Log in

Relevant bibliographies by topics / Tumor-inflammation / Books

To see the other types of publications on this topic, follow the link: Tumor-inflammation.

Books on the topic 'Tumor-inflammation'

Author: Grafiati

Published: 1 April 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 16 books for your research on the topic 'Tumor-inflammation.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

S, Grewal Iqbal, ed. Therapeutic targets of the TNF superfamily. New York: Springer Science+Business Media, 2009.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

S, Grewal Iqbal, ed. Therapeutic targets of the TNF superfamily. New York: Springer Science+Business Media, 2009.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Marialuisa, Melli, and Parente Luca, eds. Cytokines and lipocortins in inflammation and differentiation: Proceedings of the International Conference on Molecular and Cellular Biology of IL-1, TNF, and Lipocortins in Inflammation and Differentiation, held in Siena, Italy, October 22-25, 1989. New York, NY: Wiley-Liss, 1990.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Harald, Tschesche, ed. Proteinases in inflammation and tumor invasion: Review articles including those from an international conference, Bielefeld, Federal Republic of Germany, March 14-16, 1985. Berlin: De Gruyter, 1986.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

E, Sim, ed. Humoral factors. Oxford: IRL Press at Oxford University Press, 1993.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Dougan, Michael Lawrence. Balancing oncogenic inflammation and anti-tumor immunity in the development of novel immune therapies for cancer. 2009.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

Grewal, Iqbal S. Therapeutic Targets of the TNF Superfamily. Springer London, Limited, 2009.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

Grewal, Iqbal S. Therapeutic Targets of the TNF Superfamily: Volume 647. Springer, 2010.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Tschesche, Harald. Proteinases in Inflammation and Tumor Invasion: Review Articles Incl. Those from an Internat. Conference, Bielefeld, March 14 - 16 1985. De Gruyter, Inc., 1986.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Tschesche, Harald. Proteinases in inflammation and tumor Invasion: Review articles incl. those from an internat. conference, Bielefeld, March 14 - 16 1985. De Gruyter, Inc., 2020.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

11

Tschesche, Harald. Proteinases in Inflammation and Tumor Invasion: Review Articles Including Those from an International Conference Bielefeld, Federal Republic of Germ. Walter De Gruyter Inc, 1986.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

12

(Editor), Marialuisa Melli, and Luca Parente (Editor), eds. Cytokines and Lipocortins in Inflammation and Differentiation: Proceedings of the International Conference on Molecular and Cellular Biology of Il-1, (Progress in Clinical & Biological Research). Wiley-Liss, 1990.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

13

Woolf, Eric C., and Adrienne C. Scheck. Ketogenic Diet as Adjunctive Therapy for Malignant Brain Cancer. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0013.

Full text

Abstract:

Malignant brain tumors are devastating, and increased survival requires new therapeutic modalities. Metabolic dysregulation results in an increased need for glucose in tumor cells, suggesting that reduced tumor growth could be achieved with decreased glucose availability either through pharmacological means or use of a high-fat, low-carbohydrate ketogenic diet (KD). KD provides increased blood ketones to support energy needs of normal tissues and has been shown to reduce tumor growth, angiogenesis, inflammation, peritumoral edema, migration, and invasion. Furthermore, this diet can enhance the activity of radiation and chemotherapy in a mouse model of glioma, thus increasing survival. In vitro studies indicate that increasing ketones in the absence of glucose reduction can also inhibit cell growth and potentiate the effects of radiation. Thus, emerging data provide strong support for the use of KD in the treatment of malignant gliomas and thus far has led to a limited number of clinical trials.

APA, Harvard, Vancouver, ISO, and other styles

14

Epenetos, Agamemnon A., and Massimo Pignatelii. Cell Adhesion Molecules in Cancer and Inflammation (Cell Adhesion and Communication, Vol 1). CRC, 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

15

Proteinases in Inflamation and Tumour Invasion. Walter de Gruyter & Co, 1986.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

16

Keum, NaNa, Mingyang Song, Edward L. Giovannucci, and A. Heather Eliassen. Obesity and Body Composition. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0020.

Full text

Abstract:

In 2014, an estimated 1.9 billion adults worldwide were either overweight (BMI 25–29.9) or obese (BMI ≥30). The so-called obesity epidemic began in high-income, English-speaking countries in the early 1970s, but soon spread globally; more than one-third (38%) of all adults and 600,000 children under age five are overweight or obese, as are two-thirds (69%) of adults in the United States. Excessive body fat is a major cause of type 2 diabetes, hypertension, cardiovascular and liver disease, among other disorders, and has been designated a definite cause of at least fourteen cancer sites: breast (postmenopausal), colorectum, endometrium, esophagus (adenocarcinoma), gallbladder, kidney (renal cell), pancreas, gastric cardia, liver, ovary, prostate (advanced tumors), multiple myeloma, thyroid, and meningioma. Mechanisms by which adipose tissue are thought to promote tumor growth include the endocrine and metabolic effects of fat on sex hormones, growth factors, and inflammation, as well as local chemical or mechanical injury of gastrointestinal organs.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!