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Relevant bibliographies by topics / Tumor-inflammation / Books

Books on the topic 'Tumor-inflammation'

To see the other types of publications on this topic, follow the link: Tumor-inflammation.

Author: Grafiati

Published: 1 April 2023

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1

S, Grewal Iqbal, ed. Therapeutic targets of the TNF superfamily. New York: Springer Science+Business Media, 2009.

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2

S, Grewal Iqbal, ed. Therapeutic targets of the TNF superfamily. New York: Springer Science+Business Media, 2009.

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3

International Conference on Molecular and Cellular Biology of IL-1, TNF, and Lipocortins in Inflammation and Differentiation (1989 Siena, Italy). Cytokines and lipocortins in inflammation and differentiation: Proceedings of the International Conference on Molecular and Cellular Biology of IL-1, TNF, and Lipocortins in Inflammation and Differentiation, held in Siena, Italy, October 22-25, 1989. Edited by Melli Marialuisa and Parente Luca. New York, NY: Wiley-Liss, 1990.

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4

Harald, Tschesche, ed. Proteinases in inflammation and tumor invasion: Review articles including those from an international conference, Bielefeld, Federal Republic of Germany, March 14-16, 1985. Berlin: De Gruyter, 1986.

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5

E, Sim, ed. Humoral factors. Oxford: IRL Press at Oxford University Press, 1993.

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6

Dougan, Michael Lawrence. Balancing oncogenic inflammation and anti-tumor immunity in the development of novel immune therapies for cancer. 2009.

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7

Grewal, Iqbal S. Therapeutic Targets of the TNF Superfamily. Springer London, Limited, 2009.

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8

Grewal, Iqbal S. Therapeutic Targets of the TNF Superfamily: Volume 647. Springer, 2010.

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9

Tschesche, Harald. Proteinases in Inflammation and Tumor Invasion: Review Articles Incl. Those from an Internat. Conference, Bielefeld, March 14 - 16 1985. De Gruyter, Inc., 1986.

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10

Tschesche, Harald. Proteinases in inflammation and tumor Invasion: Review articles incl. those from an internat. conference, Bielefeld, March 14 - 16 1985. De Gruyter, Inc., 2020.

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11

Tschesche, Harald. Proteinases in Inflammation and Tumor Invasion: Review Articles Including Those from an International Conference Bielefeld, Federal Republic of Germ. Walter De Gruyter Inc, 1986.

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12

(Editor), Marialuisa Melli, and Luca Parente (Editor), eds. Cytokines and Lipocortins in Inflammation and Differentiation: Proceedings of the International Conference on Molecular and Cellular Biology of Il-1, (Progress in Clinical & Biological Research). Wiley-Liss, 1990.

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13

Woolf, Eric C., and Adrienne C. Scheck. Ketogenic Diet as Adjunctive Therapy for Malignant Brain Cancer. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0013.

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Abstract:

Malignant brain tumors are devastating, and increased survival requires new therapeutic modalities. Metabolic dysregulation results in an increased need for glucose in tumor cells, suggesting that reduced tumor growth could be achieved with decreased glucose availability either through pharmacological means or use of a high-fat, low-carbohydrate ketogenic diet (KD). KD provides increased blood ketones to support energy needs of normal tissues and has been shown to reduce tumor growth, angiogenesis, inflammation, peritumoral edema, migration, and invasion. Furthermore, this diet can enhance the activity of radiation and chemotherapy in a mouse model of glioma, thus increasing survival. In vitro studies indicate that increasing ketones in the absence of glucose reduction can also inhibit cell growth and potentiate the effects of radiation. Thus, emerging data provide strong support for the use of KD in the treatment of malignant gliomas and thus far has led to a limited number of clinical trials.

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14

Epenetos, Agamemnon A., and Massimo Pignatelii. Cell Adhesion Molecules in Cancer and Inflammation (Cell Adhesion and Communication, Vol 1). CRC, 1995.

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15

Proteinases in Inflamation and Tumour Invasion. Walter de Gruyter & Co, 1986.

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16

Keum, NaNa, Mingyang Song, Edward L. Giovannucci, and A. Heather Eliassen. Obesity and Body Composition. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0020.

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In 2014, an estimated 1.9 billion adults worldwide were either overweight (BMI 25–29.9) or obese (BMI ≥30). The so-called obesity epidemic began in high-income, English-speaking countries in the early 1970s, but soon spread globally; more than one-third (38%) of all adults and 600,000 children under age five are overweight or obese, as are two-thirds (69%) of adults in the United States. Excessive body fat is a major cause of type 2 diabetes, hypertension, cardiovascular and liver disease, among other disorders, and has been designated a definite cause of at least fourteen cancer sites: breast (postmenopausal), colorectum, endometrium, esophagus (adenocarcinoma), gallbladder, kidney (renal cell), pancreas, gastric cardia, liver, ovary, prostate (advanced tumors), multiple myeloma, thyroid, and meningioma. Mechanisms by which adipose tissue are thought to promote tumor growth include the endocrine and metabolic effects of fat on sex hormones, growth factors, and inflammation, as well as local chemical or mechanical injury of gastrointestinal organs.

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