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Relevant bibliographies by topics / Tumor; Gene expression

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Academic literature on the topic 'Tumor; Gene expression'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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Journal articles on the topic "Tumor; Gene expression"

1

Ruan, Xiaogang, Yingxin Li, Jiangeng Li, Daoxiong Gong, and Jinlian Wang. "Tumor-specific gene expression patterns with gene expression profiles." Science in China Series C 49, no. 3 (June 2006): 293–304. http://dx.doi.org/10.1007/s11427-006-0293-1.

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Peterson, Carsten, and Markus Ringnér. "Analyzing tumor gene expression profiles." Artificial Intelligence in Medicine 28, no. 1 (May 2003): 59–74. http://dx.doi.org/10.1016/s0933-3657(03)00035-6.

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Chen, Xin, Siu Tim Cheung, Samuel So, Sheung Tat Fan, Christopher Barry, John Higgins, Kin-Man Lai, et al. "Gene Expression Patterns in Human Liver Cancers." Molecular Biology of the Cell 13, no. 6 (June 2002): 1929–39. http://dx.doi.org/10.1091/mbc.02-02-0023.

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Hepatocellular carcinoma (HCC) is a leading cause of death worldwide. Using cDNA microarrays to characterize patterns of gene expression in HCC, we found consistent differences between the expression patterns in HCC compared with those seen in nontumor liver tissues. The expression patterns in HCC were also readily distinguished from those associated with tumors metastatic to liver. The global gene expression patterns intrinsic to each tumor were sufficiently distinctive that multiple tumor nodules from the same patient could usually be recognized and distinguished from all the others in the large sample set on the basis of their gene expression patterns alone. The distinctive gene expression patterns are characteristic of the tumors and not the patient; the expression programs seen in clonally independent tumor nodules in the same patient were no more similar than those in tumors from different patients. Moreover, clonally related tumor masses that showed distinct expression profiles were also distinguished by genotypic differences. Some features of the gene expression patterns were associated with specific phenotypic and genotypic characteristics of the tumors, including growth rate, vascular invasion, and p53 overexpression.

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Greiner, Johannes, Janine Müller, Jörg Ebmeyer, and Holger Sudhoff. "Gene expression profiling reveals expression of tumor-relevant." Journal of Laryngology & Otology 130, S3 (May 2016): S114. http://dx.doi.org/10.1017/s0022215116004126.

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Lichtor, Terry, George J. Dohrmann, and Mark E. Gurney. "Cytokine Gene Expression by Human Gliomas." Neurosurgery 26, no. 5 (May 1, 1990): 788–93. http://dx.doi.org/10.1227/00006123-199005000-00009.

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Abstract Two glioma tumor lines and specimens from five patients with gliomas were analyzed to determine genic expression of four growth factors found in human brain. Messenger RNA encoding for interleukin-1β, interleukin-6, and basic fibroblast growth factor was found to be expressed in significant amounts in some of these tumors, while mRNA for interleukin-3 was found in small quantities in only the tumor lines. Multiple species of mRNA for basic fibroblast growth factor were found. Expression of growth factor genes may play a role in the growth of human gliomas.

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Granzow, M., D. Berrar, W. Dubitzky, A. Schuster, F. J. Azuaje, and R. Eils. "Tumor classification by gene expression profiling." ACM SIGBIO Newsletter 21, no. 1 (April 2001): 16–22. http://dx.doi.org/10.1145/381371.381384.

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Marx, J. "TUMOR ANGIOGENESIS: Gene Expression Patterns Identified." Science 289, no. 5482 (August 18, 2000): 1121a—1122. http://dx.doi.org/10.1126/science.289.5482.1121a.

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Iwamoto, Akemi, Masahide Ikeguchi, Sachico Matsumoto, Youji Hukumoto, Masashi Inoue, Tomohiro Ozaki, Masayuki Ataka, et al. "Tumor Cyclooxygenase-2 Gene Suppresses Local Immune Responses in Patients with Hepatocellular Carcinoma." Tumori Journal 92, no. 2 (March 2006): 130–33. http://dx.doi.org/10.1177/030089160609200208.

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Aims and Background In several neoplastic diseases including hepatocellular carcinoma (HCC) immunosuppression is correlated with disease stage, progression and outcome. Moreover, recent studies have demonstrated that cyclooxygenase-2 (COX-2) enhances tumor growth in HCCs. The present study analyzed the correlation between local immune responses and COX-2 gene expression levels in patients with primary HCCs. Methods Fresh tissues were obtained from 59 patients who underwent resection of an HCC. The COX-2 gene expression levels were quantified by real-time reverse transcriptase-polymerase chain reaction (RT-PCR) and compared with the CD8+ T cell densities detected by immunohistochemistry. Results COX-2 gene expression was detected in 35 of the 59 tumors. The CD8+ T cell density in COX-2-expressing tumors (6.1 cells/high-power field (HPF), x200 magnification) was suppressed compared with that in non-COX-2-expressing tumors (13.6 cells/HPF, P = 0.009). Tumor COX-2 gene expression was associated with a poorer disease-free survival rate. Conclusions Elevation of the tumor COX-2 level is correlated with the suppression of local immune responses in HCCs, suggesting that COX-2 plays a role in early tumor recurrence in the residual liver in patients after HCC resection.

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Xiong, Momiao, Wuju Li, Jinying Zhao, Li Jin, and Eric Boerwinkle. "Feature (Gene) Selection in Gene Expression-Based Tumor Classification." Molecular Genetics and Metabolism 73, no. 3 (July 2001): 239–47. http://dx.doi.org/10.1006/mgme.2001.3193.

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Bakary, Nermeen. "Calcitriol Revises Aromatase Gene Expression in Ehrlich Solid Tumor Bearing Mice Exposed to Low Dose Gamma Radiation." Cancer Research and Cellular Therapeutics 5, no. 1 (February 12, 2021): 01–09. http://dx.doi.org/10.31579/2640-1053/074.

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Background: Dysregulation of aromatase expression had been monitored in many types of cancer. Our study aimed to evaluate the possible role of calcitriol (Cal; Vit D3-OH) or/and low dose of gamma radiation in regulation of aromatase gene expression and the regression of tumor proliferation in murine model (EST; Ehrlich solid tumor bearing mice). Methods: Mice with ≈1 cm3 EST were received (i.p. injection) day after day repeated doses of Calcitriol (Cal) (0.05µg/mouse) for 14day or/and exposed to 0.5 Gy gamma radiation (low dose) delivered as one shot at dose rate 0.48 Gy/min. Results: Our results demonstrated that, mRNA expression of aromatase, levels of cyclooxygenase (COX2) and prostaglandin (PGE2) in addition to volume of the tumor are significantly decreased while caspase 3 level is significantly increased in EST mice treated with Cal or/and exposed to 0.5 Gy gamma ray compared to untreated EST bearing mice. However, the most pronounced improvements in all of the measured parameters were obviously indicated in EST mice group treated with Cal and exposed to gamma radiation. This was accomplished by suppression of inflammatory markers which cause down regulation in aromatase mRNA expression as well as augmenting apoptosis by inducing Caspase3 concentration. Conclusion: It could be concluded that the exposure to low dose gamma radiation potentiate the action of Calcitriol against tumor growth in the subjected murine model which represent a prospective policy for the management of solid tumor and decreasing the possibilities of tumor drug resistance.

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Dissertations / Theses on the topic "Tumor; Gene expression"

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Beijnum, Judith Rosina van. "Gene expression profiling of tumor angiogenesis." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 2006. http://arno.unimaas.nl/show.cgi?fid=7710.

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Tan, Ern Yu. "Loss of protein folding gene expression in human tumors." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670106.

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Petty, Aaron. "Novel MIG-7 expression increases tumor cell invasion and tumor progression." Online access for everyone, 2008. http://www.dissertations.wsu.edu/Thesis/Spring2008/a_petty_040908.pdf.

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Askew, David. "Changes in macrophage functions and gene expression during tumor growth." Diss., This resource online, 1993. http://scholar.lib.vt.edu/theses/available/etd-05042006-164512/.

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Wykoff, Charles C. "The hypoxia-regulated transcriptome and its expression in cancer." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365288.

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Wang, Fuli. "Identification of tumor suppressor genes using the approach of gene inactivation test /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-798-7/.

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Clark, Aaron J. "The Expression and Function of Wilms' Tumor 1 in Malignant Glioma." VCU Scholars Compass, 2006. http://hdl.handle.net/10156/1665.

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Yamaga, Yuichi. "Gene expression profile of Dclk1+ cells in intestinal tumors." Kyoto University, 2019. http://hdl.handle.net/2433/236595.

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Karlgren, Maria. "Novel extrahepatic P450 enzymes with emphasis on the tumor specific CYP2W1 /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-139-5/.

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Kiss, Nimrod G. B. "DNA methylation and gene expression patterns in adrenal medullary tumors." Stockholm : Department of Molecular Medicin and Surgery, Karolinska Institutet, 2009. http://diss.kib.ki.se/2009/978-91-7409-750-4/.

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Books on the topic "Tumor; Gene expression"

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Bar-Eli, Menashe, ed. Regulation of Gene Expression in the Tumor Environment. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-8341-9.

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Hellberg, Dan. Histological and serological tumor markers, and gene expression, and their clinical usefulness in cancers. Hauppauge, NY: Nova Science Publishers, 2009.

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Slack, Frank J. MicroRNAs in development and cancer. London: Imperial College Press, 2011.

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Moldoveanu, Andrei Ion. Effects of prolonged endurance exercise on the gene expression and plasma levels of interleukin-1 beta, interleukin-6, and tumor necrosis factor-alpha. Ottawa: National Library of Canada, 1999.

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Hendrix, Mary. Maspin. Georgetown, Tex: Landes Bioscience, 2001.

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Triton Biosciences-UCLA Symposium (1986 Steamboat Springs, Colo.). Growth factors, tumor promoters, and cancer genes: Proceedings of a Triton Biosciences-UCLA Symposium held in Steamboat Springs, Colorado, April 6-13, 1986. Edited by Colburn Nancy H, Moses Harold L, and Stanbridge Eric J. New York: Liss, 1988.

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H, Colburn Nancy, Moses Harold L, and Stanbridge Eric J, eds. Growth factors, tumor promoters and cancer genes: Proceedings of a Triton Biosciences UCLA Symposium held in Steamboat Springs, Colorado, April 6-13, 1986. New York: Liss, 1988.

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Anobile, Cristina J. Cytokine regulation of human leukocyte antigen DR[alpha] (HLA-DR[alpha]) gene expression in human tumour celllines. [s.l.]: typescript, 1996.

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Venables, Julian P. Alternative splicing in cancer. Trivandrum, Kerala, India: Transworld Research Network, 2006.

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10

Cancer systems biology. Boca Raton: CRC Press, 2010.

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Book chapters on the topic "Tumor; Gene expression"

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Razorenova, Olga V., and Amato J. Giaccia. "Hypoxia, Gene Expression, and Metastasis." In The Tumor Microenvironment, 43–58. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6615-5_3.

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Benz, Edward J., Katherine A. High, Karen Lomax, Catherine Stolle, Thomas A. Rado, Jay W. Schneider, and Robert W. Mercer. "Studies of Gene Expression During Granulocyte Maturation." In Tumor Cell Differentiation, 79–103. Totowa, NJ: Humana Press, 1987. http://dx.doi.org/10.1007/978-1-4612-4594-0_6.

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Komatsu, Yoji, Koji Tsuboi, Yoshihiko Yoshii, and Tadao Nose. "The Correlation Between flg Gene Expression and the Progression of Glioma." In Brain Tumor, 203–9. Tokyo: Springer Japan, 1996. http://dx.doi.org/10.1007/978-4-431-66887-9_20.

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Steeg, P. S., G. Bevilacqua, A. M. Rosengard, M. E. Sobel, V. Cioce, and L. A. Liotta. "Altered Gene Expression in Tumor Metastasis: The nm23 Gene." In Cancer Metastasis, 48–52. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74236-1_7.

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Freije, J. M. P., N. J. MacDonald, and P. S. Steeg. "Differential Gene Expression in Tumor Metastasis: Nm23." In Current Topics in Microbiology and Immunology, 215–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-61109-4_10.

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Aitken, Stuart, and Ruthrothaselvi Bharathavikru. "Bioinformatic Analysis of Next-Generation Sequencing Data to Identify WT1-Associated Differential Gene and Isoform Expression." In The Wilms' Tumor (WT1) Gene, 211–19. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-4023-3_18.

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Yamasaki, Toshiki, Kouzo Moritake, Yasuhiko Akiyama, Masako Fukuda, and Seiichi Nagao. "Experimental Analysis of Proto-Oncogene and Histocompatibility Antigen Gene Expression During Brain Tumor Progression." In Brain Tumor, 315–25. Tokyo: Springer Japan, 1996. http://dx.doi.org/10.1007/978-4-431-66887-9_33.

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Höfler, Heinz, Katja Specht, and Karl-Friedrich Becker. "Molecular Analysis of Gene Expression in Tumor Pathology." In Advances in Experimental Medicine and Biology, 19–26. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0081-0_3.

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Wang, Shulin, Ji Wang, Huowang Chen, and Boyun Zhang. "SVM-Based Tumor Classification with Gene Expression Data." In Advanced Data Mining and Applications, 864–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/11811305_94.

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Iozzo, R. V., and I. Cohen. "Altered proteoglycan gene expression and the tumor stroma." In Proteoglycans, 199–214. Basel: Birkhäuser Basel, 1994. http://dx.doi.org/10.1007/978-3-0348-7545-5_12.

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Conference papers on the topic "Tumor; Gene expression"

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Laug, Walter E. "HETEROGENOUS EXPRESSION OF PLASMINOGEN ACTIVATOR (PA) GENES IN THE HUMAN SARCOMA CELL LINE HT1080." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644395.

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Tumor cell derived PA activities are of crucial importance for tissue invasion and destruction by tumor cells. Therefore, we studied the expression of the PA genes in HT1080 cells using immunoenzymatic methods and specific PA gene probes.Immunenzymatic methods allowed only for the detection of urokinase like PA (u-PA) activities in HT1080 cells which was suppressed by treatment of the cells with dexamethasone (10-7 m). Despite the lack of u-PA activities, the cells still degraded extracellular tissue glycoproteins. Northern blot analysis with specific PA gene probe showed that HT1080 cells express both tissue type PA (t-PA) and u-PA. The enzymatic activities of t-PA were most likely masked by the simultaneous production of inhibitors of PA (PAI). Treatment of HT1080 cells with dexamethasone resulted in increased transcription of t-PA and decreased expression of the u-PA gene, explaining the unchanged tissue destruction by dexamethasone treated HT1080 cells.Cell clones secreting either large or small amounts of enzymatic PA activities were isolated from the parental HT1080 cell line using a fibrin agarose overlay technique.The expression of the u-PA gene was enhanced in high secreting PA clones compared to low secreting PA clones when analyzed on Northern blots. This heterogenous expression of the u-PA gene within the HT1080 cell line was confirmed by in situ hybridization with a specific u-PA gene probe.These findings demonstrate that PA gene expression can be missed with immunenzymatic methods due to simultaneous production of inhibitors of PA. In addition our results show that the expression of a given PA gene may be heterogenous on the cellular level within an established tumor cell line. These findings, therefore, suggest cellular variation of PA gene expression in tumor which may be of fundamental importance for tissue invasion and metastasis by cancer cells.

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Buchanan, Cara F., Elizabeth Voigt, Christopher S. Szot, Joseph W. Freeman, Pavlos P. Vlachos, and Marissa Nichole Rylander. "Shear Stress Mediates Angiogenic Gene Expression in a Microfluidic Tumor Vascular Model." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80286.

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While research has shown that the fluid mechanics of the tumor vasculature reduce transport and uptake of therapeutics, the underlying role of these stresses in regulating tumor-endothelial cell signaling and neovascularization are not well understood. Understanding the reciprocal interaction between endothelial and tumor cells to mediate angiogenesis, and the effect of fluid shear on this process, may offer insight into the development of improved treatment modalities to control highly vascularized tumors. We have previously shown that breast cancer cells cultured under 2D, static conditions with endothelial cells significantly increase expression of pro-angiogenic factors vascular endothelial growth factor (VEGF) and angiopoietin 2 (ANG2) [1]. These preliminary results motivated the investigation of tumor-endothelial cross-talk under 3D, dynamic co-culture conditions.

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Yuan, Fang, Chris Pua, Pei Zhong, and Yunbo Liu. "HIFU-Induced Gene Activation in a Cell-Embedded Tissue Mimicking Phantom." In ASME 2007 International Mechanical Engineering Congress and Exposition. ASMEDC, 2007. http://dx.doi.org/10.1115/imece2007-43996.

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The synergistic integration of high intensity focused ultrasound (HIFU) thermal ablation and HIFU-induced gene therapy represents a promising approach in improving the overall efficacy and quality of cancer therapy. Previous studies have demonstrated that HIFU can induce GFP gene activation under the control of hsp70B promoter in a murine tumor model [1]. Thermal stress has been identified as the primary mechanism to regulate the gene expression. However, the natural heterogeneity and opacity of solid tumors has hindered direct correlation of site-specific gene expression level with in situ thermal dosimetry. We have developed a homogeneous and transparent cell-embedded tissue mimicking phantom as an alternative for simultaneous assessment of temperature distribution, HIFU lesion formation, and gene expression.

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Islam, Atiq, Khan M. Iftekharuddin, and E. Olusegun George. "Gene Expression Based CNS Tumor Prototype for Automatic Tumor Detection." In 2006 Fortieth Asilomar Conference on Signals, Systems and Computers. IEEE, 2006. http://dx.doi.org/10.1109/acssc.2006.354869.

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Trager, Megan H., Emanuelle Rizk, Sharon Rose, Branden Lau, Ben Fullerton, Kuixi Zhu, Jaya Pradhan, et al. "Abstract PO063: Transcriptomic analysis identifies changes in gene expression in Actinic Keratoses after treatment with imiquimod and differential gene expression." In Abstracts: AACR Virtual Special Conference: Tumor Immunology and Immunotherapy; October 19-20, 2020. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/2326-6074.tumimm20-po063.

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Lyu, Boyu, and Anamul Haque. "Deep Learning Based Tumor Type Classification Using Gene Expression Data." In BCB '18: 9th ACM International Conference on Bioinformatics, Computational Biology and Health Informatics. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3233547.3233588.

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Yang, Wen-Hui, Dao-Qing Dai, and Hong Yan. "Generalized Discriminant Analysis for Tumor Classification with Gene Expression Data." In Proceedings of 2006 International Conference on Machine Learning and Cybernetics. IEEE, 2006. http://dx.doi.org/10.1109/icmlc.2006.259021.

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Wang, Lu-yong, and Zhuowen Tu. "Lung Tumor Diagnosis and Subtype Discovery by Gene Expression Profiling." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.259539.

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Wang, Lu-yong, and Zhuowen Tu. "Lung Tumor Diagnosis and Subtype Discovery by Gene Expression Profiling." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.4398792.

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Phuong, Nguyen Minh, and Nguyen Xuan Vinh. "Normalized EM algorithm for tumor clustering using gene expression data." In 2008 8th IEEE International Conference on Bioinformatics and BioEngineering (BIBE). IEEE, 2008. http://dx.doi.org/10.1109/bibe.2008.4696683.

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Reports on the topic "Tumor; Gene expression"

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Klauber-DeMore, Nancy. Characterization of Gene Expression in Human Breast Tumor Endothelium. Fort Belvoir, VA: Defense Technical Information Center, May 2008. http://dx.doi.org/10.21236/ada486849.

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Collart, F. R., C. B. Chubb, B. L. Mirkin, and E. Huberman. Increased IMP dehydrogenase gene expression in solid tumor tissues and tumor cell lines. Office of Scientific and Technical Information (OSTI), July 1992. http://dx.doi.org/10.2172/10148922.

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Swafford, D. S., J. Tesfaigzi, and S. A. Belinsky. Expression of the p16{sup INK4a} tumor suppressor gene in rodent lung tumors. Office of Scientific and Technical Information (OSTI), December 1995. http://dx.doi.org/10.2172/381388.

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Furbert-Harris, Paulette. Eosinophil Granular Protein(s) Modulate Tumor Metastasis Marker Gene Expression. Fort Belvoir, VA: Defense Technical Information Center, May 2007. http://dx.doi.org/10.21236/ada473779.

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Xiao, Hua. Regulation of Estrogen-Responsive Gene Expression and Tumor Suppression by Transcriptional Cofactors. Fort Belvoir, VA: Defense Technical Information Center, May 2005. http://dx.doi.org/10.21236/ada437873.

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Carey, Michael F. Interrogating Androgen Receptor Mediated Gene Expression and Tumor Progression by Molecular Imaging. Fort Belvoir, VA: Defense Technical Information Center, October 2005. http://dx.doi.org/10.21236/ada443777.

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Carey, Michael F. Interrogating Androgen Receptor Mediated Gene Expression and Tumor Progression by Molecular Imaging. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada431376.

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Carey, Michael F. Interrogating Androgen Receptor Mediated Gene Expression and Tumor Progression by Molecular Imaging. Fort Belvoir, VA: Defense Technical Information Center, October 2003. http://dx.doi.org/10.21236/ada419790.

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Xiao, Hua. Regulation of Estrogen-Responsive Gene Expression and Tumor Suppression by Transcriptional Cofactors. Fort Belvoir, VA: Defense Technical Information Center, May 2004. http://dx.doi.org/10.21236/ada426879.

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Murphy, Maureen E. Regulation of IAP (Inhibitor of Apoptosis) Gene Expression by the p53 Tumor Suppressor Protein. Fort Belvoir, VA: Defense Technical Information Center, May 2005. http://dx.doi.org/10.21236/ada436891.

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