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Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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1

Lin, Phoebe, and Eric Suhler, eds. Treatment of Non-infectious Uveitis. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-22827-9.

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2

Kanski, Jack J. Uveitis: A colour manual of diagnosis and treatment. London: Butterworths, 1987.

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3

1934-, Nozik Robert A., ed. Surgical treatment of ocular inflammatory disease. Philadelphia: Lippincott, 1988.

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4

New treatments in non-infectious uveitis. Basel: Karger, 2012.

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5

Diagnosis and Treatment of Uveitis. Saunders, 2002.

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6

Diagnosis and Treatment of Uveitis. Jaypee Brothers Medical Publishers (P) Ltd., 2013. http://dx.doi.org/10.5005/jp/books/11822.

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7

Lin, Phoebe, and Eric Suhler. Treatment of Non-infectious Uveitis. Springer, 2019.

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8

Diagnosis And Treatment Of Uveitis. Jaypee Brothers Medical Publishers, 2013.

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9

Yang, Peizeng. Atlas of Uveitis: Diagnosis and Treatment. Springer Singapore Pte. Limited, 2020.

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10

Yang, Peizeng. Atlas of Uveitis: Diagnosis and Treatment. Springer Singapore Pte. Limited, 2021.

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11

Hughes, Edward, Miles Stanford, and Dania Qatarneh. Uveitis and medical ophthalmology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199672516.003.0007.

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This chapter focuses on uveitis and medical ophthalmology. It details uveal anatomy, before discussing anterior uveitis, intermediate uveitis, and posterior uveitis. Next, it discusses posterior uveitis and then specific non-infectious posterior uveitides before moving on to infectious uveitis (viral, parasitic, fungal, and bacterial) and HIV-related disease. It continues by examining systemic associations with uveitis, including sarcoidosis, Behcets disease, multiple sclerosis, and the seronegative arthritides ankylosing spondylitis, Reiter syndrome, psoriatic arthritis, and inflammatory bowel disease. In addition, it discusses scleritis and episcleritis, ocular inflammatory disorders (systemic treatment, biological agents and periocular treatments, intraocular treatments), rheumatological disease, vasculitis, cardiovascular disease and the eye, and endocrine, respiratory, and skin diseases in ophthalmology.
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12

Rao, Narsing A., Julie Schallhorn, and Damien C. Rodger. Posterior Uveitis: Advances in Imaging and Treatment. Springer, 2019.

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13

Kanski, Jack J. Uveitis: A Colour Manual of Diagnosis and Treatment (Colour Manuals in Ophthalmology). Butterworth-Heinemann, 1987.

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14

Hughes, Edward, and Miles Stanford. Medical ophthalmology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199237593.003.0005.

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The chapter begins with an introduction to uveal anatomy. The following areas of clinical knowledge are then discussed: anterior uveitis, intermediate uveitis, posterior uveitis, specific non-infectious posterior uveitides, viral infectious uveitis, parasitic infectious uveitis, fungal infectious uveitis, bacterial infectious uveitis, HIV-related disease, the systemic associations of uveitis, scleritis and episcleritis, systemic treatment of ocular inflammatory disorders, biological agents and periocular treatments of ocular inflammatory disorders, intraocular treatments of ocular inflammatory disorders, rheumatological disease, vasculitis, cardiovascular disease and the eye, endocrine diseases in ophthalmology, and respiratory and skin diseases in ophthalmology. Practical skills are also addressed, namely orbital-floor injection, posterior sub-Tenon injection, and intravitreal injection. The chapter concludes with a case-based discussion on scleritis.
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15

J, Huang John, and Gaudio Paul A, eds. Ocular inflammatory disease and uveitis manual: Diagnosis and treatment. Philadelphia: Wolters Kluwer/Lippincott Williams & Wilkins Health, 2010.

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16

Rosenbaum, James T. Extra-articular manifestations: uveitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0016.

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Acute anterior uveitis (AAU) is the most common, extra-articular, clinical manifestation of spondyloarthritis. In some patients, it is the dominant manifestation. This chapter reviews the definition of uveitis, the epidemiology of uveitis, the differential diagnosis, the relationship of HLA-B27 to uveitis, laboratory testing for suspected HLA-B27-associated uveitis, treatment considerations, and experimental models in which both uveitis and spondylitis co-exist. Although acute anterior uveitis rarely precedes spondyloarthritis, acute anterior uveitis is often the clue that chronic back pain is due to sacroiliac inflammation. Uveitis is sometimes the dominant clinical manifestation of spondyloarthritis and it can influence the choice for appropriate therapy.
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17

Uveitis: A Practical Guide to the Diagnosis and Treatment of Intraocular Inflammation. Springer, 2017.

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18

Papaliodis, George N. Uveitis: A Practical Guide to the Diagnosis and Treatment of Intraocular Inflammation. Springer London, Limited, 2016.

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19

Coates, Laura C., Arthur Kavanaugh, and Christopher T. Ritchlin. Treatment algorithm and treat to target. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0032.

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This chapter covers the evidence for treatment algorithms and treatment to target in PsA. Evidence for treatment algorithms including step up vs step down approaches to prescribing and early vs delayed treatment is discussed. EULAR recommendations for treating to target in SpA are outlined with a summary of the level of evidence available at that time. Key outcome measures that could be utilized as targets in PsA are reviewed with discussion of their merits and deficiencies. A detailed description of the first treat to target study in PsA is presented: the TICOPA study. The impact of comorbidities on treatment decisions is discussed, both related SpA conditions such as uveitis and inflammatory bowel disease, and non-SpA comorbidities such as the metabolic syndrome and liver disease. Finally, suggestions for translation into clinical practice are outlined, highlighting the need for multi-speciality collaborative working, full assessment of disease activity and subsequent optimal treatment.
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20

Miserocchi, E., G. Modorati, and C. S. Foster, eds. New Treatments in Noninfectious Uveitis. S. Karger AG, 2012. http://dx.doi.org/10.1159/isbn.978-3-8055-9987-0.

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21

Foster, Brogan, and Paul A. Brogan. Juvenile idiopathic arthritis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738756.003.0003.

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This chapter describes the JIA subtypes, uveitis, prognostic indicators, the spectrum of JIA in adults and updated chapters on the genetics and immunology of JIA. There are updated sections on treatment approaches, pathways (including reference to NICE and guidance from North America and Europe) and disease activity scores.
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22

Michet, Clement J., Kenneth G. Moder, and William W. Ginsburg. Rheumatology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0681.

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Major rheumatologic diseases are reviewed in detail, including symptoms, diagnosis, and treatment. Rheumatoid arthritis, Sjogren syndrome, Felty syndrome, osteoarthritis, fibromyalgia, and vasculitic syndromes like giant cell arteritis, polyarteritis nodosa, Takayasu arteritis, and Wegener granulomatosis are included. Drugs used to treat rheumatic disease are also highlighted. They include NSAIDs, antimalarials, and glucocorticosteroids. Crystalline arthropathies, such as hyperuricemia and gout, are another class of rheumatologic disease, as are spondyloarthropathies, systemic lupus erythematosus, uveitis, and Behcet syndrome.
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23

Foster, Brogan, and Paul A. Brogan. Systemic diseases. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738756.003.0004.

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This chapter covers the full spectrum of systemic diseases in paediatric rheumatology including: the systemic vasculitides (HSP, Kawasaki disease, PAN, ANCA-associated vasculitis, Takayasu arteritis, Behçet's disease, cerebral vasculitis, and many others); juvenile SLE; scleroderma; JDM; overlap syndromes; antiphospholipid syndrome; sarcoid; and paediatric uveitis. In addition, it provides updated descriptions and treatment approaches for autoinflammatory diseases, including recently described diseases such as DADA, SAVI, CANDLE, and many others. Other systemic diseases described in detail include mucopolysaccharidoses and mucolipidoses; musculoskeletal features of chromosomal abnormalities; cystic fibrosis; and inflammatory bowel disease. Treatment guidelines for all these systemic diseases have been fully updated, and aligned with recent evidence-based/consensus European guidance.
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24

Petrushkin, Harry, and Miles Stanford. The eye. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0024.

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The ophthalmologist has a large part to play in the management of many rheumatological diseases. These diseases can cause a number of symptoms from mild ocular discomfort to sudden blindness. In addition, many rheumatological diseases have helpful ophthalmic signs, which can aid diagnosis. This chapter has been written to help rheumatologists identify these signs and symptoms. We have started by summarizing the common pathology found in patients with rheumatological diseases (dry eye syndromes, conjunctivitis, episcleritis, scleritis, uveitis, and optic neuropathy). This has been arranged working backwards from the front of the eye towards the retina and optic nerve. The rheumatological conditions that give rise to ophthalmic signs (giant cell arteritis, systemic lupus erythematosus, polyarteritis nodosa, Wegner's granulomatosis, systemic sclerosis, rheumatoid arthritis, seronegative arthropathies, sarcoidosis, and Behçet’s disease) have then been summarized, including a section of paediatric conditions (juvenile idiopathic arthritis, spondyloarthropathies, and multisystemic illness). Finally, treatment regimes and recent guidelines have been covered for the screening of uveitis in juvenile idiopathic arthritis and the management of patients taking hydroxychloroquine. We hope that both rheumatologists in training and consultants find this chapter a useful clinical aid, and that it encourages them to look closely for subtle signs that will help improve the management of their patients.
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25

Petrushkin, Harry, and Miles Stanford. The eye. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0024_update_001.

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The ophthalmologist has a large part to play in the management of many rheumatological diseases. These diseases can cause a number of symptoms from mild ocular discomfort to sudden blindness. In addition, many rheumatological diseases have helpful ophthalmic signs, which can aid diagnosis. This chapter has been written to help rheumatologists identify these signs and symptoms. We have started by summarizing the common pathology found in patients with rheumatological diseases (dry eye syndromes, conjunctivitis, episcleritis, scleritis, uveitis, and optic neuropathy). This has been arranged working backwards from the front of the eye towards the retina and optic nerve. The rheumatological conditions that give rise to ophthalmic signs (giant cell arteritis, systemic lupus erythematosus, polyarteritis nodosa, Wegner’s granulomatosis, systemic sclerosis, rheumatoid arthritis, seronegative arthropathies, sarcoidosis, and Behçet’s disease) have then been summarized, including a section of paediatric conditions (juvenile idiopathic arthritis, spondyloarthropathies, and multisystemic illness). Finally, treatment regimes and recent guidelines have been covered for the screening of uveitis in juvenile idiopathic arthritis and the management of patients taking hydroxychloroquine. We hope that both rheumatologists in training and consultants find this chapter a useful clinical aid, and that it encourages them to look closely for subtle signs that will help improve the management of their patients.
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26

Haghighi, Afshin Borhani, and Bernadette Kalman. Other Proven and Putative Autoimmune Disorders of the CNS. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0094.

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Behcet’s Disease (BD) is a multiorgan disorder characterized by oral and genital ulceration, uveitis, and dermatological symptoms. BD is most prevalent in the Mediterranean countries and East Asia, but also occurs in Europe and North America. The etiology remains unknown. Evidence suggests that BD is an autoimmune disorder with complex traits. Neuro-Behcet’s Syndome (NBS) develops in about 5% to 30% of patients with BD and presents with parenchymal or nonparenchymal pathology. The course of NBS is highly variable. Treatment strategies include modulations of the immune response and tissue degeneration, along with symptomatic medications. Main directions of current research include genomic studies, biomarker discovery, and inventive drug- development strategies.
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27

Siebert, Stefan, Sengupta Raj, and Alexander Tsoukas. Extra-articular manifestations of axial spondyloarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198755296.003.0008.

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Axial spondyloarthritis (axSpA) is associated with a number of extra-articular manifestations (EAMs) reflecting shared clinical, genetic, and pathophysiologicalfeatures. The EAMs are considered part of the spondyloarthritis spectrum and should be differentiated from complications arising as a consequence of the disease (covered in a separate chapter). The key EAMs of axSpA are inflammatory bowel disease (IBD), uveitis, and psoriasis. EAMs carry their own morbidity and often warrant treatment in their own right. Furthermore, the presence of EAMs may influence the choice of therapy used to treat the underlying axSpA. Clinicians should ensure that the well-recognized EAMs of axSpA are actively and regularly enquired about when reviewing patients with axSpA.
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28

DeAugustinas, M., and A. Kiely. Endophthalmitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0017.

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Endophthalmitis refers to inflammation of both the anterior and posterior intraocular chambers and their structures. This vision-threatening condition occurs in three principal scenarios: penetrating ocular trauma, after intraocular surgery, and in systemically infected (often immunocompromised) patients. Endophthalmitis presents with marked intraocular inflammation, often with hypopyon. Patients report pain and significant vision loss out of proportion to typical post-operative complaints. It is distinguished from uveitis by both history and slit lamp examination. Endophthalmitis is an ophthalmic emergency. Same day ophthalmology consult/referral is mandatory. Vision is threatened over the course of hours. Treatment includes prompt intravitreal injection of antibiotics by an ophthalmologist. Systemic therapy should occur only in in endogenous infections, continuing until cultures clear.
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29

Baildam, Eileen. Juvenile idiopathic arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0116.

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Juvenile idiopathic arthritis (JIA) is defined as arthritis lasting for 6 weeks or more presenting in childhood at any age up to 17 years. Arthritis is diagnosed clinically by the presence of joint pain, stiffness, and swelling with inflammation limiting the range of individual joint movement. There are subtypes that tend to follow distinct courses and with phenotypes that vary widely from a serious systemic inflammatory disorder of systemic JIA to single-joint monoarthritis. The differential diagnosis of JIA is wide and the best chance of long-term remission is where treatment is started as early as possible. However, there is often delay in diagnosis in childhood and there is no single reliable diagnostic test so pattern recognition is fundamental. There are associated disorders such as silent uveitis that must be screened for and managed as part of essential multidisciplinary care. Systemic JIA is complicated by potentially life-threatening macrophage activation syndrome that is often underdiagnosed but where the diagnosis is based on easy clinical tests and where awareness is vital. This chapter covers descriptions of the classification criteria for chronic arthritis in children, clinical presentations and likely course for the various subtypes.
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30

Sieper, Joachim. Ankylosing spondylitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0113.

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Ankylosing spondylitis (AS) is a chronic inflammatory disease predominantly of the sacroiliac joint (SIJ) and the spine. It starts normally in the second decade of life and has a slight male predominance. The prevalence is between 0.2 and 0.8% and is strongly dependent on the prevalence of HLA B27 in a given population. For the diagnosis of AS, the presence of radiographic sacroiliitis is mandatory. However, radiographs do not detect active inflammation but only structural bony damage. Most recently new classification criteria for axial spondyloarthritis (SpA) have been developed by the Assessement of Spondylo-Arthritis international Society (ASAS) which cover AS but also the earlier form of non-radiographic axial SpA. MRI has become an important new tool for the detection of subchondral bone marrow inflammation in SIJ and spine and has become increasingly important for an early diagnosis. HLA B27 plays a central role in the pathogenesis but its exact interaction with the immune system has not yet been clarified. Besides pain and stiffness in the axial skeleton patients suffer also from periods of peripheral arthritis, enthesitis, and uveitis. New bone formation as a reaction to inflammation and subsequent ankylosis of the spine determine long-term outcome in a subgroup of patients. Currently only non-steroidal anti-inflammatory drugs (NSAIDs) and tumour necrosis factor (TNF) blockers have been proven to be effective in the medical treatment of axial SpA, and international ASAS recommendations for the structured management of axial SpA have been published based on these two types of drugs. Conventional disease-modifying anti-rheumatic drugs (DMARDs) such as methotrexate are not effective.
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31

Sieper, Joachim. Axial spondyloarthropathies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0113_update_003.

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Axial spondyloarthritis (axSpA) is a chronic inflammatory disease predominantly of the sacroiliac joint (SIJ) and the spine. It starts normally in the second decade of life and has a slight male predominance. The prevalence is between 0.2% and 0.8% and is strongly dependent on the prevalence of HLA-B27 in a given population. AxSpA can be split in patients with radiographic axSpA (also termed ankylosing spondylitis (AS)) and in patients with non-radiographic axSpA (nr-axSpA). For the diagnosis of AS, the presence of radiographic sacroiliitis is mandatory. However, radiographs do not detect active inflammation but only structural bony damage. Most recently new classification criteria for axSpA have been developed by the Assessment of Spondylo-Arthritis International Society (ASAS) which cover AS but also the earlier form of nr-axSpA. MRI has become an important new tool for the detection of subchondral bone marrow inflammation in SIJ and spine and has become increasingly important for an early diagnosis. HLA-B27 plays a central role in the pathogenesis but its exact interaction with the immune system has not yet been clarified. Besides pain and stiffness in the axial skeleton patients suffer also from periods of peripheral arthritis, enthesitis, and uveitis. New bone formation as a reaction to inflammation and subsequent ankylosis of the spine determine long-term outcome in a subgroup of patients. Currently only non-steroidal anti-inflammatory drugs (NSAIDs) and tumour necrosis factor (TNF) blockers have been proven to be effective in the medical treatment of axial SpA, and international ASAS recommendations for the structured management of axial SpA have been published based on these two types of drugs. Conventional disease-modifying anti-rheumatic drugs (DMARDs) such as methotrexate are not effective.
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