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Books on the topic 'Ubiquitin ligase'

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Author: Grafiati
Published: 4 June 2021
Last updated: 21 February 2023

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1

Di, Napoli Mario, and Wójcik Cezary 1968-, eds. The ubiquitin proteasome system in the central nervous system: From physiology to pathology : 2008 update. Hauppauge, NY: Nova Science, 2009.

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2

Hyŏmnyŏktan, Koryŏ Taehakkyo Sanhak. E3, ubiquitin ligase chŏhaeje rŭl wihan E1-E2-E3-substrate cognate pair network chŏngnip kisul kaebal kwa i rŭl iyong han tanangsŏng sinjŭnghugun (ADPKD) ch'iryoje kaebal yŏn'gu =: Study on E1-E2-E3-substrate cognate pair network for E3 ligase inhibitor and application. [Seoul]: Pogŏn Pokchi kajokpu, 2008.

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3

Inuzuka, Hiroyuki, and Wenyi Wei. SCF and APC E3 Ubiquitin Ligases in Tumorigenesis. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-05026-3.

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4

Inuzuka, Hiroyuki. SCF and APC E3 ubiquitin ligases in tumorigenesis. Cham: Springer, 2014.

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5

Harris, Edward T. Ubiquitin Ligase: New Insights, Emerging Roles and Clinical Implications. Nova Science Publishers, Incorporated, 2017.

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6

Groettrup, Marcus. Conjugation and Deconjugation of Ubiquitin Family Modifiers. Springer, 2010.

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7

Groettrup, Marcus. Conjugation and Deconjugation of Ubiquitin Family Modifiers. Springer, 2016.

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8

Conjugation And Deconjugation Of Ubiquitin Family Modifiers. Springer, 2010.

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9

Smith, Derek B. Investigating a role for the ubiquitin ligase Itch in lymphocyte development. 2005.

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10

Plant, Pamela J. A role for the C2 domain of the ubiquitin-protein ligase Nedd4. 2000.

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11

Patton, E. Elizabeth. E3 ubiquitin protein ligase complexes that regulate G1-phase in budding yeast. 2001.

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12

(Editor), Leonidas Stefanis, and J. N. Keller (Editor), eds. The Proteasome in Neurodegeneration. Springer, 2006.

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13

Stefanis, Leonidas, and J. N. Keller. Proteasome in Neurodegeneration. Springer London, Limited, 2007.

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14

Atfield, Alexandre David. The E3 ubiquitin ligase Cbl-b is essential for the induction of in vivo T-cell anergy. 2005.

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15

Atfield, Alexandre David. The E3 ubiquitin ligase Cbl-b is essential for the induction of in vivo T-cell anergy. 2005.

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16

(Editor), Mario Di Napoli, and Cezary Wojcik (Editor), eds. The Ubiquitin Proteasome System in the Central Nervous System: From Physiology to Pathology. Nova Science Pub Inc, 2008.

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17

Barañano, Kristin W. Angelman Syndrome. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0055.

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Abstract:
Angelman syndrome (AS) is a severe neurodevelopmental disorder caused by maternal deficiency of the epigenetically imprinted gene UBE3A. It is characterized by severe developmental delay, an ataxic gait disorder, an apparent happy demeanor with frequent smiling or laughing, and severe expressive language impairments. Understanding the neurobiology of AS has focused on understanding how UBE3A is regulated by neuronal activity, as well as the targets of its ubiquitin E3 ligase activity. This has led to a model of the role of UBE3A in the regulation of experience-dependent sculpting of synaptic circuits. At this time, treatment is largely supportive, but efforts directed toward reversing the epigenetic silencing machinery may lead to improved synaptic function in AS patients.
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18

Bolanos-Garcia, Victor M., Julien Licchesi, Heike Laman, and Fumiyo Ikeda, eds. E3 Ubiquitin Ligases: From Structure to Physiology. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-883-3.

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19

Kavsak, Peter. Smurfs are E3 ubiquitin protein ligases that inhibit the transforming growth factor beta signalling pathway. 2001.

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20

Willems, Andrew R. Substrate recognition, structure/function analysis, and regulation of SCF ubiquitin protein ligases in buding yeast. 2002.

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