Academic literature on the topic 'Tumoral Niche'
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Journal articles on the topic "Tumoral Niche"
Grassi, Elisa Stellaria, Viola Ghiandai, and Luca Persani. "Thyroid Cancer Stem-Like Cells: From Microenvironmental Niches to Therapeutic Strategies." Journal of Clinical Medicine 10, no. 7 (April 1, 2021): 1455. http://dx.doi.org/10.3390/jcm10071455.
Full textShah, Sumedh, Garima Yagnik, Alan Nguyen, Harsh Wadhwa, Jordan Spatz, Michael Safaee, Justin Cheng, and Manish Aghi. "TMIC-57. PRO-TUMORAL EFFECTS OF INTRA-TUMORAL NEUTROPHILS IN THE GLIOBLASTOMA MICROENVIRONMENT." Neuro-Oncology 21, Supplement_6 (November 2019): vi260. http://dx.doi.org/10.1093/neuonc/noz175.1091.
Full textJandial, Rahul, and Khairul I Ansari. "Peri-tumoral neural niche in brain metastasis from breast cancer." Integrative Cancer Science and Therapeutics 3, no. 4 (2016): 509. http://dx.doi.org/10.15761/icst.1000199.
Full textStöth, Manuel, Aida Freire Valls, Mingyi Chen, Sarah Hidding, Karl Knipper, Ying Shen, Johannes Klose, et al. "Splenectomy reduces lung metastases and tumoral and metastatic niche inflammation." International Journal of Cancer 145, no. 9 (November 2019): 2509–20. http://dx.doi.org/10.1002/ijc.32378.
Full textChung, Hyewon, Sang Wha Kim, and Seung Hyeok Seok. "Abstract B009: Tumoral activation of endothelium drives macrophages-mediated metastatic niche formation and promotes lung metastasis." Cancer Research 83, no. 2_Supplement_2 (January 15, 2023): B009. http://dx.doi.org/10.1158/1538-7445.metastasis22-b009.
Full textGarcia-Mazas, Carla, Noemi Csaba, and Marcos Garcia-Fuentes. "Biomaterials to suppress cancer stem cells and disrupt their tumoral niche." International Journal of Pharmaceutics 523, no. 2 (May 2017): 490–505. http://dx.doi.org/10.1016/j.ijpharm.2016.12.013.
Full textJansen, Caroline S., Nataliya Prokhnevska, Viraj A. Master, Martin G. Sanda, Jennifer W. Carlisle, Mehmet Asim Bilen, Maria Cardenas, et al. "An intra-tumoral niche maintains and differentiates stem-like CD8 T cells." Nature 576, no. 7787 (December 11, 2019): 465–70. http://dx.doi.org/10.1038/s41586-019-1836-5.
Full textMoffet, Joel, Oluwaseun Fatunla, James Whittle, Jones Jordan, Samuel Roberts-Thomson, Anna Pavenko, David Scoville, et al. "TMIC-36. SPATIAL ARCHITECTURE OF HIGH-GRADE GLIOMA REVEALS TUMOR HETEROGENEITY WITHIN DISTINCT DOMAINS." Neuro-Oncology 25, Supplement_5 (November 1, 2023): v286. http://dx.doi.org/10.1093/neuonc/noad179.1102.
Full textInfanger, David W., YouJin Cho, Brina S. Lopez, Sunish Mohanan, S. Chris Liu, Demirkan Gursel, John A. Boockvar, and Claudia Fischbach. "Glioblastoma Stem Cells Are Regulated by Interleukin-8 Signaling in a Tumoral Perivascular Niche." Cancer Research 73, no. 23 (October 11, 2013): 7079–89. http://dx.doi.org/10.1158/0008-5472.can-13-1355.
Full textXiang, Lisha, and Daniele Gilkes. "The Contribution of the Immune System in Bone Metastasis Pathogenesis." International Journal of Molecular Sciences 20, no. 4 (February 25, 2019): 999. http://dx.doi.org/10.3390/ijms20040999.
Full textDissertations / Theses on the topic "Tumoral Niche"
Grégoire, Murielle. "Polynucléaires neutrophiles, cellules stromales, lymphocytes B : interaction tripartite dans la niche des lymphomes B." Thesis, Rennes 1, 2014. http://www.theses.fr/2014REN1S156/document.
Full textFor long time, neutrophils have only been considered as cells involved in the innate immune response. More recently, in descriptive publications, neutrophils were found in the microenvironment of many solid cancers, hypothesizing that they could also play a role in tumorigenesis and cancer progression. These studies highlighted the prognostic value of their frequency, but few of them focused on the functional characterization of these cells in tumor growth. In many cancers, including germinal centre-derived B-cell lymphomas, tumor cells are dependent on their microenvironment to proliferate and survive. In this study, we focused on the role of neutrophils in the progression of B-cell lymphomas, and for the first time we demonstrated that neutrophils directly support the growth and survival of tumor Bcells. In addition, we highlighted the existence of bidirectional cooperation between neutrophils and stromal cells. In one hand stromal cells support the survival of neutrophils. On the other hand, neutrophils induce a lymphoid stroma phenotype which is well known to enhance their supportive effect on tumor cells. This study demonstrates that neutrophils are a significant component of the tumor microenvironment and may be considered as a potential therapeutic target for the treatment of B-cell lymphomas
Laviron, Marie. "Etude de la modulation des niches de macrophages au cours du développement tumoral et en réponse à la chimiothérapie." Electronic Thesis or Diss., Sorbonne université, 2021. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2021SORUS225.pdf.
Full textMacrophages represent the most abundant population within the tumor and their accumulation is associated with bad prognosis in most cancers. They form a group of sub-populations that vary in terms of origin, localization and function. Those characteristics, defining the interaction between the macrophage and its environment, constitute the macrophage niche. Macrophage niches have been described in different tissues at homeostasis but their evolution during tumor development remains to be elucidated. My thesis project aims at characterizing macrophage niches and focuses on two parts; the evolution of macrophage niches during tumor development; and the impact of chemotherapy on macrophage niches, and specifically the role of Treg on their polarization in this setting. Through those projects, we highlight that the heterogeneity of macrophages is directly linked to the diversity of tumor spatial niches. Macrophage polarization is dictated by signals derived from the niche. We also suggest that chemotherapy favors Treg and macrophage interactions, and that Treg depletion post-chemotherapy leads to the repolarization of macrophages towards a pro-inflammatory phenotype, associated with better tumor control. This work sheds light on the importance of the relationship between the macrophage and the tumor for the induction of its functions, and could identify specific populations to target to restore an efficient anti-tumor immune response
Gualtieri, Marco. "In vivo analysis and manipulation of an invasive brain tumour." Electronic Thesis or Diss., Sorbonne université, 2022. http://www.theses.fr/2022SORUS388.
Full textPrimary brain tumours are extremely aggressive, and often incurable. Interestingly, cells sharing many of the features of neural stem cells (NSCs) have been identified in several primary brain tumors. These cells are coined cancer stem cells (CSCs), and display self-renewal potential with illimited proliferation. Tumours strongly depend on a cellular microenvironment, which results in part from the remodelling of pre-existing populations, such as glial cells and blood vessels (the blood-brain barrier). The project uses the Drosophila Central Nervous System as an in vivo model to track brain tumors during the entire life of the host. The fly NSCs are a well-characterised stem cell model from which tumous can be generated during development, are CSC-driven and can survive and proliferate extensively during adulthood. In this system I can discriminate between different cell populations within the niche, and explore their behavior with respect to the CSCs during tumor growth. In particular I am interested in the mechanisms of cellular interactions happening in glial cells at the interface with the tumors. My results show that a sub-population of glial cells (cortex glia) undergoes apoptosis upon tumor growth, a mechanism that appears to promote tumor propagation. Interestingly, preventing cortex glia death leads to reduced tumor growth, suggesting that the tumour is at least in part required to eliminate glia to grow. In return, precocious killing of cortex glia favors tumor growth, pinpointing a reciprocal relationship between these two cell populations. Performing a transcriptional analysis of cortex glia during tumor growth has revealed multiple signalling pathways with a changing expression, and whose functional relevance is currently being assessed. In parallel, taking advantage of a published tumor transcriptome, I have selected several potential candidates mediating protein protein interactions at the interface between the tumour and the glia cells. Finally, I have also evaluated the role of known Rab proteins in the context of tumor development. This on-going work will shed light on how CNS tumors progress within and invade the healthy tissue
Deynoux, Margaux. "Incidence de l'hypoxie sur le métabolisme oxydatif des leucémies aiguës myéloïdes : établissement et caractérisation d'un modèle in vitro de niche leucémique." Thesis, Tours, 2019. http://www.theses.fr/2019TOUR3303.
Full textIn acute myeloid leukemia, a high level of ROS is known to favor blasts proliferation, whereas a low level promotes stem cells quiescence. The low oxygenation, or hypoxia, of the bone marrow niche could contribute to chemoresistance of AML cells by reducing the oxidative stress. Hypoxia-inducible factors (HIF) are involved in the control of the cell metabolism and antioxidant enzymes. HIFs inhibition leads to AML cells stress and death. The purpose of this work was to study a link between hypoxia, oxidative metabolism and chemoresistance in an in vitro model of leukemic cell culture. The acquisition of a hypoxic profile by hematopoietic stem cells (HSC) cultured with medullary mesenchymal stromal cells (MSC), has been shown. We hypothesized that AML cells may also acquire such profile in a coculture with human MSCs. To demonstrate that, we cultivated primary AML cells or the MV4-11 cell line on primary human MSCs or the HS-27a cell line. Like HSCs, we identified three leukemic populations according to their adhesion capacity to MSCs: in suspension, adherent to MSCs and embedded in MSCs. Embedded cells, the most adherent, have stronger CXCR4 expression compared to the others. They are also 2- to 7-fold more resistance to cytarabine. However, no change in the stem cell phenotype profile and in the clonogenic, repopulation or xenograft capacities, could be associated with the embedded cells compared to other populations. In contrast, embedded cells present a hypoxic profile, a weak proliferation with increased G0 phase, and lower ROS level that may rely on lower mitochondrial mass. This suggests that chemoresistance mainly relies on hypoxia or cell metabolism rather than a higher stem cell capacity. Furthermore, we have shown that acriflavine, a non-specific HIF inhibitor, could synergize with the cytarabine to eliminate embedded chemoresistant cells. Our results show that the MSC supernatant or a simple contact are not sufficient to induce metabolic change and resistance to cytarabine. We assume that hypoxia in the niche may modulate the oxidative metabolism and the chemoresistance by direct mechanisms and/or indirect ones through CXCR4 expression, a chemokine receptor shown to be involved in the regulation of the oxidative stress in HSC
Lim, Chetana. "Microenvironnement et angiogénèse : implications dans la stratégie onco-chirurgicale des métastases hépatiques synchrones des cancers colorectaux." Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCC018/document.
Full textAt the time of the diagnosis of colorectal cancer, nearly 25% of patients have synchronous liver metastases. When this tumor is asymptomatic, the question of surgical strategy (primary tumor first versus liver-first strategy) remains debated. Current recommendations are based on agreements of experts which are by themselves based on retrospective clinical studies. The study of the tumor microenvironment has taken in recent years a major place in the field of cancer research. It leads to new paradigm with a new conception of the metastatic process. It may be possible that the microenvironment of the metastatic sites can be modulated by the primary tumor to promote the formation of the pre-“metastatic niche”. This leads to promote the growth of cancer cells and increase the metastatic potential of primary tumor. By a multidisciplinary research including fundamental, translational and clinical approaches, we have shown that primary tumor first strategy could modulate tumor angiogenesis and liver metastatic process. It is associated with improved survival of patients and efficacy of the anti-angiogenic therapy
Dagdelen, Olcay. "Nicht-invasive Dignitätsbestimmung von HNO-Tumoren mittels Positronenemissionstomographie." Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-27595.
Full textVahedi, Nadine. "Früh- und Langzeitergebnisse nach Resektion von Lebermetastasen nicht-kolorektaler Tumoren." Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-100951.
Full textKoczuła, Katarzyna Malgorzata. "Real-time metabolic flux in chronic lymphocytic leukaemia cells adapting to the hypoxic niche." Thesis, University of Birmingham, 2015. http://etheses.bham.ac.uk//id/eprint/6067/.
Full textFahy, Lucine. "Etude des conséquences d’un environnement hypoxique sur le développement et la chimiorésistance des leucémies aiguës lymphoblastiques T (LAL-T)." Thesis, Sorbonne Paris Cité, 2019. https://theses.md.univ-paris-diderot.fr/FAHY_Lucine_2_complete_20190627.pdf.
Full textBackground: T cell acute lymphoblastic leukemia (T-ALL) is a malignant hematological disorder characterized by an increased proliferation of T lymphocyte precursors that are unable to complete their maturation. Treatments are effective in 90% of children T-ALL cases. However, children with relapse have a very poor prognosis. Resistance to chemotherapy is thus a major therapeutic challenge in the treatment of T-ALL that can be driven by interactions between leukemic cells and the microenvironment. Bone marrow microenvironment, a crucial site of propagation and maintenance of leukemia, is hypoxic with oxygen levels varying from 0.1 to 5%. As hypoxic solid tumors are more resistant to radiotherapy and chemotherapy, the aims of the study were to investigate to which extent oxygen levels impact on leukemia development and chemoresistance and to characterize the implicated molecular mechanisms. Methods: Mouse models and patients derived samples of T-ALL were cultured in low (1% and 3.5%) and high (21%) oxygen levels. Growth, apoptosis, cell cycle, metabolism and chemoresistance were measured as well as in vivo leukemia propagation using immune deficient mice as ways to define the impact of oxygen levels on T-ALL.Results: Results show that hypoxia inhibits the growth of T-ALL by slowing down cell cycle progression and decreasing metabolism, making them less sensitive to anti-leukemic drugs and preserving their ability to initiate leukemia in vivo after treatment. Knocking down (KD) HIF1α counteracted the effects observed in hypoxic T-ALL and restored their chemosensitivity. Interestingly activation of mTOR was diminished in hypoxic leukemic cells and HIF-1α KD restored mTOR activation in these low O2 conditions. Moreover, inhibiting mTOR in HIF1aKD T-ALL enhanced their chemoresistance, indicating a functional relationship between these two pathways.Conclusion: This work shows that hypoxic niches can play a protective role during the treatment of T-ALL through a HIF1α/mTOR molecular loop. Inhibition of HIF1α and/or activation of the mTOR pathway may help suppress the chemoresistance of T-ALL in hypoxic niches
Kobelt, Dennis. "Klinische Studie und experimentelle Untersuchungen zur nicht-viralen Gentherapie solider Tumoren." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2012. http://dx.doi.org/10.18452/16598.
Full textCancer is one leading causes of death worldwide. Gene therapy belongs to the promising options for treatment of malignant tumors. The non-viral gene therapy is known as safer alternative to the viral gene therapy. For non-viral gene transfer the vector and the transfer technology are of crucial importance. As part of this work a clinical trial was performed to assess efficiency and safety of the non-viral jet-injection. It was shown, that this technology can be used safely in a clinical setting. As a result of this clinical trial we concluded, that vector safety and especially efficiency need further improvements. Based on this optimized non-viral vectors (minicircle, MIDGE) were compared with each other and their respective parental plasmids. The MIDGE vector showed the highest transgene expression due to increased transcription. In preparation of a clinical trial the combined treatment of hTNF-alpha gene transfer and Vindesine chemotherapy was analyzed. Again, the MIDGE vector showed the highest transgene expression. This expression led to an increased cytotoxicity of Vindesine in vitro due to an elevated apoptosis signaling. Furthermore, these results could be assigned to an in vivo model. The increased hTNF-alpha expression after MIDGE vector jet-injection in combination with Vindesine led to a significant decrease in tumor growth. Detailed analysis of systemic vector distribution in the blood and organs as well as the preclinical toxicity evaluation showed the safety of the non-viral MIDGE vector. Initial experiments were performed to show further options for stable gene expression and combined gene transfer protocols using the MIDGE vector.
Books on the topic "Tumoral Niche"
Ich will nicht, dass ihr weint: Das Krebstagebuch der 16-jährigen Jenni. Augsburg: Weltbild, 2008.
Find full textBotta, Cirino, Marco Rossi, Niels Weinhold, and Niccolò Bolli, eds. Targeting the Microenvironment Niche in Solid Tumors. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-772-6.
Full textRemagen, W., B. Spiessl, Joachim Prein, and E. Uehlinger. Atlas der Tumoren des Gesichtsschädels: Odontogene und Nicht Odontogene Tumoren. Springer London, Limited, 2013.
Find full textGünther, Sonja. Unsichere Gespräche: Zur Interaktion Von Arzt, Patienten und Nicht-Menschlichen Akteuren in der Neuroonkologie. de Gruyter GmbH, Walter, 2017.
Find full textGünther, Sonja. Unsichere Gespräche: Zur Interaktion Von Arzt, Patienten und Nicht-Menschlichen Akteuren in der Neuroonkologie. de Gruyter GmbH, Walter, 2017.
Find full textGünther, Sonja. Unsichere Gespräche: Zur Interaktion Von Arzt, Patienten und Nicht-Menschlichen Akteuren in der Neuroonkologie. de Gruyter GmbH, Walter, 2017.
Find full textBook chapters on the topic "Tumoral Niche"
Bennewith, Kevin L., Janine T. Erler, and Amato J. Giaccia. "Premetastatic Niches." In Tumor Microenvironment, 161–82. Chichester, UK: John Wiley & Sons, Ltd, 2010. http://dx.doi.org/10.1002/9780470669891.ch8.
Full textKornhuber, B. "Nicht organgebundene Tumoren." In Therapie der Krankheiten des Kindesalters, 389–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-662-10467-5_10.
Full textMcCauley, Heather A., and Géraldine Guasch. "Serial Orthotopic Transplantation of Epithelial Tumors in Single-Cell Suspension." In Stem Cell Niche, 231–45. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-508-8_20.
Full textLöser, Christoph R. "Nicht-melanozytäre benigne Tumoren." In Hauterkrankungen in der Zahnmedizin, der Mund-Kiefer-Gesichtschirurgie, der Augenheilkunde und der Hals-Nasen-Ohrenheilkunde, 149–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 2023. http://dx.doi.org/10.1007/978-3-662-66733-0_21.
Full textShiozawa, Yusuke, Russell S. Taichman, and Evan T. Keller. "Detection and Isolation of Human Disseminated Tumor Cells in the Murine Bone Marrow Stem Cell Niche." In Stem Cell Niche, 207–15. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-508-8_18.
Full textSaw, Phei Er, and Erwei Song. "Pre-Metastatic Niche: Communication Between Local and Distal Onco-Spheres." In Tumor Ecosystem, 249–66. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-1183-7_11.
Full textZimmermann, Arthur. "Pathogenic Features of Liver Metastasis: Mechanisms Involving Platelets, Tumor Stroma, Epithelial-Mesenchymal Transition, and the Premetastatic Niche." In Tumors and Tumor-Like Lesions of the Hepatobiliary Tract, 1997–2017. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26956-6_113.
Full textZimmermann, Arthur. "Pathogenic Features of Liver Metastasis: Mechanisms Involving Platelets, Tumor Stroma, Epithelial-Mesenchymal Transition, and the Pre-metastatic Niche." In Tumors and Tumor-Like Lesions of the Hepatobiliary Tract, 1–21. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26587-2_113-1.
Full textLinn, Jennifer, Martin Wiesmann, and Hartmut Brückmann. "Tumoren und nicht neoplastische intrakranielle Raumforderungen." In Atlas Klinische Neuroradiologie des Gehirns, 193–309. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-540-89569-5_3.
Full textSchäfer, Karl-Ludwig, Raihanatou Diallo, Barbara Dockhorn-Dworniczak, and Christopher Poremba. "Molekulare Grundlagen organspezifischer Tumoren: Knochen- und Weichteilsarkome." In Molekularmedizinische Grundlagen von nicht-hereditären Tumorerkrankungen, 385–403. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56297-6_13.
Full textConference papers on the topic "Tumoral Niche"
Pasquier, Jennifer, Hamda Al Thawadi, Nadine Abu Kaoud, Pegah Ghiabi, Mahtab Maleki, Bella S. Guerrouahen, and Arash Rafii. "Abstract A74: Microparticles mediate cross-talk between tumoral and endothelial cells and promote the constitution of an angiocrine pro-metastatic niche through Arf6 up regulation." In Abstracts: AACR Special Conference on Advances in Ovarian Cancer Research: From Concept to Clinic; September 18-21, 2013; Miami, FL. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1078-0432.ovca13-a74.
Full textFilatova, A. A., L. A. Alexeeva, and N. L. Mironova. "EFFECT OF CIRCULATING CELL-FREE DNA ON THE INVASIVE POTENTIAL OF MURINE MELANOMA." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-382.
Full textSokolow, AJ, D. Schiltz, and U. von Fritschen. "Phylloides Tumoren - selten aber nicht harmlos." In Wissenschaftliche Abstracts zur 40. Jahrestagung der Deutschen Gesellschaft für Senologie e.V. (DGS) Interdisziplinär. Kommunikativ. Digital. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1730226.
Full textEnderling, Heiko, Lynn Hlatky, and Philip Hahnfeldt. "Abstract 4931: Cancer stem cells in solid tumors: Symmetric division, niche size, and invasive tumor morphology." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-4931.
Full textCroucher, Peter, Weng Hua Khoo, Ryan Chai, Alex Corr, James Smith, Qihao Ren, Paul Baldock, Michelle McDonald, Sheila Stewart, and Tri G. Phan. "Abstract IA015: Niche-dependent control of tumor cell dormancy." In Abstracts: AACR Virtual Special Conference: The Evolving Tumor Microenvironment in Cancer Progression: Mechanisms and Emerging Therapeutic Opportunities; in association with the Tumor Microenvironment (TME) Working Group; January 11-12, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.tme21-ia015.
Full textMin, Wang. "Abstract 129: AIP1 suppresses tumor metastasis by regulating tumor microenvironment and metastatic niche." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-129.
Full textCarpenter, Ryan, and Jungwoo Lee. "Abstract 3748: Implantable tumor attracting niche models to study disseminated tumor cell biology." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-3748.
Full textCarpenter, Ryan, and Jungwoo Lee. "Abstract 3748: Implantable tumor attracting niche models to study disseminated tumor cell biology." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-3748.
Full textZhartun, E. S., and D. B. Nizheharodava. "EXTRACELLULAR VESICLES AS A MECHANISM OF INTERCELLULAR COMMUNICATION IN TUMOR PATHOPHYSIOLOGY." In SAKHAROV READINGS 2022: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2022. http://dx.doi.org/10.46646/sakh-2022-2-125-128.
Full textWang, Qirui, Yanqing Anna Gong, and Yi Fan. "Abstract B44: Regulation of macrophage activation by vascular niche in glioblastoma." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; October 1-4, 2017; Boston, MA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/2326-6074.tumimm17-b44.
Full textReports on the topic "Tumoral Niche"
Hinch, Lindsay. Elucidating the Tumor Suppressive Role of SLITs in Maintaining the Basal Cell Niche. Fort Belvoir, VA: Defense Technical Information Center, July 2010. http://dx.doi.org/10.21236/ada537890.
Full textHinck, Lindsay. Elucidating the Tumor Suppressive Role of SLITs in Maintaining the Basal Cell Niche. Fort Belvoir, VA: Defense Technical Information Center, July 2009. http://dx.doi.org/10.21236/ada515794.
Full textHinck, Lindsay. Elucidating the Tumor-Suppressive Role of SLITs in Maintaining the Basal Cell Niche. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada561080.
Full textMarsden, Carolyn. Mesenchymal Stem Cells in the Bone Marrow Provide a Supportive Niche for Early Disseminated Breast Tumor-Initiating Cells. Fort Belvoir, VA: Defense Technical Information Center, April 2011. http://dx.doi.org/10.21236/ada552881.
Full textKapulnik, Yoram, and Donald A. Phillips. Isoflavonoid Regulation of Root Bacteria. United States Department of Agriculture, January 1996. http://dx.doi.org/10.32747/1996.7570561.bard.
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