Relevant bibliographies by topics / Transmigration

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Transmigration'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Transmigration"

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Sulabha A. Narsapur, Sameer Choudhari, and Shrishal Totad. "Unusual transmigration of canines – report of two cases in a family." RSBO 11, no. 1 (March 30, 2015): 88–92. http://dx.doi.org/10.21726/rsbo.v11i1.823.

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Transmigration is pre-eruptive migration of tooth across the midline. The etiology of this rare anomaly is unknown. Transmigration is largely related to mandibular canines. Although maxillary canine transmigrations are found in the literature, they are still a rare entity. Objective: The aim of the present paper is to report two cases of unusual transmigrations of canines in two immediate members of a family and to report the first case of simultaneous transmigration of maxillary and mandibular canines in an Indian adolescent. Case report: A rare case of simultaneous transmigration of maxillary and mandibular canine in Indian adolescent along with bilateral transmigration of mandibular canines in her father is described here. Conclusion: Transmigration of canines in two immediate members of family needs to be further studied for familial occurrence of transmigration.
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Cuvelier, Susan L., and Kamala D. Patel. "Shear-dependent Eosinophil Transmigration on Interleukin 4–stimulated Endothelial Cells." Journal of Experimental Medicine 194, no. 12 (December 10, 2001): 1699–709. http://dx.doi.org/10.1084/jem.194.12.1699.

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Leukocyte infiltration into inflammatory sites is regulated by the expression of adhesion and activation proteins, yet the role of these proteins in shear-dependent transmigration is poorly understood. We examined eosinophil recruitment on cytokine-stimulated human umbilical vein endothelial cells (HUVECs) under laminar flow conditions. Eosinophils rapidly transmigrated on interleukin (IL)-4–, but not TNF-stimulated HUVECs. Transmigration was shear dependent, with up to 90% of eosinophils transmigrating in the presence of shear and less than 25% of cells transmigrating under static conditions. Eosinophils express CC chemokine receptor CCR3 and are responsive to various CC chemokines. The effects of chemokines are mediated primarily through Gαi, which is pertussis toxin sensitive. Greater than 65% of shear-dependent eosinophil transmigration on IL-4–stimulated HUVECs was blocked by either pertussis toxin or by an anti-CCR3 monoclonal antibody. Using reverse transcription polymerase chain reaction (RT-PCR) and Western blots, we found that IL-4–stimulated HUVECs produce both mRNA and protein for eotaxin-3. Eotaxin-3 was both released by HUVECs and expressed on the endothelial cell surface. Pretreatment of HUVECs with an anti–eotaxin-3 antibody blocked eosinophil transmigration to the same extent as an anti-CCR3 antibody. These results indicate that IL-4–stimulated HUVECs support shear-dependent eosinophil transmigration by upregulating eotaxin-3, and that surface association is critical for the role of eotaxin-3 in transmigration.
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Junaidi, Junaidi. "Transmigration In Jambi Province From The Perspective of Regional Policymakers." Jambura Agribusiness Journal 4, no. 1 (July 22, 2022): 13–22. http://dx.doi.org/10.37046/jaj.v4i1.15429.

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This study aims to analyze the current state of ex-transmigration villages and regional policy makers' perceptions regarding transmigration's existence and sustainability. The study was conducted in Jambi Province. This research uses a descriptive qualitative approach. Primary data collection was carried out through Focus Group Discussions (FGD) with policymakers at the provincial and district levels. According to the study’s results, the implementation of the transmigration program in addition to showing various successes also resulted in a number of negative excesses for the destination areas. These negative excesses are mainly related to the disparity between the transmigration area and its surrounding area, the low leverage of the transmigration area on the surrounding area, and the potential emergence of poverty in the transmigration area. Related to this, future revitalization of the transmigration program must focus on the development of transmigration areas that are functionally related to the surrounding area, based on the expertise required by the local area, and increasing the role of the community and private sector in the diversification transmigrant business patterns.
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Su, Wen-Hong, Hsiun-ing Chen, Ji-ping Huang, and Chauying J. Jen. "Endothelial [Ca2+]i signaling during transmigration of polymorphonuclear leukocytes." Blood 96, no. 12 (December 1, 2000): 3816–22. http://dx.doi.org/10.1182/blood.v96.12.3816.

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Abstract Vascular endothelium plays an important role in regulating the transendothelial migration of polymorphonuclear leukocytes (PMNs). In this study, the intracellular calcium ion ([Ca2+]i) signaling of endothelial cells (ECs) during PMN transmigration was examined at the single-cell level. Human umbilical vein ECs were cultured on a thin layer of collagen gel. The ECs were labeled with fura-2, immersed in formyl-Met-Leu-Phe, and subsequently perfused with fresh buffer to establish a gradient of chemoattractant across the EC monolayer. The entire process of PMN rolling on, adhering to, and transmigrating across the EC monolayer was recorded under both phase-contrast and fluorescence optics. The data showed the following: (1) At high concentration (approximately 3 × 106/mL), both PMN suspension and its supernatant stimulated frequent EC [Ca2+]i elevations across the monolayer; (2) when used at lower concentration (approximately 5 × 105/mL) to avoid the interference of soluble factors, PMN transmigration, but not rolling or adhesion, was accompanied by EC [Ca2+]i elevation; (3) the latter EC [Ca2+]i elevation occurred simultaneously in ECs adjacent to the transmigration site, but not in those that were not in direct contact with the transmigrating PMNs; (4) this EC [Ca2+]i elevation was an initial and required event for PMN transmigration; and (5) PMNs pretreated with 5,5′-dimethyl-1,2-bis(2-aminophenoxy)ethane-N, N, N′, N′-tetraacetic acid transmigrated with the accompanying EC [Ca2+]i elevation, but they became elongated in the collagen gel. In conclusion, PMNs induce adjacent EC [Ca2+]i signaling, which apparently mediates the “gating” step for their subsequent transmigration.
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Su, Wen-Hong, Hsiun-ing Chen, Ji-ping Huang, and Chauying J. Jen. "Endothelial [Ca2+]i signaling during transmigration of polymorphonuclear leukocytes." Blood 96, no. 12 (December 1, 2000): 3816–22. http://dx.doi.org/10.1182/blood.v96.12.3816.h8003816_3816_3822.

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Vascular endothelium plays an important role in regulating the transendothelial migration of polymorphonuclear leukocytes (PMNs). In this study, the intracellular calcium ion ([Ca2+]i) signaling of endothelial cells (ECs) during PMN transmigration was examined at the single-cell level. Human umbilical vein ECs were cultured on a thin layer of collagen gel. The ECs were labeled with fura-2, immersed in formyl-Met-Leu-Phe, and subsequently perfused with fresh buffer to establish a gradient of chemoattractant across the EC monolayer. The entire process of PMN rolling on, adhering to, and transmigrating across the EC monolayer was recorded under both phase-contrast and fluorescence optics. The data showed the following: (1) At high concentration (approximately 3 × 106/mL), both PMN suspension and its supernatant stimulated frequent EC [Ca2+]i elevations across the monolayer; (2) when used at lower concentration (approximately 5 × 105/mL) to avoid the interference of soluble factors, PMN transmigration, but not rolling or adhesion, was accompanied by EC [Ca2+]i elevation; (3) the latter EC [Ca2+]i elevation occurred simultaneously in ECs adjacent to the transmigration site, but not in those that were not in direct contact with the transmigrating PMNs; (4) this EC [Ca2+]i elevation was an initial and required event for PMN transmigration; and (5) PMNs pretreated with 5,5′-dimethyl-1,2-bis(2-aminophenoxy)ethane-N, N, N′, N′-tetraacetic acid transmigrated with the accompanying EC [Ca2+]i elevation, but they became elongated in the collagen gel. In conclusion, PMNs induce adjacent EC [Ca2+]i signaling, which apparently mediates the “gating” step for their subsequent transmigration.
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Shaw, Sunil K., Shuo Ma, Michael B. Kim, Ravi M. Rao, Charles U. Hartman, Richard M. Froio, Lin Yang, et al. "Coordinated Redistribution of Leukocyte LFA-1 and Endothelial Cell ICAM-1 Accompany Neutrophil Transmigration." Journal of Experimental Medicine 200, no. 12 (December 20, 2004): 1571–80. http://dx.doi.org/10.1084/jem.20040965.

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The leukocyte integrin lymphocyte function-associated antigen 1 (LFA-1) and its endothelial ligand intercellular adhesion molecule (ICAM)-1 play an important role in transmigration as demonstrated by in vivo and in vitro models of inflammation. Despite the prominent role, little is known concerning the distribution and dynamic behavior of these adhesion molecules during leukocyte transmigration. Therefore, we examined the spatial and temporal distribution of LFA-1 on neutrophils actively transmigrating tumor necrosis factor-α–activated human umbilical vein endothelial monolayers under shear flow. Upon neutrophil arrest, LFA-1 was evenly distributed. However, once neutrophils initiated transmigration, LFA-1 rapidly redistributed to form a ringlike cluster at the neutrophil–endothelial junctional interface through which transmigration occurred. As transmigration was completed, LFA-1 redistributed to the neutrophil uropod. Endothelial ICAM-1 and JAM-A both colocalized with the ringlike LFA-1 cluster. Further analysis of PMA-stimulated neutrophils, which increase mobility of LFA-1, showed a rapid redistribution of LFA-1 and ICAM-1, but not endothelial JAM-A. Thus, endothelial JAM-A does not appear to contribute to adhesion or transmigration in this system. This is the first demonstration that neutrophil LFA-1 rapidly redistributes to form a ringlike structure that coclusters with endothelial ICAM-1 as the neutrophil transmigrates.
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Cheng, Hu, and Ming Di. "Transmigration." Ploughshares 48, no. 1 (March 2022): 73. http://dx.doi.org/10.1353/plo.2022.0031.

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Indrayani, Nelly, and Supian Ramli. "The Impact of Transmigration on The Development of Christianization in West Pasaman (1953-1980)." Criksetra: Jurnal Pendidikan Sejarah 12, no. 1 (February 27, 2023): 93–107. http://dx.doi.org/10.36706/jc.v12i1.19430.

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Abstract : This study reveals the social history of transmigration's effects on the chirstianization of pasaman. This chirstianization took place in 1953, for the existence of christians who have settled in about the 20th century. These christians came from java, and it's mainly in transmigration resettlement areas. In progress until the end of 1980, chirstian activity look dinamics, so that christian could engage in various activity of life. The study uses historical science research methods of heuristic, critisim, interpretation, and historiography. Studies have found that, the social movement of christian resulted from transmigration trough all walks of life in the pasaman. The christian movement in the pasaman included education, place of worship, youth and art, and socioeconomic society. A propesive action is that without resorting to anarchy in a persuasive or inviting approach. This effrot has brough various froms of infrastructure to all sector of life.Keywords : Impact, Transmigration,Christianization, Pasaman.
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Su, Wen-Hong, Hsiun-ing Chen, and Chauying J. Jen. "Differential movements of VE-cadherin and PECAM-1 during transmigration of polymorphonuclear leukocytes through human umbilical vein endothelium." Blood 100, no. 10 (November 15, 2002): 3597–603. http://dx.doi.org/10.1182/blood-2002-01-0303.

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Most existing evidence regarding junction protein movements during transendothelial migration of leukocytes comes from taking postfixation snap shots of the transendothelial migration process that happens on a cultured endothelial monolayer. In this study, we used junction protein–specific antibodies that did not interfere with the transendothelial migration to examine the real-time movements of vascular endothelial–cadherin (VE-cadherin) and platelet/endothelial cell adhesion molecule-1 (PECAM-1) during transmigration of polymorphonuclear leukocytes (PMNs) either through a cultured endothelial monolayer or through the endothelium of dissected human umbilical vein tissue. In either experimental model system, both junction proteins showed relative movements, not transient disappearance, at the PMN transmigration sites. VE-cadherin moved away to different ends of the transmigration site, whereas PECAM-1 opened to surround the periphery of a transmigrating PMN. Junction proteins usually moved back to their original positions when the PMN transmigration process was completed in less than 2 minutes. The relative positions of some junction proteins might rearrange to form a new interendothelial contour after PMNs had transmigrated through multicellular corners. Although transmigrated PMNs maintained good mobility, they only moved laterally underneath the vascular endothelium instead of deeply into the vascular tissue. In conclusion, our results obtained from using either cultured cells or vascular tissues showed that VE-cadherin–containing adherent junctions were relocated aside, not opened or disrupted, whereas PECAM-1–containing junctions were opened during PMN transendothelial migration.
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Mingé, Jeanine M., and Richard Lee Smith. "Metempsychosis." Departures in Critical Qualitative Research 3, no. 2 (2013): 162–68. http://dx.doi.org/10.1525/dcqr.2014.3.2.162.

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This fusion of photographs and poetry, a form of arts-based inquiry, explores metempsychosis, or the transmigration of the soul, which the artists experienced at Burning Man 2013. Within this ritual space the artists experienced a metempsychosis without a physical death, entering the playa as one spiritual form and leaving as another. The artists hope to create a passageway or opening for readers to experience their own transmigrations.
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Dissertations / Theses on the topic "Transmigration"

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Lam, Siu-wing Cynthia. "Transmigration of Hong Kongers to Canada, 1990-2004." Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36598343.

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Lam, Siu-wing Cynthia, and 林兆泳. "Transmigration of Hong Kongers to Canada, 1990-2004." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B36598343.

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Roulson, Jo-An. "Bone marrow endothelial transmigration of prostate carcinoma cells." Thesis, University of Manchester, 2008. https://www.research.manchester.ac.uk/portal/en/theses/bone-marrow-endothelial-transmigration-of-prostate-carcinoma-cells(997acbf2-bbbc-455b-bb84-b439ffb9f839).html.

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Levang, Patrice. "La terre d'en face : la transmigration en Indonésie /." Paris : Éd. de l'ORSTOM, Institut de recherche scientifique pour le développement en coopération, 1997. http://catalogue.bnf.fr/ark:/12148/cb36198299m.

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Reymond, Nicolas. "Les nectines, une nouvelle famille de molécules d'adhérence : Implication dans la migration trans-endothéliale des leucocytes." Aix-Marseille 2, 2004. http://www.theses.fr/2004AIX22032.

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Thompson, Richard Damian. "Mechanisms of leukocyte transmigration in rat and murine microcirculation." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252277.

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Popple, Amy Lee. "The tumour microenvironment influences antigen specific T cell transmigration." Thesis, University of Nottingham, 2012. http://eprints.nottingham.ac.uk/12584/.

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T cell infiltration into tumours is essential for tumour antigen recognition and tumour cell elimination. The aim of this study was to develop a better understanding of T cell infiltration into tumours, focusing on two opposing arms of an immune response, anti-tumour CD8 Tcells and Regulatory T cells (Tregs). Activated CD4 T helper cells are also of importance but could not be studied due to the time constraints of the project. The effect of T cell signalling at the immunological synapse following interactions between T cells and APCs presenting cognate antigen have been well studied [1]. The endothelium is neither a stereotypical APC nor simply a passive filter barrier for non-cognate infiltrating T cells. The endothelium can activel influence the development of an inflammatory response depending on the functional state of both the endothelium and interacting T cells (resting versus recently activated T cells)and the type of interactions (cognate versus non-cognate). The hypothesis was that recognition of antigens presented in the context of major histocompatibility complex (MHC)molecules by endothelium aids T cell transmigration and hence infiltration into tissues, including into tumours. In this study, the data highlights that high avidity TRP-2 specific CD8 T cell transmigration across murine lung endothelium requires recognition of TRP-2 peptide presented by the endothelium, aiding recruitment of antigen-specific T cells into tissues in the absence of endothelial cell killing. In order for antigen specific T cells to migrate into the tumour, the tumour endothelium therefore needs to present tumour antigens. In addition to CD8 T cells, high numbers of Tregs have been found within tumours but the key mediators for this recruitment remain uncertain. The data shows a novel mechanism for Treg transmigration where cognate antigen-specific recognition of self-peptides was required for transmigration with preferential transmigration of Tregs across syngeneic rather than allogeneic endothelium. Upregulation of major histocompatibility complex (MHC) class II and adhesion molecules, by IFN-γ and TNF-α, together with a gradient of the tumour associated chemokine CXCL12 were also pre-requisites for efficient Treg transmigration. Previous studies have shown that high CXCL12 expression can induce fugetaxis of tumour cells leading to efficient metastatic spread and a poor prognosis. These results would suggest that low levels of CXCL12 and recognition of self peptides presented by self MHC on endothelial cells allows efficient migration of Tregs whereas higher levels of CXCL12 may promote tumour metastases and lead to fugetaxis of the Tregs leading to an even worse prognosis. In conclusion recognition of cognate antigen presented by endothelium enhances antigenspecific transmigration of CD8 and Regulatory T cells. This study therefore reports a novel mechanism for T cell subset infiltration into tumours where high avidity CD8 T cells require recognition of cognate tumour antigen presented on tumour endothelium in the context of MHC class I and conversely regulatory T cell infiltration into tumours depends on the repertoire of self-peptides presented on tumour endothelium in the context of MHC class II. Alteration of antigen presentation or MHC expression on tumour endothelium therefore represents a mechanism whereby T cell infiltration can be altered to re-direct anti-tumour immune responses.
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Evans, Nicholas J. "Aliens en route : European transmigration through Britain, 1836-1914." Thesis, University of Hull, 2006. http://hydra.hull.ac.uk/resources/hull:8092.

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This thesis discusses the agencies, transport systems, and infrastructure that enabled more than 3.15 million Europeans to emigrate to the United States, Canada, and South Africa through Britain between 1836 and 1914. Rather than travelling directly from the European mainland, these transmigrants broke their journeys by travelling to Britain where they boarded another vessel that conveyed them across the Atlantic. The control that Britain exerted over both the short-sea and long-haul passenger routes thus involved was as important to British maritime commerce as similar controls over freight or direct long-haul passenger routes to the far-flung corners of the British Empire. However the crucial significance of the transmigrant business to the British merchant marine has been largely overlooked in recent historiography, and it is this lacuna that the present dissertation seeks to redress. The study is split into three sections. The first part quantifies the patterns of the transmigrant business, answering questions such as: what were the origins of the migrants and what routes did they use to reach Britain? When did they come? Where in Britain did they land, where were they bound, and where did they re-embark? Having charted these issues, the thesis turns in the second section to investigate how the transmigrant business developed and evolved, paying particular attention to the factors that conditioned the market throughout the 78 year period. Finally, the thesis examines the significance of the transmigrant business to British ports serving as conduits for the passenger movement, to the companies involved in transporting the aliens, and to the migrants themselves. By exploring these issues this thesis has made a significant contribution to migrant and maritime historiography in the following ways. First, it has broadened the chronological and geographical focus of migrancy back from the 1880s to the 1830s and stressed Scandinavian as well as Central/East European movements. Second, it has demonstrated how European transmigrants were as important to British shipping companies as were British emigrants seeking to settle in Britain's overseas dominions. Third, immigration to Britain has been incorrectly conceptualised because historians and social commentators fail to take account of the onward movement of aliens arriving in Britain and assume instead that most were permanent settlers. Fourth, the primacy of Britain's maritime links to the United States was more important for the passenger business than has been previously acknowledged. Finally, this study disproves theories by immigrant historians that centres of alien settlement across Northern Britain arose because they were situated along the transmigrant corridor between the Humber and Mersey. In reality many of the trains carrying transmigrants never passed through the towns and cities where large-scale immigration took place. By combining a mixture of global, national and local studies, and a longer chronology, this thesis offers an important intersection of transport and maritime studies that shows how transmigration has been under appreciated by both maritime and migrants historians alike.
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Reyat, Jasmeet Singh. "Regulation of lymphocyte transmigration by ADAM10 and TspanC8 tetraspanins." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6650/.

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The passage of leukocytes across blood vessel walls plays a key role in the immune response to infection in inflammatory conditions. ADAM10 is a ubiquitously expressed molecular scissor that proteolytically cleaves key cell surface proteins including vascular endothelial (VE)-cadherin and transmembrane chemokines. Their shedding by ADAM10 promotes leukocyte transmigration in cell line models, however the precise mechanism behind ADAM I O's involvement is unknown. ADAM10 associates with six different membrane organising tetraspanins (Tspan5/10/14/15/17/33) termed the TspanC8s. These tetraspanins regulate ADAM10 enzymatic maturation and trafficking to the cell surface and emerging evidence indicates that different TspanC8s can promote ADAM10 cleavage of specific substrates. It was hypothesised that ADAM10 promotes leukocyte transmigration by cleaving one of its endothelial substrates and one or more of the TspanC8s could facilitate this process. The aim of this thesis was to test this hypothesis using in vitro leukocyte adhesion assays with primary human leukocytes and human umbilical vein endothelial cells (HUVECs). siRNA knockdown or pharmacological inhibition of ADAM10 on HUVECs impaired the transmigration of lymphocytes, but not neutrophils or monocytes. ADAM10 knockdown/inhibition caused a reduction in VE-cadherin shedding and an increase in VE-cadherin surface expression. Partial knockdown of VE-cadherin, in the presence of ADAM10 knockdown/inhibition, reduced VE-cadherin levels to normal and restored basal lymphocyte transmigration. Systematic knockdown of TspanC8s in HUVECs revealed that the presence of either Tspan5 or Tspan17 was sufficient to maintain basal lymphocyte transmigration and reduced VE-cadherin surface levels. Tspan5 and Tspan17 are functionally uncharacterised, but they are the most highly related TspanC8s by sequence (78% amino acid identity) and may share a common role in lymphocyte transmigration by regulation of ADAM10 and VE-cadherin.
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Wechsung, Katja [Verfasser]. "Transmigration von CD4+ T-Zellen durch Lebersinusendothel / Katja Wechsung." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2014. http://d-nb.info/1052221343/34.

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Books on the topic "Transmigration"

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Barbara, Górska, Puntos Jani Konstantinovski 1961-, Gonway Teresa, and Międzynarodowe Centrum Kultury w Krakowie, eds. Transmigracje =: Transmigration. Kraków: Międzynarodowe Centrum Kultury, 2011.

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Brooks, Ann, and Ruth Simpson. Emotions in Transmigration. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334.

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Nergaard, Siri. Translation and Transmigration. London ; New York : Routledge, 2021. | Series: New perspectives in translation and interpreting studies: Routledge, 2020. http://dx.doi.org/10.4324/9781003141716.

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Sutardi, Winoto, ed. Historiography on transmigration. [Jakarta]: Secretariat General, Dept. of Transmigration, 1987.

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Fasbender, Karl. Selected articles on transmigration, 1987.: Transmigration in East Kalimantan. Bielefeld: Universität Bielefeld, Fakultät für Soziologie, Forschungsschwerpunkt Entwicklungssoziologie, 1988.

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Indonesia. Basic stipulations for transmigration. [Jakarta]: Directorate of Foreign Information Services, Dept. of Information, Repulic of Indonesia, 1996.

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William, Barton. The transmigration of souls. New York: Warner Books, 1996.

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Negeri, Indonesia Departemen Luar, ed. Transmigration: Issues and perspectives. [Jakarta: Dept. of Foreign Affairs, Republic of Indonesia, 1995.

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Indonesia. Direktorat Jenderal Penyiapan Pemukiman., ed. Transmigration project preparation: Proposal for World Bank participation under Transmigration V Loan. [Jakarta]: The Directorate General, 1989.

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Smith, Brian. John Locke, Territory, and Transmigration. Abingdon, Oxon ; New York, NY : Taylor & Francis Group, 2021.: Routledge India, 2020. http://dx.doi.org/10.4324/9780429326547.

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Book chapters on the topic "Transmigration"

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Knox, Shauna. "Subjective Transmigration." In The Black Subaltern, 44–54. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003226802-4.

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Brooks, Ann, and Ruth Simpson. "Introduction: Understanding Emotions in Transmigration in Southeast Asia, Europe and the United States." In Emotions in Transmigration, 1–13. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_1.

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Brooks, Ann, and Ruth Simpson. "The Significance of Emotions in Contemporary Social Theorizing." In Emotions in Transmigration, 14–30. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_2.

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Brooks, Ann, and Ruth Simpson. "Gender, Emotions and Migration in a European Context." In Emotions in Transmigration, 31–51. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_3.

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Brooks, Ann, and Ruth Simpson. "Dirty Work, Identity and Emotions: The Polish Experience." In Emotions in Transmigration, 52–70. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_4.

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Brooks, Ann, and Ruth Simpson. "The Feminization of Migration and Emotions in Transmigration in Southeast Asia." In Emotions in Transmigration, 71–89. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_5.

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Brooks, Ann, and Ruth Simpson. "Agency in the Construction of Emotions in Transmigration in Different Cultural and Work Contexts." In Emotions in Transmigration, 90–106. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_6.

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Brooks, Ann, and Ruth Simpson. "‘Unseen America’: Citizenship and the Politics of Migration in California." In Emotions in Transmigration, 107–28. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_7.

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Brooks, Ann, and Ruth Simpson. "‘California Dreamin”: Transformation and Identity in the Experiences of Migrants into the San Francisco Bay Area." In Emotions in Transmigration, 129–57. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_8.

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Brooks, Ann, and Ruth Simpson. "Conclusion." In Emotions in Transmigration, 158–74. London: Palgrave Macmillan UK, 2013. http://dx.doi.org/10.1057/9781137284334_9.

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Conference papers on the topic "Transmigration"

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Tajiri, Rikiya, and Kiyoshi Tomimatsu. "Japanese text transmigration presentation." In ACM SIGGRAPH 2008 posters. New York, New York, USA: ACM Press, 2008. http://dx.doi.org/10.1145/1400885.1400909.

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2

Xue, Jiaxing, Jean Gao, and Liping Tang. "Phagocyte Transmigration Modeling Using System Dynamic Controls." In 2007 IEEE 7th International Symposium on BioInformatics and BioEngineering. IEEE, 2007. http://dx.doi.org/10.1109/bibe.2007.4375604.

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Xue, Jiaxing, Jean Gao, and Liping Tang. "A hybrid computational model for phagocyte transmigration." In 2008 8th IEEE International Conference on Bioinformatics and BioEngineering. IEEE, 2008. http://dx.doi.org/10.1109/bibe.2008.4696731.

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4

Yulmardi, Yulmardi, and Erfit Erfit. "Analysis of Individual Characteristics, Transmigration Aspects and Transmigrant Employment Structures (Study in ex-Transmigration Villages within Jambi Province)." In First Padang International Conference On Economics Education, Economics, Business and Management, Accounting and Entrepreneurship (PICEEBA 2018). Paris, France: Atlantis Press, 2018. http://dx.doi.org/10.2991/piceeba-18.2018.34.

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5

Zemans, Rachel L., Natalie Briones, Jazalle McClendon, and Gregory P. Downey. "Beta-Catenin Mitigates Epithelial Injury Induced By Neutrophil Transmigration." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4342.

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6

García de Alba, Carolina, Carina Becerril, Silvia Reyes, Moises Selman, and Annie Pardo. "MMP-8 Is A Central Player In Fibrocytes Transmigration." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5270.

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7

Fu, Y., A. M. J. Vandongen, T. Bourouina, W. T. Park, M. Y. Je, J. M. Tsai, D. L. Kwong, and A. Q. Liu. "A study of cancer cell metastasis using microfluidic transmigration device." In 2012 IEEE 25th International Conference on Micro Electro Mechanical Systems (MEMS). IEEE, 2012. http://dx.doi.org/10.1109/memsys.2012.6170300.

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8

Yoshida, Kenji, Shoichi Saito, Koichi Mouri, and Hiroshi Matsuo. "Orthros: A High-Reliability Operating System with Transmigration of Processes." In 2013 IEEE 19th Pacific Rim International Symposium on Dependable Computing (PRDC). IEEE, 2013. http://dx.doi.org/10.1109/prdc.2013.54.

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9

Kang, Mingon, Jean Gao, and Liping Tang. "Nonlinear RANSAC Optimization for Parameter Estimation with Applications to Phagocyte Transmigration." In 2011 Tenth International Conference on Machine Learning and Applications (ICMLA). IEEE, 2011. http://dx.doi.org/10.1109/icmla.2011.104.

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10

Kang, Mingon, Jean X. Gao, and Liping Tang. "Computational modeling of phagocyte transmigration during biomaterial-mediated foreign body responses." In 2010 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2010. http://dx.doi.org/10.1109/bibm.2010.5706638.

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