Relevant bibliographies by topics / Upper digestive tract cancer

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Academic literature on the topic 'Upper digestive tract cancer'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Upper digestive tract cancer"

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Reed, P. I. "Micronutrients and upper digestive tract cancer." European Journal of Cancer Prevention 6, no. 5 (October 1997): 494. http://dx.doi.org/10.1097/00008469-199710000-00037.

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M., PRABHAKARAN. "Upper Aero Digestive Tract Cancer: A Detailed Exploration Using Computer Vision Techniques." Journal of Research on the Lepidoptera 51, no. 2 (June 25, 2020): 648–65. http://dx.doi.org/10.36872/lepi/v51i2/301124.

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Turati, Federica, Marta Rossi, Claudio Pelucchi, Fabio Levi, and Carlo La Vecchia. "Fruit and vegetables and cancer risk: a review of southern European studies." British Journal of Nutrition 113, S2 (April 2015): S102—S110. http://dx.doi.org/10.1017/s0007114515000148.

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High intakes of fruit and vegetables may reduce the risk of cancer at several sites. Evidence has been derived mainly from case–control studies. We reviewed the relationship between consumption of vegetables and fruit and the risk of several common cancers in a network of Italian and Swiss case–control studies including over 10 000 cases of fourteen different cancers and about 17 000 controls. Data were suggestive of a protective role of vegetable intake on the risk of several common epithelial cancers. OR for the highest compared with the lowest levels of consumption ranged from 0·2 (larynx, oral cavity and pharynx) to 0·9 (prostate). Inverse associations were found for both raw and cooked vegetables, although for upper digestive tract cancers the former were somewhat stronger. Similar inverse associations were found for cruciferous vegetables. Frequent consumption of allium vegetables was also associated with reduced risk of several cancers. Fruit was a favourable correlate of the risk of several cancers, particularly of the upper digestive tract, with associations generally weaker than those reported for vegetables. A reduced risk of cancers of the digestive tract and larynx was found for high consumption of citrus fruit. Suggestive protections against several forms of cancer, mainly digestive tract cancers, were found for high consumption of apples and tomatoes. High intakes of fibres, flavonoids and proanthocyanidins were inversely related to various forms of cancer. In conclusion, data from our series of case–control studies suggested a favourable role of high intakes of fruit and vegetables in the risk of many common cancers, particularly of the digestive tract. This adds evidence to the indication that aspects of the Mediterranean diet may have a favourable impact not only on CVD, but also on several common (epithelial) cancers, particularly of the digestive tract.
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Bosetti, Cristina, Claudio Pelucchi, and Carlo La Vecchia. "Diet and cancer in Mediterranean countries: carbohydrates and fats." Public Health Nutrition 12, no. 9A (September 2009): 1595–600. http://dx.doi.org/10.1017/s1368980009990425.

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AbstractObjectiveSeveral aspects of the diet characteristic of the Mediterranean countries are considered favourable not only on cardiovascular disease, but also on cancer risk. We considered some aspects of the Mediterranean diet (including, in particular, the consumption of olive oil and carbohydrates) on cancer risk.Design, Setting and SubjectsData were derived from a series of case-control studies, conducted in Italy since the early 1990s, on over 10 000 cases of thirteen cancer sites and over 17 000 controls.ResultsOlive oil, and other mono- and unsaturated fats, appear to be favourable indicators of breast, ovarian, colorectal, but mostly of upper aero-digestive tract cancers. Whole grain foods are also related to reduced risk of upper aero-digestive tract and various other cancers. In contrast, refined grain intake and, consequently, glycaemic index and glycaemic load were associated to increased risk for several cancer sites. Fish, and hence a diet rich in n-3 polyunsaturated fatty acids, tended to be another favourable diet indicator, while frequent red meat intake was directly related to some common neoplasms. An a priori defined Mediterranean diet score was inversely related to upper digestive and respiratory tract cancers.ConclusionsThese data provide additional evidence that major characteristics of the Mediterranean diet favourably affect cancer risk.
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Song, Yinghao, Yulei Qu, Lu Li, Mengxuan Xing, Baohui Jia, Yingjie Ma, and Yong Zhang. "Application Value of the Diagnosis during Early Carcinoma of Upper Digestive Tract Based on Optical Enhanced Endoscopic Technique." Computational and Mathematical Methods in Medicine 2022 (July 19, 2022): 1–6. http://dx.doi.org/10.1155/2022/9587070.

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Objective. The diagnostic value of optical enhanced endoscopy in early cancer of upper digestive tract was studied by comparing the disease accuracy, tumor type, invasion, and various surgical indicators between the two groups. Methods. 188 patients with early upper gastrointestinal cancer treated in our hospital from January 2020 to February 2021 were selected as the research objects. The patients were randomly divided into the observation group and control group with 94 cases in each group. Results. The accuracy of early detection of early carcinoma of upper digestive tract in the observation group was 94.68% and that in the control group was 76.60%. The accuracy of the observation group was significantly higher than that in the control group, with statistical significance ( P < 0.05 ). In the observation group, 36 cases of early gastric cancer, 28 cases of early esophageal cancer, and 30 cases of early colorectal cancer were detected; 25 cases of early gastric cancer, 19 cases of early esophageal cancer, and 28 cases of early colorectal cancer were detected; 26 cases of early carcinoma of upper digestive tract infiltration were detected; and 68 cases were not detected, and the detection rate was 27.66%, which was higher than 9.57% in the control group, and the difference was statistically significant ( P < 0.05 ). After different methods of treatment, no death occurred in all patients. Except for the operation time, the surgical indexes of the observation group were better than the control group, the difference was statistically significant ( P < 0.05 ). Conclusion. Optical enhanced endoscopic technique had obvious effect in the diagnosis of patients with early cancer of upper digestive tract, it was helpful to improve the clinical detection rate of early carcinoma of upper digestive tract and had certain diagnostic ability for the invasion depth of early cancer of high upper gastrointestinal tract, which was conducive to the detection of clinical invasion lesions and had high clinical promotion and application value.
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Jiang, Xingyu, Qi Liang, Huanhuan Xu, Shouyong Gu, and Lingxiang Liu. "The Association of Waist Circumference with the Prevalence and Survival of Digestive Tract Cancer in US Adults: A Population Study Based on Machine Learning Methods." Computational and Mathematical Methods in Medicine 2022 (October 6, 2022): 1–11. http://dx.doi.org/10.1155/2022/2492488.

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Aims. This paper aims to investigate the relationship of waist circumference (WC) with digestive tract cancer morbidity and mortality. Methods. Based on the data from a nationally representative US population survey, we summarized the prevalence of digestive tract cancer and all-cause mortality of cancer patients across WC quartiles. Adjusted logistic regression and restricted spline curve were used to analyze WC and the prevalence of digestive tract cancer. Moreover, Cox regression and the Kaplan-Meier curve were applied to investigate the association of WC with all-cause mortality. We also attempted to make a model to predict cancer happening. Results. This paper included a total of 34,041 participants, with digestive tract cancer observed in 265 (0.7%) individuals. WC was positively associated with digestive tract cancer morbidity after full adjustment of covariates (OR: 1.72 and 95% CI: 1.41-2.10). Also, individuals in the highest WC group had a higher risk of digestive tract cancer (Q4, OR: 2.71 and 95% CI: 1.48-5.00). Moreover, no significant association was observed in upper digestive cancer, and WC was associated with a longer survival time once diagnosed (hazard ratio (HR): 0.50 and 95% CI: 0.28-0.92). Finally, the model we made proved to be effective. Conclusion. High WC is a risk factor for digestive tract cancer with or without adjusting for body mass index, especially those located in the lower digestive tract. However, once digestive tract cancer has been diagnosed, patients with higher WC showed better survival outcomes. Moreover, machine learning methods can be used to predict digestive tract cancer risk in the future.
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Gupta, Bhawna, Ratilal Lalloo, and Newell W. Johnson. "Life course models for upper aero-digestive tract cancer." International Dental Journal 65, no. 3 (June 2015): 111–19. http://dx.doi.org/10.1111/idj.12167.

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Vecchia, C. L., S. Franceschi, A. Favero, R. Talamini, E. Negri, K. K. Cheng, C. Cummins, et al. "Alcohol intake and cancer of the upper digestive tract." BMJ 318, no. 7193 (May 8, 1999): 1289. http://dx.doi.org/10.1136/bmj.318.7193.1289b.

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Farinati, F., R. Cardin, M. Zordan, F. Valiante, A. J. Garro, P. Burra, P. Venier, D. Nitti, A. G. Levis, and R. Naccarato. "Alcohol metabolism in the upper digestive tract." European Journal of Cancer Prevention 1 (October 1992): 25–32. http://dx.doi.org/10.1097/00008469-199210003-00004.

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Haguenoer, J. M., S. Cordier, C. Morel, J. L. Lefebvre, and D. Hemon. "Occupational risk factors for upper respiratory tract and upper digestive tract cancers." Occupational and Environmental Medicine 47, no. 6 (June 1, 1990): 380–83. http://dx.doi.org/10.1136/oem.47.6.380.

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Dissertations / Theses on the topic "Upper digestive tract cancer"

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Sharpe, Katharine H. "Socioeconomic inequalities in lung and upper aero-digestive tract cancer incidence in Scotland." Thesis, University of Glasgow, 2018. http://theses.gla.ac.uk/8887/.

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Socioeconomic inequality in cancer risk and incidence burden has received limited attention compared to genetic and behavioural risk factors. Where they have been studied, the temporal relationship between socioeconomic factors and cancer risk has been under explored due to the mainly cross-sectional nature of most research. Moreover, the inter-relationships of the multiple measures of socioeconomic status and, in particular, area and individual measures and their interaction with risk behaviours have also had limited attention. The overarching aim of this thesis was to investigate socioeconomic inequalities in the risk of lung and upper aero-digestive tract cancers and the relationship between this risk and socioeconomic status, area and individual based measures of socioeconomic circumstances, and behaviours such as smoking, alcohol consumption, diet and exercise. To understand and quantify the relative contribution by age, sex and tumour subtype to the socioeconomic inequalities of all cancer risk, a descriptive epidemiological study of cancer incidence in Scotland (2000-07) was undertaken. Age standardised rates per 100,000 population were calculated by direct standardisation to the European standard. A linear regression model was used to calculate the Slope Index of Inequality (SII) and Relative Index of Inequality (RII) which were employed to rank tumour and subtype contribution to all cancer risk socioeconomic inequalities by age for each sex for lung and upper aero-digestive tract (UADT) cancers separately. There were 216,305 cases excluding nonmelanoma skin cancer (all cancer) comprising 37,274 lung, 8,216 head and neck and 6,534 oesophageal cancers classified into anatomical or morphological subtypes. Socioeconomic circumstances were measured using the Scottish Index of Multiple Deprivation (SIMD). Analyses were partitioned by five-year age group and sex. RII was adapted to rank the contribution of each tumour type to all cancer socioeconomic inequalities and to examine subtype by age and sex simultaneously. The rank was defined as the proportion of all cancer socioeconomic inequality. All cancer socioeconomic inequality was greater for males than females (RII=0.366; female RII=0.279). The combination of lung and UADT socioeconomic inequalities contributed 91% and 81% respectively to all cancer socioeconomic inequality. For both sexes lung and UADT subtypes showed significant socioeconomic inequalities (P < 0.001) except oesophageal adenocarcinoma in males (P=0.193); for females, socioeconomic inequality was borderline significant (P=0.048). Although RII rank differed by sex, all lung and larynx subtypes contributed the most to all cancer socioeconomic inequality with RII rank for oral cavity, oesophagus–squamous cell and oropharynx following. For males 40-44 years old, socioeconomic inequalities increased abruptly peaking at 55-59 years. For females, socioeconomic inequalities gradually peaked 10 years later. In both sexes, the socioeconomic inequalities peak age preceded age of peak incidence. This study showed that socioeconomic inequalities in lung and UADT cancers vary greatly by age, tumour subtype and sex; these variations were likely to largely reflect differences between the sexes in risk behaviours which vary by birth cohort and are socioeconomically patterned. Longitudinal data enabled exploration of the temporal relationship between socioeconomic status and cancer incidence. An investigation of several individual and a single area-based measure of socioeconomic circumstances was undertaken in the second study of this thesis. The effect of country of birth, marital status, one area socioeconomic circumstances measure (Carstairs) and five individual socioeconomic variables (economic activity, education, occupational social class, car ownership, household tenure) on the risk associated with lung, UADT and all cancer combined (excluding non-melanoma skin cancer) were explored. A linked dataset using the Scottish Longitudinal Study and Scottish Cancer Registry was created to follow 203,658 cohort members aged 15+ years from 1991-2006. Relative risks (RR) were calculated using Poisson regression models by sex offset for person-years of follow-up. There were 21,832 first primary tumours (including 3,505 lung and 1,206 UADT cancers). Regardless of cancer, economic inactivity (versus activity) was associated with increased risk (male: RR 1.14 95% CI 1.10, 1.18; female: RR 1.06 95% CI 1.02, 1.11). For lung cancer, area deprivation remained significant after full adjustment suggesting that the area deprivation cannot be fully explained by individual variables. Not having a qualification (versus degree) was associated with increased lung cancer risk; likewise for UADT cancer risk (females only). Occupational social class associations were most pronounced and elevated for UADT risk. No car access (versus ownership) was associated with increased risk (excluding all cancer risk for males). Renting accommodation (versus home ownership) was associated with increased lung cancer risk, UADT cancer risk for males only and all cancer risk for females only. Regardless of cancer group, elevated risk was associated with no education and living in deprived areas. This study demonstrated that different and independent socioeconomic variables were inversely associated (greater incidence with lower socioeconomic circumstances) with different cancer risks in both sexes; no one socioeconomic variable had a dominant risk association or captured all aspects of socioeconomic circumstances or the full life-course. The association of multiple socioeconomic variables was likely to reflect the complexity and multifaceted nature of low socioeconomic circumstances as well as the various roles of these dimensions over the life-course. A final study investigated the role of behaviours (smoking, alcohol, diet and exercise) on the association of low socioeconomic circumstances with all cancer risk and lung and upper aero-digestive tract cancers combined (LUADT). The Scottish Cancer Registry and Scottish Health Survey data were linked to create a population study (1995-2011). There were 42,983 adults over 16 years old who were followed for 3,750,611 person-years. There were 2,130 first primary cancers diagnosed including 453 LUADT cancers. Poisson regression models, minimally adjusted by age and sex, were developed to estimate the risk association between five individual socioeconomic variables (economic activity, highest qualification, occupational social class, car ownership and housing tenure), one area-based socioeconomic indicator (SIMD) and all cancer and LUADT cancer. A further socioeconomic indicator was developed to reflect multiple low socioeconomic circumstances. This was defined as the count, at the individual participant level, of socioeconomic variables in the highest risk category. A similar multiple high risk behaviour derived variable, defined as the count of highest risk category for the following variables: current smoking status, units of alcohol consumed in a week, daily fruit and vegetable consumption and exercise sessions per week, was also calculated at the individual participant level. The minimally adjusted Poisson models were successively adjusted for behaviours (smoking, alcohol, diet and exercise) to establish any remaining contribution to cancer risk not explained by behaviour. Multiple low socioeconomic circumstances were very strongly associated with increased risk for both cancer groups. For all cancer risk, the elevated risk was nearly fully attenuated for all categories of multiple low socioeconomic circumstances when adjusted for smoking only. For LUADT cancer and in the minimally adjusted model, the risk increased in a dose-response manner.
The risk associated with LUADT cancer for study participants in the highest category of multiple low socioeconomic circumstances was more than three-times greater when compared to their affluent counterparts (RR 3.35 95% CI 2.26, 4.97); this elevated risk remained at 86% compared to those with no socioeconomic disadvantage, even after full adjustment for smoking, alcohol, diet and exercise behaviours. When looking at single socioeconomic status (SES) indicators, only those who rented accommodation from a local authority remained at a 50% increased risk of LUADT cancer even after adjustment for all the behaviours (RR 1.50 95% CI 1.05, 2.16). This study demonstrated that smoking is a major inequality issue and a significant cancer risk which is socially patterned. Further analytical research is required to fully understand the pathways and mechanisms between socioeconomic circumstance and lung and upper aerodigestive cancer risk.
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Gupta, Bhawna. "Life-course approach to behavioural risk factors and quality of life for cancers of the upper aero-digestive tract in an Indian population: a case-control study." Thesis, Griffith University, 2018. http://hdl.handle.net/10072/371971.

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The incidence and mortality rates of cancers of upper aero-digestive tract (UADT) remain significantly high in India. These cancers, which occur more commonly in later adulthood, are influenced by social and lifestyle behaviours carried out during childhood, adolescence and early adult life. Thus, life-course epidemiology attempts to assess varying health effects of various risk factors, according to timing, duration and frequency of exposure, which may give important clues to the causes of cancer. These cancers have serious impacts on quality of life (QoL) as they involve anatomical structures essential for mastication, speaking, cosmetic appearance and psychological wellbeing. Moreover, these cancers frequently present late and progress rapidly. Therefore, there is a need to develop a model for cost-effective screening and detection of individuals at high-risk of these cancers in the near future as well as early detection of cancer cases. Thereby, a study was designed with the following hypotheses:  Early age at initiation, frequency and duration of use of tobacco in its all forms, exposure to second hand tobacco smoke (SHS) at home and drinking alcohol has a linear dose-response relationship with the incidence of UADT cancers.  There is an association between tobacco, alcohol drinking, diet, oral hygiene and anthropometry measures with incidence of oral cancers.  Cancer site, staging, gender and age at diagnosis have an impact on QoL.  A risk-factor based screening model for UADT cancers has strong predictive ability to detect high-risk individuals. A bi-centre hospital-based frequency matched case-control study was conducted in Pune, Maharashtra State, India, from June 2014 and May 2015. Cases were histopathologically confirmed new cases of squamous cell carcinoma of UADT. Controls were patients diagnosed with a disease other than UADT or any other cancer, selected from the same hospital during the same period as the cases were recruited. Data were collected by medical-record abstraction, face-face interviews and by visual inspection of the oral cavity. A closed-ended questionnaire with a life-course perspective was used to collect patient‟s self-reported information on sociodemographics, lifestyle habits (chewing and smoking tobacco, drinking alcohol, SHS, oral hygiene) and QoL. The interviewer recorded anthropometry measures and number of missing teeth. Unconditional logistic regression was used to estimate the odds ratios (ORs) and their corresponding 95% confidence intervals (CIs). A reciever-operater characterstic curve was plotted against sensitivity and false-positive rate to produce a cut off point for the presence of UADT cancers. Examination of all QoL domains was done using oneway analysis of variance and the Bonferroni adjustments for post-hoc comparisons. Data were analysed by the Statistical Package for Social Sciences version 22. A total of 480 patients participated in the study. Chewing tobacco emerged as the strongest predictor for UADT cancers (OR=7.61; 95% CI 4.65-12.45) followed by smoking and drinking alcohol. Exposure to SHS during childhood (<16 years) rather than after ≥16 years increased the risk significantly (OR=4.05; 95% CI 2.06-7.95). There is a linear dose-response relationship between duration, frequency and early age at initiation for lifestyle risk factors (chewing and smoking tobacco; drinking alcohol) with incidence of UADT cancers in adulthood. Combined effects of tobacco and alcohol consumption habits elevated the risk (OR=12.05; 95% CI 4.61-31.49) in comparison to never users of these habits. Furthermore, the combination of these three lifestyle risk factors accounted for 86.82% of population attributable risk. Overall, the most affected QoL domains were anxiety and mood both among cases and controls. Oropharynx and hypopharynx cancer cases had the worst mean scores across all QoL domains. Stage IV cancer patients showed the worst QoL. Amongst UADT cancers in Pune, cancer of the oral cavity was the most common. Chewing tobacco showed higher odds (OR=8.51; 95% CI 4.90-14.77) for oral cancer risk as compared to UADT cancers. Poor oral hygiene emerged as significant predictor for oral cancer risk (OR=6.98; 95% CI 3.72-13.05). A screening model was derived for detection of individuals at high-risk for UADT cancers. This model has high sensitivity (93.5%), specificity (71.1%), false positive rate (28.8%), false negative rate (6.4%), positive predictive value (74.8%) and negative predictive value (96.6%). Our research recognises the framework of life-course influences of early exposure to behavioural risk-factors as independent and combined predictors of UADT cancers. The significantly compromised QoL in UADT cancer needs to be incorporated as an outcome measure in an individualized approach to therapeutic and palliative care planning of these cases to enable a better quality of survival. If validated in other studies, our proposed screening model can be applicable to many other high-risk UADT cancer populations with behavioural risk factors similar to our study population.
Thesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Dentistry and Oral Health and Menzies Health Institute Queensland
Griffith Health
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Väkeväinen, Satu. "Local acetaldehyde production as a pathogenetic factor for upper digestive tract cancers in humans." Helsinki : University of Helsinki, 2002. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/vakevainen/.

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Cui, Yan. "Polymorphism of xeroderma pigmentosum group G and dietary flavonoid intake on the risk of lung and upper aero-digestive tract cancers." Diss., Restricted to subscribing institutions, 2005. http://proquest.umi.com/pqdweb?did=954047411&sid=1&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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Chen, Jun 1969. "The association between dietary intake and the risk of cancers of the upper aero-digestive tract : a case-control study in Brazil." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=29422.

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Cancers of the upper aero-digestive tract (UADT) rank as the fifth most common neoplastic disease worldwide. Two identified risk contributors are consumption of tobacco and alcohol. Among all other potential etiological factors, diet has long been recognized to play an important role in the development of cancers of the UADT. Data from a multi-centre, hospital-based case-control study conducted in Brazil were used to assess the association of dietary intake with the risk of cancers of the UADT. Dietary assessment was made in terms of estimated intake of nutrients, specific foods and food groups. After adjusting for the effects of alcohol and tobacco consumption as well as empirical confounders, protective effects against cancer of the mouth (Odds Ratio (OR) = 0.61, 95% confidence interval (95% CI): 0.4--1.0) and the pharynx (OR = 0.51, 95%CI: 0.3--0.9) were found for consumption of citric fruits. (Abstract shortened by UMI.)
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Shaikh, Mushfiq Hassan. "The Role of Human Papillomaviruses in the Aetiopathogenesis of Head and Neck Cancer in South Asia, and Approaches to Treatment." Thesis, Griffith University, 2017. http://hdl.handle.net/10072/366967.

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Malignancies of the upper aero-digestive tract are a major public health problem, especially in South Asia. The major risk factors in South Asia remain smoked/smokeless tobacco, areca nut, alcohol abuse and poor diet, with limited evidence for human papillomavirus (HPV). Although HPV-associated head and neck squamous cell carcinoma (HNSCC) is well documented in the western world, studies on South Asian populations are few and inconsistent. However, the incidence of HPV-associated head and neck cancer (HNC) has increased in recent years. Certain high-risk types of HPV infection are regarded as well-established risk factors for cervical cancer and a subset of HNSCC; however, their true role and importance in the progression of HNSCC remain unclear. Although HPV-associated HNC patients generally have a better prognosis than those with HPV-negative disease, current chemo- and radio-therapies are largely non-specific and have considerable toxicities. RNA interference (RNAi), which has shown great promise as a highly specific therapy for other diseases, has potential for treating HPV-associated HNC, especially if disease progression is dependent on the continual expression of HPV oncogenes.
Thesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Dentistry and Oral Health
Griffith Health
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DALMARTELLO, MICHELA. "A LATENT VARIABLE APPROACH TO DIETARY PATTERNS RESEARCH." Doctoral thesis, Università degli Studi di Milano, 2019. http://hdl.handle.net/2434/612183.

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INTRODUCTION The dietary pattern approach is useful to study the effect of the overall diet on health outcomes, through considering the network of complex interactions between foods or nutrients. The main methods traditionally used to identify dietary patterns are principal components analysis, factor analysis, principal components factor analysis and cluster analysis. Latent class analysis (LCA) is a latent variable approach, that has some advantages in comparison to the previous methods. Unlike principal component, factor and principal component factor analysis, it can be used to classify individuals into mutually exclusive groups conceived as dietary patterns and differently from cluster analysis, which has the same aim of grouping subjects, it permits quantification of the uncertainty of class membership, and assessment of goodness of fit. Moreover, it allows for adjustment for covariates directly in the pattern identification. OBJECTIVES As latent class analysis has rarely been applied in dietary pattern studies, the aim of this research is to apply the recent developments of the techniques to this area of research. We targeted to address the issue of dietary pattern identification in the case-control setting using latent class analysis and latent class trees. We provided estimation of pattern sizes and their characterization, taking into account correlations between dietary variables (local dependencies), and covariate adjustment. We also evaluated the robustness of the identified dietary patterns to total non-alcoholic energy intake adjustment, for different types of correction. Finally, we illustrated the method’s properties in the assessment of the relation between the identified dietary patterns and selected health outcomes, given the all the above. DIETARY PATTERNS AND THE RISK OF ORAL AND PHARYNGEAL CANCER We analyzed data from a multicentric case-control study on oral and pharyngeal cancer (OPC) carried out between 1992 and 2009, including 946 cases and 2492 hospital controls. Information on diet was collected through a food frequency questionnaire (FFQ). Using LCA, we identified 4 dietary patterns, conceived as mutually exclusive groups of people who shared a common dietary behaviour within groups. The first pattern, labelled ‘Prudent pattern’, showed higher probability of consuming more leafy and fruiting vegetables, citrus fruit and all other kinds of fruits, tea while showing lower probability of consuming red meat. The second pattern, that we named ‘Western pattern’, reported higher consumption of red meat and lower consumption of fruits, cruciferous and fruiting vegetables. We termed the third pattern ‘Lower consumers-combination pattern’ as people in it were less likely to eat fruits, leafy and fruiting vegetables, pulses, potatoes, fish, white and red meat, bread and tea/decaffeinated coffee. The last pattern had higher probability to eating fruiting, leafy and other vegetables, white and red meat and bread, while showed a lower probability to consume coffee, tea, processed meat, cheese, fish, sugary drinks and desserts. We called this last pattern ‘Higher consumers-combination pattern’. Dietary patterns were adjusted for total non-alcoholic energy intake and correlation between certain foods item (sugar-coffee, soups-pulses) was allowed during classes identification. Compared to the Prudent pattern, the Western and the Lower consumers-combination ones were positively related to the risk of OPC (OR=2.56, 95% CI: 1.90 – 3.45 and OR=2.23, 95% CI: 1.64 – 3.02). Higher consumers-combination pattern didn’t differ significantly from the Prudent pattern (OR=1.28, 95% CI: 0.92 – 1.77). ENERGY INTAKE ADJUSTMENT IN DIETARY PATTERN RESEARCH USING LATENT CLASS ANALYSIS Using data from the multicentric case-control study on OPC (Italy, 1992-2009), we identified and compared dietary patterns adjusting or not for total non-alcoholic energy intake in the classes identification phase of the analysis. Three possible ways to correct for total energy intake in class identification were presented, corresponding to different hypothesis on the effect of this variable. In general unadjusted and adjusted solutions were comparable. The main difference was related to the patterns that showed highest/lowest non-alcoholic energy intake, that resulted in a variation of number of classes (4/5/7 patterns for the different adjusted solutions and 5 patterns for the unadjusted one). Then, to determine the effect of adjustment in predicting an health outcome, we compared the effect of unadjusted dietary patterns, unadjusted dietary patterns with non-alcoholic energy intake variable also included in the model as a confounder, and adjusted dietary patterns on the risk of OPC . Differences in the estimations for the distinct solutions were found when Odds Ratios (ORs) were not corrected for known/potential risk factors. In general, adjustments for non-alcoholic energy intake results in a mitigation of the effects, thus remaining in the same order. When adjusting for known/potential risk factors, estimations of ORs and related confidence intervals (CIs) remained consistent in all the models we fitted. In the end, specific suggestions on how to perform energy correction in dietary patterns research using LCA were delivered, basing on the results of the current analysis. DIETARY PATTERNS INSPECTION THROUGH LATENT CLASS TREE We analyzed data from two Italian case–control studies, the first included 946 cases with OPC and 2492 hospital controls, and the second included 304 cases with squamous cell carcinoma of the esophagus (ESCC) and 743 hospital controls. In our application of latent class analysis on the combined dataset of the two case-control studies (Italy, 1992-2009), we found the best fit for a solution that was difficult to interpret and included minor differences between clusters. To address these issues, the Latent Class Tree method was proposed. Three fit statistics (AIC, AIC3, BIC) were used for their different level of penalty that resulted in different lengths of the tree and consequently, different granularity in the analysis. For the first split we allowed for a 4-class solution which identified a pattern characterized by high intake of leafy and fruiting vegetable and fruits (‘Prudent pattern’), a pattern with a high intake of red meat and low intake of certain fruits and vegetables (‘Western pattern’) and two patterns which showed a combination-type of diet. The first ‘combination’ pattern showed a low intake of the majority of foods (‘Lower consumers-combination pattern’), and the other one high intake of various foods (‘Higher consumers-combination pattern’). Compared to the Prudent pattern, the Western one was positively related to OPC (OR=1.91, 95% CI: 1.41-2.58) and to ESCC (OR=3.22, 95% CI: 1.78 – 5.82). The Lower consumers-combination pattern was positively associated to OPC (OR=2.14, 95% CI: 1.58-2.91) and to ESCC (OR=2.85, 95% CI: 1.47-5.55). No significant association was found between the Higher consumers-combination pattern and OPC (1.04, 95% CI: 0.74-1.46) and ESCC (OR=0.89, 95% CI: 0.39-1.99). In the ‘Prudent pattern’ branch of the tree, at the third level, we found two classes that differed in the risk of both cancer types. These two classes differed mainly for the intake of citrus fruit, showing respectively, OR=1.85, 95% CI:1.07-3.19 for OPC and OR=5.37, 95% CI: 1.48-19.44 for ESCC for the class that reported low intake of citrus fruit with respect to the class which exhibit a high intake of citrus fruit. No other significant differences were found between the other pairs of classes at any other level of the tree. CONCLUSION We presented latent class methods as powerful tools to determine dietary patterns conceived as mutually exclusive homogeneous groups of subjects which shared common dietary habits. These methods exhibit some advantages, with respect to classical approaches, that can address important issues in dietary pattern research. For example, it is possible to obtain estimation for pattern prevalence in the population, and to perform energy intake adjustment in the pattern identification phase of the analysis. Moreover, class formation inspection, comparison between different solutions and the analysis of subgroups that may be relevant for the research at hand are features offered by the newly developed latent class tree approach.
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Patronieri, Alexandre Tadeu. "Tendências de incidência do câncer das vias aéreas e digestivas superiores segundo 18 registros de câncer de base populacional com destaque ao município de São Paulo. 1969-1999." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/6/6132/tde-07032007-095710/.

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Introdução: O câncer das Vias Aéreas e Digestivas Superiores compreendem um grupo de tumores que se distribuem por diversas regiões anatômicas. Para a maioria das localizações os principais fatores de risco são o tabaco e o álcool. Como os fatores de risco são conhecidos espera-se que as diversas campanhas de prevenção e detecção precoce estivessem propiciando à diminuição da incidência. Objetivos Estimar em algumas localidades do mundo as tendências da incidência dos tumores de lábio, língua, boca, glândulas salivares, orofaringe, nasofaringe e hipofaringe de 1969 a 1999. Métodos Analisou-se os coeficientes de incidência de 18 Registros de Câncer de Base Populacional que possuíam toda a série histórica publicada no “Cancer Incidence in Five Continents”.utilizando-se modelos de regressão polinomial.Resultados Houve tendência de aumento, estatisticamente significativo, para o câncer de língua e boca em São Paulo em mulheres e em homens de diversos países europeus e localidades dos EUA. Em contrapartida houve queda em Porto Rico, Mumbai e Cingapura. O câncer de lábio decresceu na maioria das localidades em homens. Entretanto em países do norte da Europa e em Iowa houve aumento em mulheres. O câncer de orofaringe aumentou em homens na Europa e em Iowa e decresceu em Cali, Quebec, e localidades da Ásia. O câncer de hipofaringe apresentou tendência crescente em várias localidades da Europa em homens e queda na Suécia, Inglaterra, Cingapura e Porto Rico. Conclusão os tumores de língua, boca e orofaringe continuam apresentando tendências de crescimento para diversas localidades. Por isso há a necessidade de campanhas propiciando a prevenção e a detecção precoce.
Introduction: Upper Aerodigestive Tract Cancer comprises a group of neoplasms which spread over many anatomic regions. Considering most of the locations, the worst risk coefficients are tobacco and alcohol. Taken the coefficients are known, prevention and precocious detection campaigns are expected to lessen the incidence statistics. Objectives. To estimate the trends on lip, oral cavity (tongue and mouth), salivary gland, oropharyngeal, nasopharyngeal and hypopharyngeal cancer incidences in some locations worldwide between 1969 - 1999. Methods Incidence coefficient data of eighteen records from the Population Database Cancer Registry which had its history published in “Cancer Incidence in Five Continents” has been analysed applying polynomial regression models. Results There has been a statistically significant increase to the number of oral cavity cancer in Sao Paulo among women; in European countries as well as in some North American cities, the increase was detected among both men and women. On the other hand, there has been a decrease in Puerto Rico, Mumbai and Singapore. The incidence of lip cancer decreased in most locations (among men). However, an increase has been detected in North Europe and in Iowa, USA (among women). Oropharyngeal cancer has increased in number among men in Europe and Iowa, USA; it decreased in number in Cali, Quebec, and other areas in Ásia. Hypopharyngeal cancer has shown to be an increasing trend in many Europeans countries among men, but a decreasing one in Sweden, England, Singapore and Puerto Rico. Conclusion Oral cavity and oropharyngeal neoplasms trends proceed grow in many locations. Finally, the need for prevention and precocious detection campaigns is urgent.
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Paget-Bailly, Sophie. "Facteurs de risque professionnels des cancers des voies aéro-digestives supérieures : Synthèse des données épidémiologiques et analyse d’une étude cas-témoins, l’étude Icare." Thesis, Paris 11, 2012. http://www.theses.fr/2012PA11T061/document.

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Contexte : L’amiante est maintenant une cause avérée de cancer du larynx, mais le rôle des expositions professionnelles dans la survenue des cancers des voies aéro-digestives supérieures (VADS) reste largement méconnu. Bien que plusieurs études aient rapporté des associations entre les expositions professionnelles et ces cancers, il est difficile de synthétiser les résultats et d’en tirer des conclusions définitives. Objectifs : (1) Le premier objectif est de synthétiser les données épidémiologiques disponibles sur les associations entre les cancers de la cavité buccale, du pharynx et du larynx (les cancers des VADS les plus fréquents) et les expositions professionnelles ; (2) le second objectif est, à partir des données d’une large étude cas-témoins, l'étude Icare, d'identifier les professions ou industries présentant des risques élevés de cancer des VADS, puis d'étudier le rôle de certaines expositions professionnelles suspectées (amiante, laines minérales (LM), poussières de ciment, silice). Matériel et méthodes : (1) Une recherche bibliographique et des séries de méta-analyses ont été réalisées pour certaines expositions professionnelles suspectées. (2) L'étude Icare est une étude cas-témoins en population générale française incluant 2415 cas de cancer des VADS et 3555 témoins. L'historique professionnel complet des sujets a été recueilli, avec une description détaillée de chaque emploi exercé. L'évaluation des expositions à l'amiante, aux LM, aux poussières de ciment et à la silice sur l’ensemble de la vie professionnelle a été réalisée à l'aide des matrices emplois-expositions développées dans le cadre du programme Matgéné (Institut de Veille Sanitaire). Résultats : Des méta-risques relatifs (méta-RR) significativement augmentés ont été observés pour le cancer du larynx et les expositions aux hydrocarbures aromatiques polycycliques (HAP), aux gaz d'échappement de moteurs, au travail dans l'industrie textile et dans l'industrie du caoutchouc, et pour le cancer de la cavité buccale et du pharynx et les expositions à l'amiante, aux HAP et aux gaz d'échappement de moteurs. Des augmentations de risque significatives ont été observées pour plusieurs professions et secteurs d'activité exposant notamment aux nuisances présentant des méta-RR augmentés. Les résultats des analyses par nuisance confirment l'association entre l'amiante et le cancer du larynx et suggèrent également une association avec les cancers de la cavité buccale et du pharynx. Une association avec les poussières de ciment est également suggérée. Les résultats ne sont pas en faveur d’un rôle de l’exposition aux LM et à la silice.Conclusion : Ce travail renforce l’hypothèse d’un rôle des expositions professionnelles dans la survenue des cancers des VADS. Dans l’ensemble, nos résultats suggèrent des associations entre le risque de cancer des VADS et les expositions à l'amiante, aux HAP, aux poussières de ciment, et le travail dans l'industrie du caoutchouc
Background: There is sufficient evidence that asbestos causes laryngeal cancer, but overall the role of occupational exposures in the etiology of head and neck cancer (HNC) remains largely unknown. Although several studies have reported associations between occupational exposures and HNC, it is difficult to draw firm conclusions. Objectives: (1) To summarize available epidemiologic data on occupational exposures and cancers of the oral cavity (OC), pharynx and larynx (the most frequent HNC); (2) using data from a large case-control study, to identify occupations and industries with an increased risk of HNC, then to investigate the role of some suspected occupational exposures (asbestos, mineral wools (MW), cement dust, silica). Methods: (1) A literature research and a series of meta-analyses were performed. (2) The Icare study is a French population-based case-control study including 2415 HNC cases and 3555 controls. Complete and detailed occupational histories were collected. Analyses by job title were conducted. Job exposure matrices, developed by the Occupational Health Department of the French Institute for Public Health Surveillance (InVS), were used to assess lifetime occupational exposure to asbestos, MW, cement dust and silica. Results: Significantly increased meta-relative risks (meta-RR) were obtained considering laryngeal cancer and exposures to polycyclic aromatic hydrocarbons (PAH), engine exhausts, working in the textile industry and the rubber industry, and for cancer of the OC and pharynx and exposures to asbestos, PAH and engine exhausts. Significantly increased risks were found for several jobs and industries, some of them entailing exposures to agents for which meta-RR were increased. Analyses for specific occupational exposures confirmed the association between asbestos and laryngeal cancer and showed an association with the risk of oral cavity and pharyngeal cancer. An association with exposure to cement dust was also suggested. The results did not support an association between HNC risk and exposure to MW or silica. Conclusion: This work emphasizes the role of occupational exposures in HNC. Overall, our results suggest associations between HNC and exposure to asbestos, PAH, cement dust, and work in the rubber industry
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Ventayol-García, Tania. "Clonal expansion in the human upper gastrointestinal tract." Thesis, Queen Mary, University of London, 2013. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8694.

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The high incidence of gastrointestinal cancers in the general population and the presence of premalignant dysplastic precursor lesions in the gastrointestinal tract make the gastrointestinal tract an ideal environment to study cancer clonality and clonal expansion. Background: Intestinal metaplastic (IM) glands in the human stomach are clonal, contain multiple stem cells and spread by fission. This mechanism of gland fission causes field cancerisation. We hypothesised that gastric adenocarcinoma (GA) progresses through a series of genetic events arising from a founder mutation. A process analogous to niche succession may also take place in the normal oesophagus. We hypothesise that oesophageal squamous cell cancer occurs by a process of field cancerisation of the oesophagus. RHBDF2 has been identified as the gene responsible for tylosis with oesophageal carcinoma (TOC). We hypothesise that RHBDF2 germline gain of function mutations might be lost during tumour progression in TOC and this might affect iRhom2 localisation in the cell. Methods and results: A cohort of 23 patients with dysplasia and a cohort of 51 GA patients were screened for genes accounting for 75% of all somatic mutations previously reported in GA. Only 13% of dysplastic patients and 31.4% of GA patients had mutations. Three dysplastic patients and six GA patients were analysed by microdissection. Small gastric cancer foci in a cohort of hereditary diffuse gastric cancer (HDGC) patients (n=5) were also screened by laser-capture microdissection (LCM) for mutations in TP53. A cohort of 30 patients was screened for common mutations in OSCC and for RHBDF2 mutations. 36.36% of the patients presented mutations. Three patients with mutations were randomly selected and areas of oesophageal squamous cell dysplasia and OSCC were analysed by LCM. Three TOC patients were also analysed by LCM and immunohistochemistry was performed for iRhom2 and ADAM17. Conclusions: The usual mutational events established for GA development during the metaplasiadysplasia- carcinoma sequence (MCS) do not fit the results from either of our two LCM mutation studies in the human stomach. Dysplasia was shown to be clonal and GA demonstrates genetic heterogeneity through clonal evolution. Field cancerisation could not be detected in HDGC using TP53 as a clonal marker. The low incidence of OSCC patients with mutations implies that other genes may be involved in the premalignant pathway leading to OSCC. Oesophageal squamous cell dysplasia and OSCC demonstrate clonal expansion through tumour progression. RHBDF2 mutations do not occur in sporadic OSCC but germline RHBDF2 mutations can be lost during tumour progression in TOC patients with LOH in 17q. Overall, the somatic mutation theory of carcinogenesis seems to hold true for both the progression to GA and OSCC, as both carcinomas seem to evolve from a single mutated stem cell and acquire genetic heterogeneity as the tumours evolve.
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Books on the topic "Upper digestive tract cancer"

1

M, Daly John, Hennessy T. P. J, and Reynolds John V, eds. Management of upper gastrointestinal cancer. London: W.B. Saunders, 1999.

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Kirk, R. M. Complications of surgery of the upper gastrointestinal tract. London: Baillière Tindall, 1986.

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Meyers, Morton A. Neoplasms of the digestive tract: Imaging, staging, and management. Edited by Meyers Morton A. Philadelphia: Lippincott-Raven, 1998.

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Fong, Yuman, Pierre A. Clavien, and M. G. Sarr. Atlas of Upper Abdominal Surgery. Berlin: Springer-Verlag GmbH & Co. KG, 2004.

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Jansen, Marnix, and Nicholas A. Wright, eds. Stem Cells, Pre-neoplasia, and Early Cancer of the Upper Gastrointestinal Tract. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-41388-4.

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Price, Tim. A diagnostic atlas of tumors of the upper aero-digestive tract: A transnasal video endoscopic approach. New York, NY: Informa Healthcare, 2012.

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Morson, Basil C. Precancerous lesions of the gastrointestinal tract: A histological classification. London: Baillière Tindall, 1985.

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The effective management of upper gastrointestinal malignancies. London: Aesculapius Medical Press, 2005.

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Rikiya, Fujita, ed. Early cancer of the gastrointestinal tract: Endoscopy, pathology, and treatment. Tokyo: Springer, 2006.

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R, Galle P., ed. Disease progression and carcinogenesis in the gastrointestinal tract: Proceedings of Falk Symposium 132 (part III of the Gastroenterology Week), held in Freiburg, Germany, October 9-10, 2002. Dordrecht: Kluwer Academic, 2003.

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Book chapters on the topic "Upper digestive tract cancer"

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Skinner, Heath D., Qiongrong Chen, Elena Elimova, Roopma Wadhwa, Shumei Song, and Jaffer A. Ajani. "Predictive Biomarkers for Therapy in Adenocarcinoma of the Upper Digestive Tract." In Biomarkers in Cancer Screening and Early Detection, 118–29. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118468869.ch10.

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Hiraishi, Mamoru, Toshiro Konishi, Ken-Ichi Mafune, Takeshi Miyama, Tooru Hirata, Kiyoshi Mori, Haruhiro Nishina, and Yasuo Idezuki. "Endoscopic Treatment for the Cancer and Submucosal Tumor of the Upper Gastrointestinal Tract." In Recent Advances in Management of Digestive Cancers, 464–66. Tokyo: Springer Japan, 1993. http://dx.doi.org/10.1007/978-4-431-68252-3_130.

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Snijders, P. J. F., A. J. C. van den Brule, C. J. L. M. Meijer, and J. M. M. Walboomers. "Papillomaviruses and Cancer of the Upper Digestive and Respiratory Tracts." In Current Topics in Microbiology and Immunology, 177–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78487-3_10.

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Altorki, N. K., K. Subbaramaiah, and A. J. Dannenberg. "Cyclooxygenase-2: A Target for the Prevention and Treatment of Cancers of the Upper Digestive Tract." In COX-2, 107–23. Basel: KARGER, 2003. http://dx.doi.org/10.1159/000071369.

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Weisburger, J. H. "Nutrition and Cancer Prevention: Gastrointestinal Cancer." In Digestive Tract Tumors, 265–74. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5149-8_36.

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Loh, John P. "Upper Digestive Tract Radiographic Imaging." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 2952–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_508.

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Bardi, Georgia, and Sverre Heim. "Tumors of the digestive tract." In Cancer Cytogenetics, 373–400. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118795569.ch14.

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Bardi, Georgia, and Sverre Helm. "Tumors of the Digestive Tract." In Cancer Cytogenetics, 429–61. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2010. http://dx.doi.org/10.1002/9781118010136.ch13.

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Oguro, Yanao, and Takao Sakita. "The Endoscopic Diagnosis of Early Gastric Cancer." In Digestive Tract Tumors, 111–18. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5149-8_13.

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Drasar, B. S., and P. G. S. Cook. "Intestinal Bacteria and the Initiation of Cancer." In Digestive Tract Tumors, 221–28. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5149-8_30.

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Conference papers on the topic "Upper digestive tract cancer"

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Gualberto, Antonio, Tenghui Chen, Catherine Scholz, and Jason Luke. "Abstract P110: Neandertal introgressions contribute to upper aero-digestive tract tumor patient survival and identify patients who may benefit from STING agonist treatment." In Abstracts: AACR-NCI-EORTC Virtual International Conference on Molecular Targets and Cancer Therapeutics; October 7-10, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1535-7163.targ-21-p110.

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Huang, Shiang-Fu, Huei-Tzu Chien, Sou-De Cheng, Chun-Ta Liao, and Hung-Ming Wang. "Abstract 3255: The roles of ALDH2, ADH1B and ADH1C gene polymorphism in predicting upper digestive tract multiple primary malignancies in head and neck cancer patients." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-3255.

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Ouazzani, S., A. Lemmers, J. M. Gonzalez, J. Closset, I. El Moussaoui, J. Deviere, and M. Barthet. "DIGESTIVE NEO-EPITHELIZATION AFTER ENDOSCOPIC STENTING FOR COMPLETE UPPER DIGESTIVE TRACT DISUNION." In ESGE Days 2022. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1745107.

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Brunet Mas, E., E. Martínez Bauer, P. Garcia Iglesias, L. Hernández, J. Da Costa, G. Llibre, A. Soria, et al. "MUCOSAL ENDOSCOPIC RESECTION OF LARGE LESIONS OF THE UPPER DIGESTIVE TRACT." In ESGE Days 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1681939.

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"Clinical Study Based on Cancer-Related Anemia in Digestive Tract Tumors." In 2020 International Conference on Social Sciences and Social Phenomena. Scholar Publishing Group, 2020. http://dx.doi.org/10.38007/proceedings.0001065.

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Mathialagan, Prabhakaran, and Malathy Chidambaranathan. "Morphological and Characteristic Analysis of Upper Aero-Digestive Tract Tumour: Revealing Uncovered Facts in Digital Pathology*." In 2021 International Conference on Computing, Communication, and Intelligent Systems (ICCCIS). IEEE, 2021. http://dx.doi.org/10.1109/icccis51004.2021.9397133.

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Mu, Hai-Bin, Man-Ling Zhang, Xiao-Qiang Zhang, Feng-Qiu Zhang, De-Jia Kong, and Li-Bin Tang. "Experience of curing serious obstruction of advanced-stage upper digestive tract tumor using laser under endoscope." In 1997 Shanghai International Conference on Laser Medicine and Surgery, edited by Jing Zhu. SPIE, 1998. http://dx.doi.org/10.1117/12.330141.

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Dinali, Mohamed El, Johannes Braegelmann, Thomas Stricker, Christopher Brown, Ravi Salgia, Everett Vokes, Kevin White, and Tanguy Seiwert. "Abstract A207: Whole transcriptome profiling in upper aerodigestive tract cancers." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 15-19, 2009; Boston, MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/1535-7163.targ-09-a207.

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Gu, Yubei, and Zhong Jie. "IDDF2019-ABS-0154 Upper gastrointestinal tract involvement in crohn’s disease in china: frequency, endoscopic features, related factors, and prognosis." In International Digestive Disease Forum (IDDF) 2019, Hong Kong, 8–9 June 2019. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2019. http://dx.doi.org/10.1136/gutjnl-2019-iddfabstracts.169.

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Ren, Xiaoxue, Yifan Wu, Mingle Huang, Yubin Xie, Ming Kuang, Xiaoxing Li, and Lixia Xu. "IDDF2022-ABS-0194 Patient-derived organoids predict chemotherapy response in biliary tract cancer." In Abstracts of the International Digestive Disease Forum (IDDF), Hong Kong, 2–4 September 2022. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2022. http://dx.doi.org/10.1136/gutjnl-2022-iddf.25.

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Reports on the topic "Upper digestive tract cancer"

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Mizrahi, Itzhak, and Bryan A. White. Exploring the role of the rumen microbiota in determining the feed efficiency of dairy cows. United States Department of Agriculture, October 2011. http://dx.doi.org/10.32747/2011.7594403.bard.

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Expanding world hunger calls for increasing available food resources. Ruminants have the remarkable ability to convert human-indigestible plant biomass into human-digestible food products, due to a complex microbiome residing in the rumen compartment of their upper digestive tract. One way to tackle the problem of diminishing food resources is to increase the animals' energetic efficiency, i.e., the efficiency with which they convert energy from feed, thereby increasing food availability while lowering the environmental burden, as these animals would produce more and eat less. We hypothesize that the cow's feed efficiency is dependent on the taxonomic composition, coding capacity and activity of its reticulorumenmicrobiota. To test this hypothesis, three aims are defined: (1) Evaluation of the feed efficiency of 146 dairy cows and defining two groups representing the highest and lowest 25% using the Israeli group's unique facility; (2) Comparing these two groups for microbiota diversity, identity and coding capacity using next-generation sequencing and metagenomic approaches; (3) Comparing the reticulorumenmicrobiota metabolic activity parameters. We measured feed efficiency in 146 milking cows and analyzed the taxonomic composition, gene content, microbial activity and metabolomic composition of rumen microbiomes from the 78 most extreme animals. Lower richness of microbiome gene content and taxa was tightly linked to higher feed efficiency. Microbiome genes and species accurately predicted the animals' feed-efficiency phenotype. Specific enrichment of microbes and metabolic pathways in each of these microbiome groups resulted in increasing valuable metabolites and decreasing unusable ones such as methane in efficient animals. This ecological and mechanistic understanding of the rumen microbiome could lead to an increase in available food resources and environmentally friendly livestock agriculture.
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Ureteroscopic management of upper tract urothelial cancer. BJUI Knowledge, April 2018. http://dx.doi.org/10.18591/bjuik.0455.

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