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Relevant bibliographies by topics / Toxicodynamie

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Journal articles

Academic literature on the topic 'Toxicodynamie'

Author: Grafiati

Published: 22 June 2023

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Journal articles on the topic "Toxicodynamie"

1

Kasteel, Emma E. J., Sandra M. Nijmeijer, Keyvin Darney, et al. "Acetylcholinesterase inhibition in electric eel and human donor blood: an in vitro approach to investigate interspecies differences and human variability in toxicodynamics." Archives of Toxicology 94, no. 12 (2020): 4055–65. http://dx.doi.org/10.1007/s00204-020-02927-8.

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Abstract In chemical risk assessment, default uncertainty factors are used to account for interspecies and interindividual differences, and differences in toxicokinetics and toxicodynamics herein. However, these default factors come with little scientific support. Therefore, our aim was to develop an in vitro method, using acetylcholinesterase (AChE) inhibition as a proof of principle, to assess both interspecies and interindividual differences in toxicodynamics. Electric eel enzyme and human blood of 20 different donors (12 men/8 women) were exposed to eight different compounds (chlorpyrifos,

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2

Boak, Lauren M., Craig R. Rayner, M. Lindsay Grayson, et al. "Clinical Population Pharmacokinetics and Toxicodynamics of Linezolid." Antimicrobial Agents and Chemotherapy 58, no. 4 (2014): 2334–43. http://dx.doi.org/10.1128/aac.01885-13.

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ABSTRACTThrombocytopenia is a common side effect of linezolid, an oxazolidinone antibiotic often used to treat multidrug-resistant Gram-positive bacterial infections. Various risk factors have been suggested, including linezolid dose and duration of therapy, baseline platelet counts, and renal dysfunction; still, the mechanisms behind this potentially treatment-limiting toxicity are largely unknown. A clinical study was conducted to investigate the relationship between linezolid pharmacokinetics and toxicodynamics and inform strategies to prevent and manage linezolid-associated toxicity. Forty

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3

Blanchette, Alexander D., Sarah D. Burnett, Fabian A. Grimm, Ivan Rusyn, and Weihsueh A. Chiu. "A Bayesian Method for Population-wide Cardiotoxicity Hazard and Risk Characterization Using an In Vitro Human Model." Toxicological Sciences 178, no. 2 (2020): 391–403. http://dx.doi.org/10.1093/toxsci/kfaa151.

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Abstract Human induced pluripotent stem cell (iPSC)-derived cardiomyocytes are an established model for testing potential chemical hazards. Interindividual variability in toxicodynamic sensitivity has also been demonstrated in vitro; however, quantitative characterization of the population-wide variability has not been fully explored. We sought to develop a method to address this gap by combining a population-based iPSC-derived cardiomyocyte model with Bayesian concentration-response modeling. A total of 136 compounds, including 54 pharmaceuticals and 82 environmental chemicals, were tested in

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4

Kavlock, Robert J., and Gabriel L. Plaa. "Session summary: toxicodynamic interactive mechanisms." Toxicology 105, no. 2-3 (1995): 235–36. http://dx.doi.org/10.1016/0300-483x(95)03218-5.

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5

Ashauer, Roman, and Colin Brown. "TOXICODYNAMIC ASSUMPTIONS IN ECOTOXICOLOGICAL HAZARD MODELS." Environmental Toxicology and Chemistry preprint, no. 2008 (2007): 1. http://dx.doi.org/10.1897/07-642.

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6

Ashauer, Roman, and Colin D. Brown. "TOXICODYNAMIC ASSUMPTIONS IN ECOTOXICOLOGICAL HAZARD MODELS." Environmental Toxicology and Chemistry 27, no. 8 (2008): 1817. http://dx.doi.org/10.1897/07-642.1.

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7

Serkova, Natalie J., and Uwe Christians. "Biomarkers for Toxicodynamic Monitoring of Immunosuppressants." Therapeutic Drug Monitoring 27, no. 6 (2005): 733–37. http://dx.doi.org/10.1097/01.ftd.0000179846.30342.65.

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8

Gots, Ronald E., and Suellen W. Pirages. "Multiple Chemical Sensitivities: Psychogenic or Toxicodynamic Origins." International Journal of Toxicology 18, no. 6 (1999): 393–400. http://dx.doi.org/10.1080/109158199225107.

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The multiple chemical sensitivity (MCS) phenomenon can cause significant dysfunction and symptomatology and presents a difficult challenge for patient management. Central to the MCS debate is whether this phenomenon results from a primary emotional response to perceived chemical exposures or from pathological interactions between chemicals and biological systems. Those who believe the latter argue that toxic interactions result in physiological impairment and that subsequent emotional problems derive from such impairment. Distinguishing between psychogenic (emotional) or a toxicodynamic (chemi

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9

Reeves, Andrew L. "Beryllium: Toxicological Research of the Last Decade." Journal of the American College of Toxicology 8, no. 7 (1989): 1307–13. http://dx.doi.org/10.3109/10915818909009122.

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10

Danilov-Danilyan, V. I., and O. M. Rozental. "Logistic Model of Population Toxicodynamics." Water Resources 49, no. 2 (2022): 231–39. http://dx.doi.org/10.1134/s0097807822020038.

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Abstract The effect of pollutant in water on a population of aquatic organisms as a function of exposure time is studied. Natural assumptions are formulated regarding the character of this process, primarily, the linear relationship between the rate of decrease in the population, on the one hand, and the population size and the number of organisms killed by intoxication, on the other hand. The formulated assumptions are used to construct a model of population toxicodynamics, which describes the kinetics of suppression of the population by a logistic function. The results of model calculations

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