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Journal articles on the topic 'Unilateral brain damage'

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Published: 14 March 2023

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1

Winstein, C. "Motor learning after unilateral brain damage." Neuropsychologia 37, no. 8 (July 1, 1999): 975–87. http://dx.doi.org/10.1016/s0028-3932(98)00145-6.

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2

Schapiro, Anna C., James L. McClelland, Stephen R. Welbourne, Timothy T. Rogers, and Matthew A. Lambon Ralph. "Why Bilateral Damage Is Worse than Unilateral Damage to the Brain." Journal of Cognitive Neuroscience 25, no. 12 (December 2013): 2107–23. http://dx.doi.org/10.1162/jocn_a_00441.

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Human and animal lesion studies have shown that behavior can be catastrophically impaired after bilateral lesions but that unilateral damage often produces little or no effect, even controlling for lesion extent. This pattern is found across many different sensory, motor, and memory domains. Despite these findings, there has been no systematic, computational explanation. We found that the same striking difference between unilateral and bilateral damage emerged in a distributed, recurrent attractor neural network. The difference persists in simple feedforward networks, where it can be understood in explicit quantitative terms. In essence, damage both distorts and reduces the magnitude of relevant activity in each hemisphere. Unilateral damage reduces the relative magnitude of the contribution to performance of the damaged side, allowing the intact side to dominate performance. In contrast, balanced bilateral damage distorts representations on both sides, which contribute equally, resulting in degraded performance. The model's ability to account for relevant patient data suggests that mechanisms similar to those in the model may operate in the brain.
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3

Doricchi, Fabrizio, Gaspare Galati, Lara DeLuca, Daniele Nico, and Francesca D’Olimpio. "Horizontal space misrepresentation in unilateral brain damage." Neuropsychologia 40, no. 8 (January 2002): 1107–17. http://dx.doi.org/10.1016/s0028-3932(02)00010-6.

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4

Doricchi, Fabrizio, Alessandra Onida, and Paola Guariglia. "Horizontal space misrepresentation in unilateral brain damage." Neuropsychologia 40, no. 8 (January 2002): 1118–28. http://dx.doi.org/10.1016/s0028-3932(02)00011-8.

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5

Soffer, Dov, Eldad Melamed, Yaakov Assaf, and Shamay Cotev. "Hemispheric brain damage in unilateral status epilepticus." Annals of Neurology 20, no. 6 (December 1986): 737–40. http://dx.doi.org/10.1002/ana.410200616.

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6

Ludwig, Casimir J. H., Stephen H. Butler, Stephanie Rossit, Monika Harvey, and Iain D. Gilchrist. "Modelling contralesional movement slowing after unilateral brain damage." Neuroscience Letters 452, no. 1 (March 2009): 1–4. http://dx.doi.org/10.1016/j.neulet.2009.01.033.

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7

Kucharska-Pietura, K., B. Loza, and G. Kopacz. "Emotional control in schizophrenia and unilateral brain damage." European Neuropsychopharmacology 11 (January 2001): S284. http://dx.doi.org/10.1016/s0924-977x(01)80368-0.

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8

Noetzel, Michael J. "Theophylline Neurotoxicity Resulting in Significant Unilateral Brain-damage." Developmental Medicine & Child Neurology 27, no. 2 (November 12, 2008): 242–45. http://dx.doi.org/10.1111/j.1469-8749.1985.tb03775.x.

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9

Gandour, J., S. Ponglorpisit, F. Khunadorn, S. Dechongkit, P. Boongird, and R. Boonklam. "Stop voicing in thai after unilateral brain damage." Aphasiology 6, no. 6 (November 1992): 535–47. http://dx.doi.org/10.1080/02687039208249490.

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10

Gandour, Jack, Siripong Potisuk, Suvit Ponglorpisit, Sumalee Dechongkit, Fuangfa Khunadorn, and Prasert Boongird. "Tonal Coarticulation in Thai after Unilateral Brain Damage." Brain and Language 52, no. 3 (March 1996): 505–35. http://dx.doi.org/10.1006/brln.1996.0027.

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11

Zaidel, Dahlia W. "Neuronal connectivity, regional differentiation, and brain damage in humans." Behavioral and Brain Sciences 22, no. 5 (October 1999): 854–55. http://dx.doi.org/10.1017/s0140525x99502197.

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When circumscribed brain regions are damaged in humans, highly specific impairments in language, memory, problem solving, and cognition are observed. Neurosurgery such as “split brain” or hemispherectomy, for example, has shown that encompassing regions, the left and right cerebral hemispheres, each control human behavior in unique ways. Observations stretching over 100 years of patients with unilateral focal brain damage have revealed, without the theoretical benefits of “cognitive neuroscience” or “cognitive psychology,” that human behavior is indeed controlled by the brain and its neurons.
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12

Blanke, Olaf, Theodor Landis, Christophe Mermoud, Laurent Spinelli, and Avinoam B. Safran. "Direction-selective motion blindness after unilateral posterior brain damage." European Journal of Neuroscience 18, no. 3 (August 2003): 709–22. http://dx.doi.org/10.1046/j.1460-9568.2003.02771.x.

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13

Borod, Joan C., Ronald L. Bloom, Adam M. Brickman, Luba Nakhutina, and Elizabeth A. Curko. "Emotional Processing Deficits in Individuals With Unilateral Brain Damage." Applied Neuropsychology 9, no. 1 (March 2002): 23–36. http://dx.doi.org/10.1207/s15324826an0901_4.

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14

Sundet, Kjetil. "Sex differences in cognitive impairment following unilateral brain damage." Journal of Clinical and Experimental Neuropsychology 8, no. 1 (January 1986): 51–61. http://dx.doi.org/10.1080/01688638608401296.

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15

HERMSDORFER, J., S. ULRICH, C. MARQUARDT, G. GOLDENBERG, and N. MAI. "Prehension With the Ipsilesional Hand After Unilateral Brain Damage." Cortex 35, no. 2 (1999): 139–61. http://dx.doi.org/10.1016/s0010-9452(08)70791-3.

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16

Caggiano, Pietro, Nicoletta Beschin, and Gianna Cocchini. "Personal Neglect Following Unilateral Right and Left Brain Damage." Procedia - Social and Behavioral Sciences 140 (August 2014): 164–67. http://dx.doi.org/10.1016/j.sbspro.2014.04.403.

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17

Gandour, J., S. Dechongkit, S. Ponglorpisit, F. Khunadorn, and P. Boongird. "Intraword Timing Relations in Thai after Unilateral Brain Damage." Brain and Language 45, no. 2 (August 1993): 160–79. http://dx.doi.org/10.1006/brln.1993.1041.

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18

GANDOUR, J. "Lexical tones in Thai after unilateral brain damage*1." Brain and Language 43, no. 2 (August 1992): 275–307. http://dx.doi.org/10.1016/0093-934x(92)90131-w.

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19

Buchanan, Tony W., Daniel Tranel, and Ralph Adolphs. "Memories for emotional autobiographical events following unilateral damage to medial temporal lobe." Brain 129, no. 1 (November 16, 2005): 115–27. http://dx.doi.org/10.1093/brain/awh672.

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20

Tompkins, Connie A., Richard Boada, and Kathrine McGarry. "The Access and Processing of Familiar Idioms by Brain-Damaged and Normally Aging Adults." Journal of Speech, Language, and Hearing Research 35, no. 3 (June 1992): 626–37. http://dx.doi.org/10.1044/jshr.3503.626.

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Idiom interpretation tasks are routinely used in the clinical evaluation of adults with brain damage, and idiom processing has received increasing attention in the psycholinguistic literature. Clinical evidence suggests that adults with unilateral right-hemisphere damage (RHD) are insensitive to nonliteral meanings conveyed by idiomatic expressions and other figurative forms. However, this portrayal is derived from their terminal responses to tasks that reflect multiple aspects of mental operations (off-line measures), obscuring the source of poor performance. This study used an on-line word-monitoring task to assess RHD, left-hemisphere-damaged, and normally aging adults' implicit knowledge of familiar idiomatic expressions. Brain-damaged subjects performed similarly to normal controls on this task, even though the clinical subjects fared poorly by comparison on an off-line idiom definition measure. These results suggest that adults with unilateral brain damage can activate and retrieve familiar idiomatic forms, and that their idiom-interpretation deficits most likely reflect impairment at some later stage of information processing. Further, error analysis of idiom-definition performance did not support the customary characterization of RHD adults as excessively literal responders The paper discusses clinical implications of the nature and use of idiom interpretation tasks.
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21

Cristinzio, C., K. N'Diaye, M. Seeck, P. Vuilleumier, and D. Sander. "Integration of gaze direction and facial expression in patients with unilateral amygdala damage." Brain 133, no. 1 (October 14, 2009): 248–61. http://dx.doi.org/10.1093/brain/awp255.

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22

TARTAGLIONE, A., M. L. INGLESE, F. BANDINI, L. SPADAVECCHIA, K. HAMSHER, and E. FAVALE. "HEMISPHERE ASYMMETRY IN DECISION MAKING ABILITIES: AN EXPERIMENTAL STUDY IN UNILATERAL BRAIN DAMAGE." Brain 114, no. 3 (June 1, 1991): 1441–56. http://dx.doi.org/10.1093/brain/114.3.1441.

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23

Trauner, D. A. "Behavioural profiles of children and adolescents after pre- or perinatal unilateral brain damage." Brain 124, no. 5 (May 1, 2001): 995–1002. http://dx.doi.org/10.1093/brain/124.5.995.

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24

Larrabee, Glenn J. "Another look at viq-piq scores and unilateral brain damage." International Journal of Neuroscience 29, no. 1-2 (January 1986): 141–48. http://dx.doi.org/10.3109/00207458608985644.

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25

Kempler, Daniel, Diana VanLancker, Virginia Marchman, and Elizabeth Bates. "Idiom comprehension in children and adults with unilateral brain damage." Developmental Neuropsychology 15, no. 3 (January 1999): 327–49. http://dx.doi.org/10.1080/87565649909540753.

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26

Grabowska, Anna, Anna Nowicka, Olga Szymańska, and Iwona Szatkowska. "Subjective contour illusion: sex-related effect of unilateral brain damage." Neuroreport 12, no. 10 (July 2001): 2289–92. http://dx.doi.org/10.1097/00001756-200107200-00048.

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27

Lukauskiene, R., R. Ruseckaite, and B. Mickiene. "624 Object recognition changes in patients with unilateral brain damage." International Journal of Psychophysiology 30, no. 1-2 (September 1998): 237. http://dx.doi.org/10.1016/s0167-8760(98)90623-7.

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28

Trauner, Doris A. "Hemispatial neglect in young children with early unilateral brain damage." Developmental Medicine & Child Neurology 45, no. 3 (February 13, 2007): 160–66. http://dx.doi.org/10.1111/j.1469-8749.2003.tb00924.x.

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29

Borod, J. C., E. Koff, M. P. Lorch, and M. Nicholas. "Channels of Emotional Expression in Patients With Unilateral Brain Damage." Archives of Neurology 42, no. 4 (April 1, 1985): 345–48. http://dx.doi.org/10.1001/archneur.1985.04060040055011.

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30

Tartaglione, Antonio, Giovanni Bino, Marcello Manzino, Luciano Spadavecchia, and Emilio Favale. "Simple reaction-time changes in patients with unilateral brain damage." Neuropsychologia 24, no. 5 (January 1986): 649–58. http://dx.doi.org/10.1016/0028-3932(86)90004-7.

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31

Klepousniotou, Ekaterini, and Shari R. Baum. "Unilateral brain damage effects on processing homonymous and polysemous words." Brain and Language 93, no. 3 (June 2005): 308–26. http://dx.doi.org/10.1016/j.bandl.2004.10.011.

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32

KUCHARSKA-PIETURA, KATARZYNA, and ANTHONY S. DAVID. "The perception of emotional chimeric faces in patients with depression, mania and unilateral brain damage." Psychological Medicine 33, no. 4 (May 2003): 739–45. http://dx.doi.org/10.1017/s0033291702007316.

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Background. Judgements made on chimeric faces elicit reliably a perceptual bias to the left hemispace, presumed to be due to right hemisphere dominance for emotional processes. Major depressive illness has been shown to attenuate this bias. The aim of this work was to examine lateral perceptual bias in bipolar I and II patients in a hypomanic state and unipolar depressed patients and those with unilateral hemisphere damage following stroke.Method. Sixty patients with DSM-IV affective disorder (30 bipolar I or II, currently hypomanic, 30 unipolar depressives), 30 right brain-damaged patients, 30 left brain-damaged patients and 30 healthy controls were given the Happy–Sad Chimeric Faces Test.Results. Right hemisphere damaged and unipolar depressed patients both showed a significantly reduced left hemispatial bias (LHB) compared to controls, bipolars and left brain-damaged patients. No significant difference in mean LHB between controls and both hypomanics and left brain-damaged patients was found. There was no significant association between LHB and clinical variables.Conclusions. The results suggest a physiological distinction between bipolar and unipolar depression. The significantly diminished left hemifacial bias in depressed patients suggests right hemisphere dysfunction.
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33

Oliveri, M., P. M. Rossini, R. Traversa, P. Cicinelli, M. M. Filippi, P. Pasqualetti, F. Tomaiuolo, and C. Caltagirone. "Left frontal transcranial magnetic stimulation reduces contralesional extinction in patients with unilateral right brain damage." Brain 122, no. 9 (September 1999): 1731–39. http://dx.doi.org/10.1093/brain/122.9.1731.

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34

Walker, Robin, and Jason B. Mattingley. "Pathological completion: The blind leading the mind?" Behavioral and Brain Sciences 21, no. 6 (December 1998): 778–79. http://dx.doi.org/10.1017/s0140525x98561754.

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The taxonomy proposed by Pessoa et al. should be extended to include “pathological” completion phenomena in patients with unilateral brain damage. Patients with visual field defects (hemianopias) may “complete” whole figures, while patients with parietal lobe damage may “complete” partial figures. We argue that the former may be consistent with the brain “filling-in” information, and the latter may be consistent with the brain ignoring the absence of information.
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35

Winstein, C. J., and S. Onla-or. "Effects of Unilateral Brain Damage on the Control of Aiming Movements." Neurology Report 20, no. 4 (1996): 21. http://dx.doi.org/10.1097/01253086-199620040-00019.

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36

Kucharska-Pietura, K., and B. Loza. "The assessment of lateral preference in schizophrenia and unilateral brain damage." European Neuropsychopharmacology 11 (January 2001): S284—S285. http://dx.doi.org/10.1016/s0924-977x(01)80369-2.

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37

Hermsdörfer, J., and G. Goldenberg. "Ipsilesional deficits during fast diadochokinetic hand movements following unilateral brain damage." Neuropsychologia 40, no. 12 (January 2002): 2100–2115. http://dx.doi.org/10.1016/s0028-3932(02)00048-9.

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38

Kucharska-Pietura, Katarzyna, Mary L. Phillips, Wojciech Gernand, and Anthony S. David. "Perception of emotions from faces and voices following unilateral brain damage." Neuropsychologia 41, no. 8 (January 2003): 1082–90. http://dx.doi.org/10.1016/s0028-3932(02)00294-4.

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39

Barrett, Anna M. "Treatment of Unilateral Neglect in Patients With Right Hemisphere Brain Damage." Perspectives on Neurophysiology and Neurogenic Speech and Language Disorders 10, no. 4 (December 2000): 18–26. http://dx.doi.org/10.1044/nnsld10.4.18.

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40

Gandour, J., S. Ponglorpisit, S. Dechongkit, F. Khunadorn, P. Boongird, and S. Potisuk. "Anticipatory Tonal Coarticulation in Thai Noun Compounds after Unilateral Brain Damage." Brain and Language 45, no. 1 (July 1993): 1–20. http://dx.doi.org/10.1006/brln.1993.1030.

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41

Gandour, Jack, Suvit Ponglorpisit, Siripong Potisuk, Fuangfa Khunadorn, Prasert Boongird, and Sumalee Dechongkit. "Interaction between Tone and Intonation in Thai after Unilateral Brain Damage." Brain and Language 58, no. 1 (June 1997): 174–96. http://dx.doi.org/10.1006/brln.1997.1768.

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42

Mosidze, Vakhtang M., Rusudan A. Mkheidze, and Malhas A. Makashvili. "Disorders of visuo-spatial attention in patients with unilateral brain damage." Behavioural Brain Research 65, no. 1 (November 1994): 121–22. http://dx.doi.org/10.1016/0166-4328(94)90081-7.

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43

Hart, Emerson, and Michelle Woodbury. "Visual Search Strategy and Balance in Children After Unilateral Brain Damage." Archives of Physical Medicine and Rehabilitation 104, no. 3 (March 2023): e64. http://dx.doi.org/10.1016/j.apmr.2022.12.186.

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44

Braun, C. M. J., I. Montour-Proulx, S. Daigneault, I. Rouleau, S. Kuehn, M. Piskopos, G. Desmarais, F. Lussier, and C. Rainville. "Prevalence, and Intellectual Outcome of Unilateral Focal Cortical Brain Damage as a Function of Age, Sex and Aetiology." Behavioural Neurology 13, no. 3-4 (2002): 105–16. http://dx.doi.org/10.1155/2002/634764.

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Neurologists and neuropsychologists are aware that aging men are more at risk than women for brain damage, principally because of the well known male-predominant risk for cardiovascular disease and related cerebrovascular accidents. However, a disproportion in prevalence of brain damage between the sexes in childhood may be less suspected. Furthermore, sex-specific risk for other aetiologies of brain damage may be little known, whether in the pediatric or adult populations. Proposals of a sex difference in cognitive recovery from brain damage have also been controversial. Six hundred and thirty five “consecutive” cases with cortical focal lesions including cases of all ages and both sexes were reviewed. Aetiology of the lesion was determined for each case as was postlesion IQ. Risk was highly male prevalent in all age groups, with a predominance of cardiovascular aetiology explaining much of the adult male prevalence. However, several other aetiological categories were significantly male prevalent in juveniles (mitotic, traumatic, dysplasic) and adults (mitotic, traumatic). There was no sex difference in outcome (i.e., postlesion IQ) of these cortical brain lesions for the cohort as a whole, after statistical removal of the influence of lesion extent, aetiology and presence of epilepsy. Mechanisms potentially responsible for sex differences in prevalence, aetiology of brain damage, and recovery, are reviewed and discussed.
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45

Sakaguchi, Yukitoshi, and Yoshio Sakurai. "Paradoxical Enhancement of Spatial Learning Induced by Right Hippocampal Lesion in Rats." Symmetry 13, no. 11 (November 10, 2021): 2138. http://dx.doi.org/10.3390/sym13112138.

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The left–right hemispheric differences in some brain functions are well known in humans. Among them, savant syndrome has unique features, such as exceptional abilities in vision, memory, computation, and music, despite brain abnormalities. In cases of acquired savant and transient savant, brain damage or inhibition is often seen in the left hemisphere, suggesting a link between left hemispheric dysfunction and these talents. On the other hand, some functional left–right differences have been reported in rodent brains, and therefore, unilateral damage in rodents may also result in savant-like enhancements. In the present study, we examined the effects of hippocampal damage on spatial learning in rats with left, right, or bilateral hippocampal lesion. The results showed that learning performance was impaired in the bilateral lesion group, and there was no significant difference in the left lesion group, while performance was enhanced in the right lesion group. These results suggest that damage to the right hippocampus in rats may lead to savant-like enhancement in learning and memory. The construction of the savant model through these results will contribute to the neuroscientific elucidation of the paradoxical phenomenon observed in savants, that some abilities are enhanced despite their brain dysfunction.
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46

Borod, J. C., E. Koff, M. P. Lorch, M. Nicholas, and J. Welkowitz. "Emotional and non-emotional facial behaviour in patients with unilateral brain damage." Journal of Neurology, Neurosurgery & Psychiatry 51, no. 6 (June 1, 1988): 826–32. http://dx.doi.org/10.1136/jnnp.51.6.826.

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47

Delis, Dean C., Mark G. Kiefner, and Alan J. Fridlund. "Visuospatial dysfunction following unilateral brain damage: Dissociations in hierarchical and hemispatial analysis." Journal of Clinical and Experimental Neuropsychology 10, no. 4 (August 1988): 421–31. http://dx.doi.org/10.1080/01688638808408250.

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48

Nass, Ruth, and Diane Koch. "Temperament differences in toddlers with early unilateral right‐ and left‐brain damage." Developmental Neuropsychology 3, no. 2 (January 1987): 93–99. http://dx.doi.org/10.1080/87565648709540367.

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49

Salazar-López, E., B. J. Schwaiger, and J. Hermsdörfer. "Lesion correlates of impairments in actual tool use following unilateral brain damage." Neuropsychologia 84 (April 2016): 167–80. http://dx.doi.org/10.1016/j.neuropsychologia.2016.02.007.

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50

Mihaltchev, P., P. S. Archambault, A. G. Feldman, and M. F. Levin. "Control of double-joint arm posture in adults with unilateral brain damage." Experimental Brain Research 163, no. 4 (February 3, 2005): 468–86. http://dx.doi.org/10.1007/s00221-004-2202-9.

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