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Relevant bibliographies by topics / Twardzik

Academic literature on the topic 'Twardzik'

Author: Grafiati

Published: 27 July 2024

Last updated: 28 July 2024

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Journal articles on the topic "Twardzik"

1

van Zoelen, E. J., W. J. van de Ven, H. J. Franssen, T. M. van Oostwaard, P. T. van der Saag, C. H. Heldin, and S. W. de Laat. "Neuroblastoma cells express c-sis and produce a transforming growth factor antigenically related to the platelet-derived growth factor." Molecular and Cellular Biology 5, no. 9 (September 1985): 2289–97. http://dx.doi.org/10.1128/mcb.5.9.2289-2297.1985.

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Mouse neuroblastoma Neuro-2A cells produce transforming growth factors during exponential growth in a defined hormone-free medium, which, on Bio-Gel columns in 1 M HAc, elute at a molecular size of 15 to 20 kilodaltons (kDa). These neuroblastoma-derived transforming growth factors have strong mitogenic activity, but they do not compete with epidermal growth factor for receptor binding (E. J. J. van Zoelen, D. R. Twardzik, T. M. J. van Oostwaard, P. T. van der Saag, S. W. de Laat, and G. J. Todaro, Proc. Natl. Acad. Sci. U.S.A. 81:4085-4089, 1984). In this study approximately 80% of the mitogenic activity was immunoprecipitated by antibodies raised against platelet-derived growth factor (PDGF). Immunoblotting indicated a true molecular size of 32 kDa for this PDGF-like growth factor. Analysis of poly(A)+ RNA from Neuro-2A cells demonstrated the expression of the c-sis oncogene in this cell line, whereas in vitro translation of the RNA yielded a 20-kDa protein recognized by anti-PDGF antibodies. Separation by reverse-phase high-pressure liquid chromatography demonstrated the presence of two distinct mitogenic activities in neuroblastoma-derived transforming growth factor preparations, one of which is antigenically related to PDGF. Both activities had the ability to induce anchorage-independent growth in normal rat kidney cells, both in the presence and in the absence of epidermal growth factor. It is concluded that Neuro-2A cells express c-sis with concomitant production and secretion of a PDGF-like growth factor, which plays a role in the induction of phenotypic transformation on normal rat kidney cells.

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2

van Zoelen, E. J., W. J. van de Ven, H. J. Franssen, T. M. van Oostwaard, P. T. van der Saag, C. H. Heldin, and S. W. de Laat. "Neuroblastoma cells express c-sis and produce a transforming growth factor antigenically related to the platelet-derived growth factor." Molecular and Cellular Biology 5, no. 9 (September 1985): 2289–97. http://dx.doi.org/10.1128/mcb.5.9.2289.

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Mouse neuroblastoma Neuro-2A cells produce transforming growth factors during exponential growth in a defined hormone-free medium, which, on Bio-Gel columns in 1 M HAc, elute at a molecular size of 15 to 20 kilodaltons (kDa). These neuroblastoma-derived transforming growth factors have strong mitogenic activity, but they do not compete with epidermal growth factor for receptor binding (E. J. J. van Zoelen, D. R. Twardzik, T. M. J. van Oostwaard, P. T. van der Saag, S. W. de Laat, and G. J. Todaro, Proc. Natl. Acad. Sci. U.S.A. 81:4085-4089, 1984). In this study approximately 80% of the mitogenic activity was immunoprecipitated by antibodies raised against platelet-derived growth factor (PDGF). Immunoblotting indicated a true molecular size of 32 kDa for this PDGF-like growth factor. Analysis of poly(A)+ RNA from Neuro-2A cells demonstrated the expression of the c-sis oncogene in this cell line, whereas in vitro translation of the RNA yielded a 20-kDa protein recognized by anti-PDGF antibodies. Separation by reverse-phase high-pressure liquid chromatography demonstrated the presence of two distinct mitogenic activities in neuroblastoma-derived transforming growth factor preparations, one of which is antigenically related to PDGF. Both activities had the ability to induce anchorage-independent growth in normal rat kidney cells, both in the presence and in the absence of epidermal growth factor. It is concluded that Neuro-2A cells express c-sis with concomitant production and secretion of a PDGF-like growth factor, which plays a role in the induction of phenotypic transformation on normal rat kidney cells.

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3

Schrack, Jennifer, and Elena Fazio. "EXPANDING INSIGHTS INTO LATE-LIFE DISABILITY USING OBJECTIVE ASSESSMENTS IN NHATS." Innovation in Aging 7, Supplement_1 (December 1, 2023): 165. http://dx.doi.org/10.1093/geroni/igad104.0541.

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Abstract The National Health and Aging Trends Study (NHATS) is a NIA-funded national panel study that conducts annual in-home interviews with a representative sample of US Medicare beneficiaries ages 65 years and older. Designed as a platform for the scientific study of late life disability trends and trajectories, NHATS fosters research to guide efforts to maximize health, promote independent functioning, and enhance quality of life at older ages. NHATS has collected performance-based measures of physical function since its inception (2011). In 2021 NHATS responded to expanded interest in objective measures of sensory loss and physical activity, by adding measures of vision (visual acuity, contrast sensitivity), hearing (pure tone audiometry), and physical activity (accelerometry). This symposium will introduce these measures and demonstrate their scientific application to answer key research questions related to physical and cognitive health. Dr. Schrack will present an overview of the accelerometry data collection procedures, as well as results by key demographic and health-related features. Dr. Twardzik will examine the association between features of participants’ social and physical environments and patterns of accelerometry-based physical activity. Dr. Martinez-Amezcua will examine the association between hearing and phenotypic frailty. Dr. Davoudi will present evidence on the links among sensory and motor function and risk of dementia. Finally, Dr. Wanigatunga will describe the association between measures of hand and leg strength and dementia incidence. Collectively, these presentations will highlight population-level associations of objective measures of health and function with physical and cognitive outcomes to advance the science of disability in older adults.

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4

Gentry, L. E., M. N. Lioubin, A. F. Purchio, and H. Marquardt. "Molecular events in the processing of recombinant type 1 pre-pro-transforming growth factor beta to the mature polypeptide." Molecular and Cellular Biology 8, no. 10 (October 1988): 4162–68. http://dx.doi.org/10.1128/mcb.8.10.4162-4168.1988.

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Recently, the simian type 1 transforming growth factor beta (TGF-beta 1) cDNA was expressed at high levels in Chinese hamster ovary (CHO) cells by dihydrofolate reductase-induced gene amplification (L.E. Gentry, N.R. Webb, G.J. Lim, A.M. Brunner, J.E. Ranchalis, D.R. Twardzik, M.N. Lioubin, H. Marquardt, and A.F. Purchio, Mol. Cell. Biol. 7:3418-3427, 1987). We have now purified and characterized the recombinant proteins released by these cells. Analyses of the precursor proteins by amino acid sequencing identified potentially important proteolytic processing sites. Signal peptide cleavage occurs at the Gly-29-Leu-30 peptide bond of pre-pro-TGF-beta 1, yielding pro-TGF-beta 1 (30 to 390). In addition, proteolytic processing of the precursor to yield mature TGF-beta 1 occurs at the dibasic cleavage site immediately preceding Ala-279, indicating that CHO cells possess the appropriate processing enzyme. Greater than 95% of the biological activity detected in the conditioned medium of the CHO transfectant was due to mature, properly processed growth factor. Highly purified recombinant TGF-beta 1 had the same specific biological activity as natural TGF-beta 1. The concentration of TGF-beta 1 required for half-maximal inhibition of Mv1Lu mink lung epithelial cell growth was approximately 1 to 2 pM. Purified precursor inhibited mink lung cell proliferation at 50 to 60 pM concentrations. The purified precursor preparation was shown to consist of pro-TGF-beta 1 (30 to 390), the pro region of the precursor (30 to 278), and mature TGF-beta 1 (279 to 390) interlinked by at least one disulfide bond with the pro portion of the precursor. These recombinant forms of TGF-beta1 should prove useful for further structural and functional studies.

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5

Gentry, L. E., M. N. Lioubin, A. F. Purchio, and H. Marquardt. "Molecular events in the processing of recombinant type 1 pre-pro-transforming growth factor beta to the mature polypeptide." Molecular and Cellular Biology 8, no. 10 (October 1988): 4162–68. http://dx.doi.org/10.1128/mcb.8.10.4162.

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Recently, the simian type 1 transforming growth factor beta (TGF-beta 1) cDNA was expressed at high levels in Chinese hamster ovary (CHO) cells by dihydrofolate reductase-induced gene amplification (L.E. Gentry, N.R. Webb, G.J. Lim, A.M. Brunner, J.E. Ranchalis, D.R. Twardzik, M.N. Lioubin, H. Marquardt, and A.F. Purchio, Mol. Cell. Biol. 7:3418-3427, 1987). We have now purified and characterized the recombinant proteins released by these cells. Analyses of the precursor proteins by amino acid sequencing identified potentially important proteolytic processing sites. Signal peptide cleavage occurs at the Gly-29-Leu-30 peptide bond of pre-pro-TGF-beta 1, yielding pro-TGF-beta 1 (30 to 390). In addition, proteolytic processing of the precursor to yield mature TGF-beta 1 occurs at the dibasic cleavage site immediately preceding Ala-279, indicating that CHO cells possess the appropriate processing enzyme. Greater than 95% of the biological activity detected in the conditioned medium of the CHO transfectant was due to mature, properly processed growth factor. Highly purified recombinant TGF-beta 1 had the same specific biological activity as natural TGF-beta 1. The concentration of TGF-beta 1 required for half-maximal inhibition of Mv1Lu mink lung epithelial cell growth was approximately 1 to 2 pM. Purified precursor inhibited mink lung cell proliferation at 50 to 60 pM concentrations. The purified precursor preparation was shown to consist of pro-TGF-beta 1 (30 to 390), the pro region of the precursor (30 to 278), and mature TGF-beta 1 (279 to 390) interlinked by at least one disulfide bond with the pro portion of the precursor. These recombinant forms of TGF-beta1 should prove useful for further structural and functional studies.

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6

Qamar Khan, Hania, Mohammad Adil, Syed Suhail Amin, Mohd Mohtashim, Roopal Bansal, and Fatima Tuz Zahra. "Bilateral morphea en coup de sabre: a rare presentation of linear morphea." Dermatology Review 108, no. 1 (2021): 59–66. http://dx.doi.org/10.5114/dr.2021.105896.

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7

Szkudlarek, Marcin. "Twardzina układowa – wielorakie zastosowania badania ultrasonograficznego Komentarz redakcyjny do artykułu G. Praconia, M. Płazy, M. Walentowskiej-Janowicz, I. Sudoł-Szopińskiej pt.: Wartość badania ultrasonograficznego w diagnostyce twardziny ograniczonej – opis przypadku klinicznego." Journal of Ultrasonography 16, no. 64 (February 20, 2016): 99–101. http://dx.doi.org/10.15557/jou.2016.0012.

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8

Peregud-Pogorzelska, Małgorzata, Beata Trzcińska-Budkiewicz, Jarosław Kaźmierczak, Maciej Wielusiński, Marek Zieliński, and Marek Brzosko. "Śluzak prawego przedsionka u pacjenta z twardziną układową." Folia Cardiologica 11, no. 6 (January 17, 2017): 563–66. http://dx.doi.org/10.5603/fc.2016.0116.

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9

Zoń-Giebel, Aleksandra, and Tomasz Czerw. "Fotofereza pozaustrojowa w leczeniu chorych na twardzinę układową." Forum Reumatologiczne 5, no. 1 (April 4, 2019): 27–32. http://dx.doi.org/10.5603/fr.2019.0004.

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10

Maciaszczyk, Katarzyna, Tomasz Durko, Elżbieta Waszczykowska, Bożena Bartkowiak-Dziankowska, Anna Pajor, Anna Erkiert-Polguj, and Magdalena Józefowicz-Korczyńska. "Stan układu przedsionkowego u pacjentów z twardziną układową." Otolaryngologia Polska 65, no. 3 (May 2011): 202–7. http://dx.doi.org/10.1016/s0030-6657(11)70676-x.

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Dissertations / Theses on the topic "Twardzik"

1

Twardziok, Monika [Verfasser]. "Mechanism of action of Viscum album L. extracts in Ewing sarcoma / Monika Twardziok." Berlin : Freie Universität Berlin, 2016. http://d-nb.info/1081367091/34.

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2

Twardziok, Sven [Verfasser]. "Die Piggeldat-Datenbank als Fundament zur Untersuchung der Auswirkungen von Nahrungsfaktoren bei Ferkeln / Sven Twardziok." Berlin : Freie Universität Berlin, 2015. http://d-nb.info/1075493668/34.

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3

Twardziok, Sven Olaf [Verfasser]. "Die Piggeldat-Datenbank als Fundament zur Untersuchung der Auswirkungen von Nahrungsfaktoren bei Ferkeln / Sven Twardziok." Berlin : Freie Universität Berlin, 2015. http://nbn-resolving.de/urn:nbn:de:kobv:188-fudissthesis000000099862-4.

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Books on the topic "Twardzik"

1

Bouncin' with Bartok: The incomplete works of Richard Twardzik. Toronto: Mercury Press, 2008.

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2

Chambers, Jack. Bouncin' With Bartok: The Incomplete Works Of Richard Twardzik (Jazz from Berkeley Hills). Berkeley Hills Books, 2006.

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Book chapters on the topic "Twardzik"

1

"Richard Twardzik’s Trio." In Essays on the Peripheries, 263–66. punctum books, 2021. http://dx.doi.org/10.2307/jj.2353947.30.

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