Relevant bibliographies by topics / Vascular

Academic literature on the topic 'Vascular'

Author: Grafiati
Published: 4 June 2021
Last updated: 14 March 2023

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Journal articles on the topic "Vascular"

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Ascaso Martorel, Joaquín, and Joan Pcdrol Solanes. "De plantis vascularibus praesertim ibericis. III." Acta Botanica Malacitana 26 (December 1, 2001): 213–16. http://dx.doi.org/10.24310/abm.v26i0.7421.

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Moura-Ribeiro, Maria Valeriana L., Cristiane M. Rocha, Walter L. M. Fernandes, and Marilisa M. Guerreiro. "Meningites bacterianas agudas complicações vasculares: vascular complications." Arquivos de Neuro-Psiquiatria 51, no. 4 (November 1993): 507–10. http://dx.doi.org/10.1590/s0004-282x1993000400014.

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Os autores apresentam o estudo de quatro crianças que tiveram arterite como complicação vascular de meningite bacteriana aguda. Apresentam revisão dos mecanismos fisiopatológicos envolvidos nas alterações vasculares que têm trazido reais progressos no entendimento dessas complicações.
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A, Bajaj. "Vascular and Neurogenic-Cobb Syndrome." Open Access Journal of Gynecology 5, no. 1 (January 9, 2020): 1–5. http://dx.doi.org/10.23880/oajg-16000206.

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Cobb syndrome is an exceptional, non-inherited, genetic disorder characteristically constituted by vascular anomalies and neurological deficits. Spinal arteriovenous malformations appear in concurrence with cutaneous vascular lesions within the corresponding dermatome. Dermatome specific port wine stain upon the trunk, arteriovenous malformation, angioma, angiokeratoma, angiolipoma, cavernous haemangioma or lymphatic malformation is discerned in accompaniment with hyperreflexia, limb paresis, muscular cramps, sensory loss, bladder and bowel dysfunction, sudden paraplegia or subarachnoid haemorrhage. Spinal vascular lesions of Cobb syndrome can be adequately determined with magnetic resonance imaging (MRI), computerized tomography (CT) scan, plain radiography or angiography. Cobb syndrome can be appropriately managed with sclerotherapy, endovascular embolization, oral corticosteroids or surgical extermination of vascular lesions.
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Sánchez Ferrer, C. F., I. Valencia Fernández, and C. Peiró Vallejo. "Adipokines, ageing, and vascular damage." ANALES RANM 139, no. 139(03) (2023): 223–28. http://dx.doi.org/10.32440/ar.2022.139.03.rev01.

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The main physiologic function of adipokines on the energy metabolism and the cardiovascular homeostasis, among others, are well known. In the last years, our laboratory is providing increasing evidence about the possible role of adipokines as mediators of vascular damage, by inducing different pro-inflammatory and pro-senescent mechanisms. The secretion of adipokines like visfatin or dipeptidylpeptidase-4 (DPP4) by adipose tissue, particularly by the visceral fat, is enhanced in obese and type 2 diabetic patients. Both adipokines are enzymes metabolically active that can induce the activation of specific receptors, namely type 4 “toll like receptors” (TLR4) for visfatin and “protease activated receptor 2” (PAR2) for DPP4. Stimulation of such receptors is triggering well known pro-inflammatory and pro-senescent mechanisms, like the nuclear transcription factor-κB (NF-κB) or the inflammasome NLRP3 (“nucleotide-binding, leucine-rich-repeat, pyrin domain containing 3”), a cell structure that transforms the immature forms of classic cytokines in their active derivates, interleukin-1β (IL-1β) or interleukin 18 (IL-18). It is worth to note that these classic cytokines are the final effectors for the harmful effects of the adipokines and its blockade can be a very relevant therapeutic approach. In our experiments, the specific antagonism of IL-1 receptors with anakinra prevents the inflammatory and senescent effects evoked by visfatin and DPP4. Moreover, this finding is in agreement with data from other researchers, as well as with the results of the CANTOS clinical trial, which demonstrate a very important cardioprotective cardiovascular effect mediated by the anti-inflammatory effect of the monoclonal antibody canakinumab. On the other hand, we have also provided experimental evidence about possible vasculopotective adipokines, such angiotensin-(1-7), which is able to induce, through activation of Mas receptors, the expression of klotho protein and the activation of antioxidant pathways, like the well known Nrf2-HO-1 (“nuclear factor erythroid-2 y hemoygenase-1”).
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Ielapi, Nicola, Noemi Licastro, Martina Catana, Umberto Marcello Bracale, and Raffaele Serra. "Vascular Nursing and Vascular Surgery." Annals of Vascular Surgery 68 (October 2020): 522–26. http://dx.doi.org/10.1016/j.avsg.2020.05.038.

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Țandrău, Mircea Călin, Cristina Vîrjan, Oana Știrbu, and Daniela-Adriana Oatiș. "EVALUATION OF VASCULAR STIFFNESS SECONDARY TO VASCULAR CALCIFICATIONS IN AMBULATORY DIALYSIS PATIENTS." Romanian Medical Journal 68, no. 2 (June 30, 2021): 208–18. http://dx.doi.org/10.37897/rmj.2021.2.13.

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Arterial stiffness provides an indirect picture of vascular calcifications in terms of the effect they have on the systemic circulation and consequently on the heart. The present study sought to demonstrate the usefulness of outpatient investigation of vascular calcifications in dialysis patients using pulse wave velocity (PWV) determination. The aim of the study was to determine arterial resistance in patients with chronic dialysis kidney disease. The average study period was 48.5 ± 19.0 (SD) months. The study included 203 patients on dialysis with BCRT. The determination of arterial stiffness was measured at an interval of one hour after the dialysis session. The study highlighted the presence of an increased cardiovascular risk related to the presence of vascular calcifications without being able to establish a direct linear and causal link between their presence, location and the risk of cardiovascular events. The importance of highlighting calcifications and the consequences they generate means the possibility of influencing the main elements that can generate acute cardiovascular events. The results obtained and the correlations performed give an overview of the pathology studied, providing premises for therapeutic influence and risk assessment.
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Arthur, Adam S., I. Josh Abecassis, Karl R. Abi-Aad, Felipe C. Albuquerque, Rami O. Almefty, Rami James N. Aoun, Daniel L. Barrow, et al. "Vascular." Operative Neurosurgery 17, Supplement_1 (May 17, 2019): S76—S118. http://dx.doi.org/10.1093/ons/opz088.

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Varty, K., K. E. Allen, L. Jones, R. D. Sayers, J. D. Morgan, D. A. Ratliff, P. R. F. Bell, et al. "Vascular." Irish Journal of Medical Science 161, S11 (November 1992): 44–46. http://dx.doi.org/10.1007/bf02943727.

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Bonnici-Mallia, Anne M., Christopher Barbara, and Rahul Rao. "Vascular cognitive impairment and vascular dementia." InnovAiT: Education and inspiration for general practice 11, no. 5 (March 16, 2018): 249–55. http://dx.doi.org/10.1177/1755738018760649.

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Vascular dementia is the second-most-common type of dementia. Vascular cognitive impairment is a term encompassing vascular dementia as well as milder forms of pre-dementia cognitive impairment related to vascular damage that do not meet the criteria for a diagnosis of dementia. Early diagnosis of cerebrovascular disease, including silent infarcts and small vessel disease is difficult, but important, as modification of cardiovascular risk factors is the mainstay of management to decrease further insults and deterioration, and maintain the patient’s independence within their family and social unit.
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Ursache, Robertas, Jung-ok Heo, and Ykä Helariutta. "Plant Vascular Biology 2013: vascular trafficking." Journal of Experimental Botany 65, no. 7 (January 15, 2014): 1673–80. http://dx.doi.org/10.1093/jxb/ert462.

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Dissertations / Theses on the topic "Vascular"

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Purroy, Rodríguez Marina. "Canvis vasculars associats a l'envelliment en models animals de demència vascular." Doctoral thesis, Universitat de Barcelona, 2021. http://hdl.handle.net/10803/673556.

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INTRODUCCIÓ: El deteriorament cognitiu d’origen vascular, i més concretament la demència vascular (DV), s’associa al compromís persistent del flux sanguini cerebral i representa la segona causa més freqüent de demència al món, després de la malaltia d’Alzheimer. La DV és una malaltia caracteritzada per múltiples i discretes lesions isquèmiques, lesions difuses a la substància blanca (leucoaraiosi) i hemorràgies cerebrals, entre d’altres. S’han dedicat gran quantitat d’esforços en la recerca dels mecanismes fisiopatològics subjacents a aquesta patologia però encara no es coneixen amb precisió. Les tècniques de neuroimatge, com la imatge per ressonància (MRI), van suposar un gran avenç en el diagnòstic d’aquesta patologia. Per aquest motiu, en aquest treball de tesi doctoral s’ha proposat l’estudi longitudinal de canvis estructurals i funcionals vasculars al llarg del temps en animals d’experimentació, en el curs dels símptomes de la malaltia, i sota la influència de diversos factors de risc, mitjançant diferents tècniques de MRI per trobar nous biomarcadors no invasius per la DV, i per avançar en l’estudi de la seva fisiopatologia, així com en el diagnòstic precoç i la monitorització de l’efectivitat de noves teràpies. MATERIAL I MÈTODES: Es van utilitzar un total de 174 ratolins de dues soques, ratolins ApoE-/-, que tenen predisposició a desenvolupar aterosclerosi amb l’edat, i els ratolins control C57Bl6 de tres edats diferents, des de l’adult jove (6 mesos) fins a la vellesa (18 mesos) incloent una edat intermitja (12 mesos). Es va realitzar un seguiment longitudinal amb tècniques de MRI per estudiar diferents característiques de les lesions vasculars, que es van correlacionar proves de comportament. La validació de les tècniques de MRI es va realitzar mitjançant tincions d’immunohistoquímica per MBP i la clarificació de talls gruixuts de cervell (2 mm) per quantificació de la microvasculatura. RESULTATS: Es van detectar alteracions relacionades amb aterosclerosi i hipoperfusió en longitud i tortuositat de les principals artèries que irriguen el cervell mitjançant angiografia TOF. També en relació a la irrigació del cervell, es van trobar canvis en el flux sanguini cerebral mitjançant perfusió ASL, associats a les tres condicions estudiades (envelliment, aterosclerosi i hipoperfusió). Així mateix, les tres condicions experimentals van provocar alteracions en les fibres de mielina, detectades amb l’anàlisi dels diferents components dels tensors de difusió (DTI). Aquests canvis es van trobar a estructures relacionades amb les alteracions en les proves de conducta, com ara en ansietat, 11 memòria de reconeixement, debilitat muscular i pertorbacions en la marxa. D’altra banda, l’índex Q va mostrar menys sensibilitat que l’anàlisi histopatològic per a l’estudi de la densitat microvascular. Tot i això, va servir per detectar canvis en la densitat microvascular associats a l’edat. Els canvis en el gruix de l’escorça cerebral (atròfia cortical) mesurats per MRI va resultar ser un bon biomarcador de la mort neuronal produïda per l’envelliment i la hipoperfusió. CONCLUSIONS: Els resultats d’aquest estudi confirmen la utilitat de la MRI com a eina per avaluar els canvis estructurals i funcionals de la neuropatologia de l’envelliment, l’aterosclerosi i la hipoperfusió en recerca traslacional.
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Adams, Gregory Nicholas. "Prolylcarboxypeptidase protects from vascular dysfunction and promotes vascular repair." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1346973249.

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Núñez, Do Rio Joan M. "Vascular pattern characterization in colonoscopy images." Doctoral thesis, Universitat Autònoma de Barcelona, 2015. http://hdl.handle.net/10803/325145.

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El càncer de còlon és el tercer amb més incidència al món i el segon tipus de tumor maligne més comú a Europa. Les tècniques d'exploració directa del còlon han demostrat la seva eficiència en la reducció del nombre de víctimes mortals, permetent la detecció de pòlips en estadis prematurs. Entre les diferents tècniques d'exploració, la colonoscòpia és considerada actualment l'estàndard clínic, tot i que diferents estudis revelen la incidència d'alguns factors en la qualitat de l'exploració. La navegació al llarg del còlon i el recte evidencia una sèrie de reptes per als endoscopistes que provoquen un augment en la taxa d'errors. L'acurada inspecció del còlon ha de ser certificada per tal de minimitzar les possibilitats que alguna lesió no sigui detectada. La inspecció de les imatges de colonoscòpia pot aportar informació crucial per als endoscopistes i donar suport a la navegació durant el procediment. Els vasos sanguinis i els seus patrons de ramificació poden aportar potencial descriptiu per desenvolupar marcadors biomètrics. Els marcadors anatòmics basats en vasos sanguinis podrien ser utilitzats per identificar escenes en vídeos de colonoscòpia i donar suport per a la navegació generant una seqüència d'imatges ordenades en el recorregut de les seccions del colon. Verificant la presència de contingut vascular a l'escena endoluminal també és possible certificar una acurada inspecció de les mucoses i millorar la localització de pòlips. Considerant els usos potencials de la descripció dels vasos sanguinis, aquesta contribució estudia la caracterització del contingut vascular i l'anàlisi de la capacitat descriptiva dels seus patrons de ramificació. La caracterització dels vasos sanguinis en imatges de colonoscòpia suposa reptes importants. L'escena endoluminal inclou diferents objectes amb característiques similars, fet que dificulta el desenvolupament de models diferents per a cadascun d'aquests objectes. Per afrontar aquestes dificultats, proposem l'ús dels patrons de ramificació dels vasos sanguinis com a trets a baix nivell per a la descripció de formes. Hem creat dues bases de dades d'imatges que inclouen la segmentació manual dels arbres vasculars, així com la localització manual de dos tipus de punts d'interès: encreuaments i punts finals. Presentem un model per a la caracterització dels punts d'encreuament en patrons binaris. La implementació del model ens permet desenvolupar un mètode de localització de punts d'encreuament. El mètode supera els algorismes existents a la literatura en experiments en dues bases de dades: una de creació pròpia i la base de dades DRIVE, d'imatges de fons d'ull. En el segon cas, hem creat una extensió amb la localització manual dels punts d'encreuament. Pel fet que volem explorar la capacitat descriptiva de patrons vasculars i punts d'encreuament, proposem una aproximació basada en grafs per crear marcadors anatòmics. En el context de la localització de pòlips, establim un nou mètode per inhibir la influència dels vasos sanguinis en l’extracció d'informació de baix nivell. Els resultats mostren que la nostra metodologia disminueix la influència dels vasos sanguinis, augmenta la informació als pòlips i millora els mètodes de localització de pòlips.
Colorectal cancer is the third most common cancer worldwide and the second most common malignant tumor in Europe. Screening tests have shown to be very effective in reducing the amount of deaths since they allow an early detection of polyps. Among the different screening techniques, colonoscopy is considered the gold standard although clinical studies mention several problems that have an impact in the quality of the procedure. The navigation through the rectum and colon track can be challenging for the physicians which can increase polyp miss rates. The thorough visualization of the colon track must be ensured so that the chances of missing lesions are minimized. The visual analysis of colonoscopy images can provide important information to the physicians and support their navigation during the procedure. Blood vessels and their branching patterns can provide descriptive power to potentially develop biometric markers. Anatomical markers based on blood vessel patterns could be used to identify a particular scene in colonoscopy videos and to support endoscope navigation by generating a sequence of ordered scenes through the different colon sections. By verifying the presence of vascular content in the endoluminal scene it is also possible to certify a proper inspection of the colon mucosa and to improve polyp localization. Considering the potential uses of blood vessel description, this contribution studies the characterization of the vascular content and the analysis of the descriptive power of its branching patterns. Blood vessel characterization in colonoscopy images is shown to be a challenging task. The endoluminal scene is conformed by several objects whose similar characteristics hinders the development of particular models for each of them. To overcome such difficulties we propose the use of the blood vessel branching characteristics as low-level features for pattern description. We created two data sets including manually labeled vessel information as well as manual ground truths of two types of keypoint landmarks: junctions and endpoints. We present a model to characterize junctions in binary patterns. The implementation of the junction model allows us to develop a junction localization method. The proposed method outperforms the available algorithms in the literature in experiments in both, our newly created colon vessel data set, and in DRIVE retinal fundus image data set. In the latter case, we created manual ground truth of junction coordinates. Since we want to explore the descriptive potential of junctions and vessels, we propose a graph-based approach to create anatomical markers. In the context of polyp localization, we present a new method to inhibit the influence of blood vessels in the extraction of low-level profile information. The results show that our methodology decreases vessel influence, increases polyp information and leads to an improvement in state-of-the-art polyp localization performance.
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Sunman, Wayne. "The role adenosine in vascular steal in peripheral vascular disease." Thesis, University of Liverpool, 1997. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.264365.

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Rudström, Håkan. "Iatrogenic Vascular Injuries." Doctoral thesis, Uppsala universitet, Kärlkirurgi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-194346.

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Iatrogenic vascular injuries (IVIs) and injuries associated with vascular surgery can cause severe morbidity and death. The aims of this thesis were to study those injuries in the Swedish vascular registry (Swedvasc), the Swedish medical injury insurance where insurance claims are registered, the Population and Cause of death registries, and in patient records, in order to explore preventive strategies. Among 87 IVIs during varicose vein surgery 43 were venous, mostly causing bleeding in the groin. Among 44 arterial injuries, only 1/3 were detected intraoperatively. Accidental arterial stripping predominated, with poor outcome. Four patients died, all after venous injuries. IVIs increased over time, and constitute more than half of the vascular injuries registered in the Swedvasc. Lethal outcome was more common (4.9%) among patients suffering IVIs than among non-iatrogenic vascular injuries (2.5%). Risk factors for death were age, diabetes, renal insufficiency and obstructive lung-disease. Fifty-two patients died within 30 days after IVI. The most common lethal IVIs were puncture during endovascular procedures (n=24, 46%), penetrating trauma during open surgery (11) and occlusion after compression (6). Symptoms were peripheral ischemia (n=19), external bleeding (14), and hypovolemic chock without external bleeding (10). Most died within two weeks (n=36, 69%). After >2 weeks the IVI as a cause of death was uncertain. Among 193 insurance claims after vascular surgery during 2002-2007, nerve injuries (91) and wound infections (22) dominated. Most patients suffered permanent injuries, three died. Patients with insurance claims were correctly registered in the Swedvasc in 82%. In 32 cases of popliteal artery injury during knee arthroplasty symptoms were bleeding (n=14), ischaemia (n=7) and false aneurysm formation (n=11). Only twelve injuries (38%) were detected intraoperatively. Patency at 30 days was 97%, but only seven (22%) patients had complete recovery. Six of those had intraoperative diagnosis of popliteal injury and immediate vascular repair. In conclusion, registration of IVIs is increasing and outcome is often negatively affected by diagnostic and therapeutic delay. Not all fatalities after IVIs are attributable to the injury itself. The most common causes of insurance claims after vascular surgery were nerve injuries, and 82% were correctly registered in Swedvasc.
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Salanga, Matthew Charles. "EMBRYONIC VASCULAR DEVELOPMENT." Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/203435.

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The formation of the embryonic vasculature is essential for life. The components driving this process are well conserved across vertebrate species. At the core of vascular development is the specification of endothelial precursor cells from nascent mesoderm. Transcription factors of the ETS family are important regulators of endothelial specification. In this document we characterize the role of the ETS transcription factors, ETV2, during embryonic vascular development.Expression analysis shows that Etv2 is highly expressed in hematopoietic and endothelial precursor cells in the Xenopus embryo. In gain-of-function experiments, ETV2 is sufficient to activate ectopic expression of vascular endothelial markers. In addition, ETV2 activated expression of hematopoietic genes representing the myeloid but not the erythroid lineage. Loss-of-function studies indicate that ETV2 is required for expression of all endothelial markers examined. However, knockdown of ETV2 has no detectable effects on expression of either myeloid or erythroid markers. This contrasts with studies in mouse and zebrafish where ETV2 is required for development of the myeloid lineage. Our studies confirm an essential role for ETV2 in endothelial development, but also reveal important differences in hematopoietic development between organisms.Although ETV2 is a pivotal molecule in development it remains unidentified in the chicken genome. We hypothesize that chicken Etv2 is expressed in the early Gallus embryo, and is necessary for endothelial specification consistent with its role in other species. To test this hypothesis we attempted to amplify Etv2 transcripts from Gallus embryos using degenerate PCR. Disappointingly this strategy did not reveal a putative Etv2 candidate. However, some important findings were uncovered, including the cloning of a previously uncharacterized Gallus ETS protein, SPDEF. Additionally the identification of an annotation error mis-identifying Ets gene "Erf" as "Etv3" (also an Ets gene) provided details on gene arrangement previously unknown. The workflow described could be used in future studies for the identification of other members of gene families that exhibit gaps, keeping in mind the goal of the study and the limitations of each technology.
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Lugano, Roberta. "Low density lipoproteins, vascular smooth muscle cell function and vascular remodeling." Doctoral thesis, Universitat Pompeu Fabra, 2013. http://hdl.handle.net/10803/283471.

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High levels of circulating low-density lipoproteins (LDL) are one of the major cardiovascular risk factors. Hypercholesterolemia induces endothelial dysfunction and chronic intimal inflammatory cell accumulation, hallmarks of the initiation of atherosclerosis. Additionally, growing human atherosclerotic plaques show proliferation and migration of vascular smooth muscle cells (VSMC) towards the intima producing remodeling of the vascular wall. However, those plaques that are most prone to rupture show a progressive loss of VSMC becoming soft and vulnerable and these lipid-rich high risk plaques cause clinical episodes resulting in morbid or fatal ischemic events. The mechanisms involved in the transformation of a plaque into a vulnerable VSMC-depleted atheroma have not been completely elucidated. Lipid-rich-VSMC have an impaired vascular repair function due to changes in cytoskeleton proteins. However, the effects of LDL on VSMC function during plaque remodeling and vascular repair are not fully understood. Thus, the aim of this thesis was to investigate early changes directly induced by LDL on VSMC phenotype and function and to identify the molecular mechanisms involved. This thesis demonstrates that the cardiovascular risk of hypercholesterolemia involves the interaction of LDL with VSMC and the regulation at a molecular level of different pathways that converge in the cell’s migratory capacity. Migratory function of lipid-loaded VSMC can be restored by inhibition of 3-hydroxy-methylglutaryl coenzyme A (HMG-CoA) through a Rho kinase and myosin light chain phosphatase dependent mechanism. In addition, the studies performed in this thesis show that LDL affect VSMC adhesion, migration and cytoskeleton dynamics through the abrogation of the urokinase-plasminogen activator (uPA)/uPA receptor (uPAR) system function and by modulation of HSP27 phosphorylation and subcellular localization.
El nivel elevado de lipoproteínas de baja densidad (LDL), uno de los principales factores de riesgo cardiovascular, conllevan a una disfunción endotelial y acumulación crónica de células inflamatorias en la íntima arterial en la etapa inicial de desarrollo de la arterosclerosis. Además, la progresión de las placas arterioscleróticas se caracteriza por un proceso de remodelado vascular consecuencia de la proliferación y migración de células musculares lisas vasculares (CML) en la íntima. Sin embargo, las placas ateroscleróticas con mayor susceptibilidad a la ruptura presentan una pérdida progresiva de CML, siendo estas placas ricas en lípidos y altamente vulnerables las que provocan eventos isquémicos mórbidos o fatales. Hoy día desconocemos todavía los mecanismos involucrados en la transformación de las placas en ateromas vulnerables. Las CML ricas en lípidos presentan alteraciones en su capacidad de reparación vascular debido a alteraciones en proteínas del citoesqueleto. Sin embargo, los efectos de las LDL en la función de las CML durante el remodelado de las placas y reparación vascular se desconocen en gran medida. Por ello, el objetivo de esta tesis ha sido investigar los cambios iniciales inducidos directamente por las LDL en el fenotipo y la función de las CML e identificar los mecanismos moleculares involucrados. Esta tesis demuestra que el riesgo cardiovascular de la hipercolesterolemia implica la interacción entre LDL y CML y la regulación a nivel molecular de diferentes vías de señalización que convergen en la migración celular. La capacidad de migración de CML cargadas de lípidos puede restituirse mediante la inhibición de la 3-hidroxi-3-metilglutaril coenzima-A (HMG-CoA), a través de un mecanismo dependiente de la quinasa Rho. Además, los estudios realizados en esta tesis demuestran que las LDL afectan la adhesión, migración y dinámica de formación del citoesqueleto de las CML a través de la alteración de la función del sistema del activador del plasminogeno tipo uroquinasa (uPA)/uPA receptor (uPAR) y mediante la modulación de la fosforilación y localización subcelular de la HSP27.
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Kara, Kerim. "A 3-d Vascular Connectivity Tracking And Vascular Network Extraction Toolkit." Master's thesis, METU, 2011. http://etd.lib.metu.edu.tr/upload/12613201/index.pdf.

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Angiography is an invasive procedure since contrast medium is injected into circulatory system of patients and the mostly preferred technique is X-ray angiography. For diagnosis, treatment planning, and risk assessment purposes, interventional radiologists utilize visual inspection to determine connectivity relations between vessels. This situation leads angiography to be more invasive, since it requires additional injection of contrast medium and X-ray dose. This thesis work presents a 3-D vascular connectivity tracking toolkit for automated extraction of vascular networks in 3-D medical images. The proposed method automatically extracts the vascular network connected to a user-defined point in a user-defined direction, and requires no further user interaction. The toolkit prevents additional injection of contrast agent and X-ray dose, saves time for the interventional radiologist. While the algorithm is applicable on all 3-D angiography images, performance of the method is observed on 3-D catheter angiography image of cerebrovascular structures. The algorithm iteratively tracks gravity centers of vascular branches in the user-defined direction, preserving connection to the user-defined point. Curvy branches are tracked even if they have discontinuous portions. Since this tracking method does not depend on lumen diameter and intensity differences, branches with stenoses and branches having large intensity difference can be successfully tracked. Skeletonization and junction detection methods are described, which are used to detect the sub branches, indirectly connected to the point. These methods are capable of handling bifurcations, trifurcations, and junctions having more branches. However, false junctions occurring due to superposition of vessels are not eliminated.
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Allcock, Graham Harvey. "Functional characterisation of endothelin receptors in vascular and non-vascular tissues." Thesis, Queen Mary, University of London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244194.

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Wardell, Claire Frances. "Mechanisms of transmitter release in vascular and non-vascular smooth muscle." Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.303665.

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Books on the topic "Vascular"

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Savage, Mark William. Vascular reactivity, insulin resistance and vascular disease. Manchester: University of Manchester, 1996.

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J, Vinken P., and Toole James F. 1925-, eds. Vascular diseases. Amsterdam: Elsevier Science Publishers, 1988.

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Vascular aphasia. Cambridge, Mass: MIT Press, 1986.

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Lee, Byung-Boong, Peter Gloviczki, Francine Blei, and Jovan N. Markovic, eds. Vascular Malformations. Boca Raton : CRC Press, [2020]: CRC Press, 2019. http://dx.doi.org/10.1201/9780367255343.

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Baker, Andrew H. Vascular Disease. New Jersey: Humana Press, 1999. http://dx.doi.org/10.1385/1592592473.

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Paul, Robert H., Ronald Cohen, Brian R. Ott, and Stephen Salloway, eds. Vascular Dementia. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1592598242.

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Scurr, John H. Vascular surgery. London: Cavendish Publishing, 1999.

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Gasser, T. Christian. Vascular Biomechanics. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-70966-2.

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Vivanco, Fernando, ed. Vascular Proteomics. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-405-0.

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Geroulakos, George, and Bauer Sumpio, eds. Vascular Surgery. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-65936-7.

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Book chapters on the topic "Vascular"

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McBride, Andrew, Amy M. Singer, and Holly Beach. "Vascular." In Sports-related Fractures, Dislocations and Trauma, 683–92. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-36790-9_39.

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Yang, Aaron Jay, and Nitin B. Jain. "Vascular." In Pain Medicine, 73–74. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-43133-8_19.

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Paño, Blanca, and Pedro Seguí. "Vascular." In Learning Ultrasound Imaging, 187–212. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30586-3_7.

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Awaad, Yasser M. "Vascular." In Absolute Pediatric Neurology, 705–28. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78801-2_28.

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Jones, Brandt D. "Vascular." In Passing the General Surgery Oral Board Exam, 133–41. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7663-4_16.

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Jin, Kwang Nam, Eun-Ah Park, and Whal Lee. "Vascular." In Dual Source CT Imaging, 103–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-15134-7_10.

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Han, Dan, Lan Yan, and Hui Duan. "Vascular." In Dual Source CT Imaging, 111–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-15134-7_11.

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Cheah, Foong Koon, and John Huang. "Vascular." In Dual Source CT Imaging, 119–25. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-15134-7_12.

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Peiling, Li, Chai Ruimei, Wang Qiang, and Xu Ke. "Vascular." In Dual Source CT Imaging, 127–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-15134-7_13.

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Shin, Cheong-Il, Eun-Ah Park, and Whal Lee. "Vascular." In Dual Source CT Imaging, 135–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-15134-7_14.

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Conference papers on the topic "Vascular"

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"Clinical: Vascular." In Proceedings of UK Radiological Conference 2013. The British Institute of Radiology, 2013. http://dx.doi.org/10.1259/conf-pukrc.2013.vascular.

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"Clinical: Vascular." In Proceedings of UK Radiological Conference 2012. The British Institute of Radiology, 2012. http://dx.doi.org/10.1259/conf-pukrc.2012.3.vascular.

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Klepac, Nataša. "Vascular dementia." In Rijeka Forum on Neurodegenerative Diseases (2 ; 2018 ; Rijeka). Hrvatska akademija znanosti i umjetnosti, 2019. http://dx.doi.org/10.21857/mzvkptz479.

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Cole, Jim, Ian Bond, and Andrew Lawrie. "Active Thermal Management of FRP Composites via Embedded Vascular Networks." In ASME 2019 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/smasis2019-5555.

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Abstract Fibre-reinforced polymer (FRP) composite materials are limited in high temperature applications by the matrix glass transition temperature, Tg. At and above this temperature, significant mechanical performance is lost, and degradation processes accelerated. This research explores the use of internal passages, or vascules, within the laminate to carry a coolant fluid, absorbing heat energy and cooling the material. A custom thermal chamber and four-point flexural test fixture were developed to perform in-situ thermo-mechanical testing. Vascular and non-vascular carbon/epoxy specimens were manufactured, containing arrays of four 1.1 mm diameter vascules. Specimens were exposed to temperatures from ambient to 170 °C (Tg = 200 °C). Flexural modulus varied little with temperature across all tests. Non-vascular specimens at 170 °C showed a reduction in ultimate strength of 21 % compared to under ambient conditions. The presence of vascules caused a small improvement in flexural modulus and strength, due to displacement of a small number of 0° fibre tows further from the neutral axis as a result of the manufacturing process. At 15 L·min−1 coolant flow, vascular specimens showed full retention of strength compared to non-vascular specimens at ambient, demonstrating the potential mechanical performance benefits.
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Verisokin, Andrey Yu, Darya V. Verveyko, and Dmitry E. Postnov. "Simulation of Propagated Vascular Responses at the Vascular Bifurcation." In 2020 11th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2020. http://dx.doi.org/10.1109/esgco49734.2020.9158145.

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Zahadat, Payam, Daniel Nicolas Hofstadler, and Thomas Schmickl. "Vascular morphogenesis controller." In GECCO '17: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3071178.3071247.

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White, Rodney A., George Kopchok, and Geoffrey H. White. "Laser Vascular Welding." In 1988 Los Angeles Symposium--O-E/LASE '88, edited by Michael W. Berns. SPIE, 1988. http://dx.doi.org/10.1117/12.945334.

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Sturm, Deborah, Lisa Xu, and Michael E. Kress. "Visualizing vascular structures." In Electronic Imaging '97, edited by Divyendu Sinha. SPIE, 1997. http://dx.doi.org/10.1117/12.270347.

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Bonura, Eric, Junchieh J. Tsay, Allison S. Friedenberg, Nishay Chitkara, and Laura E. Evans. "Vascular Ultrasound Education." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4305.

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Synn, A., C. de Margerie-Mellon, S. Y. Jeong, A. Bankier, F. N. Rahaghi, G. R. Washko, R. San Jose Estepar, P. VanderLaan, and M. B. Rice. "Pulmonary Vascular Pruning on Computed Tomography and Histologic Vascular Remodeling." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3680.

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Reports on the topic "Vascular"

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Passariello, Fausto, Bruno Amato, Pier Luigi Antignani, Guillermo Bonvini, Giuseppe Botta, Alberto Caggiati, Massimo Cappelli, et al. Informed Consensus in Vascular Procedures. A VASCULAB Project. Fondazione Vasculab, October 2005. http://dx.doi.org/10.24019/2005.icivp.

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Ethicon, Inc. Vascular Anastomosis. Touch Surgery Simulations, 2015. http://dx.doi.org/10.18556/touchsurgery/2015.s0097.

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Morrison, W., W. Winters, P. Leonard, and B. Miller. Vascular Occlusive Device prototyping. Office of Scientific and Technical Information (OSTI), April 1997. http://dx.doi.org/10.2172/770388.

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Passariello, Fausto, ed. Informed Consensus in Vascular Procedures. Fondazione Vasculab, December 2006. http://dx.doi.org/10.24019/2006.icivp.

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It is an open project, which has the aim of writing protocols for the informed consensus in invasive and non invasive vascular procedures. Versions in several languages are scheduled. English and Italian initially. Later other languages will follow, as soon as the translation will be technically possible. The project is organised into Sections. There is an initial index of the Proposed Sections, but users can by themselves propose other ones. Anyway, the Section is officially constituted as soon as they are gathered the subscriptions of the Section Coordinator and of others in a number which is sufficient to carry on the project.
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Sackschewsky, Michael R., and Janelle L. Downs. Vascular Plants of the Hanford Site. Office of Scientific and Technical Information (OSTI), September 2001. http://dx.doi.org/10.2172/789922.

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Pumiglia, Kevin. NF1 Signal Transduction and Vascular Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, May 2014. http://dx.doi.org/10.21236/ada605985.

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Sackschewsky, Michael R., and Janelle L. Downs. Vascular Plants of the Hanford Site. Office of Scientific and Technical Information (OSTI), September 2001. http://dx.doi.org/10.2172/965728.

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Alexander, Jonathan. Lymphatic Vascular-Based Therapy for IBD. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada567582.

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Pumiglia, Kevin. NF1 Signal Transduction and Vascular Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, May 2015. http://dx.doi.org/10.21236/ada620935.

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Faris, Gregory W. Breast Cancer Detection Using Optical Vascular Fusion. Fort Belvoir, VA: Defense Technical Information Center, June 2005. http://dx.doi.org/10.21236/ada455282.

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You might also be interested in the extended bibliographies on the topic 'Vascular' for particular source types:
Journal articles Dissertations / Theses Books
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