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Books on the topic 'Tumor; Gene expression'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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1

Bar-Eli, Menashe, ed. Regulation of Gene Expression in the Tumor Environment. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-8341-9.

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2

Hellberg, Dan. Histological and serological tumor markers, and gene expression, and their clinical usefulness in cancers. Hauppauge, NY: Nova Science Publishers, 2009.

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3

Slack, Frank J. MicroRNAs in development and cancer. London: Imperial College Press, 2011.

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4

Moldoveanu, Andrei Ion. Effects of prolonged endurance exercise on the gene expression and plasma levels of interleukin-1 beta, interleukin-6, and tumor necrosis factor-alpha. Ottawa: National Library of Canada, 1999.

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5

Hendrix, Mary. Maspin. Georgetown, Tex: Landes Bioscience, 2001.

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6

Triton Biosciences-UCLA Symposium (1986 Steamboat Springs, Colo.). Growth factors, tumor promoters, and cancer genes: Proceedings of a Triton Biosciences-UCLA Symposium held in Steamboat Springs, Colorado, April 6-13, 1986. Edited by Colburn Nancy H, Moses Harold L, and Stanbridge Eric J. New York: Liss, 1988.

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7

H, Colburn Nancy, Moses Harold L, and Stanbridge Eric J, eds. Growth factors, tumor promoters and cancer genes: Proceedings of a Triton Biosciences UCLA Symposium held in Steamboat Springs, Colorado, April 6-13, 1986. New York: Liss, 1988.

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8

Anobile, Cristina J. Cytokine regulation of human leukocyte antigen DR[alpha] (HLA-DR[alpha]) gene expression in human tumour celllines. [s.l.]: typescript, 1996.

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9

Venables, Julian P. Alternative splicing in cancer. Trivandrum, Kerala, India: Transworld Research Network, 2006.

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10

Cancer systems biology. Boca Raton: CRC Press, 2010.

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11

L, Moses Harold, Lengyel Peter 1929-, Stiles Charles D, and Genentech Inc, eds. Growth inhibitory and cytotoxic polypeptides ; proceedings of a Genentech-Smith, Kline & French-Triton Biosciences-UCLA Symposium held in Keystone, Colorado, January 24-30, 1988. New York: A.R. Liss, 1989.

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12

Frank, Steven A. Dynamics of cancer: Incidence, inheritance, and evolution. Princeton, N.J: Princeton University Press, 2007.

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13

Michael, Botchan, Grodzicker Terri, and Sharp Phillip A, eds. DNA tumor viruses: Control of gene expression and replication. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1986.

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14

Said, Harun M., Adrian Staab, and Carsten Hagemann. Brain-Cancer Associated Tumor Marker Genes Expression Pattern in Humans. Nova Science Publishers, Incorporated, 2011.

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15

Grodzicker, Terri, and Michael Botchan. DNA Tumor Viruses: Control of Gene Expression and Replication (Cancer Cells, 4). Cold Spring Harbor Laboratory Pr, 1986.

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16

Chen, Wenbiao. Estrogen-related gene expression in normal and tumor target tissues: Syrian hamster renal tumorigenesis and kidney tumor. 1993.

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17

Hendrix, Mary. Maspin (Medical Intelligence Unit, Unit 32). Eurekah.com, 2001.

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18

Hendrix, Mary. Maspin (Medical Intelligence Unit, Unit 32). Landes Bioscience, 2002.

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19

(Editor), M. Yaniv, and J. Ghysdael (Editor), eds. Oncogenes as Transcriptional Regulators: Volume 1: Retroviral Oncogenes (Progress in Gene Expression). Birkhauser, 1997.

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20

DiCiommo, David P. A novel, DNA-based alphavirus gene expression system for rapid recombinant protein purification and virus-based gene delivery to retina and retinoblastoma tumor cells. 2002.

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21

P, Villarreal Luis, and ICN-UCI International Conference on Virology (1989 : Newport Beach, Calif.), eds. Common mechanisms of transformation by small DNA tumor viruses. Washington, DC: American Society for Microbiology, 1989.

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22

Menon, Deepa U. Autism and Intellectual Disabilities. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0053.

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PTEN (phosphatase and tensin homologue) on chromosome 10q23.3 is a tumor suppressor gene that encodes for a dual specificity phosphatase that regulates the phosphatidylinositol 3- kinase pathway and has an important role in brain development by affecting neuronal survival, neurite outgrowth, synaptic plasticity, and learning memory. Germline mutations of the PTEN gene have been implicated in a group of related tumor syndromes with autosomal dominant inheritance and variable expression and include the Cowden syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome, and Juvenile Polyposis syndrome. These syndromes are collectively called the PTEN hamartoma tumor syndromes (PHTS) because they have a predisposition to tumors and hamartomas. PTEN germ line mutations have also been recently linked to autism and macrocephaly and the prevalence of PTEN mutation in children with autism spectrum disorder, and macrocephaly is reported to range from 1.1% to 16.7%.

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23

Menashe, Bar-Eli, ed. Regulation of gene expression in the tumor environment: Regulation of melanoma progression by the microenvironment: the roles of PAR-1 and PAFR. Dordrecht: Springer, 2008.

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24

Mammoser, Aaron. Primary and Secondary Glioblastoma. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0127.

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Glioblastoma, formerly glioblastoma multiforme, is synonymous with WHO grade IV astrocytoma and is the most commonly diagnosed astrocytoma; it carries with it significant clinical, histologic, and molecular heterogeneity, with subtypes of the tumor and important new mutations associated with it characterized over the previous decade. Gene expression profiling has identified four tumor subgroups associated with specific mutational patterns, age of onset, and prognosis. The discovery of isocitrate dehydrogenase (IDH) mutations has led to further delineation between primary and secondary glioblastoma. Despite promising new investigational treatments, glioblastoma remains an incurable and fatal tumor.

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25

MicroRNAs in Breast Cancer. Nova Science Publishers, Incorporated, 2010.

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26

Gregory, Bock, Marsh Joan, and Ciba Foundation, eds. Genetic analysis of tumour suppression. Chichester: Wiley, 1989.

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27

Expression Profiling of Human Tumors: Diagnostic and Research Applications. Humana Press, 2003.

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28

Marc, Ladanyi, and Gerald William L, eds. Expression profiling of human tumors: Diagnostic and research applications. Totowa, N.J: Humana Press, 2003.

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29

Marc, Ladanyi, and Gerald William L, eds. Expression profiling of human tumors: Diagnostic and research applications. Totowa, N.J: Humana Press, 2003.

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30

Potter, M. Mechanisms in B-Cell Neoplasia 1994 (Current Topics in Microbiology and Immunology). Springer-Verlag, 1994.

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31

Mechanisms in B-cell neoplasia 1994. Berlin: Springer-Verlag, 1995.

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32

Michael, Potter, Melchers F. 1936-, National Cancer Institute (U.S.), and Workshop on Mechanisms in B-Cell Neoplasia (12th : 1994 : Bethesda, Maryland ), eds. Mechanisms in B-cell neoplasia 1994: [12th workshop, Bethesda, MD, April 18-20, 1994]. Berlin: Springer-Verlag, 1995.

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33

Potter, M., and Michael Potter. Mechanisms in B-Cell Neoplasia 1994. SPRINGER-VERLAG, 1995.

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34

Enrico, Mihich, Croce Carlo, and Pezcoller Symposium on the Biology of Tumors (1997 : 7, 19Rovereto, Trento, Italy), eds. The biology of tumors. New York: Plenum Press, 1998.

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35

Wang, Edwin. Cancer Systems Biology. Taylor & Francis Group, 2017.

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36

Gay, Steffen, and Michel Neidhart. Epigenetics. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0039.

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In higher eukaryotic organisms epigenetic modifications are crucial for proper chromatin folding and thereby proper regulation of gene expression. Epigenetics include DNA methylation, histone modifications, and microRNAs. First described in tumors, the involvement of aberrant epigenetic modifications has been reported also in other diseases, i.e. metabolic, psychiatric, inflammatory, and autoimmune. Deregulation of epigenetic mechanisms occurred in patients with rheumatoid arthritis, systemic lupus erythematosus, and scleroderma. Many questions remain: e.g. what is the cause of these epigenetic changes and how can we interfere in the pathological process? Here we discuss whether supplementation with methyl donors could represent a novel therapeutic concept for such diseases.

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37

(Editor), Klaus-Michael Debatin, and Simone Fulda (Editor), eds. Apoptosis and Cancer Therapy: From Cutting-edge Science to Novel Therapeutic Concepts. Wiley-VCH, 2006.

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38

M, Debatin K., and Fulda Simone, eds. Apoptosis and cancer therapy: From cutting-edge science to novel therapeutic concepts. Weinheim: Wiley-VCH, 2006.

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39

Vogelstein, Bert, and Kenneth W. Kinzler. The Genetic Basis of Human Cancer. 2nd ed. McGraw-Hill Professional, 2002.

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40

The Genetic Basis of Human Cancer. McGraw-Hill Professional, 2002.

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41

Merl, Dan, Joseph Lucas, Joseph Nevins, Haige Shen, and Mike West. Trans-study projection of genomic biomarkers in analysis of oncogene deregulation and breast cancer. Edited by Anthony O'Hagan and Mike West. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780198703174.013.6.

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This article focuses on the use of Bayesian concepts and methods in the trans-study projection of genomic biomarkers for the analysis of oncogene deregulation in breast cancer. The objective of the study is to determine the extent to which patterns of gene expression associated with experimentally induced oncogene pathway deregulation can be used to investigate oncogene pathway activity in real human cancers. This is often referred to as the in vitro to in vivo translation problem, which is addressed using Bayesian sparse factor regression analysis for model-based translation and refinement of in vitro generated signatures of oncogene pathway activity into the domain of human breast tumour tissue samples. The article first provides an overview of the role of oncogene pathway deregulation in human cancers before discussing the details of modelling and data analysis. It then considers the findings based on biological evaluation and Bayesian pathway annotation analysis.

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42

Growth inhibitory and cytotoxic polypeptides ; proceedings of a Genentech-Smith, Kline & French-Triton Biosciences-UCLA Symposium held in Keystone, Colorado, ... symposia on molecular and cellular biology). A.R. Liss, 1989.

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43

Dynamics of Cancer: Incidence, Inheritance, and Evolution (Princeton Series in Evolutionary Biology). Princeton University Press, 2007.

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44

Frank, Steven A. Dynamics of Cancer: Incidence, Inheritance, and Evolution (Princeton Series in Evolutionary Biology). Princeton University Press, 2007.

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45

Jacqueline, Boultwood, and Fidler Carrie, eds. Molecular analysis of cancer. Totowa, N.J: Humana Press, 2002.

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46

Parker, M. Growth Regulation by Nuclear Hormone Receptors (Cancer Surveys Series, Vol 14) (Cancer Surveys Series, Vol 14). Cold Spring Harbor Laboratory Pr, 1992.

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47

G, Parker Malcolm, ed. Growth regulation by nuclear hormone receptors. Plainview, N.Y: Cold Spring Harbor Laboratory Press, 1992.

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