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Books on the topic 'Tumours'

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Published: 4 June 2021
Last updated: 29 July 2024

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1

Oosterhuis, Jendo A., ed. Ophthalmic Tumours. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-5520-2.

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2

Yalcin, Suayib, and Kjell Öberg, eds. Neuroendocrine Tumours. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-45215-8.

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3

Puri, Prem, ed. Neonatal Tumours. London: Springer London, 1996. http://dx.doi.org/10.1007/978-1-4471-3028-4.

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4

A, Fagin James, ed. Pituitary tumours. London: Baillière Tindall, 1995.

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5

J, Johnson P., ed. Liver tumours. London: Baillière Tindall, 1999.

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6

1931-, Williams Roger, and Johnson P. J, eds. Liver tumours. London: Baillière, 1987.

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7

Prem, Puri, and Surana Rajendra FRCSI, eds. Neonatal tumours. London: Springer, 1996.

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8

M, Polak Julia, and Bloom Stephen Robert, eds. Endocrine tumours: The pathobiology ofregulatory peptide-producing tumours. Edinburgh: Churchill Livingstone, 1985.

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9

Peter, Chambers, and BACUP, eds. Understanding brain tumours. London: BACUP, 1994.

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10

Thomas, David G. T., and David I. Graham, eds. Malignant Brain Tumours. London: Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-1877-0.

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11

Neal, David E., ed. Tumours in Urology. London: Springer London, 1994. http://dx.doi.org/10.1007/978-1-4471-2086-5.

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12

Master, Farokh J. Tumours and homoeopathy. New Delhi: Jain, 1997.

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13

1930-, Bleehen Norman M., and Royal College of Radiologists, eds. Tumours of thebrain. Berlin: Springer-Verlag, 1986.

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14

Donal, O'Shea, and Bloom Stephen Robert, eds. Gastrointestinal endocrine tumours. London: Baillière Tindall, 1996.

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15

Cotton, Dennis W. K. Spread of tumours. Sheffield: Audio Visual and TelevisionCentre, University of Sheffield, 1987.

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16

1939-, Jones E. L., and Young Jennifer A, eds. Tumours: Structure & diagnosis. London, England: Harvey Miller Publishers, 1991.

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17

T, Thomas D. G., and Graham David I, eds. Malignant brain tumours. London: Springer-Verlag, 1995.

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18

J, Emmett Anthony J., and O'Rourke Michael G. E, eds. Malignant skin tumours. 2nd ed. Edinburgh: Churchill Livingstone, 1991.

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19

1951-, Neal David E., ed. Tumours in urology. London: Springer-Verlag, 1994.

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20

M, Polak Julia, and Bloom Stephen Robert, eds. Endocrine tumours: The pathobiology of regulatory peptide-producing tumours. Edinburgh: Churchill Livingstone, 1985.

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21

Hatton, William James. Stereological envmeration of tumour associated macrophages infiltrating human colorectal tumours. [S.l: The Author], 1996.

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22

Glicenstein, Julien, Jacques Ohana, and Caroline Leclercq. Tumours of the Hand. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-71834-2.

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23

Harnden, Patricia, Johnathan K. Joffe, and William G. Jones, eds. Germ Cell Tumours V. London: Springer London, 2002. http://dx.doi.org/10.1007/978-1-4471-3281-3.

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24

Bleehen, Norman M., ed. Tumours of the Brain. London: Springer London, 1986. http://dx.doi.org/10.1007/978-1-4471-3437-4.

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25

Meyer-Breiting, Erhard, and Arne Burkhardt. Tumours of the Larynx. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-71100-8.

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26

Verley, J. M., and K. H. Hollmann. Tumours of the Mediastinum. Dordrecht: Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2994-7.

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27

Mazabraud, André. Pathology of bone tumours. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-95839-7.

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28

Ribatti, Domenico, and Enrico Crivellato. Mast Cells and Tumours. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1469-4.

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29

Chapman, Catherine Eluned. Radioimmunodetection of endocrine tumours. Birmingham: University of Birmingham, 1987.

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30

Warwick, David, Roderick Dunn, Erman Melikyan, and Jane Vadher. Tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199227235.003.0017.

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Types of tumour 574Benign tumours of bone and cartilage 575Ganglia 578Benign tumours of soft tissue 582Malignant tumours 584Malignant tumours of bone and cartilage 585Soft tissue sarcoma (STS) 586Metastatic tumours 590• Osteoid osteoma• Osteoblastoma• Chondroma (enchondroma, periosteal chondroma = ecchondroma)...
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31

Lamyman, Michael, and Roderick Dunn. Tumours. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757689.003.0017.

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Hand tumours occur in a spectrum from common benign conditions (such as ganglions) and rare, aggressive metastatic sarcoma. We present a clear account of benign and malignant soft tissue and bone tumours affecting the hand and upper limb, including diagnostic features, useful investigations, and classifications. Treatment plans are discussed, with some operative details for the different types of ganglia.
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32

Westmorland, Katelyn. Brain Tumours : Brain Tumor : Symptoms and Signs: Russell Watson Brain Tumour. Independently Published, 2021.

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33

Frink, Ashley. Brain Tumours : Brain Tumor : Symptoms and Signs: Russell Watson Brain Tumour. Independently Published, 2021.

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34

Laws, Edward R., Whitney W. Woodmansee, and Jay S. Loeffler. Pituitary tumours. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0018.

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Pituitary tumours are common, usually benign, lesions ordinarily well controlled by multidisciplinary management. The several subtypes of pituitary tumours reflect the hormones produced by the pituitary gland, and each may require a complex sequential programme of treatment. Modern laboratory evaluation and imaging is capable of extensively characterizing the tumours, and is the basis for the recommended therapies. The tumours that produce excess active levels of pituitary hormones may be amenable to very satisfactory medical therapy, which reduces hormone levels towards normal, and often causes shrinkage of the tumour. Surgical management is appropriate for tumours that are not producing excess hormones but by nature of their bulk can compress the optic nerves and cause visual loss. These tumours respond well to surgical management, which is usually done using the transnasal, transsphenoidal route of access. Patients with persistent or recurrent tumours and persistent hormonal excess can be effectively treated with modern techniques of radiation therapy. A multidisciplinary approach with specialists from different fields concentrating on the patient and the problem offers a comprehensive and effective solution for most patients with pituitary disorders.
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35

Archer, Nick, and Nicky Manning. Cardiac tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199230709.003.0018.

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Introduction 252Echogenic foci 254Rhabdomyoma 258Teratoma 260Fibroma 262Haemangioma 262Myxoma 262• Cardiac tumours are rare.• Identification requires subsequent monitoring for:• Fetal well-being.• Growth of the tumour.• Planning management of delivery and neonatal period.• It is important to consider the possibility of non-cardiac problems....
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36

WHO Classification of Tumours Editorial Board. Thoracic Tumours: WHO Classification of Tumours. World Health Organization, 2021.

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37

Paediatric Tumours: WHO Classification of Tumours. World Health Organization, 2023.

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38

Polak, Julia M. Endocrine Tumours: The Pathobiology of Regulatory Peptide-Producing Tumours (Current Problems in Tumour Pathology). Churchill Livingstone, 1985.

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39

Soffietti, Riccardo, Hugues Duffau, Glenn Bauman, and David Walker. Neuronal and mixed neuronal–glial tumours. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0008.

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Neuronal and mixed neuronal-glial tumours are rare tumours of the central nervous system that occur more commonly in children. Despite a generally benign course, most tumours cause medically intractable seizures, and have been denominated as ‘long-term epilepsy-associated tumours’. The World Health Organization classification distinguishes nine histological variants: dysplastic gangliocytoma of the cerebellum/Lhermitte–Duclos disease, desmoplastic infantile astrocytoma and ganglioglioma, dysembryoplastic neuroepithelial tumour, gangliocytoma and ganglioglioma, central neurocytoma and extraventricular neurocytoma, cerebellar liponeurocytoma, papillary glioneuronal tumour, rosette-forming glioneuronal tumour of the fourth ventricle, and spinal paraganglioma. Early surgery with complete resection may significantly improve the likelihood of postoperative epilepsy freedom. Conformal radiotherapy can be considered in case of patients with incompletely resected symptomatic tumours, atypical or high-grade tumours, or in the case of multiple recurrences despite resections. The role of chemotherapy in these lesions remains poorly defined, while targeted therapies are now available to impact some molecular alterations.
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40

Alapetite, Claire, Takaaki Yanagisawa, and Ryo Nishikawa. Germ cell tumours. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0014.

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Central nervous system germ cell tumours are mysterious tumours, which are common in young male adults in eastern Asia, and include germinoma, mature and immature teratoma, teratoma with malignant transformation, yolk sac tumour, embryonal carcinoma, choriocarcinoma, and mixed tumours of these components. The aetiological mechanism why they mostly develop in the pineal and neurohypophyseal region is still unknown. Their treatment is also a challenge; surgery is demanding, and sometimes biopsy would be preferred. Radiotherapy is effective, but its dose and field would be better reduced for young adults and children. Chemotherapy is effective but not enough for especially non-germinomatous tumours. This chapter presents a comprehensive discussion about those challenging diseases.
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41

Goepel, John. Pathology of testicular tumours. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0091.

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Tumours of the testis are uncommon and are usually germ cell tumours. They present most often as a scrotal mass in a young man, and are the most frequent malignant tumour in this age group. The incidence has risen over recent decades and is higher in Western Europe. A history of testicular maldescent is a significant risk factor. About 50% are pure seminoma; the remainder non-seminomas may have a single but more usually a mixed histology. Non-seminomas are all called teratoma in the British system. Metastasis readily occurs to paraaortic lymph nodes or the lungs, and some patients present with advanced metastatic disease. Radical orchidectomy is the usual treatment of the primary tumour, with chemotherapy for metastatic disease. There are other tumours that arise in the testis, which will require a different management strategy. Finally, there are tumours that arise in paratesticular tissue or the spermatic cord.
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42

Samyn, Marianne. Liver tumours. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759928.003.0066.

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The chapter on liver tumours covers the features and management of infantile haemangiomas, mesenchymal hamartoma, focal nodular hyperplasia, nodular regenerative hyperplasia, hepatoblastoma, and hepatocellular carcinoma.
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43

Archer, Nick, and Nicky Manning. Cardiac tumours. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198766520.003.0020.

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44

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Liver tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0060.

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Infantile haemangiomata 440Mesenchymal hamartoma 441Focal nodular hyperplasia (FNH) 441Nodular regenerative hyperplasia (NRH) 441Hepatoblastoma 442Hepatocellular carcinoma (HCC) 442Inflammatory pseudotumour 443Fibropolycystic liver disease 443Liver tumours in children are rare, accounting for 0.5–2% of all neoplasms in the paediatric age group....
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45

Keshav, Satish, and Alexandra Kent. Gastrointestinal tumours. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0204.

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Gastrointestinal (GI) tumours can affect any part of the GI tract, and colorectal cancer is the most common. Throughout the GI tract, chronic inflammation seems to promote the development of neoplasia: for example, chronic reflux oesophagitis is linked to oesophageal adenocarcinoma; chronic Helicobacter pylori infection is linked to gastric cancer; chronic pancreatitis is linked to pancreatic cancer; cirrhosis is linked to hepatocellular cancer; chronic biliary inflammation is linked to cholangiocarcinoma; untreated coeliac disease is linked to intestinal lymphoma; and chronic inflammatory bowel disease is linked to colorectal cancer. Symptoms depend on the location of the tumour, and occur as a result of local anatomical disruption, with consequent functional consequences and, less frequently, as a result of hormonal, metabolic, and immune effects. Weight loss is a common symptom seen in the gastroenterology outpatient clinic, given the high overall incidence of GI tumours. Often, the associated symptoms will direct the doctor to the site of a possible underlying cancer. Anaemia is another non-specific finding with a strong association with luminal cancers. Patients with anaemia with or without weight loss will normally undergo upper and lower GI investigations, usually via oesophagogastroduodenoscopy and colonoscopy (either CT or endoscopic colonoscopy). In tumours that are difficult to identify or assess the malignant potential, PET scanning can provide a large amount of information. PET scanning is a nuclear medicine scanning technique that utilizes 18F-fluorodeoxyglucose (FDG), which is taken up by metabolically active tissue. When combined with CT scanning it can provide information about both anatomical and metabolic activity. FDG is rapidly taken up by malignant tumours and, as a result, is often used for diagnosing, staging, and monitoring response in cancers.
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46

Elwell, Christine, and Kufre Sampson. Neurological tumours. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0237.

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Neurological tumours are categorized by the WHO as follows: neuroepithelial tumours (gliomas, oligodendrogliomas, ependymomas, pineal parenchymal tumours, medulloblastoma, neuronal and neuroglial tumours); cranial and paraspinal nerve tumours (schwannoma, neurofibromas); meningeal tumours (meningiomas); lymphomas; germ cell tumours (germinoma, teratoma); sellar region tumours (cranipharyngioma); and metastases. The tumours are classified according to grade. The WHO histological grading scheme used for astrocytomas is based on mitoses, nuclear pleomorphism, necrosis, and endothelial proliferation. WHO Grade I and Grade II tumours are low-grade tumours, and WHO Grade III and Grade IV tumours are high-grade tumours.
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47

Andrade, Maria João, Jadranka Separovic Hanzevacki, and Ricardo Ronderos. Cardiac tumours. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0052.

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Transthoracic and transoesophageal echocardiography represent the first-line diagnostic tools for imaging space-occupying lesions of the heart. Cardiac masses can be classified as tumours, thrombi, vegetations, iatrogenic material, or normal variants. Occasionally, extracardiac masses may compress the heart and create a mass effect. Cardiac masses may be suspected from the clinical presentation. This is the case in patients with an embolic event presumed to be of cardiac origin or in patients with infective endocarditis. Otherwise, a cardiac mass can be identified during the routine investigation of common, non-specific cardiac manifestations or as an incidental finding. In general, an integrated approach which correlates the patient’s clinical picture with the echocardiographic findings may reasonably predict the specific nature of encountered cardiac masses and, in the case of tumours, discriminate between primary versus secondary, and benign versus malignant. Furthermore, echocardiography alone or with complementary imaging modalities, can provide information to decide on the resectability of cardiac tumours and assist on planning the therapy and follow-up. Because several normal structures and variants may mimic pathological lesions, a thorough knowledge of potential sources of misinterpretation is crucial for a correct diagnosis. After surgical resection, histological investigation is mandatory to confirm the diagnosis.
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48

Ajithkumar, Thankamma, Ann Barrett, Helen Hatcher, and Natalie Cook. CNS tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235636.003.0005.

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Tumours of the CNS are characterized by a remarkable diversity of pathological type and behaviour. Although rare, they affect people of all ages and hence have a disproportionate influence on the number of years of life lost to cancer. With few exceptions there is a marked consistency of incidence worldwide. Some types of brain tumour are increasing in incidence and, after many years of therapeutic stagnation, there have recently been some important improvements in treatment and outcome. For both of these reasons the subdiscipline of neuro-oncology is of increasing relevance in cancer medicine....
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49

Ajithkumar, Thankamma, Ann Barrett, Helen Hatcher, and Natalie Cook. Thoracic tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235636.003.0006.

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Primary tracheal tumours represent <1% of all respiratory malignancies and can arise from the respiratory epithelium, salivary glands, and mesenchymal structures of the trachea. Most tumours in adults are benign while this is the case in only 30% of tumours in children. Primary tumours in adults are predominantly of adenoid cystic or squamous cell histology (Gaissert et al. 2006)....
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50

Ajithkumar, Thankamma, Ann Barrett, Helen Hatcher, and Natalie Cook. Endocrine tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235636.003.0014.

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Malignancies of the thyroid gland are the commonest endocrine malignancy but comprise <1% of cancer incidence overall (Coleman et al. 1999). The incidence is increasing slowly. The highest incidence is seen in North and Central America, and Australasia, with the lowest incidence in Africa.
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