Relevant bibliographies by topics / Unilateral brain damage

Academic literature on the topic 'Unilateral brain damage'

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Published: 14 March 2023

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Journal articles on the topic "Unilateral brain damage"

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Winstein, C. "Motor learning after unilateral brain damage." Neuropsychologia 37, no. 8 (July 1, 1999): 975–87. http://dx.doi.org/10.1016/s0028-3932(98)00145-6.

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Schapiro, Anna C., James L. McClelland, Stephen R. Welbourne, Timothy T. Rogers, and Matthew A. Lambon Ralph. "Why Bilateral Damage Is Worse than Unilateral Damage to the Brain." Journal of Cognitive Neuroscience 25, no. 12 (December 2013): 2107–23. http://dx.doi.org/10.1162/jocn_a_00441.

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Human and animal lesion studies have shown that behavior can be catastrophically impaired after bilateral lesions but that unilateral damage often produces little or no effect, even controlling for lesion extent. This pattern is found across many different sensory, motor, and memory domains. Despite these findings, there has been no systematic, computational explanation. We found that the same striking difference between unilateral and bilateral damage emerged in a distributed, recurrent attractor neural network. The difference persists in simple feedforward networks, where it can be understood in explicit quantitative terms. In essence, damage both distorts and reduces the magnitude of relevant activity in each hemisphere. Unilateral damage reduces the relative magnitude of the contribution to performance of the damaged side, allowing the intact side to dominate performance. In contrast, balanced bilateral damage distorts representations on both sides, which contribute equally, resulting in degraded performance. The model's ability to account for relevant patient data suggests that mechanisms similar to those in the model may operate in the brain.
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Doricchi, Fabrizio, Gaspare Galati, Lara DeLuca, Daniele Nico, and Francesca D’Olimpio. "Horizontal space misrepresentation in unilateral brain damage." Neuropsychologia 40, no. 8 (January 2002): 1107–17. http://dx.doi.org/10.1016/s0028-3932(02)00010-6.

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Doricchi, Fabrizio, Alessandra Onida, and Paola Guariglia. "Horizontal space misrepresentation in unilateral brain damage." Neuropsychologia 40, no. 8 (January 2002): 1118–28. http://dx.doi.org/10.1016/s0028-3932(02)00011-8.

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Soffer, Dov, Eldad Melamed, Yaakov Assaf, and Shamay Cotev. "Hemispheric brain damage in unilateral status epilepticus." Annals of Neurology 20, no. 6 (December 1986): 737–40. http://dx.doi.org/10.1002/ana.410200616.

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Ludwig, Casimir J. H., Stephen H. Butler, Stephanie Rossit, Monika Harvey, and Iain D. Gilchrist. "Modelling contralesional movement slowing after unilateral brain damage." Neuroscience Letters 452, no. 1 (March 2009): 1–4. http://dx.doi.org/10.1016/j.neulet.2009.01.033.

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Kucharska-Pietura, K., B. Loza, and G. Kopacz. "Emotional control in schizophrenia and unilateral brain damage." European Neuropsychopharmacology 11 (January 2001): S284. http://dx.doi.org/10.1016/s0924-977x(01)80368-0.

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Noetzel, Michael J. "Theophylline Neurotoxicity Resulting in Significant Unilateral Brain-damage." Developmental Medicine & Child Neurology 27, no. 2 (November 12, 2008): 242–45. http://dx.doi.org/10.1111/j.1469-8749.1985.tb03775.x.

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Gandour, J., S. Ponglorpisit, F. Khunadorn, S. Dechongkit, P. Boongird, and R. Boonklam. "Stop voicing in thai after unilateral brain damage." Aphasiology 6, no. 6 (November 1992): 535–47. http://dx.doi.org/10.1080/02687039208249490.

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Gandour, Jack, Siripong Potisuk, Suvit Ponglorpisit, Sumalee Dechongkit, Fuangfa Khunadorn, and Prasert Boongird. "Tonal Coarticulation in Thai after Unilateral Brain Damage." Brain and Language 52, no. 3 (March 1996): 505–35. http://dx.doi.org/10.1006/brln.1996.0027.

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Dissertations / Theses on the topic "Unilateral brain damage"

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McIntosh, Robert D. "Unilateral neglect : visual and manual." Thesis, Glasgow Caledonian University, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287857.

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Halligan, Peter W. "The behavioural assessment of unilateral visual neglect." Thesis, Oxford Brookes University, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.328077.

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Sullivan, Carol-Anne Maria. "The effects of lateralized stimulation on unilateral visuo-spatial neglect." Thesis, University of York, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341860.

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Bury, Scott Douglas. "Denervation facilitates motor skills learning with the "unaffected" forelimb in adult rats with unilateral sensorimotor cortex lesions /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/9134.

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Klepousniotou, Ekaterini. "On the resolution of lexical ambiguity : unilateral brain damage effects on the processing of homonymy and polysemy." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85078.

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An increasing amount of evidence suggests that both cerebral hemispheres contribute to the comprehension of semantic relations. A literature review of language abilities after right hemisphere (RH) damage reveals abnormalities in the interpretation of lexical items that have alternate meanings (Chiarello, 1991). Two major theories have been proposed to account for the lexical-semantic deficits observed in RH damaged (RHD) individuals, namely the "suppression deficit" and the "coarse semantic coding" hypotheses. By exploiting the theoretical linguistic distinction of lexical ambiguity into homonymy, metaphor and metonymy, the present investigation attempts to directly contrast the predictions of the two hypotheses. To this end, two on-line priming studies were developed, each comprising two experiments conducted with RHD patients, individuals with nonfluent aphasia subsequent to left hemisphere damage (LHD) and non-brain-damaged control participants (NC).
The first single-word priming study showed that, for all three subject groups (NC, LHD, RHD), the multiple meanings of homonymous and metonymous words can be triggered and accessed out of context, whereas for metaphorical words, context seems to be necessary in order to access secondary metaphorical meanings. The second sentence priming study showed that the RHD patients' performance contrasts with the performance of the normal control and LHD non-fluent aphasic subjects who showed both effects of context and the time-course of processing, as well as comparable processing across the three types of ambiguous words. For the RHD patients, although homonymous and metonymous words showed relatively normal patterns of activation, the subordinate meanings of metaphors were not activated, suggesting a selective problem with figurative meanings.
Taken together, these findings suggest that the lexical ambiguity processing deficit observed after RH damage is mostly evident when processing subordinate meanings of metaphorically ambiguous words (even in the presence of biasing context), although a lesion in one hemisphere does not completely disrupt the ability of individuals to appreciate the alternative meanings of ambiguous words at the single-word level. The findings were contrary to the strongest expectations of both the "coarse semantic coding" and the "suppression deficit" hypotheses of RH language abilities; however, they seem to be more consistent with a weaker version of the "suppression deficit" hypothesis according to which RH damage leads to deficits in contextual integration and selection of appropriate meanings.
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Cicerale, Alessandro. "Perception and motor control in healthy and brain damaged patients." Doctoral thesis, SISSA, 2013. http://hdl.handle.net/20.500.11767/4786.

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Wild, Celia. "Contributions of the left and right hemisphere in language : investigating the effects of unilateral brain damage (stroke) on metaphor processing." Thesis, University of Leeds, 2012. http://etheses.whiterose.ac.uk/3134/.

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It is widely accepted that the left hemisphere of the brain is specialised and dominant for language comprehension and production and that those with left hemisphere damage often display profound language disruption (Geschwind, 1965). The importance of the left hemisphere is shown by communication problems or extreme difficulty in producing speech following damage to this brain region. In contrast, following right hemisphere damage, disruption to language is less perceptible to the casual observer. The evidence base currently available acknowledges a critical role for the right hemisphere in processing inferred or implied information by maintaining relevant facts and/or suppressing irrelevant ones but the exact role of the right hemisphere and its coordination with the left is open for debate (Johns, Tooley and Traxler, 2008). Two theories have been proposed to explain communication/language difficulties in individuals with right hemisphere damage: (i) the “coarse semantic coding” hypothesis and (ii) the “suppression deficit” hypothesis. The “coarse semantic coding” hypothesis proposes that damage to the right hemisphere causes an over reliance on fine coding assumed to be undertaken by the left hemisphere in the comprehension of language, implying the recall of most literal interpretations. The “suppression deficit” hypothesis proposes that damage in the right hemisphere means multiple activations of meanings of words are not attenuated leading to ineffective suppression of inappropriate interpretations. This project investigated competing evidence for each of these hypotheses by studying the processing abilities of individuals with depressed unilateral brain function caused by stroke or innovatively produced by transcranial DC stimulation (tDCS), on semantic judgement tasks using metaphorical language. The results demonstrated the strongest of evidence for the coarse semantic coding hypothesis when the data from participants with damage to the right hemisphere, both caused by stroke and simulated by tDCS was considered. Overall, the study has furthered the understanding of the role of the right hemisphere in language comprehension and demonstrated the contribution of the tDCS methodology in the field.
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Barrette, Martine. "Contributions of the right and left hemispheres to lexical ambiguity resolution : evidence from unilaterally brain-damaged adults." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=32978.

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The present experiment was conducted to explore the time-course of lexical ambiguity resolution in normal control (NC) subjects and the nature of right hemisphere-damaged (RHD) and left hemisphere-damaged (LHD) patients' impairments in this process. NC, RHD and LHD subjects performed a cross-modal lexical decision task, in which they heard sentence contexts that were biased toward the dominant or subordinate meanings of ambiguous words occurring before the end of the sentence. Written target words related to the dominant or subordinate meaning of the ambiguous words were introduced either at the onset of the ambiguous word (immediate condition), or 1000 ms later (delayed condition). Results revealed that both dominant and subordinate meanings were primed in the immediate condition, irrespective of context type or group. In the delayed condition, only the contextually-appropriate dominant meanings were primed in normal control subjects, whereas both patient groups showed significant priming only for the contextually-inappropriate dominant meanings. Findings for the NC subjects are interpreted in support of a modular model of lexical ambiguity resolution, and more specifically of an exhaustive access view. Patients' results are discussed with reference to a delayed suppression mechanism of inappropriate meanings, which is thought to be involved in these patients' language comprehension deficits.
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Hebda-Bauer, Elaine K. "Familiarity and cue use effects of unilateral hippocampal damage in disoriented animals : a research report submitted in partial fulfillment ... for the degree of Master of Science (Medical-Surgical Nursing) ... /." 1993. http://catalog.hathitrust.org/api/volumes/oclc/68797229.html.

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Books on the topic "Unilateral brain damage"

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Robertson, Ian, and John Marshall. Unilateral Neglect: Clinical and Experimental Studies. Taylor & Francis Group, 2013.

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Robertson, Ian, and John Marshall. Unilateral Neglect: Clinical and Experimental Studies. Taylor & Francis Group, 2013.

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Robertson, Ian, and John Marshall. Unilateral Neglect: Clinical and Experimental Studies. Taylor & Francis Group, 2013.

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Robertson, Ian, and John Marshall. Unilateral Neglect: Clinical and Experimental Studies. Taylor & Francis Group, 2013.

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Robertson, Ian. Unilateral Neglect: Clinical And Experimental Studies (Brain Damage, Behaviour, and Cognition). Psychology Press, 1993.

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Robertson, Ian. Unilateral Neglect: Clinical And Experimental Studies (Brain Damage, Behaviour and Cognition Series). Psychology Press, 1993.

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Vallar, Giuseppe, and Nadia Bolognini. Unilateral Spatial Neglect. Edited by Anna C. (Kia) Nobre and Sabine Kastner. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199675111.013.012.

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Left unilateral spatial neglect is the most frequent and disabling neuropsychological syndrome caused by lesions to the right hemisphere. Over 50% of right-brain-damaged patients show neglect, while right neglect after left-hemispheric damage is less frequent. Neglect patients are unable to orient towards the side contralateral to the lesion, to detect and report sensory events in that portion of space, as well as to explore it by motor action. Neglect is a multicomponent disorder, which may involve the contralesional side of the body or of extra-personal physical or imagined space, different sensory modalities, specific domains (e.g. ‘neglect dyslexia’), and worsen sensorimotor deficits. Neglect is due to higher-order unilateral deficits of spatial attention and representation, so that patients are not aware of contralesional events, which, however, undergo a substantial amount of unconscious processing up to the semantic level. Cross-modal sensory integration is also largely preserved. Neglect is primarily a spatially specific disorder of perceptual consciousness. The responsible lesions involve a network including the fronto-temporo-parietal cortex (particularly the posterior-inferior parietal lobe, at the temporo-parietal junction), their white matter connections, and some subcortical grey nuclei (thalamus, basal ganglia). Damage to primary sensory and motor regions is not associated to neglect. A variety of physiological lateralized and asymmetrical sensory stimulations (vestibular, optokinetic, prism adaptation, motor activation), and transcranial electrical and magnetic stimulations, may temporarily improve or worsen neglect. Different procedures have been successfully developed to rehabilitate neglect, using both ‘top down’ (training the voluntary orientation of attention) and ‘bottom up’ (the above-mentioned stimulations) approaches.
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Spatial Neglect: A Clinical Handbook for Diagnosis and Treatment (Brain Damage, Behaviour, and Cognition). Psychology Press, 1999.

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Book chapters on the topic "Unilateral brain damage"

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Bartolomeo, Paolo. "Unilateral Spatial Neglect: Clinical Aspects." In Attention Disorders After Right Brain Damage, 49–83. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-5649-9_4.

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Zoccolotti, Pierluigi, Carlo Caltagirone, Anna Pecchinenda, and Elio Troisi. "Electrodermal Activity in Patients with Unilateral Brain Damage." In Progress in Electrodermal Research, 311–26. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2864-7_21.

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Etcoff, N. L. "Recognition of Emotions in Patients with Unilateral Brain Damage." In Emotions and the Dual Brain, 168–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73396-3_9.

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Kimura, Doreen, and Robert Faust. "Spontaneous Drawing in an Unselected Sample of Patients with Unilateral Cerebral Damage." In Duality and Unity of the Brain, 114–46. London: Palgrave Macmillan UK, 1987. http://dx.doi.org/10.1007/978-1-349-08940-6_9.

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Kimura, Doreen, and Robert Faust. "Spontaneous Drawing in an Unselected Sample of Patients with Unilateral Cerebral Damage." In Duality and Unity of the Brain, 114–46. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1949-8_9.

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Gainotti, Guido. "Disorders of Emotional Behaviour and of Autonomic Arousal Resulting from Unilateral Brain Damage." In Duality and Unity of the Brain, 161–79. London: Palgrave Macmillan UK, 1987. http://dx.doi.org/10.1007/978-1-349-08940-6_11.

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Gainotti, Guido. "Disorders of Emotional Behaviour and of Autonomic Arousal Resulting from Unilateral Brain Damage." In Duality and Unity of the Brain, 161–79. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1949-8_11.

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Prosiegel, M., R. Höling, M. Heintze, E. Wagner-Sonntag, and K. Wiseman. "Swallowing therapy — a prospective study on patients with neurogenic dysphagia due to unilateral paresis of the vagal nerve, Avellis’ syndrome, Wallenberg’s syndrome, posterior fossa tumours and cerebellar hemorrhage." In Re-Engineering of the Damaged Brain and Spinal Cord, 35–37. Vienna: Springer Vienna, 2005. http://dx.doi.org/10.1007/3-211-27577-0_4.

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Prosiegel, M., R. Höling, M. Heintze, E. Wagner-Sonntag, and K. Wiseman. "The localization of central pattern generators for swallowing in humans — a clinical-anatomical study on patients with unilateral paresis of the vagal nerve, Avellis’ syndrome, Wallenberg’s syndrome, posterior fossa tumours and cerebellar hemorrhage." In Re-Engineering of the Damaged Brain and Spinal Cord, 85–88. Vienna: Springer Vienna, 2005. http://dx.doi.org/10.1007/3-211-27577-0_13.

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Belfi, Amy M., Agathe Pralus, Catherine Hirel, Daniel Tranel, Barbara Tillmann, and Anne Caclin. "Investigating Musical Emotions in People with Unilateral Brain Damage." In Brain, Beauty, and Art, 170–74. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780197513620.003.0034.

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The study under discussion sought to investigate the hemispheric laterality of musical emotions: Is one hemisphere of the brain preferentially involved in recognizing emotions in music? The authors took a neuropsychological approach to answer this question by studying emotional judgments of music in people with brain damage to either hemisphere. Their results indicated that individuals with left hemisphere damage were significantly impaired in recognizing musical emotions as compared to healthy comparison participants. In contrast, individuals with right hemisphere damage were not impaired at identifying emotions in music, but rated the perceived intensity of the emotions lower for sadness and fear (as compared to joy and serenity). Their work suggests that the identification of emotions in music and the perceived intensity of the emotions expressed may rely on different hemispheres of the brain.
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Conference papers on the topic "Unilateral brain damage"

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Bush, Ronald A., and Theodore P. Williams. "Effect of optic nerve section on retinal light damage." In OSA Annual Meeting. Washington, D.C.: Optica Publishing Group, 1985. http://dx.doi.org/10.1364/oam.1985.wj45.

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Sprague-Dawley rats, age 20-24 weeks, were obtained from Zivic-Miller Laboratories with unilateral intracranial optic nerve sections which did not interrupt the blood flow or any other innervation to the eye. They were then kept in uniform illumination of 1000 lx continuously for 24 h. The outer nuclear layer (ONL) areas of the two eyes in the same animal were compared five days postexposure. Light of this intensity causes damage primarily to the photoreceptors in the retina of the albino rat. Since the ONL of the retina contains photoreceptor cell bodies, reduction in the area of this layer is proportional to the number of cells lost due to the bright light exposure. Although the ONL area was reduced in both eyes, the retina with the intact optic nerve had an ONL area which averaged ~25 % less than that in the other eye with the optic nerve cut. Controls with unilateral optic nerve sections performed in the same manner but not exposed to damaging light showed no difference in ONL area between the two eyes. A second experiment using pigmented rats gave similar results. These results suggest a role for the brain in retinal light damage.
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Araújo, Maria Tereza Corrêa de, Amanda Virgínia Oliveira Leite, Ana Clara Sousa Leal, Daniel Jonatan de Aguiar Almeida, Iris Maria de Miranda Correia, and David Plácido Lopes. "AGenesis of the internal carotid artery (ICA agenesis): an integrative review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.308.

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Introduction: The ICA agenesis is a rare anomaly, an incidental finding whose incidence is lower than 0,01%. It is predominantly asymptomatic, because of the development of a collateral circulation, though it can present nonspecific symptoms, that result from insufficient blood flow, due to the development of atherosclerosis in vessels that previously supported the blood flow in the absence of the ICA, and by the association with brain aneurysm. Objectives: Conduct an integrative review about the ICA agenesis, describing the aspects and importance of vascular changes. Methods: It is an integrative revision that includes articles indexed in the SCIELO and PUBMED databases, using descriptors, with articles from the last 5 years. Results: The ICA agenesis can be unilateral or bilateral, predominantly on the left side. The main collateral pathways are the Circle of Willis and the transcranial collateral vessels of the external carotid artery. Differential diagnosis, like total occlusion or dissection, should be excluded by the inspection of the carotid canal in the CT, because the demonstration of a normal carotid canal excludes developmental anomalies of the ICA. The MRI and the angiography, combined with CT and Doppler ultrasound allow an accurate diagnosis. Conclusion: It is important to consider the possibility of ICA agenesis in the investigation of nonspecific symptoms, when other more prevalent causes have already been discarded, since these patients have an increased incidence of several other intracranial diseases, such as cerebral aneurysms with an association of 25-44%, that if identified in time, can avoid severe damages.
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