Relevant bibliographies by topics / Transcranial tissue Doppler

Academic literature on the topic 'Transcranial tissue Doppler'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Transcranial tissue Doppler"

1

Rock, Erwin H. "Vascular Dizziness and Transcranial Doppler Ultrasonography." Annals of Otology, Rhinology & Laryngology 98, no. 7_suppl (July 1989): 3–23. http://dx.doi.org/10.1177/00034894890980s701.

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Dizziness as defined herein will include an illusion of motion caused by various degrees of ischemia to the vestibular pathway or its interconnecting pathways. “Syndrome,” such as the lateral medullary syndrome, denotes a macroinfarct, while a microinfarct or an area of incomplete infarct (where there may develop an incomplete degeneration of the neural tissue secondary to the arteriolar microatheromatous stenosis) may cause only one neurologic deficit, such as dizziness per se as the only symptom. However, the latter may presage a larger and more debilitating neurologic deficit. The transcranial Doppler, used to track sequentially the larger basal arteries of the brain, specifically the vertebrobasilar arterial system, is an addition to noninvasive diagnostic methods of separating vascular problems from other causes of dizziness.
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Craven, Claudia L., Morrakot Sae-Huang, Chandrashekar Hoskote, Laurence D. Watkins, Ugan Reddy, and Ahmed K. Toma. "Relationship between Brain Tissue Oxygen Tension and Transcranial Doppler Ultrasonography." World Neurosurgery 149 (May 2021): e942-e946. http://dx.doi.org/10.1016/j.wneu.2021.01.070.

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Lindegaard, Karl-Fredrik, Peter Grolimund, Rune Aaslid, and Helge Nornes. "Evaluation of cerebral AVM's using transcranial Doppler ultrasound." Journal of Neurosurgery 65, no. 3 (September 1986): 335–44. http://dx.doi.org/10.3171/jns.1986.65.3.0335.

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✓Blood flow velocities in basal cerebral arteries were recorded noninvasively in 28 patients with cerebral arteriovenous malformations (AVM's) and were correlated with the angiographic findings. In normal arteries remote from the AVM, flow velocities ranged from 44 to 94 cm/sec (median 65 cm/sec) with pulsatility indexes from 0.65 to 1.10 (median 0.87). This is consistent with findings in normal individuals. Arteries feeding the AVM's were identified by the high flow velocities (ranging from 75 to 237 cm/sec, median 124 cm/sec). The pulsatility index ranged from 0.22 to 0.74 (median 0.48). The difference of these results from findings in normal remote arteries was highly significant (p < 0.001). Hyperventilation tests illustrated the hemodynamic difference between an AVM and normal cerebrovascular beds. Flow velocity measurements permitted noninvasive diagnosis of AVM's in 26 of the 28 patients. Furthermore, the identification of individual feeding arteries permitted good definition of the anatomical localization of individual AVM's. Flow velocity measurements combined with computerized tomography scans are useful in the diagnosis of AVM's. With the feeding artery's configuration identified on angiography, flow velocity measurements permit a new insight into the “hemodynamic dimension” of an AVM and its possible effects on adjacent normal brain-tissue perfusion in the individual patient.
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Hertel, Frank, Christof Walter, Martin Bettag, Maria Mörsdorf, R. Loch Macdonald, Gabriele Schackert, and J. Max Findlay. "Perfusion-weighted Magnetic Resonance Imaging in Patients with Vasospasm: A Useful New Tool in the Management of Patients with Subarachnoid Hemorrhage." Neurosurgery 56, no. 1 (January 1, 2005): 28–35. http://dx.doi.org/10.1227/01.neu.0000144866.28101.6d.

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Abstract OBJECTIVE: Cerebral vasospasm (VSP) is one of the most important risk factors for the development of a delayed neurological deficit after subarachnoid hemorrhage (SAH). Perfusion-weighted magnetic resonance imaging (pwMRI) provides the possibility of detecting tissue at risk for infarction. The objective of our study was to evaluate the feasibility and impact of pwMRI in the management of SAH patients. METHODS: From a consecutive series of 180 patients experiencing SAH and treated at our institution over a 3-year period, we identified 20 who underwent pwMRI during their acute illness. For these 20 patients, the results of pwMRI were compared with the results of diffusion-weighted MRI, transcranial Doppler sonography, and neurological examinations performed at the same time and with repeated pwMRI examinations of the same patient at different times. RESULTS: Nineteen of 20 patients showed perfusion changes predominantly in the time maps. Fifteen of 19 patients with changes in pwMRI had a neurological deficit at the same time. In 7 of 15 patients with neurological deterioration, transcranial Doppler sonography showed signs of VSP, whereas all 15 patients showed alterations in pwMRI. The areas of perfusion changes in pwMRI correlated well with the neurological deficits of the patients and were larger than the areas of changed diffusion in diffusion-weighted MRI performed at the same time. There were no clinical complications with regard to the pwMRI examinations. CONCLUSION: pwMRI is safe and helpful in the management of patients with VSP after SAH. The sensitivity of pwMRI is higher than that of transcranial Doppler sonography in the detection of decreased perfusion as a result of VSP. pwMRI can detect tissue at risk before definitive infarction occurs and therefore may lead to a change of therapy in those patients.
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Telman, Gregory, Olga Namestnikov, Efim Kouperberg, Elliot Sprecher, and David Yarnitsky. "Ischemic Middle Cerebral Artery Stroke Missing the Tissue Plasminogen Activator Time Window: Transcranial Doppler Evaluation." Journal of Stroke and Cerebrovascular Diseases 17, no. 6 (November 2008): 366–69. http://dx.doi.org/10.1016/j.jstrokecerebrovasdis.2008.04.004.

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Porter, Tyrone M., Christy K. Holland, Jason M. Meunier, and George J. Shaw. "Enhancement of recombinant tissue‐plasminogen activator (rt‐PA) activity with 2‐MHz transcranial Doppler ultrasound." Journal of the Acoustical Society of America 120, no. 5 (November 2006): 3004. http://dx.doi.org/10.1121/1.4787007.

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Musahl, Christian, Hans Henkes, Zsolt Vajda, Jan Coburger, and Nikolai Hopf. "Continuous Local Intra-arterial Nimodipine Administration in Severe Symptomatic Vasospasm After Subarachnoid Hemorrhage." Neurosurgery 68, no. 6 (June 1, 2011): 1541–47. http://dx.doi.org/10.1227/neu.0b013e31820edd46.

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Abstract BACKGROUND: Cerebral vasospasm (CV) is a potentially disastrous consequence of subarachnoid hemorrhage despite medical treatment. Nimodipine is a potent drug for vessel relaxation, but side effects may preclude a sufficient dose. OBJECTIVE: To explore whether continuous local intra-arterial nimodipine administration (CLINA) can reverse vasospasm and prevent delayed ischemic neurological deficit. METHODS: Six consecutive subarachnoid hemorrhage patients (5 women; mean age, 47.2 years) with severe CV despite maximum medical therapy underwent CLINA within 2 hours after the onset of clinical symptoms. After anticoagulation, microcatheters were inserted distally in the concerning supra-aortic vessels. Glyceryl trinitrate injection (2 mg) was followed by CLINA (nimodipine 0.4 mg/h for 70-147 hours). Duration of CLINA was determined by neurological status, transcranial Doppler sonography, and partial tissue oxygen pressure values. RESULTS: In all patients, neurological deficits improved or partial tissue oxygen pressure values returned to normal and transcranial Doppler sonography confirmed a reduced blood flow velocity within 12 hours. Magnetic resonance imaging showed no ischemic lesion caused by CV. Neurological outcome was good (modified Rankin Scale score, 0–2) in 3 patients, whereas 1 patient had a moderate clinical outcome (modified Rankin Scale score, 3–4) and 2 patients had a poor outcome (modified Rankin Scale score, 5) because of the SAH. CONCLUSION: Preliminary data show that CLINA is a straightforward, effective, and safe option for patients with severe CV refractory to medical therapy. Dilation of spastic arteries starts within a few hours and is lasting. Indication for CLINA is peripheral and diffuse CV at any location.
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Gan, Lingli, Xiaoling Yin, Jiating Huang, and Bin Jia. "Transcranial Doppler analysis based on computer and artificial intelligence for acute cerebrovascular disease." Mathematical Biosciences and Engineering 20, no. 2 (2022): 1695–715. http://dx.doi.org/10.3934/mbe.2023077.

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<abstract> <p>Cerebrovascular disease refers to damage to brain tissue caused by impaired intracranial blood circulation. It usually presents clinically as an acute nonfatal event and is characterized by high morbidity, disability, and mortality. Transcranial Doppler (TCD) ultrasonography is a non-invasive method for the diagnosis of cerebrovascular disease that uses the Doppler effect to detect the hemodynamic and physiological parameters of the major intracranial basilar arteries. It can provide important hemodynamic information that cannot be measured by other diagnostic imaging techniques for cerebrovascular disease. And the result parameters of TCD ultrasonography such as blood flow velocity and beat index can reflect the type of cerebrovascular disease and serve as a basis to assist physicians in the treatment of cerebrovascular diseases. Artificial intelligence (AI) is a branch of computer science which is used in a wide range of applications in agriculture, communications, medicine, finance, and other fields. In recent years, there are much research devoted to the application of AI to TCD. The review and summary of related technologies is an important work to promote the development of this field, which can provide an intuitive technical summary for future researchers. In this paper, we first review the development, principles, and applications of TCD ultrasonography and other related knowledge, and briefly introduce the development of AI in the field of medicine and emergency medicine. Finally, we summarize in detail the applications and advantages of AI technology in TCD ultrasonography including the establishment of an examination system combining brain computer interface (BCI) and TCD ultrasonography, the classification and noise cancellation of TCD ultrasonography signals using AI algorithms, and the use of intelligent robots to assist physicians in TCD ultrasonography and discuss the prospects for the development of AI in TCD ultrasonography.</p> </abstract>
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Zivanovic, Zeljko, Andrei Alexandrov, Aleksandar Jesic, and Petar Slankamenac. "Sonothrombolysis: Is the story (t)old or just the beginning." Medical review 67, no. 1-2 (2014): 17–23. http://dx.doi.org/10.2298/mpns1402017z.

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Introduction. Intravenous administration of recombinant tissue plasminogen activator, fastest and widely feasible treatment in acute ischemic stroke induces arterial recanalization, a prerequisite for neurological recovery. The Therapeutic Role of Ultrasound and Potential Mechanism of Sonothrombolysis. Augmentation of recanalization can be achieved safely in combination with diagnostic transcranial Doppler by delivering mechanical pressure waves to the thrombus and exposing more thrombus surface to circulating drug. The addition of microspheres can further improve thrombolytic effect. Clinical Trials. International multicenter CLOTBUST trial showed that acute ischemic stroke patients treated with sonothrombolysis had higher rate of arterial recanalization and dramatic clinical recovery without increasing risk of symptomatic intracranial hemorrhage. A microsphere dose-escalation study called TUCSON showed that rates of recanalization and clinical recovery tended to be higher in target groups compared with controls. Meta-analysis of clinical trials of sonothrombolysis. Cochrane Stroke Group found that sonothrombolysis was likely to reduce death or dependency. A metaanalysis of sonothrombolysis showed that patients who received any form of sonothrombolysis had more than twofold higher likelihood of achieving complete arterial recanalization. Perspectives for sonothrombolysis - Operator-independent device for sonothrombolysis. The collaborative group of the CLOTBUST trial designed multi-transducer assembly to cover conventional windows used for transcranial Doppler examinations. Operatorindependent device can be quickly mounted by medical personnel with no prior experience in ultrasound. Sonothrombolysis for acute ischemic stroke is now tested in a pivotal efficacy multi-national trial called CLOTBUSTER. Conclusion. Ultrasound is a promising tool to enhance systemic thrombolysis.
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Gandee, Richard, and Chad Miller. "Multimodality Monitoring: Toward Improved Outcomes." Seminars in Respiratory and Critical Care Medicine 38, no. 06 (December 2017): 785–92. http://dx.doi.org/10.1055/s-0037-1608774.

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AbstractMultimodality monitoring provides insights into the critically ill brain-injured patient through the assessment of biochemical, physiological, and electrical data that provides insight into a patient's condition and what strategies may be available to limit further damage and improve the odds for recovery. Modalities utilized include evaluation of intracranial pressure along with cerebral perfusion pressure to determine adequate blood flow; continuous electroencephalography to protect the patient from seizures and to identify early functional manifestations of ischemia and toxicity; transcranial Doppler evaluation for bedside review of circulatory adequacy; tissue oxygen monitoring to establish that brain tissue is receiving adequate oxygen from blood flow; and microdialysis to evaluate the metabolic function of the tissue in areas of concern. These monitors provide insights regarding specific aspects of brain tissue and overall brain function in the critically ill patient. Although recommendations continue to evolve for therapeutic targets for each of these modalities, an effective clinician may use each of these modalities to evaluate patients on an individual basis to improve the outcome of each patient, tailoring management to provide the care needed for any unique clinical presentation.
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Book chapters on the topic "Transcranial tissue Doppler"

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Tachtsidis, Ilias, Martin Tisdall, David T. Delpy, Martin Smith, and Clare E. Elwell. "Measurement of Cerebral Tissue Oxygenation in Young Healthy Volunteers During Acetazolamide Provocation: A Transcranial Doppler and Near-Infrared Spectroscopy Investigation." In Advances In Experimental Medicine And Biology, 389–96. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-74911-2_43.

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Wunna Htay, Soe. "Management of Traumatic Brain Injury." In Trauma and Emergency Surgery. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.98981.

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Head trauma or traumatic brain injury (TBI) is one of the most serious, life-threatening conditions in trauma victims. Prompt and appropriate therapy is essential to obtain a favorable outcome. The aim of the acute care of patients with brain injury is to optimize cerebral perfusion and oxygenation and to avoid secondary brain injury. Secondary brain injury develops with times and cause further damage to nervous tissues. The common denominators of secondary injury are cerebral hypoxia and ischemia. A systemic approach such as the Advanced Trauma Life Support (ATLS) algorithm has been recommended for managing head injury patients. Quick initial assessment of the patient’s neurologic condition thoroughly is mandatory. There should be attention in evidence of intrathoracic or intraperitoneal hemorrhage in multiple traumatized patients. Optimizing the open airway and adequate ventilation depending on patient’s neurologic condition is first step in emergency therapy. Cerebral perfusion pressure should be maintained between 50 and 70 mmHg. Systemic hypotension is one of the major contributors to poor outcome after head trauma. Careful stabilization of the blood pressure with fluid resuscitation and a continuous infusion of an inotrope or vasopressor may be necessary. Standard monitoring with direct arterial blood pressure monitoring and periodical measurement of arterial blood gases, hematocrit, electrolytes, glucose, and serum osmolarity are important. Brain monitoring as with an electroencephalogram, evoked potentials, jugular venous bulb oxygen saturation (Sjo2), flow velocity measured by transcranial Doppler (TCD), brain tissue oxygenation (btPo2), and ICP monitoring may be used. The reduction of elevated ICP by means of giving barbituates, hyperventilation, diuretics and hyperosmolar fluid therapy, body posture and incremental CSF drainage are critical. Seizure prophylaxis, early enteral feeding, stress ulcer prophylaxis, prevention of hyperglycemic state, fever and prophylaxis against deep venous thrombosis in neurointensive care unit are also important after successful resuscitation of head trauma patients.
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