Academic literature on the topic 'Trypanosomiasis'

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Journal articles on the topic "Trypanosomiasis"

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Giblin, James. "Trypanosomiasis Control in African History: An Evaded Issue?" Journal of African History 31, no. 1 (March 1990): 59–80. http://dx.doi.org/10.1017/s0021853700024786.

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Social control of trypanosomiasis in African history deserves further study. The pioneering work in this field is John Ford's respected but neglected The Role of the Trypanosomiases in African Ecology (1971). While Ford's arguments have received support from recent findings in immunological, epidemiological and epizootiological research, they have rarely met with evaluation or engagement, either in historical or scientific literature. Historians have tended to describe trypanosomiasis control as a matter of avoiding contact with tsetse fly. In so doing they have implicitly rejected the position of Ford, who regarded infrequent contacts between tsetse and mammalian hosts as necessary for the maintenance of host resistance. Ford believed that host resistance, rather than avoidance of tsetse, was the basis of trypanosomiasis control. The historical nature of Ford's work requires that a satisfactory evaluation of The Role of the Trypanosomiases make use of historical, as well as scientific, data. The evidence of trypanosomiasis and cattle-keeping from one region of north-eastern Tanzania supports Ford and suggests that other explanations of trypanosomiasis control are inadequate. The Tanzanian evidence shows that precolonial societies coexisted with, but could not avoid, tsetse. They could not eradicate tsetse because scarcity of water prevented permanent occupation of large areas. Tsetse and trypanosomiasis did not prevent cattle-keeping, but helped to keep the region's cattle population low and confined it to relatively densely settled neighbourhoods. Social control of trypanosomiasis collapsed during the pre-Second World War period of colonial rule. Economic and political developments were primarily responsible for a series of famines between 1894 and 1934. Famine-induced depopulation allowed steady spread of tsetse and wildlife reservoirs of trypanosomes into formerly cultivated areas which had been free of tsetse before the colonial period.
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Morais, Mayara Castro de, Jucieudo Virgulino de Souza, Carlos da Silva Maia Bezerra Filho, Silvio Santana Dolabella, and Damião Pergentino de Sousa. "Trypanocidal Essential Oils: A Review." Molecules 25, no. 19 (October 6, 2020): 4568. http://dx.doi.org/10.3390/molecules25194568.

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Trypanosomiases are diseases caused by parasitic protozoan trypanosomes of the genus Trypanosoma. In humans, this includes Chagas disease and African trypanosomiasis. There are few therapeutic options, and there is low efficacy to clinical treatment. Therefore, the search for new drugs for the trypanosomiasis is urgent. This review describes studies of the trypanocidal properties of essential oils, an important group of natural products widely found in several tropical countries. Seventy-seven plants were selected from literature for the trypanocidal activity of their essential oils. The main chemical constituents and mechanisms of action are also discussed. In vitro and in vivo experimental data show the therapeutic potential of these natural products for the treatment of infections caused by species of Trypanosoma.
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Pereira, Glaécia AN, Lucianna H. Santos, Steven C. Wang, Luan C. Martins, Filipe S. Villela, Weiting Liao, Marco A. Dessoy, et al. "Benzimidazole inhibitors of the major cysteine protease of Trypanosoma brucei." Future Medicinal Chemistry 11, no. 13 (July 2019): 1537–51. http://dx.doi.org/10.4155/fmc-2018-0523.

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Aim: Limitations in available therapies for trypanosomiases indicate the need for improved medicines. Cysteine proteases cruzain and rhodesain are validated targets for treatment of Chagas disease and human African trypanosomiasis. Previous studies reported a benzimidazole series as potent cruzain inhibitors. Results & methodology: Considering the high similarity between these proteases, we evaluated 40 benzimidazoles against rhodesain. We describe their structure-activity relationships (SAR), revealing trends similar to those observed for cruzain and features that lead to enzyme selectivity. This series comprises noncovalent competitive inhibitors (best Ki = 0.21 μM against rhodesain) and micromolar activity against Trypanosoma brucei brucei. A cheminformatics analysis confirms scaffold novelty, and the inhibitors described have favorable predicted physicochemical properties. Conclusion: Our results support this series as a starting point for new human African trypanosomiasis medicines.
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Kirchhoff, L. V. "Trypanosomiasis." Current Opinion in Infectious Diseases 2, no. 5 (October 1989): 639–43. http://dx.doi.org/10.1097/00001432-198910000-00005.

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Kirchhoff, Louis V. "Trypanosomiasis." Current Opinion in Infectious Diseases 3, no. 3 (June 1990): 414–19. http://dx.doi.org/10.1097/00001432-199006000-00017.

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Taverne, J. "Trypanosomiasis." Trends in Parasitology 18, no. 4 (April 2002): 189. http://dx.doi.org/10.1016/s1471-4922(02)02274-2.

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Bentivoglio, M. "Trypanosomiasis." Journal of the Neurological Sciences 357 (October 2015): e465-e466. http://dx.doi.org/10.1016/j.jns.2015.09.175.

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Chimelli, Leila, and Francesco Scaravilli. "Trypanosomiasis." Brain Pathology 7, no. 1 (January 1997): 599–611. http://dx.doi.org/10.1111/j.1750-3639.1997.tb01077.x.

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VICKERMAN, KEITH. "The Trypanosomiases (ed. Maudlin, I., Holmes, P. H. & Miles, M. A.), pp. 624. International CABI Publishing, UK, 2004. ISBN 0 85199 475 X. £99.50 (US$185.00)." Parasitology 131, no. 3 (August 16, 2005): 436–37. http://dx.doi.org/10.1017/s0031182005238581.

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Back in the early 1960s, when the curtain was falling on British colonial administration in Africa, the newly-created Ministry of Overseas Development decided to gather together for posterity the expertise and experience of authorities on tsetse and trypanosomiasis control. Weighing in at three and a half pounds, the resulting publication, ‘The African Trypanosomiases’ edited by Colonel Hugh Mulligan and published in 1969, has since been a baseline not only for investigators in the field but also for pure scientists working on related problems at the laboratory bench. The editors of the present volume were inspired by the enormous progress made in trypanosomiasis research over the last thirty years to produce ‘an update of Mulligan’ – so, how do the two books compare? Well, amazingly, their weights are exactly the same – but content and coverage are, as might be expected, very different.
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Rojas-Pirela, Maura, Ulrike Kemmerling, Wilfredo Quiñones, Paul A. M. Michels, and Verónica Rojas. "Antimicrobial Peptides (AMPs): Potential Therapeutic Strategy against Trypanosomiases?" Biomolecules 13, no. 4 (March 26, 2023): 599. http://dx.doi.org/10.3390/biom13040599.

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Trypanosomiases are a group of tropical diseases that have devastating health and socio-economic effects worldwide. In humans, these diseases are caused by the pathogenic kinetoplastids Trypanosoma brucei, causing African trypanosomiasis or sleeping sickness, and Trypanosoma cruzi, causing American trypanosomiasis or Chagas disease. Currently, these diseases lack effective treatment. This is attributed to the high toxicity and limited trypanocidal activity of registered drugs, as well as resistance development and difficulties in their administration. All this has prompted the search for new compounds that can serve as the basis for the development of treatment of these diseases. Antimicrobial peptides (AMPs) are small peptides synthesized by both prokaryotes and (unicellular and multicellular) eukaryotes, where they fulfill functions related to competition strategy with other organisms and immune defense. These AMPs can bind and induce perturbation in cell membranes, leading to permeation of molecules, alteration of morphology, disruption of cellular homeostasis, and activation of cell death. These peptides have activity against various pathogenic microorganisms, including parasitic protists. Therefore, they are being considered for new therapeutic strategies to treat some parasitic diseases. In this review, we analyze AMPs as therapeutic alternatives for the treatment of trypanosomiases, emphasizing their possible application as possible candidates for the development of future natural anti-trypanosome drugs.
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Dissertations / Theses on the topic "Trypanosomiasis"

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Rossi, B. C. "Macrophage function in African trypanosomiasis." Thesis, Brunel University, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.373784.

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Milligan, Paul. "Population dynamics of African trypanosomiasis." Thesis, University of Salford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306017.

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Smuts, Celia Margaretha. "Development of tools to improve the detection of Trypanoma evansi in Australia /." Murdoch University Digital Theses Program, 2009. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20090709.113425.

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Bailey, Wendi. "The diagnosis of human African trypanosomiasis." Thesis, University of Liverpool, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260319.

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Tchamo, Cesaltina da Conceicao Lopes Menete. "Evaluation of the pathogenicity in goats of Trypanosoma congolense from Matutuine, Mozambique." Pretoria : [s.n.], 2007. http://upetd.up.ac.za/thesis/available/etd-04212008-143822/.

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Ibrahim, Hasan Mohamed Saleh. "New therapeutic strategies against trypanosomiasis and leishmaniasis." Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/1158/.

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Leishmaniasis and African Trypanosomiasis are diseases caused by the Kinetoplastida parasites of Leishmania sp. and Trypanosoma sp. respectively. Control and management of these diseases, which affect a significant number of people in the tropics and subtropical areas of the world, is beset with numerous problems such as drug toxicity, affordability and the emergence and spread of parasites resistance to most of the routinely used drugs. This situation calls for an urgent search for new drugs that would address these concerns. Based on report of excellent antimicrobial activities against other parasites and the possession of other known good values, analogues of choline and curcumin were thoroughly assessed in this study for their potential as antitrypanosomal and antileishmanial drugs. Standard methods such as the Alamar Blue, propidium iodide and direct microscopy methods were used to determine the susceptibility of the parasites to the different analogues. Toxicity tests were performed to determine the effect of these compounds on Human Embryonic Kidney (HEK) cells. The presence of mediated transport of these compounds across the parasite plasma membrane was investigated using the classical uptake technique. In order to investigate the possible mechanism of antiparasitic action of the compounds, this study employed flow cytometry to assess the mitochondrial membrane potential Ym, as well as parameters such as production of reactive oxygen species (ROS), the permeability of the plasma membrane and any effects of the test copounds on the parasite’s cell cycle. Five out of 7 choline compounds tested in this study had EC50 values of 0.13-1.8 µM against T. brucei, 0.14-6.9 µM against L. major, L. mexicana promastigotes and 1.69-12.9 µM against L. mexicana amastigotes. With regard to the curcuminoid compounds, 35 out of 98 tested were observed to exhibit trypanocidal activity better than the original curcumin with EC50 values between 0.05 and 1 µM. Against Leishmania, most of the compounds displayed higher antiparasitic activity than curcumin but lower than observed against trypanosomes. The activity of choline analogues was very similar against L. mexicana and L. major promastigotes (P>0.05), and much higher than against L. mexicana amastigotes. Interestingly, some of the compounds displayed EC50 values below that of pentamidine, the routinely used drug. Assessment of parasite growth pattern in the presence of choline analogues showed that two of the compounds, T1 and MS1, are fast acting, killing the population of BSF T. b. brucei within 8 h with the onset of cell death at 2-4 hours of treatment. In contrast, the other three choline compounds observed to have antiparasitic activities acted more slowly, completely killing the trypanosome population after more than 30 hours of incubation. However, all the choline compounds appeared to rapidly inhibit trypanosome proliferation. The choline compounds exhibited low toxic effects against HEK cell line T29, with the selectivity index (S.I.) being high for some of the compounds. The curcumin compounds, too, were observed to have generally similar or lower toxicity against the human cells than the parent curcumin compound (AS-HK001), which in itself is not considered toxic and routinely used in food. Investigations on the toxicological and pharmacological effects of the curcumin compounds on the survival and the glutathione and protein content of primary murine hepatocytes showed no significant difference between hepatocyte cells treated with curcuminoid compounds AS-HK001, AS-HK009, and AS-HK014 compared with controls. We also investigated how choline and its analogues enter the trypanmosome. Evidence gathered in this study strongly suggests that unlike in Leishmania species and Plasmodium, choline transporters are not expressed in the bloodstream form of T. b. brucei. It was also conclusively shown that the P2, high affinity pentamidine transporter (HAPT) and low affinity pentamidine transporter (LAPT) do not play any significant role in the uptake of this compound. Lacking radiolabeled forms of the choline analogues, this study could not identify a definitive route of uptake of this class of compounds into the parasite. Analysis of cell cycle progression, by flow cytometry, showed trypanosomes in the G1, S, and G2/M stages. Curcuminoids do not appear to cause any important changes in the proportion of cells in G1, S or G2/M phase of the cell cycle. Cells exposed to various concentrations of some curcumin compounds, such as AS-HK014 and AS-HK096, showed a rapid increase in cell permeability, reaching between 80% and 90% in 4 hours. The permeability was observed to increase with increasing drug concentration and/or the incubation time. Investigations of cell membrane permeability also showed that choline analogues caused plasma membrane defects which could probably lead to cell death. With regard to the effect of the compound on mitochondrial membrane potential Ym the dicationic choline compounds, including M38, G25, T4 and MS1, were observed to have pronounced effects on Ym with an onset as early as 8 h of contact and we believe the mitochondria could be the main target of these compounds rather than indicating the induction of apoptosis, as the action of the test compounds was not associated with the production of reactive oxygen species. Indeed, both choline and curcumin analogues reduced the production of reactive oxygen species in T. b. brucei cultures. Furthermore, there were no major defects in choline phospholipid metabolism upon treatment with the choline compounds, suggesting that phospholipid metabolism is not the target of the anti-trypanocidal activity of these compounds. Preliminary results with infected ICR mice infected with T. b. brucei did not reveal significant in vivo activity of the three curcumin compounds on blood parasitemia when they were injected intra-peritoneally with two doses of 50 mg/kg body weight. With reference to evidence obtained in this study, it can firmly be concluded that analogues of choline and curcumin display highly promising antiparasitic activities and are generally non-toxic to human cells. Information provided in this thesis could therefore assist in the further development of these classes of compounds as lead compounds against kinetoplastid diseases. We strongly recommend that further investigation be carried out to understand the full mechanism of action of these compounds in order to facilitate this strategy.
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Nyasulu, Yohane. "The study of human trypanosomiasis in Malawi." Thesis, University of Salford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304724.

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Isobe, Hiroyuki. "Medizin und Kolonialgesellschaft : die Bekämpfung der Schlafkrankheit in den deutschen "Schutzgebieten" vor dem Ersten Weltkrieg /." Berlin : Lit, 2009. http://opac.nebis.ch/cgi-bin/showAbstract.pl?u20=9783825816032.

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Matemba, Lucas E. "Epidemiology of human African trypanosomiasis in western Tanzania." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/24915.

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This thesis started by reviewing the existing sleeping sickness historical records in Tanzania with the aim of exploring the evidence for the existence of Trypanosoma brucei gambiense in Tanzania. Findings from the available historical data did not provide sufficient evidence for the existence of T. b. gambiense sleeping sickness in Tanzania.
The thesis further estimated under-reporting of T. b. rhodesiense in endemic areas of Tanzania using an established model. Using data from a 2000-2004 outbreak of T. b. rhodesiense in Urambo, the model predicts 46% underreporting. All unreported cases were assumed to be undetected deaths as sleeping sickness is invariable fatal if left untreated. These underreporting findings were then used to recalibrate the burden of T. b. rhodesiense (using Disability-Adjusted Life Years – DALYs), as a metric. The burden imposed to rural communities by rhodesiense sleeping sickness is high. The costs of hospitalization are very high considering the long duration of hospital stay (26 days mean hospital stay) for sleeping sickness patients. Finally the thesis investigated spatial and behavioural risk factors for T. b. rhodesiense sleeping sickness in Urambo district, through a matched case control study both at the village and within village scales. Statistically significant cluster was observed at the village level (P = 0.001). However there was no significant spatial association in an individual village’s analysis. There was an increased risk of sleeping sickness in homesteads with a previous history of the disease (P < 0.001). Presence of wild animals in the villages (P<0.001) and forest visits (P = 0.001) were also significantly associated with sleeping sickness in the district.
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Stebeck, Caroline Elizabeth. "The identification and characterization of two unique membrane-associated molecules of African trypanosomes." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1996. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq21950.pdf.

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Books on the topic "Trypanosomiasis"

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A, Newton B., ed. Trypanosomiasis. London: Churchill Livingstone for the British Council, 1985.

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Tyler, Kevin M., and Michael A. Miles, eds. American Trypanosomiasis. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9206-2.

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Magez, Stefan, and Magdalena Radwanska, eds. Trypanosomes and Trypanosomiasis. Vienna: Springer Vienna, 2014. http://dx.doi.org/10.1007/978-3-7091-1556-5.

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G, Hide, ed. Trypanosomiasis and leishmaniasis: Biology and control. Wallingofrd, Oxon, UK: CAB International, 1997.

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R, Tizard Ian, ed. Immunology and pathogenesis of trypanosomiasis. Boca Raton, Fla: CRC Press, 1985.

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1943-, Maudlin Ian, Holmes P. H, and Miles Michael A, eds. The trypanosomiases. Wallingford, Oxfordshire, UK: CABI Pub., 2004.

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Desta, Abeba. Trypanosomiasis and tsetse flies (1907-1979) =: Les trypanosomiases et les glossines (1907-1979). Addis Ababa, Ethiopia: Documentation Centre, International Livestock Centre for Africa, 1988.

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Rossi, Bartira Consini. Macrophage function in African trypanosomiasis. Uxbridge: Brunel University, 1986.

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Milligan, Paul. Population dynamics of African Trypanosomiasis. Salford: University of Salford, 1991.

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Jordan, Anthony M. Trypanosomiasis control and African rural development. London: Longman, 1986.

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Book chapters on the topic "Trypanosomiasis"

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O’Hara, James E., Igor UsUpensky, N. J. Bostanian, John L. Capinera, Reg Chapman, Carl S. Barfield, Marilyn E. Swisher, et al. "Trypanosomiasis." In Encyclopedia of Entomology, 3949. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_2580.

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Carod-Artal, Francisco Javier. "Trypanosomiasis." In International Neurology, 290–92. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444317008.ch81.

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Bittencourt, A. L., and T. G. Ashworth. "Trypanosomiasis." In Tropical Pathology, 653–704. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-57863-2_15.

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Carod-Artal, Francisco Javier. "Trypanosomiasis." In International Neurology, 272–75. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118777329.ch72.

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Eltorai, Ibrahim M. "Trypanosomiasis." In Rare Diseases and Syndromes of the Spinal Cord, 283–86. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-45147-3_86.

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Kaya, Mahmut. "Trypanosomiasis." In Uveitis, 225–28. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-52974-1_49.

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Gooch, Jan W. "Trypanosomiasis." In Encyclopedic Dictionary of Polymers, 930. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_15025.

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Warren, Kenneth S., and Adel A. F. Mahmoud. "American Trypanosomiasis." In Geographic Medicine for the Practitioner, 106–11. New York, NY: Springer New York, 1985. http://dx.doi.org/10.1007/978-1-4613-8578-3_16.

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Long, E. G. "African Trypanosomiasis." In Laboratory Diagnosis of Infectious Diseases, 731–38. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3898-0_75.

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Mehlhorn, Heinz. "African Trypanosomiasis." In Encyclopedia of Parasitology, 68. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_84.

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Conference papers on the topic "Trypanosomiasis"

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W. Bradosty, Sarwan, Ahmad K. Maigari, Nasir T. Dabo, and Salisu Ibrahim. "Application of Body Condition Scorings to Effective Detection of African Trypanosomiasis in Camels and Cattle." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.709.

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Trypanosomiasis is a parasitic disease that is transmitted by tsetse flies. However, because of the limitation of conventional parasitological methods, conclusive epidemiological inferences on trypanosomiasis is challenging, leaving a high proportions of the disease to remain undetected which leads to difficulty in monitoring and strategic control. The present study therefore, employed the use of molecular methods to detect trypanosomes in trade camels and cattle, along line analysis of their body condition scores (BCS). Results of the study indicated that, all the infected camels and majority of the infected cattle had poor BCS. The average packed cell volume (PCV) of infected animals was lower than the average PCV of uninfected animals. Findings from this study revealed an infection rate of 48.75% with the most frequently encountered species being Trypanosoma vivax (18.75%), followed by T. brucei (12.50%), T. congolense (8.75%), T. evansi (6.25%) and mixed infection involving T. brucei and T. congolense (2.50%). Conclusively, animals with poor BCS are more susceptible suggesting that, the use of BCS may improve the quality of evaluation of trypanosomiasis in animals, especially for large scale epidemiological study.
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"Targeting peroxisomal transport in trypanosoma." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.566.

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Human infection with Trypanosoma parasites (Chagas disease and Human African Trypanosomiasis) affects around 10 million people worldwide resulting in life-threatening disease. Treatment options are limited to historic drugs characterized by significant side effects and decreasing efficacy while new drug development efforts are largely neglected. Here, we review drug discovery effort in human trypanosomiasis undertaken in academia. Peroxisomal (Pex) transport system was validated as a target in Chagas disease and a number of compounds were delivered which have shown promising results in animal experiments. Future perspectives of exploring the Pex system in anti-trypanosoma drug development are discussed.
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Vahekeni, N., M. Neto Pedro, K. Kayimbo Malilo, P. van Eeuwijk, P. Mäser, G. João Pedro, J. Théophile, E. Wolfram, E. da Costa, and J. Falquet. "Use of herbal medicine in the management of trypanosomiasis in Angola." In GA 2017 – Book of Abstracts. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1608565.

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"Antimalarial, Anti-trypanosomiasis, Anti-HIV and Cytotoxicity Studies of Some Ferrocenyl Schiff bases." In Nov. 27-28, 2017 South Africa. EARES, 2017. http://dx.doi.org/10.17758/eares.eap1117039.

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MADSEN, T., D. I. WALLACE, and N. ZUPAN. "SEASONAL FLUCTUATION IN TSETSE FLY POPULATIONS AND HUMAN AFRICAN TRYPANOSOMIASIS: A MATHEMATICAL MODEL." In International Symposium on Mathematical and Computational Biology. WORLD SCIENTIFIC, 2013. http://dx.doi.org/10.1142/9789814520829_0004.

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Melo, Ana C. A. "Prospecting of olfactory proteins from the American trypanosomiasis vector Rhodnius prolixus(Heteroptera: Reduviidae)." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.105298.

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Dardonville, Christophe, Francisco José Fueyo González, Carolina Izquierdo García, Teresa Díaz Ayuga, Godwin Ebiloma, Emmanuel Balogun, Kiyoshi Kita, and Harry de Koning. "Targeting the Trypanosome Alternative Oxidase (TAO) as Promising Chemotherapeutic Approach for African Trypanosomiasis." In 3rd International Electronic Conference on Medicinal Chemistry. Basel, Switzerland: MDPI, 2017. http://dx.doi.org/10.3390/ecmc-3-04641.

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Al-Hakak, Zahra M. "Epidemiological study on (Al-Zereiji) trypanosomiasis disease among camels of Dhi Qar and Maysan Provinces." In 2ND INTERNATIONAL CONFERENCE OF MATHEMATICS, APPLIED SCIENCES, INFORMATION AND COMMUNICATION TECHNOLOGY. AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0161691.

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Maseleno, Andino, Md Mahmud Hasan, Norjaidi Tuah, Fauzi, and Muhammad Muslihudin. "Fuzzy Logic and Dempster-Shafer belief theory to detect the risk of disease spreading of African Trypanosomiasis." In 2015 Fifth International Conference on Digital Information Processing and Communications (ICDIPC). IEEE, 2015. http://dx.doi.org/10.1109/icdipc.2015.7323022.

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Marcelino Pacheco Neto, Joao, and Otavio Noura Teixeira. "Reconhecimento de Padrões em Imagens de Triatomíneos Usando Redes Neurais Artificiais com Algoritmo Backpropagation." In Computer on the Beach. Itajaí: Universidade do Vale do Itajaí, 2020. http://dx.doi.org/10.14210/cotb.v11n1.p455-460.

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Abstract:
Chagas disease, also known as American trypanosomiasis isone of the consequences of the human infection caused by theflagellate protozoan called Trypanosoma cruzi transmittedby the barbeiro of the subfamily Triatominae known as triatomines.In the Lower Tocantins region of the state of Para,three genera of barbers transmitting the disease are found.Searching for a way to automate the manual recognition process,this work aimed to implement a Model of Recognitionand Classification of Images of barbers found in the LowerTocantins region in order to recognize the genus of the insectthrough the use of Artificial Neural Networks PerceptronMulti-layered and performing training with Backpropagationalgorithm, helping to identify the transmitters. In themiddle of this recognition, the Digital Image Processing isperformed to extract important characteristics relevant to theclassification. This entire process is performed in MATLABsoftware through scripts and the creation of the ArtificialNeural Network in the toolbox called Pattern RecognitionApp.
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Reports on the topic "Trypanosomiasis"

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Silas Lendzele, Sevidzem. A nationwide study of Stomoxys (Diptera: Muscidae), potential mechanical vector of animal trypanosomiasis in Cameroon. Peeref, April 2023. http://dx.doi.org/10.54985/peeref.2304p8166036.

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Clarkson, Allen B., and Jr. Development of a New Chemotherapy for Human African Trypanosomiasis by Using an Animal Model: Suramin with DL-Alpha-Difluoromethylornithine. Fort Belvoir, VA: Defense Technical Information Center, September 1989. http://dx.doi.org/10.21236/ada237231.

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