Relevant bibliographies by topics / Tumor

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Tumor'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Tumor"

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Mathew, George, and Shwetha Jose. "PRIMARY TUMORS AND TUMOR-LIKE LESIONS OF BONE IN CHILDREN." International Journal of Integrative Medical Sciences 4, no. 6 (November 3, 2017): 507–11. http://dx.doi.org/10.16965/ijims.2017.112.

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Légrádi, Mária, Jenő Mojzes, Endre Kálmán, Evelin Csernus, and Zita Battyáni. "Abrikossoff`s tumor – Granular cell tumor." Bőrgyógyászati és Venerológiai Szemle 88, no. 2 (April 30, 2012): 56–59. http://dx.doi.org/10.7188/bvsz.2012.88.2.4.

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Rodin, William Carl Ivar, Patrik Sundström, Filip Ahlmanner, Elinor Bexe Lindskog, and Marianne Quiding Järbrink. "Potent anti-tumor effector functions in tumor-infiltrating MAIT and γδ T cells isolated from colon cancer patients." Journal of Immunology 202, no. 1_Supplement (May 1, 2019): 138.13. http://dx.doi.org/10.4049/jimmunol.202.supp.138.13.

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Abstract In colon cancer, tumor progression and patient outcome is affected by the cytokine balance in the tumors. IFNγ, TNFα and Granzyme B expression are associated with favorable patient outcome, while high IL-17 expression is associated with accelerated tumor progression. However, knowledge of the regulation and activation of unconventional T cell subsets in colon tumors is limited. The aim of this study was to characterize unconventional T cells in colon tumors and unaffected tissue, determine their capacity to produce cytokines affecting tumor progression as well as the In vitro cytotoxic capabilities of MAIT and γδ T cells. Using flow cytometry, we show that MAIT cells accumulate in colon tumors and that the frequencies of γδ T cells are reduced in the tumor epithelium. Using polyclonal stimulation, we show that IFNγ production by tumour infiltrating MAIT cells is impaired whilst tumour infiltrating γδ T have an increased expression of IFNγ, TNFα and Granzyme B. IL-17 expression was also elevated in tumour infiltrating γδ T cells, but at lower levels than the TH1 - associated cytokines. Tumor infiltrating MAIT cells had an exhausted (PD-1highTim-3+) phenotype compared to MAIT cells from unaffected tissue. Analyzing cytokine expression, we show that while no single molecule is lost the polyfunctional capacity of tumour infiltrating MAIT cells is decreased compared to MAIT cells from unaffected tissue. Altogether, this study shows that γδ T cells and MAIT cells contribute to the cytokine balance in colon tumors with a TH1 – dominated profile and that they have potent cytotoxic capacity, which may reduce tumor progression and improve patient outcome.
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Patel, Falguni T., Bhoomi A. Shah, Nisarg R. Parikh, and Ratigar N. Gonsai. "Steroid cell tumor : A rare ovarian tumor." Annals of Pathology and Laboratory Medicine 6, no. 8 (August 26, 2019): C88–90. http://dx.doi.org/10.21276/apalm.2500.

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Raheja, Aastha, Deepti Goswami, Pooja Sakhija, Gauri Gandhi, and Bidhisha Singhla. "Steroid Cell Tumor: A Rare Ovarian Tumor." Annals of Applied Bio-Sciences 5, no. 2 (June 2018): C1–3. http://dx.doi.org/10.21276/aabs.2167.

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Sengupta, Sanjay, Subrata Pal, Biplab Kr Biswas, Srabani Chakrabarti, Kingshuk Bose, and Sritanu Jana. "Collision tumor of ovary: a rare combination of dysgerminoma and serous cystadenocarcinoma." Bangladesh Journal of Medical Science 13, no. 2 (March 3, 2014): 224–27. http://dx.doi.org/10.3329/bjms.v13i2.14955.

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Collision tumors of ovary are rare neoplasms and most commonly consist of a teratoma with mucinous tumor. Combination of papillary serous cystadenocarcinoma and dysgerminoma was yet to be reported. A twenty years female patient presented with a large tumor of right ovary. Microscopically it was diagnosed as a collision tumor of ovary composed of dysgerminoma and serous cystadenocarcinoma. Mixed tumour can arise from divergent differentiation of a single type of stem cell. But components of collision tumor must arise from separate clones. Possibility of collision tumour should always kept in mind during assessment of difficult ovarian tumors to avoid diagnostic error. DOI: http://dx.doi.org/10.3329/bjms.v13i2.14955 Bangladesh Journal of Medical Science Vol.13(2) 2014 p.224-227
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Milas, L. "Tumor Bed Effect in Murine Tumors: Relationship to Tumor Take and Tumor Macrophage Content." Radiation Research 123, no. 2 (August 1990): 232. http://dx.doi.org/10.2307/3577551.

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Punam, Saudagar. "Deep Learning Approach for Brain Tumor Classification." International Journal for Research in Applied Science and Engineering Technology 9, no. VI (June 30, 2021): 3094–98. http://dx.doi.org/10.22214/ijraset.2021.35648.

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Tumors are complex. There are a lot of variations in sizes and location of tumor. This makes it really hard for complete understanding of tumor. Brain tumour is the abnormal growth of cells inside the brain cranium which limits the functioning of brain. Now a days, medical images processing is a most challenging and developing field. Automated detection of tumor in MRI is extremely crucial because it provides information about abnormal tissues which is important for planning treatment. The conventional method for defect detection in resonance brain images is time consuming. So, automated tumor detection methods are developed because it would save radiologist time and acquire a tested accuracy. The MRI brain tumor detection is complicated task due to complexity and variance of tumors.There are many previously implemented approaches on detecting these kinds of brain tumors. In this paper, we used and implement Convolutional Neural Network (CNN) which is one among the foremost widely used deep learning architectures for classifying a brain tumor into four types. i.e Glioma , Meningioma, Pituitary and No tumour. CNN may be used to effectively locate most cancers cells in brain via MRI. classification.
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Malik, Shan Nawaz, Mohammad Khursheed Alam, Mariyam Shahina, Salman Siddique, and Vishnu Das Prabhu. "Calcifying Epithelial Odontogenic Tumor (Ceot)." Bangladesh Journal of Medical Science 13, no. 1 (December 24, 2013): 14–19. http://dx.doi.org/10.3329/bjms.v13i1.17378.

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Calcifying epithelial odontogenic tumor is a rare benign epithelial odontogenic lesion that comprises from 0.2% to 1.1 of all odontogenic tumors. In the past a number of different names have been given to this lesion, such as calcifying ameloblastoma, cystic complex odontoma, uncommon ameloblastoma with calcifications and others. There is a need to study and explore various aspects of this tumour, this article gives a broad idea of the various aspects of this tumor and which aspect of this tumour needs more investigation. DOI: http://dx.doi.org/10.3329/bjms.v13i1.17378 Bangladesh Journal of Medical Science Vol. 13 No. 01 January2014: 14-19
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Barreda Bolaños, Fernando, Humberto Liu Bejarano, Lidia Rodriguez Briceño, Bruno Salmon Alva, Edith Sulca Flores, Luis Taxa Rojas, and Francisco Berrospi Espinoza. "Tumor sólido pseudopapilar de páncreas: Tumor de Frantz." Horizonte Médico (Lima) 18, no. 2 (December 31, 2018): 80–85. http://dx.doi.org/10.24265/horizmed.2018.v18n2.12.

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Dissertations / Theses on the topic "Tumor"

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Igney, Frederik. "Tumor Counterattack: Apoptose-vermittelte Immunsuppression durch Tumore." [S.l. : s.n.], 2002. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB10283671.

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Cataldo, A. "ANTI-TUMOR ACTIVITY OF CPG-ODN IN OVARIAN XENOGRAFT TUMORS." Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/229558.

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Synthetic oligodeoxynucleotides expressing CpG motifs (CpG-ODN), Toll-like receptor 9 (TLR9) agonists, are able to induce innate/adaptive immune responses and can enhance the antitumor activity of DNA-damaging chemotherapy and radiation therapy in preclinical mouse models. It was recently reported that peritumoral CpG-ODN treatment in preclinical models of ovarian cancer, activating TLR-9 expressing cells in tumor microenvironment, induces modulation of DNA repair genes and sensitizes cancer cells to DNA-damaging Cisplatin treatment. In this thesis we investigated whether this treatment induces modulation of miRNAs in tumor cells and their relevance to chemotherapy response. Array analysis identified 20 differentially expressed miRNAs (16 down- and 4 up-regulated) in human IGROV-1 ovarian tumor cells from CpG-ODN-treated mice versus controls. Evaluation of the role of the 3 most differentially expressed miRNAs on sensitivity to Cisplatin of IGROV-1 cells revealed significant increased Cisplatin cytotoxicity upon ectopic expression of hsa-miR-302b (up-modulated in our array), but no increased effect upon reduced expression of hsa-miR-424 or hsa-miR-340 (down-modulated in our array). The impact of expression levels of all 20 differentially expressed miRNAs were associated with time to replase and overall survival probability in two data sets of ovarian cancer patients treated with platinum. It was found that hsa-miR-302b expression was significantly associated with time to relapse or overall survival in these patients. Use of bio-informatics tools identified 19 mRNAs potentially targeted by hsa-miR-302b, including HDAC4 gene, which has been reported to mediate Cisplatin sensitivity in ovarian cancer. Both HDAC4 mRNA and protein levels were significantly reduced in IGROV-1 cells overexpressing hsa-miR-302b. Altogether, these findings indicate that hsa-miR-302b acts as a ‘‘chemosensitizer’’ in human ovarian carcinoma cells and may represent a biomarker able to predict response to Cisplatin treatment. Moreover, the identification of miRNAs that improve sensitivity to chemotherapy provides the experimental underpinning for their possible future clinical use. In the second part of this thesis we tested the efficacy of CpG-ODN in combination with other possible therapeutic agents in ovarian carcinoma ascites-bearing athymic mice, to mimic clinical treatment situations in advanced human ovarian disease. Mice injected i.p. with IGROV-1 ovarian cancer cells were treated at different stages of ascites progression for 4 weeks with CpG-ODN alone or in combination with Bevacizumab, Polyinosinic:Polycytidylic acid (Poly(I):Poly(C)), Gefitinib, Cetuximab and Cisplatin. In mice treated when ascitic fluid began to accumulate, CpG-ODN combined with Bevacizumab, Poly(I): Poly(C) or Gefitinib did not significantly increase Median Survival Times (MST), as compared with that using CpG-ODN alone, whereas MST in mice treated with CpG-ODN plus Cetuximab was significantly increased (>103 days for combination vs 62 days for CpG alone; P = 0.0008), with 4/8 mice alive at the end of the experiment. In mice showing evident and established ascites, evaluated with increase of abdominal volume and body weight (27.9 ± 0.8 g after vs 23 ± 1.1 g before tumor cell injection), treatment with Cisplatin in addition to CpG-ODN/Cetuximab led to significantly increased MST (105.5 days; P = 0.001), with all mice still alive at 85 days, over that using CpG ODN/Cetuximab (66 days), Cetuximab/Cisplatin (18.5 days), Cisplatin (23 days) or saline (16 days). At a very advanced stage of disease (body weight: 31.4 ± 0.9 g), when more than half of control mice had to be sacrificed 6 days after starting treatments, the triple-combination therapy still increased MST (45 days; P = 0.0089) vs controls. These data indicated that CpG-ODN combination therapies that enhance the immune response in the tumor microenvironment and concomitantly target tumor cells are highly efficacious even in experimental advanced malignancies. Although differences in the distribution of TLR9 in mice and humans and the enrichment of this receptor on innate immune cells of athymic mice must be considered, our results indicate a promising strategy to treat ovarian cancer patients with bulky ascites.
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Swanson, Kristin Rae. "Mathematical modeling of the growth and control of tumors /." Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/6764.

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Lu, Dan. "Tumor priming enhances particle delivery to and transport in solid tumors." Columbus, Ohio : Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1144936804.

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Tai, Kai-chun Dora, and 戴啟真. "Krukenberg tumours of colorectal origin: experience of a tertiary referral centre and review of theliterature." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B4562043X.

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Butler, Savannah E. "TUMOR-INTRINSIC INFLAMMATORY PATHWAYS ASSOCIATED WITH TUMOR DORMANCY AND RECURRENCE." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/4753.

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The successful treatment of breast cancer is limited due to a fraction of tumor cells escaping drug-treatment by entering a dormant state, only to relapse years or decades later at distant sites. Host-driven chronic inflammatory cells such as M2 macrophages play an important role in tumorigenesis, but the role of tumor-intrinsic inflammatory signaling involved in tumor dormancy and recurrence is unknown. We sought to determine the role of tumor-intrinsic inflammatory pathways in mouse mammary carcinoma cells (MMC) treated with Adriamycin (ADR), a clinically relevant chemotherapeutic drug. We found that ADR-induced dormant tumor cells autonomously produced pro-inflammatory cytokines, in vitro. MMC treated with Chloroquine (CQ) prior to ADR treatment displayed a delay in relapse, or prolonging of dormancy, when compared to ADR-treated MMC. Additional gene array data showed predicated activation of NF-κB p65 in ADR-treated dormant MMC that eventually relapsed. These data suggest that the anti-inflammatory function of CQ led to prolonged dormancy. To test this, we investigated the role of inflammatory signaling pathways directly by shRNA-mediated knockdown and CRISPR-Cas9-mediated knockout of NF-κB p65 in MMC. We found that knockdown of NF-κB p65 resulted in fewer dormant cells after ADR treatment and reduced rate of relapse, in vitro. NF-κB p65 was also found to reduce the immunomodulatory effects of ADR, with shNF-κB p65 showing increased upregulation of neu upon ADR treatment. Additionally, we found NF-κB p65 to be associated with a higher infiltration of CD8+ T cells and anti-tumor T cell responses. Our findings suggest a dual role of tumor-intrinsic NF-κB p65 pathway, allowing for escape from drug treatment through dormancy which leads to relapse, but also for proper lymphocyte infiltration and subsequent anti-tumor activity.
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Casiraghi, Nicola. "Quantitative analyses to study tumor clones dynamics and tumor heterogeneity." Doctoral thesis, Università degli studi di Trento, 2017. https://hdl.handle.net/11572/368498.

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Prostate cancer is a highly heterogeneous disease and its manifestations can vary from indolent localized tumor to widespread metastases. This heterogeneity is also observed at the molecular level both inter- and intra-patient. Intra-patient heterogeneity in the clinical setting of men with castration resistant prostate cancer (CRPC) might be informative in terms of treatment decision. Here I present analytical work on two approaches relevant to the characterization of intra-patient heterogeneity and applied to unpublished CRPC patients sequencing data. The first is based on the genome wide interrogation of multiple metastatic and primary tissue biopsies from single patients. I present genomic analyses to decipher the content of multiple tumor biopsies from CRPC patients and provide comparisons to highlight similarities and differences and to identify alternative patterns of aberrations. The second approach, alternative to tissue biopsies that might under-represent the genomic landscape of the patient’s disease, relies on liquid biopsies, a minimally invasive test that is also amenable to serial sampling. Liquid biopsies contain circulating cell free DNA (cfDNA) released from widespread tumor cells, potentially uncovering the full tumor landscape. By using next generation sequencing on cfDNA obtained from plasma, I developed strategies aimed at systematically tracking the reiterative process of genetic diversification leading to disease evolution and to detect genomic aberrations. I specifically focused on an ad hoc computational procedure (ABEMUS) to detect somatic point mutations that could emerge under treatment pressure and as drug resistance mechanism. The work I present is relevant to the context of precision oncology that exploits detailed patient-specific molecular information to diagnose and follow cancer progression with the ultimate goal of promptly guiding treatment decisions to improve clinical outcome with transdisciplinary strategies. The analytical work I developed can be applied to the study of any tumor type.
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Casiraghi, Nicola. "Quantitative analyses to study tumor clones dynamics and tumor heterogeneity." Doctoral thesis, University of Trento, 2017. http://eprints-phd.biblio.unitn.it/2626/2/Disclaimer_Casiraghi.pdf.

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Prostate cancer is a highly heterogeneous disease and its manifestations can vary from indolent localized tumor to widespread metastases. This heterogeneity is also observed at the molecular level both inter- and intra-patient. Intra-patient heterogeneity in the clinical setting of men with castration resistant prostate cancer (CRPC) might be informative in terms of treatment decision. Here I present analytical work on two approaches relevant to the characterization of intra-patient heterogeneity and applied to unpublished CRPC patients sequencing data. The first is based on the genome wide interrogation of multiple metastatic and primary tissue biopsies from single patients. I present genomic analyses to decipher the content of multiple tumor biopsies from CRPC patients and provide comparisons to highlight similarities and differences and to identify alternative patterns of aberrations. The second approach, alternative to tissue biopsies that might under-represent the genomic landscape of the patient’s disease, relies on liquid biopsies, a minimally invasive test that is also amenable to serial sampling. Liquid biopsies contain circulating cell free DNA (cfDNA) released from widespread tumor cells, potentially uncovering the full tumor landscape. By using next generation sequencing on cfDNA obtained from plasma, I developed strategies aimed at systematically tracking the reiterative process of genetic diversification leading to disease evolution and to detect genomic aberrations. I specifically focused on an ad hoc computational procedure (ABEMUS) to detect somatic point mutations that could emerge under treatment pressure and as drug resistance mechanism. The work I present is relevant to the context of precision oncology that exploits detailed patient-specific molecular information to diagnose and follow cancer progression with the ultimate goal of promptly guiding treatment decisions to improve clinical outcome with transdisciplinary strategies. The analytical work I developed can be applied to the study of any tumor type.
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Diego, Ángeles Pedro, Ximena Vega, and José Palacios. "Tumor mucoso apendicular." Asociación Colombiana de Cirugía, 2016. http://hdl.handle.net/10757/615473.

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Los tumores mucosos apendiculares tienen baja incidencia y comúnmente se diagnostican en el estudio anatomo-patólogico después de la apendicectomía. Se reporta el caso de una mujer de 41 años de edad, con un cuadro clínico de ocho meses de evolución, caracterizado por dolor abdominal de tipo opresivo, difuso y de gran intensidad en el hemiabdomen inferior, acompañado de náuseas. Después de cinco meses de iniciado este cuadro clínico, se evidenció una masa en la fosa iliaca derecha; el dolor se agudizó e intensificó, y las náuseas continuaron, por lo cual fue remitida al hospital. En los exámenes practicados se observó una masa quística compleja abdomino-pélvica de origen indeterminado, y la tomografía computadorizada de abdomen fue sugestiva de mucocele apendicular. Con estos hallazgos, se optó por el tratamiento quirúrgico por laparotomía, consistente en hemicolectomía derecha, con resección parcial de íleon, epiplectomía, histerectomía y salpigooforectomía bilateral.
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Petty, Aaron. "Novel MIG-7 expression increases tumor cell invasion and tumor progression." Online access for everyone, 2008. http://www.dissertations.wsu.edu/Thesis/Spring2008/a_petty_040908.pdf.

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Books on the topic "Tumor"

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Noel, Tuazon, ed. Tumor. Los Angeles, Calif: Archaia Entertainment, 2010.

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Eissa, Saad. Tumor markers. London: Chapman & Hall, 1998.

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Santini-Araujo, Eduardo, Ricardo K. Kalil, Franco Bertoni, and Yong-Koo Park, eds. Tumors and Tumor-Like Lesions of Bone. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-28315-5.

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Santini-Araujo, Eduardo, Ricardo K. Kalil, Franco Bertoni, and Yong-Koo Park, eds. Tumors and Tumor-Like Lesions of Bone. London: Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6578-1.

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Bhutia, Sujit Kumar, ed. Autophagy in tumor and tumor microenvironment. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6930-2.

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C, Ghosh Bimal, and Ghosh Luna, eds. Tumor markers and tumor-associated antigens. New York: McGraw-Hill, 1987.

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Margaret, Hanausek, and Walaszek Zbigniew, eds. Tumor marker protocols. Totowa, N.J: Humana Press, 1998.

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Fan, Z. Hugh. Circulating tumor cells: Isolation and analysis. Hoboken, New Jersey: John Wiley & Sons, Inc., 2016.

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Birbrair, Alexander, ed. Tumor Microenvironment. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-62658-7.

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Birbrair, Alexander, ed. Tumor Microenvironment. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73119-9.

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Book chapters on the topic "Tumor"

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Lu, Jian-Qiang, and Arthur W. Clark. "Tumor-to-Tumor Metastasis: Extracranial Tumor Metastatic to Intracranial Tumors." In Tumors of the Central Nervous system, Volume 3, 35–46. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1399-4_4.

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Soun, Jennifer, Lu-Aung Yosuke Masudathaya, Arabdha Biswas, and Daniel S. Chow. "The Role of Artificial Intelligence in Neuro-oncology Imaging." In Machine Learning for Brain Disorders, 963–76. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3195-9_30.

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AbstractDiagnostic imaging is widely used to assess, characterize, and monitor brain tumors. However, there remain several challenges in each of these categories due to the heterogeneous nature of these tumors. This may include variations in tumor biology that relate to variable degrees of cellular proliferation, invasion, and necrosis that in turn have different imaging manifestations. These variations have created challenges for tumor assessment, including segmentation, surveillance, and molecular characterizations. Although several rule-based approaches have been implemented that relates to tumor size and appearance, these methods inherently distill the rich amount of tumor imaging data into a limited number of variables. Approaches in artificial intelligence, machine learning, and deep learning have been increasingly leveraged to computer vision tasks, including tumor imaging, given their effectiveness for solving image-based challenges. This objective of this chapter is to summarize some of these advances in the field of tumor imaging.
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Tsui, Wilson M. S., Takahiro Mori, and Masaki Iwai. "Liver Tumor I: Benign Tumors and Tumor-Like Lesions." In Diagnosis of Liver Disease, 211–33. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-6806-6_16.

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Hegazi, Tarek M., and Jim S. Wu. "Tumor/Tumor-Like Lesions." In Musculoskeletal MRI, 203–30. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-26777-3_7.

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Kalil, Ricardo K. "Glomus Tumor." In Tumors and Tumor-Like Lesions of Bone, 487–94. London: Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6578-1_34.

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Zhang, Yaxia, and Andrew E. Rosenberg. "Glomus Tumor." In Tumors and Tumor-Like Lesions of Bone, 505–10. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-28315-5_37.

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Bährle-Rapp, Marina. "Tumor." In Springer Lexikon Kosmetik und Körperpflege, 569. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_10811.

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George, Bernard, and Claude Laurian. "Tumor." In The Vertebral Artery, 58–76. Vienna: Springer Vienna, 1987. http://dx.doi.org/10.1007/978-3-7091-6967-4_6.

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Peterson, Hamlet A. "Tumor." In Physeal Injury Other Than Fracture, 115–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-22563-5_4.

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Arampatzis, Adamantios, Lida Mademli, Thomas Reilly, Mike I. Lambert, Laurent Bosquet, Jean-Paul Richalet, Thierry Busso, et al. "Tumor." In Encyclopedia of Exercise Medicine in Health and Disease, 883. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4565.

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Conference papers on the topic "Tumor"

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Chopra, Seema. "Sclerosing sex cord stromal tumour of the ovary: A rare variant of ovarian neoplasms in childhood and adolescence." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685321.

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Case Report: 19 yr old unmarried girl c/o abdominal distension, loss of appetite and Irregular menstrual cycles x 5 months. USG: gross ascites, liver, Lobulated isoechoic mass in right adnexa, 7x5 cm, abutting right ovary. CA125: 1297 U/ml. FNAC Degenerated crushed cells & stromal fragments. Few scattered benign oval/spindle cells. Laparoscopy f/b laparotomy: 6 litres of straw colored asciic fluid drained. Uterus, left adnexa normal. Rt ovarian mass 6x7 cm, bilobed, arising from ovary. Solid, stuck in POD Adherent to gut. Right oophrectomy done. CA-125: 22 u/ml on day 6 post op. HPE – Sclerosing stromal tumor. Discussion: Sclerosing sex cord stromal tumour of the ovary is a rare tumor; accounts for 6% of ovarian stromal tumors Over a 100 reported tumors in literature. 80% of SST seen in second and third decade of life. Essentially a benign tumour, Usually a unilateral nonfunctioning tumor. Few cases with elevated serum CA-125 and hormonal abnormalities have been reported. Endocrine alterations caused by secretion of estrogen, progesterone or testosterone; induction of precocious puberty. Conclusion: Unilateral oophrectomy is the treatment. No recurrence of the tumor in the patients treated by oophorectomy or by conservative resection of the tumor. Excision of the tumor isfollowed by normal menses, pregnancy has also been reported.
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Savita, Pannu, and Khullar Harsha. "Two interesting cases of granulosa cell tumor: A case report." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685326.

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Introduction: Granulosa cell tumor (GCT) is an ovarian malignancy that arise from granulosa cells of the ovary. This tumour is a type of the sex cord-gonadal stromal tumour. GCT have good prognosis in comparison with other epithelial tumors. Methodology: Two cases of granulosa cell tumors were diagnosed in sir Ganga ram hospital, Rajendernagar, New Delhi in December 2015 and January 2016. The patient’s age, clinical manifestations, radiological and histopathological findings were evaluated. One was in perimenopausal age group and other case was in postmenopausal age group. The clinical manifestations were menorrhagia and abdominal pain. Ultrasonographically, in one case focal hypoechoic zone showing peripheral hypervascularity with possibility of old hemorrhage follicular cyst was seen and in other case of granulosa cell tumors was both solid and cystic areas were seen. Histologically, variety of patterns like diffuse, trabecular, nodular, sheets, nests and fascicular patterns with nuclear grooving in ovarian tissue. In addition endometrial findings were suggestive of simple hyperplasia without atypia. Treatment modalility used was surgery i.e., Total hysterectomy and bilateral salpingo-oophorectomy in both cases. Conclusion: Granulosa cell tumor of the ovary is a rare ovarian malignancy. Endometrial pathology to rule out endometrial carcinomaspecially when postmenopausal bleeding is concomitant finding is advised. Radical surgery is usually not required.
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Savita, Pannu, and Khullar Harsha. "Two interesting cases of granulosa cell tumor: A case report." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685309.

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Introduction: Granulosa cell tumor (GCT) is an ovarian malignancy that arise from granulosa cells of the ovary. This tumour is a type of the sex cord-gonadal stromal tumour. GCT have good prognosis in comparison with other epithelial tumors. Methodology: Two cases of granulosa cell tumors were diagnosed in sir Ganga ram hospital, Rajender Nagar, New Delhi in December 2015 and January 2016. The patient’s age, clinical manifestations, radiological and histopathological findings were evaluated. One was in perimenopausal age group and other case was in postmenopausal age group. The clinical manifestations were menorrhagia and abdominal pain. Ultrasonographically, in one case focal hypoechoic zone showing peripheral hypervascularity with possibility of old hemorrhage follicular cyst was seen and in other case of granulosa cell tumors was both solid and cystic areas were seen. Histologically, variety of patterns like diffuse, trabecular, nodular, sheets, nests and fascicular patterns with nuclear grooving in ovarian tissue. In addition endometrial findings were suggestive of simple hyperplasia without atypia. Treatment modalility used was surgery i.e. Total hysterectomy and bilateral salpingo-oophorectomy in both cases. Conclusion: Granulosa cell tumor of the ovary is a rare ovarian malignancy. Endometrial pathology to rule out endometrial carcinoma specially when postmenopausal bleeding is concomitant finding is advised. Radical surgery is usually not required.
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Pyrz, Matthew, and James Baish. "Effect of Tumor Heterogeneity on Interstitial Pressure and Fluid Flow." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14089.

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Delivery of drugs to solid tumors is often impeded by the elevation of the interstitial fluid pressure at the tumor core and the loss of plasma from the tumor to the surrounding normal tissue. Interstitial pressure in tumors is abnormally high because tumor blood vessels are highly permeable relative to their normal counterparts and tumors lack functional lymphatic vessels that can absorb the excess fluid loss [1]. As a result, there is a net outward flow of plasma-like fluid through the interstitial spaces of the tumor toward the exterior of the tumor where functional lymphatics may be found (Fig. 1).
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Singh, Anurag, and Prachi Chauhan. "A REVIEW ON BRAIN TUMOR MRI IMAGE SEGMENTATION." In Computing for Sustainable Innovation: Shaping Tomorrow’s World. Innovative Research Publication, 2024. http://dx.doi.org/10.55524/csistw.2024.12.1.53.

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A brain tumor represents one of the most perilous medical conditions. Consequently, swift and precise detection of brain tumors is imperative. Employing automated techniques for identifying brain tumor tissue aids in containing the proliferation of tumor cells within the system. MRI technology is utilized to capture high-resolution images of both the individual's anatomy and cancerous tissues, offering superior image quality compared to other imaging modalities. However, pinpointing brain tumors in MRI images poses challenges due to the intricate nature of the brain. Processing MRI images and segmenting brain tumors therein constitute complex tasks. Automated segmentation in image processing serves as a method for partitioning an image into distinct regions. This process stands as a critical and indispensable phase in the analysis of brain tumor magnetic resonance imaging. Moreover, the utilization of advanced image processing techniques facilitates the enhancement of MRI images, improving their clarity and aiding in the identification of subtle abnormalities associated with brain tumors. This enhanced image quality is particularly crucial for detecting small or deeply situated tumors that may otherwise go unnoticed.
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LeBrun, Alexander, Navid Manuchehrabadi, Anilchandra Attaluri, Ronghui Ma, and Liang Zhu. "Tumor Geometry and SAR Distribution Generated From MicroCT Images for Tumor Temperature Elevation Simulation in Magnetic Nanoparticle Hyperthermia." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14505.

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Previous investigations in magnetic nanoparticle hyperthermia for cancer treatments have demonstrated that particle size, particle coating, and magnetic field strength and frequency determine its heating generation capacity. However, once the nanoparticles are manufactured, the spatial distribution of the nanostructures dispersed in tissue dominates the spatial temperature elevation during heating. 1–3 Therefore, understanding the distribution of magnetic nanoparticles in tumors is critical to develop theoretical models to predict temperature distribution in tumors during hyperthermia treatment. An accurate description of the nanoparticle distribution and the tumor geometry will greatly enhance the simulation accuracy of the heat transfer process in tumors, which is crucial for generating an optimal temperature distribution that can prevent the occurrence of heating under-dosage in the tumor and overheating in the healthy tissue. Recently studies by our group have demonstrated that the nanoparticle concentration distribution in tumors can be visualized via microCT image due to the density elevation of the presence of magnetic nanoparticles. 4 The problem is the intensive memory requirements to directly import the microCT images to numerical simulation software packages such as COMSOL. Although commercial software packages exist to handle detailed entities inside tumors, they are very expensive to purchase. In addition, having very small entities at the micrometer level inside the tumor geometry may provide challenge to numerical simulation software to accept the generated geometry.
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Tuteja, Geetanjali, S. Unmesh, S. Shree, and S. Rudra. "Juvenile granulosa cell tumor." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685332.

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The differential diagnosis for precocious puberty in a young female includes peripheral causes. This case report documents a rare cause of isosexual precocious puberty, a juvenile granulosa cell tumour of the ovary–and a brief literature review. A one year-old baby girl presented with mass abdomen, vaginal discharge and rapid onset of pubertal development. She underwent an exploratory laparotomy for tumour resection. Pathology reported a juvenile granulosa cell tumour of the ovary. Early stage granulosa cell tumor surgically treated has good prognosis. Adjuvant chemotherapy is not indicated in this setting.
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Tuteja, Geetanjali, S. Unmesh, S. Shree, and S. Rudra. "Juvenile granulosa cell tumor." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685322.

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The differential diagnosis for precocious puberty in a young female includes peripheral causes. This case report documents a rare cause of isosexual precocious puberty, a juvenile granulosa cell tumour of the ovary–and a brief literature review. A one year-old baby girl presented with mass abdomen, vaginal discharge and rapid onset of pubertal development. She underwent an exploratory laparotomy for tumour resection. Pathology reported a juvenile granulosa cell tumour of the ovary. Early stage granulosa cell tumor surgically treated has good prognosis. Adjuvant chemotherapy is not indicated in this setting.
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Bulja, Deniz, and Sandra Vegar-Zubović. "IMIDŽING MAGNETNOM REZONANCOM U EVALUACIJI INTRADURALNIH TUMORA KIČME." In Okrugli sto “Tumori centralnog nervnog sistema”. Academy of Sciences and Arts of Bosnia and Herzegovina, 2021. http://dx.doi.org/10.5644/pi2021.197.03.

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Uvod: Spinalni tumor predstavlja patološku tkivnu masu smještenu unutar kičmene moždine, kičmenog kanala ili vertebralne kolumne. Incidenca primarnih spinalnih tumora je mala i prema navodima iz literature iznosi oko 0,58 do 0,71 na 100.000 pacijenta u godini. Preko dvije trećine svih primarnih spinalnih tumora otpada na tumore intraduralne ekstramedularne lokacije, a ostatak na intraduralne intramedularne tumore. Uvođenjem MRI u kliničku praksu došlo je do revolucije u karakterizaciji spinalnih tumora kroz detaljnu i direktnu vizualizaciju koštane srži i kičmene moždine u više ravni, čime je omogućeno ranije otkrivanje i liječenje kako ekstraduralnih tako i intraduralnih tumora. Iako imidžing putem CT-a daje pouzdane informacije u pogledu procjene matriksa tumora i koštanih promjena kod ekstraduralne lokacije, MRI je metod izbora u procjeni mekotkivne ekstenzije tumora, infiltracije koštane srži i eventualne intraspinalne propagacije. Ciljevi: 1. Prikazati ulogu imidžinga magnetnom rezonancom kod evaluacije intraduralnih spinalnih tumora evaluiranih na Klinici za radiologiju Kliničkog centra Univerziteza u Sarajevu putem predavanja u vidu slikovnog eseja. 2. Klasificirati primarne i sekundarne spinalne tumore u odnosu na njihovo porijeklo iz ekstraduralnog, intraduralnog ekstramedularnog i intraduralnog intramedularnog kompartmenta uz razmatranje dijagnotičkog pristupa u kliničkoj obradi pacijenata s intraduralnim spinalnim tumorima. 3. Pružiti pregled MRI karakteristika različitih primarnih spinalnih tumora intraduralne lokalizacije.
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Jethwani, Umesh, and Divya Jethwani. "Sertoli cell tumor of ovary: A rare case report." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685324.

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Introduction: Sertoli-Leydig cell tumor (SLCT) is a rare ovarian tumor, Constitute less than 0.5% of ovarian tumors. Most tumors are unilateral, confined to the ovaries. They are seen during the second and third decades of life. They are characterized by the presence of testicular structures that produce androgens. Patients have symptoms of virilization (depending on the quantity of androgen). Case Report: A 42-year-old woman presented Amenorrhea for 14 months. Change in her voice for 1 year and Excessive hair growth on her face, chest, and limbs for the last 2 months. She complained of vague abdominal discomfort. No history of anorexia, weight loss, increased libido. Her medical and family history was unremarkable. On examination - Hirsutism and clitoromegaly. Lump of size 10x8 cm palpable in left iliac fossa. Vaginal examination revealed a firm and mobile cystic mass in the right adnexa. An ultrasound examination of the pelvis showed a 17x 13x 9-cm heterogeneous solid cystic mass replacing the left ovary. The right ovary and the uterus were normal. CECT Scan Abdomen-Large heterogenous encapsulated solid soft tissue mass lesions containing areas of calcification arising from left ovary of size 17x13x10.6cm causing displacement of urinary bladder and surrounding bowel loops. Serum testosterone level -2 ng/mL (normal, 0.2–1.2 ng/mL); (DHEAS), CA 125, and alpha fetoprotein (AFP) -normal. On Laparotmy-Large mass of size 17 X 13 cm arising from left adnexa. Uterus and right ovary grossly normal. Total Abdominal hysterectomy, B/L Salpingo-opherectomy and infracolic omentectomy was done. Peritoneal washing were sent for cytologic examination for malignant cells. No liver metastasis. The post operative period was uneventful. Histopathology revealed- confirmed it be Sertoli Leydig cell tumor. 3month follow up – resolution of her virilization symptoms. No increase of her hirsutism. Repeat testosterone levels - within normal range. Conclusion: Only few cases of SLCT have been reported till date Prognosis depends on extent of disease, stage of disease, tumour differentiation, grade. The treatment should be individualized according to the location, state of spread and the patient’s condition.
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Reports on the topic "Tumor"

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Condeelis, John. Isolation of Motile Tumor Cells From Live Breast Tumors. Fort Belvoir, VA: Defense Technical Information Center, June 2001. http://dx.doi.org/10.21236/ada395259.

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Condeelis, John. Isolation of Motile Tumor Cells from Live Breast Tumors. Fort Belvoir, VA: Defense Technical Information Center, June 2002. http://dx.doi.org/10.21236/ada412991.

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Liu, Hanli. Non-Invasive Monitoring of Breast Tumor Oxygenation: A Key to Tumor Therapy Planning and Tumor Prognosis. Fort Belvoir, VA: Defense Technical Information Center, September 2001. http://dx.doi.org/10.21236/ada399998.

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Liu, Hanli. Non-Invasive Monitoring of Breast Tumor Oxygenation: A Key to Tumor Therapy Planning and Tumor Prognosis. Fort Belvoir, VA: Defense Technical Information Center, September 2002. http://dx.doi.org/10.21236/ada413009.

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Albertson, B., and S. Binney. Pituitary tumor evaluation. Office of Scientific and Technical Information (OSTI), November 1995. http://dx.doi.org/10.2172/421339.

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Stein, Stanley, and Michael J. Leibowitz. Autologous Tumor Vaccination. Fort Belvoir, VA: Defense Technical Information Center, July 2002. http://dx.doi.org/10.21236/ada407668.

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Baldwin, Albert S. Promotion of Tumor-Initiating Cells in Primary and Recurrent Breast Tumors. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613713.

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Baldwin, Albert S. Promotion of Tumor-Initiating Cells in Primary and Recurrent Breast Tumors. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada596410.

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Pacheco-Ojeda, Luis, Carolina Sáenz-Gómez, Stalin Cañizares-Quisiguiña, Tatiana Borja-Herrera, Juan Carlos Vallejo-Garzón, and Sergio Poveda. Function Sparing Conservative Approach of a Low-Grade Chondrosarcoma of the Larynx: Case Report and Literature Review. Science Repository, March 2024. http://dx.doi.org/10.31487/j.scr.2024.01.04.

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Background: Laryngeal cancer is relatively uncommon in Ecuador. Usually epithelial in origin, the most frequent histological type is squamous cell carcinoma. The most common mesenchymal tumor is chondrosarcoma. Most laryngeal chondrosarcomas are treated with total laryngectomy, but a conservative function sparing resection is recommended in low-grade limited tumors. Case Report: In a 68-year-old female nonsmoker patient, a small tumor was found in the posterior left aspect of the cricoid cartilage in a computed tomography (CT) performed immediately after an unexpected difficulty to pass the endotracheal tube for a thoracoscopic biopsy of 4 cm tumor of the left lung, in another hospital. The patient underwent, then, an initial tracheostomy, a total thyroidectomy for a goiter and a biopsy of the tumor of the cricoid cartilage whose pathological study was inconclusive. One month later, a low-grade neuroendocrine pulmonary tumor was completed resected. Two years later, a CT scan showed the cricoid lesion with the same characteristics. At endoscopic video laryngoscopy, two subglottic masses that narrowed the airway in approximately 60% of the normal caliber, were observed located at the posterior and left walls. An intraluminal resection was performed through a transcricoid anterior approach. The pathological diagnosis was a low-grade chondrosarcoma. Tracheal decannulation was performed one month later. At an endoscopic video laryngoscopy performed six months post-operatively, the tracheal caliber and mucosa were normal. The patient remained with normal voice and breathing. Conclusion: We report the second case of chondrosarcoma of the larynx in our country, treated by a conservative approach.
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Nie, Daotai. Targeting Tumor Oct4 to Deplete Prostate Tumor and Metastasis Initiating Cells. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613791.

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