Log in

Relevant bibliographies by topics / Transcription factors / Journal articles

To see the other types of publications on this topic, follow the link: Transcription factors.

Journal articles on the topic 'Transcription factors'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 December 2024

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Transcription factors.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Wilson, Nicola K., Fernando J. Calero-Nieto, Rita Ferreira, and Berthold Göttgens. "Transcriptional regulation of haematopoietic transcription factors." Stem Cell Research & Therapy 2, no. 1 (2011): 6. http://dx.doi.org/10.1186/scrt47.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Geng, Yanbiao, Peter Laslo, Kevin Barton, and Chyung-Ru Wang. "Transcriptional Regulation ofCD1D1by Ets Family Transcription Factors." Journal of Immunology 175, no. 2 (July 7, 2005): 1022–29. http://dx.doi.org/10.4049/jimmunol.175.2.1022.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Senecal, Adrien, Brian Munsky, Florence Proux, Nathalie Ly, Floriane E. Braye, Christophe Zimmer, Florian Mueller, and Xavier Darzacq. "Transcription Factors Modulate c-Fos Transcriptional Bursts." Cell Reports 8, no. 1 (July 2014): 75–83. http://dx.doi.org/10.1016/j.celrep.2014.05.053.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

BARNES, P. J., and I. M. ADCOCK. "Transcription factors." Clinical Experimental Allergy 25, s2 (November 1995): 46–49. http://dx.doi.org/10.1111/j.1365-2222.1995.tb00421.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Hawkins, R. "Transcription Factors." Journal of Medical Genetics 33, no. 12 (December 1, 1996): 1054. http://dx.doi.org/10.1136/jmg.33.12.1054-a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Papavassiliou, Athanasios G. "Transcription Factors." New England Journal of Medicine 332, no. 1 (January 5, 1995): 45–47. http://dx.doi.org/10.1056/nejm199501053320108.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Locker, J. "Transcription Factors." Biomedicine & Pharmacotherapy 52, no. 1 (January 1998): 47. http://dx.doi.org/10.1016/s0753-3322(97)86247-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Locker., J. "Transcription Factors." Journal of Steroid Biochemistry and Molecular Biology 64, no. 5-6 (March 1998): 316. http://dx.doi.org/10.1016/s0960-0760(96)00245-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Handel, Malcolm L., and Laila Girgis. "Transcription factors." Best Practice & Research Clinical Rheumatology 15, no. 5 (December 2001): 657–75. http://dx.doi.org/10.1053/berh.2001.0186.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Parker, C. S. "Transcription factors." Current Opinion in Cell Biology 1, no. 3 (June 1989): 512–18. http://dx.doi.org/10.1016/0955-0674(89)90013-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Zhang, Yuli, and Linlin Hou. "Alternate Roles of Sox Transcription Factors beyond Transcription Initiation." International Journal of Molecular Sciences 22, no. 11 (May 31, 2021): 5949. http://dx.doi.org/10.3390/ijms22115949.

Full text

Abstract:

Sox proteins are known as crucial transcription factors for many developmental processes and for a wide range of common diseases. They were believed to specifically bind and bend DNA with other transcription factors and elicit transcriptional activation or repression activities in the early stage of transcription. However, their functions are not limited to transcription initiation. It has been showed that Sox proteins are involved in the regulation of alternative splicing regulatory networks and translational control. In this review, we discuss the current knowledge on how Sox transcription factors such as Sox2, Sry, Sox6, and Sox9 allow the coordination of co-transcriptional splicing and also the mechanism of SOX4-mediated translational control in the context of RNA polymerase III.

APA, Harvard, Vancouver, ISO, and other styles

12

Bloor, Adrian, Ekaterina Kotsopoulou, Penny Hayward, Brian Champion, and Anthony Green. "RFP represses transcriptional activation by bHLH transcription factors." Oncogene 24, no. 45 (June 27, 2005): 6729–36. http://dx.doi.org/10.1038/sj.onc.1208828.

Full text

APA, Harvard, Vancouver, ISO, and other styles

13

Zhang, Lang, Haoyue Yu, Pan Wang, Qingyang Ding, and Zhao Wang. "Screening of transcription factors with transcriptional initiation activity." Gene 531, no. 1 (November 2013): 64–70. http://dx.doi.org/10.1016/j.gene.2013.07.054.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Thiel, Gerald, Lisbeth A. Guethlein, and Oliver G. Rössler. "Insulin-Responsive Transcription Factors." Biomolecules 11, no. 12 (December 15, 2021): 1886. http://dx.doi.org/10.3390/biom11121886.

Full text

Abstract:

The hormone insulin executes its function via binding and activating of the insulin receptor, a receptor tyrosine kinase that is mainly expressed in skeletal muscle, adipocytes, liver, pancreatic β-cells, and in some areas of the central nervous system. Stimulation of the insulin receptor activates intracellular signaling cascades involving the enzymes extracellular signal-regulated protein kinase-1/2 (ERK1/2), phosphatidylinositol 3-kinase, protein kinase B/Akt, and phospholipase Cγ as signal transducers. Insulin receptor stimulation is correlated with multiple physiological and biochemical functions, including glucose transport, glucose homeostasis, food intake, proliferation, glycolysis, and lipogenesis. This review article focuses on the activation of gene transcription as a result of insulin receptor stimulation. Signal transducers such as protein kinases or the GLUT4-induced influx of glucose connect insulin receptor stimulation with transcription. We discuss insulin-responsive transcription factors that respond to insulin receptor activation and generate a transcriptional network executing the metabolic functions of insulin. Importantly, insulin receptor stimulation induces transcription of genes encoding essential enzymes of glycolysis and lipogenesis and inhibits genes encoding essential enzymes of gluconeogenesis. Overall, the activation or inhibition of insulin-responsive transcription factors is an essential aspect of orchestrating a wide range of insulin-induced changes in the biochemistry and physiology of insulin-responsive tissues.

APA, Harvard, Vancouver, ISO, and other styles

15

Yamada, Yasuyuki, and Fumihiko Sato. "Transcription Factors in Alkaloid Engineering." Biomolecules 11, no. 11 (November 18, 2021): 1719. http://dx.doi.org/10.3390/biom11111719.

Full text

Abstract:

Plants produce a large variety of low-molecular-weight and specialized secondary compounds. Among them, nitrogen-containing alkaloids are the most biologically active and are often used in the pharmaceutical industry. Although alkaloid chemistry has been intensively investigated, characterization of alkaloid biosynthesis, including biosynthetic enzyme genes and their regulation, especially the transcription factors involved, has been relatively delayed, since only a limited number of plant species produce these specific types of alkaloids in a tissue/cell-specific or developmental-specific manner. Recent advances in molecular biology technologies, such as RNA sequencing, co-expression analysis of transcripts and metabolites, and functional characterization of genes using recombinant technology and cutting-edge technology for metabolite identification, have enabled a more detailed characterization of alkaloid pathways. Thus, transcriptional regulation of alkaloid biosynthesis by transcription factors, such as basic helix–loop–helix (bHLH), APETALA2/ethylene-responsive factor (AP2/ERF), and WRKY, is well elucidated. In addition, jasmonate signaling, an important cue in alkaloid biosynthesis, and its cascade, interaction of transcription factors, and post-transcriptional regulation are also characterized and show cell/tissue-specific or developmental regulation. Furthermore, current sequencing technology provides more information on the genome structure of alkaloid-producing plants with large and complex genomes, for genome-wide characterization. Based on the latest information, we discuss the application of transcription factors in alkaloid engineering.

APA, Harvard, Vancouver, ISO, and other styles

16

Bakshi, Madhunita, and Ralf Oelmüller. "WRKY transcription factors." Plant Signaling & Behavior 9, no. 2 (February 2014): e27700. http://dx.doi.org/10.4161/psb.27700.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Massague, J. "Smad transcription factors." Genes & Development 19, no. 23 (December 1, 2005): 2783–810. http://dx.doi.org/10.1101/gad.1350705.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Steinke, John, and Larry Borish. "Beyond Transcription Factors." Allergy & Clinical Immunology International - Journal of the World Allergy Organization 16, no. 01 (2004): 20–27. http://dx.doi.org/10.1027/0838-1925.16.1.20.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Engelkamp, Dieter. "Pathological transcription factors." Trends in Genetics 16, no. 5 (May 2000): 233–34. http://dx.doi.org/10.1016/s0168-9525(99)01963-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Yeh, Jennifer E., Patricia A. Toniolo, and David A. Frank. "Targeting transcription factors." Current Opinion in Oncology 25, no. 6 (November 2013): 652–58. http://dx.doi.org/10.1097/01.cco.0000432528.88101.1a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Wolffe, A. "Architectural transcription factors." Science 264, no. 5162 (May 20, 1994): 1100–1101. http://dx.doi.org/10.1126/science.8178167.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Rushton, Paul J., Imre E. Somssich, Patricia Ringler, and Qingxi J. Shen. "WRKY transcription factors." Trends in Plant Science 15, no. 5 (May 2010): 247–58. http://dx.doi.org/10.1016/j.tplants.2010.02.006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Warren, Alan J. "Eukaryotic transcription factors." Current Opinion in Structural Biology 12, no. 1 (February 2002): 107–14. http://dx.doi.org/10.1016/s0959-440x(02)00296-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Tan, Song, and Timothy J. Richmond. "Eukaryotic transcription factors." Current Opinion in Structural Biology 8, no. 1 (February 1998): 41–48. http://dx.doi.org/10.1016/s0959-440x(98)80008-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Reese, Joseph C. "Basal transcription factors." Current Opinion in Genetics & Development 13, no. 2 (April 2003): 114–18. http://dx.doi.org/10.1016/s0959-437x(03)00013-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Wolberger, Cynthia. "Combinatorial transcription factors." Current Opinion in Genetics & Development 8, no. 5 (October 1998): 552–59. http://dx.doi.org/10.1016/s0959-437x(98)80010-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Struhl, Kevin. "Yeast transcription factors." Current Opinion in Cell Biology 5, no. 3 (June 1993): 513–20. http://dx.doi.org/10.1016/0955-0674(93)90018-l.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Crunkhorn, Sarah. "Targeting transcription factors." Nature Reviews Drug Discovery 18, no. 1 (December 28, 2018): 18. http://dx.doi.org/10.1038/nrd.2018.231.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

D'Arcangelo, Gabriel la, and Tom Curran. "Smart transcription factors." Nature 376, no. 6538 (July 1995): 292–93. http://dx.doi.org/10.1038/376292a0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Latchman, D. S. "Eukaryotic transcription factors." Biochemical Journal 270, no. 2 (September 1, 1990): 281–89. http://dx.doi.org/10.1042/bj2700281.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Latchman, David S. "Inhibitory transcription factors." International Journal of Biochemistry & Cell Biology 28, no. 9 (September 1996): 965–74. http://dx.doi.org/10.1016/1357-2725(96)00039-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

Carter, Matthew E., and Anne Brunet. "FOXO transcription factors." Current Biology 17, no. 4 (February 2007): R113—R114. http://dx.doi.org/10.1016/j.cub.2007.01.008.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Polyanovsky, Oleg L., and Alexander G. Stepchenko. "Eukaryotic transcription factors." BioEssays 12, no. 5 (May 1990): 205–10. http://dx.doi.org/10.1002/bies.950120503.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

WATANABE, A., M. ARAI, N. KOITABASHI, M. YAMAZAKI, K. NIWANO, and M. KURABAYASHI. "Mitochondrial transcription factors regulate SERCA2 gene transcription." Journal of Molecular and Cellular Cardiology 41, no. 6 (December 2006): 1049. http://dx.doi.org/10.1016/j.yjmcc.2006.08.046.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Becskei, Attila. "Tuning up Transcription Factors for Therapy." Molecules 25, no. 8 (April 20, 2020): 1902. http://dx.doi.org/10.3390/molecules25081902.

Full text

Abstract:

The recent developments in the delivery and design of transcription factors put their therapeutic applications within reach, exemplified by cell replacement, cancer differentiation and T-cell based cancer therapies. The success of such applications depends on the efficacy and precision in the action of transcription factors. The biophysical and genetic characterization of the paradigmatic prokaryotic repressors, LacI and TetR and the designer transcription factors, transcription activator-like effector (TALE) and CRISPR-dCas9 revealed common principles behind their efficacy, which can aid the optimization of transcriptional activators and repressors. Further studies will be required to analyze the linkage between dissociation constants and enzymatic activity, the role of phase separation and squelching in activation and repression and the long-range interaction of transcription factors with epigenetic regulators in the context of the chromosomes. Understanding these mechanisms will help to tailor natural and synthetic transcription factors to the needs of specific applications.

APA, Harvard, Vancouver, ISO, and other styles

36

Chávez, Joselyn, Damien P. Devos, and Enrique Merino. "Complementary Tendencies in the Use of Regulatory Elements (Transcription Factors, Sigma Factors, and Riboswitches) in Bacteria and Archaea." Journal of Bacteriology 203, no. 2 (October 19, 2020): e00413-20. http://dx.doi.org/10.1128/jb.00413-20.

Full text

Abstract:

ABSTRACTIn prokaryotes, the key players in transcription initiation are sigma factors and transcription factors that bind to DNA to modulate the process, while premature transcription termination at the 5′ end of the genes is regulated by attenuation and, in particular, by attenuation associated with riboswitches. In this study, we describe the distribution of these regulators across phylogenetic groups of bacteria and archaea and find that their abundance not only depends on the genome size, as previously described, but also varies according to the phylogeny of the organism. Furthermore, we observed a tendency for organisms to compensate for the low frequencies of a particular type of regulatory element (i.e., transcription factors) with a high frequency of other types of regulatory elements (i.e., sigma factors). This study provides a comprehensive description of the more abundant COG, KEGG, and Rfam families of transcriptional regulators present in prokaryotic genomes.IMPORTANCE In this study, we analyzed the relationship between the relative frequencies of the primary regulatory elements in bacteria and archaea, namely, transcription factors, sigma factors, and riboswitches. In bacteria, we reveal a compensatory behavior for transcription factors and sigma factors, meaning that in phylogenetic groups in which the relative number of transcription factors was low, we found a tendency for the number of sigma factors to be high and vice versa. For most of the phylogenetic groups analyzed here, except for Firmicutes and Tenericutes, a clear relationship with other mechanisms was not detected for transcriptional riboswitches, suggesting that their low frequency in most genomes does not constitute a significant impact on the global variety of transcriptional regulatory elements in prokaryotic organisms.

APA, Harvard, Vancouver, ISO, and other styles

37

Poulat, Francis. "Non-Coding Genome, Transcription Factors, and Sex Determination." Sexual Development 15, no. 5-6 (2021): 295–307. http://dx.doi.org/10.1159/000519725.

Full text

Abstract:

In vertebrates, gonadal sex determination is the process by which transcription factors drive the choice between the testicular and ovarian identity of undifferentiated somatic progenitors through activation of 2 different transcriptional programs. Studies in animal models suggest that sex determination always involves sex-specific transcription factors that activate or repress sex-specific genes. These transcription factors control their target genes by recognizing their regulatory elements in the non-coding genome and their binding motifs within their DNA sequence. In the last 20 years, the development of genomic approaches that allow identifying all the genomic targets of a transcription factor in eukaryotic cells gave the opportunity to globally understand the function of the nuclear proteins that control complex genetic programs. Here, the major transcription factors involved in male and female vertebrate sex determination and the genomic profiling data of mouse gonads that contributed to deciphering their transcriptional regulation role will be reviewed.

APA, Harvard, Vancouver, ISO, and other styles

38

Pan, Yanyun, Jin Dai, Minwei Jin, Qiujun Zhou, Xiaoliang Jin, and Jinjie Zhang. "Transcription factors in tanshinones: Emerging mechanisms of transcriptional regulation." Medicine 103, no. 47 (November 22, 2024): e40343. http://dx.doi.org/10.1097/md.0000000000040343.

Full text

Abstract:

Transcription factors play a crucial role in the biosynthesis of tanshinones, which are significant secondary metabolites derived from Salvia miltiorrhiza, commonly known as Danshen. These compounds have extensive pharmacological properties, including anti-inflammatory and cardioprotective effects. This review delves into the roles of various transcription factor families, such as APETALA2/ethylene response factor, basic helix-loop-helix, myeloblastosis, basic leucine zipper, and WRKY domain-binding protein, in regulating the biosynthetic pathways of tanshinones. We discuss the emerging mechanisms by which these transcription factors influence the synthesis of tanshinones, both positively and negatively, by directly regulating gene expression or forming complex regulatory networks. Additionally, the review highlights the potential applications of these insights in enhancing tanshinone production through genetic and metabolic engineering, setting the stage for future advancements in medicinal plant research.

APA, Harvard, Vancouver, ISO, and other styles

39

Gao, T. W., W. W. Zhang, A. P. Song, C. An, J. J. Xin, J. F. Jiang, Z. Y. Guan, F. D. Chen, and S. M. Chen. "Phylogenetic and transcriptional analysis of chrysanthemum GRAS transcription factors." Biologia Plantarum 62, no. 4 (June 27, 2018): 711–20. http://dx.doi.org/10.1007/s10535-018-0816-1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Fox, Rebecca M., and Deborah J. Andrew. "Transcriptional regulation of secretory capacity by bZip transcription factors." Frontiers in Biology 10, no. 1 (November 17, 2014): 28–51. http://dx.doi.org/10.1007/s11515-014-1338-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Cai, Bin, Cheng-Hui Li, Ai-Sheng Xiong, Ri-He Peng, Jun Zhou, Feng Gao, Zhen Zhang, and Quan-Hong Yao. "DGTF: A Database of Grape Transcription Factors." Journal of the American Society for Horticultural Science 133, no. 3 (May 2008): 459–61. http://dx.doi.org/10.21273/jashs.133.3.459.

Full text

Abstract:

The database of grape transcription factors (DGTF) is a plant transcription factor (TF) database comprehensively collecting and annotating grape (Vitis L.) TF. The DGTF contains 1423 putative grape TF in 57 families. These TF were identified from the predicted wine grape (Vitis vinifera L.) proteins from the grape genome sequencing project by means of a domain search. The DGTF provides detailed annotations for individual members of each TF family, including sequence feature, domain architecture, expression information, and orthologs in other plants. Cross-links to other public databases make its annotations more extensive. In addition, some other transcriptional regulators were also included in the DGTF. It contains 202 transcriptional regulators in 10 families.

APA, Harvard, Vancouver, ISO, and other styles

42

Seo, Hyungseok, Joyce Chen, Edahí González-Avalos, Daniela Samaniego-Castruita, Arundhoti Das, Yueqiang H. Wang, Isaac F. López-Moyado, et al. "TOX and TOX2 transcription factors cooperate with NR4A transcription factors to impose CD8+ T cell exhaustion." Proceedings of the National Academy of Sciences 116, no. 25 (May 31, 2019): 12410–15. http://dx.doi.org/10.1073/pnas.1905675116.

Full text

Abstract:

T cells expressing chimeric antigen receptors (CAR T cells) have shown impressive therapeutic efficacy against leukemias and lymphomas. However, they have not been as effective against solid tumors because they become hyporesponsive (“exhausted” or “dysfunctional”) within the tumor microenvironment, with decreased cytokine production and increased expression of several inhibitory surface receptors. Here we define a transcriptional network that mediates CD8+ T cell exhaustion. We show that the high-mobility group (HMG)-box transcription factors TOX and TOX2, as well as members of the NR4A family of nuclear receptors, are targets of the calcium/calcineurin-regulated transcription factor NFAT, even in the absence of its partner AP-1 (FOS-JUN). Using a previously established CAR T cell model, we show that TOX and TOX2 are highly induced in CD8+ CAR+ PD-1high TIM3high (“exhausted”) tumor-infiltrating lymphocytes (CAR TILs), and CAR TILs deficient in both TOX and TOX2 (Tox DKO) are more effective than wild-type (WT), TOX-deficient, or TOX2-deficient CAR TILs in suppressing tumor growth and prolonging survival of tumor-bearing mice. Like NR4A-deficient CAR TILs, Tox DKO CAR TILs show increased cytokine expression, decreased expression of inhibitory receptors, and increased accessibility of regions enriched for motifs that bind activation-associated nuclear factor κB (NFκB) and basic region-leucine zipper (bZIP) transcription factors. These data indicate that Tox and Nr4a transcription factors are critical for the transcriptional program of CD8+ T cell exhaustion downstream of NFAT. We provide evidence for positive regulation of NR4A by TOX and of TOX by NR4A, and suggest that disruption of TOX and NR4A expression or activity could be promising strategies for cancer immunotherapy.

APA, Harvard, Vancouver, ISO, and other styles

43

Hampsey, Michael. "Molecular Genetics of the RNA Polymerase II General Transcriptional Machinery." Microbiology and Molecular Biology Reviews 62, no. 2 (June 1, 1998): 465–503. http://dx.doi.org/10.1128/mmbr.62.2.465-503.1998.

Full text

Abstract:

SUMMARY Transcription initiation by RNA polymerase II (RNA pol II) requires interaction between cis-acting promoter elements and trans-acting factors. The eukaryotic promoter consists of core elements, which include the TATA box and other DNA sequences that define transcription start sites, and regulatory elements, which either enhance or repress transcription in a gene-specific manner. The core promoter is the site for assembly of the transcription preinitiation complex, which includes RNA pol II and the general transcription fctors TBP, TFIIB, TFIIE, TFIIF, and TFIIH. Regulatory elements bind gene-specific factors, which affect the rate of transcription by interacting, either directly or indirectly, with components of the general transcriptional machinery. A third class of transcription factors, termed coactivators, is not required for basal transcription in vitro but often mediates activation by a broad spectrum of activators. Accordingly, coactivators are neither gene-specific nor general transcription factors, although gene-specific coactivators have been described in metazoan systems. Transcriptional repressors include both gene-specific and general factors. Similar to coactivators, general transcriptional repressors affect the expression of a broad spectrum of genes yet do not repress all genes. General repressors either act through the core transcriptional machinery or are histone related and presumably affect chromatin function. This review focuses on the global effectors of RNA polymerase II transcription in yeast, including the general transcription factors, the coactivators, and the general repressors. Emphasis is placed on the role that yeast genetics has played in identifying these factors and their associated functions.

APA, Harvard, Vancouver, ISO, and other styles

44

Granadino, B., C. Perez-Sanchez, and J. Rey-Campos. "Fork Head Transcription Factors." Current Genomics 1, no. 4 (December 1, 2000): 353–82. http://dx.doi.org/10.2174/1389202003351319.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

IMAGAWA, Masayoshi. "Transcription Factors in Eukaryotes." Seibutsu Butsuri 33, no. 3 (1993): 154–58. http://dx.doi.org/10.2142/biophys.33.154.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Wilanowski, Tomasz, and Sebastian Dworkin. "Transcription Factors in Cancer." International Journal of Molecular Sciences 23, no. 8 (April 18, 2022): 4434. http://dx.doi.org/10.3390/ijms23084434.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Rusk, Nicole. "Transcription factors without footprints." Nature Methods 11, no. 10 (September 29, 2014): 988–89. http://dx.doi.org/10.1038/nmeth.3128.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Williams, Ruth. "Imaging individual transcription factors." Journal of Cell Biology 177, no. 6 (June 4, 2007): 946a. http://dx.doi.org/10.1083/jcb.1776rr1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Huang, H., and D. J. Tindall. "Dynamic FoxO transcription factors." Journal of Cell Science 120, no. 15 (July 17, 2007): 2479–87. http://dx.doi.org/10.1242/jcs.001222.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

KAMBE, FUKUSHI, and HISAO SEO. "Thyroid-Specific Transcription Factors." Endocrine Journal 44, no. 6 (1997): 775–84. http://dx.doi.org/10.1507/endocrj.44.775.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!