenon ; 4. involves a comparison of published Japanese and Caucasian coronary vasomotor reactivity studies and 5. involved an observational study of patients presenting with acute ST elevation.
Thesis (Ph.D.)--University of Adelaide, Dept. of Medicine, 2000
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Jaghoori, Amenah. "Sex-dependent differences in vasomotor responses of older male and female humans." Thesis, 2015. http://hdl.handle.net/2440/95310.

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Background and Aims: Sex differences have been observed in several cardiovascular diseases, in terms of mortality and morbidity. Female patients experience worse clinical outcomes than their male counterparts. Although multiple mechanisms may be involved, sex differences in vascular reactivity of large and small blood vessels have not been investigated. This thesis aims to assess sex-dependent difference in vasoconstrictor responses of human vessels isolated from a variety of vascular beds from older patients (mean age 68 years) with and without existing coronary artery disease. Specific aims include evaluation of :(1a) sex differences in vascular responses of internal mammary artery (IMA) and saphenous vein (SV) segments from male and female patients undergoing CABG and (1b) mechanisms underlying sex dependent vascular responses. (2) sex differences in microvascular reactivity of vessels isolated from mediastinal and peripheral subcutaneous areas in patients with CAD. (3a) sex difference in vascular reactivity of subcutaneous microvessels from patients with no known CAD, undergoing elective non-cardiac surgery. (3b) subcutaneous microvascular reactivity of males and females patients with CAD to those without known CAD. Methods: This thesis used wire myography technique to assess functional changes in vasoconstrictor responses of isolated large conduit and small blood vessels. Concentration-response curves were formed for various vasoconstrictors including phenylephrine, serotonin, endothelin-1 and the thromboxane mimetic, U46619. Western blot analysis was employed to measure the biochemical parameters, including receptor abundance endothelin-1. Summary of major findings: Female IMA segments display hypersensitive responses to serotonergic and α₁-adrenergic receptor stimulation, compared to males. Blocking eNOS and/or cyclooxygenase revealed that prostaglandins account for in the observed α₁-adrenergic mediated sex differences. Biochemical analysis revealed increased density of 5HT2A [2A in subscript] receptors in the female IMA. Similar sex differences were observed in the pericardial microvessels of the same patient cohort, with females showing increased sensitivity to serotonergic and α₁-adrenergic receptor stimulation. Interestingly, no sex differences were observed in the peripheral subcutaneous microvessels of patients with existing CAD. In patients without known CAD, female subcutaneous microvessels were hypersensitive to serotonergic and α₁-adrenergic receptor stimulation, compared to matched males. When compared to subcutaneous microvessels of male and female patients without known CAD, male and female CAD patients exhibited increased sensitivity to a1- adrenergic agonist. Male CAD patients were also hypersensitive to serotonin and the thromboxane A₂ mimetic, U46619, relative to those without known CAD. Conclusions: For the first time, in a population cohort with a mean age of 68 years, female vascular hyper-reactivity in both large graft arteries (IMA) and microvessels has been demonstrated. Female vascular hypersensitivity is consistently seen in response to serotonergic and α₁-adrenergic receptor agonist. In part, this may be due to sex-differences in prostanoid activity. The IMA hyper-reactivity in the group of older women may contribute to their poorer outcomes following CABG and microvascular differences amongst patients without documented cardiovascular disease may pre-dispose them to hypertension.
Thesis (Ph.D.) -- University of Adelaide, School of Medicine, 2015
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Ku, ByungMo. "The effect of acute consumption of a flavonol-rich cocoa drink on cerebral vasomotor reactivity in African Americans." Thesis, 2014. http://hdl.handle.net/2152/26359.

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African Americans (AA) are at great risk of cardiovascular diseases (CVD) which can lead to brain damage, dementia, and endothelial dysfunction. Decreased nitric oxide (NO) bioavailability contributes cardiovascular disease in AA population. Flavonols of the subclass known as flavonoids that have several beneficial effects on cerebral blood flow and cerebral vasomotor reactivity (CVMR). This study investigated the effects of the acute consumption of a flavanol-rich cocoa drink on CVMR. Ten non-smoking African American (6 males and 3 females) participants were randomly recruited. The subjects participated in two experimental sessions which were separated before and after the consumption of cocoa drink. For the pre-session, baseline CVMR was measured by the hypercapnia rebreathing (CVMR test) prior to the consumption of the cocoa drink and the again at 2h after consumption of one serving of the cocoa drink (45g of cocoa mixed with 8oz of cold water). Cerebral vascular conductance (CVC) was significantly increased in the post-study during hypercapnia rebreathing compared with the pre-study(post-study: 3.649 ± 1.833 CVC % of baseline/mmHg, pre-study: 2.483 ± 1.418 CVC % of baseline/mmHg vs. P < 0.05) Thus, CVMR was significantly increased in the post-study after the acute consumption of a flavonol-rich cocoa drink compared to the pre-study in AA.
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Lee, E.-Jian, and 李宜堅. "Cerebral Blood Flow Velocity and Vasomotor Reactivity before and after Shunting Surgery in Patients with Normal Pressure Hydrocephalus." Thesis, 1997. http://ndltd.ncl.edu.tw/handle/48826035231997811722.

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碩士
國立成功大學
醫學工程學系
86
ABSTRACTOBJECTIVE:The aim of the study was to evaluate the hemodynamic changes and their correlationwith the clinical results by measurement of cerebral blood flow velocity (CBFV) andcerebral vasomotor reserve before and after shunt placement in normal pressurehydrocephalus (NPH).METHODS:Ten demented patients with clinical signs suggestive of NPH received examinationsof blood flow velocity (BFV) and vasomotor reactivity (VMR) of the anterior cerebralartery (ACA) and the middle cerebral artery (MCA) by transcranial Doppler sonographywith carbogen testing before and after shunt treatment. Computerized tomography (CT),clinical assessment and neuropsychological grading were performed prior to and at 3months following surgery. A control group consisting of 7 patients, who underwentlumbar spine surgery, was included to establish baseline data for BFV and VMR values.RESULTS:Compared to the control group, the preoperative CBF studies revealed the NPH patienthad no significant decrease of BFVs in both the MCA and the ACA (P > 0.05), but hadsignificant decrease of carbogen VMR in both those two vessels (P < 0.05). Aftershunting, there were no significant changes of the BFVs in the 2 vessels as comparedto the pre-shunting data (P > 0.05). The post-shunting VMR of ACA was significantlyhigher than the pre-shunting one (p < 0.05), but there was no significance in thatof MCA (p > 0.05). Seven of the ten patients shown mentality or more symptomsimprovement were considered as good results (responsive to shunt). The remaining 3patients, who had consistent symptoms without recognizable problems, were consideredas bad results (shunt failure). Both the value of post-shunting VMR in ACA and thepost-shunting improvement of VMR in MCA of the 7 shunt- responsive patients weresignificantly higher than those of shunt-failure patients (p < 0.05). Within the tenpatients, five patients with gait improvement showed significantly in the value ofpost-shunting VMR of ACA and the post-shunting improvement of VMR for both ACA andMCA by comparing those patients without gait improvement (p < 0.05, respectively).CONCLUSION:Our results support that the patient with NPH did not have decreased BFVs, but hadvarious degrees of impaired vasomotor reserve in both the ACA and the MCA, increasingthe risk of ischemic brain insult. Shunt placement improves the VMR in responsivepatients, consequently preventing from the ischemic insult. Post-shunting increaseof VMR accompanies with the improvement of functional state in shunt-responsivepatients; however, post-shunting increase of VMR in the MCA only, and in both the ACAand the MCA are associated with symptomatic improvement in mentality, and improvementin gait, respectively. These close relationships have implications for prognosticimportance and pathophysiology in NPH.Key Words: Normal pressure hydrocephalus, Cerebral blood flow velocity, Vasomotorreactivity.
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Li, Yi-Jian, and 李宜堅. "Cerebral Blood Flow Velocity and Vasomotor Reactivity before and after Shunting Surgery in Patients with Normal Pressure Hydrocephalus." Thesis, 1997. http://ndltd.ncl.edu.tw/handle/24831229639771129866.

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Juan, Chun-Jung, and 阮春榮. "Impact of Inhaled CO2 on fMRI Experiments and Measurements of Cerebral Vasomotor Reactivity, Cerebral Blood Flow, Cerebral Blood Volume and Cerebral Blood Oxygenation." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/86259081192540330716.

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博士
國立臺灣大學
電機工程學研究所
95
The neural activity involves energy demand and energy supply. During neural activation, the energy source glucose and oxygen are supplied by increase cerebral blood flow via a regulation of the cerebral microcirculatory units. In addition to neural activation, the cerebral microcirculation is regulated by many other factors including CO2. It is very likely that the neural activation related cerebral microcirculatory regulation is overwhelmed by the external CO2 perturbation. In this thesis, we explore the effect of inhaled CO2 on cerebral vascular and microcirculatory regulations and on the visual fMRI study. Specially, we propose a hybrid pulse sequence that allows accurate measurement of cerebral blood volume, cerebral blood flow and cerebral blood oxygenation simultaneously. Experimental results are consistent with prior fMRI and PET studies and the basic understanding of cerebral microcirculatory regulation on physiology.
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Juan, Chun-Jung. "Impact of Inhaled CO2 on fMRI Experiments and Measurements of Cerebral Vasomotor Reactivity, Cerebral Blood Flow, Cerebral Blood Volume and Cerebral Blood Oxygenation." 2007. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-2406200723165800.

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Hurr, Chansol. "Impaired cerebral vascular function in college-aged African Americans and Caucasian Americans : potential role of Vitamin D and arterial stiffness." 2013. http://hdl.handle.net/2152/21800.

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African Americans have increased risk for cardiovascular and cerebral vascular disease relative to Caucasian Americans. While it is generally accepted that arteries become stiffer at a younger age in African Americans; less is known regarding cerebral vascular function / reactivity (CVMR) to hypercapnia in African Americans. Furthermore, little is known regarding the relationship between arterial stiffness and CVMR, particularly in young healthy adults. We hypothesized that African Americans have stiffer arteries (i.e. arterial stiffness) and reduced CVMR during hypercapnia relative to Caucasian Americans. We also hypothesized that there would be a negative relationship between arterial stiffness and CVMR. Lastly, we hypothesized that these responses would be related to a decrease in Vitamin D status in this population and there would be correlation between Vitamin D status and CVMR. In 11 African American and 19 Caucasian American subjects central arterial stiffness was indexed from carotid-femoral pulse wave velocity (PWV). CVMR was assessed by the cerebral vascular conductance (CVC) response to rebreathing-induced hypercapnia. Vitamin D status was assessed from plasma 25(OH) Vitamin D. PWV was elevated in the African Americans (African American: 581.16 ± 27.7 cm/sec vs. Caucasian American: 502.98 ± 17.6 cm/sec; P < 0.01). CVMR was significantly reduced during hypercapnic rebreathing in the African Americans (African American: 3.05 ± 0.38% of baseline/mmHg vs. Caucasian American: 5.09 ± 0.29% of baseline/mmHg; P < 0.001). When data from all subjects was included there was a trend towards a negative relationship (R = 0.32, P = 0.10) between PWV and CVMR. Vitamin D status was significantly lower in African Americans (African American: 14.96 ± 0.97 ng/ml vs. Caucasian American: 32.73 ± 0.99 ng/ml; P < 0.001); however, there was no significant relationship between Vitamin D status and CVMR (R = 0.23 P = 0.23). In conclusion, these data indicate that African Americans have impaired cerebral vascular responses to hypercapnia, stiffer arteries, and lower Vitamin D status when compared with Caucasian Americans. In addition, there may be a negative relationship between CVMR and PWV; however, no significant correlation between Vitamin D status and vascular function including PWV or CVMR was observed in this study.
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Books on the topic "Vasomotor reactivity"

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Ouimet, Terry M. Effects of aerobic exercise training on cardiovascular reactivity to psychological stress in adolescent males. 1995.

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The effects of aerobic exercise on cardiovascular reactivity and baroreflex response in women with parental history of hypertension. 1993.

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Book chapters on the topic "Vasomotor reactivity"

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Aaslid, Rune. "Cerebral Autoregulation and Vasomotor Reactivity." In Handbook on Neurovascular Ultrasound, 216–28. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000092434.

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Rinsky, Brenda. "Transcranial Doppler Protocols and Procedures: Vasomotor Reactivity." In Neurovascular Sonography, 229–39. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-96893-9_15.

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Castro, Pedro, and Elsa Azevedo. "Acute Neurologic Injury in ICU: Vasomotor Reactivity Testing by Transcranial Doppler (TCD/TCCS)." In Neurosonology in Critical Care, 333–40. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81419-9_19.

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Wolf, Marc E. "Functional TCD: Regulation of Cerebral Hemodynamics - Cerebral Autoregulation, Vasomotor Reactivity, and Neurovascular Coupling." In Translational Neurosonology, 40–56. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000366236.

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Ringelstein, E. B., W. Grosse, A. Mauckner, R. Schneider, W. M. Glöckner, and S. Matentzoglu. "Do Hemorheological Properties Influence Vasomotor Reactivity of the Brain Arteries? A Transcranial Doppler Study During Hypo-, Normo- and Hypercapnia." In Cerebral Ischemia and Hemorheology, 452–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71787-1_54.

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Conference papers on the topic "Vasomotor reactivity"

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Zirak, Peyman, Raquel Delgado-Mederos, Lavinia Dinia, Joan Martí-Fábregas, and Turgut Durduran. "Cerebral vasomotor reactivity in micro- and macro-vasculature of patients with severe steno-occlusive internal carotid artery lesions." In Biomedical Optics. Washington, D.C.: OSA, 2012. http://dx.doi.org/10.1364/biomed.2012.jm3a.28.

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Chen, Ching-Kun, Shoou-Jeng Yeh, Shyan-Lung Lin, Yu-Liang Hsu, and Hsing-Cheng Chang. "Innovated noninvasive assessment of Parkinson's disease based on measurement of cerebral vasomotor reactivity with transcranial Doppler ultrasonography and capnography." In 2018 IEEE International Conference on Applied System Innovation (ICASI). IEEE, 2018. http://dx.doi.org/10.1109/icasi.2018.8394519.

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