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1

Brandes, Steven B., ed. Urethral Reconstructive Surgery. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-103-1.

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2

Schreiter, F., and G. H. Jordan, eds. Urethral Reconstructive Surgery. Berlin/Heidelberg: Springer-Verlag, 2006. http://dx.doi.org/10.1007/3-540-29385-x.

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3

Brandes, Steven B., and Allen F. Morey, eds. Advanced Male Urethral and Genital Reconstructive Surgery. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-7708-2.

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4

Khattar, Nikhil, Rishi Nayyar, and Arabind Panda, eds. Female Bladder Outlet Obstruction and Urethral Reconstruction. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-8521-0.

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5

Falkiner, F. R. The insertion and management of indwelling urethral catheters. [London]: [Royal College of Physicians], 1993.

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6

Droller, Michael J. Urothelial tumors. Hamilton, Ont: B C Decker, 2004.

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7

Dogra, Vikram S., and Gregory T. MacLennan, eds. Genitourinary Radiology: Kidney, Bladder and Urethra. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-84800-245-6.

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8

George, N. J. R., and J. A. Gosling, eds. Sensory Disorders of the Bladder and Urethra. London: Springer London, 1986. http://dx.doi.org/10.1007/978-1-4471-1392-8.

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9

Sand, Peter K. Urodynamics and the evaluation of female incontinence: A practical guide. London: Springer-Verlag, 1995.

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10

1958-, Bracewell Michael, ed. The postcard art of Gilbert & George, 1972-1989: The urethra postcard art of Gilbert & George, 2009. Munich: DelMonico Books/Prestel, 2011.

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11

1930-, Mackay Bruce, ed. Atlas of bladder pathology. New York: Igaku-Shoin, 1991.

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12

Brodie, David A. Therapy markets for gastrointestinal and urinary bladder cancers. Waltham, Mass: Decision Resources, 1998.

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13

G, Notley R., ed. Transurethral resection. 4th ed. Oxford: Isis Medical Media, 1998.

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14

Schreuder, Michiel F. Posterior urethral valves. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0354.

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Posterior urethral valves is the most common congenital cause of lower urinary tract obstruction in males, and a common cause (15–17%) for end-stage renal disease in childhood. Most commonly, posterior urethral valves is suspected on basis of a screening antenatal ultrasound. Ultrasound will not detect posterior urethral valves itself, but recognizes the consequences of lower urinary tract obstruction with a dilated thick-walled bladder and dilation of the prostatic portion of the urethra. After birth, urine drainage has to be secured by placement of a bladder catheter, and imaging is needed to confirm the presence of the urethral valves and estimate the degree of damage to the kidney. Consequences of posterior urethral valves depend on the degree of renal dysplasia and bladder dysfunction. Prevention or minimization of such consequences by intrauterine urine drainage has not definitively shown a benefit of early vesico-amniotic shunting.
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15

Urethral reconstruction. Philadelphia: Saunders, 2002.

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16

Turner-Warwick, Richard T., Richard Inman, and Christopher R. Chapple. Urethral Reconstruction. Springer London, Limited, 2020.

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17

Osman, Nadir I., and Christopher R. Chapple. Urethral diverticula. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0042.

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Urethral diverticula (UD) are not uncommon, affecting up to 6% of the female population, but are in the majority of cases asymptomatic and of no clinical significance. They are thought to arise as a consequence of infection, obstruction, and subsequent rupture of a periurethral gland. UD often pose a significant diagnostic challenge, as symptoms are largely non-specific and easily confused with other conditions such as bladder pain syndrome and recurrent urinary tract infection. As such, both misdiagnosis and delayed diagnosis are common, to the frustration of both patients and surgeons. Traditional methods of diagnosis, such as voiding cystourethrogram, relied upon urethral catheterization and contrast instillation, and were associated with poor anatomical detail. Recent advances in imaging, particularly magnetic resonance imaging, have improved the diagnosis and staging of UD, and have allowed for more accurate preoperative planning.
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18

Mundy, Anthony R., and Daniela E. Andrich. Urethral strictures. Edited by Anthony R. Mundy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0050.

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Urethral strictures are common and almost all urologists will deal with them on a regular if not daily basis. They have always been common and the history of the subject stretches back to 3,000 BC. Urethral dilators have been found in the tombs of the pharaohs so that they might be able to catheterize themselves or dilate their own strictures in the afterlife. Urethrotomy and dilatation are two of the most frequently performed procedures in urology. But these are usually only palliative, and curative treatment by urethroplasty is performed by very few urologists. In part this is because most strictures are bulbar strictures and most non-bulbar strictures are seen only by reconstructive urologists; but in part this represents a somewhat ambivalent attitude of most urologists to urethral stricture disease. In this chapter, we will attempt to clarify the current approach to this problem.
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19

Urethral reconstructive surgery. Heidelberg: Springer, 2005.

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20

Reconstructive Urethral Surgery. Springer, 2005.

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21

Lippincott. Nursims Pediatric Urethral. Lippincott Williams & Wilkins, 1995.

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22

Hutton, Kim, and Ashok Daya Ram. Disorders of the urethra. Edited by David F. M. Thomas. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0117.

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Most disorders of the urethra in children are congenital in origin and affect boys more commonly than girls. They include; posterior urethral valves (PUV), anterior urethral valves, anterior urethral diverticulum, syringocele, urethral atresia, megalourethra, urethral web, urethral polyp, and urethral duplication. Urethral strictures may be congenital or acquired. Most cases of PUV are now diagnosed prenatally. Postnatal management comprises bladder drainage, correction of any metabolic disturbance, prevention of infection (UTI), and endoscopic valve ablation. Careful follow up is required with the aim of preventing urosepsis and preserving renal function. Persisting bladder dysfunction (‘valve’ bladder) can threaten renal function and should be managed aggressively. Chronic renal failure ultimately affects a third of boys with PUV, of whom 10–20% require renal transplantation during the course of childhood. PUV may also present clinically with recurrent UTI, urinary incontinence, or unexplained lower urinary tract symptoms.
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23

Norsworthy, Gary D. Urethral Obstruction in Male Cats. Teton New Media, 2000.

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24

Norsworthy, Gary D. Urethral Obstruction in Male Cats. Teton New Media, 2000.

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25

Urethral Reconstructive Surgery (Current Clinical Urology). Humana Press, 2008.

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26

Brandes, Steven B., and Allen F. Morey. Advanced Male Urethral and Genital Reconstructive Surgery. Humana, 2013.

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27

Endoscopic Diagnosis and Treatment in Urethral Pathology. Elsevier, 2016. http://dx.doi.org/10.1016/c2013-0-19326-9.

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28

Brandes, Steven B., and Allen F. Morey. Advanced Male Urethral and Genital Reconstructive Surgery. Humana, 2016.

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29

Daly, Donna, and Christopher Chapple. Anatomy, neurophysiology, and pharmacological control mechanisms of the bladder. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0034.

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The lower urinary tract has two main functions; the collection and low pressure storage of urine and periodical controlled elimination of urine at an appropriate time. In order to achieve continence during bladder filling and storage and produce efficient and effective bladder emptying, there is accurate coordination between opening and closing of the urethral sphincters and contraction of the detrusor smooth muscle. The process of micturition has two phases: the storage/filling phase and the voiding phase. The analogy for the transition between these two phases has been described as an on-off circuit, rather akin to flicking a light switch, between synchronous bladder contraction and urethral outlet relaxation, and vice versa. These phases are regulated by a complex, integration of somatic and autonomic efferent and afferent mechanisms that coordinate the activity of the bladder and urethra. This chapter provides an overview of our current understanding of these complex mechanisms.
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30

Wilson, John W., and Lynn L. Estes. Sexually Transmitted Diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199797783.003.0121.

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• Abrupt-onset, purulent urethral discharge and dysuria are more common with Neisseria gonorrhoeae than with Chlamydia trachomatis and other nongonococcal urethritis (NGU) pathogens• Mucopurulent or purulent urethral discharge and dysuria, which can occur with any pathogen, often impede clinical distinction• Gram stain of urethral discharge shows >5 leukocytes per high-power field (HPF)...
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31

Geavlete, Petrisor Aurelian. Endoscopic Diagnosis and Treatment in Urethral Pathology: Handbook of Endourology. Elsevier Science & Technology, 2015.

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32

Female Urinary Incontinence in Practice (In Practice). Royal Society of Medicine Press, 2004.

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33

Keyes, E. L. The Venereal Diseases: Including The Structure Of The Male Urethra. Kessinger Publishing, LLC, 2007.

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34

Bladder and urethra. Salt Lake City, UT: Medicode, Med-Index Division, 1994.

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35

Alessandro, Riva, Testa Riva Francesca, and Motta Pietro M, eds. Ultrastructure of the male urogenital glands: Prostate, seminal vesicles, urethral, and bulbourethral glands. Boston: Kluwer Academic Publishers, 1994.

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36

Rosemary, Crow, and University of Surrey. Nursing Practice Research Unit., eds. A Study of patients with an indwelling urethral catheter and related nursing practice. Guildford: University of Surrey, Nursing Practice Research Unit, 1986.

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37

George, N. J. R. 1946- and Gosling J. A. 1939-, eds. Sensory disorders of the bladder and urethra. Berlin: Springer-Verlag, 1986.

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38

Thomas, James, Tanya Monaghan, and Prarthana Thiagarajan. Practical procedures. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199593972.003.0018_update_001.

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Using this chapterInfiltrating anaesthetic agentsHand hygieneConsentAseptic techniqueSubcutaneous and intramuscular injectionsIntravenous injectionsVenepunctureSampling from a central venous catheterArterial blood gas (ABG) samplingPeripheral venous cannulationFemoral venous catheter insertionCentral venous access: internal jugular veinCentral venous access: subclavian veinCentral venous access: ultrasound guidanceIntravenous infusionsArterial line insertionFine needle aspiration (FNA)Lumbar punctureMale urethral catheterizationFemale urethral catheterizationBasic airway managementOxygen administrationPeak expiratory flow rate (PEFR) measurementInhaler techniqueNon-invasive ventilationPleural fluid aspirationPneumothorax aspirationChest drain insertion (Seldinger)Recording a 12-lead ECGCarotid sinus massageVagal manoeuvresTemporary external pacingDC cardioversionPericardiocentesisNasogastric tube insertionAscitic fluid sampling (ascitic tap)Abdominal paracentesis (drainage)Sengstaken–Blakemore tube insertionBasic interrupted suturingCleaning an open woundApplying a backslabManual handling
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39

Reynard, John, and Ben Turney. Bladder stones. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0030.

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The majority of bladder stones in Western practice are secondary to underlying pathology—bladder outlet obstruction due to benign prostatic enlargement in men and urethral obstruction from pelvic prolapse or cystocele in women, chronic infection in the neuropathic or augmented bladder, or in the neobladder. While the pathology of endemic bladder stones remains as it always was, the advent of augmentation cystoplasty and rising use of the neobladder after cystectomy has, through a different pathological mechanism, led to a rise in frequency of bladder stones. The mode of presentation of bladder stones and diagnostic technique are reviewed in this chapter. Treatment options are determined, to a significant degree, by the clinical context in which the stone arise, the major determinant of the approach to such stones being the calibre of the conduit (urethra or Mitrofanoff) through which access to the bladder is achieved.
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40

Frenier, Susan L. Urethral pressure response to alpha adrenergic agonist and antagonist drugs in the normal male cat. 1990.

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41

Publications, ICON Health. The Official Patient's Sourcebook on Urethral Cancer: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2002.

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42

Marks, Steven L. The effects of phenoxybenzamine and acepromazine maleate on urethral pressure profiles of anesthetized healthy male cats. 1993.

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43

Fox, Grenville, Nicholas Hoque, and Timothy Watts. Endocrinology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198703952.003.0016.

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This chapter provides background information on fetal and neonatal renal development, physiology, and function. Detailed information is given on management of common, antenatally diagnosed, renal anomalies (renal pelvis dilatation/hydronephrosis); post-natal diagnoses (hypospadius, hydrocele); posterior urethral valves; polycystic kidney disease; and rarer diagnoses. There is a guideline on the management of acute renal failure in the newborn, and information on dialysis.
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44

Ultrastructure of Male Urogenital Glands: Prostate, Seminal Vesicles, Urethral, and Bulbourethral Glands (Electron Microscopy in Biology and Medicine). Springer, 1994.

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45

Carton, James. Urological pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0011.

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This chapter discusses urological pathology and covers diseases of the urinary tract managed by urologists. This includes genitourinary malformations, urinary tract infection, urinary tract obstruction, urinary calculi, cystic renal diseases, benign renal tumours, renal cell carcinoma (RCC), childhood renal tumours, urothelial carcinoma, benign prostatic hyperplasia (BPH), prostate carcinoma, testicular germ cell tumours, testicular non-germ cell tumours, paratesticular diseases, urethral diseases, penile diseases, and scrotal diseases.
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46

Corcos, Jacques. The Urinary Sphincter. Informa Healthcare, 2001.

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47

Comprehensive Urology. Mosby, 2001.

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48

Norris, Marion. Patients' perceptions of how to care for their in-dwelling urethral catheter, plus obtaining further supplies and help: An investigative study. 1990.

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49

Cystitis: A Time to Heal with Yoga & Accupressure, An Eight Week Exercise Program with Special Information on Interstitial Cystitis & Urethral Syndrome. 1st Books Library, 2003.

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50

Mundy, Anthony R., and Daniela E. Andrich. Lower urinary tract reconstruction. Edited by Anthony R. Mundy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0049.

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In clinical practice, most reconstructive urological procedures performed are for urethral strictures and otherwise on the lower urinary tract. Bladder augmentation and substitution are widely used by urologists and increasingly so in other subspecialties across the spectrum of urology for what used to be reconstructive problems, but new problems have developed to challenge reconstructive urologists, requiring a different approach but based on the same general principles. Thus, as augmentation and substitution cystoplasty become increasingly part of ‘general urology’, so reconstructive urologists are adapting these principles to deal with the complications of the treatment of prostate cancer, gynaecological cancer and rectal cancer.
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