Journal articles: 'Vascular endothelial growth factor'

1

Ferrara, Napoleone. "Vascular Endothelial Growth Factor." Arteriosclerosis, Thrombosis, and Vascular Biology 29, no. 6 (June 2009): 789–91. http://dx.doi.org/10.1161/atvbaha.108.179663.

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Wirostko, Barbara M. "Vascular Endothelial Growth Factor." Ophthalmology 114, no. 10 (October 2007): 1954–55. http://dx.doi.org/10.1016/j.ophtha.2007.05.018.

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Ferrara, N. "Vascular endothelial growth factor." European Journal of Cancer 32, no. 14 (December 1996): 2413–22. http://dx.doi.org/10.1016/s0959-8049(96)00387-5.

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Zachary, Ian. "Vascular endothelial growth factor." International Journal of Biochemistry & Cell Biology 30, no. 11 (November 1998): 1169–74. http://dx.doi.org/10.1016/s1357-2725(98)00082-x.

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5

Clauss, Matthias, and Wolfgang Schaper. "Vascular Endothelial Growth Factor." Circulation Research 86, no. 3 (February 18, 2000): 251–52. http://dx.doi.org/10.1161/01.res.86.3.251.

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Ferrara, Napoleone. "Vascular endothelial growth factor." Trends in Cardiovascular Medicine 3, no. 6 (November 1993): 244–50. http://dx.doi.org/10.1016/1050-1738(93)90046-9.

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Yip-Schneider, Michele, and C. Max Schmidt. "Vascular Endothelial Growth Factor." Journal of the American College of Surgeons 219, no. 3 (September 2014): 591–92. http://dx.doi.org/10.1016/j.jamcollsurg.2014.06.009.

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Aziz, Reem A. Abdel. "Serum Vascular Endothelial Growth Factor in Children with Beta Thalassemia Major." Journal of Medical Science And clinical Research 04, no. 12 (December 28, 2016): 14955–63. http://dx.doi.org/10.18535/jmscr/v4i12.107.

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Rama, K., and D. Kanmani. "Implication of VEGF (Vascular Endothelial Growth Factor) in Epithelial Ovarian Neoplasms." Indian Journal of Pathology: Research and Practice 5, no. 3 (2016): 283–90. http://dx.doi.org/10.21088/ijprp.2278.148x.5316.8.

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Arkonac, Burak M., Lauren C. Foster, Nicholas E. S. Sibinga, Cam Patterson, Kaihua Lai, Jer-Chia Tsai, Mu-En Lee, Mark A. Perrella, and Edgar Haber. "Vascular endothelial growth factor induces heparin-binding epidermal growth factor-like growth factor in vascular endothelial cells." Journal of Biological Chemistry 273, no. 15 (April 1998): 9352. http://dx.doi.org/10.1016/s0021-9258(18)49640-8.

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Arkonac, Burak M., Lauren C. Foster, Nicholas E. S. Sibinga, Cam Patterson, Kaihua Lai, Jer-Chia Tsai, Mu-En Lee, Mark A. Perrella, and Edgar Haber. "Vascular Endothelial Growth Factor Induces Heparin-binding Epidermal Growth Factor-like Growth Factor in Vascular Endothelial Cells." Journal of Biological Chemistry 273, no. 8 (February 20, 1998): 4400–4405. http://dx.doi.org/10.1074/jbc.273.8.4400.

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Barus, Jimmy, Ismail Setyopranoto, Ahmad Sadewa, and Samekto Wibowo. "The Role of Vascular Endothelial Growth Factor in Diabetic Polyneuropathy." International Journal of Medical Reviews and Case Reports 2, Reports in Surgery and Dermatolo (2019): 1. http://dx.doi.org/10.5455/ijmrcr.vascular-endothelial-growth-factor-diabetic-polyneuropathy.

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Waisbourd, Michael, Anat Loewenstein, Michaella Goldstein, and Igal Leibovitch. "Targeting Vascular Endothelial Growth Factor." Drugs & Aging 24, no. 8 (2007): 643–62. http://dx.doi.org/10.2165/00002512-200724080-00003.

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Maitland, Michael L., Xing Jian Lou, Jacqueline Ramirez, Apurva A. Desai, Dorit S. Berlin, Howard L. McLeod, Ralph R. Weichselbaum, Mark J. Ratain, Russ B. Altman, and Teri E. Klein. "Vascular endothelial growth factor pathway." Pharmacogenetics and Genomics 20, no. 5 (May 2010): 346–49. http://dx.doi.org/10.1097/fpc.0b013e3283364ed7.

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Krupitskaya, Yelena, and Heather Wakelee. "Vascular Endothelial Growth Factor Pathway." Journal of Thoracic Oncology 4, no. 11 (November 2009): S1071—S1073. http://dx.doi.org/10.1097/01.jto.0000361755.32660.a1.

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Bertelmann, Thomas, Stephan Schulze, Reka Bölöni, Walter Sekundo, Sebastian Irle, Thomas Stief, and Stefan Mennel. "Intravitreal vascular endothelial growth factor." Graefe's Archive for Clinical and Experimental Ophthalmology 252, no. 4 (February 4, 2014): 583–88. http://dx.doi.org/10.1007/s00417-014-2577-7.

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17

Cai, Weibo. "Multimodality imaging of vascular endothelial growth factor and vascular endothelial growth factor receptor expression." Frontiers in Bioscience 12, no. 8-12 (2007): 4267. http://dx.doi.org/10.2741/2386.

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Lazarus, A., and E. Keshet. "Vascular Endothelial Growth Factor and Vascular Homeostasis." Proceedings of the American Thoracic Society 8, no. 6 (November 1, 2011): 508–11. http://dx.doi.org/10.1513/pats.201102-021mw.

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19

Hagberg, Carolina, Annika Mehlem, Annelie Falkevall, Lars Muhl, and Ulf Eriksson. "Endothelial Fatty Acid Transport: Role of Vascular Endothelial Growth Factor B." Physiology 28, no. 2 (March 2013): 125–34. http://dx.doi.org/10.1152/physiol.00042.2012.

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Dietary lipids present in the circulation have to be transported through the vascular endothelium to be utilized by tissue cells, a vital mechanism that is still poorly understood. Vascular endothelial growth factor B (VEGF-B) regulates this process by controlling the expression of endothelial fatty acid transporter proteins (FATPs). Here, we summarize research on the role of the vascular endothelium in nutrient transport, with emphasis on VEGF-B signaling.

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20

Zhao, B., G. Smith, J. Cai, A. Ma, and M. Boulton. "Vascular endothelial growth factor C promotes survival of retinal vascular endothelial cells via vascular endothelial growth factor receptor-2." British Journal of Ophthalmology 91, no. 4 (August 30, 2006): 538–45. http://dx.doi.org/10.1136/bjo.2006.101543.

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21

A S, Dr Divya. "Plasma Vascular Endothelial Growth Factor (VEGF) In Ischemic Stroke – A Comparative Study." Journal of Medical Science And clinical Research 05, no. 03 (May 23, 2017): 19274–81. http://dx.doi.org/10.18535/jmscr/v5i3.152.

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22

Arisakti, Miatina Artisnita, and Muhtarum Yusuf. "Correlation between vascular endothelial growth factor expression and cervical lymph node carcinoma." International Journal of Psychosocial Rehabilitation 24, no. 02 (February 13, 2020): 4072–80. http://dx.doi.org/10.37200/ijpr/v24i2/pr200728.

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23

Matsuura, Akira, Seinosuke Kawashima, Wataru Yamochi, Ken-ichi Hirata, Tsutomu Yamaguchi, Noriaki Emoto, and Mitsuhiro Yokoyama. "Vascular Endothelial Growth Factor Increases Endothelin-Converting Enzyme Expression in Vascular Endothelial Cells." Biochemical and Biophysical Research Communications 235, no. 3 (June 1997): 713–16. http://dx.doi.org/10.1006/bbrc.1997.6885.

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24

Burke, P. A., K. Lehmannbruinsma, and J. S. Powell. "Vascular Endothelial Growth Factor Causes Endothelial Proliferation After Vascular Injury." Biochemical and Biophysical Research Communications 207, no. 1 (February 1995): 348–54. http://dx.doi.org/10.1006/bbrc.1995.1194.

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Enholm, B. "Vascular Endothelial Growth Factor-C A Growth Factor for Lymphatic and Blood Vascular Endothelial Cells." Trends in Cardiovascular Medicine 8, no. 7 (October 1998): 292–97. http://dx.doi.org/10.1016/s1050-1738(98)00026-7.

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26

Yeh, John, Beom Su Kim, and Jennifer Peresie. "Ovarian vascular endothelial growth factor and vascular endothelial growth factor receptor patterns in reproductive aging." Fertility and Sterility 89, no. 5 (May 2008): 1546–56. http://dx.doi.org/10.1016/j.fertnstert.2007.06.032.

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27

Ishida, Haruhiko, Kiyoshi Doi, Keita Katata, and Kenichi Nibu. "Expression of Vascular Endothelial Growth Factor (VEGF) in Nasal Polyps." Nihon Bika Gakkai Kaishi (Japanese Journal of Rhinology) 43, no. 2 (2004): 194–99. http://dx.doi.org/10.7248/jjrhi1982.43.2_194.

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28

Cao, Yihai, Hua Chen, Li Zhou, Ming-Ko Chiang, Bela Anand-Apte, James A. Weatherbee, Yongda Wang, Faye Fang, John G. Flanagan, and Monica Lik-Shing Tsang. "Heterodimers of Placenta Growth Factor/Vascular Endothelial Growth Factor." Journal of Biological Chemistry 271, no. 6 (February 9, 1996): 3154–62. http://dx.doi.org/10.1074/jbc.271.6.3154.

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29

Olofsson, B., K. Pajusola, A. Kaipainen, G. von Euler, V. Joukov, O. Saksela, A. Orpana, R. F. Pettersson, K. Alitalo, and U. Eriksson. "Vascular endothelial growth factor B, a novel growth factor for endothelial cells." Proceedings of the National Academy of Sciences 93, no. 6 (March 19, 1996): 2576–81. http://dx.doi.org/10.1073/pnas.93.6.2576.

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30

Elkar, Buğra, Mustafa Barış, Meryem Çalışır, Yasemin Çakır, Safiye Aktaş, Merve Tütüncü, M. Kürşat Şimşek, Mustafa Seçil, Osman Yılmaz, and Zekiye Altun. "Endothelial Dysfunction in Breast Cancer In-Vivo Model." Proceedings 2, no. 25 (December 6, 2018): 1536. http://dx.doi.org/10.3390/proceedings2251536.

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Although the endothelial dysfunction is related with tumor development, there is no consensus on the suppressive or supportive effect on tumor growth. The goal of the present study was to evaluate endothelial dysfunction related factors in animal breast cancer model that was developed by administrating endothelial nitric oxide synthase blocking agent, Nitro-L-arginine methyl ester hydrochloride (L-NAME). Endothelial dysfunction related main factors such as nitric oxide synthase, interleukin-6, vascular endothelial growth factor receptor-2 and vascular endothelial cadherin were investigated by immunohistochemically in tumor and carotid artery tissues. In tumor tissues apoptosis was determined by TUNEL assay. The level of endothelin-1 in blood was measured by ELISA. İntima-media thickness of carotid artery was evaluated with Doppler-USG measurements. As a result, in this study it was shown that vascular endothelial growth factor receptor-2, endothelin-1, endothelial nitric oxide synthase, interleukin-6, vascular endothelial cadherin and E-selectin molecules play a role in reducing breast tumor growth based on endothelial dysfunction.

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31

Jabbour, Mark N., James B. Elder, Christian G. Samuelson, Shabnam Khashabi, Florence M. Hofman, Steven L. Giannotta, and Charles Y. Liu. "ABERRANT ANGIOGENIC CHARACTERISTICS OF HUMAN BRAIN ARTERIOVENOUS MALFORMATION ENDOTHELIAL CELLS." Neurosurgery 64, no. 1 (January 1, 2009): 139–48. http://dx.doi.org/10.1227/01.neu.0000334417.56742.24.

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Abstract OBJECTIVE To identify and characterize the phenotypic and functional differences of endothelial cells derived from cerebral arteriovenous malformations (AVM), as compared with endothelial cells derived from a normal brain. METHODS Isolated AVM brain endothelial cells and control brain endothelial cells were evaluated immunohistochemically for expression of the endothelial cell markers von Willebrand factor and CD31, as well as angiogenic factors including vascular endothelial growth factor A, interleukin-8, and endothelin-1. Vascular endothelial growth factor receptors 1 and 2 were also evaluated using immunohistochemistry techniques. Functional assays evaluated cell proliferation, cytokine production, tubule formation, and cell migration using the modified Boyden chamber technique. RESULTS Endothelial cells derived from AVMs expressed high levels of vascular endothelial growth factor A and significantly overexpressed the vascular endothelial growth factor receptors 1 and 2 (P < 0.05), as compared with control endothelial cells. In addition, comparison to control brain endothelial cells demonstrated that AVM brain endothelial cells proliferated faster, migrated more quickly, and produced aberrant tubule-like structures. CONCLUSION Endothelial cells derived from cerebral AVMs are highly activated cells overexpressing proangiogenic growth factors and exhibiting abnormal functions consistent with highly activated endothelial cells.

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32

Masood, Rizwan, Ethel Cesarman, D. Lynne Smith, Parkash S. Gill, and Ornella Flore. "Human Herpesvirus-8-Transformed Endothelial Cells Have Functionally Activated Vascular Endothelial Growth Factor/Vascular Endothelial Growth Factor Receptor." American Journal of Pathology 160, no. 1 (January 2002): 23–29. http://dx.doi.org/10.1016/s0002-9440(10)64344-1.

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33

Benzaquen, L. R., A. Nicholson-Weller, and J. A. Halperin. "Terminal complement proteins C5b-9 release basic fibroblast growth factor and platelet-derived growth factor from endothelial cells." Journal of Experimental Medicine 179, no. 3 (March 1, 1994): 985–92. http://dx.doi.org/10.1084/jem.179.3.985.

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Interactions between endothelium and vascular smooth muscle cells play a major role in the biology of the blood vessel wall. Growth factors released from endothelial cells control in part the normal and pathological proliferation of vascular smooth muscle cells. Endothelial deposits of C5b-9 proteins, the membrane attack complex of complement (MAC), have been found in a variety of pathological tissues in which cell proliferation is an early characteristic abnormality, including atherosclerosis. We have explored a possible bridging role for terminal complement C5b-9 proteins in eliciting focal signals for cell proliferation by releasing growth factors from endothelial cells. We found that both bovine aortic and human umbilical vein cells respond to the MAC by releasing basic fibroblast growth factor and platelet-derived growth factor. These mitogens stimulate DNA synthesis in Swiss 3T3, vascular smooth muscle, and glomerular mesangial cells. Based on these findings, we propose that complement-induced release of mitogens from endothelial cells is a novel pathogenic mechanism for proliferative disorders.

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34

Roberts, W. G., and G. E. Palade. "Increased microvascular permeability and endothelial fenestration induced by vascular endothelial growth factor." Journal of Cell Science 108, no. 6 (June 1, 1995): 2369–79. http://dx.doi.org/10.1242/jcs.108.6.2369.

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The vascular endothelial growth factor (VEGF) was originally described as vascular permeability factor due to its ability to increase microvascular permeability to plasma proteins. However, the vessel types (arteriolar, venular, and capillary) affected by VEGF and the modification of endothelial morphology in response to increased permeability induced by VEGF in vivo have not been precisely documented. By topical application or intradermal injection of recombinant human VEGF-165 we find that VEGF increases the permeability of postcapillary venules as well as muscular venules and capillaries. Surprisingly, we also find that endothelia of small venules and capillaries become fenestrated within 10 minutes of VEGF application. Fenestrations appeared in vascular beds which do not normally have fenestrated endothelium, namely the cremaster muscle and skin. Histamine, saline, and heat-inactivated VEGF do not cause fenestrations. Increased permeability is completely inhibited when VEGF is cleared by immunoprecipitation with anti-VEGF monoclonal antibodies. The VEGF effect on permeability is unlike that of any other mediator described to date since both muscular venules and capillaries are affected.

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35

MASUDA, Haruchika. "Vascular Endothelial Growth Factor and Platelets." Internal Medicine 39, no. 7 (2000): 527–28. http://dx.doi.org/10.2169/internalmedicine.39.527.

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36

Zouggari, Yasmine. "Le VEGF (vascular endothelial growth factor)." Sang thrombose vaisseaux 21, no. 9 (November 2009): 494–96. http://dx.doi.org/10.1684/stv.2009.0434.

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37

D'Amore, Patricia A. "Vascular Endothelial Cell Growth Factor-A." American Journal of Pathology 171, no. 1 (July 2007): 14–18. http://dx.doi.org/10.2353/ajpath.2007.070385.

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38

Jackson, Tanisha A., Harry E. Taylor, Deva Sharma, Stephen Desiderio, and Sonye K. Danoff. "Vascular Endothelial Growth Factor Receptor-2." Journal of Biological Chemistry 280, no. 33 (June 7, 2005): 29856–63. http://dx.doi.org/10.1074/jbc.m500335200.

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39

Nilsson, Monique, and John V. Heymach. "Vascular Endothelial Growth Factor (VEGF) Pathway." Journal of Thoracic Oncology 1, no. 8 (October 2006): 768–70. http://dx.doi.org/10.1097/01243894-200610000-00003.

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40

Tateishi, Ukihide. "Vascular Endothelial Growth Factor–related Angiogenesis." Radiology 235, no. 3 (June 2005): 1084–85. http://dx.doi.org/10.1148/radiol.2353042072.

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41

Rosengart, Todd K., Edgar G. Chedrawy, Gerald Patejunas, and Mauricio Retuarto. "Vascular endothelial growth factor before cells." Journal of Thoracic and Cardiovascular Surgery 129, no. 3 (March 2005): 696. http://dx.doi.org/10.1016/j.jtcvs.2004.11.018.

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42

Hoeben, Ann, Bart Landuyt, Martin S. Highley, Hans Wildiers, Allan T. Van Oosterom, and Ernst A. De Bruijn. "Vascular Endothelial Growth Factor and Angiogenesis." Pharmacological Reviews 56, no. 4 (December 2004): 549–80. http://dx.doi.org/10.1124/pr.56.4.3.

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43

Silvestre, Jean-Sébastien. "Vascular Endothelial Growth Factor and Angiogenesis." Circulation 127, no. 16 (April 23, 2013): 1644–46. http://dx.doi.org/10.1161/circulationaha.113.002336.

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44

Nilsson, Monique, and John V. Heymach. "Vascular Endothelial Growth Factor (VEGF) Pathway." Journal of Thoracic Oncology 1, no. 8 (October 2006): 768–70. http://dx.doi.org/10.1016/s1556-0864(15)30404-4.

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45

Dyck, P. J., J. Engelstad, and A. Dispenzieri. "Vascular endothelial growth factor and POEMS." Neurology 66, no. 1 (January 9, 2006): 10–12. http://dx.doi.org/10.1212/01.wnl.0000194614.56025.ec.

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46

Isner, Jeffrey M. "Vascular Endothelial Growth Factor in Angiogenesis." Vascular Medicine Review vmr-6, no. 4 (November 1995): 311–22. http://dx.doi.org/10.1177/1358863x9500600407.

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47

Nelson, Peter T., and Gregory A. Jicha. "Cerebrospinal Fluid Vascular Endothelial Growth Factor." JAMA Neurology 72, no. 5 (May 1, 2015): 502. http://dx.doi.org/10.1001/jamaneurol.2015.34.

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48

I. Vento, C. H. J. Wolff, P. J. Sal, S. "Vascular Permeability Factor/Vascular Endothelial Growth Factor in Nasal Polyps." Acta Oto-Laryngologica 120, no. 543 (January 2000): 170–74. http://dx.doi.org/10.1080/000164800454314.

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Creamer, D., MH Allen, RW Groves, and JNWN Barker. "Circulating vascular permeability factor/vascular endothelial growth factor in erythroderma." Lancet 348, no. 9034 (October 1996): 1101. http://dx.doi.org/10.1016/s0140-6736(05)64447-9.

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50

Stepanova, T. V., A. N. Ivanov, N. E. Tereshkina, E. B. Popyhova, and D. D. Lagutina. "MARKERS OF ENDOTHELIAL DYSFUNCTION: PATHOGENETIC ROLE AND DIAGNOSTIC SIGNIFICANCE." Russian Clinical Laboratory Diagnostics 64, no. 1 (April 29, 2019): 34–41. http://dx.doi.org/10.18821/0869-2084-2019-64-1-34-41.

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Endothelial dysfunction (ED) is considered one of the pathogenetic mechanisms of a whole range of diseases. Detection of specific biochemical markers in the blood is an effective way to ED diagnostics that characterize the vascular endothelium state. This review highlights the pathogenetic role of the factors synthesized by endotheliocytes whose level changes in biological fluids reflect violations of the endothelium basic physiological properties: vasomotor function, thromboresistance, angiogenesis regulation, barrier and adhesion functions. In particular, the participation of nitric oxide metabolites, asymmetric dimethylarginine, endothelin-1, metabolic products of arachidonic acid, von Willebrand factor, thrombomodulin, vascular endothelial growth factor, vasohibine-1 and adhesion molecules in the onset and development of ED are reviewed. The diagnostic significances of factors damaging endothelium, such as C-reactive protein, homocysteine and 8-hydroxy-2’-deoxyguanosine, are discussed. In addition, the literature data of recent years about the prospects of clinical implication the detection of the above-mentioned factors which indicates structural and functional endothelial cells damage are given. Particular attention is paid to the ED markers detection prognostic significance and the possibility of their practical use for the ED diagnosis. The search of literature for the current review was conducted in RSIC, CyberLeninka, Scopus, Web of Science, MedLine and PubMed databases from 2012 to 2018 using the following keywords: endothelial dysfunction, nitric oxide, asymmetric dimethylarginine, endothelin-1, prostacyclin, thromboxane A2, epoxyeicosatrienoic acids, von Willebrand factor, thrombomodulin, vascular endothelial growth factor, vasohibin-1, adhesive molecules, C-reactive protein, homocysteine, and 8-hydroxy-2-deoxyguanosine.

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Journal articles on the topic 'Vascular endothelial growth factor'

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