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Books on the topic 'Ulcerative syndrome'

Author: Grafiati

Published: 18 May 2024

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1

University, Barkatullah, ed. Investigations on the epizoolitic ulcerative syndrome of fishes in the water bodies in and around Bhopal with special references to Halali reservoir: Project completion report. [Bhopal: Barktullah University], 1997.

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2

Sindermann, Carl J. Epizootic ulcerative syndromes in coastal/estuarine fish. Woods Hole, Mass: U.S. Department of Commerce, National Oceanic and Atmospheric Administration, National Marine Fisheries Service, Northeast Fisheries Center, 1988.

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3

Epizootic Ulcerative Fish Disease Syndrome. Elsevier, 2016. http://dx.doi.org/10.1016/c2014-0-03152-8.

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4

Kar, Devashish. Epizootic Ulcerative Fish Disease Syndrome. Elsevier Science & Technology Books, 2015.

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5

Kar, Devashish. Epizootic Ulcerative Fish Disease Syndrome. Elsevier Science & Technology Books, 2015.

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6

Publications, Black. IBD Journal: Irritable Bowel Syndrome - Food Diary and Symptoms Tracker for Ulcerative Colitis, Crohn's Disease and Other Digestive Disorders. Independently Published, 2021.

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7

Monastyrsky, Konstantin. Fiber Menace: The Truth About the Leading Role of Fiber in Diet Failure, Constipation, Hemorrhoids, Irritable Bowel Syndrome, Ulcerative Colitis, Crohn's Disease, and Colon Cancer. Ageless Press, 2005.

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8

Publication, Asifesisler. Colitis and Crohn's Disease Journal and Planner: Log Your Pain Levels, Meals, Symptoms and More. Use for Irritable Bowel Syndrome , Ulcerative Colitis, Celiac Log Book Diary. Independently Published, 2022.

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9

Sparks, Orora P. My IBD Food Journal: Food Diary and Tracker for Ulcerative Colitis, Crohns, IBS and Other Digestive Disorders Symptom Management Log Food Sensitivity Journal Book Ibs Awareness Eating for Ibs Food Journal for Intolerance down Syndrome Diary. Independently Published, 2021.

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10

Jacquet, Gabrielle, and Lawrence Page. Odontogenic Infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0013.

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Odontogenic infections often arise from dental caries (usually the mandibular teeth) or from dental extraction. Acute necrotizing ulcerative gingivitis (ANUG) is more common in immunocompromised patients. These infections may spread into the parapharyngeal and retropharyngeal spaces, involving the airway and mediastinum. Airway management is critical as odontogenic infections can compromise airways via mass effect. Complications include the following: abscess, facial or orbital cellulitis/abscess, intracranial invasion, Ludwig’s angina, Lemierre syndrome, carotid artery erosion, descending necrotizing mediastinitis, airway compromise, hematogenous dissemination to distant organs, intraoral or dentocutaneous fistula formation, and cardiovascular disease. Antibiotics are not a substitute for definitive airway management. In addition, many cases of odontogenic infection will require surgical drainage, either at the bedside in the emergency department or in the operating room. Prior to this, consider using a nerve block to obtain anesthesia to the affected area of the face. Patients with necrotizing infections need emergent surgery with wide local debridement.

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11

Yurdakul, Sebahattin, Emire Seyahi, and Hasan Yazici. Behçet’s syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0135.

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Behçet's syndrome is a systemic inflammatory panvasculitis (affecting all sizes of vessels) of unknown aetiology. It is in vogue to include it among the systemic autoinflammatory conditions. Behçet's syndrome is more frequent along the ancient 'Silk Route' across Asia than it is in Western countries. The usual onset is the second or third decade, equally affecting either gender. However, young patients and male patients have more severe disease. Almost all patients have recurrent oral ulceration. Scar-forming genital ulcers, a variety of skin lesions including acneiform, erythema nodosum-like lesions, arthritis, potentially blinding panuveitis, thrombophlebitis, gastrointestinal disease, central nervous system (CNS) involvement, and life-threatening bleeding pulmonary artery aneurysms are seen. The pathergy phenomenon is a heightened tissue inflammatory response. The strongest genetic association is with HLA B51. There are immunological aberrations but not prominent enough to call it an autoimmune disease. Similarly, Behçet's syndrome does not fit easily into the broad concept of autoinflammatory diseases. The histopathology is also non-specific and the diagnosis is mainly clinical. Differentiation from Crohn's disease is very difficult. In more than one-half of the patients the disease burns out in time, thus only symptomatic therapy is indicated in some patients. However, eye involvement, pulmonary vascular disease, thrombophilic complications, CNS involvement, and gastrointestinal disease need prompt recognition and treatment. Brief courses of glucocorticosteroids along with immunosuppressives including the newer biologicals, interferon, and colchicine are commonly used. However, controlled clinical trials are not available for some of these medications especially when thrombophilia, CNS, and gastrointestinal disease are present.

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12

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Sexually transmitted infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0018.

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This chapter covers the diagnosis and management of sexually transmitted infections, including bacterial vaginosis, with causes including vaginal discharge, vulvovaginal candidiasis, and trichomoniasis. The chapter also covers vulvovaginal candidiasis, genital warts or anogenital warts caused by human papillomavirus, tropical genital ulceration (which is commoner in patients presenting with sexually transmitted infections in the developing world and is an important factor in the spread of HIV), genital herpes, pelvic inflammatory disease, toxic shock syndrome, gonorrhoea, chlamydia, trichomoniasis, and syphilis.

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13

Coppola, Silvia, and Franco Valenza. Inhalation injury in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0107.

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Inhalation injury represents one of the most serious associated injuries complicating the care of thermally-injured patient. It can result in severe respiratory failure and acute respiratory distress syndrome (ARDS) by three mechanisms—thermal or chemical injury, and impairment of systemic oxygen supply. Thermal injury can cause erythema, ulceration, and progressive, life-threatening oedema, particularly of the upper airways. Chemical injury is due to irritants or cytotoxic compounds, and depends on the material burned, the temperature of the fire, and the amount of oxygen present in the fire environment. It is responsible for irritation, ulceration, and oedema of the mucosal surface, and the initiation of a lung inflammatory reaction when small particles reach the alveoli. Moreover, the increased vascular permeability, and the reduced surfactant production carry a significant risk in the development of pneumonia and ARDS. Bronchospasm and upper airway oedema can occur rapidly, while lower airway oedema can be asymptomatic for up to 24 hours. Lung imaging techniques may not reveal injured areas for the first 24–48 hours. Fibre optic bronchoscopy is considered to be the most direct diagnostic method for the definitive diagnosis of inhalation injury. The patient management includes airways assessment, adequate fluid resuscitation, and mechanical ventilation when required. All victims of smoke inhalation should be always evaluated for cyanide and carbon monoxide poisoning.

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14

Ahmed, Osmanuddin. Management of Chronic Iliocaval Thrombosis. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0036.

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Chronic deep venous thrombosis (DVT) is defined by thrombus persisting beyond 28 days of initial onset and represents a condition that leads to long-standing venous hypertension, valvular incompetence, and ultimately chronic venous insufficiency and occlusion. As chronic thrombus organizes, it contracts and becomes adherent to the vein wall, causing scarring and consequent atresia of the lumen. The sequelae of such disease are manifested by limb edema, pain, discoloration, exercise intolerance, and ulceration—all encompassed within a clinical spectrum known as post-thrombotic syndrome. Attempts at recanalization with venoplasty and stenting of chronic inferior vena cava (IVC) and iliac occlusions are performed to restore the main venous outflow of the extremities to reverse or arrest the morbidity associated with this disease. This chapter discusses the interventional management of chronic iliocaval thrombosis.

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15

Saha, Sudip. Septic Thrombophlebitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0021.

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Septic (suppurative) thrombophlebitis is venous thrombosis in the setting of bacteremia. There is usually a degree of perivascular inflammation seen on histology. Septic thrombophlebitis occurs most commonly with intravenous catheters. However, most cases of infection related to intravenous catheters are not complicated by septic thrombophlebitis. Catheter-related septic thrombophlebitis includes erythema, tenderness, and/or drainage at the site of an intravenous catheter. Jugular vein septic thrombophlebitis, also known as Lemierre’s syndrome, is a subset of septic thrombophlebitis. This condition can affect otherwise young, healthy adults and is often preceded by pharyngitis with tonsillar and peritonsillar involvement, dental infections, or infectious mononucleosis. Presentation of jugular vein septic thrombophlebitis includes high fevers, rigors, respiratory distress, ulceration or erythema of the oropharynx, and tenderness and swelling of the neck. Primary treatment of thrombophlebitis includes removal of infected materials, intravenous antibiotics, and possible anticoagulation.

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