Relevant bibliographies by topics / Transplantation immunology; Cellular rejection

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  1. Journal articles
  2. Dissertations / Theses
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  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Transplantation immunology; Cellular rejection'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Transplantation immunology; Cellular rejection"

1

Korsgren, Olle. "Acute cellular xenograft rejection." Xenotransplantation 4, no. 1 (February 1997): 11–19. http://dx.doi.org/10.1111/j.1399-3089.1997.tb00159.x.

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Bolton, E. M., J. A. Gracie, J. D. Briggs, J. Kampinga, and J. A. Bradley. "Cellular requirements for renal allograft rejection in the athymic nude rat." Journal of Experimental Medicine 169, no. 6 (June 1, 1989): 1931–46. http://dx.doi.org/10.1084/jem.169.6.1931.

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This study has examined the ability of adoptively transferred CD4+ and CD8+ T cells to mediate rejection of a fully allogeneic DA renal graft in the PVG nude rat. Transfer, at the time of transplantation, of naive CD4+ T cells caused rapid graft rejection and primed CD4+ cells were several times more potent. In contrast, naive or specifically sensitized CD8+ cells were entirely ineffective at mediating renal allograft rejection. Whereas nonrejecting grafts showed only a mild cellular infiltrate, rejecting grafts in CD4+ reconstituted animals showed a substantial infiltrate and many of the infiltrating cells had a phenotype (MRC OX8+, MRC OX19-), consistent with NK cells. Experiments using a mAb (HIS 41) against an allotypic determinant of the leukocyte common antigen confirmed that the majority (greater than 80%) of the cellular infiltrate in rejecting grafts derived from the host rather than from the CD4+ inoculum. Infiltrating mononuclear cells, obtained from rejecting allografts 7 d after transplantation in CD4+-injected PVG nude hosts, showed high levels of in vitro cytotoxicity against not only kidney donor strain Con A blasts but also third-party allogeneic Con A blasts, as well as against both NK and LAK susceptible targets. When splenocytes from nontransplanted nude PVG rats were tested in vitro they also demonstrated high levels of lytic activity against both NK and LAK susceptible targets as well as allogeneic Con A blasts, which were not susceptible to lysis by spleen cells from euthymic rats. These findings suggest that injected CD4+ cells may cause renal allograft rejection by the recruitment of extrathymically derived, widely alloreactive cells into the kidney in this model of graft rejection.
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Pinchuk, A. V., R. V. Storozhev, I. V. Dmitriev, N. V. Shmarina, G. A. Nefedova, R. Sh Muslimov, and Yu S. Teterin. "Cellular rejection of pancreaticoduodenal graft." Russian Journal of Transplantology and Artificial Organs 20, no. 3 (September 17, 2018): 80–86. http://dx.doi.org/10.15825/1995-1191-2018-3-80-86.

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Aim. The evaluation of donor’s duodenum histological examination in diagnosis of pancreaticoduodenal graft rejection.Materials and methods. The 35-yo patient with terminal diabetic nephropathy undergone simultaneous retroperitoneal kidney and pancreas transplantation with enteric exocrine drainage of the graft via inter-duodenal anastomosis. When performing the esophagogastroduodenoscopy 2 years posttransplant we implemented histologic examination of the duodenum of the graft.Results. We diagnosed and verified severe cellular rejection of pancreaticoduodenal graft. Successful etiopathogenetic treatment of acute rejection of the graft (pulse therapy with glucocorticoids) was performed.Discussion. The diagnostic value of donor’s duodenum morphological examination in the diagnosis of pancreaticoduodenal graft rejection, the efficacy of anti-rejection treatment were performed in this case.
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Mandel, T. E., J. Kovarik, M. Koulmanda, and H. M. Georgiou. "Cellular rejection of fetal pancreas grafts: Differences between alio- and xenograft rejection." Xenotransplantation 4, no. 1 (February 1997): 2–10. http://dx.doi.org/10.1111/j.1399-3089.1997.tb00158.x.

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Sindhi, Rakesh, Chethan Ashokkumar, and Brandon W. Higgs. "Cellular alloresponses for rejection-risk assessment after pediatric transplantation." Current Opinion in Organ Transplantation 16, no. 5 (October 2011): 515–21. http://dx.doi.org/10.1097/mot.0b013e32834a94e3.

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Thude, Hansjörg, Petra Tiede, Martina Sterneck, Sven Peine, Björn Nashan, and Martina Koch. "CD28 gene polymorphisms and acute cellular rejection after liver transplantation." Human Immunology 81, no. 12 (December 2020): 675–78. http://dx.doi.org/10.1016/j.humimm.2020.10.002.

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7

Fryer, Jonathan P., Joseph R. Leventhal, Agustin P. Dalmasso, Sally Chen, Pamela A. Simone, Jose Jessurun, Lin Hong Sun, Nancy L. Reinsmoen, and Arthur J. Matas. "Cellular rejection in discordant xenografts when hyperacute rejection is prevented: analysis using adoptive and passive transfer." Transplant Immunology 2, no. 2 (June 1994): 87–93. http://dx.doi.org/10.1016/0966-3274(94)90033-7.

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8

Koutsokera, Angela, Liran Levy, Prodipto Pal, Ani Orchanian-Cheff, and Tereza Martinu. "Acute Cellular Rejection: Is It Still Relevant?" Seminars in Respiratory and Critical Care Medicine 39, no. 02 (March 26, 2018): 181–98. http://dx.doi.org/10.1055/s-0037-1617424.

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AbstractDespite significant progress in the field of transplant immunology, acute cellular rejection (ACR) remains a very frequent complication after lung transplantation (LTx), with almost 30% of LTx recipients experiencing at least one episode of treated ACR during the first year of follow-up. Most episodes respond to the first-line immunosuppressive treatment and are rarely a direct cause of death. However, the association of ACR with later adverse outcomes, such as chronic lung allograft dysfunction, bronchial stricture, and infectious complications associated with the intensification of immunosuppression, negatively impacts long-term survival. The burden imposed on patients and health-care resources is even higher in cases of refractory or recurrent ACR, which accelerates lung function decline. Although important laboratory and clinical research conducted over the last two decades has improved our understanding of the mechanisms underlying ACR, there are still many uncertainties about the risk factors for ACR, the optimal monitoring strategies, and the prediction of long-term outcomes. These knowledge gaps contribute to the large variability in clinical practice among LTx centers, which renders multicenter studies of ACR challenging. In this review, we summarize current evidence on the epidemiology, pathogenesis, and risk factors of ACR. We describe diagnostic and therapeutic approaches that are currently used in the clinical practice and also review promising diagnostic tools that are under investigation. Associations between ACR and other adverse outcomes of LTx are examined. Finally, within each topic of discussion, we highlight the main areas of controversy and opportunities for future research.
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Kumar, Senthil, Nihar Mohapatra, Deeplaxmi Purushottam Borle, Ashok Choudhury, Shashwat Sarin, and Ekta Gupta. "Non invasive diagnosis of acute cellular rejection after liver transplantation – Current opinion." Transplant Immunology 47 (April 2018): 1–9. http://dx.doi.org/10.1016/j.trim.2018.02.002.

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Keßler, B., M. Kurome, N. Klymiuk, A. Wünsch, J. Seissler, and E. Wolf. "New transgenic pigs for xenotransplantation, part 2: Strategies to overcome cellular rejection." Xenotransplantation 18, no. 1 (January 2011): 65. http://dx.doi.org/10.1111/j.1399-3089.2010.00607_7.x.

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Dissertations / Theses on the topic "Transplantation immunology; Cellular rejection"

1

McLean, Adam George. "Patterns of graft infiltration and cytokine gene expression during the first ten days of kidney transplantation." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390513.

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Engstrand, Mats. "Cellular Immune Responses to Allografts and Cytomegalovirus." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3441.

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Rajasekar, Magaral R. "Mechanisms of tolerance induction in experimental renal transplantation." Thesis, University of Newcastle Upon Tyne, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295540.

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Duguid, I. G. M. "Prevention of corneal graft rejection with monoclonal antibodies." Thesis, University of Aberdeen, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387460.

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This thesis aims to place corneal allograft rejection in the context of general transplantation immunology, examine the role of lymphocyte subsets in the rejection process and consider the potential application of monoclonal antibody therapy in clinical corneal graft rejection. The literature relating to the current clinical practice of corneal grafting, with particular reference to corneal allograft rejection, is reviewed in chapter 1 to present the extent of the problem. Chapter 2 then reviews the mechanisms of allograft rejection from the literature of transplantation immunology, much of which has arisen from studies of kidney, heart, pancreatic islets and liver in animal models. The materials and methods are described in detail in chapter 3, and only the relevant experimental design is detailed in the Materials and Methods sections of the succeeding chapters. The experimental mouse model of transplanting corneal tissue into the renal subcapsular is evaluated in chapter 4, demonstrating that isografts survive indefinitely whereas allografts are rejected typically by 30 days. Pretransplant sensitisation decreased allograft survival time to 10 days. Immunohistochemistry demonstrated the presence of CD4+ and CD8+ lymphocytes and macrophages at the rejection site. Heterotopic corneal graft recipients were then treated with various monoclonal antibody regimes. Chapter 5 demonstrates that allograft survival can be increased by either anti-CD4 or anti-CD8 therapy, providing near total depletion of the respective lymphocyte subset is achieved. Xenograft rejection is shown to depend on mainly CD4+ lymphocytes in chapter 6, with no benefit being found of depleting the CD8+ subset in addition. A mild immunosuppressive effect of anti-Vβ8 monoclonal antibody is demonstrated and discussed in chapter 7. The final chapter discusses these results in the light of recent, related work in other transplant systems, and presents a case for a trial of intracameral pan-T-cell monoclonal antibody treatment.
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Bathgate, Andrew John. "The pre-transplant prediction of acute cellular rejection following liver transplantation." Thesis, University of Edinburgh, 2000. http://hdl.handle.net/1842/22544.

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The aim of this thesis was to investigate the effect of various parameters in patients with chronic liver disease pretransplant which may influence the occurrence of acute rejection post transplant. This may be useful in tailoring immunosuppression to avoid adverse effects in patients less likely to develop acute rejection. The role of cytokines in acute rejection is not clear but animal and human studies had suggested that tumour necrosis factor alpha (TNF-a) played some role. Polymorphisms in the genes encoding TNFa, interleukin 10 and transforming growth factor beta1 (TGFb1) which influence in vitro production of cytokines were examined in transplant patients. This showed an increase in the TNFa 2 polymorphism at position -308 in patients with acute rejection but no association with IL-10 or TGFb1 polymorphisms. Pretransplant levels of TNFa and IL-10 were measured following stimulation of peripheral blood mononuclear cells with lipopolysaccharide from patients with chronic liver disease. PBMC were preincubated with different immunosupressants. There was increased production of stimulated TNFa pretransplant in patients who went on to develop acute rejection. No relationship was found between IL-10 production and acute rejection. There were differences in the effects of tacrolimus, cyclosporin and dexamethasone on the production of both cytokines. The pretransplant immune status of patients was assessed by contact sensitisation to diphenylcyclopropenone (DPC). This demonstrated that patients unable to mount an immune response to DPC did not require treatment for acute rejection following liver transplantation. It also demonstrated a correlation between the strength of reaction to DPC and the severity of acute rejection.
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O'Flaherty, Emmett Nathay. "Evaluation of a role for FAS ligand in transplantation." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311109.

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Rosenblum, Joshua Michael. "Novel Roles for Chemokines in Acute Cardiac Allograft Rejection." Case Western Reserve University School of Graduate Studies / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=case1244063137.

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Sleater, Michelle Leigh. "Cellular and molecular effector mechanisms of islet allograft rejection /." Connect to full text via ProQuest. IP filtered, 2006.

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Thesis (Ph.D. in Immunology) -- University of Colorado at Denver and Health Sciences Center, 2006.
Typescript. Includes bibliographical references (leaves 151-168). Free to UCDHSC affiliates. Online version available via ProQuest Digital Dissertations;
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MacEachern, Mary Christina. "Indirect presentation in allograft rejection and the potential for immune intervention." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246249.

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Salah, Adeeb Ahmed Kassim. "Application of Complement Component 4d Immunohistochemistry to ABO-Compatible and ABO-Incompatible Liver Transplantation." Kyoto University, 2015. http://hdl.handle.net/2433/199180.

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Books on the topic "Transplantation immunology; Cellular rejection"

1

Hyperacute xenograft rejection. New York: Springer-Verlag, 1995.

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2

Kelso, Anne. Cytokines in transplantation. Austin: R.G. Landes, 1996.

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3

Thayer, Meryman Harold, ed. Transplantation: Approaches to graft rejection : proceedings of the XVIII Annual Scientific Symposium of the American Red Cross, Washington, DC, April 28-30, 1986. New York: Liss, 1986.

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4

Alexander, J. Wesley. Transplantation tolerance induction. New York: Springer, 1996.

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5

Van den Elsen, Peter J., 1951-. The human T-cell receptor repertoire and transplantation. New York: Springer Verlag, 1995.

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6

L, Touraine J., Fondation Marcel Mérieux, and Université Claude Bernard, eds. Rejection and tolerance: Proceedings of the 25th Conference on Transplantation and Clinical Immunology, 24-26 May 1993. Dordrecht: Kluwer Academic Publishers, 1994.

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7

Takumi, Takeuchi, ed. The Th1, Th2 paradigm and transplantation tolerance. Austin: R.G. Landes, 1994.

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Conference on Transplantation and Clinical Immunology (28th 1996). Late graft loss: Proceedings of the 28th Conference on Transplantation and Clinical Immunology, 3-5 June, 1996. Dordrecht: Kluwer Academic Publishers, 1997.

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Conference on Transplantation and Clinical Immunology (29th 1997). Retransplantation: Proceedings of the 29th Conference on Transplantation and Clinical Immunology, 9-11 June 1997. Dordrecht: Kluwer Academic Publishers, 1998.

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Bone marrow transplantation across major genetic barriers. Hackensack, New Jersey [u.a.]: World Scientific, 2010.

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Book chapters on the topic "Transplantation immunology; Cellular rejection"

1

Rose, Marlene L., Sudhir Kushwaha, and Deirdre Cunningham. "Immunology of Cellular and Humoral Rejection after Cardiac Transplantation." In Cardiac Allograft Rejection, 21–47. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1649-1_2.

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Neil, Desley A. H., and Alexandra J. Rice. "Pathology of Acute Cellular Rejection." In The Pathology of Cardiac Transplantation, 199–217. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46386-5_13.

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Havryk, Adrian. "Cellular Rejection: Is it Still Relevant?" In Essentials in Lung Transplantation, 67–77. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90933-2_6.

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Roden, Anja C., and Henry D. Tazelaar. "Pathology of Lung Rejection: Cellular and Humoral Mediated." In Lung Transplantation, 209–30. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91184-7_13.

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Häyry, Pekka. "Cellular and molecular mechanisms of allograft rejection." In Organ Transplantation 1990, 5–9. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3386-9_2.

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Mayo, George L., Andrew M. Posselt, Luis Campos, Barbara C. Deli, Sean P. Mayo, Clyde F. Barker, and Ali Naji. "Induction of Donor-Specific Tolerance by Intrathymic Cellular Transplantation." In Rejection and Tolerance, 227–44. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-0802-7_23.

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Fung, John J., and J. Andrew Bradley. "Clinical Transplantation and the Immunology of Organ Rejection." In Immunology for Surgeons, 155–66. London: Springer London, 2002. http://dx.doi.org/10.1007/978-1-4471-0201-4_8.

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Lease, Erika D., and Ganesh Raghu. "Management of Cellular and Humoral Rejection: Prevention, Diagnosis, and Treatment." In Lung Transplantation, 195–207. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91184-7_12.

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Dadhania, Darshana, Tara K. Sigdel, Thangamani Muthukumar, Choli Hartono, Minnie M. Sarwal, and Manikkam Suthanthiran. "Molecular Characterization of Rejection in Solid Organ Transplantation." In Manual of Molecular and Clinical Laboratory Immunology, 1132–49. Washington, DC, USA: ASM Press, 2016. http://dx.doi.org/10.1128/9781555818722.ch118.

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Renkonen, R., I. Lautenschlager, and P. Häyry. "Cellular and Molecular Mechanism of Rejection and Role of Viral Infection." In The HLA System in Clinical Transplantation, 327–38. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77506-2_25.

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Conference papers on the topic "Transplantation immunology; Cellular rejection"

1

Vasileva, A., D. Abelson, J. Matelski, J. Wu, E. deHaas, Q. Huang, R. Nadj, et al. "Airway Oscillometry for Early Detection of Acute Cellular Rejection Following Lung Transplantation." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7576.

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