Log in

Relevant bibliographies by topics / Transient receptor potential receptors

Academic literature on the topic 'Transient receptor potential receptors'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Transient receptor potential receptors.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Transient receptor potential receptors"

1

Shioya, Takanobu, Kazuhiro Sato, Masaaki Sano, and Hiroyuki Watanabe. "Transient receptor potential (TRP) channel and cough." Folia Pharmacologica Japonica 131, no. 6 (2008): 417–22. http://dx.doi.org/10.1254/fpj.131.417.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Zhang, Hongyu, Peter J. Wickley, Sayantani Sinha, Ian N. Bratz, and Derek S. Damron. "Propofol Restores Transient Receptor Potential Vanilloid Receptor Subtype-1 Sensitivity via Activation of Transient Receptor Potential Ankyrin Receptor Subtype-1 in Sensory Neurons." Anesthesiology 114, no. 5 (May 1, 2011): 1169–79. http://dx.doi.org/10.1097/aln.0b013e31820dee67.

Full text

Abstract:

Background Cross talk between peripheral nociceptors belonging to the transient receptor potential vanilloid receptor subtype-1 (TRPV1) and ankyrin subtype-1 (TRPA1) family has been demonstrated recently. Moreover, the intravenous anesthetic propofol has directly activates TRPA1 receptors and indirectly restores sensitivity of TRPV1 receptors in dorsal root ganglion (DRG) sensory neurons. Our objective was to determine the extent to which TRPA1 activation is involved in mediating the propofol-induced restoration of TRPV1 sensitivity. Methods Mouse DRG neurons were isolated by enzymatic dissociation and grown for 24 h. F-11 cells were transfected with complementary DNA for both TRPV1 and TRPA1 or TRPV1 only. The intracellular Ca concentration was measured in individual cells via fluorescence microscopy. After TRPV1 desensitization with capsaicin (100 nM), cells were treated with propofol (1, 5, and 10 μM) alone or with propofol in the presence of the TRPA1 antagonist, HC-030031 (0.5 μM), or the TRPA1 agonist, allyl isothiocyanate (AITC; 100 μM); capsaicin was then reapplied. Results In DRG neurons that contain both TRPV1 and TRPA1, propofol and AITC restored TRPV1 sensitivity. However, in DRG neurons containing only TRPV1 receptors, exposure to propofol or AITC after desensitization did not restore capsaicin-induced TRPV1 sensitivity. Similarly, in F-11 cells transfected with both TRPV1 and TRPA1, propofol and AITC restored TRPV1 sensitivity. However, in F-11 cells transfected with TRPV1 only, neither propofol nor AITC was capable of restoring TRPV1 sensitivity. Conclusions These data demonstrate that propofol restores TRPV1 sensitivity in primary DRG neurons and in cultured F-11 cells transfected with both the TRPV1 and TRPA1 receptors via a TRPA1-dependent process. Propofol's effects on sensory neurons may be clinically important and may contribute to peripheral sensitization to nociceptive stimuli in traumatized tissue.

APA, Harvard, Vancouver, ISO, and other styles

3

White, John P. M., Mario Cibelli, Antonio Rei Fidalgo, Cleoper C. Paule, Faruq Noormohamed, Laszlo Urban, Mervyn Maze, and Istvan Nagy. "Role of Transient Receptor Potential and Acid-sensing Ion Channels in Peripheral Inflammatory Pain." Anesthesiology 112, no. 3 (March 1, 2010): 729–41. http://dx.doi.org/10.1097/aln.0b013e3181ca3179.

Full text

Abstract:

Pain originating in inflammation is the most common pathologic pain condition encountered by the anesthesiologist whether in the context of surgery, its aftermath, or in the practice of pain medicine. Inflammatory agents, released as components of the body's response to peripheral tissue damage or disease, are now known to be collectively capable of activating transient receptor potential vanilloid type 1, transient receptor potential vanilloid type 4, transient receptor potential ankyrin type 1, and acid-sensing ion channels, whereas individual agents may activate only certain of these ion channels. These ionotropic receptors serve many physiologic functions-as, indeed, do many of the inflammagens released in the inflammatory process. Here, we introduce the reader to the role of these ionotropic receptors in mediating peripheral pain in response to inflammation.

APA, Harvard, Vancouver, ISO, and other styles

4

Andrzejewska, Angelika, Klaudia Staszak, Marta Kaczmarek-Ryś, Ryszard Słomski, and Szymon Hryhorowicz. "Understanding cannabinoid receptors: structure and function." Folia Biologica et Oecologica 14 (December 30, 2018): 1–13. http://dx.doi.org/10.1515/fobio-2017-0004.

Full text

Abstract:

The endocannabinoid system (ECS) consists of the endocannabinoids, cannabinoid receptors and the enzymes that synthesize and degrade endocannabinoids. The whole EC system plays an important role in the proper functioning of the central and autonomic nervous system. ECS is involved in the regulation of the body energy and in the functioning of the endocrine system. It can affect on the regulation of emotional states, motoric movement, operations of the endocrine, immune and digestive system. Many of the effects of cannabinoids are mediated by G coupled –protein receptors: CB1, CB2 and GPR55 but also of transient receptor potential channels (TRPs) which not only induce the sensation of pain but also support inflammation via secretion of pro-inflammatory neuropeptides. In this review work we briefly summarize the role and action of cannabinoid receptors CB1 and CB2, protein-coupled receptor 55 (GPR55) and transient receptor potential vanilloid 1 (TRPV1).

APA, Harvard, Vancouver, ISO, and other styles

5

Toschi, Andrea, Giorgia Galiazzo, Andrea Piva, Claudio Tagliavia, Gemma Mazzuoli-Weber, Roberto Chiocchetti, and Ester Grilli. "Cannabinoid and Cannabinoid-Related Receptors in the Myenteric Plexus of the Porcine Ileum." Animals 11, no. 2 (January 21, 2021): 263. http://dx.doi.org/10.3390/ani11020263.

Full text

Abstract:

An important piece of evidence has shown that molecules acting on cannabinoid receptors influence gastrointestinal motility and induce beneficial effects on gastrointestinal inflammation and visceral pain. The aim of this investigation was to immunohistochemically localize the distribution of canonical cannabinoid receptor type 1 (CB1R) and type 2 (CB2R) and the cannabinoid-related receptors transient potential vanilloid receptor 1 (TRPV1), transient potential ankyrin receptor 1 (TRPA1), and serotonin receptor 5-HT1a (5-HT1aR) in the myenteric plexus (MP) of pig ileum. CB1R, TRPV1, TRPA1, and 5-HT1aR were expressed, with different intensities in the cytoplasm of MP neurons. For each receptor, the proportions of the immunoreactive neurons were evaluated using the anti-HuC/HuD antibody. These receptors were also localized on nerve fibers (CB1R, TRPA1), smooth muscle cells of tunica muscularis (CB1R, 5-HT1aR), and endothelial cells of blood vessels (TRPV1, TRPA1, 5-HT1aR). The nerve varicosities were also found to be immunoreactive for both TRPV1 and 5-HT1aR. No immunoreactivity was documented for CB2R. Cannabinoid and cannabinoid-related receptors herein investigated showed a wide distribution in the enteric neurons and nerve fibers of the pig MP. These results could provide an anatomical basis for additional research, supporting the therapeutic use of cannabinoid receptor agonists in relieving motility disorders in porcine enteropathies.

APA, Harvard, Vancouver, ISO, and other styles

6

Manolache, Alexandra, Teodora Stratulat, and Alexandru Babeș. "Modulation of Transient Receptor Potential (TRP) channels by tyrosine phosphorylation." Reviews in Biological and Biomedical Sciences 3, no. 1 (July 4, 2020): 77–87. http://dx.doi.org/10.31178/rbbs.2020.3.1.5.

Full text

Abstract:

Transient Receptor Potential (TRP) channels are a superfamily of polymodal, non-selective receptors, expressed in the nervous system and several other tissues, where they play many physiological or pathological roles. TRP channels are sensitive to a diverse range of stimuli, such as temperature, osmolarity, oxidative stress, external compounds and intracellular signaling molecules. The activity of TRP channels can be modulated by protein phosphorylation, including tyrosine phosphorylation. In this review, we present the studies carried out so far regarding the modulation of TRP channels by tyrosine phosphorylation.

APA, Harvard, Vancouver, ISO, and other styles

7

Ditting, Tilmann, Roland Veelken, and Karl F. Hilgers. "Transient Receptor Potential Vanilloid Type 1 Receptors in Hypertensive Renal Damage." Hypertension 52, no. 2 (August 2008): 213–14. http://dx.doi.org/10.1161/hypertensionaha.108.116129.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Shabir, Saqib, William Cross, Lisa A. Kirkwood, Joanna F. Pearson, Peter A. Appleby, Dawn Walker, Ian Eardley, and Jennifer Southgate. "Functional expression of purinergic P2 receptors and transient receptor potential channels by the human urothelium." American Journal of Physiology-Renal Physiology 305, no. 3 (August 1, 2013): F396—F406. http://dx.doi.org/10.1152/ajprenal.00127.2013.

Full text

Abstract:

In addition to its role as a physical barrier, the urothelium is considered to play an active role in mechanosensation. A key mechanism is the release of transient mediators that activate purinergic P2 receptors and transient receptor potential (TRP) channels to effect changes in intracellular Ca2+. Despite the implied importance of these receptors and channels in urothelial tissue homeostasis and dysfunctional bladder disease, little is known about their functional expression by the human urothelium. To evaluate the expression and function of P2X and P2Y receptors and TRP channels, the human ureter and bladder were used to separate urothelial and stromal tissues for RNA isolation and cell culture. RT-PCR using stringently designed primer sets was used to establish which P2 and TRP species were expressed at the transcript level, and selective agonists/antagonists were used to confirm functional expression by monitoring changes in intracellular Ca2+ and in a scratch repair assay. The results confirmed the functional expression of P2Y4 receptors and excluded nonexpressed receptors/channels (P2X1, P2X3, P2X6, P2Y6, P2Y11, TRPV5, and TRPM8), while a dearth of specific agonists confounded the functional validation of expressed P2X2, P2X4, P2Y1, P2Y2, TRPV2, TRPV3, TRPV6 and TRPM7 receptors/channels. Although a conventional response was elicited in control stromal-derived cells, the urothelial cell response to well-characterized TRPV1 and TRPV4 agonists/antagonists revealed unexpected anomalies. In addition, agonists that invoked an increase in intracellular Ca2+ promoted urothelial scratch repair, presumably through the release of ATP. The study raises important questions about the ligand selectivity of receptor/channel targets expressed by the urothelium. These pathways are important in urothelial tissue homeostasis, and this opens the possibility of selective drug targeting.

APA, Harvard, Vancouver, ISO, and other styles

9

Blythe, Sarah N., Jeremy F. Atherton, and Mark D. Bevan. "Synaptic Activation of Dendritic AMPA and NMDA Receptors Generates Transient High-Frequency Firing in Substantia Nigra Dopamine Neurons In Vitro." Journal of Neurophysiology 97, no. 4 (April 2007): 2837–50. http://dx.doi.org/10.1152/jn.01157.2006.

Full text

Abstract:

Transient high-frequency activity of substantia nigra dopamine neurons is critical for striatal synaptic plasticity and associative learning. However, the mechanisms underlying this mode of activity are poorly understood because, in contrast to other rapidly firing neurons, high-frequency activity is not evoked by somatic current injection. Previous studies have suggested that activation of dendritic N-methyl-d-aspartate (NMDA) receptors and/or G-protein-coupled receptor (GPCR)-mediated reduction of action potential afterhyperpolarization and/or activation of cation channels underlie high-frequency activity. To address their relative contribution, transient high-frequency activity was evoked using local electrical stimulation (1 s, 10–100 Hz) in brain slices prepared from p15–p25 rats in the presence of GABA and D2 dopamine receptor antagonists. The frequency, pattern, and morphology of action potentials evoked under these conditions were similar to those observed in vivo. Evoked activity and reductions in action potential afterhyperpolarization were diminished greatly by application of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) or NMDA receptor selective antagonists and abolished completely by co-application of AMPA and NMDA antagonists. In contrast, application of glutamatergic and cholinergic GPCR antagonists moderately enhanced evoked activity. Dendritic pressure-pulse application of glutamate evoked high-frequency activity that was similarly sensitive to antagonism of AMPA or NMDA receptors. Taken together, these data suggest that dendritic AMPA and NMDA receptor-mediated synaptic conductances are sufficient to generate transient high-frequency activity in substantia nigra dopamine neurons by rapidly but transiently overwhelming the conductances underlying action potential afterhyperpolarization and/or engaging postsynaptic voltage-dependent ion channels in a manner that overcomes the limiting effects of afterhyperpolarization.

APA, Harvard, Vancouver, ISO, and other styles

10

Montell, Craig. "Drosophila sensory receptors—a set of molecular Swiss Army Knives." Genetics 217, no. 1 (January 1, 2021): 1–34. http://dx.doi.org/10.1093/genetics/iyaa011.

Full text

Abstract:

Abstract Genetic approaches in the fruit fly, Drosophila melanogaster, have led to a major triumph in the field of sensory biology—the discovery of multiple large families of sensory receptors and channels. Some of these families, such as transient receptor potential channels, are conserved from animals ranging from worms to humans, while others, such as “gustatory receptors,” “olfactory receptors,” and “ionotropic receptors,” are restricted to invertebrates. Prior to the identification of sensory receptors in flies, it was widely assumed that these proteins function in just one modality such as vision, smell, taste, hearing, and somatosensation, which includes thermosensation, light, and noxious mechanical touch. By employing a vast combination of genetic, behavioral, electrophysiological, and other approaches in flies, a major concept to emerge is that many sensory receptors are multitaskers. The earliest example of this idea was the discovery that individual transient receptor potential channels function in multiple senses. It is now clear that multitasking is exhibited by other large receptor families including gustatory receptors, ionotropic receptors, epithelial Na+ channels (also referred to as Pickpockets), and even opsins, which were formerly thought to function exclusively as light sensors. Genetic characterizations of these Drosophila receptors and the neurons that express them also reveal the mechanisms through which flies can accurately differentiate between different stimuli even when they activate the same receptor, as well as mechanisms of adaptation, amplification, and sensory integration. The insights gleaned from studies in flies have been highly influential in directing investigations in many other animal models.

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Transient receptor potential receptors"

1

Liapi, Anastasia. "Cloning of the vanilloid-like receptor VR-L and investigation of its interaction with members of the transient receptor potential family of receptors." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270624.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Stokes, Alexander James. "Regulatory interactions of transient receptor potential channels." Thesis, University of Warwick, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.418114.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Abe, Junji. "Localization and desensitization of transient receptor potential M8." 京都大学 (Kyoto University), 2007. http://hdl.handle.net/2433/135954.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Kim, Ju Young. "M1 muscarinic acetylcholine receptor regulation of endogenous transient receptor potential-canonical, subtype 6 (TRPC6) channels." Connect to resource, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1117570788.

Full text

Abstract:

Thesis (Ph. D.)--Ohio State University, 2005.
Title from first page of PDF file. Document formatted into pages; contains xviii, 178 p.; also includes graphics. Includes bibliographical references (p. 163-178). Available online via OhioLINK's ETD Center

APA, Harvard, Vancouver, ISO, and other styles

5

Clark, Natalie Carol. "Evaluation of the roles of adrenomedullin1 (AM1) and transient receptor potential vanilloid1 (TRPV1) receptors in an LPS model of sepsis." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434778.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Friedrich, Olaf. "Biochemische und funktionelle Charakterisierung des potentiellen Calciumionenkanalproteins Maus-transient-receptor-Potential-1[beta] [Maus-transient-receptor-Potential-1beta] (mTRP1[beta] [(mTRP1beta)]." [S.l. : s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=963048163.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Cao, De-Shou. "Role of transient receptor potential (TRP) channels in nociception /." Available to subscribers only, 2009. http://proquest.umi.com/pqdweb?did=1967913291&sid=2&Fmt=2&clientId=1509&RQT=309&VName=PQD.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Cao, Deshou. "Role of Transient Receptor Potential (TRP) Channels in Nociception." OpenSIUC, 2009. https://opensiuc.lib.siu.edu/dissertations/71.

Full text

Abstract:

Transient receptor potential (TRP) channels play an important role in sensory and nonsensory functions. TRPVanilloid 1 and TRPVanilloid 4 are proposed to be involved in inflammation-induced pain. TRPV1 is extensively studied and it is specifically involved in inflammatory thermal hypersensitivity. Mechanical hypersensitivity is one of the significant components of nociception. Several receptors have been proposed to underlie mechanosensation. The molecular entities responsible for mechanosensation are not fully understood. In this study, I have characterized the properties of TRPV4, a putative mechanosensitive ion channel expressed in dorsal root ganglion (DRG) neurons and nonsensory tissues. First, I have investigated the expression and function of TRPV4 and TRPV1 in the DRG neuronal cell bodies as well as their central terminals and determined the modulation by protein kinase C (PKC). Both TRPV4 and TRPV1 are expressed in DRG and laminae I and II of the spinal dorsal horn (DH). Ca2+ fluorescence imaging and whole-cell patch-clamp experiments showed that both capsaicin-induced TRPV1 response and 4alpha-phorbol 12, 13-didecanoate (4alpha-PDD)-induced TRPV4 response were observed in a proportion of the same DRG neurons, suggesting their co-expression. Incubation of DRG neurons with phorbol 12, 13-dibutyrate (PDBu), a PKC activator, resulted in a significantly greater potentiation of TRPV4 currents than TRPV1 currents. In HEK cells heterologously expressing TRPV4, PDBu potentiated TRPV4-mediated single-channel current activity. In patch-clamped DH neurons, the application of 4alpha-PDD at the first sensory synapse increased the frequency but not the amplitude of the miniature excitatory postsynaptic currents (mEPSCs), suggesting a presynaptic locus of action. 4alpha-PDD-induced increase in the frequency of mEPSC was further facilitated by PDBu. These results suggest that TRPV4 in the central terminals modulates synaptic transmission and is regulated by PKC. Second, I have studied the mechanosensitivity of TRPV4 in cell-attached patches by applying direct mechanical force via the patch pipette. In TRPV4 expressing HEK cells, the application of negative pressure evoked single-channel current activity in a reversible manner and the channel activity was enhanced after incubation with PDBu. TRPV4 has been shown to be activated by hypotonicity. Here I show that negative pressure exaggerated hypotonicity-induced single-channel current activity. However, in similar experimental conditions, cells expressing TRPV1 did not respond to mechanical force. TRP channels are also expressed in non-sensory regions and the role of these channels is not fully understood. Both TRPV4 and TRPV1 are expressed in the hippocampus. Using whole-cell patch-clamp techniques, I have found that 4alpha-PDD increased the frequency, but not the amplitude of mEPSCs in cultured hippocampal neurons, suggesting a presynaptic site of action. Interestingly, the application of capsaicin had no effect on synaptic transmission in hippocampal neuronal cultures. Finally, I have investigated the expression and function of TRP channels in diabetes because TRP channels have been shown to be involved in peripheral neuropathy as well as vascular complications in diabetes. ROS production plays a critical role in the progress of diabetes. I propose that lower levels of ROS up-regulate the expression TRP channels in the early stages of diabetes, leading to hyperalgesia, and higher levels of ROS or chronic exposure to ROS down-regulate TRP channels in the late stages of diabetes, resulting in hypoalgesia. I have found that the expression of TRPV1 and phospho p38 (p-p38) MAPK was increased in DRG of streptozotocin (STZ)-injected diabetic and non-diabetic hyperalgesic mice. An increase in TRPV1 and p-p38 MAPK levels was induced by STZ or H2O2 treatment in stably TRPV1 expressing HEK cells, suggesting the involvement of STZ-ROS-p38MAPK pathway. TRPV4 has been reported to be involved in vasodilatation by shear stress in blood vessels. Here, I have demonstrated that TRPV4 is expressed in lymphatic endothelial cells (LECs). Treatment with low concentration of H2O2 enhanced the expression of TRPV4 at mRNA and protein levels in LECs, suggesting that mild levels of ROS up-regulate TRPV4 expression. In diabetes, beta cell dysfunction is responsible for decreased insulin release. TRPV4 is expressed in RINm5F (beta cell line), islets and pancreas. It has been shown that hypotonicity induced insulin release in beta cell lines, which was mediated by activation of stretch-activated channels, raising the possibility of the involvement of TRPV4, a mechanosensitive channel. Therefore, I have studied the functional role of TRPV4 in beta cells. Incubation with 4alpha-PDD enhanced insulin release in RINm5F cells, suggesting TRPV4 regulates insulin secretion from pancreatic beta cells. Since TRPV4 expression levels are decreased in diabetes, insulin secretion from beta cells may be impaired. In summary, TRPV1, a thermosensitive channel, and TRPV4, a mechanosensitive channel, contribute to thermal and mechanical hyperalgesia, respectively in the early stage of DPN through their up-regulation by ROS-p38 MAPK and insulin/IGF-1 pathways. Due to the mechanical sensitivity of TRPV4 channel, the up-regulation in the early stage and down-regulation in the late stage may be involved in the development of vascular complications and regulation of insulin release in diabetes.

APA, Harvard, Vancouver, ISO, and other styles

9

Starr, Annalouise Bertina. "Mechanisms involved in transient receptor potential vanilloid receptor 1 (TRPV 1) mediated vasoactive responses." Thesis, King's College London (University of London), 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.444546.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Agustus, Daniel Joseph. "Transient receptor potential signalling in normal human urothelial cell cultures." Thesis, University of York, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.489187.

Full text

Abstract:

The urothelium is a highly compliant, transitional epithelium that enables the bladder to accommodate urine during filling by alteration of its surface area. The urothelium was traditionally thought of as a passive barrier to ions and solutes. More recent data suggested that the urothelium may have a sensory function arising from the urothelial stretch-triggered release of chemical mediators such as ATP, as demonstrated in rabbit and cat. intracellular caicium signalling known to induce vesicle fusion and ATP release from neuronal cells; a similar mechanism may be responsible for urothelial ATP release.

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "Transient receptor potential receptors"

1

service), SpringerLink (Online, ed. Transient Receptor Potential Channels. Dordrecht: Springer Science+Business Media B.V., 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Islam, Md Shahidul, ed. Transient Receptor Potential Channels. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0265-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Flockerzi, Veit, and Bernd Nilius, eds. Transient Receptor Potential (TRP) Channels. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-34891-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Nilius, Bernd, and Veit Flockerzi, eds. Mammalian Transient Receptor Potential (TRP) Cation Channels. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-05161-1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Nilius, Bernd, and Veit Flockerzi, eds. Mammalian Transient Receptor Potential (TRP) Cation Channels. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54215-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Wang, Yizheng, ed. Transient Receptor Potential Canonical Channels and Brain Diseases. Dordrecht: Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-024-1088-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Szallasi, Arpad. TRP channels in health and disease: Implications for diagnosis and therapy. Hauppauge, N.Y: Nova Science Publishers, 2010.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

Nilius, Bernd, and Veit Flockerzi. Transient Receptor Potential Channels. Springer, 2010.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Islam, MD Shahidul. Transient Receptor Potential Channels. Springer, 2011.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

J, Abramowitz, Flockerzi Veit, and Nilius B, eds. Transient receptor potential (TRP) channels. Berlin: Springer, 2007.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Book chapters on the topic "Transient receptor potential receptors"

1

Baez-Nieto, David, Juan Pablo Castillo, Constantino Dragicevic, Osvaldo Alvarez, and Ramon Latorre. "Thermo-TRP Channels: Biophysics of Polymodal Receptors." In Transient Receptor Potential Channels, 469–90. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_26.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Trebak, M., L. Lemonnier, J. T. Smyth, G. Vazquez, and J. W. Putney. "Phospholipase C-Coupled Receptors and Activation of TRPC Channels." In Transient Receptor Potential (TRP) Channels, 593–614. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-34891-7_35.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Digel, Ilya. "Primary Thermosensory Events in Cells." In Transient Receptor Potential Channels, 451–68. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_25.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Brinkmeier, Heinrich. "TRP Channels in Skeletal Muscle: Gene Expression, Function and Implications for Disease." In Transient Receptor Potential Channels, 749–58. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_39.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Wong, Ching-On, and Xiaoqiang Yao. "TRP Channels in Vascular Endothelial Cells." In Transient Receptor Potential Channels, 759–80. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_40.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Li, Minghui, Yong Yu, and Jian Yang. "Structural Biology of TRP Channels." In Transient Receptor Potential Channels, 1–23. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Liu, Yi, and Ning Qin. "TRPM8 in Health and Disease: Cold Sensing and Beyond." In Transient Receptor Potential Channels, 185–208. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_10.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Colletti, Grace A., and Kirill Kiselyov. "TRPML1." In Transient Receptor Potential Channels, 209–19. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_11.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Flores, Emma N., and Jaime García-Añoveros. "TRPML2 and the Evolution of Mucolipins." In Transient Receptor Potential Channels, 221–28. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_12.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Noben-Trauth, Konrad. "The TRPML3 Channel: From Gene to Function." In Transient Receptor Potential Channels, 229–37. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0265-3_13.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Transient receptor potential receptors"

1

Cheon, Dae Young, Joo-Hee Kim, Young-Suk Jang, Yong Il Hwang, Sunghoon Park, Dong-Gyu Kim, Seung-Hun Jang, and Ki-Suck Jung. "The activation of transient receptor potential melastatin 8 (TRPM8) receptors of bronchial epithelial cells induces airway inflammation in bronchial asthma." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa3997.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Flajolet, Pauline, Sara J. Bonvini, Bilel Dekkak, Nadja Kobold, John J. Adcock, Peter Bradding, Maria G. Belvisi, and Mark A. Birrell. "Investigating Transient Receptor Potential V4 channel induced bronchospasm." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3875.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Jiang, Nan, Brian Y. Cooper, and Michael I. Nemenov. "Noninvasive diode laser activation of transient receptor potential proteins and nociceptors." In Biomedical Optics (BiOS) 2007, edited by Michael R. Hamblin, Ronald W. Waynant, and Juanita Anders. SPIE, 2007. http://dx.doi.org/10.1117/12.699204.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Mahapatra, Chitaranjan. "Simulation study of transient receptor potential current in urinary bladder over activity." In SAC 2018: Symposium on Applied Computing. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3167132.3167442.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Toumpanakis, Dimitrios, Athanasia Chatzianastasiou, Vyronia Vassilakopoulou, Eleftheria Mizi, Stamatios Theocharis, and Theodoros Vassilakopoulos. "Transient receptor potential vanilloid 4 channels mediate resistive breathing-induced acute lung injury." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3298.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Stengel, Peter W., and David O. Calligaro. "Bronchopulmonary Actions Of Transient Receptor Potential Vanilloid-1 (TRPV1) Agonists In Guinea Pigs." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5540.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Kuronuma, Koji, Mitsuo Otsuka, Masato Wakabayashi, Takeshi Yoshioka, Yasuhide Morioka, Tomofumi Kobayashi, Masami Kameda, Hirofumi Chiba, and Hiroki Takahashi. "Role of transient receptor potential vanilloid 4 in the therapeutic antifibrotic effects of pirfenidone." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa1289.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Bajoriunas, Vytis, Ieva Bajoriuniene, and Edgaras Stankevicius. "Late Breaking Abstract - Transient Receptor Potential Vanilloid subtype 4 in patients with lung cancer." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3938.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Baxter, Matthew D., Mark A. Birrell, and Maria G. Belvisi. "The Role Of Transient Receptor Potential Vanilloid 1 (TRPV1) In Tobacco Smoke Induced Airway Inflammation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6410.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Baxter, Matthew D., Maria G. Belvisi, and Mark A. Birrell. "The Role Of Transient Receptor Potential Melastatin 2 (TRPM2) In Murine Models Of Airway Inflammation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6411.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!