Relevant bibliographies by topics / UBAP2L

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  2. Dissertations / Theses
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Academic literature on the topic 'UBAP2L'

Author: Grafiati
Published: 23 September 2022
Last updated: 28 September 2022

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Journal articles on the topic "UBAP2L"

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Ye, Tao, Jing Xu, Ling Du, Wenhui Mo, Yiming Liang, and Jinglin Xia. "Downregulation of UBAP2L Inhibits the Epithelial-Mesenchymal Transition via SNAIL1 Regulation in Hepatocellular Carcinoma Cells." Cellular Physiology and Biochemistry 41, no. 4 (2017): 1584–95. http://dx.doi.org/10.1159/000470824.

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Background/Aims: Dysregulation of ubiquitin-associated protein 2-like (UBAP2L) has been reported in tumors, but its role in hepatocellular carcinoma (HCC) progression is unclear. Methods: The expression levels of UBAP2L in HCC tissues and HCC cell lines were detected by western blot and quantitative real-time (qRT) PCR. The effects of UBAP2L expression on HCC cell biological traits, including migration and invasion, were investigated by wound healing assay and matrigel transwell assay. Simultaneously, the expression of epithelial-mesenchymal transition (EMT) markers including E-cadherin, CK-18, N-cadherin, Vimentin, Claudin7 and the promoter activity of E-cadherin were detected by western blot and qRT-PCR. Subsequently, role of SNAIL1 in UBAP2L-mediated EMT and the mechanism underlying UBAP2L-mediated SNAIL1 expression were further investigated. Results: UBAP2L was overexpressed in human HCC tissues compared with peri-tumoral tissues. Downregulation of UBAP2L inhibited migration, invasion and the EMT in highly metastatic HCC cell lines. Furthermore, UBAP2L knockdown inhibited expression of the transcriptional repressor SNAIL1 and its ability to bind to the E-cadherin promoter via SMAD2 signaling pathway, which in turn resulted in increased E-cadherin expression. Additionally, bioinformatics analysis showed that expression of UBAP2L is correlated with poor prognosis in patients with HCC. Conclusions: UBAP2L plays a critical role in maintenance of the metastatic ability of HCC cells via SNAIL1 Regulation and is predictive of a poor clinical outcome.
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Bordeleau, Marie-Eve, Jalila Chagraoui, Romain Aucagne, Simon Girard, Éric Bonneil, Martin Sauvageau, Amélie Faubert, Pierre Thibault, and Guy Sauvageau. "Ubap2l-Bmi-1-Rnf2 Define a Novel Polycomb Complex Essential For Self-Renewal Of Hematopoietic Stem Cells." Blood 122, no. 21 (November 15, 2013): 736. http://dx.doi.org/10.1182/blood.v122.21.736.736.

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Abstract The polycomb group protein Bmi-1 is a well known determinant of hematopoietic stem cell function. Bmi-1-/- mice display severe hematopoietic defects, including progressive loss of hematopoietic cells from the bone marrow. Bmi-1 is dispensable for hematopoietic stem cell specification, but essential for their maintenance, an effect attributable to its ability to promote HSC self-renewal. The mechanism by which Bmi-1 regulates this process is not completely understood. Bmi-1 has been shown to repress the INK4A/ARF locus encoding the cell cycle inhibitors p16ink4a and p19arf , to interact with the E4F1 protein and to regulate the DNA damage response pathway, however experimental manipulation of these proteins/pathways only partially rescues the hematopoietic defects of the Bmi-1-/-mice. It thus appears that the mechanism by which Bmi-1 regulates HSC self-renewal remains to be determined. Towards this goal, we purified Bmi-1 containing protein complexes from cellular extracts and identified Bmi-1 interaction partners by mass spectrometry. We observed that the protein Ubap2l, which has never been shown to associate with Bmi-1 and for which no link with polycomb group protein function has been described, was consistently found in Bmi-1-containing protein complexes. Immunoprecipitation experiments revealed that Ubap2l indirectly associates with Bmi-1 via an interaction with the polycomb group protein Rnf2. We then evaluated the possibility that Ubap2l might be involved in the regulation of HSC activity. We observed that Ubap2l transcripts are more abundant in primitive HSC populations compared to total BM. CFC assays performed with BM cells infected with Ubap2l shRNAs revealed that Ubap2l knockdown causes a modest and progressive loss of progenitor activity when cells are kept in culture, with multipotent and bipotent progenitors being substantially more affected than unipotent progenitors. We transplanted these cells in mice and observed a gradual decrease in the percentage of donor derived cells expressing Ubap2l shRNAs in the peripheral blood of the recipient mice, with the most striking effect observed 16 weeks post-transplantation in the BM. Bmi-1 has been shown to regulate the proliferative capacity of both progenitor and stem cells, and its deletion in BM cells is known to dramatically reduce the reconstitution activity of these cells at early time points following transplantation. In contrast, Ubap2l appears to preferentially regulate LTR-HSC activity. We tested the effects of Ubap2l silencing on leukemic cells in vivo and observed that a reduction of Ubap2l levels in these cells had an important impact on their ability to reconstitute recipient mice, suggesting that Ubap2l also plays a role in leukemic stem cell activity. We determined if the mechanism by which Ubap2l regulates HSC activity is related to Bmi-1 function by simultaneously introducing Bmi-1 cDNA and Ubap2l shRNAs in BM cells and found that Bmi-1 is able to rescue the long-term reconstitution defect caused by Ubap2l downregulation in these cells. We observed that Ubap2l silencing does not significantly affect the expression of the known Bmi-1 targets p16ink4a and p19arf, implying that Ubap2l regulates HSC activity via a Bmi-1-dependent mechanism that does not involve repression of the INK4A/ARF locus. One explanation for the two Bmi-1 dependent mechanisms at play in the regulation of HSC activity could be that Bmi-1 is part of two separate protein complexes, each regulating different aspects of hematopoietic cell function. To test this hypothesis, we fractionated cellular extracts and were indeed able to resolve two distinct Bmi-1 containing protein complexes, distinguishable by the presence of Ubap2l. Based on the results we obtained, we propose a model in which two different Bmi-1 containing protein complexes regulate hematopoietic stem cell function. An Ubap2l-independent complex, which is most likely involved in the repression of the INK4A/ARF locus, and could be responsible for the effects of Bmi-1 on multipotent progenitors and STR-HSCs, and an Ubap2l-dependent complex, which operates via a yet to be defined mechanism unrelated to p16Ink4a and p19Arf, and would account for the effects of Bmi-1 on LTR-HSC activity. These results position Ubap2l as a key regulator of LTR-HSC activity and unveil a novel protein complex mediating the effects of Bmi-1 on LTR-HSCs. Disclosures: No relevant conflicts of interest to declare.
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Bordeleau, Marie-Eve, Romain Aucagne, Jalila Chagraoui, Simon Girard, Nadine Mayotte, Éric Bonneil, Pierre Thibault, et al. "UBAP2L is a novel BMI1-interacting protein essential for hematopoietic stem cell activity." Blood 124, no. 15 (October 9, 2014): 2362–69. http://dx.doi.org/10.1182/blood-2014-01-548651.

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Carlston, Colleen, Robin Weinmann, Natalia Stec, Simona Abbatemarco, Francoise Schwager, Jing Wang, Huiwu Ouyang, Collin Y. Ewald, Monica Gotta, and Christopher M. Hammell. "PQN-59 antagonizes microRNA-mediated repression during post-embryonic temporal patterning and modulates translation and stress granule formation in C. elegans." PLOS Genetics 17, no. 11 (November 22, 2021): e1009599. http://dx.doi.org/10.1371/journal.pgen.1009599.

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microRNAs (miRNAs) are potent regulators of gene expression that function in a variety of developmental and physiological processes by dampening the expression of their target genes at a post-transcriptional level. In many gene regulatory networks (GRNs), miRNAs function in a switch-like manner whereby their expression and activity elicit a transition from one stable pattern of gene expression to a distinct, equally stable pattern required to define a nascent cell fate. While the importance of miRNAs that function in this capacity are clear, we have less of an understanding of the cellular factors and mechanisms that ensure the robustness of this form of regulatory bistability. In a screen to identify suppressors of temporal patterning phenotypes that result from ineffective miRNA-mediated target repression, we identified pqn-59, an ortholog of human UBAP2L, as a novel factor that antagonizes the activities of multiple heterochronic miRNAs. Specifically, we find that depletion of pqn-59 can restore normal development in animals with reduced lin-4 and let-7-family miRNA activity. Importantly, inactivation of pqn-59 is not sufficient to bypass the requirement of these regulatory RNAs within the heterochronic GRN. The pqn-59 gene encodes an abundant, cytoplasmically-localized, unstructured protein that harbors three essential “prion-like” domains. These domains exhibit LLPS properties in vitro and normally function to limit PQN-59 diffusion in the cytoplasm in vivo. Like human UBAP2L, PQN-59’s localization becomes highly dynamic during stress conditions where it re-distributes to cytoplasmic stress granules and is important for their formation. Proteomic analysis of PQN-59 complexes from embryonic extracts indicates that PQN-59 and human UBAP2L interact with orthologous cellular components involved in RNA metabolism and promoting protein translation and that PQN-59 additionally interacts with proteins involved in transcription and intracellular transport. Finally, we demonstrate that pqn-59 depletion reduces protein translation and also results in the stabilization of several mature miRNAs (including those involved in temporal patterning). These data suggest that PQN-59 may ensure the bistability of some GRNs that require miRNA functions by promoting miRNA turnover and, like UBAP2L, enhancing protein translation.
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Huang, Chuyu, Yan Chen, Huaiqian Dai, Huan Zhang, Minyu Xie, Hanbin Zhang, Feilong Chen, Xiangjin Kang, Xiaochun Bai, and Zhenguo Chen. "UBAP2L arginine methylation by PRMT1 modulates stress granule assembly." Cell Death & Differentiation 27, no. 1 (May 21, 2019): 227–41. http://dx.doi.org/10.1038/s41418-019-0350-5.

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He, Jing, Yuanping Chen, Lu Cai, Zelei Li, and Xiaoqing Guo. "UBAP2L silencing inhibits cell proliferation and G2/M phase transition in breast cancer." Breast Cancer 25, no. 2 (December 1, 2017): 224–32. http://dx.doi.org/10.1007/s12282-017-0820-x.

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Cirillo, Luca, Adeline Cieren, Sofia Barbieri, Anthony Khong, Françoise Schwager, Roy Parker, and Monica Gotta. "UBAP2L Forms Distinct Cores that Act in Nucleating Stress Granules Upstream of G3BP1." Current Biology 30, no. 4 (February 2020): 698–707. http://dx.doi.org/10.1016/j.cub.2019.12.020.

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Huang, Rui, Qixia Yang, and Tiantian Wang. "Norcantharidin-blocked ANXA2P2 inhibits fibroblast proliferation by increasing UBAP2L mRNA stability through LIN28B." Life Sciences 279 (August 2021): 119645. http://dx.doi.org/10.1016/j.lfs.2021.119645.

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Lin, Sihan, Zhiyong Yan, Qiaofei Tang, and Shuang Zhang. "Ubiquitin-associated protein 2 like (UBAP2L) enhances growth and metastasis of gastric cancer cells." Bioengineered 12, no. 2 (November 25, 2021): 10232–45. http://dx.doi.org/10.1080/21655979.2021.1982308.

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Li, Qian, Wei Wang, Yu-Chen Hu, Tian-Tian Yin, and Jie He. "Knockdown of Ubiquitin Associated Protein 2-Like (UBAP2L) Inhibits Growth and Metastasis of Hepatocellular Carcinoma." Medical Science Monitor 24 (October 6, 2018): 7109–18. http://dx.doi.org/10.12659/msm.912861.

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Dissertations / Theses on the topic "UBAP2L"

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Liao, Yongrong. "Role of novel ubiquitin-related factors in cell cycle progression." Thesis, Strasbourg, 2021. http://www.theses.fr/2021STRAJ060.

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L'ubiquitylation est une modification post-traductionnelle qui joue de nombreuses fonctions importantes dans les cellules. Au cours de mon doctorat, je me suis concentré sur deux composants du système d'ubiquitine : l'enzyme de déubiquitination (DUB) ubiquitine carboxyl-terminal esterase L3 (UCHL3) et la protéine à domaine de liaison à l'ubiquitine (UBD) ubiquitin associated protein 2 like (UBAP2L, également connue sous le nom de NICE4). Mes études ont montré que l'UCHL3 est nécessaire au maintien de la forme nucléaire des cellules humaines et à la ségrégation des chromosomes. Au niveau moléculaire, UCHL3 interagit physiquement avec Aurora B et la déubiquitylise, régulant ainsi sa localisation au niveau des kinétochores et son interaction avec MCAK. Dans mon deuxième projet, j'ai identifié une nouvelle fonction d'UBAP2L dans la régulation des protéines liées au syndrome de retard mental du X fragile (FXRPs) et dans l'homéostasie des complexes de pores nucléaires (NPCs), qui, étonnamment, est indépendante de la liaison à l'ubiquitine d'UBAP2L. Au lieu de cela, j'ai découvert que les arginines dans le domaine arginine-glycine-glycine (RGG) de UBAP2L sont nécessaires pour l'interaction avec les FXRPs, et médient la fonction sur les FXRPs et les nucléoporines (Nups) au début de la phase G1
Ubiquitylation is a posttranslational modification which plays many important functions in cells. During my PhD study, I focused on two components of the ubiquitin system : the deubiquitinating enzyme (DUB) ubiquitin carboxyl-terminal esterase L3 (UCHL3) and the ubiquitin-binding domain (UBD) protein ubiquitin associated protein 2 like (UBAP2L, also known as NICE4). My studies showed that UCHL3 is necessary for maintaining proper nuclear shape of human cells and chromosome segregation. At the molecular level, UCHL3 physically interacts with and deubiquitylates Aurora B, thereby regulating its localization at the kinetochores and interaction with MCAK. In my second project, I identified a novel function of UBAP2L in the regulation of Fragile X mental retardation syndrome-related proteins (FXRPs) and in the nuclear pore complexes (NPCs) homeostasis, which surprisingly, is independent of UBAP2L ubiquitin-binding. Instead, I found that the arginines within the arginine–glycine–glycine (RGG) domain of UBAP2L are required for the interaction with FXRPs, and mediate the function on FXRPs and nucleoporins (Nups) in early G1 phase
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Stefani, Flavia. "Characterising the function of ubiquitin associated protein 1 (UBAP1)." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/characterising-the-function-of-ubiquitin-associated-protein-1-ubap1(cb81f935-7b1a-4b84-b806-fcd212fc1b84).html.

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Inactivating EGF signalling is key to modulating cell growth and avoiding cancer. To do this, the EGF receptor is ubiquitinated, internalized and sorted to lysosome for degradation. This latter process is coordinated by the endosomal sorting complex required for transport (ESCRT) machinery, a multi-complex protein machinery divided into four groups: ESCRT-0, I, II, III. ESCRTs recognise ubiquitinated cargoes and sort them from the limiting membrane of intermediate vesicles of maturing endosomes. In mammalian cells, the ESCRT machinery is also involved in other membrane related events, such as cytokinesis and viral budding. Certain ESCRTs, such as ESCRT-0, seem to be specifically required for cargo sorting to lysosomes, whereas other downstream ESCRTs, such as ESCRT-I, are required for all the cellular processes where the ESCRT machinery is involved. The existence of multiple variants of ESCRT-I components may suggest that ESCRT-I itself exists in different variants, each specific for a different membrane-based event. A bioinformatic study suggested Ubiquitin Associated Protein1 (UBAP1)as a novel variant of the ESCRT-I component MVB12. Moreover, a preliminary Y2H study identified UBAP1 as a potential binding partner of the ESCRT machinery regulator, HDPTP. This study aims to characterise UBAP1 as a variant MVB12 and a novel member of ESCRT-I. The results show that loss of UBAP1 impairs EGFR trafficking to lysosomes and causes the accumulation of ubiquitinated proteins on aberrant vacuolar structures. In cells, UBAP1 is incorporated in a complex with the ESCRT-I members TSG101, VPS28 and VPS37A. Importantly, UBAP1 uses three tandem ubiquitin associated (UBA) domains to bind ubiquitin and this activity is key for UBAP1 to function in cells. UBAP1 binds HDPTP via a peptide motif located about 100 aa. proximal to the tandem UBA domains. Altogether, the data shown in this thesis suggest that UBAP1 represents a subunit of an endosome-specific ESCRT-I complex, whose function may be coordinated by the ESCRT machinery regulator HDPTP.
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Chmielarska, Katarzyna. "Biochemical and cell biological characterisation of Sumo E1 activating enzyme Aos1-Uba2." Diss., [S.l.] : [s.n.], 2005. http://edoc.ub.uni-muenchen.de/archive/00006302.

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Moscoso, Blanco Milagros Yohanna. "Nivel de autocuidado y su relación con las complicaciones cardiovasculares del adulto hipertenso que asiste al consultorio de cardiología del policlínico municipal de Jesús María - UBAP - Es Salud - enero 2017." Bachelor's thesis, Universidad Nacional Mayor de San Marcos, 2017. https://hdl.handle.net/20.500.12672/6521.

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Determina si el nivel de autocuidado se relaciona con las complicaciones cardiovasculares del adulto hipertenso que asiste al consultorio de cardiología del Policlínico Municipal de Jesús María, UBAP - EsSalud. La muestra es de 59 adultos hipertensos. Encuentra que entre el nivel de autocuidado y las complicaciones cardiovasculares existe una relación indirecta y significativa al obtener un valor de -0.862; es decir a mejor nivel de autocuidado menor riesgo de complicaciones cardiovasculares. Así mismo, se logró comprobar que existe una relación alta de 86.2% entre ambas variables.
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Andia, Condori Noelia Corina. "Hallazgos radiológicos según clasificación BI-RADS en la primera mamografía de tamizaje en pacientes atendidas en el Servicio de Mamografía de la Unidad Básica de Atención Primaria UBAP Zárate - EsSalud 2016." Bachelor's thesis, Universidad Nacional Mayor de San Marcos, 2018. https://hdl.handle.net/20.500.12672/8192.

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Determina los hallazgos radiológicos según clasificación BI-RADS en la primera mamografía de tamizaje en pacientes atendidas en el servicio de mamografía del UBAP Zárate – EsSalud, de enero a setiembre del 2016. Realiza una investigación de tipo observacional y descriptiva. Es retrospectivo y de acuerdo al periodo y secuencia del estudio es transversal. Se utiliza como instrumento la hoja de recolección de datos para la recopilación de información proveniente de las fichas de factores de riesgo e informes mamográficos que cumplieron los criterios de selección. En el instrumento se registraron los datos ginecológicos y hallazgos radiológicos de las mamografías. Se estudiaron 183 fichas de factores de riesgo e informes mamográficos de pacientes que en su mayoría fueron adultas mayores de 50 años, con una edad media de 56. Presentaron menarquía entre los 12 y 15 años y el 71% se encontraba en la menopausia. Los hallazgos radiológicos con mayor frecuencia fueron las calcificaciones con el 56%, seguido de los nódulos representado por el 37%, la asimetría focal con el 6% y la distorsión de arquitectura presentó sólo el 1%. La categoría con mayor frecuencia fue el BI-RADS 2 con del 47%, seguido del BIRADS 1 con el 43%, el BI-RADS 3 presentó el 10% y para el BI-RADS 4 y BIRADS 5 el 0% de casos. Los nódulos y las calcificaciones en su mayoría fueron clasificados como BI-RADS 2, mientras que la distorsión de arquitectura y la asimetría focal en la totalidad pertenecieron a la categoría BI-RADS 3. Concluye que las calcificaciones pertenecientes a la clasificación BI-RADS 2 es el hallazgo radiológico con mayor frecuencia, en la primera mamografía de tamizaje en pacientes atendidas en el servicio de mamografía del UBAP Zárate – EsSalud durante los meses de enero a setiembre del 2016.
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Delgadillo, Penadillo Alan Cristopher. "Valor predictívo del diagnóstico ultrasonográfico en la detección de anomalías congénitas más frecuentes en gestantes de 11 a 14 semanas atendidas en la unidad básica de atención primaria de salud - Barranco (UBAP-ESSALUD) durante el periodo enero – marzo del año 2013." Bachelor's thesis, Universidad Nacional Mayor de San Marcos, 2015. https://hdl.handle.net/20.500.12672/4319.

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INTRODUCCIÓN: Las anomalías congénitas constituyen un capítulo cada vez más importante tanto para la obstetricia como para la neonatología, la genética y la biología; debido a su relativa alta frecuencia, a la posibilidad de su diagnóstico prenatal y a los enormes progresos que se están realizando para comprender mejor su epidemiología, fisiopatología y sobre todo a la posibilidad de diagnóstico cada vez más precoz y en ocasiones, tratarlas. OBJETIVOS: Determinar el valor predictivo del diagnóstico ultrasonográfico en la detección de anomalias congénitas más frecuentes en gestantes de 11 a 14 semanas atendidas en la Unidad Básica de Atención Primaria de Barranco (UBAP). Lima. Enero- Marzo, 2013. DISEÑO Y METODOS: El presente estudio es de tipo descriptivo, de corte transversal, retrospectivo y cuantitativo. Se seleccionó 113 historias clínicas de pacientes gestantes, se recolectó la información en un formato de recolección de datos, ordenando las variables según Edad, Paridad y resultados de exámenes ecográficos, y en algunos casos el resultado de la amniocentesis (Prueba de Oro) para la comparación posterior con los resultados ecográficos. Los datos fueron pasados al software SPSS v.21, se ordenaron y etiquetaron las variables para su posterior análisis. RESULTADOS: La edad promedio de pacientes es 31,5 años; el 50% tiene 31 años o más; la varianza es de 50,7; las distancias promedio de cada edad con respecto a la media es de 7,1; el marcador ecográfico más frecuente encontrado fue: translucencia nucal aumentada 2,7% y ausencia de hueso nasal 0,9%. CONCLUSIONES: La evaluación de anomalías congénitas por ultrasonografía debe considerarse un método fiable como diagnostico precoz ya que presenta un valor predictivo mayor al 90%. El valor predictivo positivo y el valor predictivo negativo de la ultrasonografía en contraste a los casos confirmados mediante la amniocentesis (prueba de oro) es 96% y 100% respectivamente; La Sensibilidad y especificidad de la ultrasonografía es del 80% y 100% respectivamente.
INTRODUCTION: Congenital anomalies constitute an increasingly important for both obstetrics and neonatology for the chapter, genetics and biology; because of its relatively high frequency, the possibility of prenatal diagnosis and the tremendous progress being made to better understand the epidemiology, pathophysiology and especially the possibility of increasing early diagnosis and sometimes treat them. OBJECTIVES: To determine the predictive value of ultrasound diagnosis in detecting most common congenital anomalies in pregnant women between 11 and 14 weeks served in the Basic Unit of Primary Care Barranco (UBAP). Lima. January-March, 2013. DESIGN AND METHODS: This study is descriptive, cross-sectional, retrospective and quantitative nature. 113 medical records of pregnant patients were selected, information was collected in a data collection form, ordering the variables as age, parity and results of ultrasound examinations, and in some cases the result of amniocentesis (Test de Oro) for the subsequent comparison with ultrasound results. Data were passed to SPSS v.21 software; they were sorted and labeled variables for further analysis. RESULTS: The mean age of patients was 31.5 years; 50% have 31 years or more; the variance is 50.7; the average distances of age with respect to each average is 7.1; the most frequent ultrasound marker was found: 2.7% increased nuchal translucency and nasal bone absence of 0.9%. CONCLUSIONS: The evaluation of congenital anomalies by ultrasound should be considered a reliable method as early diagnosis because it has a higher predictive value 90%. The positive predictive value and negative predictive value of ultrasonography in contrast to cases confirmed by amniocentesis (gold standard) is 96% and 100% respectively; the sensitivity and specificity of ultrasonography is 80% and 100% respectively. KEYWORDS: conventional ultrasound, sensitivity, specificity, positive predictive value, negative predictive value, congenital anomaly.
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Moutty, Marie Christine. "Mechanisms of the intracellular localization of the SUMO-activating enzyme Aos1/Uba2." Doctoral thesis, 2010. http://hdl.handle.net/11858/00-1735-0000-0006-B513-0.

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Chmielarska, Katarzyna [Verfasser]. "Biochemical and cell biological characterisation of Sumo E1 activating enzyme Aos1-Uba2 / Katarzyna Chmielarska." 2005. http://d-nb.info/982677634/34.

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Moutty, Marie Christine [Verfasser]. "Mechanisms of the intracellular localization of the SUMO-activating enzyme Aos1-Uba2 / submitted by Marie Christine Moutty." 2010. http://d-nb.info/1007525940/34.

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Books on the topic "UBAP2L"

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Raghav, Rangeya. Ubaal. Delhi: Kitabghar, 1988.

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Mauricio Ubal: Con la raíz a la intemperie. Montevideo: Cangrejo Solar, 2003.

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Book chapters on the topic "UBAP2L"

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Fernández-Ramírez, Fernando. "UBA2 (Ubiquitin-Like Modifier-Activating Enzyme 2)." In Encyclopedia of Signaling Molecules, 5813–17. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101918.

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Fernández-Ramírez, Fernando. "UBA2 (Ubiquitin-Like Modifier-Activating Enzyme 2)." In Encyclopedia of Signaling Molecules, 1–5. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101918-1.

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Vaishnavi Reddy, M., N. Sai Pooja Reddy, and J. V. R. Ravindra. "UBAPS: Inexact Unsigned Binary 5:2 Compressor Towards Power Efficient and High Speed for Three-Stage FIR Filter." In Lecture Notes in Networks and Systems, 607–17. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-7345-3_52.

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"UBA2." In Encyclopedia of Signaling Molecules, 5813. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_104055.

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SABRAS, Jean, Gilles PERAUDEAU, René BERJOAN, and Claude MONTY. "OXYGEN DIFFUSION, OXYGEN VACANCY CONCENTRATIONS AND CHEMICAL DIFFUSION IN Y1−uBa2−vCu3−wO7−δ." In High Tc Superconductor Materials, 239–46. Elsevier, 1990. http://dx.doi.org/10.1016/b978-0-444-88884-6.50032-8.

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Conference papers on the topic "UBAP2L"

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Malinovská, Kristína, L'udovít Malinovský, Pavel Krsek, Svatopluk Kraus, and Igor Farkaš. "UBAL." In CIIS 2019: 2019 The 2nd International Conference on Computational Intelligence and Intelligent Systems. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3372422.3372443.

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