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Journal articles on the topic 'Vasodilatace'

Author: Grafiati

Published: 28 June 2021

Last updated: 14 February 2022

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1

Liu, Qian, Ling Zhang, Qiyuan Shan, Yuxia Ding, Zhaocai Zhang, Meifei Zhu, and Yuanjie Mao. "Total flavonoids from Astragalus alleviate endothelial dysfunction by activating the Akt/eNOS pathway." Journal of International Medical Research 46, no. 6 (August 31, 2017): 2096–103. http://dx.doi.org/10.1177/0300060517717358.

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Objective To investigate the vasodilative and endothelial-protective effects and the underlying mechanisms of total flavonoids from Astragalus (TFA). Methods The vasodilative activities of TFA were measured with a myograph ex vivo using rat superior mesenteric arterial rings. The primary human umbilical vein endothelial cell (HUVEC) viabilities were assayed using the cell counting kit-8 after hypoxia or normoxia treatment with or without TFA. Akt, P-Akt, eNOS, P-eNOS, Erk, P-Erk, Bcl-2 and Bax expression were analyzed using western blotting. Results TFA showed concentration-dependent vasodilative effects on rat superior mesenteric arterial rings, but had no effects on normal or potassium chloride precontracted arterial rings. TFA did not affect HUVEC viabilities in normoxia, but dramatically promoted cell proliferation in the concentration range of 1 to 30 µg/mL under hypoxia. Moreover, TFA significantly increased the ratios of P-Akt/Akt and P-eNOS/eNOS in vascular endothelial cells under hypoxic conditions, but did not change the P-Erk/Erk or Bcl-2/Bax ratios. Conclusions TFA might exhibit vasorelaxant and endothelial-protective effects via the Akt/eNOS signaling pathway.

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2

Zhilinskaya, I. N., V. G. Platonov, I. P. Ashmarin, and O. I. Kiselev. "Antiviral activity of some vasodilative preparations." Bulletin of Experimental Biology and Medicine 121, no. 2 (February 1996): 154–56. http://dx.doi.org/10.1007/bf02446623.

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3

Warner, Eve L., Franco Galasso, Carl I. Thompson, and Francis L. Belloni. "Vasodilative and anti-adrenergic effects of adenosine in diabetic rat hearts." Canadian Journal of Physiology and Pharmacology 70, no. 1 (January 1, 1992): 13–19. http://dx.doi.org/10.1139/y92-003.

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To determine the vasodilative and negative inotropic effects of adenosine in hearts of diabetic rats, isolated hearts, perfused at constant perfusion pressure (Langendorff technique), were prepared from age-matched control Wistar rats and rats made diabetic 10 weeks prior to study by a single injection of streptozotocin (65 mg∙kg−1, i.p.). Adenosine and nitroprusside each increased coronary inflow when administered either as bolus injections or as infusions. Coronary flow responses to nitroprusside were unchanged in diabetic hearts. Coronary flow responses of diabetic hearts to adenosine injections were unchanged, but responses to adenosine infusions tended to be larger than in normal hearts. Diabetes had no significant effect on the EC50 for either vasodilator. Adenosine inhibited the inotropic effect of isoproterenol (enhanced left ventricular (LV) pressure (P) and LV dP/dtmax) in normal hearts, independently of its vasodilative action. This negative inotropic action of adenosine appeared equally strong in diabetic hearts. We conclude that adenosine's coronary vasodilative and anti-β-adrenergic, negative inotropic effects in the rat heart were not diminished after 10 weeks of streptozotocin-induced diabetes mellitus. Thus, earlier reports of diminished adenosine dilative efficacy in experimental diabetes may have been unique to those particular models.Key words: experimental diabetes mellitus, coronary, adenosine, isoproterenol, myocardial contraction.

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4

Cupp, Mary S., Jose M. C. Ribeiro, and Eddie W. Cupp. "Vasodilative Activity in Black Fly Salivary Glands." American Journal of Tropical Medicine and Hygiene 50, no. 2 (February 1, 1994): 241–46. http://dx.doi.org/10.4269/ajtmh.1994.50.241.

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5

Takagi, S., H. Goto, Y. Shimada, K. Nakagomi, Y. Sadakane, Y. Hatanaka, and K. Terasawa. "Vasodilative effect of perillaldehyde on isolated rat aorta." Phytomedicine 12, no. 5 (May 2005): 333–37. http://dx.doi.org/10.1016/j.phymed.2003.08.004.

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6

Grimminger, F., H. Olschewski, F. Rose, H. A. Ghofrani, N. Weissmann, R. T. Schermuly, D. Walmrath, and W. Seeger. "Schwere pulmonale Hypertonie - Vasodilative Therapie in der Lungenstrombahn." Pneumologie 54, no. 4 (April 2000): 160–69. http://dx.doi.org/10.1055/s-2000-9083.

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7

Bi, Yue-Feng, Hai-Wei Xu, Xiao-Qing Liu, Xiao-Juan Zhang, Zhen-Ji Wang, and Hong-Min Liu. "Synthesis and vasodilative activity of tanshinone IIA derivatives." Bioorganic & Medicinal Chemistry Letters 20, no. 16 (August 2010): 4892–94. http://dx.doi.org/10.1016/j.bmcl.2010.06.076.

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8

Rosenberry, Ryan, Darian Trojacek, Susie Chung, Daisha J. Cipher, and Michael D. Nelson. "Interindividual differences in the ischemic stimulus and other technical considerations when assessing reactive hyperemia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 317, no. 4 (October 1, 2019): R530—R538. http://dx.doi.org/10.1152/ajpregu.00157.2019.

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Reactive hyperemia is an established, noninvasive technique to assess microvascular function and a powerful predictor of all-cause and cardiovascular morbidity and mortality. Emerging evidence from our laboratory suggests a close link between reactive hyperemia and the metabolic rate of the ischemic limb and the existence of large interindividual differences contributing to markedly different stimuli to vasodilate. Here we relate forearm tissue desaturation (i.e., the ischemic stimulus to vasodilate, measured by near-infrared spectroscopy) to brachial artery hyperemic velocity and flow (measured using duplex ultrasound) across a wide range of ischemic stimuli. Twelve young and 11 elderly individuals were prospectively studied. To recapitulate conventional vascular occlusion testing, reactive hyperemia was first assessed using a standard 5-min occlusion period. Then, to evaluate the dose dependence of tissue ischemia on reactive hyperemia, we randomly performed 4-, 6-, and 8-min cuff occlusions in both groups. In all cases, peak velocity, as well as the 5-s average velocity, immediately after the cuff occlusion was significantly higher in the young than the elderly group; however, tissue desaturation was also much more pronounced in the young group ( P < 0.05), representing a greater ischemic stimulus. Remarkably, when reactive hyperemia was adjusted for the ischemic vasodilatory stimulus, group differences in reactive hyperemia were abrogated. Together, these data challenge conventional interpretations of reactive hyperemia and show that the ischemic stimulus to vasodilate varies across individuals and that the level of reactive hyperemia is often coupled to the magnitude of tissue desaturation.

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9

Zegarra, Jaime, Mónica Meza, Carla Cornejo, Willy Porras, Alfredo Díaz, Omar Heredia, Wilson Rojas, et al. "Amlodipino y choque vasodilatado en una unidad de cuidados intensivos de un hospital general. Reporte de caso." Revista Medica Herediana 28, no. 2 (July 4, 2017): 101. http://dx.doi.org/10.20453/rmh.v28i2.3110.

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Los calcio antagonistas son fármacos usados para diferentes patologías médicas; sin embargo la intoxicación puede ser grave. Presentamos el caso de una mujer joven intoxicada por amlodipino quien cursó con choque vasodilatado y disfunción multiorgánica, en quien se usó vasopresores múltiples a dosis por encima de las habituales para estabilizarla.

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10

Leuchte, Hanno H., Jens Michalek, Oezcan Soenmez, Tobias Meis, Shani Haziraj, Vera Cavalli, Dorian Bevec, and Jürgen Behr. "Preserved pulmonary vasodilative properties of aerosolized brain natriuretic peptide." Pulmonary Pharmacology & Therapeutics 22, no. 6 (December 2009): 548–53. http://dx.doi.org/10.1016/j.pupt.2009.07.002.

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11

Bi, Yue-Feng, Zhen-Ji Wang, Ruo-Fei Guan, Yu-Ting Ye, Yuan-Yuan Chen, Yan-Bing Zhang, and Hong-Min Liu. "Design, synthesis and vasodilative activity of tanshinone IIA derivatives." Bioorganic & Medicinal Chemistry Letters 22, no. 15 (August 2012): 5141–43. http://dx.doi.org/10.1016/j.bmcl.2012.05.014.

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12

Park, Kyung Hee, Yoon Hee Kim, Seok Hwa Yoon, Jung Un Lee, Hae Ja Kim, and Sae Jin Choi. "Vasodilative Effects of Propofol on Isolated Pulmonary Artery in Rats." Korean Journal of Anesthesiology 31, no. 6 (1996): 677. http://dx.doi.org/10.4097/kjae.1996.31.6.677.

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13

Iwai, Takeshi, Hisanori Takanashi, Masaaki Kurata, Kentaro Asanuma, and Tadashi Asano. "Vasodilative mechanism of [Leu13]-motilin in canine isolated mesenteric artery." Japanese Journal of Pharmacology 76 (1998): 145. http://dx.doi.org/10.1016/s0021-5198(19)40688-4.

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14

Leuchte, Hanno H., Carlos Baezner, Rainer A. Baumgartner, Olaf Muehling, Claus Neurohr, and Juergen Behr. "Residual pulmonary vasodilative reserve predicts outcome in idiopathic pulmonary hypertension." Heart 101, no. 12 (May 25, 2015): 972–76. http://dx.doi.org/10.1136/heartjnl-2015-307529.

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15

MIZUTANI, NOBORU. "Longterm effect of vasodilative .BETA. blockers on congestive heart failures." Rinsho yakuri/Japanese Journal of Clinical Pharmacology and Therapeutics 29, no. 1/2 (1998): 275–76. http://dx.doi.org/10.3999/jscpt.29.275.

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16

Nakamura, Kumi, Hiroshi Toda, Kiyoshi Terasako, Masahiro Kakuyama, Yoshio Hatano, Kenjiro Mori, and Kenji Kangawa. "Vasodilative Effect of Adrenomedullin in Isolated Arteries of the Dog." Japanese Journal of Pharmacology 67, no. 3 (1995): 259–62. http://dx.doi.org/10.1254/jjp.67.259.

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17

Mercuri, M., R. Tang, M. Espeland, R. Byington, M. G. Bond, and N. Borhani. "Does isradipine vasodilate common carotid arteries? the MIDAS database." Atherosclerosis 109, no. 1-2 (September 1994): 294. http://dx.doi.org/10.1016/0021-9150(94)94176-9.

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18

Radharani, Radhika. "Kompres Jahe Hangat dapat Menurunkan Intensitas Nyeri pada Pasien Gout Artritis." Jurnal Ilmiah Kesehatan Sandi Husada 11, no. 1 (June 30, 2020): 573–78. http://dx.doi.org/10.35816/jiskh.v11i1.349.

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Latar Belakang: Gout artritis adalah suata proses peradangan karena terjadinya peradangan di sekitar sendi. Salah satu terapi non-farmakologi pada pasien gout artritis adalah dengan memberikan kompres jahe hangat. Efek panas dari kompres jahe hangat akan menyebabkan vasodilatasi pembuluh darah dan meningkatkan aliran darah ke tubuh dengan rasa sakit yang mengakibatkan penurunan rasa sakit Tujuan : mengetahui lebih lanjut tentang kompres jahe hangat yang dapat menurunkan intensitas nyeri pada pasien gout artritis. Metode : Menggunakan studi literatur dari jurnal baik nasional maupun internasional dengan cara meringkas topic pembahasan dan membandingkan hasil yang disajikan dalam artikel. Hasil : Kompres jahe hangat dapat mengurangi nyeri pada gout artritis. Kompres jahe hangat adalah pengobatan tradisional atau terapi alternatif untuk mengurangi nyeri gout artritis. Jahe mengandung enzim siklo-oksigenasi yang dapat mengurangi peradangan pada pasien dengan gout artritis , selain itu jahe juga memiliki efek farmakologis berupa sensasi panas dan pedas, di mana panas ini dapat meredakan rasa sakit, kekakuan dan kejang otot atau terjadinya vasodilatasi pembuluh darah, manfaat maksimal akan dicapai dalam waktu 20 menit setelah aplikasi kompres jahe hangat di lokasi nyeri. Kesimpulan : Kompres jahe hangat dapat mengurangi nyeri radang pada pasien gout artritis. Kompres jahe adalah pengobatan tradisional atau terapi alternatif untuk mengurangi nyeri radang sendi gout. Kompres jahe hangat mengandung enzim siklooksigenase yang dapat mengurangi peradangan dan nyeri pada penderita gout artritis

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19

Uchida, Sae, and Harumi Hotta. "Acupuncture Affects Regional Blood Flow in Various Organs." Evidence-Based Complementary and Alternative Medicine 5, no. 2 (2008): 145–51. http://dx.doi.org/10.1093/ecam/nem051.

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In this review, our recent studies using anesthetized animals concerning the neural mechanisms of vasodilative effect of acupuncture-like stimulation in various organs are briefly summarized. Responses of cortical cerebral blood flow and uterine blood flow are characterized as non-segmental and segmental reflexes. Among acupuncture-like stimuli delivered to five different segmental areas of the body; afferent inputs to the brain stem (face) and to the spinal cord at the cervical (forepaw), thoracic (chest or abdomen), lumbar (hindpaw) and sacral (perineum) levels, cortical cerebral blood flow was increased by stimuli to face, forepaw and hindpaw. The afferent pathway of the responses is composed of somatic groups III and IV afferent nerves and whose efferent nerve pathway includes intrinsic cholinergic vasodilators originating in the basal forebrain. Uterine blood flow was increased by cutaneous stimulation of the hindpaw and perineal area, with perineal predominance. The afferent pathway of the response is composed of somatic group II, III and IV afferent nerves and the efferent nerve pathway includes the pelvic parasympathetic cholinergic vasodilator nerves. Furthermore, we briefly summarize vasodilative regulation of skeletal muscle blood flow via a calcitonin gene-related peptide (CGRP) induced by antidromic activation of group IV somatic afferent nerves. These findings in healthy but anesthetized animals may be applicable to understanding the neural mechanisms improving blood flow in various organs following clinical acupuncture.

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20

Matsunaga, Kimihiro, Masaoki Shibuya, and Yasushi Ohizumi. "Graminone B, a Novel Lignan with Vasodilative Activity from Imperata cylindrica." Journal of Natural Products 57, no. 12 (December 1994): 1734–36. http://dx.doi.org/10.1021/np50114a020.

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21

Seki, Koh-ichi, Yoichi Noya, Yusuke Mikami, Shinji Taneda, Akira K. Suzuki, Yuji Kuge, and Kazue Ohkura. "Isolation and identification of new vasodilative substances in diesel exhaust particles." Environmental Science and Pollution Research 17, no. 3 (June 26, 2009): 717–23. http://dx.doi.org/10.1007/s11356-009-0207-4.

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22

Gansser, Rolf E., Adam Schneeweiss, Marija Weiss, and Kurt F. Bachmann. "Hemodynamic and coronary vasodilative action of two nitroglycerin oral spray formulations." Cardiovascular Drugs and Therapy 4, no. 2 (March 1990): 475–80. http://dx.doi.org/10.1007/bf01857756.

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23

Leuchte, Hanno H., Tobias Meis, Michal El-Nounou, Jens Michalek, and Jürgen Behr. "Inhalation of endothelin receptor blockers in pulmonary hypertension." American Journal of Physiology-Lung Cellular and Molecular Physiology 294, no. 4 (April 2008): L772—L777. http://dx.doi.org/10.1152/ajplung.00405.2007.

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Endothelin 1 (ET-1) is a potent pulmonary vasoconstrictor and mediator of lung diseases. Antagonism of the ET-1-mediated effects has become an important therapeutic approach. ET-1 (A and B) receptors are differentially distributed in the lung vasculature. Whereas the ETA receptors mainly mediate vasoconstriction, the endothelial ETB receptor seems to have vasodilative properties. We sought to determine if antagonism of ET receptors can be achieved by inhalation of specific blockers in a model of ET-1-mediated pulmonary hypertension.

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24

Katzenschlager, Reinhold, Burgmann, Ehringer, and Minar. "Vasospasmus: Eine seltene Komplikation bei Homozystinurie." Vasa 29, no. 1 (February 1, 2000): 84–86. http://dx.doi.org/10.1024/0301-1526.29.1.84.

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We report about a patient suffering from multiple spastic stenoses of varying degree of both iliac arteries and from occlusion of the left tibiofibular trunc. Laboratory investigations revealed increased levels of homocysteine and the diagnosis of homocystinuria was confirmed by fibroblast cell culture. The spasms responded well to vasodilative therapy with nitroglycerine, molsidomine (cGMP mediated) and prostaglandine E1 but not with nifedipine (Ca influx blocker). Our review of literature demonstrated that this arterial spastic abnormality is a very rare complication in patients suffering from homocystinuria.

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25

Gorenflo, Matthias, Herbert E. Ulmer, and Konrad Brockmeier. "Successful closure of the arterial duct in the setting of rubella syndrome." Cardiology in the Young 12, no. 2 (March 2002): 200–202. http://dx.doi.org/10.1017/s1047951102000434.

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A 9-year-old boy, with significant left-to-right shunting across a large duct in the context of rubella syndrome, was tested during catheterization to establish the feasability of occluding the duct with a device. The testing, including temporary closure of the duct and monitoring of pulmonary vascular reactivity to vasodilative substances, lead to the decision to implant an Amplatzer occluder. Sixteen months later, there was no residual shunting across the duct, and pulmonary arterial pressures had normalised. It remains unclear why the patient had not developed irreversible pulmonary vascular disease.

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26

BEYER, M. "P1781 Oestrogen receptor alpha mediates the acute vasodilative effects of 17?-oestradiol." European Heart Journal 24, no. 5 (March 2003): 342. http://dx.doi.org/10.1016/s0195-668x(03)94919-0.

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27

Kitajima, T. "Acute cigarette smoking reduces vasodilative effect induced by sympathetic block in dogs." Regional Anesthesia and Pain Medicine 26, no. 1 (January 2001): 41–45. http://dx.doi.org/10.1053/rapm.2001.16163.

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28

GEWIRTZ, H., R. A. OLSSON, and A. S. MOST. "Role of adenosine in mediating the coronary vasodilative response to acute hypoxia." Cardiovascular Research 21, no. 2 (February 1, 1987): 81–89. http://dx.doi.org/10.1093/cvr/21.2.81.

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29

Kase, Noriko, Hitomi Kubo, Tetsuaki Yamaura, and Haruo Ohnishi. "Vasodilative action of F-0401, a new anti-cerebrodamage agent in vitro." Japanese Journal of Pharmacology 55 (1991): 411. http://dx.doi.org/10.1016/s0021-5198(19)39620-9.

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30

Kitajima, Toshimitsu, Yasuhisa Okuda, Mutsuo Mishio, Shinsuke Hamaguchi, Shigeki Yamaguchi, and Yoshiyuki Kimura. "Acute Cigarette Smoking Reduces Vasodilative Effect Induced by Sympathetic Block in Dogs." Regional Anesthesia and Pain Medicine 26, no. 1 (January 2001): 41–45. http://dx.doi.org/10.1097/00115550-200101000-00010.

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31

Uchida, Sae. "Cholinergic Vasodilative System in the Cerebral Cortex: Effects of Acupuncture and Aging." Journal of Acupuncture and Meridian Studies 7, no. 4 (August 2014): 173–79. http://dx.doi.org/10.1016/j.jams.2014.02.006.

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32

Kang, Bo-Rui, Sen Li, Shuai Mao, Yong-Xiao Cao, and San-Qi Zhang. "Discovery of novel 2-benzylisoquinolin-1(2H)-ones as potent vasodilative agents." Bioorganic & Medicinal Chemistry Letters 25, no. 24 (December 2015): 5808–12. http://dx.doi.org/10.1016/j.bmcl.2015.10.018.

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33

Crystal, George J., Xiping Zhou, Juozas Gurevicius, Edward A. Czinn, M. Ramez Salem, Syed Alam, Agnieszka Piotrowski, and Guochang Hu. "Direct Coronary Vasomotor Effects of Sevoflurane and Desflurane in In Situ Canine Hearts." Anesthesiology 92, no. 4 (April 1, 2000): 1103–13. http://dx.doi.org/10.1097/00000542-200004000-00029.

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Background An extracorporeal system was used to investigate the direct coronary vasomotor effects of sevoflurane and desflurane in vivo. The role of the adenosine triphosphate-sensitive potassium channels (KATP channels) in these effects was evaluated. Methods Twenty-one open-chest, anesthetized (fentanyl-midazolam) dogs were studied. The left anterior descending coronary artery was perfused at controlled pressure (80 mmHg) with normal arterial blood or arterial blood equilibrated with either sevoflurane or desflurane. Series 1 (n = 16) was divided into two groups of equal size on the basis of whether sevoflurane (1.2, 2.4, and 4.8%) or desflurane (3.6, 7.2, and 14.4%) was studied. The concentrations for the anesthetics corresponded to 0.5, 1.0, and 2.0 minimum alveolar concentration (MAC), respectively. Coronary blood flow (CBF) was measured with an ultrasonic, transit-time transducer. Local coronary venous samples were obtained and used to evaluate changes in myocardial oxygen extraction (EO2). In series 2 (n = 5), changes in CBF by 1 MAC sevoflurane and desflurane were assessed before and during intracoronary infusion of the KATP channel inhibitor glibenclamide (100 microg/min). Results Intracoronary sevoflurane and desflurane caused concentration-dependent increases in CBF (and decreases in EO2) that were comparable. Glibenclamide blunted significantly the anesthetic-induced increases in CBF. Conclusions Sevoflurane and desflurane have comparable coronary vasodilative effects in in situ canine hearts. The KATP channels play a prominent role in these effects. When compared with data obtained previously in the same model, the coronary vasodilative effects of sevoflurane and desflurane are similar to those of enflurane and halothane but considerably smaller than that of isoflurane.

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Deem, Steven, Seong Su Kim, Jin-Hye Min, Randy Eveland, Jennifer Moulding, Sabrina Martyr, Xunde Wang, Erik R. Swenson, and Mark T. Gladwin. "Pulmonary vascular effects of red blood cells containing S-nitrosated hemoglobin." American Journal of Physiology-Heart and Circulatory Physiology 287, no. 6 (December 2004): H2561—H2568. http://dx.doi.org/10.1152/ajpheart.00310.2004.

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The role of S-nitrosated hemoglobin (SNO-Hb) in the regulation of blood flow is a central and controversial question in cardiopulmonary physiology. In the present study, we investigate whether intact human red blood cells (RBCs) synthesized to contain high SNO-Hb levels are able to export nitric oxide bioactivity and vasodilate the pulmonary circulation, and whether SNO-Hb dependent vasodilation occurs secondary to an intrinsic oxygen-linked, allosteric function of Hb. RBCs containing supraphysiological concentrations (100–1,000× normal) of SNO-Hb (SNO-RBCs) were synthesized and added to isolated, perfused rat lungs during anoxic or normoxic ventilation, and during normoxic ventilation with pulmonary hypertension induced by the thromboxane mimetic U-46619. SNO-RBCs produced dose-dependent pulmonary vasodilation compared with control RBCs during conditions of both normoxic (U-46619) and hypoxic pulmonary vasoconstriction. These effects were associated with a simultaneous, rapid, and temperature-dependent loss of SNO from Hb. Both vasodilatory effects and the rate of SNO-Hb degradation were independent of oxygen tension and Hb oxygen saturation. Furthermore, these effects were not affected by inhibition of the RBC membrane band 3 protein (anion exchanger-1), a putative membrane facilitator of NO export from RBCs. Whereas these data support observations by multiple groups that synthesized SNO-Hb can vasodilate, this effect is not under intrinsic oxygen-dependent allosteric control, nor likely to be relevant in the pulmonary circulation at normal physiological concentrations.

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35

Akiyoshi, Junko, Satoshi Ieiri, Takanori Nakatsuji, and Tomoaki Taguchi. "Direct vasodilative effect of FK506 on porcine mesenteric artery in small bowel transplantation." Journal of Pediatric Surgery 44, no. 12 (December 2009): 2322–26. http://dx.doi.org/10.1016/j.jpedsurg.2009.07.060.

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36

Groeneweg, George, Sjoerd Niehof, Feikje Wesseldijk, Frank J. P. M. Huygen, and Freek J. Zijlstra. "Vasodilative Effect of Isosorbide Dinitrate Ointment in Complex Regional Pain Syndrome Type 1." Clinical Journal of Pain 24, no. 1 (January 2008): 89–92. http://dx.doi.org/10.1097/ajp.0b013e318156db3b.

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37

Zuo, Sai-Jie, Sen Li, Rui-Hong Yu, Guo-Xun Zheng, Yong-Xiao Cao, and San-Qi Zhang. "Discovery of novel 3-benzylquinazolin-4(3 H )-ones as potent vasodilative agents." Bioorganic & Medicinal Chemistry Letters 24, no. 24 (December 2014): 5597–601. http://dx.doi.org/10.1016/j.bmcl.2014.10.092.

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38

Jun, Tao, P. Danna, E. Piccaluga, R. Seregni, A. Colombo, S. Porcellini, M. Viecca, and C. Florentini. "Lack of NO formation is involved in the vasodilative response to contrast media." International Journal of Angiology 9, no. 01 (April 24, 2011): 42–45. http://dx.doi.org/10.1007/bf01616330.

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39

McPherson, David D., Sarah Sirna, Steven M. Collins, Allan F. Ross, John R. Moyers, Bonnie J. Kane, Loren F. Hiratzka, Melvin L. Marcus, and Richard E. Kerber. "Can Atherosclerotic Coronary Arteries Vasodilate? An Intraoperative High-Frequency Epicardial Echocardiographic Study." American Journal of Cardiology 76, no. 1-2 (July 1995): 21–25. http://dx.doi.org/10.1016/s0002-9149(99)80794-2.

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40

Baron, A. D. "Hemodynamic actions of insulin." American Journal of Physiology-Endocrinology and Metabolism 267, no. 2 (August 1, 1994): E187—E202. http://dx.doi.org/10.1152/ajpendo.1994.267.2.e187.

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There is accumulating evidence that insulin has a physiological role to vasodilate skeletal muscle vasculature in humans. This effect occurs in a dose-dependent fashion within a half-maximal response of approximately 40 microU/ml. This vasodilating action is impaired in states of insulin resistance such as obesity, non-insulin-dependent diabetes, and elevated blood pressure. The precise physiological role of insulin-mediated vasodilation is not known. Data indicate that the degree of skeletal muscle perfusion can be an important determinant of insulin-mediated glucose uptake. Therefore, it is possible that insulin-mediated vasodilation is an integral aspect of insulin's overall action to stimulate glucose uptake; thus defective vasodilation could potentially contribute to insulin resistance. In addition, insulin-mediated vasodilation may play a role in the regulation of vascular tone. Data are provided to indicate that the pressor response to systemic norepinephrine infusions is increased in obese insulin-resistant subjects. Moreover, the normal effect of insulin to shift the norepinephrine pressor dose-response curve to the right is impaired in these patients. Therefore, impaired insulin-mediated vasodilation could further contribute to the increased prevalence of hypertension observed in states of insulin resistance. Finally, data are presented to indicate that, via a yet unknown interaction with the endothelium, insulin is able to increase nitric oxide synthesis and release and through this mechanism vasodilate. It is interesting to speculate that states of insulin resistance might also be associated with a defect in insulin's action to modulate the nitric oxide system.(ABSTRACT TRUNCATED AT 250 WORDS)

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Muchlisin, Muchlisin. "PENGARUH SINAR ULTRAVIOLET TERHADAP PROSES PENYEMBUHAN LUKA : SISTEMATICAL REVIEW." Jurnal Ilmu Keperawatan Medikal Bedah 2, no. 1 (May 31, 2019): 27. http://dx.doi.org/10.32584/jikmb.v2i1.218.

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AbstrakPendahuluan : Luka bagi kebanyakan orang adalah suatu hal yang sangat mengganggu dan menyebabkan ketidaknyamanan baik fisik ataupun psikis serta meningkatkan morbiditas bagi pasien yang terkena luka. Suatu paradigma baru dalam perawatan luka yang tidak beracun, minimal invasif dan ekonomis namun tetap mendukung penyembuhan luka yang optimal satu cara yang digunakan adalah menggunakan sinar ultraviolet sebagai terapi modalitas dalam mendukung proses kesembuhan perawatan luka pasien terutama pasien dengan luka infeksi dan hasilnya sangat signifikan dalam mendukung kesembuhan dalam perawatan luka infeksi dibandingkan dengan perawatan luka biasa tanpa kombinasi dengan sinar ultraviolet.Tujuan : Mengetahui gambaran mengenai pengaruh sinar ultraviolet terhadap proses penyembuhan lukaMetode : Metode yang digunakan dalam penulisan ini adalah studi literature, 3 jurnal terindeks science direct, 1 jurnal terindeks pubmed, 1 jurnal dari google scholar.Hasil dan Pembahasan : Sinar UV (tipe B) bermanfaat dalam mengurangi jumlah eksudat pada jenis luka infeksi dan mampu memperbaiki penampilan luka dan kedalaman luka menjadi lebih baik dibandingkan dengan kelompok kontrol. Secara khusus pada ulkus yang terinfeksi karena jumlah eksudat dapat menurun secara tidak langsung dapat mengurangi bau dan mempercepat proses penyembuhan luka. Sinar UV mengubah fungsi seluler, meningkatkan permeabilitas dinding sel dengan mengubah bentuk protein, merangsang produksi berbagai bahan kimia seperti prostaglandin dan asam arakidonat, dan meningkatkan produksi adenosin trifosfat. Eritema yang hasilnya meningkatkan vasodilatasi lokal, oksigenasi jaringan, dan pelepasan histamin.Kesimpulan : Sinar ultraviolet ( tipe B dan Tipe C ) mampu mempercepat proses penyembuhan luka, mengurangi jumlah eksudat, meningkatkan vasodilatasi, membunuh kuman patogen pada luka sehingga secara tidak langsung juga berpengaruh terhadap peranan menurunkan angka LOS, menurunkan cost efektiveness ( pembiayaan ), meningkatkan rasa nyaman dengan menurunkan nyeri pada luka, menurunkan bau yang pada luka karena produksi eksudat akibat infeksi luka serta meningkatkan angka harapan hidup pasien. Kata kunci : Ultraviolet, Wound Care, Wound Healing

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Elvira, Dwitya. "DIAGNOSIS DAN TATALAKSANA HEPATOPULMONARY SYNDROME." Majalah Kedokteran Andalas 38, no. 1 (May 20, 2015): 57. http://dx.doi.org/10.22338/mka.v38.i1.p57-65.2015.

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AbstrakSirosis hepatis dan penyakit hati kronik merupakan penyebab kematian terbanyak di seluruh dunia. Tingginya angka morbiditas dan mortalitas sirosis berhubungan dengan komplikasinya yang bersifat sistemik. Salah satu komplikasi sirosis dapat mengenai paru berupa sindrom hepatopulmonar atau hepatopulmonary syndrome. Hepatopulmonary syndrome (HPS) didefinisikan sebagai trias yang terdiri dari kegagalan hati stadium lanjut, hipoksemia arterial serta dilatasi intravaskular pulmonar tanpa disertai penyakit kardiopulmonar. Patogenesis HPS masih belum diketahui pasti, namun diduga terjadi gangguan metabolisme zat vasoaktif paru yang menimbulkan vasodilatasi vaskuler paru. Manifestasi klinis HPS berupa dispneu yang khas dengan tanda kegagalan hati dan hipertensi portal. Modalitas diagnostik HPS adalah radiologi thorax, analisa gas darah, contrast enhanced echocardiography (CEE), nuclear scanning dengan Tc-99m dan angiografi paru. Penatalaksanaan HPS terutama bertujuan menurunkan vasodilatasi intrapulmonar, meningkatkan oksigenasi arterial dan mengurangi keluhan. Deteksi dini terhadap komplikasi sirosis mutlak diperlukan dalam mencegah dan mengurangi angka morbiditas dan mortalitas.Abstract Liver cirrhosis and chronic liver disease are the leading cause of death worldwide. The high morbidity and mortality associated with their systemic complications. One of the complications of cirrhosis is hepatopulmonary syndrome. Hepatopulmonary syndrome (HPS) is defined as the triad of advanced-stage liver failure, arterial hypoxemia and pulmonary intravascular dilatation without cardiopulmonary disease. The pathogenesis of HPS is still not known for sure, but suspected metabolic disorder pulmonary vasoactive substances that cause pulmonary vascular vasodilatation. The clinical manifestations of HPS is typical dispneu with signs of liver failure and portal hypertension. HPS diagnostic modalities are radiology thorax, blood gas analysis, contrast enhanced echocardiography (CEE), nuclear scanning with Tc-99m and pulmonary angiography. HPS management aims primarily to lower intrapulmonar vasodilation, improving arterial oxygenation and reduce complaints. Early detection of complications of cirrhosis is absolutely necessary in preventing and reducing morbidity and mortality.

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Habedank, Dirk, Joerg C. Schefold, Carolin Bernhardt, Tim Karhausen, Wolfram Doehner, Stefan D. Anker, and Petra Reinke. "Vasodilation and Exercise Capacity in Patients with End-Stage Renal Disease: A Prospective Proof-of-Concept Study." Cardiorenal Medicine 7, no. 1 (September 21, 2016): 50–59. http://dx.doi.org/10.1159/000449174.

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Background: Previous data have pointed to the fact that vascular function is significantly impaired in patients with end-stage renal disease (ESRD). We aimed to better characterise vasodilation and exercise capacity in both ESRD and chronic heart failure (CHF) patients. Methods: A total of 30 ESRD patients (23 male; mean age 45.7 ± 9.9 years) were included in a prospective proof-of-concept study at a tertiary care academic centre. The patients underwent forearm venous plethysmography with post-ischaemic peak blood flow (PF) and flow-dependent flow (FDF) testing as well as cardiopulmonary exercise testing during the morning of the day following the last haemodialysis. After matching for age, gender, and body mass index, the data were compared to 30 patients with CHF and 20 age-matched healthy controls. Results: PF in ESRD patients was reduced when compared to that in CHF patients (12.5 ± 4.2 vs. 15.6 ± 6.9 ml/100 ml/min; p = 0.048) and healthy controls (26.4 ± 9.3 ml/100 ml/min; p < 0.001). When compared to controls, FDF was significantly reduced in ESRD patients (7.6 ± 3.1 vs. 6.0 ± 2.5 ml/100 ml/min; p = 0.03), but not in CHF patients, whereas resting blood flow did not differ between the ESRD, CHF, and healthy control groups. In contrast to indices of vasodilative capacity, maximum exercise capacity (peakVO2) was higher in ESRD when compared to CHF patients (23.8 ± 7.3 vs. 18.8 ± 5.2 ml/min/kg), but significantly impaired when compared to controls (32.8 ± 6.7 ml/min/kg; p < 0.001). Conclusion: In this proof-of-concept study, exercise capacity was relatively preserved, while vasodilative capacity was substantially impaired in ESRD patients. Additional studies are warranted to examine the underlying mechanisms and potential clinical implications of our findings.

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LaRouere, Michael J., Jonathan S. Sillman, Mary T. Tsai, and Alfred L. Nuttall. "The Effect of Pentoxifylline on Cochlear Blood Flow." Otolaryngology–Head and Neck Surgery 106, no. 1 (January 1992): 87–91. http://dx.doi.org/10.1177/019459989210600133.

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Pentoxifylline, a phosphodiesterase inhibitor and hemorrheologic agent has been found to increase oxygen delivery to ischemic tissue. Intravenous pentoxifylline was administered to normal guinea pigs in order to assess the effect of pentoxifylline on cochlear blood flow and to elucidate its mechanism of action. Intravenous pentoxifylline was found to acutely increase cochlear blood flow in a dose-dependent manner. In normal animals, the effect appeared strongly related to the rheologic properties of this agent rather than a vasodilative action. Normovolemic hemodilution with 75% dextran resulted in no Increase in cochlear blood flow during Infusion of pentoxifylline, whereas the application of nitroprusside over the round window failed to abolish the effect of pentoxifylline.

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De Rose, A. F., G. F. Oppezzi, S. Scotto, U. Repetto, S. Quattrini, and M. Lillo. "Follow-up e considerazioni fisiopatologiche sulla fibrosi dei corpi cavernosi dopo autoiniezione con PGE1." Urologia Journal 61, no. 1_suppl (January 1994): 106–9. http://dx.doi.org/10.1177/039156039406101s30.

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12 of the 24 patients who for 4 years have been practising PGE1 self-injection, underwent pharmaco-erection with papaverine (20 mg) then, two days after, with PGE1 (10 μg). In both bases we did an echoDoppler registration looking at arterial growth until the plateau was reached. In 6 patients corpus cavernosum biopsy was done using a 21 G bioptic needle. In this study the Authors suggest that the corpus cavernosum involution is caused not by the chemical-physical nature of the drug but by its hypertensive action. The PGE1 vasodilative action seems to be less quick than the papaverine one (50″ vs 10″). This fact would allow the cavernula to adapt more easily to the hypertensive state.

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Yamanaaa, Katsumi, Toshiaki Akira, Takeshi Uchida, Masahiro Watanabe, and Yoshio Kagitani. "Ameliorative effects of vasodilative prostaglandins against thrombus-induced circulatory disorder in rat femoral artery." Japanese Journal of Pharmacology 67 (1995): 274. http://dx.doi.org/10.1016/s0021-5198(19)47059-5.

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Hong, Show-Jen, Kwou-Yeung Wu, and Ing-Jun Chen. "Ocular hypotensive and vasodilative effects of two β-adrenergic blockers with intrinsic sympathomimetic activity." Current Eye Research 17, no. 7 (January 1998): 700–707. http://dx.doi.org/10.1080/02713689808951246.

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Kobayashi, Motomasa, Kazuyoshi Kawazoe, and Isao Kitagawa. "Aragupetrosine a, a new vasodilative macrocyclic quinolizidine alkaloid from an Okinawan marine sponge sp." Tetrahedron Letters 30, no. 31 (January 1989): 4149–52. http://dx.doi.org/10.1016/s0040-4039(00)99345-6.

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Shimizu, Shogo, Takahiro Shimizu, Youichirou Higashi, and Motoaki Saito. "Effect of alpha1 adrenoceptor antagonist on lower urinary tract symptoms as a vasodilative agent." Proceedings for Annual Meeting of The Japanese Pharmacological Society 92 (2019): 3—S25–1. http://dx.doi.org/10.1254/jpssuppl.92.0_3-s25-1.

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Cho, Hidetsura, Masaru Ueda, Keiyuu Shima, Akira Mizuno, Mariko Hayashimatsu, Yoshiko Ohnaka, Yumi Takeuchi, Mikiko Hamaguchi, and Kazuo Aisaka. "Dihydropyrimidines: novel calcium antagonists with potent and long-lasting vasodilative and anti-hypertensive activity." Journal of Medicinal Chemistry 32, no. 10 (October 1989): 2399–406. http://dx.doi.org/10.1021/jm00130a029.

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